Database : MEDLINE
Search on : polyradiculoneuropathy [Words]
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[PMID]: 28449973
[Au] Autor:Sandhu SK; Hua W; MaCurdy TE; Franks RL; Avagyan A; Kelman J; Worrall CM; Ball R; Nguyen M
[Ad] Address:Center for Biologics Evaluation and Research, Food and Drug Administration, Silver Spring, MD, USA. Electronic address: sukhminder.sandhu@fda.hhs.gov.
[Ti] Title:Near real-time surveillance for Guillain-Barré syndrome after influenza vaccination among the Medicare population, 2010/11 to 2013/14.
[So] Source:Vaccine;35(22):2986-2992, 2017 05 19.
[Is] ISSN:1873-2518
[Cp] Country of publication:Netherlands
[La] Language:eng
[Ab] Abstract:BACKGROUND: Guillain-Barré syndrome (GBS) is a serious acute demyelinating disease that causes weakness and paralysis. The Food and Drug Administration (FDA) began collaborating with the Centers for Medicare and Medicaid Services (CMS) to develop near real-time vaccine safety surveillance capabilities in 2006 and has been monitoring for the risk of GBS after influenza vaccination for every influenza season since 2008. METHODS: We present results from the 2010/11 to 2013/14 influenza seasons using the Updating Sequential Probability Ratio Test (USPRT), with an overall 1-sided α of 0.05 apportioned equally using a constant alpha-spending plan among 20 consecutive weekly tests, 5 ad hoc tests, and a 26th final end of season test. Observed signals were investigated using the self-controlled risk interval (SCRI) design. RESULTS: Over 15 million people were vaccinated in each influenza season. In the 2010/11 influenza season, we observed an elevated GBS risk during the season, with an end of season SCRI analysis finding a nonsignificant increased risk (RR=1.25, 95% CI: 0.96-1.63). A sensitivity analysis applying the positive predictive value of the ICD-9 code for GBS from the 2009/10 season estimated a RR=1.98 (95% CI: 1.42-2.76). Although the 2010/11 influenza vaccine suggested an increased GBS risk, surveillance of the identical vaccine in the 2011/12 influenza season did not find an increased GBS risk after vaccination. No signal was observed in the subsequent three influenza seasons. CONCLUSIONS: Conducting near real-time surveillance using USPRT has proven to be an excellent method for near real-time GBS surveillance after influenza vaccination, as demonstrated by our surveillance efforts during the 2010/11-2013/14 influenza seasons. In the 2010/2011 influenza season, in addition to the 2009 H1N1 influenza pandemic, using near real-time surveillance we were able to observe a signal early in the influenza season and the method has now become routine.
[Mh] MeSH terms primary: Guillain-Barre Syndrome/epidemiology
Influenza Vaccines/adverse effects
Medicare
Population Surveillance/methods
[Mh] MeSH terms secundary: Aged
Aged, 80 and over
Centers for Medicare and Medicaid Services (U.S.)
Computer Systems
Female
Guillain-Barre Syndrome/etiology
Humans
Influenza Vaccines/administration & dosage
Male
Risk Assessment
United States/epidemiology
United States Food and Drug Administration
Vaccination
[Pt] Publication type:JOURNAL ARTICLE
[Nm] Name of substance:0 (Influenza Vaccines)
[Em] Entry month:1802
[Cu] Class update date: 180308
[Lr] Last revision date:180308
[Js] Journal subset:IM
[Da] Date of entry for processing:170429
[St] Status:MEDLINE

  2 / 8502 MEDLINE  
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[PMID]: 29157616
[Au] Autor:Mauermann ML
[Ad] Address:Department of Neurology, Mayo Clinic, 200 First Street Southwest, Rochester, MN 55905. Electronic address: Mauermann.Michelle@mayo.edu.
[Ti] Title:The Peripheral Neuropathies of POEMS Syndrome and Castleman Disease.
[So] Source:Hematol Oncol Clin North Am;32(1):153-163, 2018 02.
[Is] ISSN:1558-1977
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Polyneuropathy, organomegaly, endocrinopathy, monoclonal plasma cell disorder, skin changes (POEMS) syndrome is a rare paraneoplastic disorder. The polyneuropathy can be the presenting symptom and is typically a painful, motor-predominant polyradiculoneuropathy often mimicking chronic inflammatory demyelinating polyradiculoneuropathy. The presence of a lambda monoclonal protein, elevated vascular endothelial growth factor, systemic features, and treatment resistance are clues to the diagnosis. Castleman disease (CD) is seen in a subset of these patients, and when present the neuropathy is similar but less severe. In contrast, in those patients with purely CD, the neuropathy is often a mild, painless distal sensory neuropathy.
[Mh] MeSH terms primary: Castleman Disease
POEMS Syndrome
[Mh] MeSH terms secundary: Castleman Disease/diagnosis
Castleman Disease/pathology
Castleman Disease/physiopathology
Castleman Disease/therapy
Humans
POEMS Syndrome/diagnosis
POEMS Syndrome/pathology
POEMS Syndrome/physiopathology
POEMS Syndrome/therapy
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Em] Entry month:1801
[Cu] Class update date: 180307
[Lr] Last revision date:180307
[Js] Journal subset:IM
[Da] Date of entry for processing:171122
[St] Status:MEDLINE

