Database : MEDLINE
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[PMID]: 29517196
[Au] Autor:Woltanowska M; Wincewicz A; Woltanowski P; Sulkowski S
[Ti] Title:Professional interest in dermatopathology of Stanislaw Ostrowski ­ the only one State Polish President among physicians. Professional interest in dermatopathology of Stanislaw Ostrowski - the only one State Polish President among physiciansesident among physicians.
[So] Source:Pol J Pathol;68(4):277-283, 2017.
[Is] ISSN:1233-9687
[Cp] Country of publication:Poland
[La] Language:eng
[Ab] Abstract:President of prewar Lvov and Polish Republic on Exile, associate professor Stanislaw Ostrowski was a dermatologist with a keen interest in dermatopathology. This study was based on original resources, which - mainly reports of his own authorship - were focused on dermatopathology. Stanislaw Ostrowski provided excellent description of naevus epitheliomatosus sebaceus Wolters-Friboes both in Polish and German to be cited after decades in renowned handbooks of dermatopahtology published by Springer Verlag. His scientific output also includes meticulous presentation of Fox-Fordyce disease (apocrine miliaria) as well as gold-induced skin changes to Polish readership. Thus, this study documents dermatopahtological achievements of Stanislaw Ostrowski - the unifying statesman of society of Lvov and Polish emigration in London.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180308
[Lr] Last revision date:180308
[St] Status:In-Data-Review

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[PMID]: 28749746
[Au] Autor:Kravvas G; Veitch D; Al-Niaimi F
[Ad] Address:a Department of Dermatology , University College London Hospital , London , UK.
[Ti] Title:The increasing relevance of biofilms in common dermatological conditions.
[So] Source:J Dermatolog Treat;29(2):202-207, 2018 Mar.
[Is] ISSN:1471-1753
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Biofilms are diverse groups of microorganisms encased in a self-produced matrix that offers protection against unfavorable conditions and antibiotics. METHODS: We performed a literature search using the MEDLINE electronic database. Only original articles published in English were considered for review. RESULTS: Biofilms have been implicated in the pathogenesis of acne, eczema, hidradenitis suppurativa, onychomycosis, miliaria, and impetigo. Adverse dermal-filler reactions are also linked to biofilms. Strict aseptic technique and prophylactic antibiotics are recommended in order to avoid such complications. Finally, biofilms are implicated in wounds, mainly chronic and diabetic, where they impede healing and cause recurrent infections. Several novel anti-biofilm agents and wound debridement have been shown to be beneficial. CONCLUSIONS: Biofilms are a significant cause of disease with wide implications in the field of dermatology. Several novel treatments have been found to be effective against biofilms, depending on the underlying microbes and type of disease.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1707
[Cu] Class update date: 180219
[Lr] Last revision date:180219
[St] Status:In-Process
[do] DOI:10.1080/09546634.2017.1360989

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[PMID]: 29314202
[Au] Autor:Nagai H; Nishigori C
[Ad] Address:Division of Dermatology, Department of Internal Related, Graduate School of Medicine, Kobe University, Kobe, Japan.
[Ti] Title:Neutrophilic superficial eccrine ductitis: Proposal of a new disease concept.
[So] Source:Pediatr Dermatol;, 2018 Jan 05.
[Is] ISSN:1525-1470
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:A 3-year-old Japanese boy presented with a 3-month history of itchy erythematous papules on his trunk and forearms, the histologic findings of which were characterized by predominantly neutrophilic inflammation within and around the eccrine sweat ducts and obliteration and disruption of the superficial eccrine sweat ducts. Although the skin disorder had some clinical and histopathologic similarity to miliaria and neutrophilic eccrine hidradenitis, it was ultimately conformed to be neither disorder. Based on this and a case report in the Japanese literature of a 1-year-old boy with erythematous eruptions whose clinical and histopathologic findings were similar to those in our case, we propose the skin disorder in our case, referred to as "neutrophilic superficial eccrine ductitis," as a unique entity.
[Pt] Publication type:CASE REPORTS
[Em] Entry month:1801
[Cu] Class update date: 180109
[Lr] Last revision date:180109
[St] Status:Publisher
[do] DOI:10.1111/pde.13386

