Database : MEDLINE
Search on : Lateral and Medullary and Syndrome [Words]
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[PMID]: 29426369
[Au] Autor:Ogawa T; Shojima Y; Kuroki T; Eguchi H; Hattori N; Miwa H
[Ad] Address:Department of Neurology, Juntendo University Nerima Hospital, 3-1-10 Takanodai, Nerima, Tokyo, 177-8521, Japan.
[Ti] Title:Cervico-shoulder dystonia following lateral medullary infarction: a case report and review of the literature.
[So] Source:J Med Case Rep;12(1):34, 2018 Feb 10.
[Is] ISSN:1752-1947
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Secondary cervical dystonia is induced by organic brain lesions involving the basal ganglia, thalamus, cerebellum, and brain stem. It is extremely rare to see cervical dystonia induced by a medullary lesion. CASE PRESENTATION: We report a case of an 86-year-old Japanese woman who developed cervical dystonia following lateral medullary infarction. She developed sudden-onset left upper and lower extremity weakness, right-side numbness, and dysarthria. Brain magnetic resonance imaging revealed an acute ischemic lesion involving the left lateral and dorsal medullae. A few days after her stroke, she complained of a taut sensation in her left neck and body, and cervico-shoulder dystonia toward the contralateral side subsequently appeared. Within a few weeks, it disappeared spontaneously, but her hemiplegia remained residual. CONCLUSIONS: To date, to the best of our knowledge, there has been only one reported case of cervical dystonia associated with a single medullary lesion. It is interesting to note the similarities in the clinical characteristics of the previously reported case and our patient: the involvement of the dorsal and caudal parts of the medullary and associated ipsilateral hemiplegia. The present case may support the speculation that the lateral and caudal regions of the medulla may be the anatomical sites responsible for inducing cervical dystonia.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180216
[Lr] Last revision date:180216
[St] Status:In-Process
[do] DOI:10.1186/s13256-018-1561-y

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[PMID]: 29433118
[Au] Autor:Himeno T; Takeshima S; Kubo S; Hara N; Tanaka A; Kuriyama M
[Ad] Address:Department of Neurology, Brain Attack Center, Ota Memorial Hospital.
[Ti] Title:[Trigeminal Herpes Zoster Presenting with High-intensity Signals for the Spinal Trigeminal Nucleus and Tract on Diffusion-weighted Image: A Case Report of Stroke Mimic].
[So] Source:Brain Nerve;70(2):161-164, 2018 Feb.
[Is] ISSN:1881-6096
[Cp] Country of publication:Japan
[La] Language:jpn
[Ab] Abstract:A 50-year-old woman was admitted to our hospital with dysesthesia on the right upper portion of her face and a headache. Diffusion-weighted brain magnetic resonance imaging (MRI) revealed high-intensity signals in the dorsolateral portion of the medulla oblongata. She was diagnosed with lateral medullary infarction and was intravenously treated with sodium ozagrel. On the second day of hospitalization, she had nausea and vomiting and showed nystagmus to all directions, suggesting damage to the vestibular nucleus. These manifestations coincided with partial symptoms of lateral medullary syndrome. On the third day of hospitalization, a rash appeared on the region of skin innervated by the first and second branches of the right trigeminal nerve. A reevaluation of the MRI findings indicated the presence of a lesion of the right spinal trigeminal nucleus and tract. She was treated with acyclovir for 14 days, and was discharged without any residual symptoms. Varicella zoster virus-DNA was detected in her cerebrospinal fluid. This disease mimics the presentation of a stroke and is important for differential diagnosis. (Received August 1, 2017; Accepted September 14, 2017; Published February 1, 2018).
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180212
[Lr] Last revision date:180212
[St] Status:In-Data-Review
[do] DOI:10.11477/mf.1416200969

