Database : MEDLINE Search on : Muscle and Neoplasms [Words] References found : 36709 [refine] Displaying: 1 .. 10   in format [Detailed]

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[PMID]: 23617935 [Au] Autor: Nonaka K; Miyazawa M; Ban S; Aikawa M; Akimoto N; Koyama I; Kita H [Ad] Address: Department of Gastroenterology, Saitama Medical University International Medical Center, 1397-1 Yamane, Hidaka, 350-1298, Japan. hkita@saitama-med.ac.jp. [Ti] Title: Different healing process of esophageal large mucosal defects by endoscopic mucosal dissection between with and without steroid injection in an animal model. [So] Source: BMC Gastroenterol;13:72, 2013. [Is] ISSN: 1471-230X [Cp] Country of publication: England [La] Language: eng [Ab] Abstract: BACKGROUND: Stricture formation is one of the major complications after endoscopic removal of large superficial squamous cell neoplasms of the esophagus, and local steroid injections have been adopted to prevent it. However, fundamental pathological alterations related to them have not been well analyzed so far. The aim of this study was to analyze the time course of the healing process of esophageal large mucosal defects resulting in stricture formation and its modification by local steroid injection, using an animal model. METHODS: Esophageal circumferential mucosal defects were created by endoscopic mucosal dissection (ESD) for four pigs. One pig was sacrificed five minutes after the ESD, and other two pigs were followed-up on endoscopy and sacrificed at the time of one week and three weeks after the ESD, respectively. The remaining one pig was followed-up on endoscopy with five times of local steroid injection and sacrificed at the time of eight weeks after the ESD. The esophageal tissues of all pigs were subjected to pathological analyses. RESULTS: For the pigs without steroid injection, the esophageal stricture was completed around three weeks after the ESD on both endoscopy and esophagography. Histopathological examination of the esophageal tissues revealed that spindle-shaped α-smooth muscle actin (SMA)-positive myofibroblasts arranged in a parallel fashion and extending horizontally were identified at the ulcer bed one week after the ESD, and increased contributing to formation of the stenotic luminal ridge covered with the regenerated epithelium three weeks after the ESD. The proper muscle layer of the stricture site was thinned with some myocytes which seemingly showed transition to the myofibroblast layer. By contrast, for the pig with steroid injection, esophageal stricture formation was not evident with limited appearance of the spindle-shaped myofibroblasts, instead, appearance of stellate or polygocal SMA-positive stromal cells arranged haphazardly in the persistent granulation tissue of the ulcer site. CONCLUSIONS: Proliferation of spindle-shaped myofibroblasts arranged in a parallel fashion is likely to play an important role in stricture formation after circumferential mucosal defects by esophageal ESD, which may be related to the thinning of the proper muscle layer in the healing course of the defects. Local steroid injection seems to be effective to prevent the stricture through the modification of this process. [Pt] Publication type: JOURNAL ARTICLE [Em] Entry month: 1305 [Js] Journal subset: IM [St] Status: In-Data-Review [do] DOI: 10.1186/1471-230X-13-72

