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Search on : Hypoglossal and Nerve and Diseases [Words]
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[PMID]: 29452069
[Au] Autor:Shibata A; Kimura M; Ishibashi K; Umemura M
[Ad] Address:Clinical Fellow, Department of Oral and Maxillofacial Surgery, Ogaki Municipal Hospital, Ogaki, Japan. Electronic address: akio.st83@gmal.com.
[Ti] Title:Idiopathic Isolated Unilateral Hypoglossal Nerve Palsy: A Report of 2 Cases and Review of the Literature.
[So] Source:J Oral Maxillofac Surg;, 2018 Feb 02.
[Is] ISSN:1531-5053
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Hypoglossal nerve palsy (HNP) is a common finding in neurologic diseases when associated with other cranial nerve palsies or further pathology and exhibits characteristic clinical manifestations, including unilateral atrophy of the musculature of the tongue. It occasionally appears as the initial or solitary sign of an intracranial or extracranial space-occupying lesion, head or neck injury, or vascular abnormality of the internal carotid artery. There are few cases of idiopathic isolated unilateral HNP, which should be diagnosed through exclusion. This report describes 2 patients who had different outcomes and presents a literature review of idiopathic isolated unilateral HNP. Case 1 was a 71-year-old man who was referred with a 1-month history of dysphagia and speech impairment. Intraoral examination disclosed marked left-side hemiatrophy of the tongue and deviation toward the left on protrusion. At coronal Tl-weighted magnetic resonance imaging, left-side hemiatrophy of the tongue was clearly visible through deviation of the median septum to the left. The patient was diagnosed with idiopathic isolated unilateral HNP through exclusion and was treated with steroids and mecobalamin, but he did not recover. Case 2 was a 32-year-old man complaining of tongue weakness for 2 days. On examination, left HNP was evident, with deviation of the tongue to the left on protrusion. He was diagnosed with idiopathic isolated unilateral HNP through exclusion and was treated with steroids. After 3 weeks, the patient had completely recovered. To the best of the authors' knowledge, this is the first detailed literature review on idiopathic isolated unilateral HNP. This condition is very rare but should be considered for diagnosis. It warrants a thorough and stepwise approach for etiologic diagnosis.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180304
[Lr] Last revision date:180304
[St] Status:Publisher

  2 / 936 MEDLINE  
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[PMID]: 29455904
[Au] Autor:Marino SD; Schiavone L; La Mendola FMC; Timpanaro T; Cucuzza ME; Greco F; Smilari P; Fiumara A; Praticò AD
[Ad] Address:Section of Pediatrics and Child Neuropsychiatry, Department of Clinical and Experimental Medicine, University of Catania, Catania, Italy.
[Ti] Title:Hypoglossal nerve paralysis in a child after a dental procedure.
[So] Source:Neurol Neurochir Pol;, 2018 Feb 06.
[Is] ISSN:0028-3843
[Cp] Country of publication:Poland
[La] Language:eng
[Ab] Abstract:Unilateral palsy of the hypoglossal nerve is a rare complication of orthodontic procedures. The main reported causes of HNP are: orthopedic and otorhinolaryngology surgical interventions, and in particular maneuvers involving compression or overstretching of the hypoglossal nerve, dental procedures and traumas, and also infections, motoneuron disorders, tumors, vascular diseases. Diagnosis is usually performed by electrophysiology studies (EMG-VCN), and brain magnetic resonance imaging (MRI) is useful to exclude other causes. The prognosis depends on the location and extension of the damage. Currently there is not a standardized treatment approach except the speech therapy, although, in some cases, the high-dose steroid treatment could be useful. We describe the case of a ten-year-old female, who was admitted in our Unit after a deviation of the tongue associated with dysarthria and dysphagia, occurred after the application of a mobile orthodontic device.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180219
[Lr] Last revision date:180219
[St] Status:Publisher

