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Pesquisa : A08.800.050.050.650 [Categoria DeCS]
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[PMID]:27770832
[Au] Autor:Bjorke B; Shoja-Taheri F; Kim M; Robinson GE; Fontelonga T; Kim KT; Song MR; Mastick GS
[Ad] Endereço:Department of Biology, University of Nevada, Reno, NV, 89557, USA.
[Ti] Título:Contralateral migration of oculomotor neurons is regulated by Slit/Robo signaling.
[So] Source:Neural Dev;11(1):18, 2016 10 22.
[Is] ISSN:1749-8104
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Oculomotor neurons develop initially like typical motor neurons, projecting axons out of the ventral midbrain to their ipsilateral targets, the extraocular muscles. However, in all vertebrates, after the oculomotor nerve (nIII) has reached the extraocular muscle primordia, the cell bodies that innervate the superior rectus migrate to join the contralateral nucleus. This motor neuron migration represents a unique strategy to form a contralateral motor projection. Whether migration is guided by diffusible cues remains unknown. METHODS: We examined the role of Slit chemorepellent signals in contralateral oculomotor migration by analyzing mutant mouse embryos. RESULTS: We found that the ventral midbrain expresses high levels of both Slit1 and 2, and that oculomotor neurons express the repellent Slit receptors Robo1 and Robo2. Therefore, Slit signals are in a position to influence the migration of oculomotor neurons. In Slit 1/2 or Robo1/2 double mutant embryos, motor neuron cell bodies migrated into the ventral midbrain on E10.5, three days prior to normal migration. These early migrating neurons had leading projections into and across the floor plate. In contrast to the double mutants, embryos which were mutant for single Slit or Robo genes did not have premature migration or outgrowth on E10.5, demonstrating a cooperative requirement of Slit1 and 2, as well as Robo1 and 2. To test how Slit/Robo midline repulsion is modulated, we found that the normal migration did not require the receptors Robo3 and CXCR4, or the chemoattractant, Netrin 1. The signal to initiate contralateral migration is likely autonomous to the midbrain because oculomotor neurons migrate in embryos that lack either nerve outgrowth or extraocular muscles, or in cultured midbrains that lacked peripheral tissue. CONCLUSION: Overall, our results demonstrate that a migratory subset of motor neurons respond to floor plate-derived Slit repulsion to properly control the timing of contralateral migration.
[Mh] Termos MeSH primário: Orientação de Axônios
Movimento Celular
Peptídeos e Proteínas de Sinalização Intercelular/fisiologia
Neurônios Motores/fisiologia
Proteínas do Tecido Nervoso/fisiologia
Nervo Oculomotor/crescimento & desenvolvimento
Receptores Imunológicos/fisiologia
[Mh] Termos MeSH secundário: Animais
Proteínas de Membrana/fisiologia
Mesencéfalo/fisiologia
Camundongos
Fatores de Crescimento Neural/fisiologia
Netrina-1
Receptores CXCR4/fisiologia
Transdução de Sinais
Proteínas Supressoras de Tumor/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (CXCR4 protein, mouse); 0 (Intercellular Signaling Peptides and Proteins); 0 (Membrane Proteins); 0 (Nerve Growth Factors); 0 (Nerve Tissue Proteins); 0 (Ntn1 protein, mouse); 0 (Receptors, CXCR4); 0 (Receptors, Immunologic); 0 (Robo2 protein, mouse); 0 (Robo3 protein, mouse); 0 (Slit homolog 2 protein); 0 (Slit1 protein, mouse); 0 (Tumor Suppressor Proteins); 0 (roundabout protein); 158651-98-0 (Netrin-1)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:171204
[Lr] Data última revisão:
171204
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161025
[St] Status:MEDLINE


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[PMID]:28534340
[Au] Autor:Kim JH; Hwang JM
[Ad] Endereço:Department of Radiology, Seoul National University Bundang Hospital, Seoul National University College of Medicine, Seongnam, Korea.
[Ti] Título:Imaging of Cranial Nerves III, IV, VI in Congenital Cranial Dysinnervation Disorders.
[So] Source:Korean J Ophthalmol;31(3):183-193, 2017 Jun.
