Database : MEDLINE
Search on : bronchial and diseases [Words]
References found : 63728 [refine]
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[PMID]: 29515733
[Au] Autor:El Hachimi K; Benjelloun H; Zaghba N; Yassine N
[Ad] Address:Service des Maladies Respiratoires, CHU Ibn Rochd, Casablanca, Maroc.
[Ti] Title:Métastase pleurale et pulmonaire d'une polyadénofibromatose dégénéréé: à propos d'un cas. [Pleural and lung metastasis from degenerated multiple adenofibromas: about a case].
[So] Source:Pan Afr Med J;28:115, 2017.
[Is] ISSN:1937-8688
[Cp] Country of publication:Uganda
[La] Language:fre
[Ab] Abstract:Multiple adenofibromas or adenofibromatosis is characterized by the presence of at least 3 mono- or bilateral adenofibromas increasing significantly in size, causing trophic disorders. We report the case of a 46-year old female patient who had been followed up since she was 30 years old for bilateral adenofibromatosis that required 4 surgical procedures. Patient's medical data were collected in the Department of Respiratory Diseases at the Ibn Rochd University Hospital Center, Casablanca. Basing on preoperative assessment before bilateral mastectomy, the patient underwent chest X-ray that showed opaque right hemithorax exerting compression on the mediastinum. Clinical examination showed effusion syndrome in the right hemithorax and left supraclavicular cervical adenopathy. Pleural puncture biopsy confirmed the presence of poorly differentiated invasive carcinoma in the pleura, supporting breast origin. Bronchoscopy after pleural puncture objectified infiltration of the whole bronchial tree; biopsies confirmed the anatomopathological results. The recommended treatment strategy was based on multidrug chemotherapy. Patient's evolution was marked by the occurrence of hepatic metastases. This study shows that adenofibromas require regular monitoring given the risk of trasformation to breast cancer, which is a frequent cause of pleuropulmonary metastases.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180311
[Lr] Last revision date:180311
[St] Status:In-Process
[do] DOI:10.11604/pamj.2017.28.115.13599

  2 / 63728 MEDLINE  
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[PMID]: 29448087
[Au] Autor:Malinska D; Szymanski J; Patalas-Krawczyk P; Michalska B; Wojtala A; Prill M; Partyka M; Drabik K; Walczak J; Sewer A; Johne S; Luettich K; Peitsch MC; Hoeng J; Duszynski J; Szczepanowska J; van der Toorn M; Wieckowski MR
[Ad] Address:Nencki Institute of Experimental Biology, Polish Academy of Sciences, 3 Pasteur Street, 02-093 Warsaw, Poland.
[Ti] Title:Assessment of mitochondrial function following short- and long-term exposure of human bronchial epithelial cells to total particulate matter from a candidate modified-risk tobacco product and reference cigarettes.
[So] Source:Food Chem Toxicol;115:1-12, 2018 Feb 13.
[Is] ISSN:1873-6351
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Mitochondrial dysfunction caused by cigarette smoke is involved in the oxidative stress-induced pathology of airway diseases. Reducing the levels of harmful and potentially harmful constituents by heating rather than combusting tobacco may reduce mitochondrial changes that contribute to oxidative stress and cell damage. We evaluated mitochondrial function and oxidative stress in human bronchial epithelial cells (BEAS 2B) following 1- and 12-week exposures to total particulate matter (TPM) from the aerosol of a candidate modified-risk tobacco product, the Tobacco Heating System 2.2 (THS2.2), in comparison with TPM from the 3R4F reference cigarette. After 1-week exposure, 3R4F TPM had a strong inhibitory effect on mitochondrial basal and maximal oxygen consumption rates compared to TPM from THS2.2. Alterations in oxidative phosphorylation were accompanied by increased mitochondrial superoxide levels and increased levels of oxidatively damaged proteins in cells exposed to 7.5 µg/mL of 3R4F TPM or 150 µg/mL of THS2.2 TPM, while cytosolic levels of reactive oxygen species were not affected. In contrast, the 12-week exposure indicated adaptation of BEAS-2B cells to long-term stress. Together, the findings indicate that 3R4F TPM had a stronger effect on oxidative phosphorylation, gene expression and proteins involved in oxidative stress than TPM from the candidate modified-risk tobacco product THS2.2.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180310
[Lr] Last revision date:180310
[St] Status:Publisher

  3 / 63728 MEDLINE  
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[PMID]: 29524252
[Au] Autor:Jiang X; Shen C; Dai Y; Jiang F; Li S; Shen X; Hu Y; Li F
[Ad] Address:Developmental and Behavioral Pediatric & Child Primary Care Department, Ministry of Education-Shanghai Key Laboratory of Children's Environmental Health, Xinhua Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200092, China.
