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[PMID]: 29188542
[Au] Autor:Tanabe N; Hiraoka E; Kataoka J; Naito T; Matsumoto K; Arai J; Norisue Y
[Ad] Address:Department of Internal Medicine, Tokyo Bay Urayasu Ichikawa Medical Center, 3-4-32, Todaijima, Urayasu-city, Chiba, 279-0001, Japan.
[Ti] Title:Wet Beriberi Associated with Hikikomori Syndrome.
[So] Source:J Gen Intern Med;33(3):384-387, 2018 Mar.
[Is] ISSN:1525-1497
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Wet beriberi, characterized by high cardiac output with predominantly right-sided heart failure and lactic acidosis, is a disease caused by thiamine deficiency, and is rarely seen in modern society. However, patients with social withdrawal syndrome, also known as hikikomori syndrome, may be a new population at risk of thiamine deficiency. Hikikomori syndrome, first recognized in Japan, is becoming a worldwide issue. A 39-year-old Japanese patient was brought to our hospital, with a 3-week history of progressive shortness of breath and generalized edema. The patient had right-sided high-output heart failure, lactic acidosis, and Wernicke-Korsakoff syndrome. Because of his history of social isolation, we diagnosed hikikomori syndrome according to the Japanese government's definition, which is as follows: lifestyle centered at home; no interest or willingness to attend school or work; persistence of symptoms beyond 6 months; and exclusion of other psychiatric and developmental disorders. Considering his diagnosis of hikikomori syndrome and social isolation, we suspected malnutrition, particularly thiamine deficiency, and successfully treated him. Clinicians should be aware of the potential risk of thiamine deficiency associated with hikikomori syndrome and initiate thiamine replacement in cases of high-output heart failure associated with lactic acidosis.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[St] Status:In-Data-Review
[do] DOI:10.1007/s11606-017-4208-6

  2 / 2145 MEDLINE  
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[PMID]: 29489643
[Au] Autor:Lei Y; Zheng MH; Huang W; Zhang J; Lu Y
[Ad] Address:Department of Emergency Medicine.
[Ti] Title:Wet beriberi with multiple organ failure remarkably reversed by thiamine administration: A case report and literature review.
[So] Source:Medicine (Baltimore);97(9):e0010, 2018 Mar.
[Is] ISSN:1536-5964
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:RATIONALE: Circulatory failure, especially with low systemic vascular resistance (SVR), as observed in septic shock, thyrotoxicosis, and anemia, is a particular pattern that should suggest thiamine (vitamin B1) deficiency. The clinical picture of wet beriberi secondary to thiamine deficiency only demonstrates non-specific clinical manifestations. For a diagnosis of wet beriberi, medical history is very important. Interestingly, imprisonment was also found to be related to thiamine deficiency. This article presents a rare case of wet beriberi associated with multiple organ failure (MOF) in a prison patient with years of heavy alcohol consumption. PATIENT CONCERNS: The patient reported repetitive symptoms of nausea, vomiting, respiratory distress, and palpitations for a period of 1 month; dyspnea and edema for 5 days; and decreased blood pressure and urine volume for 2 days. DIAGNOSES: The heart failure patient had a history of dietary deficiency. Right heart catheterization showed high cardiac output (CO) and low SVR. Measurement of serum thiamine concentration was low. The most important factor was that the hemodynamic indices were remarkably reversed by thiamine administration. INTERVENTIONS: The patient started treatment with thiamine (100 mg) by intramuscular injection, together with basic supportive care. OUTCOMES: The hemodynamic indices improved within 12 hours after thiamine administration. Echocardiographic examinations revealed right ventricular function improvement within a few days, which were normal within a month. LESSONS: A diagnosis of wet beriberi should be considered for a prison patient who has unexplained heart failure, lactic acidosis, and/or MOF. Moreover, the patient should be empirically given thiamine administration without delay.
[Mh] MeSH terms primary: Beriberi/complications
Beriberi/drug therapy
Multiple Organ Failure/complications
Multiple Organ Failure/drug therapy
Thiamine/therapeutic use
[Mh] MeSH terms secundary: Adult
Alcoholism/complications
Beriberi/etiology
Beriberi/physiopathology
Cardiac Output
Humans
Male
Multiple Organ Failure/etiology
Multiple Organ Failure/physiopathology
Prisoners
Thiamine Deficiency/complications
Thiamine Deficiency/drug therapy
Vascular Resistance
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE; REVIEW
[Nm] Name of substance:X66NSO3N35 (Thiamine)
[Em] Entry month:1803
[Cu] Class update date: 180305
[Lr] Last revision date:180305
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:180301
[St] Status:MEDLINE
[do] DOI:10.1097/MD.0000000000010010

