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[PMID]: 29486759
[Au] Autor:Baptista CS; Monteiro C; Fernandes H; Canadas A; Guardão L; Santos JC
[Ad] Address:Department of Veterinary Clinics, UPVet, Institute of Biomedical Sciences Abel Salazar, University of Porto (ICBAS-UP), Rua Jorge Viterbo Ferreira 228, 4050-313, Porto, Portugal. csbaptista@icbas.up.pt.
[Ti] Title:Acute intraparenchymal cerebral haemorrhage in an Iberian golden eagle - a case report.
[So] Source:BMC Vet Res;14(1):60, 2018 Feb 27.
[Is] ISSN:1746-6148
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: In birds there are reports of intracranial lesions but not of the clinical, computed tomographic and histopathologic features of acute intraparenchymal cerebral haemorrhage in Iberian golden eagle. CASE PRESENTATION: The following report describes a case of a 30-year-old Iberian golden eagle (Aquila chrysaetos homeyeri) with no history of trauma, presented with acute opisthotonus, left head tilt and circling, anisocoria, positional nystagmus, and ataxia. The main differential diagnosis were hypovitaminosis B or E and intracranial disease due to trauma, infection, toxins or masses. A computed tomography (CT) of the head was performed with an 8-slices scanner and evidenced a hyperdense (63-65 HU) non-enhancing homogeneous well delineated round area in the midbrain, with 6 mm in its highest diameter. The attenuation values and the non-enhancing nature of the lesion strongly suggested the diagnosis of acute intraparenchymal haemorrhage, which was histologically confirmed after necropsy. CONCLUSIONS: In birds with a central neurological dysfunction, the diagnosis of acute brain haemorrhage should be considered when the CT evidences a non-enhancing, homogeneous, well circumscribed hyperattenuated round area.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180307
[Lr] Last revision date:180307
[St] Status:In-Process
[do] DOI:10.1186/s12917-018-1379-2

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[PMID]: 29260849
[Au] Autor:Martin TJ
[Ad] Address:Department of Ophthalmology, Wake Forest University School of Medicine , Wiston-Salem, North Carolina 27157, United States.
[Ti] Title:Horner Syndrome: A Clinical Review.
[So] Source:ACS Chem Neurosci;9(2):177-186, 2018 02 21.
[Is] ISSN:1948-7193
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Horner syndrome results from an interruption of the oculosympathetic pathway. Patients with Horner syndrome present with a slightly droopy upper lid and a smaller pupil on the affected side; less commonly, there is a deficiency of sweating over the brow or face on the affected side. This condition does not usually cause vision problems or other significant symptoms, but is important as a warning sign that the oculosympathetic pathway has been interrupted, potentially with serious and even life-threatening processes. The oculosympathetic pathway has a long and circuitous course, beginning in the brain and traveling down the spinal cord to exit in the chest, then up the neck and into the orbit. Therefore, this syndrome with unimpressive clinical findings and insignificant symptoms may be a sign of serious pathology in the head, chest, or neck. This clinical review discusses how to identify the signs, confirm the diagnosis, and evaluate the many causes of Horner syndrome.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 180226
[Lr] Last revision date:180226
[St] Status:In-Process
[do] DOI:10.1021/acschemneuro.7b00405

