Database : MEDLINE
Search on : Hyperkinesis [Words]
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[PMID]: 29434120
[Au] Autor:Tsuji M; Isogai T; Okabe Y; Nishimura Y; Itagaki S; Enatsu K; Hisagi M; Nonaka T; Ninomiya M; Otsuka T; Tanaka H; Tejima T
[Ad] Address:Department of Cardiology, Tokyo Metropolitan Tama Medical Center, Japan.
[Ti] Title:Ventricular Septal Perforation: A Rare but Life-Threatening Complication Associated with Takotsubo Syndrome.
[So] Source:Intern Med;, 2018 Feb 09.
[Is] ISSN:1349-7235
[Cp] Country of publication:Japan
[La] Language:eng
[Ab] Abstract:A 71-year-old woman was admitted with dyspnea. An electrocardiogram revealed ST-segment elevation, and echocardiography showed akinesis in the left ventricular apex with hyperkinesis of the base. Coronary angiography revealed no stenosis, and left ventriculography indicated ballooning of the left ventricular apex and apical ventricular septal perforation. We diagnosed the patient with Takotsubo syndrome complicated by ventricular septal perforation, which was surgically repaired. Although ventricular septal perforation is recognized as a life-threatening complication after acute myocardial infarction, it can also occur after Takotsubo syndrome. The early recognition and management of this condition can help prevent morbidity and mortality.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180213
[Lr] Last revision date:180213
[St] Status:Publisher
[do] DOI:10.2169/internalmedicine.0014-17

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[PMID]: 29411764
[Au] Autor:Busygina MS; Vakhrushev YM
[Ad] Address:Izhevsk State Medical Academy, Ministry of Health of Russia, Izhevsk, Russia.
[Ti] Title:Kharakteristika techeniia iazvennoi bolezni zheludka i dvenadtsatiperstnoi kishki s soputstvuiushchei duodenal'noi nedostatochnost'iu. [Characteristics of the course of gastric and duodenal ulcer disease concurrent with duodenal insufficiency].
[So] Source:Ter Arkh;89(12):76-80, 2017.
[Is] ISSN:0040-3660
[Cp] Country of publication:Russia (Federation)
[La] Language:rus
[Ab] Abstract:AIM: To comprehensively study the course of gastric ulcer disease (GUD) and duodenal ulcer disease (DUD) concurrent with chronic duodenal insufficiency (CDI). MATERIAL AND METHODS: Ulcer disease (UD) was verified on the basis of the results of clinical and fibrogastroduodenoscopic examinations. The data of contrast duodenography and cavitary manometry were used to identify CDI. Gastroduodenal motor activity was investigated using the peripheral electrogastrograph EGG-4M. The results of pH measurements were employed to assess the state of gastric acid secretion and duodenal pH values. RESULTS: A comprehensive examination was made in 106 patients with UD concurrent with CDI (a study group) and 30 UD patients without CDI (a comparison group). Epigastric pain was noted in the patients with GUD in the study and comparison groups (91.5 and 84.6%, respectively), but the pain was mainly aching in the patients with concomitant CDI and more intense (77.8%) in those without this condition. In the study group, heartburn was more common in patients with GUD and DUD (75.3 and 71.4%, respectively) than in those with UD in the comparison group (28.5 and 37.5%, respectively). Helicobacter pylori tests were positive in 23.8% of the patients in the study group and in 57.2% in the comparison group. Electrogastrography indicated that the patients with GUD and CDI had bradygastria and hypokinesis on an empty stomach; the electrical activity was reduced after eating. In the comparison group, tachygastria and hyperkinesis were detected on an empty stomach; these postprandial indicators were elevated. H. pylori tests were positive in 34.7% of the patients with DUD and CDI and in 63.6% of those with DUD without CDI. The postprandial electrical activity increased in patients with DUD and decreased in the comparison group. The specific features of changes in gastric and duodenal pH values in GUD and DUD concurrent with CDI in comparison with the isolated course of UD. CONCLUSION: The immediate and long-term follow-ups show that GUD and DUD concurrent with CDI run a more persistent course; the time of ulcer healing increases and the periods of remission decrease.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180207
[Lr] Last revision date:180207
[St] Status:In-Data-Review
[do] DOI:10.17116/terarkh2017891276-80

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[PMID]: 29376981
[Au] Autor:Karakulova YV; Loginova NV
[Ad] Address:Vagner Perm State Medical University, Perm, Russia.
