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  1 / 2182 MEDLINE  
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[PMID]:29187691
[Au] Autor:Fujiwara S; Yoshimura H; Nishiya K; Oshima K; Kawamoto M; Kohara N
[Ad] Dirección:Department of Neurology, Kobe City Medical Center General Hospital.
[Ti] Título:[Tapia's syndrome following transesophageal echocardiography during an open-heart operation: a case report].
[So] Fuente:Rinsho Shinkeigaku;57(12):785-787, 2017 Dec 27.
[Is] ISSN:1882-0654
[Cp] País de publicación:Japan
[La] Idioma:jpn
[Ab] Resumen:A 67-year-old man presented with hoarseness, dysarthria and deviation of the tongue to the left side the day after the open-heart operation under general anesthesia. Brain MRI demonstrated no causal lesion, and laryngoscope showed left vocal cord abductor palsy, so we diagnosed him with Tapia's syndrome (i.e., concomitant paralysis of the left recurrent and hypoglossal nerve). His neurological symptoms recovered gradually and improved completely four months after the onset. Tapia's syndrome is a rare condition caused by the extra cranial lesion of the recurrent laryngeal branch of the vagus nerve and the hypoglossal nerve, and mostly described as a complication of tracheal intubation. In this case, transesophageal echo probe has been held in the left side of the pharynx, so compression to the posterior wall of pharynx by the probe resulted in this condition, and to the best of our knowledge, this is the first report of Tapia's syndrome due to transesophageal echocardiography during an open-heart operation. This rare syndrome should be considered as a differential diagnosis of dysarthria and tongue deviation after a procedure associated with compression to the pharynx.
[Mh] Términos MeSH primario: Procedimientos Quirúrgicos Cardíacos
Ecocardiografía Transesofágica/efectos adversos
Enfermedades del Nervio Hipogloso/etiología
Complicaciones Intraoperatorias/etiología
Parálisis de los Pliegues Vocales/etiología
[Mh] Términos MeSH secundario: Anciano
Diagnóstico Diferencial
Disartria/etiología
Ecocardiografía Transesofágica/instrumentación
Seres Humanos
Enfermedades del Nervio Hipogloso/diagnóstico
Masculino
Síndrome
Parálisis de los Pliegues Vocales/diagnóstico
[Pt] Tipo de publicación:CASE REPORTS; JOURNAL ARTICLE
[Em] Mes de ingreso:1801
[Cu] Fecha actualización por clase:180112
[Lr] Fecha última revisión:180112
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:171201
[St] Status:MEDLINE
[do] DOI:10.5692/clinicalneurol.cn-001097


  2 / 2182 MEDLINE  
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[PMID]:29187688
[Au] Autor:Abe S; Okazaki S; Tonomura S; Miyashita K; Ihara M
[Ad] Dirección:Department of Neurology, National Cerebral and Cardiovascular Center, Osaka.
[Ti] Título:[Progressive dysarthria and bilateral sensory disturbance in a case of bilateral ventrolateral pontine infarction].
[So] Fuente:Rinsho Shinkeigaku;57(12):764-768, 2017 Dec 27.
[Is] ISSN:1882-0654
[Cp] País de publicación:Japan
[La] Idioma:jpn
[Ab] Resumen:A rare case of bilateral ventrolateral pontine infarction in a 70-year-old man who developed progressive dysarthria and bilateral sensory disturbance is reported with literature review. He had been diagnosed with hypertension, dyslipidemia, and impaired glucose tolerance 10 years earlier. Ten days before admission, he was aware of the difficulty in walking and speaking, which gradually worsened. On admission he showed bilateral thermal hypoalgesia of face and lower extremities, dysarthria, dysphagia, and ataxic gait. High resolution three-dimensional MRI revealed bilateral ventrolateral pontine infarction with a large atherosclerotic plaque in the ventral side of the basilar artery, which led to a diagnosis of atherothrombotic brain infarction. The atherosclerotic plaque in the basilar artery was thought to be responsible for simultaneous occlusion of the bilateral short circumflex arteries of the pons.