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[PMID]: 29334914
[Au] Autor:Tibussek D; Ghosh S; Huebner A; Schaper J; Mayatepek E; Koehler K
[Ad] Address:Department of General Pediatrics, Neonatology and Pediatric Cardiology, University Children's Hospital, Heinrich-Heine University, Moorenstrasse 5, 40225, Düsseldorf, Germany. daniel.tibussek@gmx.net.
[Ti] Title:"Crying without tears" as an early diagnostic sign-post of triple A (Allgrove) syndrome: two case reports.
[So] Source:BMC Pediatr;18(1):6, 2018 01 15.
[Is] ISSN:1471-2431
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Triple A syndrome (or Allgrove syndrome) is a rare autosomal recessive disorder characterized by alacrima, achalasia, adrenal insufficiency and autonomic/neurological abnormalities. The majority of cases are caused by mutations in the AAAS gene located on chromosome 12q13. However, the clinical picture as well as genetic testing may be complex since symptomatology is variable and mutations cannot be identified in all clinically diagnosed patients. We present two unrelated patients with triple-A syndrome illustrating the importance of alacrima as an early clinical sign. CASE PRESENTATION: A 3.5 year old girl presented with repeated hypoglycaemic myoclonic events. Adrenal insufficiency was diagnosed. In addition, alacrima, obvious since early infancy, was incidentally reported by the mother and finally lead to the clinical diagnosis of triple A syndrome. This was confirmed by positive mutation analysis of the AAAS gene. The second patient, an 8 months old boy was presented because of anisocoria and unilateral optic atrophy. MRI revealed cerebellar vermis hypotrophy. Psychomotor retardation, failure to thrive, and frequent vomiting lead to further diagnostic work-up. Achalasia was diagnosed radiologically. In addition, the mother mentioned absence of tears since birth leading to the clinical diagnosis of triple A syndrome. In contrast to the first cases genetic testing was negative. CONCLUSION: These two patients illustrate the heterogeneity of triple A syndrome in both terms, clinical expression and genetic testing. We particularly aim to stress the importance of alacrima, which should be considered as a red flag symptom. Further differential diagnosis is required in every child affected by alacrima.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180214
[Lr] Last revision date:180214
[St] Status:In-Process
[do] DOI:10.1186/s12887-017-0973-y

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[PMID]: 29334520
[Au] Autor:Kawasaki A
[Ad] Address:Department of Neuro-Ophthalmology, University of Lausanne, Hôpital Ophtalmique Jules Gonin, Lausanne, Switzerland.
[Ti] Title:Invited Commentary: Evaluation of Horner Syndrome in the MRI Era.
[So] Source:J Neuroophthalmol;38(1):52-53, 2018 Mar.
[Is] ISSN:1536-5166
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:This Invited Commentary discusses the following article: BACKGROUND:: To identify the etiologies of adult Horner syndrome (HS) in the MRI era using a targeted evaluation approach and to assess the value and yield of targeted imaging. METHODS: A retrospective chart review was performed of 200 adult outpatients with HS, confirmed with cocaine eyedrop testing. Patients were divided into subgroups based on the presence or absence of symptoms and those who did or did not receive additional testing with hydroxyamphetamine drops. Imaging was obtained based on pharmacologic localization and/or clinical evaluation. The etiology of HS and the yield of imaging were determined in all subgroups. RESULTS: Imaging showed causative lesions in 24 of 179 (12.84%) imaged patients with HS, and 13 (69.0%) were determined "idiopathic." Of the patients who underwent testing with hydroxyamphetamine drops (132 patients), 86 had a postganglionic localization with an imaging yield of 8.1%, and 46 had preganglionic cause with an imaging yield of 21.7%. Fifty-three patients (26.5%) never noticed ptosis/anisocoria before examination, and the imaging yield in this subgroup was 2.8%. Eighteen of the 200 patients (9.0%) had serious pathology, including carotid artery dissection, brain, or neck mass, and 6 of these (31.6%) had acute symptoms and/or pain. CONCLUSION: HS is most often idiopathic with serious pathology being relatively infrequent. When determining etiology, the absence of symptoms is not predictive of the pathology. However, acute onset of symptoms and/or pain are possible indicators for serious pathology. Localizing the lesion using hydroxyamphetamine drops whenever obtainable and available is still an efficient way to target imaging evaluation.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180213
[Lr] Last revision date:180213
[St] Status:In-Data-Review
[do] DOI:10.1097/WNO.0000000000000626

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[PMID]: 29426462
[Au] Autor:Nakatsuka AS; Beaver HA; Lee AG
[Ad] Address:University of Texas Medical Branch, Galveston, TX. Electronic address: austin32@hawaii.edu.
[Ti] Title:Mydriasis due to Opcon-A: An indication to avoid pharmacologic testing for anisocoria.
[So] Source:Can J Ophthalmol;53(1):e6-e7, 2018 Feb.
[Is] ISSN:1715-3360
[Cp] Country of publication:England
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180210
[Lr] Last revision date:180210
[St] Status:In-Data-Review