  3 / 936 MEDLINE  
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[PMID]: 29191538
[Au] Autor:Oushy S; Graffeo CS; Perry A; Morris JM; Carlson ML; Van Gompel JJ
[Ad] Address:Department of Neurologic Surgery, Mayo Clinic, Rochester, Minnesota, USA.
[Ti] Title:Collet-Sicard Syndrome Attributable to Extramedullary Plasmacytoma of the Jugular Foramen.
[So] Source:World Neurosurg;110:386-390, 2018 Feb.
[Is] ISSN:1878-8769
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:BACKGROUND: Collet-Sicard syndrome is a rare manifestation of skull base disease involving the jugular and hypoglossal foramina. We report the first case of Collet-Sicard attributable to extramedullary plasmacytoma-multiple myeloma (EP-MM) and the second case of EP-MM precipitating a jugular foramen syndrome (JFS)-spectrum disorder. CASE DESCRIPTION: A 59-year-old woman presented with 4 months of left aural fullness and pulsatile tenderness, positional vertigo, hoarseness, and dysphagia. Examination identified left tongue weakness and nonspecific sensory abnormalities of the ear, pharynx, and throat localizing to cranial nerves IX-XII. Imaging revealed a 3.4 × 1.4 × 2.8 cm lytic lesion extending from the left jugular foramen into the posterior fossa, for which she was referred to neurosurgery and otolaryngology for consideration of resection. A second, much smaller (1.1-cm) lytic lesion in the left posterior occipital bone was incidentally discovered by the surgeon during preoperative consultation. A stereotactic biopsy of the occipital lesion was subsequently recommended, which identified plasma cell neoplasm. Serum studies and skeletal survey were consistent with MM, and a definitive pathologic diagnosis of MM with cranial EP was confirmed by bone marrow biopsy. CONCLUSION: Tumors of the jugular foramen present with a diverse array of lower cranial nerve deficits, including Collet-Sicard syndrome, a rare subset of JFS-spectrum diseases. Paragangliomas are the most common jugular foramen neoplasms, followed by schwannomas and meningiomas; however, many other rare entities have been reported as masqueraders, and diligent work-up with consideration for preliminary biopsy is recommended, particularly in the presence of additional lesions, equivocal imaging findings, or cases arousing high suspicion.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 180213
[Lr] Last revision date:180213
[St] Status:In-Data-Review

  4 / 936 MEDLINE  
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[PMID]: 29187691
[Au] Autor:Fujiwara S; Yoshimura H; Nishiya K; Oshima K; Kawamoto M; Kohara N
[Ad] Address:Department of Neurology, Kobe City Medical Center General Hospital.
[Ti] Title:[Tapia's syndrome following transesophageal echocardiography during an open-heart operation: a case report].
[So] Source:Rinsho Shinkeigaku;57(12):785-787, 2017 Dec 27.
[Is] ISSN:1882-0654
[Cp] Country of publication:Japan
[La] Language:jpn
[Ab] Abstract:A 67-year-old man presented with hoarseness, dysarthria and deviation of the tongue to the left side the day after the open-heart operation under general anesthesia. Brain MRI demonstrated no causal lesion, and laryngoscope showed left vocal cord abductor palsy, so we diagnosed him with Tapia's syndrome (i.e., concomitant paralysis of the left recurrent and hypoglossal nerve). His neurological symptoms recovered gradually and improved completely four months after the onset. Tapia's syndrome is a rare condition caused by the extra cranial lesion of the recurrent laryngeal branch of the vagus nerve and the hypoglossal nerve, and mostly described as a complication of tracheal intubation. In this case, transesophageal echo probe has been held in the left side of the pharynx, so compression to the posterior wall of pharynx by the probe resulted in this condition, and to the best of our knowledge, this is the first report of Tapia's syndrome due to transesophageal echocardiography during an open-heart operation. This rare syndrome should be considered as a differential diagnosis of dysarthria and tongue deviation after a procedure associated with compression to the pharynx.
[Mh] MeSH terms primary: Cardiac Surgical Procedures
Echocardiography, Transesophageal/adverse effects
Hypoglossal Nerve Diseases/etiology
Intraoperative Complications/etiology
Vocal Cord Paralysis/etiology
[Mh] MeSH terms secundary: Aged
Diagnosis, Differential
Dysarthria/etiology
Echocardiography, Transesophageal/instrumentation
Humans
Hypoglossal Nerve Diseases/diagnosis
Male
Syndrome
Vocal Cord Paralysis/diagnosis
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180112
[Lr] Last revision date:180112
[Js] Journal subset:IM
[Da] Date of entry for processing:171201
[St] Status:MEDLINE
[do] DOI:10.5692/clinicalneurol.cn-001097

  5 / 936 MEDLINE  
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[PMID]: 27772673
[Au] Autor:Gold M; Fenig M
[Ad] Address:Department of Radiology, Lincoln Medical Center, New York, NY; Weill Cornell Medical Center, New York, NY.
[Ti] Title:Male With Pain in His Neck.
[So] Source:Ann Emerg Med;68(5):545-561, 2016 Nov.
[Is] ISSN:1097-6760
[Cp] Country of publication:United States
[La] Language:eng
[Mh] MeSH terms primary: Back Pain/diagnostic imaging
Cervical Vertebrae
Hypoglossal Nerve Diseases/diagnostic imaging
Neck Pain/diagnostic imaging
[Mh] MeSH terms secundary: Adult
Back Pain/etiology
Humans
Hypoglossal Nerve Diseases/etiology
Male
Neck Pain/etiology
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171211
[Lr] Last revision date:171211
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:161025
[St] Status:MEDLINE

