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Pesquisa : autacóides [Palavras]
Referências encontradas : 513 [refinar]
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  1 / 513 MEDLINE  
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[PMID]:27935242
[Au] Autor:Penson PE
[Ad] Endereço:Pharmacy & Biomolecular Sciences, Liverpool John Moores Univeristy, Liverpool, UK.
[Ti] Título:Autonomic & Autacoid Pharmacology 2016: The year in review.
[So] Source:Auton Autacoid Pharmacol;36(3-4):27, 2016 Jul.
[Is] ISSN:1474-8673
[Cp] País de publicação:England
[La] Idioma:eng
[Mh] Termos MeSH primário: Autacoides/farmacologia
Sistema Nervoso Autônomo
Políticas Editoriais
[Mh] Termos MeSH secundário: Animais
Sistema Nervoso Autônomo/efeitos de drogas
Sistema Nervoso Autônomo/fisiologia
Humanos
[Pt] Tipo de publicação:EDITORIAL
[Nm] Nome de substância:
0 (Autacoids)
[Em] Mês de entrada:1701
[Cu] Atualização por classe:170127
[Lr] Data última revisão:
170127
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161209
[St] Status:MEDLINE
[do] DOI:10.1111/aap.12051


  2 / 513 MEDLINE  
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[PMID]:27729772
[Au] Autor:Hesselink JM; Chiosi F; Costagliola C
[Ad] Endereço:University of Witten/Herdecke, Witten, Germany.
[Ti] Título:Resolvins and aliamides: lipid autacoids in ophthalmology - what promise do they hold?
[So] Source:Drug Des Devel Ther;10:3133-3141, 2016.
[Is] ISSN:1177-8881
[Cp] País de publicação:New Zealand
[La] Idioma:eng
[Ab] Resumo:Resolvins are a novel class of lipid-derived endogenous molecules (autacoids) with potent immunomodulating properties, which regulate the resolution phase of an active immune response. These modulating factors are locally produced, influencing the function of cells and/or tissues, which are produced on demand and subsequently metabolized in the same cells and/or tissues. This review is focused on certain lipid autacoids with putative relevance for ophthalmology in general and for dry eye more specifically. We also briefly investigate the concept of aliamides and the role of palmitoylethanolamide in ophthalmology, and analyze in more detail the putative role and the preclinical and clinical development of resolvins as emerging treatments for dry eye and related disorders, with a focus on one of the lead resolvin derivatives - RX-10045.
[Mh] Termos MeSH primário: Autacoides/uso terapêutico
Síndromes do Olho Seco/quimioterapia
Oftalmologia
[Mh] Termos MeSH secundário: Autacoides/administração & dosagem
Autacoides/química
Humanos
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Autacoids)
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170508
[Lr] Data última revisão:
170508
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161012
[St] Status:MEDLINE


  3 / 513 MEDLINE  
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[PMID]:27030719
[Au] Autor:Ramsden CE; Ringel A; Majchrzak-Hong SF; Yang J; Blanchard H; Zamora D; Loewke JD; Rapoport SI; Hibbeln JR; Davis JM; Hammock BD; Taha AY
[Ad] Endereço:Laboratory of Membrane Biochemistry and Biophysics, National Institute on Alcohol Abuse and Alcoholism, National Institutes of Health, Bethesda, MD, USA Department of Physical Medicine and Rehabilitation, University of North Carolina-Chapel Hill, NC, USA chris.ramsden@nih.gov.
[Ti] Título:Dietary linoleic acid-induced alterations in pro- and anti-nociceptive lipid autacoids: Implications for idiopathic pain syndromes?
[So] Source:Mol Pain;12, 2016.
