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Id: lil-708869
Autor: González-García, Mauricio; Chamorro, Julia; Jaramillo, Claudia; Casas, Alejandro; Maldonado, Darío.
Título: Sobrevida de pacientes con fibrosis pulmonar idiopática a la altura de Bogotá (2640 m) / Survival of patients with idiopathic pulmonary fibrosis at the altitude of Bogota (2640 m)
Fonte: Acta méd. colomb;39(1):15-20, ene.-mar. 2014. ilus, tab.
Idioma: es.
Resumo: Resumen Introducción: la sobrevida promedio de pacientes con fibrosis pulmonar idiopática (FPI) anivel del mar es de dos a tres años. Desconocemos su comportamiento en Bogotá, ciudad situadaa gran altura (2640 metros), donde hay mayor hipoxemia, factor asociado en la literatura con malpronóstico. El objetivo del estudio es describir, en una cohorte de pacientes con FPI, la sobreviday las características clínicas y funcionales en el momento del diagnóstico. Material y métodos: pacientes con diagnóstico de FPI confirmado por biopsia o por criteriosclínicos, radiológicos y funcionales. Se utilizó el análisis de sobrevida de Kaplan Meier y la prueba log rank. Resultados: cuarenta pacientes, 50% mujeres, con edad de 59.1±13 años y biopsia en52.5%. Al diagnóstico, PaO2: 48.5±11.2 mmHg, CVF: 61.5±16.8 %, CPT: 66.1±11.7 %, D LCO: 39.5±12.4%. La sobrevida desde el diagnóstico fue 42 meses (IC 95% 25.3-58.7 meses) ydesde el inicio de los síntomas 50 meses (IC 95% 40.3-59.7 meses). No hubo diferencias enla sobrevida por sexo, historia de tabaquismo, forma de diagnóstico (biopsia o no biopsia),variables gasométricas y de función pulmonar. Hubo una menor sobrevida en mayores de 60años (23 vs. 72 meses, p=0.03). Conclusiones: la sobrevida de pacientes con FPI en Bogotá con hipoxemia significativa aldiagnóstico, fue similar a la descrita en estudios a nivel del mar. La edad mayor de 60 años se relacionó con mal pronóstico. (Acta Med Colomb 2014; 39: 15-20).

Abstract Introduction: the mean survival of patients with idiopathic pulmonary fibrosis (IPF) at sea level is two to three years. We don't know its behavior in Bogotá, a city at high altitude (2640 meters), where there is greater hypoxemia, factor associated in the literature with poor prognosis. The objective of the study is to describe, in a cohort of patients with IPF survival and clinical and functional characteristics at the time of diagnosis. Methods: patients diagnosed with IPF confirmed by biopsy or by clinical, radiological and functional criteria. Survival analysis of Kaplan Meier and log rank test was utilized. Results: forty patients , 50% women with age 59.1 ± 13 years. 52.5% had biopsy . At diagnosis, PaO2: 48.5 ± 11.2 mmHg, FVC: 61.5 ± 16.8%, CPT: 66.1 ± 11.7%, DLCO: 39.5 ± 12.4%. Survival from diagnosis was 42 months (95% CI 25.3-58.7 months) and from the onset of symptoms 50 months (95% CI 40.3-59.7 months). There was no difference in survival by sex, smoking history, form of diagnosis (biopsy or not biopsy), blood gas and pulmonary function variables. There was a lower survival in patients older than 60 years (23 vs. 72 months, p = 0.03). Conclusions: the survival of patients with IPF in Bogota with significant hypoxemia at diagnosis was similar to that described in studies at sea level. Age older than 60 years was associated with poor prognosis. (Acta Med Colomb 2014; 39: 15-20).
Descritores: Sobrevida
Fibrose Pulmonar Idiopática
-Altitude
Hipóxia
Limites: Humanos
Masculino
Feminino
Pessoa de Meia-Idade
Responsável: CO70 - Asociación Colombiana de Medicina Interna