  3 / 2145 MEDLINE  
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[PMID]: 29477220
[Au] Autor:Lonsdale D
[Ad] Address:Cleveland Clinic, Cleveland, OH, United States. Electronic address: derricklonsdale@hotmail.com.
[Ti] Title:Thiamin.
[So] Source:Adv Food Nutr Res;83:1-56, 2018.
[Is] ISSN:1043-4526
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Starting with a brief history of beriberi and the discovery that thiamin deficiency is its cause, the symptoms and signs are reviewed. None are pathognomonic. The disease has a low mortality and a long morbidity. The appearance of the patient can be deceptive, often being mistaken for psychosomatic disease in the early stages. The chemistry of thiamin and the laboratory methodology for depicting its deficiency are outlined. The diseases associated with thiamin deficiency, apart from malnutrition, include a number of genetically determined conditions where mutations, either in the cofactor relationship or a transporter, provide the etiology. It is emphasized that such mutations are often epigenetically responsive to megadoses of thiamin or one of its derivatives. The use of thiamin in clinical practice requires a high index of suspicion on the part of the clinician since it has a part to play in eating disorders, diabetes, neurodegenerative disease, and cancer. A high rate of critical illness and postsurgery thiamin deficiency have been reported, particularly those associated with gastrointestinal bypass. Emphasis is placed on thiamin deficiency as a major cause of asymmetric dysautonomia, because of the high degree of sensitivity to thiamin deficiency in the brainstem, cerebellum, and hypothalamus. The relationship of thiamin with regional pain syndrome, eosinophilic esophagitis, its analgesic capacity, and its preventive use in obstetrics is raised as a potential issue. The role of thiamin in SIDS and autism is outlined. It is emphasized that megadose thiamin is being used as a drug, either in stimulating the damaged cofactor/enzyme combination, or mitochondria.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180225
[Lr] Last revision date:180225
[St] Status:In-Data-Review

  4 / 2145 MEDLINE  
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[PMID]: 29360523
[Au] Autor:DiNicolantonio JJ; Liu J; O'Keefe JH
[Ad] Address:Saint Luke's Mid America Heart Institute, Kansas City, MO, United States. Electronic address: jjdinicol@gmail.com.
[Ti] Title:Thiamine and Cardiovascular Disease: A Literature Review.
[So] Source:Prog Cardiovasc Dis;, 2018 Jan 31.
[Is] ISSN:1873-1740
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Thiamine is a water-soluble vitamin that plays an important role in the energy metabolism in the human body. Deficiency in thiamine can lead to neurological abnormalities and congestive heart failure (HF), known as dry beriberi and wet beriberi respectively. Several populations are at higher risk for thiamine deficiency, most notably persons with chronic alcoholism. This article aims to provide a review of current literature on the role of thiamine in the human body, the current scope of thiamine deficiency, and explore the specific effects of thiamine deficiency and supplementation on the cardiovascular system. HF as a result of thiamine deficiency can have non-specific presentations, often leading to delayed diagnosis and treatment. Having an understanding of pathophysiology of thiamine deficiency and considering thiamine deficiency as one of the differentials in patients with new onset HF of unknown etiology with the appropriate risk factors is important in clinical practice.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1801
[Cu] Class update date: 180216
[Lr] Last revision date:180216
[St] Status:Publisher