[Is] ISSN:1744-8069
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Chronic idiopathic pain syndromes are major causes of personal suffering, disability, and societal expense. Dietary n-6 linoleic acid has increased markedly in modern industrialized populations over the past century. These high amounts of linoleic acid could hypothetically predispose to physical pain by increasing the production of pro-nociceptive linoleic acid-derived lipid autacoids and by interfering with the production of anti-nociceptive lipid autacoids derived from n-3 fatty acids. Here, we used a rat model to determine the effect of increasing dietary linoleic acid as a controlled variable for 15 weeks on nociceptive lipid autacoids and their precursor n-6 and n-3 fatty acids in tissues associated with idiopathic pain syndromes. RESULTS: Increasing dietary linoleic acid markedly increased the abundance of linoleic acid and its pro-nociceptive derivatives and reduced the abundance of n-3 eicosapentaenoic acid and docosahexaenoic acid and their anti-nociceptive monoepoxide derivatives. Diet-induced changes occurred in a tissue-specific manner, with marked alterations of nociceptive lipid autacoids in both peripheral and central tissues, and the most pronounced changes in their fatty acid precursors in peripheral tissues. CONCLUSIONS: The present findings provide biochemical support for the hypothesis that the high linoleic acid content of modern industrialized diets may create a biochemical susceptibility to develop chronic pain. Dietary linoleic acid lowering should be further investigated as part of an integrative strategy for the prevention and management of idiopathic pain syndromes.
[Mh] Termos MeSH primário: Autacoides/farmacologia
Gorduras na Dieta/farmacologia
Ácido Linoleico/farmacologia
Nociceptividade/efeitos de drogas
Dor/patologia
[Mh] Termos MeSH secundário: Animais
Ácidos Graxos Ômega-3/farmacologia
Masculino
Especificidade de Órgãos/efeitos de drogas
Oxilipinas/farmacologia
Ratos Endogâmicos F344
Síndrome
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL; RESEARCH SUPPORT, N.I.H., INTRAMURAL
[Nm] Nome de substância:
0 (Autacoids); 0 (Dietary Fats); 0 (Fatty Acids, Omega-3); 0 (Oxylipins); 9KJL21T0QJ (Linoleic Acid)
[Em] Mês de entrada:1612
[Cu] Atualização por classe:161230
[Lr] Data última revisão:
161230
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160331
[St] Status:MEDLINE


  4 / 513 MEDLINE  
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[PMID]:27329171
[Au] Autor:E Penson P
[Ad] Endereço:Pharmacy & Biomolecular Sciences, Liverpool John Moores Univeristy, Liverpool, UK.
[Ti] Título:Autonomic & Autacoid Pharmacology: past, present and future.
[So] Source:Auton Autacoid Pharmacol;35(4):45, 2015 Dec.
[Is] ISSN:1474-8673
[Cp] País de publicação:England
[La] Idioma:eng
[Mh] Termos MeSH primário: Autacoides/farmacologia
Autacoides/uso terapêutico
[Mh] Termos MeSH secundário: Humanos
Farmacologia/métodos
[Pt] Tipo de publicação:EDITORIAL
[Nm] Nome de substância:
0 (Autacoids)
[Em] Mês de entrada:1701
[Cu] Atualização por classe:170117
[Lr] Data última revisão:
170117
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160622
[St] Status:MEDLINE
[do] DOI:10.1111/aap.12039


  5 / 513 MEDLINE  
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[PMID]:27028114
[Au] Autor:Keppel Hesselink JM
[Ad] Endereço:Research and development, Institute Neuropathic Pain, Spoorlaan 2a, Bosch en Duin, 3735 MV, Netherlands.
[Ti] Título:The terms 'autacoid', 'hormone' and 'chalone' and how they have shifted with time.
[So] Source:Auton Autacoid Pharmacol;35(4):51-8, 2015 Dec.