[Ti] Title:Éffektivnost' botulinoterapii v korrektsii stepeni bolevogo sindroma i kachestva zhizni patsientov s tservikal'noi distoniei. [The efficacy of botulinotherapy in the correction of the pain syndrome and quality of life of patients with cervical dystonia].
[So] Source:Zh Nevrol Psikhiatr Im S S Korsakova;117(12):33-36, 2017.
[Is] ISSN:1997-7298
[Cp] Country of publication:Russia (Federation)
[La] Language:rus
[Ab] Abstract:AIM: To evaluate the severity of pain, emotional status and humoral serotonin in patients with cervical dystonia (CD) before and after the botulinotherapy. MATERIAL AND METHODS: A simple, open, comparative study of clinical characteristics of hyperkinesis, pain and emotional status, quality of life and contents of serum and blood platelet serotonin in 48 patients (32 women and 16 men) with CD, in age from 37 to 53 years, before and one month after the botulinotherapy with disport in dose of 500--1000 U was carried out. A control group included 15 healthy people. RESULTS: All patients (100%) complained of involuntary movements and pain in the neck. The overall score on a scale of dystonic movements in the group of patients was 16,7±7,7 points, on TWSTRS - 46,48±6,2 points, on the Visual Analogue Scale, the average level of pain was 6,4±1,08 points. The degree of depression according to the Hamilton scale was significantly higher (p<0.05) compared to the control group. The level of trait and state anxiety measured with the Spielberger-Khanin scale was significantly higher (p<0.005) in patients with CD than in the controls. The correlation analysis revealed a direct dependence of the intensity of pain subscale TWSTRS with the degree of anxiety on the Hamilton scale and the amount of final points of dystonic movements. The level of serotonin in the serum was significantly lower in patients compared to the controls. After botulinotherapy, pain scores, anxiety and depression have significantly decreased and the level of blood platelet serotonin has increased. CONCLUSION: Botulinotherapy with dysport in CD patients reduces the degree of pain, depression, improves quality of life and stimulates the serotoninergic system.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180129
[Lr] Last revision date:180129
[St] Status:In-Data-Review
[do] DOI:10.17116/jnevro201711712133-36

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[PMID]: 29326495
[Au] Autor:Sezer Ö; Aydin AA; Bilge S; Arslan F; Arslan H
[Ad] Address:Department of Emergency Medicine, Amasya University Sabuncuoglu Serefeddin Training and Research Hospital, Amasya, Turkey.
[Ti] Title:Acute dystonic reaction leading to lingual hematoma mimicking angioedema.
[So] Source:Indian J Pharmacol;49(4):325-327, 2017 Jul-Aug.
[Is] ISSN:1998-3751
[Cp] Country of publication:India
[La] Language:eng
[Ab] Abstract:Lingual hematoma is a severe situation, which is rare and endangers the airway. It can develop due to trauma, vascular abnormalities, and coagulopathy. Due to its sudden development, it can be clinically confused with angioedema. In patients who applied to the doctor with complaints of a swollen tongue, lingual hematoma can be confused with angioedema, in particular, at the beginning if the symptoms occurred after drug use. It should especially be considered that dystonia in the jaw can present as drug-induced hyperkinetic movement disorder. Early recognition of this rare clinical condition and taking precautions for providing airway patency are essential. In this case report, we will discuss mimicking angioedema and caused by a bite due to dystonia and separation of the tongue from the base of the mouth developing concurrently with lingual hematoma.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180114
[Lr] Last revision date:180114
[St] Status:In-Data-Review
[do] DOI:10.4103/ijp.IJP_620_16

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[PMID]: 29278267
[Au] Autor:de Castilho LS; Abreu MHNG; Pires E Souza LGA; Romualdo LTA; Souza E Silva ME; Resende VLS
[Ad] Address:Professor Doctor of the Department of Restorative Dentistry of Dental School, Coordinator of the Extension Project "Dental Care for disabled", Federal University of Minas Gerais State, Brazil. Avenida Presidente Antônio Carlos, 6627, Campus Pampulha, CEP 31270-010, Belo Horizonte, Minas Gerais, Braz
[Ti] Title:Factors associated with anterior open bite in children with developmental disabilities.
