Database : MEDLINE
Search on : Migraine and without and Aura [Words]
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[PMID]: 29221971
[Au] Autor:Ikeda K; Aoyagi J; Hanashiro S; Sawada M; Kyuzen M; Morioka H; Ebina J; Nagasawa J; Yanagihashi M; Ishikawa Y; Miura K; Murata K; Takazawa T; Kawabe K; Iwasaki Y
[Ad] Address:Department of Neurology, Toho University Omori Medical Center, Tokyo, Japan; Department of Neurology, PL Tokyo Heath Care Center, Tokyo, Japan. Electronic address: keni@med.toho-u.ac.jp.
[Ti] Title:Preventive Treatment with Lomerizine Increases Cerebral Blood Flows during the Interictal Phase of Migraine.
[So] Source:J Stroke Cerebrovasc Dis;27(4):998-1002, 2018 Apr.
[Is] ISSN:1532-8511
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:BACKGROUND: Changes in regional cerebral blood flow (rCBF) were reported in migraineurs. However, little is known how preventive medications of migraine can influence rCBF. Lomerizine, a calcium channel blocker, has been used for migraine prophylaxis in Japan. We examined rCBF after lomerizine treatment. SUBJECTS AND METHODS: Migraine was diagnosed according to the criteria of the International Classification of Headache Disorders, Third Edition beta. Migraine subtype was classified into migraine with aura (MA) and migraine without aura (MO). Lomerizine (10 mg/day, per oral) was administered for 3 months. Headache Impact Test-6 (HIT-6) and blood pressure (BP) were compared at baseline and end point. Brain single photon emission computed tomography using Tc-ethyl cysteinate dimer was performed at the interictal period. Brain SPECT data were analyzed according to revised version of 3-dimensional stereotaxic region of interest template. Clinic-radiological variables were analyzed by paired Student's t test. RESULTS: Ten migraineurs (4 men and 6 women) participated in the present study. Mean age was 54.1 (standard deviation [SD] 10.1) years. Mean duration of migraine was 25.3 (SD 9.8) years. Migraine subtype showed 4 MA and 6 MO patients. Mean score of HIT-6 was 66.3 (SD 11.7). Lomerizine treatment decreased HIT-6 scores significantly (P < .01). BP did not differ significantly after lomerizine treatment. Lomerizine treatment increased rCBF 20% approximately in the frontal, the parietal, the temporal, and the occipital region. CONCLUSIONS: The present study indicated a significant increase in interictal rCBF after lomerizine treatment in migraineurs. The upregulation of rCBF could contribute to the antimigraine mechanism of lomerizine.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 180310
[Lr] Last revision date:180310
[St] Status:In-Process

  2 / 4161 MEDLINE  
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[PMID]: 29521155
[Au] Autor:Hipolito Rodrigues MA; Maitrot-Mantelet L; Plu-Bureau G; Gompel A
[Ad] Address:a Departments of Surgery, Gynecology, Obstetrics and Propedeutics , Universidade Federal de Ouro Preto , Ouro Preto , Brazil.
[Ti] Title:Migraine, hormones and the menopausal transition.
[So] Source:Climacteric;:1-11, 2018 Mar 09.
[Is] ISSN:1473-0804
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Migraine is a common, disabling and incapacitating headache disorder that may be triggered by many factors, such as hormones especially during the perimenopausal period, where large alterations in estradiol levels can occur. The evidence implies that hormonal fluctuations are one of the important triggers of migraine. During reproductive life and during hormonal contraception, the course of migraine can be impacted. Different types of migraine with and without aura can be variously influenced by hormones. Migraine can constitute a risk factor for stroke and this must be taken in account for menopause hormone therapy. Hormone therapy is a possible approach to prevent migraine that happens during the menopause transition. Scarce data on the various regimens and types of hormone therapy are available. Transdermal estradiol displays a more favorable profile on migraine than oral estrogens because it may provide more constant levels of estrogens.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[St] Status:Publisher
[do] DOI:10.1080/13697137.2018.1439914