  5 / 2145 MEDLINE  
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[PMID]: 29159485
[Au] Autor:Abdul Rahman S; Jeffree MS; Kamaludin F; Din IA; Yusof M; Ahmed K
[Ad] Address:Epidemic Intelligence Program (EIP), Ministry of Health Malaysia, Complex E, Federal Government Administrative Centre, 62590, Putrajaya, Malaysia.
[Ti] Title:Beriberi Outbreak Among Unauthorised Immigrants in a Detention Camp in Malaysia.
[So] Source:J Immigr Minor Health;, 2017 Nov 20.
[Is] ISSN:1557-1920
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:In February 2012, a district health authority received three inmates with bilateral leg swelling and difficulty in breathing from a detention camp for unauthorized immigrants. A case control study was conducted. Fasting blood samples of case and control groups were collected according to instructions of the laboratory for determining thiamine level, and their 1 week dietary intake was analyzed. 9% (21/226) of inmates had bilateral leg swelling, and 75% (6/8) of them had low thiamine level (< 66 mmol/L). Their diet contained mainly polished rice and other items with low thiamine contents. Bilateral leg swelling was associated with history of no meat consumption during past 3 months (OR 8.4; CI 2.2-32.1). Patients were treated with 100 mg thiamine intravenously and 5 mg orally per day for 6 weeks. All patients responded well. The management was advised to provide vitamin B complex daily, and encouraged to provide thiamine rich foods.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1711
[Cu] Class update date: 171121
[Lr] Last revision date:171121
[St] Status:Publisher
[do] DOI:10.1007/s10903-017-0674-5

  6 / 2145 MEDLINE  
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[PMID]: 29142756
[Au] Autor:Numata G; Kodera S; Kiriyama H; Nakayama A; Amiya E; Kiyosue A; Hatano M; Takimoto E; Watanabe M; Komuro I
[Ad] Address:Department of Cardiovascular Medicine, Graduate school of Medicine, The University of Tokyo, 7-3-1 Hongo, Bunkyo-ku, Tokyo, 1130033 Japan.
[Ti] Title:Usefulness of central venous saturation as a predictor of thiamine deficiency in critically ill patients: a case report.
[So] Source:J Intensive Care;5:61, 2017.
[Is] ISSN:2052-0492
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Background: Central venous oxygen saturation (ScvO ) reflects the balance of oxygen delivery and consumption. Low ScvO indicates the presence of inadequate oxygen delivery, while high ScvO indicates reduced oxygen consumption and is sometimes associated with a high mortality rate in critically ill patients from dysoxia. Thiamine is an essential cofactor in cellular aerobic metabolism. Thiamine deficiency is more prevalent than was previously thought, and underlies severe conditions in critically ill patients. However, currently, there is no rapid diagnostic test for thiamine deficiency. Considering oxygen flux, high ScvO might be associated with thiamine deficiency. Case presentation: A 70-year-old man admitted to the hospital with chief complaint of malaise and edema. He was diagnosed with heart failure with preserved ejection function and was treated with loop diuretics, which resulted in shock. Venoarterial extracorporeal membrane oxygenation and intra-aortic balloon pumping was indicated. The right heart catheter showed high ScvO , normal cardiac output, and low systemic vascular resistance. Thiamine deficiency was suspected and we started the thiamine infusion. His hemodynamic status improved after thiamine replacement. After his recovery, it was discovered that he had a 1-month history of anorexia and thiamine deficiency. His final diagnosis was beriberi. Conclusions: The current case showed the relation between thiamine deficiency and high ScvO . A literature review also suggested that thiamine deficiency is associated with high ScvO . Thiamine deficiency causes impaired tissue oxygen extraction, which could lead to high ScvO . In this context, high ScvO might serve as a predictor of thiamine deficiency in critically ill patients.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1711
[Cu] Class update date: 171119
[Lr] Last revision date:171119
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1186/s40560-017-0255-7