  6 / 936 MEDLINE  
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[PMID]: 28899572
[Au] Autor:Torbus-Paluszczak M; Mucha S; Wawrzynczyk M; Pierzchala K; Bartman W; Adamczyk-Sowa M
[Ad] Address:Department of Neurology in Zabrze, Medical University of Silesia, ul. 3-go Maja 13-15, Zabrze 41-800, Poland. Electronic address: magda.torbus@wp.pl.
[Ti] Title:Hypoglossal nerve palsy in the course of dissection of the internal carotid arteries - Case reports.
[So] Source:Neurol Neurochir Pol;, 2017 Aug 30.
[Is] ISSN:0028-3843
[Cp] Country of publication:Poland
[La] Language:eng
[Ab] Abstract:Internal carotid artery dissection (ICAD) has become an increasingly recognized cause of cerebrovascular accidents in young and middle-aged patients. We report 2 cases of hypoglossal nerve palsy in the course of dissection of the internal carotid arteries. The first patient was admitted to the Department of Neurology due to swallowing difficulty, speech articulation disorders and numbness of the right half of the tongue for 4 weeks. Extracranial vessel ultrasound (US) and transcranial colour Doppler (TCD) visualized thrombus causing occlusion of the right internal carotid artery (RICA). Angio-CT revealed a compression on right XII nerve and a dissection of the RICA. The second patient was referred to the Department of Neurology due to articulation disorders and swallowing difficulties. On admission, neurological examination revealed tongue deviation towards the right side with evidence of atrophy of the right half of the tongue, deviation of the uvula to the right side, absence of palatal and pharyngeal reflexes, rhinolalia and dysphagia. Vessel imaging was taken using angio-MR showing mural thrombus of the RICA. CONCLUSION: The diagnosis of spontaneous non-traumatic dissection of the carotid arteries is a major challenge for clinicians. ICAD must be considered for young and middle-aged patients when severe headache is preceded by the co-existence of focal neurological symptoms. The probability of ICAD increases in the presence of predisposing diseases. The final diagnosis is based on imaging studies: color duplex ultrasound, CT angiography or MR angiography.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1709
[Cu] Class update date: 170913
[Lr] Last revision date:170913
[St] Status:Publisher

  7 / 936 MEDLINE  
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[PMID]: 28527784
[Au] Autor:Andrzejewski K; Budzinska K; Kaczynska K
[Ad] Address:Laboratory of Respiration Physiology, Mossakowski Medical Research Centre, Polish Academy of Sciences, Warsaw, Poland. Electronic address: kandrzejewski@imdik.pan.pl.
[Ti] Title:Phrenic and hypoglossal nerve activity during respiratory response to hypoxia in 6-OHDA unilateral model of Parkinson's disease.
[So] Source:Life Sci;180:143-150, 2017 Jul 01.
[Is] ISSN:1879-0631
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:AIMS: Parkinson's disease (PD) patients apart from motor dysfunctions exhibit respiratory disturbances. Their mechanism is still unknown and requires investigation. Our research was designed to examine the activity of phrenic (PHR) and hypoglossal (HG) nerves activity during a hypoxic respiratory response in the 6-hydroxydopamine (6-OHDA) model of PD. MAIN METHODS: Male adult Wistar rats were injected unilaterally with 6-OHDA (20µg) or the vehicle into the right medial forebrain bundle (MFB). Two weeks after the surgery the activity of the phrenic and hypoglossal nerve was registered in anesthetized, vagotomized, paralyzed, and mechanically ventilated rats under normoxic and hypoxic conditions. Lesion effectiveness was confirmed by the cylinder test, performed before the MFB injection and 14days after, before the respiratory experiment. KEY FINDINGS: 6-OHDA lesioned animals showed a significant increase in normoxic inspiratory time. Expiratory time and total time of the respiratory cycle were prolonged in PD rats after hypoxia. The amplitude of the PHR activity and its minute activity were increased in comparison to the sham group at recovery time and during 30s of hypoxia. The amplitude of the HG activity was increased in response to hypoxia in 6-OHDA lesioned animals. The degeneration of dopaminergic neurons decreased the pre-inspiratory/inspiratory ratio of the hypoglossal burst amplitude during and after hypoxia. SIGNIFICANCE: Unilateral MFB lesion changed the activity of the phrenic and hypoglossal nerves. The altered pre-inspiratory hypoglossal nerve activity indicates modifications to the central mechanisms controlling the activity of the HG nerve and may explain respiratory disorders seen in PD, i.e. apnea.
[Mh] MeSH terms primary: Hypoglossal Nerve/metabolism
Hypoxia/metabolism
Parkinsonian Disorders/physiopathology
Phrenic Nerve/metabolism
Respiration
[Mh] MeSH terms secundary: Animals
Disease Models, Animal
Male
Oxidopamine/toxicity
Rats
Rats, Wistar
Respiration Disorders/etiology
Respiration Disorders/physiopathology
Respiration, Artificial
Time Factors
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:8HW4YBZ748 (Oxidopamine)
[Em] Entry month:1706
[Cu] Class update date: 170629
[Lr] Last revision date:170629
[Js] Journal subset:IM
[Da] Date of entry for processing:170522
[St] Status:MEDLINE

