Base de dados : LILACS
Pesquisa : A11.118.637.555.567.550.500.400 [Categoria DeCS]
Referências encontradas : 45 [refinar]
Mostrando: 1 .. 10   no formato [Detalhado]

página 1 de 5 ir para página              

  1 / 45 LILACS  
              next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Rizzo, Luiz Vicente
Texto completo
Id: biblio-1154101
Autor: Alonso, Giovana Toledo; Fomin, Denilson Stork; Rizzo, Luiz Vicente.
Título: Human follicular helper T lymphocytes critical players in antibody responses / Linfócitos T auxiliares foliculares humanos: células essenciais para a resposta de anticorpos
Fonte: Einstein (Säo Paulo);19:eRB6077, 2021. tab.
Idioma: en.
Resumo: ABSTRACT Follicular helper T lymphocytes are a subpopulation of CD4+ T lymphocytes initially identified in germinal centers of follicles found in secondary lymphoid organs. The primary function of follicular helper T lymphocytes is to help B lymphocytes' antibody production. Changing of antibody class and affinity, B cell differentiation and memory generation depend on cooperation between follicular helper T lymphocytes and B cells. In blood, follicular helper T lymphocytes are called circulating follicular helper T lymphocytes. They are considered to have specificities similar to those developed in the secondary lymphoid organs. The phenotype of human follicular helper T lymphocytes is given by simultaneous expression of the markers CXCR5, Bcl-6, CD40L, PD-1, and ICOS. In germinal centers, follicular helper T lymphocytes synthesize interleukin 21 as predominant cytokine. In blood, subpopulations of circulating follicular helper T lymphocytes can be recognized, with different expressions of the classical follicular helper T lymphocytes markers and, in addition, can express other markers such as CXCR3 and CCR6. Presently, there is great interest in follicular helper T lymphocytes and circulating follicular helper T lymphocytes in vaccination studies as indicators of immunization efficacy. In addition, follicular helper T lymphocytes are investigated as possible markers of activity in many diseases and potential therapeutic intervention. This short review describes aspects of immunobiology and quantification of follicular helper T lymphocytes and circulating follicular helper T lymphocytes, and presents a few examples of related findings in systemic lupus erythematosus, rheumatoid arthritis, HIV infection and vaccination.

RESUMO Linfócitos T auxiliares foliculares são uma subpopulação de linfócitos T CD4+ identificada inicialmente nos centros germinativos dos folículos dos órgãos linfoides secundários. Sua função primordial é auxiliar os linfócitos B na produção de anticorpos. A mudança de classe e de afinidade dos anticorpos, a diferenciação das células B e a geração de memória dependem da cooperação entre os linfócitos T auxiliares foliculares e as células B. No sangue, recebem o nome de linfócitos T auxiliares circulantes. Considera-se que possuem especificidades semelhantes às desenvolvidas nos órgãos linfoides secundários. O fenótipo dos linfócitos T auxiliares humanos é dado pela expressão conjunta dos marcadores CXCR5, Bcl-6, CD40L, PD-1 e ICOS. Nos folículos, linfócitos T auxiliares sintetizam a interleucina 21 como citocina predominante. No sangue, são descritas várias subpopulações de linfócitos T auxiliares circulantes com expressões variadas dos marcadores clássicos de linfócitos T auxiliares, além de poderem agregar outros, como CXCR3 e CCR6. Existe um enorme interesse no estudo de linfócitos T auxiliares e linfócitos T auxiliares circulantes, para a avaliação de eficácia de vacinação. São também investigados como possíveis marcadores de atividade em muitas doenças e potenciais intervenções terapêuticas. Esta breve revisão descreve aspectos da imunobiologia e da quantificação de linfócitos T auxiliares humanos e linfócitos T auxiliares circulantes, além de apresentar alguns achados relacionados em lúpus eritematoso sistêmico, artrite reumatoide, infecção por HIV e vacinação.
Descritores: Linfócitos T Auxiliares-Indutores/imunologia
Centro Germinativo/imunologia
Formação de Anticorpos
-Linfócitos B/imunologia
Limites: Humanos
Tipo de Publ: Revisão
Responsável: BR1.1 - BIREME


