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Search on : Mink and Viral and Enteritis [Words]
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[PMID]: 28110790
[Au] Autor:Zhang X; Wang J; Mao Y; Xi J; Yu Y; Liu W
[Ad] Address:State Key Laboratory of Agrobiotechnology, Department of Biochemistry and Molecular Biology, College of Biological Sciences, China Agricultural University, Beijing 100193, PR China.
[Ti] Title:Induction and suppression of type I interferon responses by mink enteritis virus in CRFK cells.
[So] Source:Vet Microbiol;199:8-14, 2017 Feb.
[Is] ISSN:1873-2542
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Mink enteritis virus (MEV) is one of the most important viral pathogens causing serious disease in mink. Type I interferon (IFN) plays a critical role in antiviral innate immunity and, for successful infection, many viruses have evolved evasive strategies against it. Here, we show that MEV infection does not evoke IFN or interferon-stimulated genes (ISGs) responses in feline kidney (CRFK) cells, and that MEV suppresses IFN production in both poly I:C-stimulated and untreated cells. In CRFK cells pre-exposure to IFN, show that infection with, and replication of, MEV remain unaffected. This inhibition appears to be mediated by the MEV nonstructural protein (NS1) with its ORI-binding domain playing a major role.
[Mh] MeSH terms primary: Feline Panleukopenia/immunology
Interferon Type I/immunology
Mink enteritis virus/physiology
[Mh] MeSH terms secundary: Animals
Cats
Cell Line
Gene Expression Regulation/drug effects
Interferon Inducers/pharmacology
Poly I-C/pharmacology
Viral Nonstructural Proteins/metabolism
Virus Replication
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Interferon Inducers); 0 (Interferon Type I); 0 (Viral Nonstructural Proteins); O84C90HH2L (Poly I-C)
[Em] Entry month:1702
[Cu] Class update date: 171116
[Lr] Last revision date:171116
[Js] Journal subset:IM
[Da] Date of entry for processing:170124
[St] Status:MEDLINE

  2 / 92 MEDLINE  
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[PMID]: 27354304
[Au] Autor:Yi L; Cheng Y; Zhang M; Cao Z; Tong M; Cheng S; Yan X
[Ad] Address:Institute of Special Wild Economic Animal and Plant Science, Chinese Academy of Agricultural Sciences, 4899 Juye Street, Changchun, Jilin Province, PR China. Electronic address: yili@caas.cn.
[Ti] Title:Identification of a novel Aleutian mink disease virus B-cell epitope using a monoclonal antibody against VP2 protein.
[So] Source:Virus Res;223:39-42, 2016 Sep 02.
[Is] ISSN:1872-7492
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Aleutian mink disease virus (AMDV) is a parvovirus that causes an immune complex-mediated disease in minks. Capsid protein VP2 is a major structural viral protein and can be used to diagnose AMDV. In this study, a specific monoclonal antibody, 1M13, was produced against the AMDV VP2 protein (amino acids 291-502). A linear VP2-protein epitope was identified by subjecting a series of partially overlapping synthesized peptides to be enzyme-linked immunosorbent assay (ELISA) analysis. The results indicated that (386)HLQQNFSTRYIYD(398) was the minimal linear epitope that could be recognized by mAb 1M13. ELISA assays revealed that mink anti-AMDV sera could also recognize the minimal linear epitope. Sequence alignments demonstrated that the linear epitope is highly conserved among AMDV strains except (386)H and is less conserved among Raccoon dog amdovirus, Gray fox amdovirus, Red fox amdovirus, Bat parvovirus and Mink enteritis parvovirus. Taken together, the generation of this VP2-specific mAb with a defined linear peptide epitope may have potential applications in the development of suitable diagnostic techniques for AMDV.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1606
[Cu] Class update date: 160829
[Lr] Last revision date:160829
[Js] Journal subset:IM
[St] Status:In-Process