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[PMID]: 23106771 [Au] Autor: De La Peña E; Hernández V; Blázquez C; Martin MD; Díaz FJ; Capitán C; Alemany I; Llorente C [Ad] Address: Department of Urology, Hospital Universitario Fundación Alcorcón, Madrid, Spain. [Ti] Title: Weight of the resected specimen after transurethral resection as a new predictive variable for recurrence of non-muscle-invasive bladder tumour. [So] Source: BJU Int;111(4 Pt B):E196-201, 2013 Apr. [Is] ISSN: 1464-410X [Cp] Country of publication: England [La] Language: eng [Ab] Abstract: OBJECTIVE: To evaluate the role of the weight of the resected specimen after transurethral resection as a predictive factor for recurrence and progression of non-muscle-invasive bladder tumour (NMIBT). PATIENTS AND METHODS: The weight of the resected tumour was measured consecutively in 144 subjects who underwent transurethral resection of bladder tumours at our institution. The median (interquartile range [IQR]) follow-up was 58 (61.3) months. The probability of recurrence and progression at 1 and 5 years were calculated using the currently accepted variables. Thresholds for the specimen weight were determined according to percentiles and receiver-operating characteristic curves. RESULTS: The median (IQR) weight of the specimen was 6 (16) g. Multivariate analysis showed that the weight of the resected specimen was an independent predictive risk factor for recurrence at a threshold value of 6 g with a hazard ratio of 1.7 (95% confidence interval: 1.048-2.761) P = 0.03. Progression was not associated with the weight of the resected specimen. CONCLUSIONS: The weight of the resected specimen is a new variable for predicting the risk of recurrence of NMIBT. Tumours weighing >6 g, according to the present data, have a 1.7-fold higher likelihood of recurrence than those tumours that weigh less. [Mh] MeSH terms primary: Cystectomy/methods Cystoscopy/methods Neoplasm Recurrence, Local/diagnosis Urinary Bladder Neoplasms/pathology Urinary Bladder/pathology [Mh] MeSH terms secundary: Aged Female Follow-Up Studies Humans Incidence Male Neoplasm Invasiveness Neoplasm Recurrence, Local/epidemiology Organ Size Predictive Value of Tests Retrospective Studies Spain/epidemiology Urinary Bladder/surgery Urinary Bladder Neoplasms/surgery [Pt] Publication type: COMPARATIVE STUDY; JOURNAL ARTICLE [Em] Entry month: 1305 [Js] Journal subset: IM [Da] Date of entry for processing: 130325 [St] Status: MEDLINE [do] DOI: 10.1111/j.1464-410X.2012.11588.x

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[PMID]: 23522238 [Au] Autor: Manley K; Gelvez S; Meldon CJ; Levai I; Malata CM; Coonar AS [Ad] Address: Department of Thoracic Surgery, Papworth Hospital, and University of Cambridge, School of Clinical Medicine, Cambridge, United Kingdom. kate.manley06@imperial.ac.uk [Ti] Title: Free deep inferior epigastric perforator flap used for management of post-pneumonectomy space empyema. [So] Source: Ann Thorac Surg;95(4):e83-5, 2013 Apr. [Is] ISSN: 1552-6259 [Cp] Country of publication: Netherlands [La] Language: eng [Ab] Abstract: Various solutions exist for management of post-pneumonectomy space empyema. We describe the use of a free deep inferior epigastric perforator (DIEP) flap to fill the space and close a pleural window. Previously, flaps involving abdominal muscle or omentum have been used for this purpose. Abdominal surgery to harvest such flaps can impair ventilatory mechanics. The DIEP flap--harvested from the abdomen, and composed primarily of skin and muscle avoids this problem, thus is a desirable technique in patients with impaired lung function. We believe this is the first report of the DIEP flap to close a postpneumonectomy empyema space. [Mh] MeSH terms primary: Abdominal Muscles/transplantation Empyema, Pleural/surgery Epigastric Arteries Free Tissue Flaps/blood supply Perforator Flap/blood supply Pneumonectomy/adverse effects Thoracoplasty/methods [Mh] MeSH terms secundary: Abdominal Muscles/blood supply Aged Carcinoma, Squamous Cell/surgery Drainage/methods Empyema, Pleural/etiology Humans Lung Neoplasms/surgery Male [Pt] Publication type: CASE REPORTS; JOURNAL ARTICLE [Em] Entry month: 1305 [Js] Journal subset: AIM; IM [Da] Date of entry for processing: 130325 [St] Status: MEDLINE