[Mh] Términos MeSH primario: Infarto Cerebral/etiología
Disartria/etiología
Puente/irrigación sanguínea
Trastornos de la Sensación/etiología
[Mh] Términos MeSH secundario: Anciano
Arteria Basilar/diagnóstico por imagen
Infarto Cerebral/diagnóstico por imagen
Progresión de la Enfermedad
Seres Humanos
Imagen por Resonancia Magnética
Masculino
Placa Aterosclerótica/complicaciones
Placa Aterosclerótica/diagnóstico por imagen
Puente/diagnóstico por imagen
[Pt] Tipo de publicación:CASE REPORTS; JOURNAL ARTICLE
[Em] Mes de ingreso:1801
[Cu] Fecha actualización por clase:180112
[Lr] Fecha última revisión:180112
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:171201
[St] Status:MEDLINE
[do] DOI:10.5692/clinicalneurol.cn-001090


  3 / 2182 MEDLINE  
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[PMID]:29240814
[Au] Autor:Godino-Llorente JI; Shattuck-Hufnagel S; Choi JY; Moro-Velázquez L; Gómez-García JA
[Ad] Dirección:Speech Communication Group, Research Laboratory of Electronics, Massachusetts Institute of Technology, Cambridge, Massachusetts, United States of America.
[Ti] Título:Towards the identification of Idiopathic Parkinson's Disease from the speech. New articulatory kinetic biomarkers.
[So] Fuente:PLoS One;12(12):e0189583, 2017.
[Is] ISSN:1932-6203
[Cp] País de publicación:United States
[La] Idioma:eng
[Ab] Resumen:Although a large amount of acoustic indicators have already been proposed in the literature to evaluate the hypokinetic dysarthria of people with Parkinson's Disease, the goal of this work is to identify and interpret new reliable and complementary articulatory biomarkers that could be applied to predict/evaluate Parkinson's Disease from a diadochokinetic test, contributing to the possibility of a further multidimensional analysis of the speech of parkinsonian patients. The new biomarkers proposed are based on the kinetic behaviour of the envelope trace, which is directly linked with the articulatory dysfunctions introduced by the disease since the early stages. The interest of these new articulatory indicators stands on their easiness of identification and interpretation, and their potential to be translated into computer based automatic methods to screen the disease from the speech. Throughout this paper, the accuracy provided by these acoustic kinetic biomarkers is compared with the one obtained with a baseline system based on speaker identification techniques. Results show accuracies around 85% that are in line with those obtained with the complex state of the art speaker recognition techniques, but with an easier physical interpretation, which open the possibility to be transferred to a clinical setting.
[Mh] Términos MeSH primario: Disartria/complicaciones
Enfermedad de Parkinson/fisiopatología
[Mh] Términos MeSH secundario: Anciano
Biomarcadores
Seres Humanos
Enfermedad de Parkinson/complicaciones
Acústica del Lenguaje
Inteligibilidad del Habla
[Pt] Tipo de publicación:JOURNAL ARTICLE
[Nm] Nombre de substancia:
0 (Biomarkers)
[Em] Mes de ingreso:1712
[Cu] Fecha actualización por clase:171229
[Lr] Fecha última revisión:171229
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:171215
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0189583


  4 / 2182 MEDLINE  
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[PMID]:29251341
[Au] Autor:Garcia-Estevez DA; Lopez-Diaz LM; Pardo-Parrado M; Cid-Rodriguez MC; Ozaita-Arteche G
[Ad] Dirección:Complexo Hospitalario de Ourense, Ourense, Espana.
[Ti] Título:[Isolated dysarthria and dysphagia as the presenting symptoms of simultaneous bilateral acute lagunar ischaemic stroke].
[Ti] Título:Disartria y disfagia aisladas como forma de presentacion de un ictus isquemico agudo lacunar bilateral y simultaneo..
[So] Fuente:Rev Neurol;66(1):33, 2018 Jan 01.