[Is] ISSN:1474-8673
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:The increase of knowledge in a particular field (endocrinology) can be understood if one follows how certain key concepts were constructed and transformed over time. To explore such construction and transformation (shifts in meaning), we studied the use of the concepts 'autacoid' and 'chalone' in a period of one century (1916-2016), since the introduction of these concepts by the British professor of physiology Sir Sharpey-Schäfer. We could identify that the use of 'autacoid' shifted from a very broad category encompassing both stimulating and inhibiting hormones, in the period 1916-1960, to a much more specific use of the term for locally produced bioactive molecules, from the 1960s onwards. Histamine was the first compound seen as an 'autacoid', followed by prostaglandins, ATP, ADP and bradykinin, and from 1993 onwards, compounds such as 'palmitoylethanolamide' were also classified as 'autacoids'. For 'chalone', a comparable shift was noticed around the 1960s, when the concept suddenly changed from the category of inhibiting hormones into a substance that is produced within a tissue, inhibiting mitosis of the cells of that tissue. For both concept shifts, we could not find any argument. Around 1980, authors started to relate autacoids to various promising indications in the field of inflammation and immune modulation. The Nobel laureate Rita Levi-Montalcini gave an extra dimension to the concept autacoid in 1993, and introduced a new class of compounds modulating mast cells, the ALIAmides (from Autacoid Local Inflammation Antagonist), of which palmitoylethanolamide was the prototype. Our exploration demonstrates that biomedical concepts can be constructed and defined differently as time goes by, while concept transformations seem to emerge without arguments.
[Mh] Termos MeSH primário: Autacoides/metabolismo
Calônios/metabolismo
Hormônios/metabolismo
[Mh] Termos MeSH secundário: Bradicinina/metabolismo
Endocanabinoides/metabolismo
Histamina/metabolismo
Humanos
Inflamação/metabolismo
Mastócitos/metabolismo
Prostaglandinas/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Autacoids); 0 (Chalones); 0 (Endocannabinoids); 0 (Hormones); 0 (Prostaglandins); 820484N8I3 (Histamine); S8TIM42R2W (Bradykinin)
[Em] Mês de entrada:1701
[Cu] Atualização por classe:170117
[Lr] Data última revisão:
170117
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160622
[St] Status:MEDLINE
[do] DOI:10.1111/aap.12037


  6 / 513 MEDLINE  
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[PMID]:25879450
[Au] Autor:Kozik A; Gogol M; Bochenska O; Karkowska-Kuleta J; Wolak N; Kamysz W; Aoki W; Ueda M; Faussner A; Rapala-Kozik M
[Ad] Endereço:Faculty of Biochemistry, Biophysics, and Biotechnology, Jagiellonian University in Krakow, Gronostajowa 7, 30-387, Krakow, Poland. andrzej.kozik@uj.edu.pl.
[Ti] Título:Kinin release from human kininogen by 10 aspartic proteases produced by pathogenic yeast Candida albicans.
[So] Source:BMC Microbiol;15:60, 2015 Mar 04.
[Is] ISSN:1471-2180
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Candida albicans yeast produces 10 distinct secreted aspartic proteases (Saps), which are some of the most important virulence factors of this pathogenic fungus. One of the suggested roles of Saps is their deregulating effect on various proteolytic cascades that constitute the major homeostatic systems in human hosts, including blood coagulation, fibrinolysis, and kallikrein-kinin systems. This study compared the characteristics of the action of all 10 Saps on human kininogens, which results in generating proinflammatory bradykinin-related peptides (kinins). RESULTS: Recombinant forms of Saps, heterologously overexpressed in Pichia pastoris were applied. Except for Sap7 and Sap10, all Saps effectively cleaved the kininogens, with the highest hydrolytic activity toward the low-molecular-mass form (LK). Sap1-6 and 8 produced a biologically active kinin-Met-Lys-bradykinin-and Sap3 was exceptional in terms of the kinin-releasing yield (>60% LK at pH 5.0 after 24 hours). Des-Arg(1)-bradykinin was released from LK by Sap9 at a comparably high yield, but this peptide was assumed to be biologically inactive because it was unable to interact with cellular B2-type kinin receptors. However, the collaborative actions of Sap9 and Sap1, -2, -4-6, and -8 on LK rerouted kininogen cleavage toward the high-yield release of the biologically active Met-Lys-bradykinin. CONCLUSIONS: Our present results, together with the available data on the expression of individual SAP genes in candidal infection models, suggest a biological potential of Saps to produce kinins at the infection foci. The kinin release during candidiasis can involve predominant and complementary contributions of two different Sap3- and Sap9-dependent mechanisms.