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Id: lil-734936
Autor: Bastidas, Alirio R; Giraldo, Luis Fernando; Benavides, Mateo; García, Rafael.
Título: Respiración de Cheyne-Stokes y bruxismo severo documentada por polisomnografía que mejora con la administración de oxígeno a 1 L/min / Cheyne-Stokes respiration and severe bruxism documented by polysomnography that improves with administration of oxygen at 1 L/min
Fonte: Acta méd. colomb;39(4):388-392, oct.-dic. 2014. ilus.
Idioma: es.
Resumo: Se presenta el caso de una mujer de 59 años con diagnóstico de encefalopatía anoxicoisquémica, quien se encuentra en estado vegetativo persistente y epilepsia secundaria de difícil control, quien en el transcurso de su evolución clínica desarrolla apnea central del sueño con índice de apnea hipopnea (IAH) de 87.86/h con patrón de respiración de Cheyne-Stokes (RCS) y bruxismo severo documentado mediante polisomnografía, alteraciones que mejoraron tras la administración de oxígeno por cánula nasal a 1 L/min. La asociación de bruxismo con respiración de Cheyne-Stokes y la respuesta simultánea y completa de las dos alteraciones a la administración de oxígeno suplementario no ha sido reportada previamente.

The case of a 59 year old female diagnosed with anoxic-ischemic encephalopathy, who is in a persistent vegetative state and secondary epilepsy difficult to control, who in the course of her clinical evolution develops central sleep apnea with apnea hypopnea index (AHI) of 87.86 / h with Cheyne-Stokes pattern and severe bruxism documented by polysomnography, alterations that improved after administration of oxygen by nasal cannula at 1 L / min., is presented. The association of bruxism with Cheyne-Stokes respiration and the simultaneous and complete response of the two alterations to the administration of supplemental oxygen has not been reported previously.
Descritores: Respiração de Cheyne-Stokes
-Apneia
Bruxismo
Polissonografia
Insuficiência Cardíaca
Hipóxia
Limites: Humanos
Feminino
Pessoa de Meia-Idade
Tipo de Publ: Relatos de Casos
Responsável: CO70 - Asociación Colombiana de Medicina Interna


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Id: lil-791068
Autor: Calderón, Julio; Carvajal, Carlos; Giraldo, Nelson; Pacheco, Carlos; Gómez, Camilo; Gallego, Diego; Jaimes, Fabián.
Título: Mortalidad y factores asociados en pacientes con síndrome de dificultad respiratoria agudo (SDRA) en un hospital universitario / Mortality and associated factors in patients with acute respiratory distress syndrome (ARDS) in a university hospital
Fonte: Acta méd. colomb;40(4):305-309, oct.-dic. 2015. ilus, tab.
Idioma: es.
Resumo: Introducción: el síndrome de dificultad respiratorio agudo (SDRA) es la manifestación más grave de compromiso pulmonar agudo. Actualmente no hay datos disponibles para documentar los factores asociados con la mortalidad en nuestro medio. Diseño del estudio: estudio de cohorte retrospectiva. Objetivo: describir los factores asociados a la mortalidad en pacientes adultos hospitalizados en la unidad de cuidado crítico con SDRA entre enero 2007 y diciembre de 2011. Métodos: se recolectaron datos con respecto a causas de SDRA, estancia hospitalaria y en la UCI, gravedad de la enfermedad crítica y el estado vital al egreso. Se realizó un análisis de regresión logística para determinar los factores asociados de manera independiente con la mortalidad hospitalaria. Resultados: se analizaron 141 pacientes con SDRA, la mediana de edad fue 44 años y 62.9% fueron hombres. La primera causa de SDRA fue el choque séptico de origen pulmonar en 49.6% y el APACHE II tuvo una mediana de 18. La mediana de la PaO2/FIO2 al inicio del SDRA fue 91.5 y a las 72 horas fue 125. La mediana de PEEP requerido al inicio de la ventilación mecánica fue de 10 cmH2O y a las 72 horas fue de 12. La estancia en la UCI fue de 13 días y la mortalidad hospitalaria fue del 54%. Los factores asociados con mortalidad fueron el choque de origen pulmonar (OR = 2.45; IC 95% = 1.04-5.77) y el puntaje APACHE II (OR = 1.05 por cada punto; IC 95% = 1.003-1.1). El nivel de PEEP igual o menor en las primeras 72 horas se comportó como un factor protector (OR = 0.36; IC 95% = 0.16-0.82). Conclusiones: el SDRA tiene una alta mortalidad en nuestro medio y los factores más fuertemente asociados con dicha mortalidad son dependientes de la enfermedad de base y de la intensidad de la respuesta biológica a la misma. (Acta Med Colomb 2015; 40 305-309).