  8 / 936 MEDLINE  
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[PMID]: 28522413
[Au] Autor:Tanaka T; Murakami K; Bando Y; Nomura T; Isonishi A; Morita-Takemura S; Tatsumi K; Wanaka A; Yoshida S
[Ad] Address:Department of Functional Anatomy and Neuroscience, Asahikawa Medical University, 2-1-1-1, Midorigaoka-higashi, Asahikawa, Hokkaido 078-8510, Japan; Department of Anatomy and Neuroscience, Nara Medical University, 840, Shijo-cho, Kashihara, Nara 634-8521, Japan. Electronic address: ttanaka@naramed-u.
[Ti] Title:Microglia support ATF3-positive neurons following hypoglossal nerve axotomy.
[So] Source:Neurochem Int;108:332-342, 2017 Sep.
[Is] ISSN:1872-9754
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Microglia are essential in developmental processes and maintenance of neuronal homeostasis. Experimental axotomy of motor neurons results in neurodegeneration, and microglia in motor nuclei become activated and migrate towards injured neurons. However, whether these activated microglia are protective or destructive to neurons remains controversial. In the present study, we transected the hypoglossal nerve in BALB/c mice, causing activating transcription factor 3 (ATF3) and growth associated protein 43 (GAP43) induction, and partial neuronal death. Inhibition of microglial accumulation by minocycline administration impaired microglial accumulation, decreased GAP43 mRNA expression, and reduced motor neuron survival. Expression of ATF3 contributed to nerve regeneration, and increased within 6 h after axotomy, prior to microglial migration. Further, microglial contact with neuronal cell bodies was associated with neuronal ATF3 expression. Colchicine administration blocked lesion-induced ATF3 transcription in axotomized neurons and microglial accumulation. In addition, perineuronal microglia-derived ciliary neurotrophic factor (CNTF) increased, indicating that perineuronal microglia in the hypoglossal nucleus protect axotomized motor neurons by releasing trophic factors. We also observed that microglia secrete CNTF and that neurons have CNTFRα and can respond to it in vitro. CNTF promote neurite elongation and neuronal survival of primary cultured neurons. Microglia make contact through unknown neuronal signals that are possibly regulated by ATF3 in hypoglossal nucleus. Moreover, they play important roles in regenerating motor neurons and are potential new therapeutic targets for motor neuron diseases.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1705
[Cu] Class update date: 170715
[Lr] Last revision date:170715
[St] Status:In-Process

  9 / 936 MEDLINE  
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[PMID]: 28415060
[Au] Autor:Inaka Y; Otani N; Nishida S; Ueno H; Tomiyama A; Tomura S; Toyooka T; Wada K; Mori K
[Ad] Address:Department of Neurosurgery, National Defense Medical College.
[Ti] Title:[A Case of Foramen Magnum Meningioma Manifesting as Hypoglossal Nerve Palsy].
[So] Source:No Shinkei Geka;45(4):345-350, 2017 Apr.
[Is] ISSN:0301-2603
[Cp] Country of publication:Japan
[La] Language:jpn
[Ab] Abstract:We report a case of foramen magnum meningioma manifesting as hypoglossal nerve palsy. A 72-year-old woman presented with progressive hypoglossal nerve palsy and lingual atrophy on the left side. Gadolinium-enhanced T1-weighted magnetic resonance imaging revealed a heterogeneously enhanced mass lesion with dural tail sign partially extending into the hypoglossal canal. The transcondylar approach was performed to expose the hypoglossal canal and resect the tumor completely. Histological examination revealed a transitional meningioma. The postoperative course was uneventful. Hypoglossal nerve palsy improved gradually after the operation.
[Mh] MeSH terms primary: Foramen Magnum/surgery
Hypoglossal Nerve Diseases/surgery
Meningeal Neoplasms/surgery
Meningioma/surgery
Skull Base Neoplasms/surgery
[Mh] MeSH terms secundary: Aged
Female
Foramen Magnum/pathology
Humans
Hypoglossal Nerve Diseases/diagnosis
Magnetic Resonance Imaging/methods
Meningeal Neoplasms/diagnosis
Meningioma/diagnosis
Skull Base Neoplasms/diagnosis
Treatment Outcome
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Em] Entry month:1708
[Cu] Class update date: 170831
[Lr] Last revision date:170831
[Js] Journal subset:IM
[Da] Date of entry for processing:170418
[St] Status:MEDLINE
[do] DOI:10.11477/mf.1436203507

