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[PMID]: 23536950 [Au] Autor: Smirnov AV; Nesterova OB; Suglobova ED; Golubev RV; Vasil'ev AN; Vasil'eva IA; Verbitskaia EV; Korosteleva NIu; Kostereva EM; Lebedeva EB; Levykina EN; Starosel'skii KG; Lazeba VA [Ti] Título: [Clinical and laboratory evaluation of the efficiency of chronic hemodialysis treatment using acidosuccinate in patients with terminal renal failure]. [So] Fuente: Ter Arkh;85(1):69-75, 2013. [Is] ISSN: 0040-3660 [Cp] País de publicación: Russia (Federation) [La] Idioma: rus [Ab] Resumen: AIM: To evaluate the efficiency of using a succinate-containing dialysis solution (SCDS) in terminal renal failure patients treated with chronic hemodialysis (CHD). SUBJECTS AND METHODS: Ninety patients from two hemodialysis units took part in the crossover study and were allocated to 2 groups. For 6 months, study group patients received CHD using SCDS and control group patients had CHD with a standard bicarbonate dialysis solution after 3-month washout period followed by decussation. The time course of changes in blood biochemical parameters, 24-hour ECG monitoring data, and quality of life indicators were estimated in the patients. RESULTS: After using acidosuccinate during hemodialysis, there was a significant reduction in the predialysis serum level of inorganic phosphate, a calcium phosphate product, gamma-glutamyl transpeptidase, urea, and aldosterone as compared to the control group. The blood concentration of total protein was also increased. After 6-month administration of acidosuccinate, the patients showed reductions in systolic blood pressure, heart rate, and the frequency and duration of ST-segment depression episodes. There were positive changes in the quality of life of patients according to the KDQOL-SF questionnaire. CONCLUSION: The use of SCDS in patients with CHD causes positive changes in a number of laboratory parameters and improves the physical and general status, and quality of life of patients. [Mh] Términos MeSH primario: Acetatos/farmacología Marcadores Biológicos/sangre Soluciones para Diálisis/farmacología Tasa de Filtración Glomerular Fallo Renal Crónico/terapia Diálisis Renal/métodos Ácido Succínico/farmacología [Mh] Términos MeSH secundario: Estudios Cruzados Femenino Estudios de Seguimiento Humanos Fallo Renal Crónico/sangre Fallo Renal Crónico/fisiopatología Masculino Mediana Edad Estudios Prospectivos Calidad de Vida Resultado del Tratamiento [Pt] Tipo de publicación: COMPARATIVE STUDY; ENGLISH ABSTRACT; JOURNAL ARTICLE; MULTICENTER STUDY; RANDOMIZED CONTROLLED TRIAL [Nm] Nombre de substancia: 0 (Acetates); 0 (Biological Markers); 0 (Dialysis Solutions); 110-15-6 (Succinic Acid) [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 130328 [St] Status: MEDLINE

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[PMID]: 23530302 [Au] Autor: Ricardo AC; Hacker E; Lora CM; Ackerson L; DeSalvo KB; Go A; Kusek JW; Nessel L; Ojo A; Townsend RR; Xie D; Ferrans CE; Lash JP; CRIC Investigators [Ad] Dirección: Department of Medicine, Section of Nephrology, University of Illinois at Chicago, USA. aricar2@uic.edu [Ti] Título: Validation of the Kidney Disease Quality of Life Short Form 36 (KDQOL-36) US Spanish and English versions in a cohort of Hispanics with chronic kidney disease. [So] Fuente: Ethn Dis;23(2):202-9, 2013. [Is] ISSN: 1049-510X [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: OBJECTIVE: Evaluate the reliability and validity of the Kidney Disease Quality of Life Short Form 36 (KDQOL-36) in Hispanics with mild-to-moderate chronic kidney disease (CKD). DESIGN: Cross-sectional SETTING: Chronic Renal Insufficiency Cohort Study PARTICIPANTS: 420 Hispanic (150 English- and 270 Spanish-speakers), and 409 non-Hispanic White individuals, matched by age (mean 57 years), sex (60% male), kidney function (mean estimated glomerular filtration rate 36ml/min/1.73m2), and