Database : MEDLINE
Search on : Scleromyxedema [Words]
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[PMID]: 29283443
[Au] Autor:Mark PB; Dekkers I; Blankestijn PJ; Leiner T; Roditi G
[Ad] Address:Institute of Cardiovascular and Medical Sciences, University of Glasgow, Glasgow, UK.
[Ti] Title:Letter on 'European dermatology forum S1-guideline on the diagnosis and treatment of sclerosing diseases of the skin, Part 2: Scleromyxedema, scleredema and nephrogenic systemic fibrosis'.
[So] Source:J Eur Acad Dermatol Venereol;32(2):e84-e85, 2018 Feb.
[Is] ISSN:1468-3083
[Cp] Country of publication:England
[La] Language:eng
[Pt] Publication type:LETTER
[Em] Entry month:1712
[Cu] Class update date: 180221
[Lr] Last revision date:180221
[St] Status:In-Data-Review
[do] DOI:10.1111/jdv.14774

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[PMID]: 29460897
[Au] Autor:Radenska-Lopovok SG; Volkova P
[Ad] Address:I.M. Sechenov First Moscow State Medical University, Moscow, Russia.
[Ti] Title:Skleromiksedema - dermatologicheskaia problema ili sistemnyi patologicheskii protsess? [Is scleromyxedema a skin problem or systemic pathological process?]
[So] Source:Arkh Patol;80(1):63-66, 2018.
[Is] ISSN:0004-1955
[Cp] Country of publication:Russia (Federation)
[La] Language:rus
[Ab] Abstract:Scleromyxedema is regarded as a rare cutaneous mucinosis from a group of lichen myxedematosus characterized by diffuse mucin deposition, sclerosis, and lichenoid eruptions in the absence of thyroid disease. The paper discusses the pathogenesis of the disease and histological changes in tissues. It underlines the need for using histochemical tests to identify acidic and neutral glycosaminoglycans and gives a differential diagnosis of this disease.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180220
[Lr] Last revision date:180220
[St] Status:In-Data-Review
[do] DOI:10.17116/patol201880163-66

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[PMID]: 29447654
[Au] Autor:Christman MP; Sukhdeo K; Kim RH; Meehan S; Rieder E; Sicco KL; Franks A
[Ad] Address:New York University Langone Health, New York.
[Ti] Title:Papular mucinosis, or localized lichen myxedematosis (LM) (discrete papular type).
[So] Source:Dermatol Online J;23(12), 2017 Dec 15.
[Is] ISSN:1087-2108
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Lichen myxedematosus is condition characterized by localized areas of dermal deposition of mucin, presenting with firm papules localized to few areas of the body. The condition needs to be excluded from scleromyxedema, which, in addition to the firm papular eruption, has areas of induration and is usually associated with a monoclonal gammopathyand systemic symptoms. We present a 62-year-old woman with a several-year history of asymptomatic, firm papules over the face and arms with no evidence of thyroid disease or a monoclonal gammopathy,which is consistent with a diagnosis of localized lichen myxedematosus, the discrete papular variant. The patient is being treated with a topical calcineurininhibitor.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180215
[Lr] Last revision date:180215
[St] Status:In-Process

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[PMID]: 29280826
[Au] Autor:Chatterjee S; Hedman BJ; Kirby DF
[Ti] Title:An Unusual Cause of Dysphagia.
[So] Source:J Clin Rheumatol;, 2017 Dec 27.
[Is] ISSN:1536-7355
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Scleredema adultorum of Buschke is a rare scleroderma mimic characterized by woody induration of the skin involving the back of the neck, interscapular region, upper and mid back, and sometimes the face, upper chest, and upper arms. Dysphagia is rare and not well characterized. We describe a case of scleredema that developed significant dysphagia affecting both the oropharyngeal phase and the esophageal phase of deglutition. Diagnostic evaluation revealed mild-moderate oropharyngeal and severe esophageal dysmotility. This is the first reported case of scleredema that exhibited severe dysmotility due to involvement of the smooth muscle of the entire esophagus. This case adds another sclerosing skin disorder to the list of causes of significant symptomatic dysphagia due to pharyngoesophageal dysmotility: systemic sclerosis, scleromyxedema, and chronic graft-versus-host disease. We hypothesize that dysmotility resulted from infiltration of the pharynx and esophagus with acid mucopolysaccharides. However, in the absence of histopathologic evidence, this is purely speculative. To develop an effective therapeutic approach, further studies are needed to understand the underlying pathophysiology of this uncommon but debilitating complication of scleredema.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171227
[Lr] Last revision date:171227
[St] Status:Publisher
[do] DOI:10.1097/RHU.0000000000000666