[Mh] Termos MeSH primário: Ácido Aspártico Proteases/química
Autacoides/química
Candida albicans/química
Proteínas Fúngicas/química
Cininogênios/química
Cininas/química
[Mh] Termos MeSH secundário: Sequência de Aminoácidos
Ácido Aspártico Proteases/genética
Bradicinina/análogos & derivados
Bradicinina/química
Candida albicans/enzimologia
Candida albicans/patogenicidade
Proteínas Fúngicas/genética
Expressão Gênica
Humanos
Concentração de Íons de Hidrogênio
Isoenzimas/química
Isoenzimas/genética
Dados de Sequência Molecular
Pichia/genética
Pichia/metabolismo
Proteólise
Proteínas Recombinantes/química
Proteínas Recombinantes/genética
Virulência
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Autacoids); 0 (Fungal Proteins); 0 (Isoenzymes); 0 (Kininogens); 0 (Kinins); 0 (Recombinant Proteins); 550-19-6 (bradykinin, Met-Lys-); EC 3.4.- (Aspartic Acid Proteases); S8TIM42R2W (Bradykinin)
[Em] Mês de entrada:1601
[Cu] Atualização por classe:150418
[Lr] Data última revisão:
150418
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150417
[St] Status:MEDLINE
[do] DOI:10.1186/s12866-015-0394-8


  7 / 513 MEDLINE  
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[PMID]:25217168
[Au] Autor:Arnardottir HH; Dalli J; Colas RA; Shinohara M; Serhan CN
[Ad] Endereço:Center for Experimental Therapeutics and Reperfusion Injury, Department of Anesthesiology, Perioperative and Pain Medicine, Harvard Institutes of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA 02115.
[Ti] Título:Aging delays resolution of acute inflammation in mice: reprogramming the host response with novel nano-proresolving medicines.
[So] Source:J Immunol;193(8):4235-44, 2014 Oct 15.
[Is] ISSN:1550-6606
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Aging is associated with an overt inflammatory phenotype and physiological decline. Specialized proresolving lipid mediators (SPMs) are endogenous autacoids that actively promote resolution of inflammation. In this study, we investigated resolution of acute inflammation in aging and the roles of SPMs. Using a self-resolving peritonitis and resolution indices coupled with lipid mediator metabololipidomics, we found that aged mice had both delayed resolution and reduced SPMs. The SPM precursor docosahexaenoic acid accelerated resolution via increased SPMs and promoted human monocyte reprogramming. In aged mice, novel nano-proresolving medicines carrying aspirin-triggered resolvins D1 and D3 reduced inflammation by promoting efferocytosis. These findings provide evidence for age-dependent resolution pathways in acute inflammation and novel means to activate resolution.
[Mh] Termos MeSH primário: Envelhecimento/imunologia
Autacoides/imunologia
Inflamação/imunologia
Nanomedicina
Peritonite/imunologia
[Mh] Termos MeSH secundário: Animais
Aspirina/farmacologia
Ácidos Docosa-Hexaenoicos/farmacologia
Ácido Eicosapentaenoico/farmacologia
Ácidos Graxos Insaturados/farmacologia
Humanos
Inflamação/quimioterapia
Mediadores da Inflamação/imunologia
Leucócitos Mononucleares/imunologia
Macrófagos/imunologia
Masculino
Metabolômica
Camundongos
Camundongos Endogâmicos BALB C
Análise de Componente Principal
Zimosan/imunologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Autacoids); 0 (Fatty Acids, Unsaturated); 0 (Inflammation Mediators); 0 (resolvin D1); 0 (resolvin D3); 25167-62-8 (Docosahexaenoic Acids); 9010-72-4 (Zymosan); AAN7QOV9EA (Eicosapentaenoic Acid); R16CO5Y76E (Aspirin)
[Em] Mês de entrada:1501
[Cu] Atualização por classe:161019
[Lr] Data última revisão:
161019
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:141004
[St] Status:MEDLINE
[do] DOI:10.4049/jimmunol.1401313


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[PMID]:22619589
[Au] Autor:Radenkovic M; Stojanovic M; Jankovic R; Topalovic M; Stojiljkovic M
[Ad] Endereço:Department of Pharmacology, Clinical Pharmacology and Toxicology, School of Medicine, University of Belgrade, P.O. Box 38, 11129 Belgrade, Serbia. mradenkovic@med.bg.ac.rs
[Ti] Título:Combined contribution of endothelial relaxing autacoides in the rat femoral artery response to CPCA: an adenosine A2 receptor agonist.