diabetes (70%). METHODS: To measure construct validity, we selected instruments, comorbidities, and laboratory tests related to at least one KDQOL-36 subscale. Reliability was determined by calculating Cronbach's alpha. RESULTS: Reliability of each KDQOL-36 subscale [SF-12 Physical Component Summary (PCS) and Mental Component Summary (MCS), Symptoms/Problems, Burden of Kidney Disease and Effects of Kidney Disease] was very good (Cronbach's alpha >0.8). Construct validity was supported by expected negative correlation between MCS scores and the Beck Depression Inventory in all three subgroups (r=-0.56 to -0.61, P<.0001). There was inverse correlation between the Symptoms/ Problems subscale and the Patient Symptom Form (r= -0.70 to -0.77, P<.0001). We also found significant, positive correlation between the PCS score and a physical activity survey (r=+0.29 to +0.38, P< or =.003); and between the PCS and MCS scores and the Kansas City Questionnaire (r= +0.31 to +0.64, P<.0001). Reliability and validity were similar across all racial/ethnic groups analyzed separately. CONCLUSION: Our findings support the use of the KDQOL-36 as a measure of HRQOL in this cohort of US Hispanics with CKD. [Mh] Términos MeSH primario: Calidad de Vida Insuficiencia Renal Crónica [Mh] Términos MeSH secundario: Anciano Estudios de Cohortes Estudios Transversales Femenino Indicadores de Salud Humanos Lenguaje Masculino Mediana Edad Estados Unidos [Pt] Tipo de publicación: JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL; RESEARCH SUPPORT, NON-U.S. GOV'T; VALIDATION STUDIES [Em] Mes de ingreso: 1304 [Cu] Fecha actualización por clase: 130513 [Lr] Fecha última revisión: 130513 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 130327 [St] Status: MEDLINE

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[PMID]: 23516853 [Au] Autor: Kharlamova UV; Il'icheva OE [Ti] Título: [Determinants of vascular wall stiffness in patients with chronic renal disease undergoing hemodialysis]. [So] Fuente: Klin Med (Mosk);90(11):44-7, 2012. [Is] ISSN: 0023-2149 [Cp] País de publicación: Russia (Federation) [La] Idioma: rus [Ab] Resumen: Examination of 109 patients with chronic renal disease undergoing hemodialysis revealed significant impairment of arterial wall distensibility (accordingly, decreased Peterson's and Young's elastic moduli, distensibility coefficient). The relative thickness of the common carotid artery and pulse wave velocity were significantly greater than in practically healthy subjects. Independent factors influencing arterial wall rigidity included age, arterial pressure, total cholesterol and homocystein, stable metabolites of nitric oxide, creatinine, calcium, phosphorus levels, calcium x phosphorus product, duration of hemodialysis, interdialytic weight gain. [Mh] Términos MeSH primario: Enfermedades Cardiovasculares/etiología Arteria Carótida Común/fisiopatología Fallo Renal Crónico/complicaciones Diálisis Renal Rigidez Vascular/fisiología [Mh] Términos MeSH secundario: Enfermedades Cardiovasculares/epidemiología Enfermedades Cardiovasculares/fisiopatología Progresión de la Enfermedad Elasticidad Femenino Humanos Incidencia Fallo Renal Crónico/fisiopatología Fallo Renal Crónico/terapia Masculino Mediana Edad Pronóstico Factores de Riesgo Federación de Rusia/epidemiología [Pt] Tipo de publicación: COMPARATIVE STUDY; ENGLISH ABSTRACT; JOURNAL ARTICLE [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 130322 [St] Status: MEDLINE