[Ti] Title:Early food allergy and respiratory allergy symptoms, and attention-deficit/hyperactivity disorder in Chinese children: a cross-sectional study.
[So] Source:Pediatr Allergy Immunol;, 2018 Mar 09.
[Is] ISSN:1399-3038
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: The relationship between food allergy and respiratory allergy, and attention-deficit/hyperactivity disorder (ADHD) in children is rarely investigated. Our objective is to determine whether early food allergy and respiratory allergy symptoms are associated with the prevalence of ADHD in Chinese school-age children. METHODS: This cross-sectional study was conducted in school-age children using cluster-stratified methods from nine cities across China between November and December 2005. A family and social environmental questionnaire including the diagnosis history of ADHD and allergic diseases (food allergy, allergic rhinitis and bronchial asthma), as well as general information was completed by parents. RESULTS: The prevalence of allergic rhinitis (20.4%) and asthma (11.6%) in the food allergy group were both significantly higher than in the non-food allergy group (9.0% and 2.8%, respectively) (both P <0.001). The multivariable analysis showed that single food allergy (OR=1.53, 95%CI: 1.13-2.05, P=0.005), food allergy complicated with allergic rhinitis or asthma (OR=3.36, 95%CI: 2.19-5.14, P<0.001), and food allergy complicated with allergic rhinitis and asthma simultaneously (OR=4.08, 95%CI: 2.05-8.11, P<0.001) were independently associated with the increased risk of ADHD. CONCLUSIONS: Early food allergy is associated with ADHD in school-age children. Early food allergy and respiratory allergy symptoms independently and synergistically contributed to higher risk of ADHD. Monitoring food allergy in early life could help the early prediction and intervention for the consequent allergy march and ADHD in children. This article is protected by copyright. All rights reserved.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180310
[Lr] Last revision date:180310
[St] Status:Publisher
[do] DOI:10.1111/pai.12888

  4 / 63728 MEDLINE  
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[PMID]: 29522998
[Au] Autor:Erbas B; Knudsen TM; Janson C; Nilsen RM; Accordini S; Benediktdottir B; Dratva J; Heinrich J; Jarvis D; Leynaert B; Matheson MC; Norbäck D; Real FG; Raherison-Semjen C; Villani S; Dharmage SC; Svanes C
[Ad] Address:Department of Public Health, La Trobe University, Melbourne, Australia. Electronic address: b.erbas@latrobe.edu.au.
[Ti] Title:Critical age windows in the impact of lifetime smoking exposure on respiratory symptoms and disease among ever smokers.
[So] Source:Environ Res;164:241-247, 2018 Mar 06.
[Is] ISSN:1096-0953
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:BACKGROUND: Despite extensive knowledge of smoking effects on respiratory disease, there is no study including all age windows of exposure among ever smokers. The objective of this study was to assess the effects from smoking exposure in utero, early childhood, adolescence and adulthood on respiratory health outcomes in adult male and female ever smokers. METHODS: Respiratory health outcomes were assessed in 10,610 participants of the European Community Respiratory Health Survey (ECRHS) I who reported a history of ever smoking by questionnaire. The associations of maternal smoking in utero, maternal smoking during childhood, age of smoking debut and pack-years of smoking with respiratory symptoms, obstructive diseases and bronchial hyperreactivity were analysed using generalized linear regression, non-linearity between age of smoking debut and outcomes were assessed by Generalized additive mixed models. RESULTS: Respiratory symptoms and asthma were more frequent in adults if their mother smoked during pregnancy, and, in men, also if mother smoked in childhood. Wheeze and ≥3 respiratory symptoms declined with later smoking debut among women [≤10 years: OR = 3.51, 95% CI 1.26, 9.73; 11-12 years: 1.57[1.01-2.44]; 13-15 years: 1.11[0.94-1.32] and ≤10 years: 3.74[1.56-8.83]; 11-12 years: 1.76[1.19-2.56]; 13-15 years: 1.12[0.94-1.35], respectively]. Effects of increasing number of packyears were pronounced in women (Chronic Obstructive Pulmonary Disease (COPD): OR/10 packyears women: 1.33 [1.18, 1.50], men: 1.14 [1.04, 1.26] p = 0.01). CONCLUSIONS: Among ever smokers, smoking exposure in each stage of the lifespan show persistent harmful effects for adult respiratory health, while women appeared to be more vulnerable to an early age of smoking debut and amount of smoking in adulthood.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[St] Status:Publisher

  5 / 63728 MEDLINE  
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[PMID]: 29522999
[Au] Autor:Pickering J; Teo TH; Thornton RB; Kirkham LA; Zosky GR; Clifford HD
[Ad] Address:Wesfarmers Centre of Vaccines and Infectious Diseases, Telethon Kids Institute, University of Western Australia, Perth, WA, Australia; School of Biomedical Sciences, University of Western Australia, Perth, WA, Australia. Electronic address: Janessa.pickering@uwa.edu.au.
