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Search on : pemphigoid and gestationis [Words]
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[PMID]: 29486593
[Au] Autor:Warshafsky C; Tron VA; Robertson D; Kives S
[Ad] Address:1 Department of Obstetrics and Gynecology, University of Toronto, Toronto, ON, Canada.
[Ti] Title:Pemphigoid Gestationis: A Case Presentation.
[So] Source:J Cutan Med Surg;:1203475418760459, 2018 Feb 01.
[Is] ISSN:1615-7109
[Cp] Country of publication:United States
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1803
[Cu] Class update date: 180228
[Lr] Last revision date:180228
[St] Status:Publisher
[do] DOI:10.1177/1203475418760459

  2 / 601 MEDLINE  
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[PMID]: 29473148
[Au] Autor:Saffari H; Zone JJ; Allen M; Leiferman KM
[Ad] Address:Immunodermatology Laboratory, Department of Dermatology, School of Medicine, University of Utah, Salt Lake City, UT, USA.
[Ti] Title:A subset of patients with pemphigoid (herpes) gestationis has serological evidence of celiac disease.
[So] Source:Int J Dermatol;, 2018 Feb 23.
[Is] ISSN:1365-4632
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:BACKGROUND: Pemphigoid (herpes) gestationis (PG) is an uncommon, self-limited disease with other autoimmune associations; however, celiac disease (CD) is not recognized as one. METHODS: From 71 patients' sera submitted for herpes gestationis factor (HGF) testing over a 5-year period, 12 were consistent with PG demonstrating HGF and increased IgG BP180 antibody levels; these sera were tested for IgA and IgG endomysial antibodies (EMA), epithelial basement membrane zone and cell surface antibodies by indirect immunofluorescence, and for IgA and IgG tissue transglutaminase (transglutaminase 2 or TG2) antibodies, IgA epidermal transglutaminase (transglutaminase 3 or TG3) antibodies, IgG BP230, and IgG desmoglein 1 and desmoglein 3 antibodies by enzyme-linked immunosorbent assays (ELISAs). RESULTS: Three of 12 patients' sera with PG (25%) had CD antibodies with positive IgA EMA and increased IgA TG2 antibody levels; two of these had positive IgG EMA, and one other had an increased IgA TG3 antibody level. CONCLUSIONS: A subset of patients with serological findings of PG also has serological evidence of CD, which may have implications in the etiopathogenesis of PG and which reveals important information about the mother's, and possibly her infant's, health.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180223
[Lr] Last revision date:180223
[St] Status:Publisher
[do] DOI:10.1111/ijd.13925

  3 / 601 MEDLINE  
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[PMID]: 29392620
[Au] Autor:Kushner CJ; Concha JSS; Werth VP
[Ad] Address:Corporal Michael J. Crescenz VAMC, Philadelphia, PA, USA.
[Ti] Title:Treatment of Autoimmune Bullous Disorders in Pregnancy.
[So] Source:Am J Clin Dermatol;, 2018 Feb 02.
[Is] ISSN:1179-1888
[Cp] Country of publication:New Zealand
[La] Language:eng
[Ab] Abstract:Autoimmune bullous diseases (AIBD), including pemphigus, bullous pemphigoid, epidermolysis bullosa acquisita, mucous membrane pemphigoid, and pemphigoid gestationis, pose significant therapeutic challenges, especially in pregnant and post-partum breastfeeding patients or those planning to conceive. Data on the safety and efficacy of therapeutic interventions during the perinatal period are lacking because randomized controlled trials are typically not performed in this setting. However, many of the treatments for AIBD are also used in other diseases, so data can be extrapolated from studies or case reports in these other patient populations. It appears that many of the treatments for AIBD can adversely affect the fetus or neonate, and alterations in immune status caused by pregnancy-associated hormonal changes can negatively impact disease control. This article summarizes and weighs the risks and benefits of the various agents used to treat AIBD during pregnancy. We also present the available information on lactation as well as effects on male fertility.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1802
[Cu] Class update date: 180202
[Lr] Last revision date:180202
[St] Status:Publisher
[do] DOI:10.1007/s40257-018-0342-0