[Is] ISSN:2092-9382
[Cp] País de publicação:Korea (South)
[La] Idioma:eng
[Ab] Resumo:Congenital cranial dysinnervation disorders are a group of diseases caused by abnormal development of cranial nerve nuclei or their axonal connections, resulting in aberrant innervation of the ocular and facial musculature. Its diagnosis could be facilitated by the development of high resolution thin-section magnetic resonance imaging. The purpose of this review is to describe the method to visualize cranial nerves III, IV, and VI and to present the imaging findings of congenital cranial dysinnervation disorders including congenital oculomotor nerve palsy, congenital trochlear nerve palsy, Duane retraction syndrome, Möbius syndrome, congenital fibrosis of the extraocular muscles, synergistic divergence, and synergistic convergence.
[Mh] Termos MeSH primário: Nervo Abducente/diagnóstico por imagem
Doenças dos Nervos Cranianos/diagnóstico
Síndrome da Retração Ocular/complicações
Imagem por Ressonância Magnética/métodos
Nervo Oculomotor/diagnóstico por imagem
Nervo Troclear/diagnóstico por imagem
[Mh] Termos MeSH secundário: Doenças dos Nervos Cranianos/etiologia
Síndrome da Retração Ocular/diagnóstico
Seres Humanos
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171012
[Lr] Data última revisão:
171012
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170524
[St] Status:MEDLINE
[do] DOI:10.3341/kjo.2017.0024


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[PMID]:28437527
[Au] Autor:Michalak SM; Whitman MC; Park JG; Tischfield MA; Nguyen EH; Engle EC
[Ad] Endereço:Department of Neurology, Boston Children's Hospital, Boston, Massachusetts, United States 2F. M. Kirby Neurobiology Center, Boston Children's Hospital, Boston, Massachusetts, United States 3Department of Neurology, Harvard Medical School, Boston, Massachusetts, United States 4University of North Car
[Ti] Título:Ocular Motor Nerve Development in the Presence and Absence of Extraocular Muscle.
[So] Source:Invest Ophthalmol Vis Sci;58(4):2388-2396, 2017 Apr 01.
[Is] ISSN:1552-5783
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Purpose: To spatially and temporally define ocular motor nerve development in the presence and absence of extraocular muscles (EOMs). Methods: Myf5cre mice, which in the homozygous state lack EOMs, were crossed to an IslMN:GFP reporter line to fluorescently label motor neuron cell bodies and axons. Embryonic day (E) 11.5 to E15.5 wild-type and Myf5cre/cre:IslMN:GFP whole mount embryos and dissected orbits were imaged by confocal microscopy to visualize the developing oculomotor, trochlear, and abducens nerves in the presence and absence of EOMs. E11.5 and E18.5 brainstems were serially sectioned and stained for Islet1 to determine the fate of ocular motor neurons. Results: At E11.5, all three ocular motor nerves in mutant embryos approached the orbit with a trajectory similar to that of wild-type. Subsequently, while wild-type nerves send terminal branches that contact target EOMs in a stereotypical pattern, the Myf5cre/cre ocular motor nerves failed to form terminal branches, regressed, and by E18.5 two-thirds of their corresponding motor neurons died. Comparisons between mutant and wild-type embryos revealed novel aspects of trochlear and oculomotor nerve development. Conclusions: We delineated mouse ocular motor nerve spatial and temporal development in unprecedented detail. Moreover, we found that EOMs are not necessary for initial outgrowth and guidance of ocular motor axons from the brainstem to the orbit but are required for their terminal branching and survival. These data suggest that intermediate targets in the mesenchyme provide cues necessary for appropriate targeting of ocular motor axons to the orbit, while EOM cues are responsible for terminal branching and motor neuron survival.