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[PMID]: 23513360 [Au] Autor: Oppenheimer M; Jensen R; Huntington MK [Ad] Dirección: Oppenheimer Endocrinology, Sioux Falls, SD, USA. [Ti] Título: When 7 isn't 7: glycohemoglobin and chronic kidney disease. [So] Fuente: S D Med;66(2):59-61, 2013 Feb. [Is] ISSN: 0038-3317 [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: Glycohemoglobin level (A1c) is widely viewed as the gold standard for assessing glycemic control. Clinical decisions are based upon it, and reimbursement is increasingly tied to it. However, there are a number of conditions which result in loss of correlation between A1c and mean blood glucose levels. Chronic kidney disease (CKD) is one condition, prevalent as a comorbidity with diabetes, that can result in an inaccurate impression of glycemic control based on measured A1c levels. We review the glycation of hemoglobin, how it is affected in CKD, and review alternative methods for the assessment of glycemic control in these patients. [Mh] Términos MeSH primario: Glucemia/metabolismo Diabetes Mellitus Tipo 2/complicaciones Nefropatías Diabéticas/complicaciones Hemoglobina A Glucosilada/metabolismo Insuficiencia Renal Crónica/sangre [Mh] Términos MeSH secundario: Marcadores Biológicos/sangre Diabetes Mellitus Tipo 2/sangre Nefropatías Diabéticas/sangre Humanos [Pt] Tipo de publicación: JOURNAL ARTICLE; REVIEW [Nm] Nombre de substancia: 0 (Biological Markers); 0 (Blood Glucose); 0 (Hemoglobin A, Glycosylated) [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 130320 [St] Status: MEDLINE

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[PMID]: 23449698 [Au] Autor: Lamb EJ; Levey AS; Stevens PE [Ad] Dirección: Clinical Biochemistry, East Kent Hospitals University NHS Foundation Trust, Canterbury, UK. elamb@nhs.net [Ti] Título: The Kidney Disease Improving Global Outcomes (KDIGO) guideline update for chronic kidney disease: evolution not revolution. [So] Fuente: Clin Chem;59(3):462-5, 2013 Mar. [Is] ISSN: 1530-8561 [Cp] País de publicación: United States [La] Idioma: eng [Mh] Términos MeSH primario: Guías de Práctica Clínica como Asunto Insuficiencia Renal Crónica/terapia [Mh] Términos MeSH secundario: Albuminuria/orina Creatinina/orina Tasa de Filtración Glomerular Humanos Insuficiencia Renal Crónica/fisiopatología Insuficiencia Renal Crónica/orina [Pt] Tipo de publicación: JOURNAL ARTICLE [Nm] Nombre de substancia: 60-27-5 (Creatinine) [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 130301 [St] Status: MEDLINE [do] DOI: 10.1373/clinchem.2012.184259

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[PMID]: 23267748 [Au] Autor: Jamal SA; Miller PD [Ad] Dirección: University of Toronto, Women's College Research Institute, Toronto, Ontario, Canada. sophie.jamal@utoronto.ca [Ti] Título: Secondary and tertiary hyperparathyroidism. [So] Fuente: J Clin Densitom;16(1):64-8, 2013 Jan-Mar. [Is] ISSN: 1094-6950 [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: We reviewed the etiology and management of secondary and tertiary hyperparathyroidism. Secondary hyperparathyroidism is characterized by an increase in parathyroid hormone (PTH) that is appropriate and in response to a stimulus, most commonly low serum calcium. In secondary hyperparathyroidism, the serum calcium is normal and the PTH level is elevated. Tertiary hyperparathyroidism is characterized by excessive secretion of PTH after longstanding secondary hyperparathyroidism, in which hypercalcemia has ensued. Tertiary hyperparathyroidism typically occurs in men and women with chronic kidney disease usually after kidney transplant. The etiology and treatment of secondary hyperparathyroidism is relatively straightforward whereas data on the management of tertiary hyperparathyroidism is limited to a few small trials with short follow-up. [Mh] Términos MeSH primario: Hiperparatiroidismo [Mh] Términos MeSH secundario: Calcimiméticos/uso terapéutico Femenino Humanos Hiperparatiroidismo/clasificación Hiperparatiroidismo/diagnóstico Hiperparatiroidismo/quimioterapia Hiperparatiroidismo/etiología Hiperparatiroidismo/terapia Hiperparatiroidismo Secundario/quimioterapia Hiperparatiroidismo Secundario/etiología Hiperparatiroidismo Secundario/fisiopatología Hiperplasia Trasplante de Riñón Masculino Glándulas Paratiroides/patología Hormona Paratiroidea/secreción Insuficiencia Renal Crónica/complicaciones Insuficiencia Renal Crónica/metabolismo [Pt] Tipo de publicación: JOURNAL ARTICLE; REVIEW [Nm] Nombre de substancia: 0 (Calcimimetic Agents); 0 (Parathyroid Hormone) [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 130204 [St] Status: MEDLINE

