Database : MEDLINE
Search on : Vasculitis [Words]
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[PMID]: 26346417
[Au] Autor:M'Saad S; Kammoun K; Yangui I; Fourati H; Feki W; Marouen F; Daoud E; Kammoun S
[Ad] Address:Service de pneumo-allergologie, CHU Hédi-Chaker, faculté de médecine de Sfax, route al Ain, Km 0,5, 3029 Sfax, Tunisie. Electronic address: msaadsameh@yahoo.fr....
[Ti] Title:Micropolyangéite, syndrome d'emphysème des sommets et fibrose pulmonaire des bases. [Combined pulmonary fibrosis and emphysema associated with microscopic polyangiitis].
[So] Source:Rev Mal Respir;33(5):391-6, 2016 May.
[Is] ISSN:1776-2588
[Cp] Country of publication:France
[La] Language:fre
[Ab] Abstract:BACKGROUND: Combined pulmonary fibrosis and emphysema (CPFE) is a rare entity of unknown etiology. It usually occurs in the context of smoking and, less commonly, connective tissue disease. However, it has been rarely previously described in the context of vasculitis. OBSERVATION: We report a case of CPFE occurring in a 44-year-old man, who was a light smoker without any previous medical history. He presented with fever, chronic cough and breathlessness that progressively evolved to acute respiratory failure. At the initial evaluation, CT scan showed emphysema and patchy bilateral areas of ground-glass opacity. Three years later, the patient simultaneously developed a honeycomb fibrosis and a microscopic polyangiitis with renal involvement justifying the introduction of an immunosuppressive treatment in combination with high dose of systemic corticosteroids. After a stabilization period of 6years, the patient gradually developed chronic respiratory failure with moderate pulmonary hypertension requiring long-term oxygen therapy and nocturnal non-invasive ventilation. CONCLUSION: The association of microscopic polyangiitis to CFPE suggests that autoimmune diseases may have a common pathogenic role in the development of emphysematous and fibrotic lesions in CPFE.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1606
[Js] Journal subset:IM
[St] Status:In-Data-Review

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[PMID]: 27179903
[Au] Autor:Mavrogeni SI; Kitas GD; Dimitroulas T; Sfikakis PP; Seo P; Gabriel S; Patel AR; Gargani L; Bombardieri S; Matucci-Cerinic M; Lombardi M; Pepe A; Aletras AH; Kolovou G; Miszalski T; van Riel P; Semb A; Gonzalez-Gay MA; Dessein P; Karpouzas G; Puntmann V; Nagel E; Bratis K; Karabela G; Stavropoulos E; Katsifis G; Koutsogeorgopoulou L; van Rossum A; Rademakers F; Pohost G; Lima JA
[Ad] Address:Onassis Cardiac Surgery Center, Athens, Greece. Electronic address: soma13@otenet.gr....
[Ti] Title:Cardiovascular magnetic resonance in rheumatology: Current status and recommendations for use.
[So] Source:Int J Cardiol;217:135-48, 2016 Aug 15.
[Is] ISSN:1874-1754
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Targeted therapies in connective tissue diseases (CTDs) have led to improvements of disease-associated outcomes, but life expectancy remains lower compared to general population due to emerging co-morbidities, particularly due to excess cardiovascular risk. Cardiovascular magnetic resonance (CMR) is a noninvasive imaging technique which can provide detailed information about multiple cardiovascular pathologies without using ionizing radiation. CMR is considered the reference standard for quantitative evaluation of left and right ventricular volumes, mass and function, cardiac tissue characterization and assessment of thoracic vessels; it may also be used for the quantitative assessment of myocardial blood flow with high spatial resolution and for the evaluation of the proximal coronary arteries. These applications are of particular interest in CTDs, because of the potential of serious and variable involvement of the cardiovascular system during their course. The International Consensus Group on CMR in Rheumatology was formed in January 2012 aiming to achieve consensus among CMR and rheumatology experts in developing initial recommendations on the current state-of-the-art use of CMR in CTDs. The present report outlines the recommendations of the participating CMR and rheumatology experts with regards to: (a) indications for use of CMR in rheumatoid arthritis, the spondyloarthropathies, systemic lupus erythematosus, vasculitis of small, medium and large vessels, myositis, sarcoidosis (SRC), and scleroderma (SSc); (b) CMR protocols, terminology for reporting CMR and diagnostic CMR criteria for assessment and quantification of cardiovascular involvement in CTDs; and (c) a research agenda for the further development of this evolving field.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1606
[Js] Journal subset:IM
[St] Status:In-Data-Review