  7 / 2145 MEDLINE  
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[PMID]: 29128624
[Au] Autor:Di Marco S; Pilati L; Brighina F; Fierro B; Cosentino G
[Ad] Address:Department of Experimental Biomedicine and Clinical Neurosciences (BioNeC), University of Palermo, Italy.
[Ti] Title:Wernicke-Korsakoff syndrome complicated by subacute beriberi neuropathy in an alcoholic patient.
[So] Source:Clin Neurol Neurosurg;164:1-4, 2017 Nov 08.
[Is] ISSN:1872-6968
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Thiamine (vitamin B1) deficiency is a common condition in alcohol abusers, which can lead to damage of both the peripheral and the central nervous systems. Here we describe the case of an alcoholic patient who presented with acute onset of ataxia, severe weakness of the four limbs, and hypoesthesia and dysesthesia of the distal portion of the upper and lower extremities. The clinical picture also included mental confusion and amnesia. A diagnosis of Wernicke-Korsakoff syndrome was made based on clinical symptoms and brain RMI findings. Electromyography and electroneurography revealed signs of subacute axonal sensory-motor polyneuropathy that were compatible with a rare acute presentation of beriberi. Patient immediately received parenteral thiamine administration, which resulted in rapid clinical amelioration of ataxia and confusion and also in a significant improvement of motor and sensory deficits. The association between Wernicke-Korsakoff syndrome and acute axonal polyneuropathy is a very rare condition that could make less recognizable the clinical picture of a thiamine deficiency. However, the diagnosis of thiamine deficiency should be suspected in every alcoholic patient presenting with acute onset symptoms of central and/or peripheral nervous system involvement. This because the immediate replacement treatment can be life-saving and reverse the clinical symptoms.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1711
[Cu] Class update date: 171112
[Lr] Last revision date:171112
[St] Status:Publisher

  8 / 2145 MEDLINE  
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[PMID]: 29064578
[Au] Autor:Adamolekun B; Hiffler L
[Ad] Address:Department of Neurology, University of Tennessee Health Science Center, Memphis, Tennessee.
[Ti] Title:A diagnosis and treatment gap for thiamine deficiency disorders in sub-Saharan Africa?
[So] Source:Ann N Y Acad Sci;, 2017 Oct 24.
[Is] ISSN:1749-6632
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Staple diets that are deficient in thiamine can result in low body thiamine levels, which may be subclinical or may manifest as a thiamine-deficiency syndrome. In many communities in the developing countries of Africa, the staple diets of polished rice or processed cassava are deficient in thiamine, and thus the communities are at high risk for marginal or frank thiamine deficiency unless their diets are supplemented by other sources of thiamine, such as protein meals and vegetables. African communities with large numbers of individuals in low socioeconomic strata are more likely to subsist on a monotonous diet of rice or cassava with minimal or no protein supplementation and are therefore particularly at risk of thiamine-deficiency disorders. Indeed, there is evidence of widespread biochemical thiamine deficiency from community-based studies in Africa. The protean manifestations of thiamine deficiency disorders in the developing countries of Africa are presented in this paper. We present evidence supporting the contention that there is a diagnosis and treatment gap for thiamine-deficiency disorders in Africa. We discuss research and clinical options for bridging the putative diagnosis and treatment gap for thiamine-deficiency disorders in the developing countries of Africa.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1710
[Cu] Class update date: 171102
[Lr] Last revision date:171102
[St] Status:Publisher
[do] DOI:10.1111/nyas.13509