  2 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Id: biblio-1236844
Autor: Rojas-Espinosa, Oscar; Jimenez-Zamudio, Luis; Arce-Paredes, Patricia.
Título: Activacion secuencial de la inmunidad celular y la inmunidad humoral en la lepra: consideraciones con base en hallazgos recientes / ?.
Fonte: s.l; s.n; 1994. 7 p. ilus, graf.
Idioma: es.
Descritores: Anticorpos Antibacterianos/biossíntese
Apresentação do Antígeno
Formação de Anticorpos
Hanseníase Virchowiana/imunologia
Hanseníase/imunologia
Linfócitos B/imunologia
Linfócitos T Auxiliares-Indutores/imunologia
Macrófagos/imunologia
Modelos Imunológicos
Mycobacterium leprae/imunologia
Reação em Cadeia da Polimerase
Limites: Animais
Humanos
Camundongos
Responsável: BR191.1 - Biblioteca e Centro de Documentação Luiza Keffer
[{"text": "BR191.1", "_a": "06703/s"}]


  3 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: biblio-839397
Autor: Sun, Changzhi; Ouyang, Hong; Luo, Renzhong.
Título: Distinct characteristics of nasal polyps with and without eosinophilia / Diferenças nas características de pólipos nasais com e sem eosinofilia
Fonte: Braz. j. otorhinolaryngol. (Impr.);83(1):66-72, Jan.-Feb. 2017. tab, graf.
Idioma: en.
Projeto: Medical Scientific Research Foundation of Guangdong Province.
Resumo: Abstract Introduction Eosinophilic and noneosinophilic Nasal polyps (NPs) are different subtypes of NPs and require different treatment methods. Objective To compare the histologic characteristics, mRNA and protein expression between Nasal Polyps with and without eosinophilia. Methods NPs tissues were obtained from eighty-six NPs patients during surgery. Eosinophilic and noneosinophilic NPs were distinguished according to immunochemical results of the specimen. The histological, mRNA and protein expression features were compared between the two groups. Results In eosinophilic NPs, we observed a significantly higher GATA-3, IL-5, IL-4, IL-13 mRNA and protein expression. In noneosinophilic NPs, IL-17, IL-23 and RORc mRNA and protein expression were increased. Immunohistochemistry tests showed, more mast cells and less neutrophils in eosinophilic NPs compared with noneosinophilic NPs. Eosinophilic NPs patient presented more severe symptom scores when compared to noneosinophilic NPs. Conclusion We demonstrate for the first time that Th2 is the predominant reaction in eosinophilic NPs while Th17 is the predominant reaction in noneosinophilic NPs. Our study may provide new treatment strategy for NPs.

Resumo Introdução Pólipos nasais (PNs) eosinofílicos e não eosinofílicos são diferentes subtipos de PNs e requerem diferentes métodos de tratamento. Objetivo Comparar as características histológicas e a expressão de mRNAs e proteínas entre PNs com e sem eosinofilia. Método Amostras de PNs foram obtidos de 86 pacientes durante a cirurgia. PNs eosinofílicos e não eosinofílicos foram diferenciados segundo os resultados imunoistoquímicos de cada amostra. As características histológicas e de expressão de mRNAs e de proteínas foram comparadas entre os dois grupos. Resultados Em PNs eosinofílicos, observamos uma expressão significativamente maior dos mRNAs e proteínas GATA-3, IL-5, IL-4 e IL-13. Nos PNs não eosinofílicos, aumentou a expressão dos mRNAs e das proteínas IL-17, IL-23 e RORc. Nos testes imunoistoquímicos, observamos maior número de mastócitos e menor número de neutrófilos nos PNs eosinofílicos, em comparação com PNs não eosinofílicos. Os pacientes com PNs eosinofílicos obtiveram escores de sintomas mais graves vs. PNs não eosinofílicos. Conclusão Demonstramos, pela primeira vez, uma reação Th2 predominante em PNs eosinofílicos e uma reação Th17 predominante em PNs não eosinofílicos. Nosso estudo pode proporcionar novas estratégias terapêuticas para a rinossinusite crônica.
Descritores: Sinusite/imunologia
Rinite/imunologia
Pólipos Nasais/imunologia
Eosinófilos/imunologia
-Sinusite/complicações
Fatores de Transcrição
Índice de Gravidade de Doença
RNA Mensageiro/metabolismo
Imuno-Histoquímica
Rinite/complicações
Pólipos Nasais/complicações
Pólipos Nasais/metabolismo
Pólipos Nasais/patologia
Doença Crônica
Citocinas/imunologia
Linfócitos T Auxiliares-Indutores/imunologia
Eosinofilia/complicações
Eosinofilia/metabolismo
Eosinofilia/patologia
Reação em Cadeia da Polimerase em Tempo Real
Limites: Humanos
Masculino
Feminino
Adulto
Tipo de Publ: Estudo Comparativo
Responsável: BR1.1 - BIREME