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[PMID]: 27287993
[Au] Autor:Wood EH; Wong RW
[Ad] Address:Byers Eye Institute at Stanford, Stanford University School of Medicine, 2452 Watson Court, Palo Alto, CA, 94303, USA. ehw@stanford.edu.
[Ti] Title:Bilateral frosted branch angiitis as the presenting sign of antiphospholipid antibody syndrome.
[So] Source:J Ophthalmic Inflamm Infect;6(1):20, 2016 Dec.
[Is] ISSN:1869-5760
[Cp] Country of publication:Germany
[La] Language:eng
[Ab] Abstract:BACKGROUND: "Frosted branch retinal angiitis" is an encompassing term for a rare, typically bilateral diffuse retinal periphlebitis that may occur in a number of varying conditions. To our knowledge, we report the first case of frosted branch angiitis as the presenting sign of antiphospholipid antibody syndrome in a 28-year-old woman. FINDINGS: This study is a retrospective case report and literature review. Serial fundus photos, fluorescein angiogram, and ocular coherence tomography taken were before and after treatment, showing resolution of diffuse retinal perivascular sheathing and macular edema along with marked improvement in visual acuity 4 months after the treatment with corticosteroids. CONCLUSIONS: Frosted branch angiitis can be seen in association with antiphospholipid antibody syndrome. Prompt recognition and treatment with corticosteroids may result in good visual prognosis, and long-term immunosuppression and additional anticoagulation may be beneficial to prevent recurrence.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1606
[Da] Date of entry for processing:160611
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1186/s12348-016-0089-9

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[PMID]: 26965982
[Au] Autor:Han SB; Lee SY; Jeong DC; Kang JH
[Ad] Address:Department of Pediatrics, The Catholic University of Korea, Seoul, Korea....
[Ti] Title:Should 2016 Criteria for Macrophage Activation Syndrome be applied in children with Kawasaki disease, as well as with systemic-onset juvenile idiopathic arthritis?
[So] Source:Ann Rheum Dis;75(7):e44, 2016 Jul.
[Is] ISSN:1468-2060
[Cp] Country of publication:England
[La] Language:eng
[Pt] Publication type:LETTER
[Em] Entry month:1606
[Js] Journal subset:IM
[St] Status:In-Data-Review
[do] DOI:10.1136/annrheumdis-2016-209395

  5 / 35430 MEDLINE  
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[PMID]: 27274829
[Au] Autor:Prendecki M; Cairns T; Pusey CD
[Ad] Address:Imperial College Renal and Transplant Centre, Hammersmith Hospital , London , UK.
[Ti] Title:Familial vasculitides: granulomatosis with polyangitis and microscopic polyangitis in two brothers with differing anti-neutrophil cytoplasm antibody specificity.
[So] Source:Clin Kidney J;9(3):429-31, 2016 Jun.
[Is] ISSN:2048-8505
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Anti-neutrophil cytoplasm antibody (ANCA)-associated vasculitis (AAV) is a group of rare autoimmune diseases. Although the aetiology of AAV is uncertain, it is likely that genetic and environmental factors contribute. We report the unusual case of two brothers presenting with AAV with differing clinical pictures and differing ANCA specificity. There is a recently identified difference in genetic risk factors associated with ANCA specificity, making it surprising that first-degree relatives develop AAV with differing clinical and serological features. Our report illustrates the complex aetiology of AAV and suggests that further research on the interaction of genetic and environmental factors is needed.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1606
[Cu] Class update date: 160610
[Lr] Last revision date:160610
[Da] Date of entry for processing:160609
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1093/ckj/sfw016

  6 / 35430 MEDLINE  
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[PMID]: 27275457
[Au] Autor:Wardrope KE; Manson L; Metcalfe W; Sullivan ED
[Ad] Address:Department of Renal Medicine, Royal Infirmary of Edinburgh, Edinburgh, UK....
[Ti] Title:Acute Respiratory Distress Syndrome and Posterior Reversible Encephalopathy Syndrome following Rituximab Therapy.
[So] Source:Case Rep Nephrol Dial;6(1):32-9, 2016 Jan-Apr.
[Is] ISSN:2296-9705
[Cp] Country of publication:Switzerland
[La] Language:eng
[Ab] Abstract:The anti-CD20 monoclonal antibody rituximab is associated with rare but significant adverse events, notably posterior reversible encephalopathy syndrome (PRES) and acute respiratory distress syndrome (ARDS). We report a case of concomitant ARDS and PRES developing after rituximab therapy for treatment of cryoglobulinaemic vasculitis. There are 7 reported cases of PRES complicating rituximab use. PRES onset varied from immediate to 21 days after administration. All patients recovered completely, and rituximab was reintroduced in half of the cases. The occurrence of ARDS in association with rituximab is rarer. Only 3 confirmed cases exist, and ARDS may occur as a delayed reaction.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1606
[Cu] Class update date: 160610
[Lr] Last revision date:160610
[Da] Date of entry for processing:160609
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1159/000444250