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[PMID]: 29110043
[Au] Autor:Hanner S; Schneiderbauer R; Enk A; Toberer F
[Ad] Address:Universitäts-Hautklinik Heidelberg, Ruprecht-Karls Universität Heidelberg, Im Neuenheimer Feld 440, 69120, Heidelberg, Deutschland. Susanne.Hanner@med.uni-heidelberg.de.
[Ti] Title:Axilläre und perimamilläre Fox-Fordyce-Erkrankung (apokrine Miliaria) bei einer 19-jährigen Patientin. [Axillary and perimamillary Fox-Fordyce disease (apocrine miliaria) in a 19-year-old woman].
[So] Source:Hautarzt;, 2017 Nov 06.
[Is] ISSN:1432-1173
[Cp] Country of publication:Germany
[La] Language:ger
[Ab] Abstract:Fox-Fordyce disease (FFD), also known as apocrine miliaria, is a rare and chronic skin disease characterized by itching and skin-colored, light brown or yellowish papules. FFD typically affects postpubertal young women between 13 and 35 years. The etiology is not completely known, but a hormonal component is in discussion. Furthermore, exacerbating factors like laser hair removal and hyperhidrosis have been described. Treatment of FFD is quite challenging, as the reported modalities mostly show limited success.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1711
[Cu] Class update date: 171107
[Lr] Last revision date:171107
[St] Status:Publisher
[do] DOI:10.1007/s00105-017-4076-3

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[PMID]: 28513486
[Au] Autor:Seghers AC; Tey HL; Tee SI; Cao T; Chong WS
[Ad] Address:National Skin Centre, Singapore.
[Ti] Title:Pegylated liposomal doxorubicin-induced miliaria crystallina and lichenoid follicular eruption.
[So] Source:Indian J Dermatol Venereol Leprol;, 2017 May 15.
[Is] ISSN:0973-3922
[Cp] Country of publication:India
[La] Language:eng
[Pt] Publication type:LETTER
[Em] Entry month:1705
[Cu] Class update date: 170517
[Lr] Last revision date:170517
[St] Status:Publisher
[do] DOI:10.4103/0378-6323.206233

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[PMID]: 28130590
[Au] Autor:Hanukoglu I; Boggula VR; Vaknine H; Sharma S; Kleyman T; Hanukoglu A
[Ad] Address:Laboratory of Cell Biology, Ariel University, Ariel, 40700, Israel. mbiochem@gmail.com.
[Ti] Title:Expression of epithelial sodium channel (ENaC) and CFTR in the human epidermis and epidermal appendages.
[So] Source:Histochem Cell Biol;147(6):733-748, 2017 Jun.
[Is] ISSN:1432-119X
[Cp] Country of publication:Germany
[La] Language:eng
[Ab] Abstract:A major function of the skin is the regulation of body temperature by sweat secretions. Sweat glands secrete water and salt, especially NaCl. Excreted water evaporates, cooling the skin surface, and Na ions are reabsorbed by the epithelial sodium channels (ENaC). Mutations in ENaC subunit genes lead to a severe multi-system (systemic) form of pseudohypoaldosteronism (PHA) type I, characterized by salt loss from aldosterone target organs, including sweat glands in the skin. In this study, we mapped the sites of localization of ENaC in the human skin by confocal microscopy using polyclonal antibodies generated against human αENaC. Our results reveal that ENaC is expressed strongly in all epidermal layers except stratum corneum, and also in the sebaceous glands, eccrine glands, arrector pili smooth muscle cells, and intra-dermal adipocytes. In smooth muscle cells and adipocytes, ENaC is co-localized with F-actin. No expression of ENaC was detected in the dermis. CFTR is strongly expressed in sebaceous glands. In epidermal appendages noted, except the eccrine sweat glands, ENaC is mainly located in the cytoplasm. In the eccrine glands and ducts, ENaC and CFTR are located on the apical side of the membrane. This localization of ENaC is compatible with ENaC's role in salt reabsorption. PHA patients may develop folliculitis, miliaria rubra, and atopic dermatitis-like skin lesions, due to sweat gland duct occlusion and inflammation of eccrine glands as a result of salt accumulation.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1701
[Cu] Class update date: 171005
[Lr] Last revision date:171005
[St] Status:In-Process
[do] DOI:10.1007/s00418-016-1535-3

