Database : MEDLINE
Search on : Shared and Paranoid and Disorder [Words]
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[PMID]: 28548577
[Au] Autor:Francois D; Bander E; D'Agostino M; Swinburne A; Broderick L; Grody MB; Salajegheh A
[Ti] Title:Folie à Deux in Monozygotic Twins with Cerebral Palsy.
[So] Source:Clin Schizophr Relat Psychoses;11(1):61-64, 2017.
[Is] ISSN:1935-1232
[Cp] Country of publication:United States
[La] Language:eng
[Mh] MeSH terms primary: Cerebral Palsy/complications
Diseases in Twins/psychology
Schizophrenia/complications
Schizophrenic Psychology
Shared Paranoid Disorder/psychology
Twins, Monozygotic/psychology
[Mh] MeSH terms secundary: Adult
Female
Humans
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Em] Entry month:1707
[Cu] Class update date: 180119
[Lr] Last revision date:180119
[Js] Journal subset:IM
[Da] Date of entry for processing:170527
[St] Status:MEDLINE
[do] DOI:10.3371/1935-1232-11.1.61

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[PMID]: 28993545
[Au] Autor:Tay JL; Li Z
[Ad] Address:Institute of Mental Health, Buangkok Green Medical Park, 10 Buangkok View, Singapore 539747.
[Ti] Title:Folie à deux by Proxy in a Father, after Physical Abuse by a Mentally Ill Daughter.
[So] Source:East Asian Arch Psychiatry;27(3):121-4, 2017 Sep.
[Is] ISSN:2224-7041
[Cp] Country of publication:China
[La] Language:eng
[Ab] Abstract:This is the first case report in a country predominated by Chinese that describes the physical abuse of a recipient by the inducer in shared delusional disorder. The report describes a 42-year-old patient who physically abused her father until he submitted to her delusions. Subsequently and for years, both sustained persecutory delusions against their neighbours. While the patient was undergoing treatment, the father continued reinforcing her delusions. There is a need to explore the possibility of any forms of abuse of the recipient by the inducer in shared psychotic disorder. This report discusses the development of such delusions in a specific case and makes recommendations for the management of similar cases.
[Pt] Publication type:CASE REPORTS
[Em] Entry month:1710
[Cu] Class update date: 171010
[Lr] Last revision date:171010
[St] Status:In-Process

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[PMID]: 28414014
[Au] Autor:Reichborn-Kjennerud T; Krueger RF; Ystrom E; Torvik FA; Rosenström TH; Aggen SH; South SC; Neale MC; Knudsen GP; Kendler KS; Czajkowski NO
[Ad] Address:Department of Mental Disorders,Norwegian Institute of Public Health,Oslo,Norway.
[Ti] Title:Do DSM-5 Section II personality disorders and Section III personality trait domains reflect the same genetic and environmental risk factors?
[So] Source:Psychol Med;47(12):2205-2215, 2017 Sep.
[Is] ISSN:1469-8978
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: DSM-5 includes two conceptualizations of personality disorders (PDs). The classification in Section II is identical to the one found in DSM-IV, and includes 10 categorical PDs. The Alternative Model (Section III) includes criteria for dimensional measures of maladaptive personality traits organized into five domains. The degree to which the two conceptualizations reflect the same etiological factors is not known. METHODS: We use data from a large population-based sample of adult twins from the Norwegian Institute of Public Health Twin Panel on interview-based DSM-IV PDs and a short self-report inventory that indexes the five domains of the DSM-5 Alternative Model plus a domain explicitly targeting compulsivity. Schizotypal, Paranoid, Antisocial, Borderline, Avoidant, and Obsessive-compulsive PDs were assessed at the same time as the maladaptive personality traits and 10 years previously. Schizoid, Histrionic, Narcissistic, and Dependent PDs were only assessed at the first interview. Biometric models were used to estimate overlap in genetic and environmental risk factors. RESULTS: When measured concurrently, there was 100% genetic overlap between the maladaptive trait domains and Paranoid, Schizotypal, Antisocial, Borderline, and Avoidant PDs. For OCPD, 43% of the genetic variance was shared with the domains. Genetic correlations between the individual domains and PDs ranged from +0.21 to +0.91. CONCLUSION: The pathological personality trait domains, which are part of the Alternative Model for classification of PDs in DSM-5 Section III, appears to tap, at an aggregate level, the same genetic risk factors as the DSM-5 Section II classification for most of the PDs.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1704
[Cu] Class update date: 170808
[Lr] Last revision date:170808
[St] Status:In-Process
[do] DOI:10.1017/S0033291717000824