  7 / 35430 MEDLINE  
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[PMID]: 27224742
[Au] Autor:Gilden D; Nagel MA
[Ad] Address:aDepartment of NeurologybDepartment of Immunology and Microbiology, University of Colorado School of Medicine, Aurora, Colorado, USA.
[Ti] Title:Varicella zoster virus triggers the immunopathology of giant cell arteritis.
[So] Source:Curr Opin Rheumatol;28(4):376-82, 2016 Jul.
[Is] ISSN:1531-6963
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:PURPOSE OF REVIEW: Giant cell arteritis (GCA) is a severe form of vasculitis in the elderly. The recent discovery of varicella zoster virus (VZV) in the temporal arteries and adjacent skeletal muscle of patients with GCA, and the rationale and strategy for antiviral and corticosteroid treatment for GCA are reviewed. RECENT FINDINGS: The clinical features of GCA include excruciating headache/head pain, often with scalp tenderness, a nodular temporal arteries and decreased temporal artery pulsations. Jaw claudication, night sweats, fever, malaise, and a history of polymyalgia rheumatica (aching and stiffness of large muscles primarily in the shoulder girdle, upper back, and pelvis without objective signs of weakness) are common. ESR and CRP are usually elevated. Diagnosis is confirmed by temporal artery biopsy which reveals vessel wall damage and inflammation, with multinucleated giant cells and/or epithelioid macrophages. Skip lesions are common. Importantly, temporal artery biopsies are pathologically negative in many clinically suspect cases. This review highlights recent virological findings in temporal arteries from patients with pathologically verified GCA and in temporal arteries from patients who manifest clinical and laboratory features of GCA, but whose temporal artery biopsies (Bx) are pathologically negative for GCA (Bx-negative GCA). Virological analysis revealed that VZV is present in most GCA-positive and GCA-negative temporal artery biopsies, mostly in skip areas that correlate with adjacent GCA pathology. SUMMARY: The presence of VZV in Bx-positive and Bx-negative GCA temporal arteries indicates that VZV triggers the immunopathology of GCA. However, the presence of VZV in about 20% of temporal artery biopsies from non-GCA postmortem controls also suggests that VZV alone is not sufficient to produce disease. Treatment trials should be performed to determine if antiviral agents confer additional benefits to corticosteroids in both Bx-positive and Bx-negative GCA patients. These studies should also examine whether oral antiviral agents and corticosteroids are as effective as intravenous acyclovir and corticosteroids. Appropriate dosage and duration of treatment also remain to be determined.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1605
[Cu] Class update date: 160610
[Lr] Last revision date:160610
[Js] Journal subset:IM
[St] Status:In-Data-Review
[do] DOI:10.1097/BOR.0000000000000292

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[PMID]: 26579594
[Au] Autor:Sandhu RK; Adams T; Sibley C; Suhler EB; Kim DH
[Ad] Address:*Casey Eye Institute, Oregon Health and Science University, Portland, Oregon; †Department of Medicine, Oregon Health and Science University, Portland, Oregon; ‡Rheumatology Division, Oregon Health and Science University, Portland, Oregon; and §Veterans Administration Portland Health Care System, Portland, Oregon.
[Ti] Title:GRANULOMATOSIS WITH POLYANGIITIS (GPA) PRESENTING WITH FROSTED BRANCH ANGIITIS.
[So] Source:Retin Cases Brief Rep;10(3):249-51, 2016.
[Is] ISSN:1937-1578
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:PURPOSE: To report a case of frosted branch angiitis in a patient with granulomatosis with polyangiitis. METHODS: Clinical case report. Imaging was obtained with pseudo-color scanning laser ophthalmoscope photographs, fluorescein angiography, spectral domain optical coherence tomography, and B-scan ultrasound. RESULTS: A 24-year-old woman with a clinical history of granulomatosis with polyangiitis who presented with acute vision loss was found to have frosted branch angiitis with concurrent posterior scleritis and orbital inflammation. These findings improved rapidly after initiation of high-dose intravenous solumedrol. CONCLUSION: This is a unique case of frosted branch angiitis associated with granulomatosis with polyangiitis. The authors are not aware of a previous report of this association. Although rare, retinal vasculitis should be recognized as a potential complication of granulomatosis with polyangiitis and can respond rapidly to prompt initiation of therapy.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1605
[Cu] Class update date: 160610
[Lr] Last revision date:160610
[Js] Journal subset:IM
[St] Status:In-Data-Review
[do] DOI:10.1097/ICB.0000000000000242