  6 / 814 MEDLINE  
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[PMID]: 29222127
[Au] Autor:Conrad EC; Jivraj I; Kardon R; Liu GT
[Ad] Address:From the Hospital of the University of Pennsylvania (E.C.C., I.J., G.T.L.), Philadelphia; Children's Hospital of Philadelphia (I.J., G.T.L.), PA; and University of Iowa and Veterans Administration (R.K.), Iowa City. erin.conrad@uphs.upenn.edu.
[Ti] Title:Unilateral periodic pupillary constriction causing alternating anisocoria.
[So] Source:Neurology;90(2):86-88, 2018 Jan 09.
[Is] ISSN:1526-632X
[Cp] Country of publication:United States
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 180109
[Lr] Last revision date:180109
[St] Status:In-Data-Review
[do] DOI:10.1212/WNL.0000000000004809

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[PMID]: 29305356
[Au] Autor:Royce L; Schulz C; Brown N
[Ad] Address:Department of General Paediatrics, Salisbury NHS Foundation Trust, Odstock Road, Salisbury, SP2 8BJ, UK.
[Ti] Title:Case of a fixed and dilated pupil: acute anisocoria secondary to aerosol ipratropium bromide.
[So] Source:Arch Dis Child;, 2018 Jan 04.
[Is] ISSN:1468-2044
[Cp] Country of publication:England
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180106
[Lr] Last revision date:180106
[St] Status:Publisher

  8 / 814 MEDLINE  
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[PMID]: 29245300
[Au] Autor:Liu Y; Li K; Huang Y; Sun J; Gao X
[Ad] Address:Department of Neurosurgery, Ningbo First Hospital, Ningbo Hospital of Zhejiang University, Ningbo, Zhejiang, China.
[Ti] Title:Treatment of the superior sagittal sinus and transverse sinus thrombosis associated with intracranial hemorrhage with the mechanical thrombectomy and thrombolytics: Case report.
[So] Source:Medicine (Baltimore);96(49):e9038, 2017 Dec.
[Is] ISSN:1536-5964
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:RATIONALE: The superior sagittal sinus (SSS) and transverse sinus are the major dural sinuses that receive a considerable amount of venous drainage. The occlusion of them has been suggested to cause intracranial hypertension, hemorrhage, and lead to potentially fatal consequences. PATIENT CONCERNS: We reported a 35-year-old woman with headache presented to our emergency department with a decreased level of consciousness and epileptic seizures. The examination of speech, higher mental function, and cranial nerve were normal. Computed tomography (CT) demonstrated both subarachnoid and intraparenchymal hemorrhage and brain edema at the right temporal lobe accompanied by high density shadow in the right transverse sinus. Digital subtraction angiography (DSA) revealed extensive thrombosis of the SSS and bilateral transverse sinus. DIAGNOSES: The SSS and transverse sinus thrombosis, accompanied by right temporal lobe hemorrhage, subarachnoid hemorrhage (SAH). INTERVENTIONS: An emergent mechanical thrombectomy by placed Solitair AB stent in the SSS, respectively, passed left and right sigmoid sinus-transverse sinus route. We removed the most clots, DSA revealed recanalization of the SSS and left transverse sinus was seen with normalization of the venous outflow, the occlusion of right transverse sinus was still present. There were 4 hours after patient back to neurosurgical intensive care unit (NICU), patient appeared anisocoria (R/L:4.0/2.5 mm), bilateral light reflexes disappeared, then we took a CT reexamination revealed intraparenchymal hemorrhage increased, brain edema was aggravated at the left temporal lobe, and mild midline shift. Subsequently, we performed decompressive hemicraniectomy and puncture the hematoma supplemented by B ultrasonic. Anticoagulation treatment was initiated 24 hours after surgery, and follow-up DSA showed gradually improved patency in the SSS and bilateral transverse sinus. OUTCOMES: Despite occlusion of the SSS and bilateral transverse sinus, patient's symptoms resolved after the operations and he was discharged without complications. LESSONS: The favorable clinical outcome after complete occlusion of the SSS and transverse sinus, accompanied by right temporal lobe hemorrhage, SAH has rarely been reported and it might be explained by our timely surgical intervention and development of compensatory cerebral collateral circulation.
[Mh] MeSH terms primary: Fibrinolytic Agents/therapeutic use
Intracranial Hemorrhages/complications
Lateral Sinus Thrombosis/therapy
Sagittal Sinus Thrombosis/therapy
Thrombectomy/methods
[Mh] MeSH terms secundary: Adult
Combined Modality Therapy
Female
Humans
Lateral Sinus Thrombosis/etiology
Sagittal Sinus Thrombosis/etiology
Superior Sagittal Sinus/surgery
Transverse Sinuses/surgery
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:0 (Fibrinolytic Agents)
[Em] Entry month:1712
[Cu] Class update date: 171226
[Lr] Last revision date:171226
[Js] Journal subset:AIM; IM
[Da] Date of entry for processing:171217
[St] Status:MEDLINE
[do] DOI:10.1097/MD.0000000000009038