  3 / 8502 MEDLINE  
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[PMID]: 29157614
[Au] Autor:Dispenzieri A; Kourelis T; Buadi F
[Ad] Address:Division of Hematology, Department of Medicine, Medicine, Mayo Clinic, 200 First Street Southwest, Rochester, MN 55905, USA. Electronic address: Dispenzieri.angela@mayo.edu.
[Ti] Title:POEMS Syndrome: Diagnosis and Investigative Work-up.
[So] Source:Hematol Oncol Clin North Am;32(1):119-139, 2018 02.
[Is] ISSN:1558-1977
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:POEMS syndrome is a rare paraneoplastic syndrome secondary to a plasma cell dyscrasia. Recognition of a combination of peripheral neuropathy, organomegaly, endocrinopathy, monoclonal plasmaproliferative disorder, skin changes, papilledema, extravascular volume overload, sclerotic bone lesions, thrombocytosis, and Castleman disease is the first step in managing the disease. Increased blood levels of vascular endothelial growth factor are usually confirmatory. This rare disorder should not be missed, especially if the patient has a putative diagnosis of chronic inflammatory polyradiculoneuropathy, a lambda restricted monoclonal gammopathy, and thrombocytosis, and is not responding as expected to immunomodulatory therapy commonly used for chronic inflammatory polyradiculoneuropathy.
[Mh] MeSH terms primary: Immunologic Factors/therapeutic use
POEMS Syndrome
[Mh] MeSH terms secundary: Humans
POEMS Syndrome/blood
POEMS Syndrome/diagnosis
POEMS Syndrome/drug therapy
POEMS Syndrome/pathology
Plasma Cells/metabolism
Plasma Cells/pathology
Vascular Endothelial Growth Factor A/blood
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Nm] Name of substance:0 (Immunologic Factors); 0 (Vascular Endothelial Growth Factor A)
[Em] Entry month:1801
[Cu] Class update date: 180307
[Lr] Last revision date:180307
[Js] Journal subset:IM
[Da] Date of entry for processing:171122
[St] Status:MEDLINE

  4 / 8502 MEDLINE  
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[PMID]: 29157613
[Au] Autor:Fajgenbaum DC; Shilling D
[Ad] Address:Division of Translational Medicine and Human Genetics, Hospital of the University of Pennsylvania, 3400 Spruce Street, Silverstein 5, Suite S05094, Philadelphia, PA 19104, USA. Electronic address: davidfa@pennmedicine.upenn.edu.
[Ti] Title:Castleman Disease Pathogenesis.
[So] Source:Hematol Oncol Clin North Am;32(1):11-21, 2018 02.
[Is] ISSN:1558-1977
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Castleman disease (CD) describes a group of heterogeneous disorders with common lymph node histopathologic features, including atrophic or hyperplastic germinal centers, prominent follicular dendritic cells, hypervascularization, polyclonal lymphoproliferation, and/or polytypic plasmacytosis. The cause and pathogenesis of the four subtypes of CD (unicentric CD; human herpesvirus-8-associated multicentric CD; polyradiculoneuropathy, organomegaly, endocrinopathy, monoclonal plasma cell disorder, and skin changes [POEMS]-associated multicentric CD; and idiopathic multicentric CD) vary considerably. This article provides a summary of our current understanding of the cause, cell types, signaling pathways, and effector cytokines implicated in the pathogenesis of each subtype.
[Mh] MeSH terms primary: Castleman Disease
Cytokines/blood
Herpesviridae Infections
Herpesvirus 8, Human/metabolism
Lymph Nodes
Neoplasm Proteins/blood
Signal Transduction
[Mh] MeSH terms secundary: Castleman Disease/blood
Castleman Disease/physiopathology
Castleman Disease/virology
Herpesviridae Infections/blood
Herpesviridae Infections/physiopathology
Humans
Lymph Nodes/metabolism
Lymph Nodes/physiopathology
Lymph Nodes/virology
[Pt] Publication type:JOURNAL ARTICLE; REVIEW
[Nm] Name of substance:0 (Cytokines); 0 (Neoplasm Proteins)
[Em] Entry month:1801
[Cu] Class update date: 180307
[Lr] Last revision date:180307
[Js] Journal subset:IM
[Da] Date of entry for processing:171122
[St] Status:MEDLINE