[Mh] Termos MeSH primário: Movimentos Oculares/fisiologia
Músculos Oculomotores/embriologia
Nervo Oculomotor/embriologia
[Mh] Termos MeSH secundário: Animais
Axônios/fisiologia
Camundongos
Microscopia Confocal
Modelos Animais
Neurônios Motores/fisiologia
Músculos Oculomotores/inervação
Nervo Oculomotor/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1707
[Cu] Atualização por classe:170719
[Lr] Data última revisão:
170719
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170425
[St] Status:MEDLINE
[do] DOI:10.1167/iovs.16-21268


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[PMID]:28296553
[Au] Autor:Bolat D; Yildiz D; Bahar S; Yürüker S; Kaymaz F; Ilgin C; Bozkurt EÜ; Karahan S; Sabanci SS
[Ad] Endereço:a Faculty of Veterinary Medicine, Department of Anatomy , Kirikkale University , Kirikkale.
[Ti] Título:A comparative study of oculomotor, trochlear and abducens nerves in Arabian foals.
[So] Source:Biotech Histochem;92(2):149-156, 2017.
[Is] ISSN:1473-7760
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:We investigated the microscopic structure of transverse sections of the oculomotor, trochlear and abducens nerves of Arabian foals using stereological methods. Bilateral nerve pairs from 2-month-old female Arabian foals were analyzed. The tissues were embedded in plastic blocks, then 1 µm thick sections were cut and stained with osmium tetroxide and methylene blue-azure II. Stereology was performed using light microscopy. Morphometry showed that the right and left pairs of nerves were similar. The transverse sectional areas of the oculomotor, trochlear and abducens nerves were 1.93 ± 0.19 mm , 0.32 ± 0.06 mm and 0.70 ± 0.08 mm , respectively. The oculomotor nerve exhibited a significantly greater number of myelinated axons (16755 ± 1279) and trochlear (2656 ± 494) and the abducens nerves (4468 ± 447). The ratio of the axon diameter to myelinated nerve fiber diameter was 0.58, 0.55 and 0.55 for the oculomotor, trochlear and abducens nerves, respectively. Of the three nerves studied, the abducens nerve exhibited the greatest nerve fiber area, myelin area, nerve and axon diameters, and myelin thickness. The ratio of small myelinated nerve fibers was greatest in the oculomotor nerve.
[Mh] Termos MeSH primário: Nervo Abducente/metabolismo
Axônios/metabolismo
Bainha de Mielina/metabolismo
Fibras Nervosas Mielinizadas/metabolismo
Nervo Oculomotor/metabolismo
[Mh] Termos MeSH secundário: Animais
Feminino
Cavalos
Microscopia/métodos
[Pt] Tipo de publicação:COMPARATIVE STUDY; JOURNAL ARTICLE
[Em] Mês de entrada:1703
[Cu] Atualização por classe:170330
[Lr] Data última revisão:
170330
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170316
[St] Status:MEDLINE
[do] DOI:10.1080/10520295.2017.1288926


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[PMID]:28153623
[Au] Autor:Jacquesson T; Frindel C; Cotton F
[Ad] Endereço:Department of Neurosurgery B, Skull Base Multi-disciplinary Unit, Neurological Hospital Pierre Wertheimer, Hospices Civils de Lyon, Lyon, France; Department of Anatomy, University of Lyon 1, Lyon, France; CREATIS Laboratory, CNRS UMR 5220 - INSERM U1044, Villeurbanne, France. Electronic address: timothee.jacquesson@neurochirurgie.fr.
[Ti] Título:Diffusion Tensor Imaging Tractography Detecting Isolated Oculomotor Nerve Damage After Traumatic Brain Injury.
[So] Source:World Neurosurg;100:707.e5-707.e7, 2017 Apr.
[Is] ISSN:1878-8769
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:A 24-year-old woman was hit by a bus and suffered an isolated complete oculomotor nerve palsy. Computed tomography scan did not show a skull base fracture. T2*-weighted magnetic resonance imaging revealed petechial cerebral hemorrhages sparing the brainstem. T2 constructive interference in steady state suggested a partial sectioning of the left oculomotor nerve just before entering the superior orbital fissure. Diffusion tensor imaging fiber tractography confirmed a sharp arrest of the left oculomotor nerve. This recent imaging technique could be of interest to assess white fiber damage and help make a diagnosis or prognosis.