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[PMID]: 27592801
[Au] Autor:Cui CY; Ishii R; Campbell DP; Michel M; Piao Y; Kume T; Schlessinger D
[Ad] Address:Laboratory of Genetics and Genomics, National Institute on Aging, National Institutes of Health, Baltimore, Maryland, USA. Electronic address: cuic@mail.nih.gov.
[Ti] Title:Foxc1 Ablated Mice Are Anhidrotic and Recapitulate Features of Human Miliaria Sweat Retention Disorder.
[So] Source:J Invest Dermatol;137(1):38-45, 2017 Jan.
[Is] ISSN:1523-1747
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Sweat glands are critical for thermoregulation. The single tubular structure of sweat glands has a lower secretory portion and an upper reabsorptive duct leading to the secretory pore in the skin. Genes that determine sweat gland structure and function are largely unidentified. Here we report that a Fox family transcription factor, Foxc1, is obligate for appreciable sweat duct activity in mice. When Foxc1 was specifically ablated in skin, sweat glands appeared mature, but the mice were severely hypohidrotic. Morphologic analysis revealed that sweat ducts were blocked by hyperkeratotic or parakeratotic plugs. Consequently, lumens in ducts and secretory portions were dilated, and blisters and papules formed on the skin surface in the knockout mice. The phenotype was strikingly similar to the human sweat retention disorder miliaria. We further show that Foxc1 deficiency ectopically induces the expression of keratinocyte terminal differentiation markers in the duct luminal cells, which most likely contribute to keratotic plug formation. Among those differentiation markers, we show that Sprr2a transcription is directly repressed by overexpressed Foxc1 in keratinocytes. In summary, Foxc1 regulates sweat duct luminal cell differentiation, and mutant mice mimic miliaria and provide a possible animal model for its study.
[Mh] MeSH terms primary: Forkhead Transcription Factors/genetics
Fox-Fordyce Disease/genetics
Hypohidrosis/physiopathology
Sweat Glands/embryology
[Mh] MeSH terms secundary: Animals
Biopsy, Needle
Cell Differentiation/genetics
Cells, Cultured
Female
Fluorescent Antibody Technique
Forkhead Transcription Factors/deficiency
Fox-Fordyce Disease/pathology
Gene Expression Regulation, Developmental
Hepatocyte Nuclear Factor 3-alpha/genetics
Humans
Hypohidrosis/genetics
Immunohistochemistry
Keratinocytes/cytology
Keratinocytes/physiology
Mice
Mice, Inbred C57BL
Mice, Knockout
Models, Animal
RNA, Messenger/genetics
Real-Time Polymerase Chain Reaction/methods
Reference Values
Sweat Glands/pathology
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Forkhead Transcription Factors); 0 (Foxa1 protein, mouse); 0 (Foxc1 protein, mouse); 0 (Hepatocyte Nuclear Factor 3-alpha); 0 (RNA, Messenger)
[Em] Entry month:1707
[Cu] Class update date: 170719
[Lr] Last revision date:170719
[Js] Journal subset:IM
[Da] Date of entry for processing:160906
[St] Status:MEDLINE

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[PMID]: 28292125
[Au] Autor:Elloudi S; Mernissi FZ
[Ad] Address:Hôpital Universitaire Hassan II, Service de Dermatologie et Vénérologie, Fès, Maroc.
[Ti] Title:Miliaire cristalline généralisée. [Widespread miliaria crystalline].
[So] Source:Pan Afr Med J;25:163, 2016.
[Is] ISSN:1937-8688
[Cp] Country of publication:Uganda
[La] Language:fre
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1703
[Cu] Class update date: 170315
[Lr] Last revision date:170315
[St] Status:In-Data-Review
[do] DOI:10.11604/pamj.2016.25.163.9745