  5 / 8502 MEDLINE  
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[PMID]: 29484273
[Au] Autor:Le Masson G; Solé G; Desnuelle C; Delmont E; Gauthier-Darnis M; Puget S; Durand-Zaleski I
[Ad] Address:Neuromuscular Diseases DepartmentUniversity Hospital PellegrinBordeauxFrance.
[Ti] Title:Home versus hospital immunoglobulin treatment for autoimmune neuropathies: A cost minimization analysis.
[So] Source:Brain Behav;8(2):e00923, 2018 Feb.
[Is] ISSN:2162-3279
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:Background: Prior clinical trials have suggested that home-based Ig treatment in multifocal motor neuropathy (MMN) and chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) and its variant Lewis-Sumner syndrome (LSS) is safe and effective and is less costly than hospital-administered intravenous immunoglobulin (IVIg). Methods: A French prospective, dual-center, cost minimization analysis was carried out to evaluate IVIg administration (5% concentrated) at home versus in hospital with regard to costs, patients' autonomy, and patients' quality of life. The primary endpoint was the overall cost of treatment, and we adopted the perspective of the payer (French Social Health Insurance). Results: Twenty-four patients aged 52.3 (12.2) years were analyzed: nine patients with MMN, eight with CIDP, and seven with LSS. IVIg (g/kg) dosage was 1.51 ± 0.43 in hospital and 1.52 ± 0.4 at home. Nine-month total costs per patient extrapolated to 1 year of treatment were €48,189 ± 26,105 versus €91,798 ± 51,125 in the home and hospital groups, respectively ( < .0001). The most frequently reported factors for choosing home treatment were the good tolerance and absence of side effects of IVIg administration, as well as a good understanding of the advantages and drawbacks of home treatment (75% of respondents). The mRankin scores before and after switch to home treatment were 1.61 ± 0.72 and 1.36 ± 0.76, respectively ( = .027). Discussion: The switch from hospital-based to home-based IVIg treatment for patients with immune neuropathy represents potentially significant savings in the management of the disease.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180301
[Lr] Last revision date:180301
[St] Status:In-Data-Review
[do] DOI:10.1002/brb3.923

  6 / 8502 MEDLINE  
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[PMID]: 29386496
[Au] Autor:Shiraishi N; Kinoshita M; Shimizu M; Sumikura H; Fukada K
[Ad] Address:Department of Neurology, Osaka General Medical Center.
[Ti] Title:[A case of chronic inflammatory demyelinating polyradiculoneuropathy presenting recurrent attacks associated with pregnancies].
[So] Source:Rinsho Shinkeigaku;58(2):127-130, 2018 Feb 28.
[Is] ISSN:1882-0654
[Cp] Country of publication:Japan
[La] Language:jpn
[Ab] Abstract:At 37 years of age, the patient initially presented with symptoms of chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) during her 1st pregnancy. She was treated with intravenous immunoglobulin (IVIg), and showed favorable recovery, becoming almost asymptomatic by the age of 38. At 39 years of age, during her puerperal period of her second pregnancy, she developed symmetrical muscle weakness and sensory disturbance of the upper and lower limbs. Nerve conduction studies revealed diffuse demyelination of peripheral nerves, and she was diagnosed with recurrence of CIDP. Once again, she showed remarkable improvement after IVIg therapy, and she has remained asymptomatic without the induction of preventative therapies. Recurrence of CIDP triggered in accordance with multiple pregnancies is extremely rare, and its clinical and electrophysiological features are presented in this report.
[Pt] Publication type:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180301
[Lr] Last revision date:180301
[St] Status:In-Process
[do] DOI:10.5692/clinicalneurol.cn-001104