  9 / 814 MEDLINE  
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[PMID]: 29217208
[Au] Autor:López López L; Ramírez Bueno A; Kubarsepp A
[Ad] Address:Servicio de Neumología, Hospital Vithas Xanit Internacional, Málaga, España. Electronic address: lydia.lopezlopez@gmail.com.
[Ti] Title:Anisocoria como síntoma asociado a neumomediastino espontáneo. Anisocoria Associated With Spontaneous Pneumomediastinum.
[So] Source:Arch Bronconeumol;, 2017 Dec 04.
[Is] ISSN:1579-2129
[Cp] Country of publication:Spain
[La] Language:eng; spa
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171208
[Lr] Last revision date:171208
[St] Status:Publisher

  10 / 814 MEDLINE  
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[PMID]: 28457112
[Au] Autor:Knyazer B; Smolar J; Lazar I; Rosenberg E; Tsumi E; Lifshitz T; Levy J
[Ad] Address:Department of Ophthalmology, Soroka University Medical Center and Faculty of Health Sciences, Ben-Gurion University of the Negev, Beer Sheva, Israel.
[Ti] Title:Iatrogenic Horner Syndrome: Etiology, Diagnosis and Outcomes.
[So] Source:Isr Med Assoc J;19(1):34-38, 2017 Jan.
[Is] ISSN:1565-1088
[Cp] Country of publication:Israel
[La] Language:eng
[Ab] Abstract:BACKGROUND: The identification and prompt diagnosis of Horner syndrome (HS) is essential for preventing permanent damage. HS may arise when a lesion presents anywhere along the three-neuron oculosympathetic pathway that begins at the posterior-lateral nuclei of the hypothalamus all the way through to the orbit. We present four cases and review the literature to familiarize the reader with the identification, diagnosis and treatment of Horner syndrome. The four patients, three adults and one child, were followed for at least 6 months following the initial diagnosis (range 6-18 months). There was partial resolution in three of the four cases, while the fourth resolved completely. There are numerous causes of HS, some of them iatrogenic. While iatrogenic cases of HR are rare in both adults and children, HS is seen more often following surgical procedures. Prompt recognition of the syndrome and correction of the offending agent may prevent permanent damage to the neuronal pathway. It is therefore recommended that practitioners be aware of the risks for development of iatrogenic HS and the signs for early detection.
[Mh] MeSH terms primary: Horner Syndrome/diagnosis
Horner Syndrome/etiology
[Mh] MeSH terms secundary: Adult
Aged
Anisocoria/etiology
Blepharoptosis/etiology
Female
Humans
Iatrogenic Disease
Infant
Male
Young Adult
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE; REVIEW
[Em] Entry month:1711
[Cu] Class update date: 171128
[Lr] Last revision date:171128
[Js] Journal subset:IM
[Da] Date of entry for processing:170501
[St] Status:MEDLINE


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