[Is] ISSN:1576-6578
[Cp] País de publicación:Spain
[La] Idioma:spa
[Pt] Tipo de publicación:JOURNAL ARTICLE
[Em] Mes de ingreso:1712
[Cu] Fecha actualización por clase:171218
[Lr] Fecha última revisión:171218
[St] Status:In-Data-Review


  5 / 2182 MEDLINE  
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[PMID]:28686046
[Au] Autor:Vesey S
[Ad] Dirección:Clinical Lead Speech and language Therapist, Pennine Care NHS Foundation Trust Trafford General Hospital, Manchester.
[Ti] Título:The challenges of dysphagia in treating motor neurone disease.
[So] Fuente:Br J Community Nurs;22(Sup7):S17-S21, 2017 Jul 01.
[Is] ISSN:1462-4753
[Cp] País de publicación:England
[La] Idioma:eng
[Ab] Resumen:Motor neurone disease (MND) is a relatively rare degenerative disorder. Its impacts are manifested in progressive loss of motor function and often accompanied by wider non-motor changes. Swallowing and speech abilities are frequently severely impaired. Effective management of dysphagia (swallowing difficulty) symptoms and nutritional care requires a holistic multidisciplinary approach. Care must be patient focused, facilitate patient decision making, and support planning towards end of life care. This article discusses the challenges of providing effective nutritional care to people living with motor neurone disease who have dysphagia.
[Mh] Términos MeSH primario: Trastornos de Deglución/enfermería
Enfermedad de la Neurona Motora/enfermería
[Mh] Términos MeSH secundario: Trastornos de Deglución/etiología
Trastornos de Deglución/fisiopatología
Disartria/etiología
Disartria/fisiopatología
Disfonía/etiología
Disfonía/fisiopatología
Nutrición Enteral
Gastrostomía
Seres Humanos
Enfermedad de la Neurona Motora/complicaciones
Enfermedad de la Neurona Motora/fisiopatología
Calidad de Vida
Cuidado Terminal
[Pt] Tipo de publicación:JOURNAL ARTICLE
[Em] Mes de ingreso:1709
[Cu] Fecha actualización por clase:170926
[Lr] Fecha última revisión:170926
[Sb] Subgrupo de revista:N
[Da] Fecha de ingreso para procesamiento:170708
[St] Status:MEDLINE
[do] DOI:10.12968/bjcn.2017.22.Sup7.S17


  6 / 2182 MEDLINE  
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[PMID]:28601293
[Au] Autor:Wallwitz U; Brock S; Schunck A; Wildemann B; Jarius S; Hoffmann F
[Ad] Dirección:Department of Neurology, Krankenhaus Martha-Maria Halle-Dölau, Röntgenstraße 1, 06120, Halle/Saale, Germany.
[Ti] Título:From dizziness to severe ataxia and dysarthria: New cases of anti-Ca/ARHGAP26 autoantibody-associated cerebellar ataxia suggest a broad clinical spectrum.
[So] Fuente:J Neuroimmunol;309:77-81, 2017 Aug 15.
[Is] ISSN:1872-8421
[Cp] País de publicación:Netherlands
[La] Idioma:eng
[Ab] Resumen:In 2010, a novel anti-neuronal autoantibody, termed anti-Ca, was described in a patient with subacute cerebellar ataxia, and Rho GTPase-activating protein 26 (ARHGAP26) was identified as the target antigen. Recently, three additional cases of anti-Ca-positive cerebellar ataxia have been published. In addition to ataxia, cognitive decline and depression have been observed in some patients. Here, we report two new cases of anti-Ca-associated autoimmune cerebellar ataxia. Patient 1 presented with dizziness and acute yet mild limb and gait ataxia. Symptoms stabilized with long-term oral corticosteroid therapy but transiently worsened when steroids were tapered. Interestingly, both initial occurrence and worsening of the patient's neurological symptoms after steroid withdrawal were accompanied by spontaneous cutaneous hematomas. Patient 2 initially presented with an increased startle response and myoclonic jerks, and subsequently developed severe limb and gait ataxia, dysarthria, oculomotor disturbances, head and voice tremor, dysphagia, cognitive symptoms and depression. Steroid treatment was started five years after disease onset. The symptoms then responded only poorly to corticosteroids. At most recent follow-up, 19 years after disease onset, the patient was wheelchair-bound. These cases extend the clinical spectrum associated with anti-ARHGAP26 autoimmunity and suggest that early treatment may be important in patients with this rare syndrome.