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[PMID]: 29252171
[Au] Autor:Patsatsi A; Lamprou F; Kokolios M; Stylianidou D; Trigoni A; Kalampalikis D; Sotiriadis D
[Ti] Title:Spectrum of Autoimmune Bullous Diseases in Northern Greece. A 4-year Retrospective Study and Review of the Literature.
[So] Source:Acta Dermatovenerol Croat;25(3):195-201, 2017 Oct.
[Is] ISSN:1847-6538
[Cp] Country of publication:Croatia
[La] Language:eng
[Ab] Abstract:Bullous Diseases Unit at the 2nd Department of Dermatology and Venereology, Aristotle University of Thessaloniki was founded with the aim to provide the optimal diagnostic approach and treatment of patients with autoimmune bullous diseases (AΙBD). We processed all AIBD files of patients diagnosed from 2011 to 2014 in order to record all epidemiological data and therapeutic manipulations during monitoring. 57 patients were diagnosed with intraepidermal and 62 with subepidermal bullous diseases. There were 51 cases (89%) of pemphigus vulgaris (PV) and 6 (11%) of pemphigus foliaceus (PF), whereas 45 (73%) patients were diagnosed with bullous pemphigoid (BP), 9 (14%) with mucous membrane pemphigoid (MMP), 3 (5%) with pemphigoid gestationis (PG), 3 (5%) with linear IgA dermatosis (LAD), 1 (2%) with epidermolysis bullosa aquisita (EBA), and 1 patient with an undefined subepidermal AIBD. The mean age of patients within the pemphigus spectrum was 57 years. In the pemphigoid spectrum, the mean age was 72 years. Comorbidities were reported with increasing frequency, as well as treatment options other than systemic corticosteroids, such as adjuvant immunosuppressive agents, which were used to achieve complete remission. This is a report from a tertiary AIBD Referral Center in northern Greece. Our data from a 4-year period contribute to the completion of the global geographic incidence map of AIBD.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171218
[Lr] Last revision date:171218
[St] Status:In-Process

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[PMID]: 29205468
[Au] Autor:Nishida E; Nishio E; Murashima H; Ishii N; Hashimoto T; Morita A
[Ad] Address:Department of Geriatric and Environmental Dermatology, Nagoya City University Graduate School of Medical Sciences, Nagoya, Japan.
[Ti] Title:Case of epidermolysis bullosa acquisita with concomitant anti-laminin-332 antibodies.
[So] Source:J Dermatol;, 2017 Dec 04.
[Is] ISSN:1346-8138
[Cp] Country of publication:England
[La] Language:eng
[Ab] Abstract:Subepidermal autoimmune blistering disease including bullous pemphigoid, pemphigoid gestationis, mucous membrane pemphigoid, anti-laminin-γ1 pemphigoid, linear immunoglobulin A bullous disease and epidermolysis bullosa acquisita (EBA), are all characterized by direct immunofluorescence microscopy or immunoglobulin deposition on the basement membrane zone. Among them, EBA is a rare acquired subepidermal autoimmune blistering disease of the skin and mucous membranes reactive with type VII collagen, a major component of the epidermal basement membrane zone. Anti-laminin-332-type mucous membrane pemphigoid has pathogenic autoantibodies against laminin-332, which is a basement membrane heterotrimeric protein composed of α3, ß3 and γ2 laminin chains. We describe a 73-year-old Japanese man presenting with multiple, annular, tense blisters on the lower legs and oral lesions. Despite the severe clinical manifestations, the disease was successfully controlled by combination therapy of oral prednisolone and mizoribine. This case was confirmed to have autoantibodies to both type VII collagen and laminin-332 α3 chain by indirect immunofluorescence of 1 mol NaCl-split normal human skin, various immunoblot analyses and enzyme-linked immunosorbent assays. This case was a rare case of EBA with concomitant anti-laminin-332 antibodies.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1712
[Cu] Class update date: 171205
[Lr] Last revision date:171205
[St] Status:Publisher
[do] DOI:10.1111/1346-8138.14169