[So] Source:Spec Care Dentist;, 2017 Dec 26.
[Is] ISSN:1754-4505
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:AIM: To investigate factors associated with anterior open bite in individuals aged from 2 to 33 years with developmental disabilities. DESIGN: This is a cross-sectional study. A total of 271 dental records were examined. The anterior open bite analyzed was determined based on clinic exam. These variables were also analyzed: gender, age, education level of mother, International Code of Diseases (ICD), mouth breathing, use of anticonvulsant drugs, hyperkinesis, pacifier use, thumb sucking, seizure, and involuntary movements. For the purposes of analysis, the individuals were categorized as being with and without anterior open bite. Variables with a p-value of < 0.25 in the bivariate analysis were incorporated into the logistic regression models. RESULTS: Mouth breathers had a 2.60-fold (95% CI: 1.35-5.01) greater chance of exhibiting anterior open bite than nasal breathers. Pacifier users are more likely to have an anterior open bite (3.32-fold, 95% CI: 1.62-6.77). Individuals with reported involuntary movements had a 2.66-fold (95% CI: 1.26-5.63) greater chance of exhibiting anterior open bite. Users of anticonvulsants drugs had a 3.05 (95% CI: 1.57-5.92) greater chance of showing anterior open bite. CONCLUSION: Involuntary movements, mouth breathing, using anticonvulsant drugs, and using pacifier are factors associated with anterior open bite in patients with developmental disabilities.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171226
[Lr] Last revision date:171226
[St] Status:Publisher
[do] DOI:10.1111/scd.12262

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[PMID]: 28745663
[Au] Autor:Mironov MB; Bobylova MY; Nekrasova IV; Krasilschikova TM; Gunchenko MM; Sarzhina MN; Petrukhin AS; Burd SG; Batisheva TT
[Ad] Address:Training Institute of the Federal Medical and Biological Agency, Moscow, Russia; Center for pediatric psychoneurology, Moscow, Russia.
[Ti] Title:Differentsial'naia diagnostika paroksizmov tonicheskogo napriazheniia myshts épilepticheskoi i neépilepticheskoi prirody u detei rannego vozrasta s zaderzhkoi psikhomotornogo razvitiia i ochagovymi nevrologicheskimi simptomami. [Differential diagnosis of paroxysms of tonic muscle tension in children of early age with delay of psychomotor development and abnormal neurologic status].
[So] Source:Zh Nevrol Psikhiatr Im S S Korsakova;117(6):4-9, 2017.
[Is] ISSN:1997-7298
[Cp] Country of publication:Russia (Federation)
[La] Language:rus
[Ab] Abstract:AIM: To study neurologic status, results of video-EEG monitoring and magnetic resonance imaging in children under 3 years old with paroxysms of tonic muscle tension. MATERIAL AND METHODS: One hundred and forty-six infants and young children with motor disturbances and different variants of clinically similar epileptic seizures, hyperkinesis and stereotypes were examined. RESULTS AND CONCLUSION: Cerebral palsy (91%), genetic and chromosomal abnormalities (6%), brain malformations (2%) were identified. Neurological status was characterized by pseudobulbar syndrome (100% of cases), hemiparesis (1%), tetraparesis (81%), diffuse muscular hypotonia (18%), intellectual and speech development delay (76%), autistic behavior (16%). During the prolong video-EEG monitoring, paroxysmal tonic muscle tensions were recorded in all patients: epileptic seizures were observed in 113 patients (77.40%), non-epileptic paroxysms in 51 (34.93%). The combination of epileptic and non-epileptic paroxysms was observed in 18 patients (12.33%). In 4 patients (2.75%), it was not possible to determine the genesis of paroxysms even during the prolong video-EEG-monitoring because of myographic artefacts. Five clinical and electroencephalographic combinations of dystonic attacks, epileptic seizures and epileptiform activity were identified. These data allow improving the diagnosis of epilepsy and avoiding unnecessary treatment with antiepileptic drugs. Our study has shown a high diagnostic value of video-EEG monitoring with the inclusion of sleep in patients with paroxysmal conditions in infancy and early childhood.