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[PMID]: 23522212 [Au] Autor: Taguchi S; Mori A; Yamabe K; Suzuki R; Nishizawa K; Hasegawa I; Irie R [Ad] Address: Department of Cardiovascular Surgery, Kawasaki Municipal Hospital, Kanagawa, Japan. staguchi@kmh.gr.jp [Ti] Title: Malignant solitary fibrous tumor of the left ventricular epicardium. [So] Source: Ann Thorac Surg;95(4):1447-50, 2013 Apr. [Is] ISSN: 1552-6259 [Cp] Country of publication: Netherlands [La] Language: eng [Ab] Abstract: Reports describing solitary fibrous tumors of the pericardium are rare. Surgical treatment was performed on a 49-year-old woman with a large pericardial mass. The mass was attached to the left ventricular wall with a broad stalk and was free of the parietal pericardium. It was apparent macroscopically that the tumor had invaded the left ventricular muscle. On histopathology, the tumor was diagnosed as a solitary fibrous tumor with low-grade malignancy. [Mh] MeSH terms primary: Cardiac Surgical Procedures/methods Heart Neoplasms/diagnosis Solitary Fibrous Tumors/diagnosis [Mh] MeSH terms secundary: Diagnosis, Differential Female Heart Neoplasms/surgery Heart Ventricles Humans Magnetic Resonance Imaging, Cine Middle Aged Pericardium Solitary Fibrous Tumors/surgery Tomography, X-Ray Computed [Pt] Publication type: CASE REPORTS; JOURNAL ARTICLE [Em] Entry month: 1305 [Js] Journal subset: AIM; IM [Da] Date of entry for processing: 130325 [St] Status: MEDLINE

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[PMID]: 23522204 [Au] Autor: CandaÈ™ F; Berber U; Yildizhan A; Yiyit N; Görür R; IÈ™itmangil T [Ad] Address: Department of Thoracic Surgery, GATA Haydarpasa Teaching Hospital, Istanbul, Turkey. fhcandas@yahoo.com [Ti] Title: Anterior mediastinal angiomyolipoma. [So] Source: Ann Thorac Surg;95(4):1431-2, 2013 Apr. [Is] ISSN: 1552-6259 [Cp] Country of publication: Netherlands [La] Language: eng [Ab] Abstract: Angiomyolipomas are benign, solitary, noninvasive mesenchymal tumors. They most often arise in the kidney. Extrarenal presentations of these tumors are in skin, orophaynx, abdominal wall, gastrointestinal tract, heart, lung, liver, uterus, penis, and spinal cord. Angiomyolipoma of the mediastinum is extremely rare and is composed of an admixture of fat, smooth muscle cells, and tortuous, thick-walled, small to medium sized blood vessels. We present a surgically confirmed case of anterior mediastinal angiomyolipoma incidentally diagnosed in an asymptomatic patient. [Mh] MeSH terms primary: Angiomyolipoma/diagnosis Mediastinal Neoplasms/diagnosis [Mh] MeSH terms secundary: Angiomyolipoma/surgery Biopsy, Needle Diagnosis, Differential Humans Male Mediastinal Neoplasms/surgery Thoracic Surgery, Video-Assisted Tomography, X-Ray Computed Young Adult [Pt] Publication type: CASE REPORTS; JOURNAL ARTICLE [Em] Entry month: 1305 [Js] Journal subset: AIM; IM [Da] Date of entry for processing: 130325 [St] Status: MEDLINE

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[PMID]: 23522203 [Au] Autor: Kazarin O; Vlodavsky E; Guralnik L; Kremer R; Lachter J; Bar-Sela G [Ad] Address: Department of Oncology, Rambam Health Care Campus and Technion-Israel Institute of Technology, Haifa, Israel. [Ti] Title: Association between esophageal leiomyomatosis and p53 mutation. [So] Source: Ann Thorac Surg;95(4):1429-31, 2013 Apr. [Is] ISSN: 1552-6259 [Cp] Country of publication: Netherlands [La] Language: eng [Ab] Abstract: Li-Fraumeni syndrome is a cancer predisposition syndrome associated with a variety of neoplasms, mainly soft tissue sarcoma, premenopausal breast cancer, brain tumors, adrenocortical carcinoma, and leukemia. Esophageal leiomyomatosis involves the presence of several rare benign neoplastic lesions composed of proliferating smooth muscle cells in the esophageal wall. The current case report presents a patient with recurrent diffuse leiomyomas of the esophagus and confirmed p53 mutation with clinical criteria of Li-Fraumenilike syndrome. [Mh] MeSH terms primary: DNA, Neoplasm/genetics Esophageal Neoplasms/genetics Esophagus/pathology Genes, p53/genetics Leiomyomatosis/genetics Mutation [Mh] MeSH terms secundary: Biopsy DNA Mutational Analysis Endoscopy, Gastrointestinal Esophageal Neoplasms/diagnosis Female Humans Leiomyomatosis/diagnosis Middle Aged Tomography, X-Ray Computed [Pt] Publication type: CASE REPORTS; JOURNAL ARTICLE [Nm] Name of substance: 0 (DNA, Neoplasm) [Em] Entry month: 1305 [Js] Journal subset: AIM; IM [Da] Date of entry for processing: 130325 [St] Status: MEDLINE