[Mh] Términos MeSH primario: Autoanticuerpos/sangre
Ataxia Cerebelosa/sangre
Mareo/sangre
Disartria/sangre
Proteínas Activadoras de GTPasa/sangre
[Mh] Términos MeSH secundario: Adulto
Anciano
Ataxia Cerebelosa/complicaciones
Ataxia Cerebelosa/diagnóstico
Mareo/complicaciones
Mareo/diagnóstico
Disartria/complicaciones
Disartria/diagnóstico
Seres Humanos
Mediana Edad
Índice de Severidad de la Enfermedad
[Pt] Tipo de publicación:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nombre de substancia:
0 (ARHGAP26 protein, human); 0 (Autoantibodies); 0 (GTPase-Activating Proteins)
[Em] Mes de ingreso:1708
[Cu] Fecha actualización por clase:170821
[Lr] Fecha última revisión:170821
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:170612
[St] Status:MEDLINE


  7 / 2182 MEDLINE  
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[PMID]:28572273
[Au] Autor:Thakore NJ; Pioro EP
[Ad] Dirección:Department of Neurology, Neuromuscular Center, Neurological Institute, Cleveland Clinic, Cleveland, Ohio, USA.
[Ti] Título:Laughter, crying and sadness in ALS.
[So] Fuente:J Neurol Neurosurg Psychiatry;88(10):825-831, 2017 Oct.
[Is] ISSN:1468-330X
[Cp] País de publicación:England
[La] Idioma:eng
[Ab] Resumen:BACKGROUND: Pseudobulbar affect (PBA) is prevalent in amyotrophic lateral sclerosis (ALS), but there is limited information on its associations and course. OBJECTIVES: Explore prevalence, associations, course and manifestations of PBA in outpatient cohort of patients with ALS and examine its relationship to depression. METHODS: Self-reported measures of PBA and depression (Center for Neurologic Study-Lability Scale (CNS-LS) and Patient Health Questionnaire (PHQ-9), respectively) were obtained from consecutive patients with ALS using tablet devices in waiting rooms (Knowledge Program). RESULTS: PBA (CNS-LS ≥13) was seen in 209/735 patients (28.4%). PBA was associated with bulbar onset and dysfunction, upper motor neuron dysfunction, cognitive impairment, depression and lower quality of life. A multivariable model that included lower bulbar and gross motor subscores, female gender, younger age and shorter duration of disease predicted PBA with 74% accuracy. CNS-LS scores increased only slowly with time. Women with PBA reported more crying than men. Crying (but not laughter) correlated with depression, and crying was associated with poorer quality of life. Exploratory factor analysis of pooled questions of CNS-LS and PHQ-9 identified three underlying factors (laughter, crying and depression) loaded on appropriate questions of the respective instruments. CONCLUSION: This study identifies associations of PBA and additionally finds PBA (especially crying-predominant PBA) more prevalent in women with ALS. Although the two self-report instruments (CNS-LS and PHQ-9) discriminate well between PBA and depression, there is significant overlap between depression and crying in PBA. Studies of PBA should stratify for gender, examine crying and laughter as separate outcomes and adjust for depression.