[Mh] MeSH terms primary: Muscle Tonus
Psychomotor Disorders/diagnosis
Seizures/diagnosis
[Mh] MeSH terms secundary: Anticonvulsants/therapeutic use
Cerebral Palsy/diagnosis
Cerebral Palsy/physiopathology
Child, Preschool
Diagnosis, Differential
Electroencephalography
Female
Humans
Hyperkinesis/diagnosis
Hyperkinesis/physiopathology
Infant
Male
Psychomotor Disorders/physiopathology
Seizures/drug therapy
Seizures/physiopathology
Sleep
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Anticonvulsants)
[Em] Entry month:1711
[Cu] Class update date: 171128
[Lr] Last revision date:171128
[Js] Journal subset:IM
[Da] Date of entry for processing:170727
[St] Status:MEDLINE
[do] DOI:10.17116/jnevro2017117614-9

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[PMID]: 28899869
[Au] Autor:Kang MS; Choi TY; Ryu HG; Lee D; Lee SH; Choi SY; Kim KT
[Ad] Address:Department of Life Sciences, Pohang University of Science and Technology, Pohang, Republic of Korea.
[Ti] Title:Autism-like behavior caused by deletion of vaccinia-related kinase 3 is improved by TrkB stimulation.
[So] Source:J Exp Med;214(10):2947-2966, 2017 Oct 02.
[Is] ISSN:1540-9538
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Vaccinia-related kinases (VRKs) are multifaceted serine/threonine kinases that play essential roles in various aspects of cell signaling, cell cycle progression, apoptosis, and neuronal development and differentiation. However, the neuronal function of is still unknown despite its etiological potential in human autism spectrum disorder (ASD). Here, we report that -deficient mice exhibit typical symptoms of autism-like behavior, including hyperactivity, stereotyped behaviors, reduced social interaction, and impaired context-dependent spatial memory. A significant decrease in dendritic spine number and arborization were identified in the hippocampus CA1 of -deficient mice. These mice also exhibited a reduced rectification of AMPA receptor-mediated current and changes in expression of synaptic and signaling proteins, including tyrosine receptor kinase B (TrkB), Arc, and CaMKIIα. Notably, TrkB stimulation with 7,8-dihydroxyflavone reversed the altered synaptic structure and function and successfully restored autism-like behavior in -deficient mice. These results reveal that plays a critical role in neurodevelopmental disorders and suggest a potential therapeutic strategy for ASD.
[Mh] MeSH terms primary: Autistic Disorder/etiology
Protein-Serine-Threonine Kinases/deficiency
Receptor, trkB/physiology
[Mh] MeSH terms secundary: Animals
CA1 Region, Hippocampal/pathology
Female
Flavanones/pharmacology
Hyperkinesis/etiology
Male
Mice
Mice, Inbred C57BL
Protein-Serine-Threonine Kinases/metabolism
Receptor, trkB/drug effects
Receptor, trkB/metabolism
Social Behavior
Stereotyped Behavior
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (7,8-dihydroxyflavanone); 0 (Flavanones); EC 2.7.10.1 (Receptor, trkB); EC 2.7.11.1 (Protein-Serine-Threonine Kinases); EC 2.7.11.1 (Vrk3 protein, mouse)
[Em] Entry month:1710
[Cu] Class update date: 171024
[Lr] Last revision date:171024
[Js] Journal subset:IM
[Da] Date of entry for processing:170914
[St] Status:MEDLINE
[do] DOI:10.1084/jem.20160974

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[PMID]: 28881029
[Au] Autor:Berezniuk I; Rodriguiz RM; Zee ML; Marcus DJ; Pintar J; Morgan DJ; Wetsel WC; Fricker LD
[Ad] Address:Department of Neuroscience, Albert Einstein College of Medicine, Bronx, New York, USA.