[Mh] Termos MeSH primário: Lesões Encefálicas Traumáticas/complicações
Lesões Encefálicas Traumáticas/diagnóstico por imagem
Imagem de Tensor de Difusão
Traumatismos do Nervo Oculomotor/complicações
Traumatismos do Nervo Oculomotor/diagnóstico por imagem
Nervo Oculomotor/diagnóstico por imagem
[Mh] Termos MeSH secundário: Acidentes de Trânsito
Diagnóstico Diferencial
Imagem de Difusão por Ressonância Magnética
Feminino
Seres Humanos
Doenças do Nervo Oculomotor/diagnóstico por imagem
Doenças do Nervo Oculomotor/etiologia
Tomografia Computadorizada por Raios X
Adulto Jovem
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170918
[Lr] Data última revisão:
170918
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170204
[St] Status:MEDLINE


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[PMID]:28110076
[Au] Autor:Takagi S; Kono Y; Nagase M; Mochio S; Kato F
[Ad] Endereço:Department of Neurology, The Jikei University School of Medicine, Japan; Department of Neuroscience, The Jikei University School of Medicine, Japan.
[Ti] Título:Facilitation of distinct inhibitory synaptic inputs by chemical anoxia in neurons in the oculomotor, facial and hypoglossal motor nuclei of the rat.
[So] Source:Exp Neurol;290:95-105, 2017 Apr.
[Is] ISSN:1090-2430
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disorder characterized by the selective loss of motor neurons in the brainstem and spinal cord. Clinical studies have indicated that there is a distinct region-dependent difference in the vulnerability of motor neurons. For example, the motor neurons in the facial and hypoglossal nuclei are more susceptible to neuronal death than those in the oculomotor nucleus. To understand the mechanism underlying the differential susceptibility to cell death of the neurons in different motor nuclei, we compared the effects of chemical anoxia on the membrane currents and postsynaptic currents in different motor nuclei. The membrane currents were recorded from neurons in the oculomotor, facial and hypoglossal nuclei in brain slices of juvenile Wistar rats by using whole-cell recording in the presence of tetrodotoxin that prevents action potential-dependent synaptic transmission. NaCN consistently induced an inward current and a significant increase in the frequency of spontaneous synaptic inputs in neurons from these three nuclei. However, this increase in the synaptic input frequency was abolished by strychnine, a glycine receptor antagonist, but not by picrotoxin in neurons from the hypoglossal and facial nuclei, whereas that in neurons from the oculomotor nucleus was abolished by picrotoxin, but not by strychnine. Blocking ionotropic glutamate receptors did not significantly affect the NaCN-induced release facilitation in any of the three motor nuclei. These results suggest that anoxia selectively facilitates glycine release in the hypoglossal and facial nuclei and GABA release in the oculomotor nucleus. The region-dependent differences in the neurotransmitters involved in the anoxia-triggered release facilitation might provide a basis for the selective vulnerability of motor neurons in the neurodegeneration associated with ALS.
[Mh] Termos MeSH primário: Nervo Facial/patologia
Nervo Hipoglosso/patologia
Hipóxia/induzido quimicamente
Hipóxia/patologia
Neurônios/patologia
Nervo Oculomotor/patologia
Sinapses/patologia
[Mh] Termos MeSH secundário: Esclerose Amiotrófica Lateral/patologia
Animais
Morte Celular/efeitos dos fármacos
Modelos Animais de Doenças
Técnicas de Patch-Clamp
Ratos
Ratos Wistar
Cianeto de Sódio
Transmissão Sináptica/efeitos dos fármacos
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
O5DDB9Z95G (Sodium Cyanide)
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170803
[Lr] Data última revisão:
170803
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170123
[St] Status:MEDLINE


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[PMID]:27923188
[Au] Autor:Kuwabara S; Sekiguchi Y; Misawa S
[Ad] Endereço:Department of Neurology, Graduate School of Medicine, Chiba University, 1-8-1 Inohana, Chuo-ku, Chiba 260-8670, Japan. Electronic address: kuwabara-s@faculty.chiba-u.jp.
[Ti] Título:Electrophysiology in Fisher syndrome.
[So] Source:Clin Neurophysiol;128(1):215-219, 2017 Jan.