  6 / 601 MEDLINE  
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[PMID]: 29159697
[Au] Autor:Kridin K
[Ad] Address:Department of Dermatology, Rambam Health Care Campus, POB 9602, 31096, Haifa, Israel. dr_kridin@hotmail.com.
[Ti] Title:Subepidermal autoimmune bullous diseases: overview, epidemiology, and associations.
[So] Source:Immunol Res;, 2017 Nov 21.
[Is] ISSN:1559-0755
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Subepidermal autoimmune bullous diseases of the skin and mucosae comprise a large group of chronic diseases, including bullous pemphigoid, pemphigoid gestationis, mucous membrane pemphigoid, linear IgA bullous dermatosis, epidermolysis bullosa acquisita, and anti-p200 pemphigoid. These diseases are characterized by an antibody response toward structural components of the basement membrane zone, resulting in subepidermal blistering. The epidemiological features of these diseases vary substantially in different regions of the world. Observational studies investigating comorbidities and associations among patients with these diseases are inconsistent and sometimes inconclusive. This review provides a brief overview regarding each one of the subepidermal autoimmune bullous diseases. In addition, it summarizes the most recent understanding of the epidemiological features and associations of this group of organ-specific autoimmune diseases.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1711
[Cu] Class update date: 171121
[Lr] Last revision date:171121
[St] Status:Publisher
[do] DOI:10.1007/s12026-017-8975-2

  7 / 601 MEDLINE  
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[PMID]: 28816740
[Au] Autor:Valencia-Guerrero A; Deng A; Dresser K; Bouliane G; Cornejo KM
[Ad] Address:Department of Pathology, University of Massachusetts Medical School, UMass Memorial Medical Center, Worcester, MA.
[Ti] Title:The Value of Direct Immunofluorescence on Proteinase-Digested Formalin-Fixed Paraffin-Embedded Skin Biopsies.
[So] Source:Am J Dermatopathol;, 2017 Aug 09.
[Is] ISSN:1533-0311
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Direct immunofluorescence (DIF) on frozen tissue (DIF-F) is the method of choice for the identification of immune deposits present in skin and other tissues. DIF can also be performed on formalin-fixed paraffin-embedded tissue (DIF-P) after antigen retrieval with proteases and has proven to be of value in renal pathology. However, its utility in skin biopsies has not been fully examined. In this study, we performed DIF-P on 60 skin biopsies that comprised of bullous pemphigoid (n = 18), pemphigoid gestationis (n = 1), pemphigus (n = 7), linear IgA disease (n = 7), vasculitis (n = 20), lupus erythematosus (n = 3), and dermatitis herpetiformis (n = 4) cases. We compared the results of DIF-P with those of DIF-F from the same patients. The diagnostic features were found in 15 of 19 (79%) pemphigoid (bullous pemphigoid and pemphigoid gestationis), 3 of 7 (43%) pemphigus, 3 of 7 (43%) linear IgA disease, 14 of 20 (70%) vasculitis, 1 of 3 (33%) lupus erythematosus, and none (0%) of the dermatitis herpetiformis cases tested. Overall, DIF-P is less sensitive than DIF-F but seems to be a valuable technique that could aid in the diagnosis of vasculitides, immunobullous, and connective tissue disorders when fresh tissue is unavailable.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1708
[Cu] Class update date: 170817
[Lr] Last revision date:170817
[St] Status:Publisher
[do] DOI:10.1097/DAD.0000000000000934