Introduction: acute respiratory distress syndrome (ARDS) is the most serious manifestation of acute pulmonary compromise. Currently no data are available to document the factors associated with mortality in our environment. Study Design: retrospective cohort study. Objective: To describe the factors associated with mortality in adult patients hospitalized in critical care unit with ARDS from January 2007 to December 2011. Methods: Data were collected with regard to causes of ARDS, hospital and ICU stay, critical illness severity and vital status at discharge. Logistic regression analysis was performed to determine factors independently associated with hospital mortality. Results: 141 patients with ARDS were analyzed; the median age was 44 years and 62.9% were men. The first cause of ARDS was septic shock of pulmonary origin in 49.6% and APACHE II had a median of 18. The median PaO2/FIO2 at the beginning of ARDS was 91.5 and at 72 hours was 125. The median PEEP required at the initiation of mechanical ventilation was 10 cmH2O and at 72 hours 12. The ICU stay was 13 days and hospital mortality was 54%. Factors associated with mortality were the shock of pulmonary origin (OR = 2.45; 95% CI = 1.04-5.77) and APACHE II (OR = 1.05 per point, 95% CI = 1.003-1.1). PEEP level equal to or less within 72 hours acted as a protective factor (OR = 0.36; 95% CI = 0.16-0.82). Conclusions: ARDS has a high mortality in our environment and the factors most strongly associated with this mortality are dependent on the underlying disease and the intensity of biological response to it. (Acta Med Colomb 2015; 40 305-309).
Descritores: Síndrome do Desconforto Respiratório do Recém-Nascido
-Respiração Artificial
Mecânica Respiratória
Mortalidade
Lesão Pulmonar Aguda
Hipóxia
Limites: Humanos
Masculino
Feminino
Adulto
Responsável: CO70 - Asociación Colombiana de Medicina Interna


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Texto completo SciELO Brasil
Agostinho, Angelo Antonio
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Id: biblio-1279497
Autor: Agostinho, Angelo Antonio; Alves, Diego Correa; Gomes, Luiz Carlos; Dias, Rosa Maria; Petrere Jr, Miguel; Pelicice, Fernando Mayer.
Título: Fish die-off in river and reservoir: A review on anoxia and gas supersaturation
Fonte: Neotrop. ichthyol;19(3):e210037, 2021. tab, graf, ilus.
Idioma: en.
Resumo: Albeit massive fish mortality has an extraordinary visual impact and is certainly a fatality, we still have rudimentary understanding on how addressing this problem in the Neotropical region. The processes that lead to fish die-off events are complex and sometimes ephemeral, which can lead to incorrect diagnosis. In this review, we discuss these events in Neotropical freshwaters, both in areas impacted by dams and natural environment, with a focus on deaths mediated by the lack of oxygen (anoxia) or the excess of dissolved gases (gas supersaturation). We examine the available knowledge about the related mechanisms, lethal thresholds for dissolved oxygen (DO) and total dissolved gases (TDG) for fish, and the sequelae of gas bubble disease (GBD). An assessment of the main mortality events in Brazil in the last 10 years is also presented, as well as the best practices for monitoring, prevention, and mitigation. Finally, it is concluded that the proliferation of hydroelectric plants in the Neotropical region might contribute to the expansion of these events and, consequently, increasing of impacts on fish conservation. We consider urgent the inclusion of this topic in the licensing processes for new hydropower projects by the environmental agencies.(AU)

Embora mortandades massivas de peixes tenham impacto visual extraordinário e sejam certamente uma fatalidade, estamos ainda engatinhando no entendimento e na solução desse problema. Os processos que levam aos eventos de mortes de peixes são complexos e algumas vezes efêmeros, o que explica diagnoses incorretas. Nessa revisão nós discutimos esses eventos em bacias hidrográficas neotropicais, tanto em barragens como em ambientes naturais, porém com o foco nas mortes por falta de oxigênio (anoxia) ou excesso de gases dissolvidos (supersaturação gasosa). Em particular são examinados o conhecimento disponível acerca dos processos que levam a essas condições, os limiares letais de oxigênio dissolvido (OD) e gases totais dissolvidos (GTD) para os peixes e as sequelas da doença das bolhas de gás (DBG). Uma avaliação dos principais eventos de mortandade que ocorreram no Brasil nos últimos 10 anos é também apresentada, bem como discutidas as melhores práticas para o monitoramento, prevenção e mitigação. Conclui-se, finalmente, que a proliferação de hidrelétricas deve contribuir com a expansão desses eventos e consequentemente com mais um fator de ameaça à biodiversidade. Consideramos urgente a inclusão desse tema nos processos de licenciamento de novos empreendimentos pelo órgão ambiental.(AU)
Descritores: Barragens
Mortalidade
Peixes/fisiologia
Hipóxia/mortalidade
-Oxigênio Dissolvido
Reservatórios de Água
Biodiversidade
Limites: Animais
Responsável: BR68.1 - Biblioteca Virginie Buff D'Ápice