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[PMID]: 23399832 [Au] Autor: Coulson-Thomas VJ; Coulson-Thomas YM; Gesteira TF; Andrade de Paula CA; Carneiro CR; Ortiz V; Toma L; Kao WW; Nader HB [Ad] Address: Departamento de Bioquímica, Universidade Federal de São Paulo, São Paulo, SP 04044-020, Brazil. vcoulsonthomas@gmail.com [Ti] Title: Lumican expression, localization and antitumor activity in prostate cancer. [So] Source: Exp Cell Res;319(7):967-81, 2013 Apr 15. [Is] ISSN: 1090-2422 [Cp] Country of publication: United States [La] Language: eng [Ab] Abstract: The stromal reaction surrounding tumors leads to the formation of a tumor-specific microenvironment, which may play either a restrictive role or a supportive role in the growth and progression of the tumors. Lumican, a small leucine-rich proteoglycan (SLRP) of the extracellular matrix (ECM), regulates collagen fibrillogenesis. Recently, lumican has also been shown to regulate cell behavior during embryonic development, tissue repair and tumor progression. The role of lumican in cancer varies according to the type of tumor. In this study we analyze the role of lumican in the pathogenesis of prostate cancer both in vivo and in vitro. Overall lumican up-regulation was observed in the primary tumors analyzed through both real-time PCR and immunostaining. The increase in lumican expression was observed in the reactive stroma surrounding prostate primary tumors with fibrotic deposition surrounding the acinar glands. In vitro analysis demonstrated that lumican inhibited both the migration and invasion of metastatic prostate cancer cells isolated from lymph node, bone and brain. Moreover, prostate cancer cells seeded on lumican presented a decrease in the formation of cellular projections, lamellipodia detected by a decreased rearrangement in ZO-1, keratin 8/18, integrin ß1 and MT1-MMP, and invadopodia detected by disruption of α-smooth muscle actin, cortactin and N-WASP. Moreover, a significant increase in prostate cancer cell invasion was observed through the peritoneum of lumican knockout mice, further demonstrating the restrictive role lumican present in the ECM has on prostate cancer invasion. In conclusion, lumican present in the reactive stroma surrounding prostate primary tumors plays a restrictive role on cancer progression, and we therefore postulate that lumican could be a valuable marker in prostate cancer staging. [Mh] MeSH terms primary: Chondroitin Sulfate Proteoglycans/biosynthesis Keratan Sulfate/biosynthesis Prostatic Neoplasms/metabolism [Mh] MeSH terms secundary: Animals Antigens, CD29/metabolism Cell Line, Tumor Cell Movement Chondroitin Sulfate Proteoglycans/deficiency Humans Keratan Sulfate/deficiency Male Mice Mice, Knockout Prostatic Neoplasms/pathology Up-Regulation [Pt] Publication type: JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL; RESEARCH SUPPORT, NON-U.S. GOV'T [Nm] Name of substance: 0 (Antigens, CD29); 0 (Chondroitin Sulfate Proteoglycans); 0 (lumican); 9056-36-4 (Keratan Sulfate) [Em] Entry month: 1305 [Js] Journal subset: IM [Da] Date of entry for processing: 130318 [St] Status: MEDLINE

