Database : MEDLINE
Search on : Epidermolysis and Bullosa and Acquisita [Words]
References found : 855 [refine]
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[PMID]: 29524251
[Au] Autor:Sawada M; Hida T; Ujiie H; Iwata H; Uhara H
[Ad] Address:Department of Dermatology, Sapporo Medical University School of Medicine, Sapporo, Japan.
[Ti] Title:A case of subepidermal autoimmune bullous disease with autoantibodies against 200-kDa and 290-kDa antigens.
[So] Source:J Eur Acad Dermatol Venereol;, 2018 Mar 10.
[Is] ISSN:1468-3083
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Epidermolysis bullosa acquisita (EBA) and anti-p200 pemphigoid are uncommon subepidermal autoimmune bullous diseases caused by autoantibodies against the 200-kDa protein and 290-kDa type VII collagen, respectively. Here we describe a patient with autoantibodies against both 200-kDa and 290-kDa antigens.A 63-year-old-man had itchy tense blisters and edematous erythemas scattered on his trunk, buttocks, extremities and soles (Fig. 1a). There were no ocular or mucosal lesions. Psoriatic skin lesions were not observed. There was no personal or family history of serious diseases. This article is protected by copyright. All rights reserved.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180310
[Lr] Last revision date:180310
[St] Status:Publisher
[do] DOI:10.1111/jdv.14927

  2 / 855 MEDLINE  
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[PMID]: 29447655
[Au] Autor:Cobos G; Mu E; Cohen J; Beasley J; Brinster N; Femia A
[Ad] Address:New York University, New York.
[Ti] Title:Epidermolysis bullosa acquisita.
[So] Source:Dermatol Online J;23(12), 2017 Dec 15.
[Is] ISSN:1087-2108
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Epidermolysis bullosa acquisita (EBA) is a rare, acquired subepidermal blistering disease. EBA is characterized by autoantibodies to collagen VII,which serves to link the epidermis to the dermis. The two most common presentations of EBA are classical noninflammatory EBA and bullous pemphigoid-like EBA. Diagnosis of EBA can be challenging as it sharesclinical and histopathologic features with other blistering diseases. Treatment is often recalcitrant and will often necessitate multiple therapies. We presenta case of a thirty-six-year-old Chinese man with EBA and review the literature.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180215
[Lr] Last revision date:180215
[St] Status:In-Process

  3 / 855 MEDLINE  
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[PMID]: 29441548
[Au] Autor:Carmichael AJ; Harman KE
[Ad] Address:Department of Dermatology, The James Cook University Hospital, Marton Road, Middlesbrough, TS4 3BW, U.K.
[Ti] Title:Orf - a potential trigger for self-limiting epidermolysis bullosa acquisita-like blistering.
[So] Source:Br J Dermatol;178(2):333, 2018 Feb.
[Is] ISSN:1365-2133
[Cp] Country of publication:England
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180214
[Lr] Last revision date:180214
[St] Status:In-Data-Review
[do] DOI:10.1111/bjd.16055

  4 / 855 MEDLINE  
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[PMID]: 29322498
[Au] Autor:Hertl M; Hashimoto T
[Ad] Address:Department of Dermatology and Allergology, Philipps University, Marburg, Germany.
[Ti] Title:Response to 'Serological diagnostics in the detection of IgG autoantibodies against human collagen VII in epidermolysis bullosa acquisita: a multicentre analysis': reply from authors.
[So] Source:Br J Dermatol;178(2):573-574, 2018 Feb.
[Is] ISSN:1365-2133
[Cp] Country of publication:England
[La] Language:eng
[Pt] Publication type:LETTER
[Em] Entry month:1801
[Cu] Class update date: 180214
[Lr] Last revision date:180214
[St] Status:In-Data-Review
[do] DOI:10.1111/bjd.16169

  5 / 855 MEDLINE  
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[PMID]: 29392620
[Au] Autor:Kushner CJ; Concha JSS; Werth VP
[Ad] Address:Corporal Michael J. Crescenz VAMC, Philadelphia, PA, USA.
[Ti] Title:Treatment of Autoimmune Bullous Disorders in Pregnancy.
[So] Source:Am J Clin Dermatol;, 2018 Feb 02.
[Is] ISSN:1179-1888
[Cp] Country of publication:New Zealand
[La] Language:eng
[Ab] Abstract:Autoimmune bullous diseases (AIBD), including pemphigus, bullous pemphigoid, epidermolysis bullosa acquisita, mucous membrane pemphigoid, and pemphigoid gestationis, pose significant therapeutic challenges, especially in pregnant and post-partum breastfeeding patients or those planning to conceive. Data on the safety and efficacy of therapeutic interventions during the perinatal period are lacking because randomized controlled trials are typically not performed in this setting. However, many of the treatments for AIBD are also used in other diseases, so data can be extrapolated from studies or case reports in these other patient populations. It appears that many of the treatments for AIBD can adversely affect the fetus or neonate, and alterations in immune status caused by pregnancy-associated hormonal changes can negatively impact disease control. This article summarizes and weighs the risks and benefits of the various agents used to treat AIBD during pregnancy. We also present the available information on lactation as well as effects on male fertility.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1802
[Cu] Class update date: 180202
[Lr] Last revision date:180202
[St] Status:Publisher
[do] DOI:10.1007/s40257-018-0342-0