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Texto completo SciELO Brasil
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Id: biblio-974926
Autor: Geng, Wujun; Jia, Danyu; Wang, Yichuan; Jin, Shenhui; Ren, Yelong; Liang, Dongdong; Zheng, Aote; Tang, Hongli; Basharat, Zarrin; Zimmer, Vincent; Stock, Simon; Zippi, Maddalena; Hong, Wandong.
Título: A prediction model for hypoxemia during routine sedation for gastrointestinal endoscopy
Fonte: Clinics;73:e513, 2018. tab, graf.
Idioma: en.
Resumo: OBJECTIVES: The current study was designed to assess the clinical predictors of hypoxemia and to develop a multivariable, predictive model for hypoxemia during routine gastrointestinal endoscopy. METHODS: In total, 308 patients were enrolled in the analysis. Demographic data, concurrent chronic disease information, anesthetic dose and Modified Observer's Assessment of Alertness/Sedation (MOAA/S) scores were collected and analyzed statistically. RESULTS: Multivariate logistic regression indicated that age (OR: 1.04; 95%CI 1.01-1.08), body mass index (BMI) (OR: 1.12; 95%CI: 1.02-1.21) and habitual snoring (OR: 3.71; 95%CI: 1.62-8.48) were independently associated with hypoxemia. A logistic regression function (LR model) was developed to predict hypoxemia considering the parameters of -7.73+0.04 age (years), +0.11 BMI, and +1.31 habitual snoring (yes or no). The area under the receiver operating characteristic (ROC) curve for the LR model was 0.76. CONCLUSIONS: The LR model, consisting of age, BMI and habitual snoring, was a useful predictor of hypoxemia during routine sedation for gastrointestinal endoscopy.
Descritores: Sedação Consciente/efeitos adversos
Endoscopia Gastrointestinal/efeitos adversos
Hipóxia/etiologia
-Valor Preditivo dos Testes
Estudos Prospectivos
Curva ROC
Endoscopia Gastrointestinal/métodos
Modelos Teóricos
Limites: Humanos
Masculino
Feminino
Pessoa de Meia-Idade
Responsável: BR1.1 - BIREME


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Texto completo SciELO Brasil
Texto completo
Id: biblio-1133469
Autor: Zhou, Xiaohui; Liu, Jing; Yang, Siyi; Su, Yanguang; Meng, Zhipeng; Hu, Yuqin.
Título: Ketamine ameliorates hypoxia-induced endothelial injury in human umbilical vein endothelial cells
Fonte: Clinics;75:e1865, 2020. graf.
Idioma: en.
Projeto: National Natural Science Foundation of China.
Resumo: OBJECTIVES: Hypoxia leads to endothelial cell inflammation, apoptosis, and damage, which plays an important role in the complications associated with ischemic cardiovascular disease. As an oxidoreductase, p66Shc plays an important role in the regulation of reactive oxygen species (ROS) production and apoptosis. Ketamine is widely used in clinics. This study was designed to assess the potential protective effect of ketamine against hypoxia-induced injury in human umbilical vein endothelial cells (HUVECs). Moreover, we explored the potential mechanism by which ketamine protected against hypoxia-induced endothelial injury. METHODS: The protective effects of ketamine against hypoxia-induced injury was assessed using cell viability and adhesion assays, quantitative polymerase chain reaction, and western blotting. RESULTS: Our data showed that hypoxia reduced HUVEC viability, increased the adhesion between HUVECs and monocytes, and upregulated the expression of endothelial adhesion molecules at the protein and mRNA levels. Moreover, hypoxia increased ROS accumulation and upregulated p66Shc expression. Furthermore, hypoxia downregulated sirt1 expression in HUVECs. Alternatively, ketamine was shown to reverse the hypoxia-mediated reduction of cell viability and increase in the adhesion between HUVECs and monocytes, ameliorate hypoxia-induced ROS accumulation, and suppress p66Shc expression. Moreover, EX527, a sirt1 inhibitor, reversed the protective effects of ketamine against the hypoxia-mediated reduction of cell viability and increase in adhesion between HUVECs and monocytes. CONCLUSION: Ketamine reduces hypoxia-induced p66Shc expression and attenuates ROS accumulation via upregulating sirt1 in HUVECs, thus attenuating hypoxia-induced endothelial cell inflammation and apoptosis.
Descritores: Espécies Reativas de Oxigênio/metabolismo
Apoptose/efeitos dos fármacos
Células Endoteliais da Veia Umbilical Humana/efeitos dos fármacos
Ketamina/farmacologia
Hipóxia
-Veias Umbilicais
Sobrevivência Celular
Estresse Oxidativo
Células Endoteliais da Veia Umbilical Humana/metabolismo
Proteína 1 de Transformação que Contém Domínio 2 de Homologia de Src
Limites: Humanos
Responsável: BR1.1 - BIREME