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[PMID]: 28399309
[Au] Autor:Bebbington P; Freeman D
[Ad] Address:UCL Division of Psychiatry, Faculty of Brain Sciences, Tottenham Court Road, London, UK.
[Ti] Title:Transdiagnostic Extension of Delusions: Schizophrenia and Beyond.
[So] Source:Schizophr Bull;43(2):273-282, 2017 03 01.
[Is] ISSN:1745-1701
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Delusion is central to the conceptualization, definition, and identification of schizophrenia. However, in current classifications, the presence of delusions is neither necessary nor sufficient for the diagnosis of schizophrenia, nor is it sufficient to exclude the diagnosis of some other psychiatric conditions. Partly as a consequence of these classification rules, it is possible for delusions to exist transdiagnostically. In this article, we evaluate the extent to which this happens, and in what ways the characteristics of delusions vary according to diagnostic context. We were able to examine their presence and form in delusional disorder, affective disorder, obsessive-compulsive disorder, borderline personality disorder, and dementia, in all of which they have an appreciable presence. There is some evidence that the mechanisms of delusion formation are, at least to an extent, shared across these disorders. This transdiagnostic extension of delusions is an argument for targeting them therapeutically in their own right. However there is a dearth of research to enable the rational transdiagnostic deployment of either pharmacological or psychological treatments.
[Mh] MeSH terms primary: Affective Disorders, Psychotic/classification
Borderline Personality Disorder/classification
Comorbidity
Delusions/classification
Dementia/classification
Obsessive-Compulsive Disorder/classification
Schizophrenia, Paranoid/classification
Schizophrenia/classification
[Mh] MeSH terms secundary: Affective Disorders, Psychotic/epidemiology
Borderline Personality Disorder/epidemiology
Delusions/epidemiology
Dementia/epidemiology
Humans
Obsessive-Compulsive Disorder/epidemiology
Schizophrenia/epidemiology
Schizophrenia, Paranoid/epidemiology
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1707
[Cu] Class update date: 170926
[Lr] Last revision date:170926
[Js] Journal subset:IM
[Da] Date of entry for processing:170412
[St] Status:MEDLINE
[do] DOI:10.1093/schbul/sbw191

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[PMID]: 28262165
[Au] Autor:Teo DC; Abraham AM; Peh AL
[Ad] Address:Department of Psychological Medicine, Changi General Hospital, 2 Simei Street 3, 529889, Singapore. Electronic address: david_teo@cgh.com.sg.
[Ti] Title:Folie à deux and Fregoli syndrome with greater severity in the 'secondary' - A case report.
[So] Source:Asian J Psychiatr;25:254-255, 2017 Feb.
[Is] ISSN:1876-2026
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Folie à deux and Fregoli syndrome are rarely seen in clinical practice. We present a case in which these rare syndromes co-occur. Remarkably, in this case the 'secondary' develops a more serious illness course than the 'primary' and reciprocally induces a de novo Fregoli delusion in the 'primary'. This case discusses how socio-cultural factors, such as interdependent family dynamics, could have precipitated this rare variant of folie à deux. It also highlights the importance of making culturally-sensitive formulations for treatment.
[Mh] MeSH terms primary: Mother-Child Relations
Schizophrenia, Paranoid/physiopathology
Shared Paranoid Disorder/physiopathology
[Mh] MeSH terms secundary: Adult Children
Female
Humans
[Pt] Publication type:CASE REPORTS; LETTER
[Em] Entry month:1703
[Cu] Class update date: 170314
[Lr] Last revision date:170314
[Js] Journal subset:IM
[Da] Date of entry for processing:170307
[St] Status:MEDLINE