  6 / 855 MEDLINE  
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[PMID]: 29352483
[Au] Autor:Watanabe M; Natsuga K; Shinkuma S; Shimizu H
[Ad] Address:Department of Dermatology, Hokkaido University Graduate School of Medicine, Sapporo, Japan.
[Ti] Title:Epidermal aspects of type VII collagen: Implications for dystrophic epidermolysis bullosa and epidermolysis bullosa acquisita.
[So] Source:J Dermatol;, 2018 Jan 20.
[Is] ISSN:1346-8138
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Type VII collagen (COL7), a major component of anchoring fibrils in the epidermal basement membrane zone, has been characterized as a defective protein in dystrophic epidermolysis bullosa and as an autoantigen in epidermolysis bullosa acquisita. Although COL7 is produced and secreted by both epidermal keratinocytes and dermal fibroblasts, the role of COL7 with regard to the epidermis is rarely discussed. This review focuses on COL7 physiology and pathology as it pertains to epidermal keratinocytes. We summarize the current knowledge of COL7 production and trafficking, its involvement in keratinocyte dynamics, and epidermal carcinogenesis in COL7 deficiency and propose possible solutions to unsolved issues in this field.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1801
[Cu] Class update date: 180120
[Lr] Last revision date:180120
[St] Status:Publisher
[do] DOI:10.1111/1346-8138.14222

  7 / 855 MEDLINE  
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[PMID]: 29267434
[Au] Autor:Zhao G; Yang Q; Zhang F
[Ad] Address:Department of Dermatology, Shandong Provincial Hospital for Skin Diseases, Shandong University, Shandong , China.
[Ti] Title:Acute renal failure in a patient with epidermolysis bullosa acquisita.
[So] Source:An Bras Dermatol;92(5 Suppl 1):14-16, 2017.
[Is] ISSN:1806-4841
[Cp] Country of publication:Brazil
[La] Language:eng
[Ab] Abstract:Epidermolysis bullosa acquisita is a severe autoimmune subepidermal bullous disease. In this report, we described for the first time a patient with epidermolysis bullosa acquisita who developed acute renal failure. There is a possibility that epidermolysis bullosa acquisita and acute renal failure's pathogenesis shared some common autoimmune pathways. Moreover, acute blood volume reduction may be another cause of prerenal kidney failure. Further studies are needed to verify our hypothesis.
[Pt] Publication type:CASE REPORTS
[Em] Entry month:1712
[Cu] Class update date: 171224
[Lr] Last revision date:171224
[St] Status:In-Process

  8 / 855 MEDLINE  
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[PMID]: 29205468
[Au] Autor:Nishida E; Nishio E; Murashima H; Ishii N; Hashimoto T; Morita A
[Ad] Address:Department of Geriatric and Environmental Dermatology, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan.
[Ti] Title:Case of epidermolysis bullosa acquisita with concomitant anti-laminin-332 antibodies.
[So] Source:J Dermatol;, 2017 Dec 04.
[Is] ISSN:1346-8138
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Subepidermal autoimmune blistering disease including bullous pemphigoid, pemphigoid gestationis, mucous membrane pemphigoid, anti-laminin-γ1 pemphigoid, linear immunoglobulin A bullous disease and epidermolysis bullosa acquisita (EBA), are all characterized by direct immunofluorescence microscopy or immunoglobulin deposition on the basement membrane zone. Among them, EBA is a rare acquired subepidermal autoimmune blistering disease of the skin and mucous membranes reactive with type VII collagen, a major component of the epidermal basement membrane zone. Anti-laminin-332-type mucous membrane pemphigoid has pathogenic autoantibodies against laminin-332, which is a basement membrane heterotrimeric protein composed of α3, ß3 and γ2 laminin chains. We describe a 73-year-old Japanese man presenting with multiple, annular, tense blisters on the lower legs and oral lesions. Despite the severe clinical manifestations, the disease was successfully controlled by combination therapy of oral prednisolone and mizoribine. This case was confirmed to have autoantibodies to both type VII collagen and laminin-332 α3 chain by indirect immunofluorescence of 1 mol NaCl-split normal human skin, various immunoblot analyses and enzyme-linked immunosorbent assays. This case was a rare case of EBA with concomitant anti-laminin-332 antibodies.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171205
[Lr] Last revision date:171205
[St] Status:Publisher
[do] DOI:10.1111/1346-8138.14169