  4 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: lil-623591
Autor: Borba, Enri S; Sterin-Borba, Leonor.
Título: Immunosuppressor response triggers by the interaction of lymphocytes with chagasic IgG
Fonte: Mem. Inst. Oswaldo Cruz;83(supl.1):280-283, Nov. 1988. graf.
Idioma: en.
Conferência: Apresentado em: Annual Meeting on Basic Research in Chagas's disease, 15, Apresentado em: Meeting of the Brazilian Society of Protozoology4, Caxambu, 7-10 Nov. 1988.
Descritores: Imunoglobulina G/imunologia
Receptores Adrenérgicos beta/fisiologia
Doença de Chagas/imunologia
-Receptores Colinérgicos
Linfócitos T Auxiliares-Indutores
Responsável: BR1.1 - BIREME


  5 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: lil-623588
Autor: Coffman, Robert.
Título: Immunoregulation by Helper T cell subsets
Fonte: Mem. Inst. Oswaldo Cruz;83(supl.1):264-267, Nov. 1988.
Idioma: en.
Conferência: Apresentado em: Annual Meeting on Basic Research in Chagas's disease, 15, Apresentado em: Meeting of the Brazilian Society of Protozoology4, Caxambu, 7-10 Nov. 1988.
Descritores: Linfócitos T Auxiliares-Indutores
Leishmaniose Visceral/imunologia
-Linfócitos T
Limites: Animais
Camundongos
Responsável: BR1.1 - BIREME


  6 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: biblio-839309
Autor: Xiao, Y; Deng, T; Shang, Z; Wang, D.
Título: Adiponectin inhibits oxidization-induced differentiation of T helper cells through inhibiting costimulatory CD40 and CD80
Fonte: Braz. j. med. biol. res = Rev. bras. pesqui. méd. biol;50(6):e6227, 2017. graf.
Idioma: en.
Projeto: Natural Science Foundation of Hubei Province.
Resumo: Adiponectin is a multifunctional adipokine that has several oligomeric forms in the blood stream, which broadly regulates innate and acquired immunity. Therefore, in this study, we aimed to observe the differentiation of T helper (Th) cells and expression of costimulatory signaling molecules affected by adiponectin. The mRNA and protein expression levels of adiponectin and its receptors in oxidized low density lipoprotein cholesterol-treated endothelial cells were assayed by real time PCR and immunofluorescence. The endothelial cells were then treated with adiponectin with or without adipoR1 or adipoR2 siRNA and co-cultured with T lymphocytes. The distribution of Th1, Th2 and Th17 subsets were assayed by flow cytometry. The effects of adiponectin on costimulatory signaling molecules HLA-DR, CD80, CD86 and CD 40 was also assayed by flow cytometry. The results showed that endothelial cells expressed adiponectin and its receptor adipoR1 and adipoR2, but not T-cadherin. Adiponectin suppressed Th1 and Th17 differentiation through adipoR1 receptor, contributed to the inhibition of CD80 and CD40, and inhibited differentiation of Th1 and Th17 by inhibiting antigen presenting action.
Descritores: Adiponectina/metabolismo
Antígeno B7-1/metabolismo
Antígenos CD40/metabolismo
Linfócitos T Auxiliares-Indutores/efeitos dos fármacos
-Adiponectina/genética
Adiponectina/farmacologia
Diferenciação Celular
Células Cultivadas
Células Endoteliais/efeitos dos fármacos
Células Endoteliais/metabolismo
Antígenos HLA-DR/metabolismo
Células Endoteliais da Veia Umbilical Humana/citologia
Lipoproteínas LDL/farmacologia
Receptores de Adiponectina/efeitos dos fármacos
Receptores de Adiponectina/metabolismo
Linfócitos T Auxiliares-Indutores/citologia
Linfócitos T Auxiliares-Indutores/metabolismo
Limites: Humanos
Recém-Nascido
Adulto
Responsável: BR1.1 - BIREME