  3 / 4161 MEDLINE  
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[PMID]: 29459398
[Au] Autor:Pezzini A; Busto G; Zedde M; Gamba M; Zini A; Poli L; Caria F; De Giuli V; Simone AM; Pascarella R; Padovani A; Padroni M; Gasparotti R; Colagrande S; Fainardi E
[Ad] Address:From the Dipartimento di Scienze Cliniche e Sperimentali, Clinica Neurologica (A.P., L.P., F.C., V.D.G., A.P.) and Sezione di Neuroradiologia, Dipartimento di Specialità Medico-Chirurgiche, Scienze Radiologiche e Sanità Pubblica (R.G.), Università degli Studi di Brescia, Italia; Dipartimento di Scie
[Ti] Title:Vulnerability to Infarction During Cerebral Ischemia in Migraine Sufferers.
[So] Source:Stroke;49(3):573-578, 2018 03.
[Is] ISSN:1524-4628
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:BACKGROUND AND PURPOSE: Cerebral hyperexcitability in migraine experiencers might sensitize brain tissue to ischemia. We investigated whether a personal history of migraine is associated with vulnerability to brain ischemia in humans. METHODS: Multicenter cohort study of patients with acute ischemic stroke who underwent a brain computed tomography perfusion and were scheduled to undergo reperfusion therapy. In a case-control design, we compared the proportion of subjects with no-mismatch, the volume of penumbra salvaged, as well as the final infarct size in a group of patients with migraine and a group of patients with no history of migraine. RESULTS: We included 61 patients with migraine (34 [55.7%] men; mean age, 52.2±15.1 years; migraine without aura/migraine with aura, 44/17) and 61 patients with no history of migraine. The proportion of no-mismatch among migraineurs was significantly higher than among nonmigraineurs (17 [27.9%] versus 7 [11.5%]; =0.039) and was more prominent among patients with migraine with aura (6 [35.3%]; =0.030) while it was nonsignificantly increased in patients with migraine without aura (11 [25.0%]; =0.114). Migraine, especially migraine with aura, was independently associated with a no-mismatch pattern (odds ratio, 2.65; 95% CI, 0.95-7.41 for migraine; odds ratio, 5.54; 95% CI, 1.28-23.99 for migraine with aura), and there was a linear decrease of the proportion of patients with migraine with aura with increasing quartiles of mismatch volumes. Patients with migraine with aura had also smaller volumes of salvaged penumbra (9.8±41.2 mL) compared with patients with migraine without aura (36.4±54.1 mL) and patients with no migraine (45.1±55.0 mL; =0.056). Conversely, there was no difference in final infarct size among the 3 migraine subgroups ( =0.312). CONCLUSIONS: Migraine is likely to increase individual vulnerability to ischemic stroke during the process of acute brain ischemia and might represent, therefore, a potential new therapeutic target against occurrence and progression of the ischemic damage.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[St] Status:In-Process
[do] DOI:10.1161/STROKEAHA.118.020554

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[PMID]: 29517685
[Au] Autor:Ning Y; Zheng R; Li K; Zhang Y; Lyu D; Jia H; Ren Y; Zou Y
[Ad] Address:Department of Neurology and Stroke Center, Dongzhimen Hospital, the First Affiliated Hospital of Beijing University of Chinese Medicine.
[Ti] Title:The altered Granger causality connection among pain-related brain networks in migraine.
[So] Source:Medicine (Baltimore);97(10):e0102, 2018 Mar.
[Is] ISSN:1536-5964
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Numerous fMRI studies have confirmed functional abnormalities in resting-state brain networks in migraine patients. However, few studies focusing on causal relationships of pain-related brain networks in migraine have been conducted. This study aims to explore the difference of Granger causality connection among pain-related brain networks in migraine without aura (MWoA) patients.Twenty two MWoA patients and 17 matched healthy subjects were recruited to undergo resting-state fMRI scanning. Independent component analysis was used to extract pain-related brain networks, and Granger causality analysis to characterize the difference of Granger causality connection among pain-related brain networks was employed.Seven pain-related brain networks were identified, and MwoA patients showed more complex Granger causality connections in comparison with healthy subjects. Two-sample t test results displayed that there was the significant difference between right-frontoparietal network (RFPN) and executive control network (ECN).This study indicates that the specific intrinsic brain Granger causality connectivity among pain-related networks in MwoA patients are affected after long-term migraine attacks.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180308
[Lr] Last revision date:180308
[St] Status:In-Process
[do] DOI:10.1097/MD.0000000000010102