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[PMID]: 23204312 [Au] Autor: Primiano G; Plantone D; Sauchelli D; Cuccagna C; Renna R; Iorio R; Servidei S [Ad] Address: Department of Neurology, Catholic University of the Sacred Heart, Rome, Italy. [Ti] Title: Resolution of muscle inflammation after tumor removal in a woman with paraneoplastic dermatomyositis. [So] Source: J Rheumatol;39(12):2359-60, 2012 Dec. [Is] ISSN: 0315-162X [Cp] Country of publication: Canada [La] Language: eng [Mh] MeSH terms primary: Breast Neoplasms/therapy Carcinoma, Papillary/therapy Dermatomyositis/therapy Paraneoplastic Syndromes/therapy [Mh] MeSH terms secundary: Adult Biopsy Breast Neoplasms/complications Breast Neoplasms/diagnosis Carcinoma, Papillary/complications Carcinoma, Papillary/diagnosis Combined Modality Therapy Dermatomyositis/diagnosis Dermatomyositis/etiology Electromyography Female Humans Magnetic Resonance Imaging Mastectomy Muscle, Skeletal/pathology Muscle, Skeletal/physiopathology Paraneoplastic Syndromes/diagnosis Paraneoplastic Syndromes/etiology Radiography, Thoracic Treatment Outcome [Pt] Publication type: CASE REPORTS; JOURNAL ARTICLE [Em] Entry month: 1305 [Js] Journal subset: IM [Da] Date of entry for processing: 121203 [St] Status: MEDLINE [do] DOI: 10.3899/jrheum.120806

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[PMID]: 22665269 [Au] Autor: Zhang J; Zhou Y; Wu Y; Ma L; Fan Y; Kang X; Shi H; Zhang J [Ad] Address: School of Life Science and Technology, Tongji University, Shanghai, 200092, China. [Ti] Title: Isolation and characterization of a novel noncoding RNA from nickel-induced lung cancer. [So] Source: Biol Trace Elem Res;150(1-3):258-63, 2012 Dec. [Is] ISSN: 1559-0720 [Cp] Country of publication: United States [La] Language: eng [Ab] Abstract: Noncoding RNAs have drawn significant attention in carcinogenesis. In this study, we identified a novel gene named nickel-related gene1 (NRG1) associated with nickel-induced cancer. By using rapid amplification of cDNA end PCR, we obtained the full length of the cDNA. The sequence was analyzed by using related bioinformatics software and comparative genomics methods. The results showed that NRG1 was located on chromosome 2q12, within intron2 of ADAMTS6, a disintegrin and metalloproteinase with thrombospondin motifs. And, NRG1 had a high level of homology (76 %) to rat LINE1 sequence RL1.3 (long interspersed middle repetitive DNA). What's more, there was no continuous open reading frame present in NRG1 sequence. Taken together, these data demonstrate that NRG1 is a novel noncoding RNA, and we predicted it may be a transposon-like gene. The identification of NRG1 emphasized the potential role of noncoding RNA in nickel carcinogenesis. [Mh] MeSH terms primary: Carcinogens/toxicity Lung Neoplasms/chemically induced Lung Neoplasms/metabolism Lung/metabolism Nickel/toxicity RNA, Untranslated/drug effects RNA, Untranslated/metabolism Retroelements/drug effects [Mh] MeSH terms secundary: ADAM Proteins/chemistry ADAM Proteins/genetics ADAM Proteins/metabolism Animals Base Sequence Female Introns Long Interspersed Nucleotide Elements Lung/drug effects Male Molecular Sequence Data Muscle Neoplasms/chemically induced Muscle Neoplasms/metabolism Muscles/drug effects Muscles/metabolism Nickel/administration & dosage RNA, Untranslated/chemistry RNA, Untranslated/isolation & purification Rats Rats, Wistar Sequence Homology, Nucleic Acid [Pt] Publication type: JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T [Nm] Name of substance: 0 (Carcinogens); 0 (RNA, Untranslated); 0 (Retroelements); 16812-54-7 (nickel sulfide); 7440-02-0 (Nickel); EC 3.4.24.- (ADAM Proteins) [Em] Entry month: 1305 [Js] Journal subset: IM [Da] Date of entry for processing: 121130 [St] Status: MEDLINE [do] DOI: 10.1007/s12011-012-9460-3