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[PMID]: 22968085 [Au] Autor: Vucic Lovrencic M; Radisic Biljak V; Bozicevic S; Prasek M; Pavkovic P; Knotek M [Ad] Dirección: Institute of Clinical Chemistry and Laboratory Medicine, Merkur Teaching Hospital, Zajceva 19, 10000 Zagreb, Croatia. vucic@idb.hr [Ti] Título: Estimating glomerular filtration rate (GFR) in diabetes: the performance of MDRD and CKD-EPI equations in patients with various degrees of albuminuria. [So] Fuente: Clin Biochem;45(18):1694-6, 2012 Dec. [Is] ISSN: 1873-2933 [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: OBJECTIVES: The aim of this study was to compare the performance of Chronic Kidney Disease Epidemiology Collaboration (CKD-EPI) and Modification of Diet in Renal Disease Study (MDRD) equations in estimating GFR in a large cohort of diabetic patients with various degrees of albuminuria. DESIGN AND METHODS: In a group of 842 diabetic patients GFR was estimated from standardized creatinine, with MDRD-Study and CKD-EPI equations, and their performance evaluated regarding clinical stages of albuminuria and chronic kidney disease (CKD). RESULTS: Patients with normoalbuminuria had higher eGFR when calculated by CKD-EPI, than MDRD-Study equation [median (IQR): 103 (91-115) vs 97 (85-113)mL/min/1.73 m(2), P=0.006, n=364], which significantly influenced the prevalence of stage 1 CKD [eGFR>90 mL/min/1.73 m(2): 76.7% (CKD-EPI) vs. 65.1% (MDRD-Study), P=0.005]. There were no differences between the eGFR values derived by two equations in patients with micro- and macroalbuminuria, and more advanced staging of CKD. CONCLUSION: CKD-EPI equation might be a superior surrogate marker of GFR in patients with normoalbuminuria and hyperfiltration and could be used as a screening tool for early renal impairment in diabetes. It's validity as a marker of progression of diabetic nephropathy merits further investigation. [Mh] Términos MeSH primario: Albuminuria/complicaciones Albuminuria/fisiopatología Diabetes Mellitus/fisiopatología Dieta Tasa de Filtración Glomerular/fisiología Insuficiencia Renal Crónica/epidemiología Insuficiencia Renal Crónica/fisiopatología [Mh] Términos MeSH secundario: Adulto Anciano Femenino Humanos Masculino Mediana Edad Insuficiencia Renal Crónica/complicaciones [Pt] Tipo de publicación: JOURNAL ARTICLE [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 121203 [St] Status: MEDLINE

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[PMID]: 22935566 [Au] Autor: Kim JK; Kim SG; Kim HJ; Song YR [Ad] Dirección: Department of Internal Medicine, Hallym University Sacred Heart Hospital, Anyang, Republic of Korea. [Ti] Título: Serum S100B protein is associated with depressive symptoms in patients with end-stage renal disease. [So] Fuente: Clin Biochem;45(18):1573-7, 2012 Dec. [Is] ISSN: 1873-2933 [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: OBJECTIVES: Depression is associated with a poorer prognosis in patients with end-stage renal disease (ESRD). Increasing evidence indicates that glial pathology and blood-brain-barrier (BBB) dysfunction are involved in the pathophysiology of depression. S100B, a protein expressed in astro- and oligodendroglia in the human brain is considered a biomarker of depression. Our objective was to investigate the relationship between S100B and depressive symptoms in patients undergoing hemodialysis (HD). DESIGN AND METHODS: Seventy-eight Korean patients undergoing chronic HD without significant neurological issues participated in a cross-sectional observation study. Depressive symptoms were assessed with the Beck Depression Inventory-II (BDI-II), and serum