  3 / 92 MEDLINE  
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[PMID]: 27212684
[Au] Autor:Mao Y; Su J; Wang J; Zhang X; Hou Q; Bian D; Liu W
[Ad] Address:State Key Laboratory of Agrobiotechnology, Department of Biochemistry and Molecular Biology, College of Biological Sciences, China Agricultural University, Beijing 100193, PR China.
[Ti] Title:Roles of three amino acids of capsid proteins in mink enteritis parvovirus replication.
[So] Source:Virus Res;222:24-8, 2016 Aug 15.
[Is] ISSN:1872-7492
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Virulent mink enteritis parvovirus (MEV) strain MEV-LHV replicated to higher titers in feline F81 cells than attenuated strain MEV-L. Phylogenetic and sequence analyses of the VP2 gene of MEV-LHV, MEV-L and other strains in GenBank revealed two evolutionary branches separating virulent and attenuated strains. Three residues, 101, 232 and 411, differed between virulent and attenuated strains but were conserved within the two branches. Site-directed mutagenesis of the VP2 gene of infectious plasmids of attenuated strain MEV-L respectively replacing residues 101 Ile and 411 Ala with Thr and Glu of virulent strains (MEV-L I101T and MEV-L A411E) increased replication efficiency but still to lower levels than MEV-LHV. However, viruses with mutation of residue 232 (MEV-L I232V and MEV-L I101T/I232V/A411E) decreased viral transcription and replication levels. The three VP2 residues 101, 232 and 411, located on or near the capsid surface, played different roles in the infection processes of MEV.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1605
[Cu] Class update date: 160801
[Lr] Last revision date:160801
[Js] Journal subset:IM
[St] Status:In-Process

  4 / 92 MEDLINE  
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[PMID]: 26993137
[Au] Autor:Mao Y; Wang J; Hou Q; Xi J; Zhang X; Bian D; Yu Y; Wang X; Liu W
[Ad] Address:State Key Laboratory of Agrobiotechnology, Department of Biochemistry and Molecular Biology, College of Biological Sciences, China Agricultural University, No. 2 Yuanmingyuan West Road, Haidian District, Beijing, 100193, China.
[Ti] Title:Comparison of biological and genomic characteristics between a newly isolated mink enteritis parvovirus MEV-LHV and an attenuated strain MEV-L.
[So] Source:Virus Genes;52(3):388-96, 2016 Jun.
[Is] ISSN:1572-994X
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:A virus isolated from mink showing clinical signs of enteritis was identified as a high virulent mink enteritis parvovirus (MEV) based on its biological characteristics in vivo and in vitro. Mink, challenged with this strain named MEV-LHV, exhibited severe pathological lesions as compared to those challenged with attenuated strain MEV-L. MEV-LHV also showed higher infection and replication efficiencies in vitro than MEV-L. Sequence of the complete genome of MEV-LHV was determined and analyzed in comparison with those in GenBank, which revealed that MEV-LHV shared high homology with virulent strain MEV SD12/01, whereas MEV-L was closely related to Abashiri and vaccine strain MEVB, and belonged to a different branch of the phylogenetic tree. The genomes of the two strains differed by insertions and deletions in their palindromic termini and specific unique mutations (especially VP2 300) in coding sequences which may be involved in viral replication and pathogenicity. The results of this study provide a better understanding of the biological and genomic characteristics of MEV and identify certain regions and sites that may be involved in viral replication and pathogenicity.
[Mh] MeSH terms primary: Genome, Viral
Mink enteritis virus/physiology
Mink/virology
Parvoviridae Infections/virology
[Mh] MeSH terms secundary: Amino Acid Sequence
Animals
Base Sequence
Cells, Cultured
China
Feces/virology
Mink enteritis virus/genetics
Mink enteritis virus/isolation & purification
Mutagenesis, Insertional
Mutation
Phylogeny
Sequence Deletion
Sequence Homology, Nucleic Acid
Virus Replication
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1704
[Cu] Class update date: 171102
[Lr] Last revision date:171102
[Js] Journal subset:IM
[Da] Date of entry for processing:160320
[St] Status:MEDLINE
[do] DOI:10.1007/s11262-016-1314-1