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[PMID]: 22639385 [Au] Autor: Yan H; Chang H [Ad] Address: Department of Central Laboratory, Fifth Hospital of Shijiazhuang, Shijiazhuang, Hebei, 050021, China. [Ti] Title: Antioxidant and antitumor activities of selenium- and zinc-enriched oyster mushroom in mice. [So] Source: Biol Trace Elem Res;150(1-3):236-41, 2012 Dec. [Is] ISSN: 1559-0720 [Cp] Country of publication: United States [La] Language: eng [Ab] Abstract: Selenium and zinc are well-known essential trace elements with potent biological functions. However, the possible health benefits of the combined administration of dietary selenium and zinc have not been studied extensively. In this study, we prepared selenium- and zinc-enriched mushrooms (SZMs) containing increased levels of selenium and zinc. The effects of SZMs on antioxidant and antitumor activities were evaluated. Mice were fed with either a control diet or a diet supplemented with SZMs or sodium selenite and zinc sulfate for 6 weeks. Antioxidant capacity was investigated by measuring the activities of antioxidant enzymes and the levels of lipid peroxide products. Results showed that treatment with SZMs significantly increased the activities of glutathione peroxidase (GPx) and superoxide dismutase and decreased the levels of malondialdehyde and lipofuscin. Furthermore, using a mouse model of lung tumors, we found that SZMs significantly decreased the number of tumor nodes with an increase in the activity of GPx. SZMs had a greater effect on the increase in both antioxidant and antitumor activities than did sodium selenite and zinc sulfate. These findings suggest that SZMs may be effective for improving antioxidant capacity and preventing tumors. [Mh] MeSH terms primary: Anticarcinogenic Agents/metabolism Antioxidants/metabolism Dietary Supplements Lung Neoplasms/prevention & control Pleurotus/chemistry Selenium/metabolism Zinc/metabolism [Mh] MeSH terms secundary: Animals Anticarcinogenic Agents/administration & dosage Anticarcinogenic Agents/adverse effects Antioxidants/administration & dosage Antioxidants/adverse effects Dietary Supplements/adverse effects Lipid Peroxides/metabolism Lipofuscin/antagonists & inhibitors Lipofuscin/metabolism Liver/enzymology Liver/metabolism Lung/enzymology Lung/metabolism Lung/pathology Lung Neoplasms/blood Lung Neoplasms/metabolism Lung Neoplasms/pathology Mice Mice, Inbred Strains Myocardium/enzymology Myocardium/metabolism Neoplasm Proteins/agonists Neoplasm Proteins/metabolism Oxidoreductases/blood Oxidoreductases/chemistry Oxidoreductases/metabolism Pleurotus/growth & development Pleurotus/metabolism Random Allocation Selenium/administration & dosage Selenium/adverse effects Sodium Selenite/adverse effects Sodium Selenite/metabolism Zinc/administration & dosage Zinc/adverse effects Zinc Sulfate/adverse effects Zinc Sulfate/metabolism [Pt] Publication type: COMPARATIVE STUDY; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T [Nm] Name of substance: 0 (Anticarcinogenic Agents); 0 (Antioxidants); 0 (Lipid Peroxides); 0 (Lipofuscin); 0 (Neoplasm Proteins); 10102-18-8 (Sodium Selenite); 7440-66-6 (Zinc); 7733-02-0 (Zinc Sulfate); 7782-49-2 (Selenium); EC 1.- (Oxidoreductases) [Em] Entry month: 1305 [Js] Journal subset: IM [Da] Date of entry for processing: 121130 [St] Status: MEDLINE [do] DOI: 10.1007/s12011-012-9454-1

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