S100B levels were measured using blood samples obtained prior to a mid-week HD session. RESULTS: The mean age of patients was 59.0 years, and the mean dialysis duration was 51.7 months. About 45% of patients undergoing HD met criteria for depression (BDI-II≥20). Serum S100B levels were significantly higher in patients with depression compared with patients without depression (115.1±45.4 vs. 66.1±35.3 pg/mL, p<0.001). S100B (r=0.556, p<0.001) and high-sensitivity C-reactive protein (hs-CRP; r=0.422, p<0.001) and ß2-microglobulin (r=0.391, p<0.001) levels were positively correlated with BDI-II scores. A multivariate regression analysis showed that both S100B and hs-CRP were significantly associated with BDI-II scores. CONCLUSIONS: The results showed a close association between S100B and depressive symptoms in patients undergoing HD. However, the mechanisms underlying this relationship are currently unknown and warrant further investigation. [Mh] Términos MeSH primario: Depresión/sangre Depresión/complicaciones Fallo Renal Crónico/sangre Fallo Renal Crónico/complicaciones Factores de Crecimiento Nervioso/sangre Proteínas S100/sangre [Mh] Términos MeSH secundario: Adulto Anciano Anciano de 80 o más Años Proteína C-Reactiva/metabolismo Femenino Humanos Modelos Lineales Masculino Mediana Edad Escalas de Valoración Psiquiátrica Adulto Joven Microglobulina-2 beta/sangre [Pt] Tipo de publicación: JOURNAL ARTICLE [Nm] Nombre de substancia: 0 (Nerve Growth Factors); 0 (S-100 calcium-binding protein beta subunit); 0 (S100 Proteins); 0 (beta 2-Microglobulin); 9007-41-4 (C-Reactive Protein) [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 121203 [St] Status: MEDLINE

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[PMID]: 22994862 [Au] Autor: Ruebner RL; Reese PP; Denburg MR; Rand EB; Abt PL; Furth SL [Ad] Dirección: Department of Pediatrics, Division of Nephrology, Children's Hospital of Philadelphia, Philadelphia, PA, USA. ruebnerr@email.chop.edu [Ti] Título: Risk factors for end-stage kidney disease after pediatric liver transplantation. [So] Fuente: Am J Transplant;12(12):3398-405, 2012 Dec. [Is] ISSN: 1600-6143 [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: Adult liver transplant (LT) recipients commonly develop advanced kidney disease. However, burden of end-stage kidney disease (ESKD) after pediatric LT has not been well-described. We performed a retrospective cohort study of pediatric LTs in the United States from 1990 to 2010. Multivariable Cox regression models were fit to determine risk factors for ESKD and death. Eight thousand nine hundred seventy six children received LTs. During median follow-up of 7.8 years, 2005 (22%) subjects died (mortality rate 26.1 cases/1000 person-years); 167 (2%) developed ESKD (incidence rate 2.2 cases/1000 person-years). Risk factors for ESKD included older age at LT (highest risk age >15 vs. < 5 years, HR = 4.94, p < 0.001), hepatitis C (HR 2.79, p = 0.004), liver re-transplant (HR 2.67, p < 0.001), eGFR pre-LT < 60 versus ≥ 60 (HR 2.37, p < 0.001), hepatitis B (HR 2.25, p = 0.027), black race (HR 1.46, p = 0.046), and male sex (HR 1.44, p = 0.022). LT recipients with ESKD had increased risk of mortality (HR 2.37, p < 0.001). Among pediatric LT recipients, rate of ESKD was lower than among adults and far exceeded by rate of death, however follow-up time in this study may underestimate lifetime burden of ESKD. Although uncommon, ESKD was highly associated with mortality. Pediatric LT recipients should be routinely monitored for kidney disease, particularly those at highest risk of ESKD. [Mh] Términos MeSH primario: Fallo Renal Crónico/mortalidad Trasplante de Riñón/mortalidad Trasplante de Hígado/mortalidad [Mh] Términos MeSH secundario: Adulto Niño Preescolar Femenino Humanos Incidencia Lactante Fallo Renal Crónico/epidemiología Fallo Renal Crónico/etiología Trasplante de Riñón/efectos adversos Trasplante de Hígado/efectos adversos Masculino Philadelphia/epidemiología Pronóstico Estudios Retrospectivos Factores de Riesgo Tasa de Supervivencia [Pt] Tipo de publicación: COMPARATIVE STUDY; JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 121203 [St] Status: MEDLINE [do] DOI: 10.1111/j.1600-6143.2012.04270.x