  5 / 92 MEDLINE  
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[PMID]: 26077544
[Au] Autor:Wilson DJ; Baldwin TJ; Whitehouse CH; Hullinger G
[Ad] Address:School of Veterinary Medicine, Department of Animal, Dairy and Veterinary Sciences, Utah State University, Logan, UT (Wilson, Baldwin, Whitehouse)Utah Veterinary Diagnostic Laboratory, Logan, UT (Hullinger) david.wilson@usu.edu.
[Ti] Title:Causes of mortality in farmed mink in the Intermountain West, North America.
[So] Source:J Vet Diagn Invest;27(4):470-5, 2015 Jul.
[Is] ISSN:1943-4936
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:The primary causes of mortality were identified in postmortem examination of 339 (90.9%) of 373 farmed mink (Neovison vison; syn. Mustela vison) from January 2009 through June 2014 at the Utah Veterinary Diagnostic Laboratory (Logan, Utah). Mink were raised under farm conditions in the Intermountain West in North America, except for 1 submission of mink from Wisconsin. In the 339 mink where cause(s) of death were established, 311 (91.7%) died from a single disease or condition, whereas 28 (8.3%) had 2 diseases or conditions contributing to death. Where cause(s) of death were evident, 11 diseases accounted for 321 (94.7%) of the diagnoses: bacterial pneumonia (67, 18.8%), Aleutian mink disease (61, 17.7%), mink viral enteritis (56, 16.2%), hepatic lipidosis (28, 8.1%), nutritional myopathy (24, 7%), bacterial enterocolitis (17, 4.9%), bacterial septicemia (16, 4.6%), starvation (15, 4.3%), epizootic catarrhal gastroenteritis of mink (14, 4.1%), pancreatitis (13, 3.8%), and bacterial metritis (10, 2.9%). In 34 (9.1%) animals, a cause of death was not evident. In an additional 16 (4.3%) of the mink, botulism was suspected from clinical history but could not be confirmed by laboratory testing. Control measures for the most common causes of death in farmed mink include testing and removal of positive animals (Aleutian mink disease), vaccination (Pseudomonas aeruginosa pneumonia, mink viral enteritis), avoidance of obesity in mink (hepatic lipidosis), and environmental management, including maintaining clean water cups, floors, feed troughs, cages, feed silos, feed truck tires, workers' shoes, dining areas for farm personnel, leather mink handling gloves, street clothes, and coveralls.
[Mh] MeSH terms primary: Aleutian Mink Disease/mortality
Mink
[Mh] MeSH terms secundary: Animals
Animals, Domestic
Idaho
Pneumonia, Bacterial/mortality
Pneumonia, Bacterial/veterinary
Pseudomonas Infections/mortality
Pseudomonas Infections/veterinary
Pseudomonas aeruginosa
Utah
Wisconsin
[Pt] Publication type:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Entry month:1602
[Cu] Class update date: 150803
[Lr] Last revision date:150803
[Js] Journal subset:IM
[Da] Date of entry for processing:150617
[St] Status:MEDLINE
[do] DOI:10.1177/1040638715586438

  6 / 92 MEDLINE  
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[PMID]: 25582057
[Au] Autor:Wang J; Cheng Y; Zhang M; Zhao H; Lin P; Yi L; Tong M; Cheng S
[Ad] Address:State Key Laboratory for Molecular Biology of Special Economic Animals, Institute of Special Animal and Plant Sciences, Chinese Academy of Agricultural Sciences, Changchun, 130112, China. tcswjk@126.com.
[Ti] Title:Development of a nanoparticle-assisted PCR (nanoPCR) assay for detection of mink enteritis virus (MEV) and genetic characterization of the NS1 gene in four Chinese MEV strains.
[So] Source:BMC Vet Res;11:1, 2015 Jan 13.
[Is] ISSN:1746-6148
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Mink enteritis virus (MEV) causes mink viral enteritis, an acute and highly contagious disease whose symptoms include violent diarrhea, and which is characterized by high morbidity and mortality. Nanoparticle-assisted polymerase chain reaction (nanoPCR) is a recently developed technique for the rapid detection of bacterial and viral DNA. Here we describe a novel nanoPCR assay for the clinical detection and epidemiological characterization of MEV. RESULTS: This assay is based upon primers specific for the conserved region of the MEV NS1 gene, which encodes nonstructural protein 1. Under optimized conditions, the MEV nanoPCR assay had a detection limit of 8.75 × 10(1) copies recombinant plasmids per reaction, compared with 8.75 × 10(3) copies for conventional PCR analysis. Moreover, of 246 clinical mink samples collected from five provinces in North-Eastern China, 50.8% were scored MEV positive by our nanoPCR assay, compared with 32.5% for conventional PCR. Furthermore no cross reactivity was observed for the nanoPCR assay with respect to related viruses, including canine distemper virus (CDV) and Aleutian mink disease parvovirus (AMDV). Phylogenetic analysis of four Chinese wild type MEV isolates using the nanoPCR assay indicated that they belonged to a small MEV clade, named "China type", in the MEV/FPLV cluster, and were closely clustered in the same location. CONCLUSIONS: Our results indicate that the MEV China type clade is currently circulating in domestic minks in China. We anticipate that the nanoPCR assay we have described here will be useful for the detection and epidemiological and pathological characterization of MEV.
[Mh] MeSH terms primary: Mink Viral Enteritis/virology
Mink enteritis virus/genetics
Mink enteritis virus/isolation & purification
Nanoparticles
Polymerase Chain Reaction/methods
Viral Nonstructural Proteins/metabolism
[Mh] MeSH terms secundary: Animals
China
Gene Expression Regulation, Viral
Genetic Variation
Mink
Mink Viral Enteritis/epidemiology
Sensitivity and Specificity
Viral Nonstructural Proteins/genetics
[Pt] Publication type:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Name of substance:0 (NS1 protein, Flavivirus); 0 (Viral Nonstructural Proteins)
[Em] Entry month:1601
[Cu] Class update date: 170220
[Lr] Last revision date:170220
[Js] Journal subset:IM
[Da] Date of entry for processing:150114
[St] Status:MEDLINE
[do] DOI:10.1186/s12917-014-0312-6