  10 / 936 MEDLINE  
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[PMID]: 28161565
[Au] Autor:Illuminati G; Schneider F; Pizzardi G; Masci F; Calio' FG; Ricco JB
[Ad] Address:The Department of Surgical Sciences, University of Rome "La Sapienza", Rome, Italy. Electronic address: giulio.illuminati@uniroma1.it.
[Ti] Title:Dual Antiplatelet Therapy Does Not Increase the Risk of Bleeding After Carotid Endarterectomy: Results of a Prospective Study.
[So] Source:Ann Vasc Surg;40:39-43, 2017 Apr.
[Is] ISSN:1615-5947
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:BACKGROUND: The purpose of this study was to evaluate the risk of bleeding and other postoperative complications of carotid endarterectomy (CEA) in patients receiving dual antiplatelet therapy (DAPT). METHODS: From January 2005 to December 2015, 188 consecutive patients undergoing CEA and receiving DAPT (aspirin 100 mg + clopidogrel 75 mg) were enrolled in a prospective study. All of them underwent coronary artery stenting with drug-eluting stents during the 6 months preceding CEA. In the entire series, DAPT was continued until the evening before CEA and resumed on the evening of the operation. All patients received intraoperative heparinization (5,000 IU before carotid clamping), which was reversed in 5 patients. In addition, all of them were given 2,000 units of enoxaparin every 12 hr after the operation, beginning 6 hr after completion of the operation, and until discharge. All the patients presented with carotid artery stenosis >70% (North American Symptomatic Carotid Endarterectomy Trial [NASCET] criteria), which was symptomatic in 42 patients (transient ischemic attack, n = 32; minor stroke, n = 10) and asymptomatic in 146. The CEA technique was standard, with prosthetic patch closure in 109 cases (58%) and eversion in 79 (42%). The primary endpoints of the study were occurrence of a postoperative cervical hematoma requiring surgical hemostasis and occurrence of cranial nerve injuries. The secondary endpoint was the combined rate of postoperative mortality, stroke, and myocardial ischemia. RESULTS: No postoperative cervical hematoma requiring surgical evacuation occurred in this series. One hypoglossal nerve palsy, regressive within 2 weeks, was observed. Postoperative mortality and neurologic and cardiac morbidity were nil. CONCLUSIONS: CEA under DAPT yields results comparable with those obtained in patients receiving a single antiplatelet treatment. No hemorrhagic complications were observed in this prospective series.
[Mh] MeSH terms primary: Aspirin/administration & dosage
Carotid Stenosis/surgery
Endarterectomy, Carotid/adverse effects
Platelet Aggregation Inhibitors/administration & dosage
Postoperative Hemorrhage/etiology
Ticlopidine/analogs & derivatives
[Mh] MeSH terms secundary: Aged
Aged, 80 and over
Anticoagulants/administration & dosage
Aspirin/adverse effects
Asymptomatic Diseases
Carotid Stenosis/complications
Carotid Stenosis/diagnostic imaging
Carotid Stenosis/mortality
Drug Therapy, Combination
Endarterectomy, Carotid/mortality
Female
Humans
Ischemic Attack, Transient/etiology
Male
Middle Aged
Platelet Aggregation Inhibitors/adverse effects
Postoperative Hemorrhage/chemically induced
Postoperative Hemorrhage/mortality
Prospective Studies
Risk Assessment
Risk Factors
Severity of Illness Index
Stroke/etiology
Ticlopidine/administration & dosage
Ticlopidine/adverse effects
Time Factors
Treatment Outcome
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Anticoagulants); 0 (Platelet Aggregation Inhibitors); A74586SNO7 (clopidogrel); OM90ZUW7M1 (Ticlopidine); R16CO5Y76E (Aspirin)
[Em] Entry month:1709
[Cu] Class update date: 170925
[Lr] Last revision date:170925
[Js] Journal subset:IM
[Da] Date of entry for processing:170206
[St] Status:MEDLINE


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