[Ti] Title:ProSAAS-derived peptides are regulated by cocaine and are required for sensitization to the locomotor effects of cocaine.
[So] Source:J Neurochem;143(3):268-281, 2017 Nov.
[Is] ISSN:1471-4159
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:To identify neuropeptides that are regulated by cocaine, we used a quantitative peptidomic technique to examine the relative levels of neuropeptides in several regions of mouse brain following daily intraperitoneal administration of 10 mg/kg cocaine or saline for 7 days. A total of 102 distinct peptides were identified in one or more of the following brain regions: nucleus accumbens, caudate putamen, frontal cortex, and ventral tegmental area. None of the peptides detected in the caudate putamen or frontal cortex were altered by cocaine administration. Three peptides in the nucleus accumbens and seven peptides in the ventral tegmental area were significantly decreased in cocaine-treated mice. Five of these ten peptides are derived from proSAAS, a secretory pathway protein and neuropeptide precursor. To investigate whether proSAAS peptides contribute to the physiological effects of psychostimulants, we examined acute responses to cocaine and amphetamine in the open field with wild-type (WT) and proSAAS knockout (KO) mice. Locomotion was stimulated more robustly in the WT compared to mutant mice for both psychostimulants. Behavioral sensitization to amphetamine was not maintained in proSAAS KO mice and these mutants failed to sensitize to cocaine. To determine whether the rewarding effects of cocaine were altered, mice were tested in conditioned place preference (CPP). Both WT and proSAAS KO mice showed dose-dependent CPP to cocaine that was not distinguished by genotype. Taken together, these results suggest that proSAAS-derived peptides contribute differentially to the behavioral sensitization to psychostimulants, while the rewarding effects of cocaine appear intact in mice lacking proSAAS.
[Mh] MeSH terms primary: Cocaine/pharmacology
Dopamine Uptake Inhibitors/pharmacology
Hyperkinesis/chemically induced
Locomotion/drug effects
Nerve Tissue Proteins/metabolism
[Mh] MeSH terms secundary: Amphetamine/pharmacology
Animals
Conditioning, Operant/drug effects
Dose-Response Relationship, Drug
Exploratory Behavior/drug effects
Female
Gene Expression Regulation/drug effects
Gene Expression Regulation/genetics
Male
Mass Spectrometry
Mice
Mice, Inbred C57BL
Mice, Knockout
Nerve Tissue Proteins/genetics
Nucleus Accumbens/drug effects
Ventral Tegmental Area/drug effects
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Dopamine Uptake Inhibitors); 0 (Nerve Tissue Proteins); 0 (Pcsk1n protein, mouse); CK833KGX7E (Amphetamine); I5Y540LHVR (Cocaine)
[Em] Entry month:1711
[Cu] Class update date: 171121
[Lr] Last revision date:171121
[Js] Journal subset:IM
[Da] Date of entry for processing:170908
[St] Status:MEDLINE
[do] DOI:10.1111/jnc.14209

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[PMID]: 28794249
[Au] Autor:Sadleir LG; Mountier EI; Gill D; Davis S; Joshi C; DeVile C; Kurian MA; Mandelstam S; Wirrell E; Nickels KC; Murali HR; Carvill G; Myers CT; Mefford HC; Scheffer IE; DDD Study
[Ad] Address:From the Department of Paediatrics and Child Health (L.G.S., E.I.M.), University of Otago, Wellington, New Zealand; Department of Neurology (D.G.), University of Sydney, Australia; Department of Neurology (S.D.), Starship Children's Health, Auckland, New Zealand; Department of Neurology (C.J.), Chil
[Ti] Title:Not all epileptic encephalopathies are Dravet syndrome: Early profound Thr226Met phenotype.
[So] Source:Neurology;89(10):1035-1042, 2017 Sep 05.