  7 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: lil-752428
Autor: SILVA, Nora; ABUSLEME, Loreto; BRAVO, Denisse; DUTZAN, Nicolás; GARCIA-SESNICH, Jocelyn; VERNAL, Rolando; HERNÁNDEZ, Marcela; GAMONAL, Jorge.
Título: Host response mechanisms in periodontal diseases
Fonte: J. appl. oral sci;23(3):329-355, May-Jun/2015. graf.
Idioma: en.
Projeto: Scientific and Technologic Investigation Resource; . Scientific and Technologic Investigation Resource.
Resumo: Periodontal diseases usually refer to common inflammatory disorders known as gingivitis and periodontitis, which are caused by a pathogenic microbiota in the subgingival biofilm, including Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, Tannerella forsythia and Treponema denticola that trigger innate, inflammatory, and adaptive immune responses. These processes result in the destruction of the tissues surrounding and supporting the teeth, and eventually in tissue, bone and finally, tooth loss. The innate immune response constitutes a homeostatic system, which is the first line of defense, and is able to recognize invading microorganisms as non-self, triggering immune responses to eliminate them. In addition to the innate immunity, adaptive immunity cells and characteristic cytokines have been described as important players in the periodontal disease pathogenesis scenario, with a special attention to CD4+ T-cells (T-helper cells). Interestingly, the T cell-mediated adaptive immunity development is highly dependent on innate immunity-associated antigen presenting cells, which after antigen capture undergo into a maturation process and migrate towards the lymph nodes, where they produce distinct patterns of cytokines that will contribute to the subsequent polarization and activation of specific T CD4+ lymphocytes. Skeletal homeostasis depends on a dynamic balance between the activities of the bone-forming osteoblasts (OBLs) and bone-resorbing osteoclasts (OCLs). This balance is tightly controlled by various regulatory systems, such as the endocrine system, and is influenced by the immune system, an osteoimmunological regulation depending on lymphocyte- and macrophage-derived cytokines. All these cytokines and inflammatory mediators are capable of acting alone or in concert, to stimulate periodontal breakdown and collagen destruction via tissue-derived matrix metalloproteinases, a characterization of the progression of periodontitis as a stage that presents a significantly host immune and inflammatory response to the microbial challenge that determine of susceptibility to develop the destructive/progressive periodontitis under the influence of multiple behavioral, environmental and genetic factors.
Descritores: Citocinas/imunologia
Doenças Periodontais/imunologia
Linfócitos T Auxiliares-Indutores/imunologia
-Imunidade Adaptativa
Ilustração Médica
Metaloproteinases da Matriz/imunologia
Doenças Periodontais/etiologia
Limites: Humanos
Tipo de Publ: Research Support, Non-U.S. Gov't
Revisão
Responsável: BR1.1 - BIREME