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[PMID]: 29489688
[Au] Autor:Akiyama H; Hasegawa Y
[Ti] Title:A trial case of medical treatment for primary headache using telemedicine.
[So] Source:Medicine (Baltimore);97(9):e9891, 2018 Mar.
[Is] ISSN:1536-5964
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:RATIONALE: Video-based treatment in telemedicine is a potential alternative to face-to-face treatment. We describe our trial use of telemedicine to treat a patient with primary headache. PATIENT CONCERNS: A 25-year-old woman visited our branch hospital with a chief complaint of recurrent headache. Our branch hospital had no headache specialist, so a headache specialist at our main hospital provided treatment remotely. DIAGNOSES: She was diagnosed with migraine without aura by the headache specialist using telemedicine. INTERVENTIONS: The branch hospital physician and the headache specialist used video conferencing to interview and examine the patient and share cranial magnetic resonance imaging (MRI). Audio and camera angle/zoom could be adjusted during interviews, and high-quality video was continuously displayed at both hospitals simultaneously without lag between audio and video. Temporal/spatial resolution was sufficient for proper neurological evaluation. MRI had sufficient image quality and resolution for detailed interpretation. A prescription for medication was issued by the branch hospital physician and confirmed by the headache specialist. OUTCOMES: Zolmitriptan was effective in treating her migraine. LESSONS: Telemedicine was useful for treating this primary headache patient and can contribute to regional health care. Future challenges in telemedicine include expanding its use to other areas within the purview of general physicians and headache specialists, covering implementation and maintenance costs, providing adequate explanations to patients and family members, ensuring security of video transmissions, maintaining patient medical records, and supervising treatment.
[Mh] MeSH terms primary: Migraine without Aura/diagnosis
Telemedicine/methods
Videoconferencing
[Mh] MeSH terms secundary: Adult
Female
Humans
Migraine without Aura/therapy
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180307
[Lr] Last revision date:180307
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:180301
[St] Status:MEDLINE
[do] DOI:10.1097/MD.0000000000009891

  6 / 4161 MEDLINE  
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[PMID]: 29371217
[Au] Autor:Jarvis S; Dassan P; Piercy CN
[Ad] Address:Imperial College Healthcare NHS Trust, London W12 0HS, UK sheba.jarvis@imperial.ac.uk.
[Ti] Title:Managing migraine in pregnancy.
[So] Source:BMJ;360:k80, 2018 01 25.
[Is] ISSN:1756-1833
[Cp] Country of publication:England
[La] Language:eng
[Mh] MeSH terms primary: Migraine with Aura/drug therapy
Pregnancy Complications/drug therapy
[Mh] MeSH terms secundary: Acetaminophen/therapeutic use
Adult
Analgesics, Non-Narcotic/therapeutic use
Antiemetics/therapeutic use
Female
Humans
Pregnancy
Sumatriptan/therapeutic use
Vasoconstrictor Agents/therapeutic use
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:0 (Analgesics, Non-Narcotic); 0 (Antiemetics); 0 (Vasoconstrictor Agents); 362O9ITL9D (Acetaminophen); 8R78F6L9VO (Sumatriptan)
[Em] Entry month:1803
[Cu] Class update date: 180306
[Lr] Last revision date:180306
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:180127
[St] Status:MEDLINE
[do] DOI:10.1136/bmj.k80