  7 / 92 MEDLINE  
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[PMID]: 25465595
[Au] Autor:Sun JZ; Wang J; Wang S; Yuan D; Li Z; Yi B; Hou Q; Mao Y; Liu W
[Ad] Address:State Key Laboratory of Agrobiotechnology, Department of Biochemistry and Molecular Biology, College of Biological Sciences, China Agricultural University, No. 2 Yuanmingyuan West Road, Haidian District, Beijing, 100193, China. sunjiazeng331@163.com.
[Ti] Title:MicroRNA miR-320a and miR-140 inhibit mink enteritis virus infection by repression of its receptor, feline transferrin receptor.
[So] Source:Virol J;11:210, 2014 Dec 03.
[Is] ISSN:1743-422X
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Mink enteritis virus (MEV) is one of the most important pathogens in the mink industry. Recent studies have shed light into the role of microRNAs (miRNAs), small noncoding RNAs of length ranging from 18-23 nucleotides (nt), as critical modulators in the host-pathogen interaction networks. We previously showed that miRNA miR-181b can inhibit MEV replication by repression of viral non-structural protein 1 expression. Here, we report that two other miRNAs (miR-320a and miR-140) inhibit MEV entry into feline kidney (F81) cells by downregulating its receptor, transferrin receptor (TfR), by targeting the 3' untranslated region (UTR) of TfR mRNA, while being themselves upregulated.
[Mh] MeSH terms primary: Antiviral Agents/metabolism
Gene Expression Regulation
MicroRNAs/metabolism
Mink enteritis virus/physiology
Receptors, Transferrin/antagonists & inhibitors
Receptors, Virus/antagonists & inhibitors
Virus Internalization
[Mh] MeSH terms secundary: Animals
Cats
Cell Line
Down-Regulation
MicroRNAs/genetics
Receptors, Transferrin/genetics
Receptors, Virus/genetics
[Pt] Publication type:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Name of substance:0 (Antiviral Agents); 0 (MicroRNAs); 0 (Receptors, Transferrin); 0 (Receptors, Virus)
[Em] Entry month:1510
[Cu] Class update date: 151028
[Lr] Last revision date:151028
[Js] Journal subset:IM
[Da] Date of entry for processing:141204
[St] Status:MEDLINE
[do] DOI:10.1186/s12985-014-0210-3

  8 / 92 MEDLINE  
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[PMID]: 25148585
[Au] Autor:Lian H; Liu Y; Li N; Wang Y; Zhang S; Hu R
[Ti] Title:Novel circovirus from mink, China.
[So] Source:Emerg Infect Dis;20(9):1548-50, 2014 Sep.
[Is] ISSN:1080-6059
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:A long-established epidemic of enteritis, caused by an unidentified pathogen distinct from parvovirus, has now been recognized in mink. In 2013, we identified a novel circovirus by degenerate PCR and fully sequenced its genome. This virus differs substantially from currently known members of the genus Circovirus and represents a new species.
[Mh] MeSH terms primary: Circovirus/classification
Circovirus/genetics
Mink/virology
[Mh] MeSH terms secundary: Animals
China
Circoviridae Infections/veterinary
Circovirus/isolation & purification
Molecular Sequence Data
Phylogeny
Sequence Analysis, DNA
Viral Proteins/genetics
[Pt] Publication type:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Name of substance:0 (Viral Proteins)
[Em] Entry month:1507
[Cu] Class update date: 150804
[Lr] Last revision date:150804
[Js] Journal subset:IM
[Da] Date of entry for processing:140823
[St] Status:MEDLINE
[do] DOI:10.3201/eid2009.140015