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[PMID]: 27328664
[Au] Autor:Arjona-Aguilera C; Rodríguez CC; Jiménez-Gallo D
[Ad] Address:Unidad de Gestión Clínica Dermatología y Venereología, Hospital Universitario Puerta del Mar, Cádiz, Spain.
[Ti] Title:sQuiz your knowledge: Generalized lesions resembling water droplets after treatment with idarubicin and all-trans retinoic acid.
[So] Source:Eur J Dermatol;26(3):329-31, 2016 Jun 01.
[Is] ISSN:1952-4013
[Cp] Country of publication:France
[La] Language:eng
[Mh] MeSH terms primary: Antineoplastic Combined Chemotherapy Protocols/adverse effects
Drug Eruptions/etiology
Miliaria/chemically induced
[Mh] MeSH terms secundary: Aged
Female
Humans
Idarubicin/administration & dosage
Idarubicin/adverse effects
Leukemia, Promyelocytic, Acute/drug therapy
Tretinoin/administration & dosage
Tretinoin/adverse effects
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:5688UTC01R (Tretinoin); ZRP63D75JW (Idarubicin)
[Em] Entry month:1706
[Cu] Class update date: 170623
[Lr] Last revision date:170623
[Js] Journal subset:IM
[Da] Date of entry for processing:160623
[St] Status:MEDLINE
[do] DOI:10.1684/ejd.2016.2816

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[PMID]: 27265071
[Au] Autor:Yang CS; Teeple M; Muglia J; Robinson-Bostom L
[Ad] Address:Department of Dermatology, Warren Alpert Medical School of Brown University, Providence, Rhode Island; Department of Dermatology, Rhode Island Hospital, Providence, Rhode Island. Electronic address: catherine_yang@brown.edu.
[Ti] Title:Inflammatory and glandular skin disease in pregnancy.
[So] Source:Clin Dermatol;34(3):335-43, 2016 May-Jun.
[Is] ISSN:1879-1131
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:A switch from cell-mediated to humoral immunity (helper T 1 [Th1] to helper T 2 [Th2] shift) during gestation plays a key role in placental immune tolerance. As a result, skin diseases that are Th2 mediated often worsen, whereas skin diseases that are Th1 mediated often improve during gestation. Also, due to fluctuations in glandular activity, skin diseases involving sebaceous and eccrine glands may flare, whereas those involving apocrine glands may improve during pregnancy. Despite these trends, inflammatory and glandular skin diseases do not always follow the predicted pattern, and courses are often diverse. We review the gestational course of inflammatory skin diseases, such as atopic dermatitis (atopic eruption of pregnancy), psoriasis, impetigo herpetiformis, urticaria, erythema annulare centrifugum, pityriasis rosea, sarcoidosis, Sweet syndrome, and erythema nodosum, as well as glandular skin diseases, including acne vulgaris, acne rosacea, perioral dermatitis, hidradenitis suppurativa, Fox-Fordyce disease, hyperhidrosis, and miliaria. For each of these diseases, we discuss the pathogenesis, clinical presentation, and management with special consideration for maternal and fetal safety.
[Mh] MeSH terms primary: Pregnancy Complications/diagnosis
Pregnancy Complications/therapy
Skin Diseases/diagnosis
Skin Diseases/therapy
[Mh] MeSH terms secundary: Acne Vulgaris/drug therapy
Acne Vulgaris/etiology
Dermatitis, Atopic/diagnosis
Dermatitis, Atopic/drug therapy
Dermatitis, Perioral/etiology
Erythema Nodosum/diagnosis
Female
Fox-Fordyce Disease/therapy
Hidradenitis Suppurativa/therapy
Humans
Pityriasis Rosea/diagnosis
Pityriasis Rosea/etiology
Pregnancy
Pregnancy Complications/etiology
Psoriasis/complications
Psoriasis/therapy
Remission, Spontaneous
Rosacea/therapy
Sarcoidosis/complications
Sarcoidosis/drug therapy
Skin Diseases/etiology
Sweet Syndrome/diagnosis
Symptom Flare Up
Urticaria/drug therapy
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1705
[Cu] Class update date: 170503
[Lr] Last revision date:170503
[Js] Journal subset:IM
[Da] Date of entry for processing:160607
[St] Status:MEDLINE


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