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[PMID]: 28152169
[Au] Autor:Georgakis MK; Dimitriou NG; Karalexi MA; Mihas C; Nasothimiou EG; Tousoulis D; Tsivgoulis G; Petridou ET
[Ad] Address:Department of Hygiene, Epidemiology and Medical Statistics, School of Medicine, National and Kapodistrian University of Athens, Athens, Greece.
[Ti] Title:Albuminuria in Association with Cognitive Function and Dementia: A Systematic Review and Meta-Analysis.
[So] Source:J Am Geriatr Soc;65(6):1190-1198, 2017 Jun.
[Is] ISSN:1532-5415
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:OBJECTIVES: Cerebral microvascular disease is considered to contribute to cognitive dysfunction. We opted to explore whether albuminuria, a marker of systemic microangiopathy, is associated with cognitive impairment, dementia, and cognitive function. DESIGN: Systematic review; independent reviewers screened 2359 articles, derived through the search strategy, for identification of observational studies quantifying an association of albuminuria with the outcomes of interest, abstracted data on study characteristics and results and evaluated studies on quality using the Newcastle-Ottawa scale. SETTING: Community. PARTICIPANTS: Adults. MESUREMENTS: Cognitive impairment and dementia, defined by validated neuropsychological tests or clinical guidelines, respectively, and cognitive function, assessed by validated instruments. RESULTS: Thirty-two eligible studies were identified. Albuminuria was associated with cognitive impairment (Odds Ratio (OR): 1.35, 95% Confidence Interval (CI): 1.19-1.53; 7,852 cases), dementia (OR: 1.35, 95% CI: 1.10-1.65; 5,758 cases), clinical Alzheimer's disease (OR: 1.37, 95% CI: 1.11-1.69; 629 cases) and vascular dementia (OR: 1.96, 95% CI: 1.16-3.31; 186 cases); the effect remained significant among longitudinal, population-based and high quality studies. Time-to-event analysis on prospective studies of non-demented at baseline individuals also showed a significant association with incident dementia (Risk Ratio: 1.52, 95% CI: 1.16-1.99; 971 cases). Worse global cognitive performance (Hedge's g: -0.13, 95% CI: -0.18, -0.09; 68,348 subjects) and accelerated cognitive decline (g: -0.20, 95% CI: -0.34, -0.07; 31,792 subjects) were noted among subjects with albuminuria, who also scored lower in executive function, processing speed, verbal fluency, and verbal memory. CONCLUSIONS: Albuminuria was independently associated with cognitive impairment, dementia and cognitive decline. The stronger effects for vascular dementia and cognitive performance in domains primarily affected by microvascular disease support that the association could be mediated by shared microvascular pathology in the kidney and the brain.
[Mh] MeSH terms primary: Albuminuria/complications
Cognition/physiology
Dementia/diagnosis
[Mh] MeSH terms secundary: Albuminuria/epidemiology
Albuminuria/urine
Cognitive Dysfunction/etiology
Dementia/epidemiology
Dementia/etiology
Humans
Neuropsychological Tests
Vascular Diseases
[Pt] Publication type:JOURNAL ARTICLE; META-ANALYSIS; REVIEW
[Em] Entry month:1708
[Cu] Class update date: 170808
[Lr] Last revision date:170808
[Js] Journal subset:IM
[Da] Date of entry for processing:170203
[St] Status:MEDLINE
[do] DOI:10.1111/jgs.14750