[Is] ISSN:1526-632X
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:OBJECTIVE: To define a distinct developmental and epileptic encephalopathy with early onset, profound impairment, and movement disorder. METHODS: A case series of 9 children were identified with a profound developmental and epileptic encephalopathy and mutation. RESULTS: We identified 9 children 3 to 12 years of age; 7 were male. Seizure onset was at 6 to 12 weeks with hemiclonic seizures, bilateral tonic-clonic seizures, or spasms. All children had profound developmental impairment and were nonverbal and nonambulatory, and 7 of 9 required a gastrostomy. A hyperkinetic movement disorder occurred in all and was characterized by dystonia and choreoathetosis with prominent oral dyskinesia and onset from 2 to 20 months of age. Eight had a recurrent missense mutation, p.Thr226Met. The remaining child had the missense mutation p.Pro1345Ser. The mutation arose de novo in 8 of 9; for the remaining case, the mother was negative and the father was unavailable. CONCLUSIONS: Here, we present a phenotype-genotype correlation for . We describe a distinct phenotype, early infantile encephalopathy, which is readily distinguishable from the well-recognized entities of Dravet syndrome and genetic epilepsy with febrile seizures plus. This disorder has an earlier age at onset, profound developmental impairment, and a distinctive hyperkinetic movement disorder, setting it apart from Dravet syndrome. Remarkably, 8 of 9 children had the recurrent missense mutation p.Thr226Met.
[Mh] MeSH terms primary: Developmental Disabilities/genetics
Epilepsy/genetics
Hyperkinesis/genetics
Mutation, Missense
NAV1.1 Voltage-Gated Sodium Channel/genetics
[Mh] MeSH terms secundary: Age of Onset
Brain/diagnostic imaging
Brain/physiopathology
Child
Child, Preschool
Developmental Disabilities/diagnostic imaging
Developmental Disabilities/physiopathology
Epilepsies, Myoclonic/genetics
Epilepsies, Myoclonic/physiopathology
Epilepsy/diagnostic imaging
Epilepsy/physiopathology
Female
Humans
Hyperkinesis/diagnostic imaging
Hyperkinesis/physiopathology
Male
Phenotype
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:0 (NAV1.1 Voltage-Gated Sodium Channel); 0 (SCN1A protein, human)
[Em] Entry month:1709
[Cu] Class update date: 170914
[Lr] Last revision date:170914
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:170811
[St] Status:MEDLINE
[do] DOI:10.1212/WNL.0000000000004331

  10 / 4352 MEDLINE  
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[PMID]: 28774770
[Au] Autor:Pruthi S; Kobrossi S; Bartaula R; Chaudhuri D
[Ad] Address:Department of Medicine, State University of New York, Upstate Medical University, Syracuse, NY, USA. Electronic address: pruthis@upstate.edu.
[Ti] Title:The misleading electrocardiogram - Midventricular Takotsubo masquerading as anterior wall STEMI.
[So] Source:Am J Emerg Med;35(10):1586.e3-1586.e4, 2017 Oct.
[Is] ISSN:1532-8171
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Midventricular Takotsubo cardiomyopathy (TC) is a relatively rare variant of stress induced cardiomyopathy, wherein there is akinesis of only the mid-ventricular segment with or without hyperkinesis of apical and basal ventricular segments. The ECG findings of TC typically do not include ST segment elevation in V1-V2 and are unlikely to be associated with reciprocal changes in inferior leads. The presence of these changes argues strongly in favor of anterior wall STEMI. In our patient, the ECG had changes strongly suggestive of STEMI, however had normal coronaries on cardiac catheterization. This case highlights the importance of carefully analyzing the ECG in emergency situations for differences between TC and STEMI and when in doubt, cardiac catheterization should be pursued.
[Mh] MeSH terms primary: Diagnostic Errors
Electrocardiography
Heart Ventricles/diagnostic imaging
ST Elevation Myocardial Infarction/diagnosis
Takotsubo Cardiomyopathy/diagnosis
[Mh] MeSH terms secundary: Diagnosis, Differential
Echocardiography
Female
Heart Ventricles/physiopathology
Humans
Middle Aged
Takotsubo Cardiomyopathy/blood
Troponin I/blood
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:0 (Troponin I)
[Em] Entry month:1710
[Cu] Class update date: 171012
[Lr] Last revision date:171012
[Js] Journal subset:IM
[Da] Date of entry for processing:170805
[St] Status:MEDLINE


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