[Ti] Title:Bacillus licheniformis in geogenic dust induces inflammation in respiratory epithelium.
[So] Source:Environ Res;164:248-254, 2018 Mar 06.
[Is] ISSN:1096-0953
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:Exposure to environmental geogenic (or earth-derived) dust can lead to more frequent and severe infections in the human airway. Particulate matter < 10 µm (PM ) is the component of air pollution that is commonly associated with the exacerbation of respiratory diseases. We have previously demonstrated that mice exposed to geogenic dust PM experienced an exacerbation of inflammatory responses to influenza A virus. Whether geogenic dust PM also exacerbates respiratory bacterial infection is not yet known, nor are the components of the dust that drive these responses. We treated airway bronchial epithelial cells (NuLi-1) with UV-irradiated geogenic dust PM from six remote Western Australian towns. High levels of IL-6 and IL-8 production were observed, as well as persistent microbial growth. 16 S rRNA sequencing of the growth identified the microbe as Bacillus licheniformis, a spore-forming, environmentally abundant bacterium. We next investigated the interaction of B. licheniformis with respiratory epithelium in vitro to determine whether this exacerbated infection with a bacterial respiratory pathogen (non-typeable Haemophilus influenzae, NTHi). Heat treatment (100 °C) of all PM samples eliminated B. licheniformis contamination and reduced epithelial inflammatory responses, suggesting that heat-labile and/or microbial factors were involved in the host response to geogenic dust PM . We then exposed NuLi-1 epithelium to increasing doses of the isolated Bacillus licheniformis (multiplicity of infection of 10:1, 1:1 or 0.1:1 bacteria: cells) for 1, 3, and 24 h. B. licheniformis and NTHi infection (association and invasion) was assessed using a standard gentamicin survival assay, and epithelial release of IL-6 and IL-8 was measured using a bead based immunoassay. B. licheniformis was cytotoxic to NuLi-1 cells at 24 h. At 3 h post-challenge, B. licheniformis elicited high IL-6 and IL-8 inflammatory responses from NuLi-1 cells compared with cells treated with heat-treated geogenic dust PM (p < 0.0001). Whilst treatment of cells with B. licheniformis increased inflammation, this did not make the cells more susceptible to NTHi infection. This study highlights that geogenic dust PM can harbour viable bacterial spores that induce inflammation in respiratory epithelium. The impact on respiratory health from inhalation of bacterial spores in PM in arid environments may be underestimated. Further investigation into the contribution of B. licheniformis and the wider dust microbiome to respiratory infection is warranted.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[St] Status:Publisher

  6 / 63728 MEDLINE  
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[PMID]: 29446293
[Au] Autor:Beygel EA; Katamanova EV; Shayakhmetov SF; Ushakova OV; Pavlenko NA; Kuks AN; Voronin DA
[Ti] Title:[The impact of the long-term exposure of industrial aerosols on clinical and functional indices of the broncho-pulmonary system in aluminum smelter workers].
[So] Source:Gig Sanit;95(12):1160-3, 2016.