  9 / 855 MEDLINE  
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[PMID]: 29182795
[Au] Autor:Guerra L; Condorelli AG; Fortugno P; Calabresi V; Pedicelli C; Di Zenzo G; Castiglia D
[Ti] Title:Epidermolysis Bullosa Acquisita in an Adult Patient with Previously Unrecognized Mild Dystrophic EB and Biallelic COL7A1 Mutations.
[So] Source:Acta Derm Venereol;, 2017 Nov 28.
[Is] ISSN:1651-2057
[Cp] Country of publication:Sweden
[La] Language:eng
[Ab] Abstract:Circulating anti-type VII collagen autoantibodies are frequently detected in patients with recessive dystrophic epidermolysis bullosa (RDEB). However, evidence supporting their pathogenic role in inducing epidermolysis bullosa acquisita (EBA) has been provided for only 1 individual with dominant dystrophic epidermolysis bullosa (DDEB). We describe here a patient who presented with dystrophic toenails since early childhood and developed trauma-induced skin blisters and oral erosions at age 26 years. Direct immunofluorescence showed IgG deposits with a u-serrated pattern along the cutaneous basement membrane zone, while no change in the expression of collagen VII could be detected by antigen mapping. High-titre anti-collagen VII antibodies were detected by enzyme-linked immunoassay (ELISA). In parallel, sequencing of epidermolysis bullosa (EB) genes identified compound heterozygous COL7A1 missense c.410G>A (p.Arg137Gln) and splicing c.3674C>T (p.Ala1225_Gln1241del) mutations, previously unrecognized in dystrophic epidermolysis bullosa (DEB). Thus, our patient had RDEB "nails-only" and developed mechanobullous EBA in adulthood. These data support a pathogenic role of circulating autoantibodies to collagen VII in inducing EBA in selected patients with DEB. Unforeseen worsening of skin symptoms in DEB should prompt laboratory investigations for EBA.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1711
[Cu] Class update date: 171128
[Lr] Last revision date:171128
[St] Status:Publisher
[do] DOI:10.2340/00015555-2851

  10 / 855 MEDLINE  
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[PMID]: 29165796
[Au] Autor:Prost-Squarcioni C; Caux F; Schmidt E; Jonkman MF; Vassileva S; Kim SC; Iranzo P; Daneshpazhooh M; Terra J; Bauer J; Fairley J; Hall R; Hertl M; Lehman JS; Marinovic B; Patsatsi A; Zillikens D; Werth V; Woodley DT; Murrell DF; International Bullous Diseases Group
[Ad] Address:Department of Dermatology and referral center for autoimmune bullous diseases, APHP, Avicenne Hospital, Bobigny, France.
[Ti] Title:International Bullous Diseases Group - Consensus on Diagnostic Criteria for Epidermolysis Bullosa Acquisita.
[So] Source:Br J Dermatol;, 2017 Nov 22.
[Is] ISSN:1365-2133
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Epidermolysis bullosa acquisita (EBA) is a complex autoimmune bullous disease disease with variable clinical presentations and multiple possible diagnostic tests making an international consensus on diagnosis of EBA needed. OBJECTIVES: To obtain an international consensus on the clinical and diagnostic criteria for EBA. METHODS: The international bullous diseases group (IBDG) met three times to discuss the clinical and diagnostic criteria for EBA. For the final voting exercise, 22 experts from 14 different countries voted on 50 different items. When more than 30% disagreed with a proposal, a discussion was held and revoting occurred. RESULTS: 48/50 proposals achieved consensus after discussion. This included 9 diagnostic criteria that are summarized in a flow chart. The IBDG was unable to determine one procedure which would be applicable worldwide. LIMITATIONS: Differential diagnosis of bullous systemic lupus erythematosus has not been addressed. CONCLUSION: This first international consensus conference established generally agreed upon clinical and laboratory criteria defining the clinical classification and diagnostic testing for EBA. Holding these voting exercises in person with the possibility of discussion prior to voting has advantages in reaching consensus over Delphi exercises with remote voting. This article is protected by copyright. All rights reserved.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1711
[Cu] Class update date: 171122
[Lr] Last revision date:171122
[St] Status:Publisher
[do] DOI:10.1111/bjd.16138


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