  3 / 1182 MEDLINE  
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[PMID]: 29217256
[Au] Autor:Yeo SS; Jang SH; Kwon JW
[Ad] Address:Department of Physical Therapy, College of Health Sciences, Dankook University, Republic of Korea. Electronic address: eangbul@hanmail.net.
[Ti] Title:Lateral medullary syndrome following injury of the vestibular pathway to the core vestibular cortex: Diffusion tensor imaging study.
[So] Source:Neurosci Lett;665:147-151, 2018 Feb 05.
[Is] ISSN:1872-7972
[Cp] Country of publication:Ireland
[La] Language:eng
[Ab] Abstract:OBJECTIVE: The parieto-insular vestibular cortex (PIVC) is a core region of vestibular input into regions of the cortex. The vestibular nuclei have reciprocal connections with the PIVC. However, little is known about injury of the core vestibular pathway to the PIVC in patients with dorsolateral medullary infarctions. In this study, using diffusion tensor tractography (DTT), we investigated injury of the neural connections between the vestibular nuclei and the PIVC in patients with typical central vestibular disorder. METHODS: Eight consecutive patients with lateral medullary syndrome and 10 control subjects were recruited for this study. To reconstruct the core vestibular pathway to the PIVC, we defined the seed region of interest (ROI) as the vestibular nuclei of the pons and the target ROI as the PIVC. Fractional anisotropy (FA), mean diffusivity (MD), and tract volume were measured. RESULT: The core vestibular pathway to the PIVC showed significantly lower tract volume in patients compared with the control group (p<0.05). By contrast, other DTI parameters did not show significant differences between the patient and control groups (p>0.05). CONCLUSION: In conclusion, injury of the core vestibular pathway to the PIVC was demonstrated in patients with lateral vestibular syndrome following dorsolateral medullary infarcts. We believe that analysis of the core vestibular pathway to the PIVC using DTT would be helpful in evaluating patients with lateral medullary syndrome.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 180206
[Lr] Last revision date:180206
[St] Status:In-Data-Review

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[PMID]: 29137102
[Au] Autor:Han J; Cao D; Ma G; Wang T; Ji Y; Kang Z
[Ad] Address:aDepartment of Neurology bDepartment of MRI, Harrison International Peace Hospital, Hengshui, Hebei, China.
[Ti] Title:Lateral medullary infarction with similar features of Brown Sequard syndrome caused by vertebrobasilar dysplasia and Klippel-Feil syndrome: A case report.
[So] Source:Medicine (Baltimore);96(45):e8647, 2017 Nov.
[Is] ISSN:1536-5964
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:RATIONALE: Patients with Klippel-Feil syndrome (KFS) are always anomaly associated with vertebrobasilar dysplasia. That may present commonly as infarction of brainstem, medulla, and cerebellum. In this article, we reported a rare case of lateral medullary infarction (LMI) with similar features of Brown Sequard syndrome caused by vertebrobasilar dysplasia and KFS, and the 2 rare conditions that are causally related. The case is being reported because of its unusual and rare presentation. PATIENT CONCERNS: A 38-year-old female presented with acute unsteadiness, along with a tendency to lean to the left side while walking or sitting, and paresthesia in the right lower limb and trunk, at 2 days before admission. She had no history of hypertension and diabetes, but had a 20 years history of neck pain and dizziness, which was related to head movement. DIAGNOSES: Brown Sequard syndrome and a lesion of the left thoracic spinal cord were suspected initially. KFS was confirmed by the cervical magnetic resonance imaging (MRI) and computed tomographic angiography (CTA) results. Transcranial Doppler (TCD) results confirmed that there was a causal link between LMI and KFS. INTERVENTIONS: The patient rejected the operation of stabilization of the cervical spine with fusion at appropriate levels. OUTCOMES: No recurrence of stroke, but neck pain and dizziness remained after 6 months of discharge. LESSONS: For such patients, the conventional treatment of cerebral infarction might be ineffective, but stabilization of the cervical spine with fusion at appropriate levels can successfully prevent further episodes of syncope and stroke.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1711
[Cu] Class update date: 171115
[Lr] Last revision date:171115
[St] Status:In-Process
[do] DOI:10.1097/MD.0000000000008647

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[PMID]: 29026772
[Au] Autor:Thomas VV; George T; Mishra AK; Mannam P; Ramya I
[Ad] Address:Department of Medicine, Christian Medical College, Vellore, Tamil Nadu, India.
[Ti] Title:Lateral medullary syndrome after a scorpion sting.
[So] Source:J Family Med Prim Care;6(1):155-157, 2017 Jan-Mar.
[Is] ISSN:2249-4863
[Cp] Country of publication:India
[La] Language:eng
[Ab] Abstract:Scorpion bites are a common problem in Southern parts of India. The sting of belonging to the family is known for being fatal. The toxidrome of scorpion sting is known for its effect on the cardiovascular system, and there have been rare reports of cerebrovascular accidents as well. We describe a case of lateral medullary syndrome secondary to scorpion sting. As per the knowledge of the authors, this is the first case report of the same.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1710
[Cu] Class update date: 171017
[Lr] Last revision date:171017
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.4103/2249-4863.214988