[Is] ISSN:0016-9900
[Cp] Country of publication:Russia (Federation)
[La] Language:rus
[Ab] Abstract:It was found that the appearance of respiratory diseases among workers was established to be associated with the impact of aerosols with the complex chemical composition, exceeding corresponding MAC. Among respiratory diseases in workers there were detected predominantly chronic non-obstructive bronchitis - 59.15%, and chronic obstructive pulmonary disease (COPD) - in 35.2% of cases. In the analysis of spirometric indices of examined patients the vast majority (76.3%) of them showed disturbances of the bronchial patency of mild to moderate degree of the pronouncement. As a result of the implementation offlexible bronchoscopy there was established the presence of a diffuse lesion of bronchi with a predominance of subatrophic and atrophic processes in the mucosa. It was found that under the increase of the concentration of HF in the air of industrial premises the probability of bronchial obstruction and deterioration of the tracheobronchial patency in aluminum smelter workers was established to elevate.
[Mh] MeSH terms primary: Air Pollutants, Occupational
Aluminum
Bronchitis, Chronic
Chemical Industry
Occupational Diseases
Pulmonary Disease, Chronic Obstructive
[Mh] MeSH terms secundary: Aerosols/adverse effects
Aerosols/analysis
Aerosols/chemistry
Air Pollutants, Occupational/analysis
Air Pollutants, Occupational/toxicity
Aluminum/analysis
Aluminum/chemistry
Bronchitis, Chronic/epidemiology
Bronchitis, Chronic/etiology
Bronchitis, Chronic/prevention & control
Chemical Industry/methods
Chemical Industry/standards
Humans
Male
Middle Aged
Occupational Diseases/epidemiology
Occupational Diseases/etiology
Occupational Diseases/prevention & control
Occupational Exposure/adverse effects
Occupational Exposure/analysis
Occupational Exposure/prevention & control
Pulmonary Disease, Chronic Obstructive/epidemiology
Pulmonary Disease, Chronic Obstructive/etiology
Pulmonary Disease, Chronic Obstructive/prevention & control
Siberia/epidemiology
Time
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Aerosols); 0 (Air Pollutants, Occupational); CPD4NFA903 (Aluminum)
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[Js] Journal subset:IM
[Da] Date of entry for processing:180216
[St] Status:MEDLINE

  7 / 63728 MEDLINE  
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[PMID]: 29446284
[Au] Autor:Kudaeva IV; Dyakovich OA; Beygel EA; Masnavieva LB; Naumova OV; Budarina LA
[Ti] Title:[Clinical, biochemical and allergological indices characterizing occupational diseases of the bronchial and pulmonary system in employees at aluminium production].
[So] Source:Gig Sanit;95(12):1142-5, 2016.
[Is] ISSN:0016-9900
[Cp] Country of publication:Russia (Federation)
[La] Language:rus
[Ab] Abstract:There are many harmful factors that possess a damaging impact on the body of employees at aluminum production. It leads to the development of bronchial asthma (BA), chronic nonobstructive bronchitis (CNB) and chronic obstructive pulmonary disease (COPD). The pathogenesis of these disorders, as well as sensitizing effect offluorine in the aluminum production is not fully understood. The purpose of this work was to study the characteristics of laboratory indices in patients with occupational diseases of the respiratory system. In workers of aluminum production with the diagnosis of occupational diseases of respiratory system (15 patients with a diagnosis of asthma, 30 CNB cases, 20 COPD patients) we evaluated the content of total protein, total cholesterol, high density lipoprotein cholesterol (HDLC), total calcium, phosphorus, ceruloplasmin, hematological indices and performed emigration of leukocytes braking test (TTEEL). Clinical and biochemical profile ofpersons with occupational asthma was characterized by a low level of total calcium and ceruloplasmin, a high concentration of phosphorus in the blood serum and inhibition of leukocyte emigration in the test with sodium fluoride. For aluminum production CNB workers characteristic active proatherogenic process was pronounced by a decrease in the HDLC level and an increase in atherogenic index; higher hematocrit value and concentration of erythrocytes, and more than 50% of cases of sensitization to the presence of sodium fluoride. COPD cases had occupational lower average concentration of hemoglobin in the erythrocyte, total protein in serum, as well as polymorphic variant response to sodium fluoride in the form of a depression and activation of leucocytes emigration.