[Mh] Términos MeSH primario: Esclerosis Amiotrófica Lateral/complicaciones
Llanto
Risa
Parálisis Seudobulbar/epidemiología
[Mh] Términos MeSH secundario: Factores de Edad
Depresión/psicología
Femenino
Seres Humanos
Masculino
Mediana Edad
Prevalencia
Calidad de Vida/psicología
Factores Sexuales
Encuestas y Cuestionarios
[Pt] Tipo de publicación:JOURNAL ARTICLE; OBSERVATIONAL STUDY
[Em] Mes de ingreso:1709
[Cu] Fecha actualización por clase:171114
[Lr] Fecha última revisión:171114
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:170603
[St] Status:MEDLINE
[do] DOI:10.1136/jnnp-2017-315622


  8 / 2182 MEDLINE  
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[PMID]:28552869
[Au] Autor:Tokida H; Shiga Y; Shimoe Y; Yamori S; Tanaka A; Kuriyama M
[Ad] Dirección:Department of Rehabilitation, Brain Attack Center Ota Memorial Hospital.
[Ti] Título:Foreign accent syndrome caused by the left precentral infarction-a case report.
[So] Fuente:Rinsho Shinkeigaku;57(6):293-297, 2017 06 28.
[Is] ISSN:1882-0654
[Cp] País de publicación:Japan
[La] Idioma:jpn
[Ab] Resumen:A 57-year-old right-handed man was admitted to our hospital because of right facial paresis and acute-onset dysarthria. He presented with non-fluent aphasia. His aphasia gradually improved, but he started speaking with a strange accent and intonation from the fifth hospital day. He was diagnosed with foreign accent syndrome (FAS), which lasted for 2 months. MRI revealed ischemic infarction with edema in the superior, middle, and inferior parts of the left precentral gyrus. One year later, MRI revealed old, small infarct lesions in the left precentral gyrus, middle frontal gyrus, and postcentral gyrus. We suspected that FAS developed because of disturbance of prosody in the speaking network on improving his aphasia. His meticulous character was probably influenced on developing FAS. The responsible lesions possibly were those in the reversible parts of the left precentral gyrus with edema on acute stage.
[Mh] Términos MeSH primario: Trastornos de la Articulación/etiología
Infarto Cerebral/complicaciones
[Mh] Términos MeSH secundario: Reacción de Fase Aguda
Afasia/etiología
Infarto Cerebral/diagnóstico por imagen
Infarto Cerebral/patología
Imagen de Difusión por Resonancia Magnética
Disartria/etiología
Edema
Parálisis Facial/etiología
Lóbulo Frontal/diagnóstico por imagen
Lóbulo Frontal/patología
Seres Humanos
Masculino
Mediana Edad
Personalidad/fisiología
Síndrome
[Pt] Tipo de publicación:CASE REPORTS; JOURNAL ARTICLE; REVIEW
[Em] Mes de ingreso:1709
[Cu] Fecha actualización por clase:171012
[Lr] Fecha última revisión:171012
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:170530
[St] Status:MEDLINE
[do] DOI:10.5692/clinicalneurol.cn-000988


  9 / 2182 MEDLINE  
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[PMID]:28443623
[Au] Autor:Hellebrekers DMEI; Sallevelt SCEH; Theunissen TEJ; Hendrickx ATM; Gottschalk RW; Hoeijmakers JGJ; Habets DD; Bierau J; Schoonderwoerd KG; Smeets HJM
[Ad] Dirección:Department of Clinical Genetics, Maastricht University Medical Centre (MUMC), Maastricht, The Netherlands.
[Ti] Título:Novel SLC25A32 mutation in a patient with a severe neuromuscular phenotype.
[So] Fuente:Eur J Hum Genet;25(7):886-888, 2017 Jun.
[Is] ISSN:1476-5438
[Cp] País de publicación:England
[La] Idioma:eng
[Ab] Resumen:In a 51-year-old patient of consanguineous parents with a severe neuromuscular phenotype of early-onset ataxia, myoclonia, dysarthria, muscle weakness and exercise intolerance, exome sequencing revealed a novel homozygous variant (c.-264_31delinsCTCACAAATGCTCA) in the mitochondrial FAD-transporter gene SLC25A32. Flavin adenine dinucleotide (FAD) is an essential co-factor for many mitochondrial enzymes and impaired mitochondrial FAD-transport was supported by a reduced oxidative phosphorylation complex II activity in the patient's muscle, decreased ATP production in fibroblasts, and a deficiency of mitochondrial FAD-dependent enzymes. Clinically, the patient showed improvement upon riboflavin treatment, which is a precursor of FAD. Our results confirm the recently reported case of SLC25A32 as a cause of riboflavin-responsive disease. Our patient showed a more severe clinical phenotype compared with the reported patient, corresponding with the (most likely) complete absence of the SLC25A32-encoding MFT (Mitochondrial Folate Transporter) protein.