[So] Source:ScientificWorldJournal;2012:143818, 2012.
[Is] ISSN:1537-744X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:We examined the contribution of endothelial relaxing factors and potassium channels in actions of CPCA, potent adenosine A(2) receptor agonist, on isolated intact male rat femoral artery (FA). CPCA produced concentration-dependent relaxation of FA, which was notably, but not completely, reduced after endothelial denudation. DPCPX, A(1) receptor antagonist, had no significant effect, while SCH 58261 (A(2A) receptor antagonist) notably reduced CPCA-evoked effect. Pharmacological inhibition of nitric oxide synthase or cyclooxygenase comparably reduced CPCA-evoked action, still in a lesser degree than after denudation. In the presence of buffer with high K(+) (100 mM), CPCA-produced relaxations were almost abolished. TEA (nonselective K(Ca) blocker), glibenclamide (K(ATP) blocker), Ba(++) (K(IR) blocker), or ouabain (Na(+)/K(+)-ATPase inhibitor) did not change CPCA-induced relaxation. Concentration-response curve for CPCA was significantly shifted to the right after the incubation of apamin (SK channel blocker). CPCA produced concentration-dependent relaxation of FA that was partly dependent on endothelial cells. Endothelium-related portion of CPCA-elicited effect was mediated by combined action of endothelial NO, prostacyclin, and EDHF after activation of endothelial A(2A) receptors. Small conductance K(Ca) channels were involved in this action.
[Mh] Termos MeSH primário: Agonistas do Receptor A2 de Adenosina/farmacologia
Adenosina/análogos & derivados
Autacoides/fisiologia
Artéria Femoral/efeitos de drogas
Receptores A2 de Adenosina/efeitos de drogas
[Mh] Termos MeSH secundário: Adenosina/farmacologia
Animais
Artéria Femoral/fisiologia
Relaxamento Muscular/fisiologia
Ratos
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Adenosine A2 Receptor Agonists); 0 (Autacoids); 0 (Receptors, Adenosine A2); 50908-62-8 (N-cyclopropyl adenosine-5'-carboxamide); K72T3FS567 (Adenosine)
[Em] Mês de entrada:1209
[Cu] Atualização por classe:150225
[Lr] Data última revisão:
150225
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:120523
[St] Status:MEDLINE
[do] DOI:10.1100/2012/143818


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[PMID]:21566204
[Au] Autor:Schindler C; Leuschner S; Schwanebeck U; Kirch W
[Ad] Endereço:Institute of Clinical Pharmacology, Medical Faculty, Technical University Dresden, Fiedlerstrasse 27, D-01307 Dresden, Germany. christoph.schindler@tu-dresden.de
[Ti] Título:Characterization of local vascular effects of the nitric oxide inhibitor NG-monomethyl-L-arginine on dorsal hand veins.
[So] Source:J Clin Pharmacol;52(6):859-69, 2012 Jun.