  8 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: lil-747107
Autor: Yüksel, Arif; Bilgir, Ferda; Bilgir, Oktay; Calan, Mehmet; Bozkaya, Giray.
Título: Increased circulating macrophage migration inhibitory factor levels are associated with coronary artery disease
Fonte: Clinics;70(3):169-172, 03/2015. tab.
Idioma: en.
Resumo: BACKGROUND: To evaluate the macrophage migration inhibitory factor and E-selectin levels in patients with acute coronary syndrome. MATERIALS/METHODS: We examined the plasma migration inhibitory factor and E-selectin levels in 87 patients who presented with chest pain at our hospital. The patients were classified into two groups according to their cardiac status. Sixty-five patients had acute myocardial infarction, and 22 patients had non-cardiac chest pain (non-coronary disease). We designated the latter group of patients as the control group. The patients who presented with acute myocardial infarction were further divided into two subgroups: ST-elevated myocardial infarction (n = 30) and non-ST elevated myocardial infarction (n = 35). RESULTS: We found higher plasma migration inhibitory factor levels in both acute myocardial infarction subgroups than in the control group. However, the E-selectin levels were similar between the acute myocardial infarction and control patients. In addition, we did not find a significant difference in the plasma migration inhibitory factor levels between the ST elevated myocardial infarction and NST-elevated myocardial infarction subgroups. DISCUSSION: The circulating concentrations of migration inhibitory factor were significantly increased in acute myocardial infarction patients, whereas the soluble E-selectin levels were similar between acute myocardial infarction patients and control subjects. Our results suggest that migration inhibitory factor may play a role in the atherosclerotic process. .
Descritores: /metabolismo
CHEMOKINE CCLABORTION APPLICANTS/metabolismo
Interferon gama/metabolismo
Neoplasias Mamárias Animais/imunologia
Esferoides Celulares/imunologia
Linfócitos T Citotóxicos/metabolismo
Linfócitos T Auxiliares-Indutores/metabolismo
-Alginatos
Antígenos de Neoplasias/imunologia
Antígenos de Neoplasias/metabolismo
Linhagem Celular Tumoral
Movimento Celular
Quitosana
/genética
CHEMOKINE CCLABORTION APPLICANTS/genética
/imunologia
CHEMOKINE CCLABORTION APPLICANTS/imunologia
Ácido Glucurônico
Granzimas/metabolismo
Ácidos Hexurônicos
Imunidade Celular
Interferon gama/genética
Interferon gama/imunologia
Neoplasias Mamárias Animais/genética
Neoplasias Mamárias Animais/metabolismo
Neoplasias Mamárias Animais/patologia
Esferoides Celulares/metabolismo
Esferoides Celulares/patologia
Linfócitos T Citotóxicos/imunologia
Linfócitos T Auxiliares-Indutores/imunologia
Microambiente Tumoral
Limites: Animais
Feminino
Camundongos
Tipo de Publ: Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Research Support, U.S. Gov't, Non-P.H.S.
Responsável: BR1.1 - BIREME


  9 / 45 LILACS  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: lil-718287
Autor: ARAUJO-PIRES, Ana Claudia; FRANCISCONI, Carolina Favaro; BIGUETTI, Claudia Cristina; CAVALLA, Franco; ARANHA, Andreza Maria Fabio; LETRA, Ariadne; TROMBONE, Ana Paula Favaro; FAVERI, Marcelo; SILVA, Renato Menezes; GARLET, Gustavo Pompermaier.
Título: Simultaneous analysis of T helper subsets (Th1, Th2, Th9, Th17, Th22, Tfh, Tr1 and Tregs) markers expression in periapical lesions reveals multiple cytokine clusters accountable for lesions activity and inactivity status
Fonte: J. appl. oral sci;22(4):336-346, Jul-Aug/2014. tab, graf.
Idioma: en.
Projeto: FAPESP; . CNPq.
Resumo: Previous studies demonstrate that the balance between pro- and anti-inflammatory mediators determines the stable or progressive nature of periapical granulomas by modulating the balance of the osteoclastogenic factor RANKL and its antagonist OPG. However, the cytokine networks operating in the development of periapical lesions are quite more complex than what the simple pro- versus anti-inflammatory mediators' paradigm suggests. Here we simultaneously investigated the patterns of Th1, Th2, Th9, Th17, Th22, Thf, Tr1 and Tregs cytokines/markers expression in human periapical granulomas. Methods: The expression of TNF-α, IFN-γ, IL-17A, IL23, IL21, IL-33, IL-10, IL-4, IL-9, IL-22, FOXp3 markers (via RealTimePCR array) was accessed in active/progressive (N=40) versus inactive/stable (N=70) periapical granulomas (as determined by RANKL/OPG expression ratio), and also to compare these samples with a panel of control specimens (N=26). A cluster analysis of 13 cytokine levels was performed to examine possible clustering between the cytokines in a total of 110 granulomas. Results: The expression of all target cytokines was higher in the granulomas than in control samples. TNF-α, IFN-γ, IL-17A and IL-21 mRNA levels were significantly higher in active granulomas, while in inactive lesions the expression levels of IL-4, IL-9, IL-10, IL-22 and FOXp3 were higher than in active granulomas. Five clusters were identified in inactive lesion groups, being the variance in the expression levels of IL-17, IL-10, FOXp3, IFN-γ, IL-9, IL-33 and IL-4 statistically significant (KW p<0.05). Three clusters were identified in active lesions, being the variance in the expression levels of IL-22, IL-10, IFN-γ, IL-17, IL-33, FOXp3, IL-21 and RANKL statistically significant (KW p<0.05). Conclusion: There is a clear dichotomy in the profile of cytokine expression in inactive and active periapical lesions. While the widespread ...
Descritores: Citocinas/análise
Granuloma Periapical/patologia
Subpopulações de Linfócitos T/metabolismo
Linfócitos T Auxiliares-Indutores/metabolismo
-Análise de Variância
Biomarcadores/análise
Doença Crônica
Citocinas/imunologia
Granuloma Periapical/imunologia
Reação em Cadeia da Polimerase em Tempo Real
Valores de Referência
Estatísticas não Paramétricas
Subpopulações de Linfócitos T/imunologia
Linfócitos T Auxiliares-Indutores/imunologia
Limites: Adulto
Feminino
Humanos
Masculino
Pessoa de Meia-Idade
Adulto Jovem
Tipo de Publ: Research Support, Non-U.S. Gov't
Responsável: BR1.1 - BIREME