  9 / 35430 MEDLINE  
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[PMID]: 27195943
[Au] Autor:Lee J; Kim DM; Yun NR; Kim YD; Park CG; Kim MW
[Ad] Address:Department of Internal Medicine, School of Medicine, Chosun University, Gwangju, Republic of Korea....
[Ti] Title:The Correlation of Endoscopic Findings and Clinical Features in Korean Patients with Scrub Typhus: A Cohort Study.
[So] Source:PLoS One;11(5):e0155810, 2016.
[Is] ISSN:1932-6203
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Scrub typhus is an infectious disease caused by Orientia tsutsugamushi-induced systemic vasculitis, but the involvement of the gastrointestinal tract and the endoscopic findings associated with scrub typhus are not well understood. We performed a prospective study and recommend performing esophagogastroduodenoscopy (EGD) for all possible scrub typhus patients, regardless of gastrointestinal symptoms. Gastrointestinal symptoms, endoscopic findings and clinical severity based on organ involvement and ICU admission were analyzed. Gastrointestinal symptoms occurred in up to 76.4% of scrub typhus patients. The major endoscopic findings were ulcers (43/127, 33.9%). Interestingly, 7.1% (9/127) of the patients presented with esophageal candidiasis. There was no correlation between the presence or absence of gastrointestinal symptoms and the endoscopic grade (P = 0.995). However, there was a positive correlation between the clinical severity and the endoscopic findings (P = 0.001). Sixty-three percent of the patients presented with erosion or ulcers on prospectively performed endoscopic evaluations, irrespective of gastrointestinal symptoms. Gastrointestinal symptoms did not reflect the need for endoscopy. Scrub typhus patients could have significant endoscopic abnormalities even in the absence of gastrointestinal symptoms.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1605
[Cu] Class update date: 160610
[Lr] Last revision date:160610
[Js] Journal subset:IM
[St] Status:In-Data-Review
[do] DOI:10.1371/journal.pone.0155810

  10 / 35430 MEDLINE  
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[PMID]: 27195791
[Au] Autor:Fry LM; Schneider DA; Frevert CW; Nelson DD; Morrison WI; Knowles DP
[Ad] Address:Animal Disease Research Unit, Agricultural Research Service, US Department of Agriculture, Pullman, Washington, United States of America....
[Ti] Title:East Coast Fever Caused by Theileria parva Is Characterized by Macrophage Activation Associated with Vasculitis and Respiratory Failure.
[So] Source:PLoS One;11(5):e0156004, 2016.
[Is] ISSN:1932-6203
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Respiratory failure and death in East Coast Fever (ECF), a clinical syndrome of African cattle caused by the apicomplexan parasite Theileria parva, has historically been attributed to pulmonary infiltration by infected lymphocytes. However, immunohistochemical staining of tissue from T. parva infected cattle revealed large numbers of CD3- and CD20-negative intralesional mononuclear cells. Due to this finding, we hypothesized that macrophages play an important role in Theileria parva disease pathogenesis. Data presented here demonstrates that terminal ECF in both Holstein and Boran cattle is largely due to multisystemic histiocytic responses and resultant tissue damage. Furthermore, the combination of these histologic changes with the clinical findings, including lymphadenopathy, prolonged pyrexia, multi-lineage leukopenia, and thrombocytopenia is consistent with macrophage activation syndrome. All animals that succumbed to infection exhibited lymphohistiocytic vasculitis of small to medium caliber blood and lymphatic vessels. In pulmonary, lymphoid, splenic and hepatic tissues from Holstein cattle, the majority of intralesional macrophages were positive for CD163, and often expressed large amounts of IL-17. These data define a terminal ECF pathogenesis in which parasite-driven lymphoproliferation leads to secondary systemic macrophage activation syndrome, mononuclear vasculitis, pulmonary edema, respiratory failure and death. The accompanying macrophage phenotype defined by CD163 and IL-17 is presented in the context of this pathogenesis.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1605
[Cu] Class update date: 160610
[Lr] Last revision date:160610
[Js] Journal subset:IM
[St] Status:In-Data-Review
[do] DOI:10.1371/journal.pone.0156004


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