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[PMID]: 27823940
[Au] Autor:Rodríguez-Cerdeira C; Sánchez-Blanco E; Sánchez-Blanco B; Carnero-Gregorio M; Pychodermatology group of CILAD
[Ad] Address:Dermatology Service, Hospital do Meixoeiro and University of Vigo, Spain; Postdoctoral researcher, University of Vigo, Spain. Electronic address: carmencerdeira33@gmail.com.
[Ti] Title:Delusional infestation.
[So] Source:Am J Emerg Med;35(2):357-360, 2017 Feb.
[Is] ISSN:1532-8171
[Cp] Country of publication:United States
[La] Language:eng
[Mh] MeSH terms primary: Delusions/diagnosis
Delusions/psychology
Hallucinations/psychology
Parasitic Diseases/psychology
Psychotic Disorders/diagnosis
Psychotic Disorders/psychology
Shared Paranoid Disorder/diagnosis
[Mh] MeSH terms secundary: Antipsychotic Agents/therapeutic use
Benzodiazepines/therapeutic use
Delusions/drug therapy
Female
Humans
Male
Middle Aged
Psychotic Disorders/drug therapy
Referral and Consultation
Shared Paranoid Disorder/drug therapy
Shared Paranoid Disorder/psychology
[Pt] Publication type:LETTER
[Nm] Name of substance:0 (Antipsychotic Agents); 12794-10-4 (Benzodiazepines); N7U69T4SZR (olanzapine)
[Em] Entry month:1704
[Cu] Class update date: 170808
[Lr] Last revision date:170808
[Js] Journal subset:IM
[Da] Date of entry for processing:161109
[St] Status:MEDLINE

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[PMID]: 27439544
[Au] Autor:Riggs S; Perry T; Dowben J; Burson R
[Ad] Address:Psychiatry Resident, Department of Psychiatry, University of Texas Health Science Center at San Antonio (UTHSCSA), San Antonio, Texas, USA.
[Ti] Title:Vive La France: Three Delusional Disorders Originally Reported in the French Medical Literature.
[So] Source:Perspect Psychiatr Care;53(1):5-9, 2017 Jan.
[Is] ISSN:1744-6163
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Delusions as a feature of psychosis have long captured the fascination of both psychiatry and the public at large. The French first described three famous delusions: the Cotard delusion, folie à deux, and Capgras delusion. In this article, we examine a case illustrating each delusion and the hallmark features of each as well as a brief discussion about the current understanding of these disorders.
[Mh] MeSH terms primary: Delusions/psychology
Schizophrenia, Paranoid/classification
Schizophrenia, Paranoid/psychology
Shared Paranoid Disorder/psychology
[Mh] MeSH terms secundary: Adult
Aged
Female
France
Humans
Male
Middle Aged
Psychiatry
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE; REVIEW
[Em] Entry month:1703
[Cu] Class update date: 170323
[Lr] Last revision date:170323
[Js] Journal subset:N
[Da] Date of entry for processing:160722
[St] Status:MEDLINE
[do] DOI:10.1111/ppc.12176