[Mh] Términos MeSH primario: Ataxia/genética
Disartria/genética
Mutación INDEL
Proteínas de Transporte de Membrana/genética
Debilidad Muscular/genética
[Mh] Términos MeSH secundario: Ataxia/diagnóstico
Ataxia/tratamiento farmacológico
Células Cultivadas
Disartria/diagnóstico
Disartria/tratamiento farmacológico
Fibroblastos/metabolismo
Seres Humanos
Masculino
Mediana Edad
Debilidad Muscular/diagnóstico
Debilidad Muscular/tratamiento farmacológico
Fenotipo
Riboflavina/metabolismo
Riboflavina/uso terapéutico
Síndrome
Complejo Vitamínico B/metabolismo
Complejo Vitamínico B/uso terapéutico
[Pt] Tipo de publicación:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nombre de substancia:
0 (Membrane Transport Proteins); 0 (SLC25A32 protein, human); 12001-76-2 (Vitamin B Complex); TLM2976OFR (Riboflavin)
[Em] Mes de ingreso:1709
[Cu] Fecha actualización por clase:170928
[Lr] Fecha última revisión:170928
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:170427
[St] Status:MEDLINE
[do] DOI:10.1038/ejhg.2017.62


  10 / 2182 MEDLINE  
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[PMID]:28400397
[Au] Autor:Solis WG; Hansen M
[Ad] Dirección:Department of Neurosurgery, John Hunter Hospital, New Lambton, New South Wales, Australia waldo.g.solis@outlook.com.
[Ti] Título:Fluorescence in a cryptococcoma following administration of 5-aminolevulinic acid hydrochloride (Gliolan).
[So] Fuente:BMJ Case Rep;2017, 2017 Apr 11.
[Is] ISSN:1757-790X
[Cp] País de publicación:England
[La] Idioma:eng
[Ab] Resumen:A 54-year-old man presented with two episodes of dysarthria and left facial droop. Both episodes resolved by the time of examination. MRI of the brain revealed a right frontotemporal, heterogeneously enhancing mass with surrounding vasogenic oedema, suggestive of a high-grade primary brain neoplasm. The patient was administered preoperative 5-aminolevulinic acid hydrochloride (Gliolan), and fluorescence-guided resection of the lesion was undertaken. infection was diagnosed from the specimen and the patient was given appropriate antifungal treatment. This is the first reported case of Gliolan-mediated fluorescence in a fungal abscess and highlights one of the potential pitfalls in fluorescence-guided surgery.
[Mh] Términos MeSH primario: Ácido Aminolevulínico/administración & dosificación
Encéfalo/diagnóstico por imagen
Criptococosis/diagnóstico
Cryptococcus gattii/aislamiento & purificación
Fármacos Fotosensibilizantes/administración & dosificación
[Mh] Términos MeSH secundario: Antifúngicos/uso terapéutico
Criptococosis/tratamiento farmacológico
Diagnóstico Diferencial
Disartria/etiología
Seres Humanos
Imagen por Resonancia Magnética
Masculino
Mediana Edad
Cirugía Asistida por Computador
[Pt] Tipo de publicación:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nombre de substancia:
0 (Antifungal Agents); 0 (Photosensitizing Agents); 88755TAZ87 (Aminolevulinic Acid)
[Em] Mes de ingreso:1706
[Cu] Fecha actualización por clase:170606
[Lr] Fecha última revisión:170606
[Sb] Subgrupo de revista:IM
[Da] Fecha de ingreso para procesamiento:170413
[St] Status:MEDLINE



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