  7 / 4161 MEDLINE  
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[PMID]: 29386181
[Au] Autor:Adelborg K; Szépligeti SK; Holland-Bill L; Ehrenstein V; Horváth-Puhó E; Henderson VW; Sørensen HT
[Ad] Address:Department of Clinical Epidemiology, Aarhus University Hospital, Denmark kade@clin.au.dk.
[Ti] Title:Migraine and risk of cardiovascular diseases: Danish population based matched cohort study.
[So] Source:BMJ;360:k96, 2018 01 31.
[Is] ISSN:1756-1833
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:OBJECTIVE: To examine the risks of myocardial infarction, stroke (ischaemic and haemorrhagic), peripheral artery disease, venous thromboembolism, atrial fibrillation or atrial flutter, and heart failure in patients with migraine and in a general population comparison cohort. DESIGN: Nationwide, population based cohort study. SETTING: All Danish hospitals and hospital outpatient clinics from 1995 to 2013. PARTICIPANTS: 51 032 patients with migraine and 510 320 people from the general population matched on age, sex, and calendar year. MAIN OUTCOME MEASURES: Comorbidity adjusted hazard ratios of cardiovascular outcomes based on Cox regression analysis. RESULTS: Higher absolute risks were observed among patients with incident migraine than in the general population across most outcomes and follow-up periods. After 19 years of follow-up, the cumulative incidences per 1000 people for the migraine cohort compared with the general population were 25 17 for myocardial infarction, 45 25 for ischaemic stroke, 11 6 for haemorrhagic stroke, 13 11 for peripheral artery disease, 27 18 for venous thromboembolism, 47 34 for atrial fibrillation or atrial flutter, and 19 18 for heart failure. Correspondingly, migraine was positively associated with myocardial infarction (adjusted hazard ratio 1.49, 95% confidence interval 1.36 to 1.64), ischaemic stroke (2.26, 2.11 to 2.41), and haemorrhagic stroke (1.94, 1.68 to 2.23), as well as venous thromboembolism (1.59, 1.45 to 1.74) and atrial fibrillation or atrial flutter (1.25, 1.16 to 1.36). No meaningful association was found with peripheral artery disease (adjusted hazard ratio 1.12, 0.96 to 1.30) or heart failure (1.04, 0.93 to 1.16). The associations, particularly for stroke outcomes, were stronger during the short term (0-1 years) after diagnosis than the long term (up to 19 years), in patients with aura than in those without aura, and in women than in men. In a subcohort of patients, the associations persisted after additional multivariable adjustment for body mass index and smoking. CONCLUSIONS: Migraine was associated with increased risks of myocardial infarction, ischaemic stroke, haemorrhagic stroke, venous thromboembolism, and atrial fibrillation or atrial flutter. Migraine may be an important risk factor for most cardiovascular diseases.
[Mh] MeSH terms primary: Cardiovascular Diseases/etiology
Migraine Disorders/complications
Myocardial Infarction/etiology
Stroke/etiology
[Mh] MeSH terms secundary: Adult
Atrial Fibrillation/epidemiology
Atrial Fibrillation/etiology
Body Mass Index
Cardiovascular Diseases/epidemiology
Cohort Studies
Comorbidity
Denmark/epidemiology
Female
Heart Failure/epidemiology
Heart Failure/etiology
Humans
Incidence
Intracranial Hemorrhages/epidemiology
Intracranial Hemorrhages/etiology
Male
Middle Aged
Migraine Disorders/diagnosis
Migraine Disorders/epidemiology
Myocardial Infarction/epidemiology
Outcome Assessment (Health Care)
Peripheral Arterial Disease/epidemiology
Peripheral Arterial Disease/etiology
Prospective Studies
Risk Factors
Smoking/epidemiology
Stroke/epidemiology
Venous Thromboembolism/epidemiology
Venous Thromboembolism/etiology
[Pt] Publication type:COMPARATIVE STUDY; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Entry month:1803
[Cu] Class update date: 180305
[Lr] Last revision date:180305
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:180202
[St] Status:MEDLINE
[do] DOI:10.1136/bmj.k96