  10 / 45 LILACS  
              first record previous record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Brasil
Texto completo
Id: lil-709444
Autor: Tossige-Gomes, R.; Ottone, V.O.; Oliveira, P.N.; Viana, D.J.S.; Araújo, T.L.; Gripp, F.J.; Rocha-Vieira, E..
Título: Leukocytosis, muscle damage and increased lymphocyte proliferative response after an adventure sprint race
Fonte: Braz. j. med. biol. res = Rev. bras. pesqui. méd. biol;47(6):492-498, 06/2014. tab, graf.
Idioma: en.
Projeto: FAPEMIG.
Resumo: The effect of an adventure sprint race (ASR) on T-cell proliferation, leukocyte count and muscle damage was evaluated. Seven young male runners completed an ASR in the region of Serra do Espinhaço, Brazil. The race induced a strong leukocytosis (6.22±2.04×103 cells/mm3 before vs 14.81±3.53×103 cells/mm3 after the race), marked by a significant increase of neutrophils and monocytes (P<0.05), but not total lymphocytes, CD3+CD4+ or CD3+CD8+ cells. However, the T-cell proliferative response to mitogenic stimulation was increased (P=0.025) after the race, which contradicted our hypothesis that ASR, as a high-demand competition, would inhibit T-cell proliferation. A positive correlation (P=0.03, r=0.79) was observed between the proliferative response of lymphocytes after the race and the time to complete the race, suggesting that the proliferative response was dependent on exercise intensity. Muscle damage was evident after the race by increased serum levels of aspartate amino transferase (24.99±8.30 vs 50.61±15.76 U/L, P=0.003). The results suggest that humoral factors and substances released by damaged muscle may be responsible for lymphocyte activation, which may be involved in muscle recovery and repair.
Descritores: Proliferação de Células/fisiologia
Leucocitose/imunologia
Músculo Esquelético/lesões
Resistência Física/imunologia
Corrida/lesões
Linfócitos T/imunologia
-Alanina Transaminase/sangue
Aspartato Aminotransferases/sangue
Citometria de Fluxo
Imunossupressão
Contagem de Leucócitos
Leucocitose/etiologia
Monócitos/imunologia
Músculo Esquelético/imunologia
Neutrófilos/imunologia
Resistência Física/fisiologia
Corrida/fisiologia
Linfócitos T Citotóxicos/fisiologia
Linfócitos T Auxiliares-Indutores/fisiologia
Fatores de Tempo
Limites: Adulto
Humanos
Masculino
Tipo de Publ: Research Support, Non-U.S. Gov't
Responsável: BR1.1 - BIREME



página 1 de 5 ir para página              
   


Refinar a pesquisa
  Base de dados : Formulário avançado   

    Pesquisar no campo  
1  
2
3
 
           



Search engine: iAH v2.6 powered by WWWISIS

BIREME/OPAS/OMS - Centro Latino-Americano e do Caribe de Informação em Ciências da Saúde