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Texto completo SciELO Chile
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Id: biblio-1124210
Autor: Sadeghi, Fatemeh; Kardar, Gholam Ali; Bolouri, Mohammad Reza; Nasri, Farzad; Sadri, Maryam; Falak, Reza.
Título: Overexpression of bHLH domain of HIF-1 failed to inhibit the HIF-1 transcriptional activity in hypoxia
Fonte: Biol. Res;53:25, 2020. tab, graf.
Idioma: en.
Projeto: Medical faculty, Iran University of Medical Sciences.
Resumo: BACKGROUND: Hypoxia inducible factor-1 (HIF-1) is considered as the most activated transcriptional factor in response to low oxygen level or hypoxia. HIF-1 binds the hypoxia response element (HRE) sequence in the promoter of different genes, mainly through the bHLH domain and activates the transcription of genes, especially those involved in angiogenesis and EMT. Considering the critical role of bHLH in binding HIF-1 to the HRE sequence, we hypothesized that bHLH could be a promising candidate to be targeted in hypoxia condition. METHODS: We inserted an inhibitory bHLH (ibHLH) domain in a pIRES2-EGFP vector and transfected HEK293T cells with either the control vector or the designed construct. The ibHLH domain consisted of bHLH domains of both HIF-1a and Arnt, capable of competing with HIF-1 in binding to HRE sequences. The transfected cells were then treated with 200 µM of cobalt chloride (CoCl2) for 48 h to induce hypoxia. Real-time PCR and western blot were performed to evaluate the effect of ibHLH on the genes and proteins involved in angiogenesis and EMT. RESULTS: Hypoxia was successfully induced in the HEK293T cell line as the gene expression of VEGF, vimentin, and ß-catenin were significantly increased after treatment of untransfected HEK293T cells with 200 µM CoCl2. The gene expression of VEGF, vimentin, and ß-catenin and protein level of ß-catenin were significantly decreased in the cells transfected with either control or ibHLH vectors in hypoxia. However, ibHLH failed to be effective on these genes and the protein level of ß-catenin, when compared to the control vector. We also observed that overexpression of ibHLH had more inhibitory effect on gene and protein expression of N-cadherin compared to the control vector. However, it was not statistically significant. CONCLUSION: bHLH has been reported to be an important domain involved in the DNA binding activity of HIF. However, we found that targeting this domain is not sufficient to inhibit the endogenous HIF-1 transcriptional activity. Further studies about the function of critical domains of HIF-1 are necessary for developing a specific HIF-1 inhibitor.
Descritores: Fatores de Transcrição Hélice-Alça-Hélice Básicos/metabolismo
Fator 1 Induzível por Hipóxia/metabolismo
Hipóxia/metabolismo
-Expressão Gênica
Ativação Transcricional/genética
Western Blotting
Fatores de Transcrição Hélice-Alça-Hélice Básicos/genética
Fator 1 Induzível por Hipóxia/genética
Células HEK293
Reação em Cadeia da Polimerase em Tempo Real
Hipóxia/genética
Limites: Humanos
Responsável: CL1.1 - Biblioteca Central


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Id: biblio-986850
Autor: Vizueth Martínez, Alberto; Moedano Álvarez, Erika Belem; Ramírez Morelos, Daniel; López Villeda, Juliette Areli; Ramírez Trejo, Diana.
Título: Sindrome de Atra / Atra syndrome
Fonte: Rev. pediatr. electrón;14(2):34-38, ago. 2017.
Idioma: es.
Resumo: La leucemia promielocítica aguda (APL) es el subtipo de leucemia mieloide aguda de mejor pronóstico en niños. Su incidencia es menor a 10%. Desde el punto de vista citogenético se observa una translocación t (15;17). En la terapéutica la incorporación del ácido transretinoico ha logrado altas tasas de remisión completa debido a la rápida desaparición de la coagulopatía y, en consecuencia, disminución de la tasa de recaídas, en comparación con el tratamiento de monoterapia. En general es un fármaco bien tolerado pero puede tener reacciones adversas; el más grave es el síndrome de ácido transretinoico (ATRA), potencialmente mortal. Las manifestaciones clínicas son: fiebre, ganancia de peso, infiltrados pulmonares, síndrome de dificultad respiratoria, derrame pleural o pericárdico, hipotensión, insuficiencia hepática y renal. El tratamiento es con suspensión del ácido transretinoico, medidas de apoyo y altas dosis de esteroides. Se presenta un caso clínico del hospital del Niño DIF con APL y Síndrome de ATRA.