[Mh] MeSH terms primary: Air Pollutants, Occupational
Aluminum
Asthma, Occupational
Chemical Industry
Pulmonary Disease, Chronic Obstructive
[Mh] MeSH terms secundary: Air Pollutants, Occupational/analysis
Air Pollutants, Occupational/toxicity
Aluminum/analysis
Aluminum/toxicity
Asthma, Occupational/blood
Asthma, Occupational/diagnosis
Asthma, Occupational/epidemiology
Biomarkers/analysis
Chemical Industry/methods
Chemical Industry/standards
Humans
Male
Middle Aged
Occupational Exposure/adverse effects
Occupational Exposure/analysis
Occupational Exposure/prevention & control
Occupational Health/statistics & numerical data
Pulmonary Disease, Chronic Obstructive/blood
Pulmonary Disease, Chronic Obstructive/diagnosis
Pulmonary Disease, Chronic Obstructive/epidemiology
Pulmonary Disease, Chronic Obstructive/etiology
Respiratory Hypersensitivity/blood
Respiratory Hypersensitivity/chemically induced
Respiratory Hypersensitivity/diagnosis
Siberia/epidemiology
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Air Pollutants, Occupational); 0 (Biomarkers); CPD4NFA903 (Aluminum)
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[Js] Journal subset:IM
[Da] Date of entry for processing:180216
[St] Status:MEDLINE

  8 / 63728 MEDLINE  
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[PMID]: 29272749
[Au] Autor:Liessi N; Cichero E; Pesce E; Arkel M; Salis A; Tomati V; Paccagnella M; Damonte G; Tasso B; Galietta LJV; Pedemonte N; Fossa P; Millo E
[Ad] Address:Department of Experimental Medicine, Section of Biochemistry, University of Genoa, Viale Benedetto XV 1, 16132 Genoa, Italy; Center of Excellence for Biomedical Research (CEBR), University of Genoa, Viale Benedetto XV 9, 16132 Genoa, Italy.
[Ti] Title:Synthesis and biological evaluation of novel thiazole- VX-809 hybrid derivatives as F508del correctors by QSAR-based filtering tools.
[So] Source:Eur J Med Chem;144:179-200, 2018 Jan 20.
[Is] ISSN:1768-3254
[Cp] Country of publication:France
[La] Language:eng
[Ab] Abstract:The most common CF mutation, F508del, impairs the processing and gating of CFTR protein. This deletion results in the improper folding of the protein and its degradation before it reaches the plasma membrane of epithelial cells. Present correctors, like VX809 only induce a partial rescue of the mutant protein. Our previous studies reported a class of compounds, called aminoarylthiazoles (AATs), featuring an interesting activity as correctors. Some of them show additive effect with VX809 indicating a different mechanism of action. In an attempt to construct more interesting molecules, it was thought to generate chemically hybrid compounds, blending a portion of VX809 merged to the thiazole scaffold. This approach was guided by the development of QSAR analyses, which were performed based on the F508del correctors so far disclosed in the literature. This strategy was aimed at exploring the key requirements turning in the corrector ability of the collected derivatives and allowed us to derive a predictive model guiding for the synthesis of novel hybrids as promising correctors. The new molecules were tested in functional and biochemical assays on bronchial CFBE41o-cells expressing F508del-CFTR showing a promising corrector activity.
[Mh] MeSH terms primary: Aminopyridines/chemistry
Aminopyridines/pharmacology
Benzodioxoles/chemistry
Benzodioxoles/pharmacology
Cystic Fibrosis Transmembrane Conductance Regulator/genetics
Mutation/drug effects
Thiazoles/chemistry
Thiazoles/pharmacology
[Mh] MeSH terms secundary: Aminopyridines/chemical synthesis
Benzodioxoles/chemical synthesis
Cell Line
Cystic Fibrosis/drug therapy
Cystic Fibrosis/genetics
Humans
Quantitative Structure-Activity Relationship
Thiazoles/chemical synthesis
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Aminopyridines); 0 (Benzodioxoles); 0 (Thiazoles); 126880-72-6 (Cystic Fibrosis Transmembrane Conductance Regulator); EGP8L81APK (lumacaftor)
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[Js] Journal subset:IM
[Da] Date of entry for processing:171223
[St] Status:MEDLINE

  9 / 63728 MEDLINE  
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[PMID]: 29521052
[Au] Autor:Krzych-Falta E; Furmanczyk K; Tomaszewska A; Olejniczak D; Samolinski B; Samolinska-Zawisza U
[Ad] Address:Department of the Prevention of Environmental Hazards and Allergology, Faculty of Health Sciences, Medical University of Warsaw, Poland.