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[PMID]: 22700181 [Au] Autor: Matsumoto T; Fukushima S; Kanasaki T; Hagino S [Ad] Dirección: Bioengineering Division, Osaka University Graduate School of Engineering Science, 1-3 Machikaneyama-cho, Toyonaka, 560-8531, Japan. matsu@me.es.osaka-u.ac.jp [Ti] Título: Relationship between aortic mineral elements and osteodystrophy in mice with chronic kidney disease. [So] Fuente: Biol Trace Elem Res;150(1-3):278-84, 2012 Dec. [Is] ISSN: 1559-0720 [Cp] País de publicación: United States [La] Idioma: eng [Ab] Resumen: In chronic kidney disease (CKD), osteodystrophy and arterial calcification often coexist. However, arterial alterations have not been addressed in CKD unaccompanied by evidence of calcification. We investigated the association of phosphate (P) and calcium (Ca) accumulation in calcification-free aortas with CKD-induced osteodystrophy. Aortic accumulation of magnesium (Mg), an inhibitor of calcification, was also examined. Male mice aged 26 weeks with CKD characterized by hyperparathyroidism and hyperphosphatemia (Nx, n = 8) and age-matched healthy male mice (shams, n = 8) were sampled for blood, and thoracic vertebrae and aortas were harvested. Bone structure and chemicals were analyzed by microcomputed tomography and infrared microspectroscopy, respectively, and aortic accumulation of P, Ca, and Mg was evaluated by plasma-atomic emission spectrometry. Volume fractions of cortical and trabecular bones were smaller in Nx than in sham animals (P < 0.05), attributed to cortical thinning and reduction in trabecular number, respectively. Bone chemicals were not different between the groups. No calcification was found in either group, but P, Ca, and Mg contents were higher in Nx than in shams (P < 0.05). The mass ratio of Ca/P was lower in Nx than in shams (P < 0.05), but that of Mg/Ca and Mg/P was not different between the groups. Aortic P and Ca contents were inversely correlated with the volume fraction of cortical bone (P < 0.05). In conclusion, the relationship of osteodystrophy with aortic P and Ca accumulation suggests the existence of a bone-vascular axis, even in calcification-free arteries in CKD. The preservation of ratios of Mg/Ca and Mg/P despite CKD development might contribute to calcification resistance. [Mh] Términos MeSH primario: Aorta Torácica/química Calcio/análisis Fósforo/análisis Insuficiencia Renal Crónica/fisiopatología Osteodistrofia Renal/fisiopatología Vértebras Torácicas/patología Calcificación Vascular/etiología [Mh] Términos MeSH secundario: Animales Aorta Torácica/metabolismo Aorta Torácica/patología Calcio/metabolismo Resistencia a la Enfermedad Diagnóstico Precoz Hiperparatiroidismo Secundario/etiología Hiperfosfatemia/etiología Magnesio/análisis Magnesio/metabolismo Masculino Ratones Ratones Consanguíneos ICR Fósforo/metabolismo Osteodistrofia Renal/etiología Osteodistrofia Renal/metabolismo Osteodistrofia Renal/radiografía Índice de Severidad de la Enfermedad Espectrofotometría Atómica Espectrofotometría Infrarroja Vértebras Torácicas/química Vértebras Torácicas/radiografía Tomografía Computarizada por Rayos X Calcificación Vascular/diagnóstico Calcificación Vascular/prevención & control [Pt] Tipo de publicación: JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T [Nm] Nombre de substancia: 7439-95-4 (Magnesium); 7440-70-2 (Calcium); 7723-14-0 (Phosphorus) [Em] Mes de ingreso: 1305 [Sb] Subgrupo de revista: IM [Da] Fecha de ingreso para procesamiento: 121130 [St] Status: MEDLINE [do] DOI: 10.1007/s12011-012-9466-x

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