The leukemia promyelocytic acute (APL) is the subtype of leukemia myeloid acute of better prognosis in children. Its incidence is less than 10%. From the point of view cytogenetic is observed a translocation t (15; 17). The addition of the acid transretinoico has achieved high rates of complete remission because of the rapid disappearance of the coagulopathy and, consequently, decrease in the rate of relapses, compared with monotherapy treatment. In general it is a well-tolerated drug but can have adverse reactions; the most serious is transretinoico acid (ATRA), potentially fatal syndrome. The manifestations are: fever, weight gain, pulmonary infiltrates, syndrome of shortness of breath, hypotension, pleural effusion or pericardial, hepatic and renal insufficiency. The treatment is with suspension of the acid transretinoico, measures of support and high doses of steroids. It presents a case clinical of the Hospital del Niño DIF with APL and syndrome of ATRA.
Descritores: Tretinoína/efeitos adversos
Leucemia Promielocítica Aguda/tratamento farmacológico
Antineoplásicos/efeitos adversos
-Derrame Pleural/induzido quimicamente
Insuficiência Respiratória/induzido quimicamente
Síndrome
Evolução Fatal
Febre/induzido quimicamente
Hepatomegalia/induzido quimicamente
Hipóxia/induzido quimicamente
Limites: Humanos
Feminino
Pré-Escolar
Tipo de Publ: Relatos de Casos
Responsável: CL126.3 - Biblioteca Campus Oriente Peñalolén


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Id: biblio-968887
Autor: Rodríguez B, Jorge; Reyes N, Manuel; Jorquera C, Rodrigo.
Título: Oxigenoterapia en pediatría / Oxygen therapy in pediatrics
Fonte: Rev. pediatr. electrón;14(1):13-25, 2017. img..
Idioma: es.
Resumo: Cada año, cerca de 6 millones de niños mueren por enfermedades prevenibles o fácilmente tratadas. El 95% se produce en países subdesarrollados, siendo la neumonía la causa más prevalente muertes en menores de 5 años (18%). La hipoxemia es la causa de los decesos en estos individuos. La aplicación de O2 como medida terapéutica para tratar o prevenirla es una de las indicaciones más frecuente que debe realizar un clínico. Este escrito trata de integrar en forma muy resumida los conceptos básicos que involucran a la oxigeno terapia, cuándo aplicar O2, con que equipos y aditamentos más apropiados.

Around 6 million children died of preventable or easily treatable diseases each year. Ninety five per cent are produced in undeveloped countries, being pneumonia the leading cause of death of children of 5 years or less (18%) and hypoxemia is the cause of dead. Oxygen therapy to treat or prevent this, as a therapeutic measure, is one of the most commons indicationsthat must be performed by aclinician. The review summarized the basic concepts of oxygen administration, when and how, with which equipment, oxygen delivery devices, dangers and its precautions.
Descritores: Oxigenoterapia/instrumentação
Oxigenoterapia/métodos
Hipóxia/prevenção & controle
-Oxigênio/administração & dosagem
Limites: Humanos
Criança
Tipo de Publ: Artigo Clássico
Responsável: CL1.1 - Biblioteca Central


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Id: biblio-1182658
Autor: Perú. Ministerio de Salud. Instituto Materno Perinatal.
Título: Atención integral materno perinatal / Perinatal maternal integral attention.
Fonte: Lima; DISEÑO GRAFICA; 1999. 163 p. ilus.
Idioma: es.
Resumo: El presente documento constituye como instrumento de capacitación, con la finalidad de compartir nuestras experiencias con los numerosos profesionales que deben enfrentar problemas similares en sus establecimientos, en todo el ámbito nacional; para ello, se mantiene en el abordaje de los temas tratados, el enfoque integral de atención de la condición fetal o perinatal
Descritores: Hemorragia
Hipóxia
Mortalidade Infantil
Mortalidade Materna
Responsável: PE18.1 - Biblioteca Central
[{"text": "PE18.1 MS/IMP 0015"}]



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