  9 / 2145 MEDLINE  
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[PMID]: 28919592
[Au] Autor:Lee J; Cho Y
[Ad] Address:Severance Dental Clinic.
[Ti] Title:The Study on the Lives and Health Conditions of Internees in Santo Thomas Camp of Philippines - Based on McAnlis's The War in Manila (1941-1945).
[So] Source:Uisahak;26(2):265-314, 2017 Aug.
[Is] ISSN:1225-505X
[Cp] Country of publication:Korea (South)
[La] Language:eng
[Ab] Abstract:When Japan invaded the Philippines, two missionary dentists (Dr. McAnlis and Dr. Boots) who were forced to leave Korea were captured and interned in the Santo Thomas camp in Manila. Japan continued to bombard and plunder the Philippines in the wake of the Pacific War following the Great East Asia policy, leading to serious inflation and material deficiency. More than 4,000 Allied citizens held in Santo Thomas camp without basic food and shelter. Santo Thomas Camp was equipped with the systems of the Japanese military medical officers and Western doctors of captivity based on the Geneva Conventions(1929). However, it was an unsanitary environment in a dense space, so it could not prevent endemic diseases such as dysentery and dengue fever. With the expansion of the war in Japan, prisoners in the Shanghai and Philippine prisons were not provided with medicines, cures and food for healing diseases. In May 1944, the Japanese military ordered the prisoners to reduce their ration. The war starting in September 1944, internees received 1000 kcal of food per day, and since January 1945, they received less than 800 kcal of food. This was the lowest level of food rationing in Japan's civilian prison camps. They suffered beriberi from malnutrition, and other endemic diseases. An averaged 24 kg was lost by adult men due to food shortages, and 10 percent of the 390 deaths were directly attributable to starvation. The doctors demanded food increases. The Japanese Military forced the prisoner to worship the emperor and doctors not to record malnourishment as the cause of death. During the period, the prisoners suffered from psychosomatic symptoms such as headache, diarrhea, acute inflammation, excessive smoking, and alcoholism also occurred. Thus, the San Thomas camp had many difficulties in terms of nutrition, hygiene and medical care. The Japanese military had unethical and careless medical practices in the absence of medicines. Dr. McAnlis and missionary doctors handled a lot of patients focusing mainly on examination, emergency treatment and provided the medical services needed by Philippines and foreigners as well as prisoners. Through out the war in the Great East Asia, the prisoners of Santo Thomas camp died of disease and starvation due to inhumane Japanese Policy. Appropriate dietary prescriptions and nutritional supplements are areas of medical care that treat patients' malnutrition and disease. It is also necessary to continue research because it is a responsibility related to the professionalism and ethics of medical professionals to urge them to observe the Geneva Convention.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1709
[Cu] Class update date: 170918
[Lr] Last revision date:170918
[St] Status:In-Data-Review
[do] DOI:10.13081/kjmh.2017.26.265

  10 / 2145 MEDLINE  
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[PMID]: 28905406
[Au] Autor:Hiffler L; Adamolekun B; Fischer PR; Fattal-Vavleski A
[Ad] Address:Médecins Sans Frontières-OCBA, Dakar Unit, Dakar, Senegal.
[Ti] Title:Thiamine content of F-75 therapeutic milk for complicated severe acute malnutrition: time for a change?
[So] Source:Ann N Y Acad Sci;1404(1):20-26, 2017 Sep.
[Is] ISSN:1749-6632
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Since community-based management of severe acute malnutrition has become the standard of care, the clinical profile of severe acutely malnourished patients admitted to hospitals or inpatient therapeutic feeding centers has changed significantly. These patients are usually very ill and often present with several comorbidities, such as shock, sepsis, and pneumonia. Complicated severe acute malnutrition patients are at risk of thiamine insufficiency, and critically ill patients have higher thiamine requirements. The thiamine content of F-75, the therapeutic milk formula used in the early stabilization phase of refeeding in patients with severe acute malnutrition, seems insufficient. Here, we discuss the need and rationale for a substantial increase in the thiamine content of F-75.
[Mh] MeSH terms primary: Milk/chemistry
Severe Acute Malnutrition/diet therapy
Thiamine Deficiency/diet therapy
Thiamine/administration & dosage
Thiamine/analysis
[Mh] MeSH terms secundary: Animals
Critical Illness/epidemiology
Critical Illness/therapy
Food, Fortified
Humans
Severe Acute Malnutrition/diagnosis
Severe Acute Malnutrition/epidemiology
Thiamine Deficiency/diagnosis
Thiamine Deficiency/epidemiology
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Nm] Name of substance:X66NSO3N35 (Thiamine)
[Em] Entry month:1710
[Cu] Class update date: 171011
[Lr] Last revision date:171011
[Js] Journal subset:IM
[Da] Date of entry for processing:170915
[St] Status:MEDLINE
[do] DOI:10.1111/nyas.13458


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