[Is] ISSN:1872-8952
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Fisher syndrome (FS), a variant of Guillain-Barré syndrome (GBS), is characterized by the clinical triad of ophthalmoplegia, ataxia, and areflexia. The lesion sites for these unique clinical features include the oculomotor nerves and group 1a neurons in the dorsal root ganglion, and the presence of FS is determined by the expression of ganglioside GQ1b in the human nervous system. Neurophysiological findings suggest that ataxia and areflexia are due to an impaired proprioceptive afferent system. Typically, the soleus H-reflex is absent and a body-sway analysis using posturography shows a 1-Hz peak, which indicates proprioception dysfunction. Sensory nerve action potentials and somatosensory-evoked potentials are abnormal in approximately 30% of FS patients, indicating the occasional involvement of cutaneous (group 2) afferents. During the disease course, approximately 15% of FS patients suffer an overlap of axonal GBS with nerve conduction abnormalities that reflect axonal dysfunction. This review summarizes electrophysiological abnormalities and their clinical significance in FS.
[Mh] Termos MeSH primário: Fenômenos Eletrofisiológicos/fisiologia
Síndrome de Miller Fisher/diagnóstico
Síndrome de Miller Fisher/fisiopatologia
Condução Nervosa/fisiologia
[Mh] Termos MeSH secundário: Ataxia/diagnóstico
Ataxia/fisiopatologia
Potenciais Somatossensoriais Evocados/fisiologia
Reflexo H/fisiologia
Seres Humanos
Nervo Oculomotor/fisiopatologia
Propriocepção/fisiologia
Reflexo Anormal/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170531
[Lr] Data última revisão:
170531
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161207
[St] Status:MEDLINE


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[PMID]:27884738
[Au] Autor:Yamaguchi K
[Ad] Endereço:Department of Pathology, Dokkyo University School of Medicine, 880 Kitakobayashi, Mibu, Tochigi 321-0293, Japan. Electronic address: katsuyukiy@cc9.ne.jp.
[Ti] Título:Development of the human oculomotor nuclear complex: Centrally-projecting Edinger-Westphal nucleus.
[So] Source:Neurosci Lett;646:8-14, 2017 Apr 12.
[Is] ISSN:1872-7972
[Cp] País de publicação:Ireland
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: The cytoarchitecturally defined Edinger-Westphal nucleus (EW) is now referred to by many investigators as the centrally-projecting EW (EWcp) in humans. Although the mature structure is well-characterized, there have been few reports describing the precise morphology of this nucleus during the second half of gestation. SUBJECTS/DESIGN: Eleven brains were examined from preterm infants, aged 20-39 postmenstrual weeks, who died of various causes. After fixation, the brains were embedded in celloidin and serial sections of 30-µm thickness were cut in the horizontal plane. Sections were stained using the Klüver-Barrera method. In addition to microscopic observations, computerized 3D reconstruction and morphometry were performed. RESULTS: From 21 weeks, the EWcp had a distinctive, complex 3D structure comprising two or three parts. The dorsal part was arcuate, half encircling the oculomotor somatic nuclei (OSN). The rostral part was the most voluminous, ventral to the rostral OSN, extending anteriorly. The caudal part was the smallest, and was composed of several neuronal groups near the ventral tip of the OSN. In three cases, the caudal part was absent. It could also be joined to the rostral part, forming a ventral part. The total volume of the EWcp increased exponentially with age, and the ventral part grew more rapidly than the dorsal part. The mean neuronal profile area increased linearly with age, and the rate of increase was almost equal between the dorsal and ventral parts. CONCLUSIONS: This study suggests that a distinctive, complex, two- or three-part 3D structure of the EWcp is preserved after mid-gestation, and that the ventral part of the EWcp may expand in volume more rapidly than the dorsal part.