  8 / 601 MEDLINE  
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[PMID]: 28779299
[Au] Autor:Amber KT; Murrell DF; Schmidt E; Joly P; Borradori L
[Ad] Address:Department of Dermatology, University of California Irvine Health, 118 Med Surg 1, Irvine, CA, 92697, USA. KAmber@UCI.edu.
[Ti] Title:Autoimmune Subepidermal Bullous Diseases of the Skin and Mucosae: Clinical Features, Diagnosis, and Management.
[So] Source:Clin Rev Allergy Immunol;, 2017 Aug 04.
[Is] ISSN:1559-0267
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Autoimmune subepidermal blistering diseases of the skin and mucosae constitute a large group of sometimes devastating diseases, encompassing bullous pemphigoid, gestational pemphigoid, mucous membrane pemphigoid, epidermolysis bullosa acquisita, and anti-p200 pemphigoid. Their clinical presentation is polymorphic. These autoimmune blistering diseases are associated with autoantibodies that target distinct components of the basement membrane zone of stratified epithelia. These autoantigens represent structural proteins important for maintenance of dermo-epidermal integrity. Bullous pemphigoid (BP) is the most common subepidermal autoimmune blistering disease of the skin and mucosae. Although the disease typically presents with a generalized blistering eruption associated with itch, atypical variants with either localized bullous lesions or "non-bullous" presentations are observed in approximately 20% of patients. A peculiar form of BP typically associated with pregnancy is pemphigoid gestationis. In anti-p200 pemphigoid, patients present with tense blisters on erythematosus or normal skin resembling BP, with a predilection for acral surfaces. These patients have antibodies targeting the 200-kDa basement membrane protein. Epidermolysis bullosa is a rare autoimmune blistering disease associated with autoantibodies against type VII collagen that can have several phenotypes including a classical form mimicking dystrophic epidermolysis bullosa, an inflammatory presentation mimicking BP, or mucous membrane pemphigoid-like lesions. Mucous membrane pemphigoid (MMP) is the term agreed upon by international consensus for an autoimmune blistering disorder, which affects one or more mucous membrane and may involve the skin. The condition involves a number of different autoantigens in the basement membrane zone. It may result in severe complications from scarring, such as blindness and strictures. Diagnosis of these diseases relies on direct immunofluorescence microscopy studies and immunoserological assays. Management of affected patients is often challenging. We will here review the clinical and immunopathological features as well as the pathophysiology of this group of organ-specific autoimmune diseases. Finally, we will discuss the diagnostic approach and the principles of management in clinical practice.
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1708
[Cu] Class update date: 170805
[Lr] Last revision date:170805
[St] Status:Publisher
[do] DOI:10.1007/s12016-017-8633-4

  9 / 601 MEDLINE  
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[PMID]: 28560301
[Au] Autor:Hallaji Z; Mortazavi H; Ashtari S; Nikoo A; Abdollahi M; Nasimi M
[Ad] Address:Department of Dermatology, Razi Hospital, Tehran University of Medical Sciences, Tehran, Iran.
[Ti] Title:Pemphigoid gestationis: Clinical and histologic features of twenty-three patients.
[So] Source:Int J Womens Dermatol;3(2):86-90, 2017 Jun.
[Is] ISSN:2352-6475
[Cp] Country of publication:Netherlands
[La] Language:eng
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1706
[Cu] Class update date: 170816
[Lr] Last revision date:170816
[St] Status:PubMed-not-MEDLINE
[do] DOI:10.1016/j.ijwd.2016.11.004

  10 / 601 MEDLINE  
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[PMID]: 28524368
[Au] Autor:Oumerzouk J; Abida N; Zaimi A; Znati K; Zbir EM; Bourazza A
[Ad] Address:Department of Neurology, Mohammed V. Rabat Military Hospital, Rabat, Morocco.
[Ti] Title:Severe pemphigoid gestationis associated with acute disseminated encephalomyelitis in the setting of a systemic disorder.
[So] Source:Australas J Dermatol;, 2017 May 19.
[Is] ISSN:1440-0960
[Cp] Country of publication:Australia
[La] Language:eng
[Ab] Abstract:Pemphigoid gestationis is a skin-specific autoimmune disorder that can sometimes present as the cutaneous manifestation of a multiorgan disease due to potentially common pathogenic mechanisms. We report a severe form of pemphigoid gestationis in a 32-year-old primigravida woman, who presented at 22 weeks of gestation with headaches and blurred vision, later developing encephalitis, intrauterine fetal demise and dilated cardiomyopathy.
[Pt] Publication type:CASE REPORTS
[Em] Entry month:1705
[Cu] Class update date: 170525
[Lr] Last revision date:170525
[St] Status:Publisher
[do] DOI:10.1111/ajd.12656


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