[Ti] Title:Probiotics: Myths or facts about their role in allergy prevention.
[So] Source:Adv Clin Exp Med;27(1):119-124, 2018 Jan.
[Is] ISSN:1899-5276
[Cp] Country of publication:Poland
[La] Language:eng
[Ab] Abstract:BACKGROUND: The hygiene hypothesis proposed by Strachan in the 1980s clearly emphasized the role of microorganisms in atopy prevention. OBJECTIVES: The study objective was to assess the preventive role of probiotics in patients with allergic rhinitis, bronchial asthma, atopic dermatitis, and/or food allergy. MATERIAL AND METHODS: The methods used in the study were the International Study of Asthma and Allergies in Childhood (ISAAC) questionnaires for 6-7- and 13-14-year-olds and the European Community Respiratory Health Survey II (ECRHS II) questionnaire targeted for the 20-44 age group. The study was conducted as part of the cross-sectional Epidemiology of Allergic Diseases in Poland study conducted in 9 Polish regions (8 urban: Warszawa, Lublin, Bialystok, Gdansk, Poznan, Wroclaw, Katowice, Kraków, and the rural regions of Zamojski and Krasnostawski counties). The study material was a group of patients diagnosed with food allergy (n = 407), atopic dermatitis (n = 311), allergic rhinitis (n = 1.353), bronchial asthma (n = 505), and healthy volunteers (n = 2,403). RESULTS: Genetic factors play an important role in the allergy development. A family history positive for chronic skin disorders increased the risk of atopic dermatitis and food allergies (OR = 1.456, CI = 1.14-1.85, p = 0.002; and OR = 1.378, CI = 1.05-1.81, p = 0.02, respectively). The consumption of products containing live bacterial cultures showed no preventive effects in any of the evaluated disorders in early childhood. Conversely, over the age of 14 years, probiotic formulations exhibit health-promoting effects and may lower the risk of allergic diseases. CONCLUSIONS: The use of probiotics in the Polish population showed no protective effect in the first years of life. The changes in dietary habits introduced during late adolescence demonstrated significantly greater preventive effects of live bacterial cultures against the development of allergic diseases.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180309
[Lr] Last revision date:180309
[St] Status:In-Data-Review
[do] DOI:10.17219/acem/65476

  10 / 63728 MEDLINE  
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[PMID]: 29384600
[Au] Autor:Kondov S; Siepe M; Beyersdorf F; von Samson-Himmelstjern P; Czerny M
[Ad] Address:Department of Cardiovascular Surgery, University Heart Center Freiburg, Faculty of Medicine, University of Freiburg, Germany.
[Ti] Title:Thoracoabdominal aortic replacement with a bovine pericardial tube graft for aortobronchial fistulation 10 years after TEVAR.
[So] Source:Multimed Man Cardiothorac Surg;2017, 2017 Dec 20.
[Is] ISSN:1813-9175
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Aortobronchial fistula after thoracic endovascular aortic repair is usually a late complication. It is associated with high mortality and its surgical management is technically challenging. This tutorial illustrates the steps involved in removing an infected stent graft and replacing it with a bovine pericardial tube graft.
[Mh] MeSH terms primary: Aortic Aneurysm, Thoracic/surgery
Aortic Diseases/surgery
Blood Vessel Prosthesis Implantation/adverse effects
Bronchial Fistula/surgery
Prosthesis-Related Infections/surgery
Vascular Fistula/surgery
[Mh] MeSH terms secundary: Animals
Aortic Diseases/etiology
Bioprosthesis
Blood Vessel Prosthesis/adverse effects
Bronchial Fistula/etiology
Cattle
Device Removal
Endovascular Procedures/adverse effects
Humans
Pericardium/transplantation
Prosthesis-Related Infections/etiology
Reoperation
Stents/adverse effects
Vascular Fistula/etiology
[Pt] Publication type:VIDEO-AUDIO MEDIA
[Em] Entry month:1803
[Cu] Class update date: 180308
[Lr] Last revision date:180308
[Js] Journal subset:IM
[Da] Date of entry for processing:180201
[St] Status:MEDLINE
[do] DOI:10.1510/mmcts.2017.027


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