[Is] ISSN:1552-4604
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Infusion of NG-monomethyl-L-arginine (L-NMMA; 6.4 µmol/min) into hand veins can cause a 20% increase in vein size in specific subjects. This study explored potential underlying mechanisms in healthy male participants. Ten healthy male participants received in phenylephrine (PE)-preconstricted veins a dose-response curve (DRC) to L-NMMA (0.2-6.4 µmol/min) without and with coinfusion of the endothelium-dependent dilator histamine, a DRC to L-arginine with and without coinfusion of L-NMMA, a DRC to NG-monomethyl-D-arginine (D-NMMA), and a DRC to L-NMMA in prostaglandin F(2α)-(PGF(2α))-preconstricted veins. Participants were classified as L-NMMA responders (R) and nonresponders (NR). Infusion of L-NMMA resulted in a maximum venodilation of 38% ± 11% (R) versus 10% ± 5% (NR; P = .005). In PGF(2α)-preconstricted veins, L-NMMA caused venodilation to 26% ± 34% (NS) in responders. Results suggest that endothelial nitric oxide synthase-mediated formation of nitric oxide (NO) from L-NMMA in doses >3.2 µmol/min and continuous PE-induced α-adrenergic stimulation resulting in release of very small amounts of NO from L-NMMA contribute to the observed L-NMMA-induced increase in vein size. Venous reactivity to L-NMMA resulting in a phenotype as R or NR is most likely genetically predetermined, which requires further study.
[Mh] Termos MeSH primário: Mãos/irrigação sanguínea
Óxido Nítrico/antagonistas & inibidores
Vasodilatadores/farmacologia
Veias/efeitos de drogas
ômega-N-Metilarginina/farmacologia
[Mh] Termos MeSH secundário: Adulto
Arginina/metabolismo
Autacoides/metabolismo
Estudos Cross-Over
Relação Dose-Resposta a Droga
Resistência a Medicamentos
Endotélio Vascular/efeitos de drogas
Endotélio Vascular/fisiologia
Inibidores Enzimáticos/farmacologia
Humanos
Infusões Intravenosas
Masculino
Óxido Nítrico/metabolismo
Doadores de Óxido Nítrico/farmacologia
Óxido Nítrico Sintase Tipo III/antagonistas & inibidores
Óxido Nítrico Sintase Tipo III/metabolismo
Estereoisomerismo
Vasoconstritores/antagonistas & inibidores
Vasoconstritores/farmacologia
Vasodilatação/efeitos de drogas
Vasodilatadores/administração & dosagem
Vasodilatadores/química
Veias/metabolismo
Adulto Jovem
ômega-N-Metilarginina/administração & dosagem
ômega-N-Metilarginina/química
[Pt] Tipo de publicação:CLINICAL TRIAL; CONTROLLED CLINICAL TRIAL; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Autacoids); 0 (Enzyme Inhibitors); 0 (Nitric Oxide Donors); 0 (Vasoconstrictor Agents); 0 (Vasodilator Agents); 27JT06E6GR (omega-N-Methylarginine); 31C4KY9ESH (Nitric Oxide); 94ZLA3W45F (Arginine); EC 1.14.13.39 (NOS3 protein, human); EC 1.14.13.39 (Nitric Oxide Synthase Type III)
[Em] Mês de entrada:1210
[Cu] Atualização por classe:131121
[Lr] Data última revisão:
131121
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:120528
[Cl] Clinical Trial:ClinicalTrial
[St] Status:MEDLINE
[do] DOI:10.1177/0091270011406277


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[PMID]:22272416
[Au] Autor:Natochin IuV
[Ti] Título:[Water-salt homeostasis--role of reflexes, hormones, incretins, and autacoids].
[So] Source:Fiziol Zh;57(5):13-5, 2011.
[Cp] País de publicação:Ukraine
[La] Idioma:rus
[Mh] Termos MeSH primário: Autacoides/fisiologia
Hormônios/fisiologia
Reflexo/fisiologia
Equilíbrio Hidroeletrolítico/fisiologia
[Mh] Termos MeSH secundário: Animais
Humanos
Incretinas/fisiologia
Concentração Osmolar
[Pt] Tipo de publicação:LECTURES; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Autacoids); 0 (Hormones); 0 (Incretins)
[Em] Mês de entrada:1203
[Cu] Atualização por classe:120124
[Lr] Data última revisão:
120124
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:120124
[St] Status:MEDLINE



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BIREME/OPAS/OMS - Centro Latino-Americano e do Caribe de Informação em Ciências da Saúde