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[PMID]: 29228499
[Au] Autor:Hoffmann JHO; Enk AH
[Ad] Address:Department of Dermatology, University of Heidelberg, Heidelberg, Germany.
[Ti] Title:High-dose intravenous immunoglobulins for the treatment of dermatological autoimmune diseases.
[So] Source:J Dtsch Dermatol Ges;15(12):1211-1226, 2017 Dec.
[Is] ISSN:1610-0387
[Cp] Country of publication:Germany
[La] Language:eng
[Ab] Abstract:Based on their immunomodulatory properties, high-dose intravenous immunoglobulins (IVIGs) are successfully used in the treatment of various dermatological autoimmune diseases, in particular pemphigus vulgaris and dermatomyositis. In autoimmune bullous diseases, IVIGs can be used in an adjuvant setting (second- or third-line therapy) once combined immunosuppressive regimens have failed. In dermatomyositis, IVIGs may already be employed as an adjuvant second-line therapy after failure of corticosteroid monotherapy. In scleromyxedema, IVIGs may be considered as first-line treatment, given the lack of effective and safe alternatives. Other potential indications for IVIGs may include severe recalcitrant cases of systemic vasculitis and systemic lupus erythematosus. Toxic epidermal necrolysis may be an indication for high-dose IVIGs if administered early. Common, readily manageable side effects include nausea, headache, fatigue, and febrile infusion reactions. Severe adverse events such as thromboembolic events, anaphylaxis, and acute renal failure are very uncommon. The risk of viral transmission is very low. Potential mechanisms of action include upregulation of inhibitory Fc receptors, reduction of the half-life of endogenous immunoglobulins due to displacement from protective receptor sites, neutralization of autoantibodies by anti-idiotypic antibodies, as well as inhibition of complement activation.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171211
[Lr] Last revision date:171211
[St] Status:In-Process
[do] DOI:10.1111/ddg.13389

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[PMID]: 29214719
[Au] Autor:Arginelli F; Paganelli A; Rongioletti F; Pellacani G; Conti A
[Ad] Address:Dermatology Clinic, Azienda Ospedaliero-Universitaria Policlinico di Modena, Italy.
[Ti] Title:Ineffectiveness of infliximab CT-P13 for the treatment of scleromyxedema: A case report.
[So] Source:Dermatol Ther;, 2017 Dec 07.
[Is] ISSN:1529-8019
[Cp] Country of publication:United States
[La] Language:eng
[Pt] Publication type:LETTER
[Em] Entry month:1712
[Cu] Class update date: 171207
[Lr] Last revision date:171207
[St] Status:Publisher
[do] DOI:10.1111/dth.12583

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[PMID]: 28961556
[Au] Autor:Long V; Tan W; Lee SSJ; Thng TGS
[Ad] Address:*Department of General Medicine, Tan Tock Seng Hospital, Singapore; †National Skin Centre, Singapore; and ‡FAMS Dip Dermatopathology (ICDP-UEMS), National Skin Centre, Singapore.
[Ti] Title:Interstitial Granulomatous Variant of Scleromyxedema-A Diagnostic Pitfall.
[So] Source:Am J Dermatopathol;, 2017 Sep 20.
[Is] ISSN:1533-0311
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Scleromyxedema is a rare disorder where patients may develop systemic manifestations such as monoclonal gammopathy, inflammatory polyarthritis, and esophageal and neurological dysfunction. Histologically, there may be atypical variants of scleromyxedema showing features resembling interstitial granuloma annulare. We report an unusual case of scleromyxedema with interstitial granulomatous pattern and highlight potential diagnostic pitfalls when encountered with such a variant.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1709
[Cu] Class update date: 170929
[Lr] Last revision date:170929
[St] Status:Publisher
[do] DOI:10.1097/DAD.0000000000000988