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[PMID]: 27277535
[Au] Autor:Cohn-Hokke PE; Holstege H; Weiss MM; van der Flier WM; Barkhof F; Sistermans EA; Pijnenburg YA; van Swieten JC; Meijers-Heijboer H; Scheltens P
[Ad] Address:Department of Clinical Genetics, VU University Medical Center, Amsterdam, The Netherlands.
[Ti] Title:A novel CCM2 variant in a family with non-progressive cognitive complaints and cerebral microbleeds.
[So] Source:Am J Med Genet B Neuropsychiatr Genet;174(3):220-226, 2017 Apr.
[Is] ISSN:1552-485X
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Lobar cerebral microbleeds are most often sporadic and associated with Alzheimer's disease. The aim of our study was to identify the underlying genetic defect in a family with cognitive complaints and multiple lobar microbleeds and a positive family history for early onset Alzheimer's disease. We performed exome sequencing followed by Sanger sequencing for validation purposes on genomic DNA of three siblings with cognitive complaints, reduced amyloid-beta-42 in CSF and multiple cerebral lobar microbleeds. We checked for the occurrence of the variant in a cohort of 363 patients with early onset dementia and/or microbleeds. A novel frameshift variant (c.236_237delAC) generating a premature stop codon in the CCM2 gene shared by all three siblings was identified. Pathogenicity of the variant was supported by the presence of cerebral cavernous malformations in two of the siblings and by the absence of the variant exome variant databases. Two siblings were homozygous for APOE-ϵ4; one heterozygous. The cognitive complaints, reduced amyloid-beta-42 in CSF and microbleeds suggest preclinical Alzheimer's disease, but the stability of the cognitive complaints does not. We hypothesize that the phenotype in this family may be due to a combination of the CCM2 variant and the APOE status. © 2016 The Authors. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics Published by Wiley Periodicals, Inc.
[Mh] MeSH terms primary: Alzheimer Disease/genetics
Carrier Proteins/genetics
[Mh] MeSH terms secundary: Adult
Amyloid beta-Peptides/genetics
Carrier Proteins/metabolism
Cerebral Hemorrhage/genetics
Cognition
Cognition Disorders/genetics
Dementia/etiology
Dementia/genetics
Female
Genetic Variation/genetics
Humans
Magnetic Resonance Imaging
Male
Middle Aged
Pedigree
Sequence Analysis, DNA
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:0 (Amyloid beta-Peptides); 0 (CCM2 protein, human); 0 (Carrier Proteins)
[Em] Entry month:1709
[Cu] Class update date: 170921
[Lr] Last revision date:170921
[Js] Journal subset:IM
[Da] Date of entry for processing:160610
[St] Status:MEDLINE
[do] DOI:10.1002/ajmg.b.32468

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[PMID]: 27906088
[Au] Autor:Vargas Alves Nunes A; Odebrecht Vargas Nunes S; Strano T; Pascolat G; Schier Doria GM; Nasser Ehlke M
[Ad] Address:Pediatrics Department, Hospital Universitário Evangélico de Curitiba, 2885 Padre Agostinho, apto 1304 torre barigui, ZIP 80710-000, Curitiba, Paraná, Brazil. dodonunes@hotmail.com.
[Ti] Title:Folie à Deux and its interaction with early life stress: a case report.
[So] Source:J Med Case Rep;10(1):339, 2016 Dec 01.
[Is] ISSN:1752-1947
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Folie à deux is a very rare psychiatric syndrome in which a psychotic symptom is transmitted from one individual to another. We present a case of folie à deux occurring during childhood, which is not an usual presentation of this syndrome. In this case, the disorder is correlated with child abuse and neglect, which possibly had a role in the development of the symptoms in our case. CASE PRESENTATION: We present a case of folie à deux between an "induced" 9-year-old black Brazilian boy and the "inducer", his grandmother. They were found to be sharing similar auditory and visual hallucinations and delusional beliefs. The boy was neglected by his parents and was being cared for by his grandmother, who had a history of mental disorder. CONCLUSIONS: The close relationship between the boy and his grandmother, the family history of first-degree psychosis, and the child abuse and neglect suffered by the boy may have altered his vulnerability to early-onset psychosis and, in this case, folie à deux.
[Mh] MeSH terms primary: Child Abuse/psychology
Grandparents/psychology
Shared Paranoid Disorder/complications
Shared Paranoid Disorder/diagnosis
Stress, Psychological/complications
Stress, Psychological/diagnosis
[Mh] MeSH terms secundary: Age of Onset
Anticonvulsants/therapeutic use
Carbamazepine/therapeutic use
Child
Delusions/complications
Hallucinations/complications
Humans
Male
Shared Paranoid Disorder/drug therapy
Shared Paranoid Disorder/psychology
Social Environment
Stress, Psychological/drug therapy
Stress, Psychological/psychology
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:0 (Anticonvulsants); 33CM23913M (Carbamazepine)
[Em] Entry month:1705
[Cu] Class update date: 170510
[Lr] Last revision date:170510
[Js] Journal subset:IM
[Da] Date of entry for processing:161202
[St] Status:MEDLINE


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