  9 / 92 MEDLINE  
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[PMID]: 24525403
[Au] Autor:Sun JZ; Wang J; Wang S; Yuan D; Birame BM; Li Z; Yi B; Liu W
[Ad] Address:State Key Laboratory of Agrobiotechnology, Department of Biochemistry and Molecular Biology, College of Biological Sciences, China Agricultural University, Beijing 100193, China.
[Ti] Title:MicroRNA profile analysis of a feline kidney cell line before and after infection with mink enteritis virus.
[So] Source:Gene;539(2):224-9, 2014 Apr 15.
[Is] ISSN:1879-0038
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:MicroRNAs (miRNAs) are small regulatory RNAs that play a significant role in eukaryotes by targeting mRNAs for cleavage or translational repression. Recent studies have also shown them to be associated with cellular changes following viral infection. Mink enteritis virus (MEV) is one of the most important viral pathogens in the mink industry. To study the involvement of miRNAs in the MEV infection process, we used Illumina's ultrahigh throughput approach to sequencing miRNA libraries from the feline kidney (F81) cell line before and after infection with MEV. Using this bioinformatics approach we identified 196 known mammalian miRNA orthologs belonging to 152 miRNA families in F81 cells. Additionally, 97 miRNA*s of these miRNAs were detected. As well as known miRNAs, 384 and 398 novel miRNA precursor candidates were identified in uninfected and MEV-infected F81 cells respectively that have not been reported in other mammals. In MEV-infected cells 3 miRNAs were significantly down-regulated and 4 up-regulated including 3 significantly. The majority (12 of 16) of randomly selected miRNA expression profiles by qRT-PCR were consistent with those identified by deep sequencing. A total of 88 miRNAs were predicted to target interferon-associated genes; 6 appear to target the 3'UTR of MEV-specific receptor transferring receptor mRNAs; and 8 to target the MEV mRNA coding region. No miRNAs coded by MEV itself were detected.
[Mh] MeSH terms primary: Biomarkers/metabolism
Feline Panleukopenia/genetics
Gene Expression Profiling
Kidney/metabolism
MicroRNAs/genetics
Mink Viral Enteritis/genetics
Mink enteritis virus/pathogenicity
[Mh] MeSH terms secundary: Animals
Cats
Cells, Cultured
Computational Biology
Feline Panleukopenia/virology
Kidney/virology
Mink Viral Enteritis/virology
Oligonucleotide Array Sequence Analysis
RNA, Messenger/genetics
Real-Time Polymerase Chain Reaction
Reverse Transcriptase Polymerase Chain Reaction
[Pt] Publication type:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Name of substance:0 (Biomarkers); 0 (MicroRNAs); 0 (RNA, Messenger)
[Em] Entry month:1405
[Cu] Class update date: 151119
[Lr] Last revision date:151119
[Js] Journal subset:IM
[Da] Date of entry for processing:140215
[St] Status:MEDLINE

  10 / 92 MEDLINE  
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[PMID]: 24463297
[Au] Autor:Yuan D; Wang J; Li Z; Mao Y; Sun JZ; Xi J; Wang S; Hou Q; Yi B; Liu W
[Ad] Address:State Key Laboratory of Agrobiotechnology, Department of Biochemistry and Molecular Biology, College of Biological Sciences, China Agricultural University, Beijing 100193, PR China.
[Ti] Title:Establishment of a rescue system for an autonomous Parvovirus mink enteritis virus.
[So] Source:Virus Res;183:1-5, 2014 Apr.
[Is] ISSN:1872-7492
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Construction and characterization of a full-length infectious clone (pMEV) of mink enteritis virus are described. Feline kidney cells (F81) were transfected with pMEV containing an engineered BamHI site that served as a genetic marker. The rescued virus was indistinguishable from its parental virus. The availability of a MEV infectious clone will facilitate studies of viral replication and pathogenicity and will permit the elucidation of determinants of the host range of the parvovirus.
[Mh] MeSH terms primary: Mink enteritis virus/growth & development
Reverse Genetics/methods
Virology/methods
[Mh] MeSH terms secundary: Animals
Cats
Cell Line
Mink enteritis virus/genetics
[Pt] Publication type:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Entry month:1411
[Cu] Class update date: 140324
[Lr] Last revision date:140324
[Js] Journal subset:IM
[Da] Date of entry for processing:140128
[St] Status:MEDLINE


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