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[PMID]: 28868289
[Au] Autor:Eckes B; Wang F; Moinzadeh P; Hunzelmann N; Krieg T
[Ad] Address:Department of Dermatology, University of Cologne, Cologne, Germany.
[Ti] Title:Pathophysiological Mechanisms in Sclerosing Skin Diseases.
[So] Source:Front Med (Lausanne);4:120, 2017.
[Is] ISSN:2296-858X
[Cp] Country of publication:Switzerland
[La] Language:eng
[Ab] Abstract:Sclerosing skin diseases represent a large number of distinct disease entities, which include systemic sclerosis, localized scleroderma, and scleredema adultorum. These pathologies have a common clinical appearance and share histological features. However, the specific interplay between cytokines and growth factors, which activate different mesenchymal cell populations and production of different extracellular matrix components, determines the biomechanical properties of the skin and the clinical features of each disease. A better understanding of the mechanisms underlying these events is prerequisite for developing novel targeted therapeutic approaches.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1709
[Cu] Class update date: 170906
[Lr] Last revision date:170906
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.3389/fmed.2017.00120

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[PMID]: 28831414
[Au] Autor:Oh SJ; Oh SH; Jun JY; Park JH; Lee JH; Lee DY; Lee JH; Yang JM
[Ad] Address:Department of Dermatology, Samsung Medical Center, Sungkyunkwan University School of Medicine, Seoul, Korea.
[Ti] Title:Paraneoplastic atypical scleromyxedema with advanced gastric cancer.
[So] Source:JAAD Case Rep;3(5):376-378, 2017 Sep.
[Is] ISSN:2352-5126
[Cp] Country of publication:United States
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1708
[Cu] Class update date: 170827
[Lr] Last revision date:170827
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1016/j.jdcr.2017.04.005

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[PMID]: 28786499
[Au] Autor:Knobler R; Moinzadeh P; Hunzelmann N; Kreuter A; Cozzio A; Mouthon L; Cutolo M; Rongioletti F; Denton CP; Rudnicka L; Frasin LA; Smith V; Gabrielli A; Aberer E; Bagot M; Bali G; Bouaziz J; Braae Olesen A; Foeldvari I; Frances C; Jalili A; Just U; Kähäri V; Kárpáti S; Kofoed K; Krasowska D; Olszewska M; Orteu C; Panelius J; Parodi A; Petit A; Quaglino P; Ranki A; Sanchez Schmidt JM; Seneschal J; Skrok A; Sticherling M; Sunderkötter C; Taieb A; Tanew A; Wolf P; Worm M; Wutte NJ; Krieg T
[Ad] Address:Department of Dermatology, Medical University of Vienna, Vienna, Austria.
[Ti] Title:European dermatology forum S1-guideline on the diagnosis and treatment of sclerosing diseases of the skin, Part 2: Scleromyxedema, scleredema and nephrogenic systemic fibrosis.
[So] Source:J Eur Acad Dermatol Venereol;31(10):1581-1594, 2017 Oct.
[Is] ISSN:1468-3083
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:The term 'sclerosing diseases of the skin' comprises specific dermatological entities which have fibrotic changes of the skin in common. These diseases mostly manifest in different clinical subtypes according to cutaneous and extracutaneous involvement and can sometimes be difficult to distinguish from each other. The present guideline focuses on characteristic clinical and histopathological features, diagnostic scores and the serum autoantibodies most useful for differential diagnosis. In addition, current strategies in the first- and advanced-line therapy of sclerosing skin diseases are addressed in detail. Part 2 of this guideline provides clinicians with an overview of the diagnosis and treatment of scleromyxedema, scleredema (of Buschke) and nephrogenic systemic sclerosis (nephrogenic fibrosing dermopathy).
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1708
[Cu] Class update date: 171023
[Lr] Last revision date:171023
[St] Status:In-Process
[do] DOI:10.1111/jdv.14466


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