  8 / 4161 MEDLINE  
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[PMID]: 29476276
[Au] Autor:Miglis MG
[Ad] Address:Department of Neurology, Division of Autonomic Disorders, Stanford University, 213 Quarry Road, M/C 5992, Palo Alto, CA, 94304, USA. mmiglis@stanford.edu.
[Ti] Title:Migraine and Autonomic Dysfunction: Which Is the Horse and Which Is the Jockey?
[So] Source:Curr Pain Headache Rep;22(3):19, 2018 Feb 23.
[Is] ISSN:1534-3081
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:PURPOSE OF REVIEW: Symptoms of autonomic dysfunction are common in patients with migraine, both during and between migraine attacks. Studies evaluating objective autonomic testing in patients have found significant, though somewhat conflicting results. The purposes of this review are to summarize and interpret the key findings of these studies, including those evaluating heart rate variability, autonomic reflex testing, and functional imaging in patients with migraine. The neuroanatomy of the central autonomic network as it relates to migraine is also reviewed. RECENT FINDINGS: Several studies have evaluated autonomic balance in migraineurs, with conflicting results on the magnitude of sympathetic versus parasympathetic dysfunction. Most studies demonstrate sympathetic impairment, with a lesser degree of parasympathetic impairment. Three trends have emerged: (1) migraine with aura tends to produce more significant autonomic dysfunction than migraine without aura, (2) sympathetic impairment is more common than parasympathetic impairment, and (3) sympathetic impairment is common in the interictal period, with increased sympathetic responsiveness during the ictal period, suggesting adrenoreceptor hypersensitivity.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1802
[Cu] Class update date: 180305
[Lr] Last revision date:180305
[St] Status:In-Process
[do] DOI:10.1007/s11916-018-0671-y

  9 / 4161 MEDLINE  
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[PMID]: 29491334
[Au] Autor:Saiki M; Suzuki K; Takekawa H; Kanaya H; Kawamoto S; Nakamura T; Hirata K
[Ad] Address:Department of Neurology, Dokkyo Medical University.
[Ti] Title:[Triptan-responsive migraine-like headache caused by cavernous sinus dural arteriovenous fistula in a 69-year-old woman with a history of migraine without aura].
[So] Source:Rinsho Shinkeigaku;, 2018 Feb 28.
[Is] ISSN:1882-0654
[Cp] Country of publication:Japan
[La] Language:jpn
[Ab] Abstract:A 69-year-old woman with a previous history of migraine without aura developed throbbing headache in the right frontal region accompanied by nausea, lasting more than 4 hours a day. The headache intensity was more severe than that of usual her migraine headaches. Administration of eletriptan in the previous hospital improved her headaches. However, one month later the patient experienced more intense headaches in the same region and then was referred to our hospital. MR angiography showed abnormal signal intensities in the cavernous sinus. Cerebral angiography revealed blood reflux to the cavernous sinus, leading to diagnosis of cavernous sinus dural arteriovenous fistula. Transvenous embolization of cavernous sinus dural arteriovenous fistula was performed, which resulted in resolution of the patient's headache. We should be aware that patients with cavernous sinus dural arteriovenous fistula can manifest migraine-like headaches without being accompanied by cranial nerve palsies.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180301
[Lr] Last revision date:180301
[St] Status:Publisher
[do] DOI:10.5692/clinicalneurol.cn-001118

  10 / 4161 MEDLINE  
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[PMID]: 29384561
[Au] Autor:Gray IN; Cristancho AG; Licht DJ; Liu GT
[Ti] Title:Ocular Dipping in a Patient With Hemiplegic Migraine.
[So] Source:J Pediatr Ophthalmol Strabismus;55:e4-e6, 2018 Jan 31.
[Is] ISSN:1938-2405
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:A 5-year-old girl presented with acute, rapidly progressive encephalopathy following minor head trauma and was found to have ocular dipping. Her encephalopathy was secondary to a channelopathy caused by a CACNA1A mutation. This is the first reported case of ocular dipping in an encephalopathic child with CACNA1A-confirmed hemiplegic migraine. [J Pediatr Ophthalmol Strabismus. 2018;55:e4-e6.].
[Mh] MeSH terms primary: Cerebral Cortex/diagnostic imaging
Eye Movements/physiology
Hemiplegia/complications
Migraine with Aura/complications
Nystagmus, Pathologic/etiology
[Mh] MeSH terms secundary: Child, Preschool
Female
Hemiplegia/diagnosis
Humans
Magnetic Resonance Imaging
Migraine with Aura/diagnosis
Nystagmus, Pathologic/diagnosis
Nystagmus, Pathologic/physiopathology
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE; VIDEO-AUDIO MEDIA
[Em] Entry month:1802
[Cu] Class update date: 180228
[Lr] Last revision date:180228
[Js] Journal subset:IM
[Da] Date of entry for processing:180201
[St] Status:MEDLINE
[do] DOI:10.3928/01913913-20171129-01


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