  7 / 8502 MEDLINE  
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[PMID]: 29269693
[Au] Autor:Yasuda K; Murase N; Ohtani R; Oka N; Nakamura M
[Ad] Address:Department of Neurology, National Hospital Organization Kyoto Medical Center.
[Ti] Title:[A case of chronic inflammatory demyelinating polyradiculoneuropathy, showing radicular pain due to tuberous hypertrophy of the spinal roots and plexuses after 20 years interval without relapsing sensorimotor symptoms].
[So] Source:Rinsho Shinkeigaku;58(1):21-24, 2018 Jan 26.
[Is] ISSN:1882-0654
[Cp] Country of publication:Japan
[La] Language:jpn
[Ab] Abstract:A 40-year-old man visited our department because of chest and back pain. He had a history of diagnosis of chronic inflammatory demyelinating polyneuropathy (CIDP) 20 years ago. He received immunosuppressive therapy and had no relapses after that. On Admission, MRI showed tuberous hypertrophy of the spinal roots, intercostal nerves, and brachial and lumbar plexuses. The genetic analysis showed no mutations in any of Charcot-Marie-Tooth related genes. He was finally diagnosed with CIDP and administration of high dose intravenous methylprednisolone relieved his chest and back pain within a few days. We present a rare case of CIDP in which showed marked enlarged spinal roots in long clinical course and have a relapse with radicular pain without sensorimotor symptoms.
[Mh] MeSH terms primary: Back Pain/etiology
Brachial Plexus/pathology
Chest Pain/etiology
Polyradiculoneuropathy, Chronic Inflammatory Demyelinating/complications
Spinal Nerve Roots/pathology
[Mh] MeSH terms secundary: Adult
Back Pain/drug therapy
Brachial Plexus/diagnostic imaging
Chest Pain/drug therapy
Humans
Hypertrophy
Infusions, Intravenous
Magnetic Resonance Imaging
Male
Methylprednisolone/administration & dosage
Polyradiculoneuropathy, Chronic Inflammatory Demyelinating/diagnostic imaging
Polyradiculoneuropathy, Chronic Inflammatory Demyelinating/drug therapy
Polyradiculoneuropathy, Chronic Inflammatory Demyelinating/pathology
Pulse Therapy, Drug
Spinal Nerve Roots/diagnostic imaging
Time Factors
Treatment Outcome
[Pt] Publication type:CASE REPORTS; JOURNAL ARTICLE
[Nm] Name of substance:X4W7ZR7023 (Methylprednisolone)
[Em] Entry month:1802
[Cu] Class update date: 180227
[Lr] Last revision date:180227
[Js] Journal subset:IM
[Da] Date of entry for processing:171223
[St] Status:MEDLINE
[do] DOI:10.5692/clinicalneurol.cn-001073

  8 / 8502 MEDLINE  
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[PMID]: 29360221
[Au] Autor:Kacar A; Bjelica B; Bozovic I; Peric S; Nikolic A; Cobeljic M; Petrovic M; Stojanov A; Djordjevic G; Vukojevic Z; Dominovic-Kovacevic A; Stojanovic M; Stevic Z; Rakocevic-Stojanovic V; Lavrnic D; Basta I
[Ad] Address:Neurology Clinic, Clinical Center of Serbia, School of Medicine, University of Belgrade, Belgrade, Serbia.
[Ti] Title:Neuromuscular disease-specific questionnaire to assess quality of life in patients with chronic inflammatory demyelinating polyradiculoneuropathy.
[So] Source:J Peripher Nerv Syst;, 2018 Jan 23.
[Is] ISSN:1529-8027
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:To date, generic questionnaires have been used to investigate quality of life (QoL) in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) patients. Although these measures are very useful, they are not usually precise enough to measure all specific characteristics of the disease. Our aim was to investigate QoL using the neuromuscular disease-specific questionnaire (individualized neuromuscular quality of life, INQoL) in a large cohort of patients with CIDP. Our study comprised 106 patients diagnosed with CIDP. INQoL questionnaire, Medical Research Council (MRC) sum score, Inflammatory Neuropathy Cause and Treatment (INCAT) disability score, Visual Analogue Pain Scale, Beck Depression Inventory, and Krupp's Fatigue Severity Scale were used in our study. Physical domains of INQoL were more affected than mental, and the overall score was 57 ± 25. Significant predictors of higher INQoL score in our patients with CIDP were severe fatigue (ß = 0.35, p < 0.01), higher INCAT disability score at time of testing (ß = 0.29, p < 0.01), and being unemployed/retired (ß = 0.22, p < 0.05). QoL was reduced in our cohort of CIDP patients, which was more pronounced in physical segments. Patients with fatigue, more severe disability, and unemployed/retired need special attention of neurologists because they could be at greater risk to have worse QoL.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1801
[Cu] Class update date: 180215
[Lr] Last revision date:180215
[St] Status:Publisher
[do] DOI:10.1111/jns.12251