[Mh] Termos MeSH primário: Núcleo de Edinger-Westphal/patologia
Neurônios/metabolismo
Nervo Oculomotor/patologia
Complexo Nuclear Oculomotor/patologia
[Mh] Termos MeSH secundário: Núcleo de Edinger-Westphal/crescimento & desenvolvimento
Seres Humanos
Nervo Oculomotor/metabolismo
Complexo Nuclear Oculomotor/crescimento & desenvolvimento
Urocortinas/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (UCN1 protein, human); 0 (Urocortins)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171010
[Lr] Data última revisão:
171010
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161126
[St] Status:MEDLINE


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[PMID]:27876664
[Au] Autor:Lim JJ; Clark HB; Grande AW
[Ad] Endereço:Department of Neurosurgery, University of Minnesota, Minneapolis, Minnesota, USA. Electronic address: limxx132@umn.edu.
[Ti] Título:Isolated Hypertrophic Neuropathy of the Oculomotor Nerve.
[So] Source:World Neurosurg;98:880.e1-880.e4, 2017 Feb.
[Is] ISSN:1878-8769
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Hypertrophic neuropathy is a rare entity commonly associated with peripheral nerve, characterized by onion bulb formations. Its cranial nerve involvement is very rare; only 7 cases have been found in the literature. CASE DESCRIPTION: A 32-year-old white man with a 5-year history of intermittent right retro-orbital headache and mild right ptosis presented to the Neurosurgery Clinic. A magnetic resonance imaging of his brain demonstrated an enhancing lesion associated with the right third nerve. He underwent biopsy of the lesion, and its pathology report confirmed the diagnosis of hypertrophic neuropathy. Within 4 months, his third nerve palsy had completely resolved and was functioning fully. CONCLUSIONS: Here, we report a first case of isolated hypertrophic neuropathy involving the oculomotor nerve.
[Mh] Termos MeSH primário: Doenças do Nervo Oculomotor/cirurgia
Nervo Oculomotor/patologia
[Mh] Termos MeSH secundário: Adulto
Encéfalo/diagnóstico por imagem
Craniotomia
Cefaleia/etiologia
Seres Humanos
Hipertrofia
Imagem por Ressonância Magnética
Masculino
Nervo Oculomotor/diagnóstico por imagem
Doenças do Nervo Oculomotor/complicações
Doenças do Nervo Oculomotor/diagnóstico por imagem
Córtex Pré-Frontal/cirurgia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170915
[Lr] Data última revisão:
170915
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161124
[St] Status:MEDLINE


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[PMID]:27859787
[Au] Autor:Park HK; Rha HK; Lee KJ; Chough CK; Joo W
[Ad] Endereço:Department of Neurosurgery, Yeouido St. Mary's Hospital, The Catholic University of Korea College of Medicine, Seoul, South Korea.
[Ti] Título:Microsurgical Anatomy of the Oculomotor Nerve.
[So] Source:Clin Anat;30(1):21-31, 2017 Jan.
[Is] ISSN:1098-2353
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The oculomotor nerve supplies the extraocular muscles. It also supplies the ciliary and sphincter pupillae muscles through the ciliary ganglion. The nerve fibers leave the midbrain through the most medial part of the cerebral peduncle and enter the interpeduncular cistern. After the oculomotor nerve emerges from the interpeduncular fossa, it enters the cavernous sinus slightly lateral and anterior to the dorsum sellae. It enters the orbit through the superior orbital fissure, after exiting the cavernous sinus, to innervate the extraocular muscles. Therefore, knowledge of the detailed anatomy and pathway of the oculomotor nerve is critical for the management of lesions located in the middle cranial fossa and the clival, cavernous, and orbital regions. This review describes the microsurgical anatomy of the oculomotor nerve and presents pictures illustrating this nerve and its surrounding connective and neurovascular structures. Clin. Anat. 30:21-31, 2017. © 2016 Wiley Periodicals, Inc.
[Mh] Termos MeSH primário: Nervo Oculomotor/anatomia & histologia
[Mh] Termos MeSH secundário: Seres Humanos
Microcirurgia
Nervo Oculomotor/cirurgia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1703
[Cu] Atualização por classe:170302
[Lr] Data última revisão:
170302
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161119
[St] Status:MEDLINE
[do] DOI:10.1002/ca.22811



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BIREME/OPAS/OMS - Centro Latino-Americano e do Caribe de Informação em Ciências da Saúde