  6 / 1182 MEDLINE  
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[PMID]: 28931039
[Au] Autor:Bright FM; Vink R; Byard RW; Duncan JR; Krous HF; Paterson DS
[Ad] Address:Discipline of Anatomy and Pathology, Adelaide Medical School, University of Adelaide, Adelaide, SA, Australia.
[Ti] Title:Abnormalities in substance P neurokinin-1 receptor binding in key brainstem nuclei in sudden infant death syndrome related to prematurity and sex.
[So] Source:PLoS One;12(9):e0184958, 2017.
[Is] ISSN:1932-6203
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Sudden infant death syndrome (SIDS) involves failure of arousal to potentially life threatening events, including hypoxia, during sleep. While neuronal dysfunction and abnormalities in neurotransmitter systems within the medulla oblongata have been implicated, the specific pathways associated with autonomic and cardiorespiratory failure are unknown. The neuropeptide substance P (SP) and its tachykinin neurokinin-1 receptor (NK1R) have been shown to play an integral role in the modulation of homeostatic function in the medulla, including regulation of respiratory rhythm generation, integration of cardiovascular control, and modulation of the baroreceptor reflex and mediation of the chemoreceptor reflex in response to hypoxia. Abnormalities in SP neurotransmission may therefore result in autonomic dysfunction during sleep and contribute to SIDS deaths. [125I] Bolton Hunter SP autoradiography was used to map the distribution and density of the SP, NK1R to 13 specific nuclei intimately related to cardiorespiratory function and autonomic control in the human infant medulla of 55 SIDS and 21 control (non-SIDS) infants. Compared to controls, SIDS cases exhibited a differential, abnormal developmental profile of the SP/NK1R system in the medulla. Furthermore the study revealed significantly decreased NK1R binding within key medullary nuclei in SIDS cases, principally in the nucleus tractus solitarii (NTS) and all three subdivisions of the inferior portion of the olivo-cerebellar complex; the principal inferior olivary complex (PIO), medial accessory olive (MAO) and dorsal accessory olive (DAO). Altered NK1R binding was significantly influenced by prematurity and male sex, which may explain the increased risk of SIDS in premature and male infants. Abnormal NK1R binding in these medullary nuclei may contribute to the defective interaction of critical medullary mechanisms with cerebellar sites, resulting in an inability of a SIDS infant to illicit appropriate respiratory and motor responses to life threatening challenges during sleep. These observations support the concept that abnormalities in a multi-neurotransmitter network within key nuclei of the medullary homeostatic system may underlie the pathogenesis of a subset of SIDS cases.
[Mh] MeSH terms primary: Brain Stem/pathology
Infant, Premature/metabolism
Medulla Oblongata/pathology
Olivary Nucleus/pathology
Receptors, Neurokinin-1/metabolism
Substance P/metabolism
Sudden Infant Death/pathology
[Mh] MeSH terms secundary: Brain Stem/metabolism
Cohort Studies
Female
Humans
Infant
Infant, Newborn
Male
Medulla Oblongata/metabolism
Olivary Nucleus/metabolism
Protein Binding
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Receptors, Neurokinin-1); 33507-63-0 (Substance P)
[Em] Entry month:1710
[Cu] Class update date: 171017
[Lr] Last revision date:171017
[Js] Journal subset:IM
[Da] Date of entry for processing:170921
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0184958

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[PMID]: 28922849
[Au] Autor:Bright FM; Byard RW; Vink R; Paterson DS
[Ad] Address:Faculty of Health and Medical Science, School of Medicine, University of Adelaide, Adelaide, South Australia, Australia; Department of Pathology, Boston Children's Hospital and Harvard Medical School, Boston, Massachusetts; Sansom Institute for Health Research, University of South Australia, Adelaid
[Ti] Title:Medullary Serotonin Neuron Abnormalities in an Australian Cohort of Sudden Infant Death Syndrome.
[So] Source:J Neuropathol Exp Neurol;76(10):864-873, 2017 Oct 01.
[Is] ISSN:1554-6578
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Serotonin (5-hydroxytryptamine [5-HT]) neurons in the medulla oblongata project extensively to key autonomic and respiratory nuclei in the brainstem and spinal cord regulating critical homeostatic functions. Multiple abnormalities in markers of 5-HT function in the medulla in sudden infant death syndrome (SIDS) have been reported, informing the hypothesis that at least a subset of SIDS cases is caused by deficits in 5-HT function resulting in impaired homeostatic responses to potentially life-threatening events during sleep. To investigate medullary 5-HT defects in SIDS further, we undertook qualitative analysis immunohistochemical assessment of 5-HT neuron expression within the medulla of SIDS infants (n41) and nonSIDS controls (n = 28) in an independent cohort from Forensic Science South Australia. Compared with controls SIDS cases had significantly higher 5-HT neuron numbers and density in addition to significantly altered 5-HT neuron morphology. Thus, for the first time, we replicated and corroborated previous observations of a significant abnormality in medullary 5-HT neuron expression in SIDS in a separate independent SIDS cohort. This study further supports the hypothesis that medullary 5-HT defects contribute to the pathogenesis of a subset of SIDS victims and provides additional evidence of a more complex abnormality in 5-HT neuron dysfunction specifically within the different caudal and rostral medullary 5-HT domains.
[Mh] MeSH terms primary: Medulla Oblongata/pathology
Neurons/pathology
Serotonin/metabolism
Sudden Infant Death/epidemiology
Sudden Infant Death/pathology
[Mh] MeSH terms secundary: Australia/epidemiology
Cell Count
Cohort Studies
Databases, Factual/statistics & numerical data
Female
Humans
Infant
Infant, Newborn
Male
Neurons/metabolism
Risk Factors
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:333DO1RDJY (Serotonin)
[Em] Entry month:1710
[Cu] Class update date: 171002
[Lr] Last revision date:171002
[Js] Journal subset:IM
[Da] Date of entry for processing:170920
[St] Status:MEDLINE
[do] DOI:10.1093/jnen/nlx071