  9 / 8502 MEDLINE  
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[PMID]: 29435815
[Au] Autor:Härtig F; Ross M; Dammeier NM; Fedtke N; Heiling B; Axer H; Décard BF; Auffenberg E; Koch M; Rattay TW; Krumbholz M; Bornemann A; Lerche H; Winter N; Grimm A
[Ad] Address:Department of Neurology, Tübingen University Hospital, University of Tübingen, Hoppe-Seyler-Str. 3, 72076, Tubingen, Germany.
[Ti] Title:Nerve Ultrasound Predicts Treatment Response in Chronic Inflammatory Demyelinating Polyradiculoneuropathy-a Prospective Follow-Up.
[So] Source:Neurotherapeutics;, 2018 Feb 12.
[Is] ISSN:1878-7479
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:As reliable biomarkers of disease activity are lacking, monitoring of therapeutic response in chronic inflammatory demyelinating polyradiculoneuropathy (CIDP) remains a challenge. We sought to determine whether nerve ultrasound and electrophysiology scoring could close this gap. In CIDP patients (fulfilling EFNS/PNS criteria), we performed high-resolution nerve ultrasound to determine ultrasound pattern sum scores (UPSS) and predominant echotexture nerve conduction study scores (NCSS) as well as Medical Research Council sum scores (MRCSS) and inflammatory neuropathy cause and treatment disability scores (INCAT) at baseline and after 12 months of standard treatment. We retrospectively correlated ultrasound morphology with nerve histology when available. 72/80 CIDP patients featured multifocal nerve enlargement, and 35/80 were therapy-naïve. At baseline, clinical scores correlated with NCSS (r = 0.397 and r = 0.443, p < 0.01), but not or hardly with UPSS (Medical Research Council sum scores MRCSS r = 0.013, p = 0.332; inflammatory neuropathy cause and treatment disability scores INCAT r = 0.053, p = 0.048). Longitudinal changes in clinical scores, however, correlated significantly with changes in both UPSS and NCSS (r = 0.272-0.414, p < 0.0001). Combining nerve/fascicle size with echointensity and histology at baseline, we noted 3 distinct classes: 1) hypoechoic enlargement, reflecting active inflammation and onion bulbs; 2) nerve enlargement with additional hyperechogenic fascicles/perifascicular tissue in > 50% of measured segments, possibly reflecting axonal degeneration; and 3) almost no enlargement, reflecting "burned-out" or "cured" disease without active inflammation. Clinical improvement after 12 months was best in patients with pattern 1 (up to 75% vs up to 43% in pattern 2/3, Fisher's exact test p < 0.05). Nerve ultrasound has additional value not only for diagnosis, but also for classification of disease state and may predict treatment response.
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180213
[Lr] Last revision date:180213
[St] Status:Publisher
[do] DOI:10.1007/s13311-018-0609-4

  10 / 8502 MEDLINE  
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[PMID]: 27779466
[Au] Autor:Dirlikov E; Kniss K; Major C; Thomas D; Virgen CA; Mayshack M; Asher J; Mier-Y-Teran-Romero L; Salinas JL; Pastula DM; Sharp TM; Sejvar J; Johansson MA; Rivera-Garcia B
[Ti] Title:Guillain-Barré Syndrome and Healthcare Needs during Zika Virus Transmission, Puerto Rico, 2016.
[So] Source:Emerg Infect Dis;23(1):134-136, 2017 01.
[Is] ISSN:1080-6059
[Cp] Country of publication:United States
[La] Language:eng
[Ab] Abstract:To assist with public health preparedness activities, we estimated the number of expected cases of Zika virus in Puerto Rico and associated healthcare needs. Estimated annual incidence is 3.2-5.1 times the baseline, and long-term care needs are predicted to be 3-5 times greater than in years with no Zika virus.
[Mh] MeSH terms primary: Disease Outbreaks
Guillain-Barre Syndrome/epidemiology
Health Services Needs and Demand/statistics & numerical data
Models, Statistical
Zika Virus Infection/epidemiology
[Mh] MeSH terms secundary: Forecasting
Guillain-Barre Syndrome/complications
Guillain-Barre Syndrome/therapy
Guillain-Barre Syndrome/virology
Humans
Incidence
Long-Term Care/statistics & numerical data
Monte Carlo Method
Population Surveillance
Puerto Rico/epidemiology
Zika Virus/pathogenicity
Zika Virus/physiology
Zika Virus Infection/complications
Zika Virus Infection/therapy
Zika Virus Infection/virology
[Pt] Publication type:JOURNAL ARTICLE
[Em] Entry month:1802
[Cu] Class update date: 180209
[Lr] Last revision date:180209
[Js] Journal subset:IM
[Da] Date of entry for processing:161026
[St] Status:MEDLINE
[do] DOI:10.3201/eid2301.161290


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