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[PMID]: 28878917
[Au] Autor:Aynaci O; Gok F; Yosunkaya A
[Ad] Address:Department of Anesthesiology and ReanimationIntensive Care UnitMeram Medical FacultyNecmettin Erbakan University42080KonyaTurkey.
[Ti] Title:Management of a patient with Opalski's syndrome in intensive care unit.
[So] Source:Clin Case Rep;5(9):1518-1522, 2017 Sep.
[Is] ISSN:2050-0904
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Opalski syndrome is a rare vascular brainstem syndrome which is accepted as a variant of Wallenberg syndrome. Opalski syndrome should be considered in acute conditions in which typical symptoms of lateral medullary infarct are accompanied by ipsilateral hemiparesis. Other brain stem syndromes are distinguished from Opalski syndrome by the presence of contralateral hemiparesis.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1709
[Cu] Class update date: 170910
[Lr] Last revision date:170910
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1002/ccr3.1111

  9 / 1182 MEDLINE  
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[PMID]: 28786694
[Au] Autor:Jaimes A; García-Sáez R; Gutiérrez-Viedma Á; Cuadrado ML
[Ad] Address:1 Department of Neurology, 16267 Hospital Clínico San Carlos , Madrid, Spain.
[Ti] Title:Case report: Wallenberg's syndrome, a possible cause of symptomatic epicrania fugax.
[So] Source:Cephalalgia;:333102417726334, 2017 Jan 01.
[Is] ISSN:1468-2982
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Background Epicrania fugax has been described as a primary headache. Nevertheless, a symptomatic form was recently found in a patient with a skull base meningioma abutting the trigeminal nerve. Here we report on a patient with facial pain with the features of epicrania fugax occurring after Wallenberg's syndrome. Case report A 53-year-old man suffered a right-sided dorsolateral medullary ischaemic stroke. Nine months later, he presented with brief electric shock-like paroxysms of pain stemming from the right eye and radiating to the ipsilateral forehead, the temple or the cheek in a zigzag trajectory. Some episodes were accompanied by ipsilateral conjunctival injection and eyelid oedema. Treatment with eslicarbazepine abolished the pain. Conclusion A pain with the features of epicrania fugax may be associated with medullary lesions. This finding suggests that the central trigeminal pathways and/or the spinal trigeminal nucleus may play an important role in the pathophysiology of this type of pain.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1708
[Cu] Class update date: 171118
[Lr] Last revision date:171118
[St] Status:Publisher
[do] DOI:10.1177/0333102417726334

  10 / 1182 MEDLINE  
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[PMID]: 28769139
[Au] Autor:Shenavandeh S; Petramfar P
[Ad] Address:Shiraz University of Medical Sciences, Departments of Internal Medicine, Division of Rheumatology, Iran.
[Ti] Title:Three atypical manifestations of granulomatosis with polyangiitis: lateral medullary syndrome, anterior cheek mass and melting scleritis of eye.
[So] Source:Reumatologia;55(3):145-150, 2017.
[Is] ISSN:0034-6233
[Cp] Country of publication:Poland
[La] Language:eng
[Ab] Abstract:Granulomatosis with polyangiitis (GPA, formerly Wegener granulomatosis) is a vasculitis with various organ involvement. There have been a few cases of CNS stroke and rare cases of lateral medullary infarction (LMI) as a manifestation of GPA. Also there have been reports of sinuses, nose and laryngeal masses mistakenly referred as carcinomas and subsequently GPA was diagnosed in their pathological reports. Another severe fulminant manifestation can be necrotizing scleritis leading to perforation of sclera. Therefore, here we present some rare and fulminant manifestations of GPA in 3 separate cases for further emphasis of the unusual manifestations of GPA that should always be kept in mind.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1708
[Cu] Class update date: 170806
[Lr] Last revision date:170806
[St] Status:In-Data-Review
[do] DOI:10.5114/reum.2017.68915


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