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[PMID]:27795769
[Au] Autor:Doghmi N; Elkoundi A; Meskine A; Benakrout A; Baite A; Haimeur C
[Ad] Endereço:Service de Réanimation Médicale, Pôle Anesthésie-Réanimation, Hôpital Militaire Med V, Rabat, Maroc.
[Ti] Título:[Epilepsy revealing chorea-acanthocytosis: about a case].
[Ti] Título:Epilepsie révélant une chorée-acanthocytose: à propos d'un cas..
[So] Source:Pan Afr Med J;24:172, 2016.
[Is] ISSN:1937-8688
[Cp] País de publicação:Uganda
[La] Idioma:fre
[Ab] Resumo:Chorea-acanthocytosis (ChAc) is an extremely rare autosomal recessive disorder caused by mutations in the VSP13A gene on chromosome 9q21. It is characterized by neurological symptoms, psychiatric manifestations and multisystem involvement resulting in myopathy, axonal neuropathy and presence of spiculated red blood cells or acanthocytes. Rarely, epilepsy may be the early symptom in these patients. This can lead to serious delays in diagnosis. We here report the case of a patient with this disease who had seizures several years before the onset of typical manifestations.
[Mh] Termos MeSH primário: Acantócitos/patologia
Epilepsia/etiologia
Neuroacantocitose/diagnóstico
[Mh] Termos MeSH secundário: Adulto
Diagnóstico Tardio
Seres Humanos
Masculino
Neuroacantocitose/complicações
Neuroacantocitose/fisiopatologia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1703
[Cu] Atualização por classe:170310
[Lr] Data última revisão:
170310
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161101
[St] Status:MEDLINE


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[PMID]:27061618
[Au] Autor:Oikawa S; Ito S; Odajima C; Horibe Y; Urano S; Suzuki K; Minegishi M; Itoh T; Shibasaki I; Shimizu H
[Ad] Endereço:Japanese Red Cross Tohoku Block Blood Center, Miyagi 981-3206, Japan.
[Ti] Título:Reproducible delayed appearance of platelet clumps and acanthocytes in blood components collected from a single donor.
[So] Source:Transfus Med;26(1):69-70, 2016 Feb.
[Is] ISSN:1365-3148
[Cp] País de publicação:England
[La] Idioma:eng
[Mh] Termos MeSH primário: Acantócitos
Remoção de Componentes Sanguíneos
Doadores de Sangue
Plaquetas
Segurança do Sangue
Agregação Plaquetária
[Mh] Termos MeSH secundário: Adulto
Seres Humanos
Masculino
Fatores de Tempo
[Pt] Tipo de publicação:CASE REPORTS; LETTER
[Em] Mês de entrada:1612
[Cu] Atualização por classe:161230
[Lr] Data última revisão:
161230
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160411
[St] Status:MEDLINE
[do] DOI:10.1111/tme.12274


  3 / 295 MEDLINE  
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[PMID]:26147873
[Au] Autor:Pacheco JM; Yilmaz M; Rice L
[Ad] Endereço:Division of Hematology and Oncology, Department of Internal Medicine, Baylor College of Medicine, 1709 Dryden, Suite 5.82A, Houston, Texas, 77030.
[Ti] Título:How low is too low: Statin induced hemolysis.
[So] Source:Am J Hematol;91(2):267, 2016 Feb.
[Is] ISSN:1096-8652
[Cp] País de publicação:United States
[La] Idioma:eng
[Mh] Termos MeSH primário: Abetalipoproteinemia/induzido quimicamente
Acantócitos/efeitos dos fármacos
Atorvastatina Cálcica/efeitos adversos
Hemólise/efeitos dos fármacos
Inibidores de Hidroximetilglutaril-CoA Redutases/efeitos adversos
[Mh] Termos MeSH secundário: Abetalipoproteinemia/sangue
Abetalipoproteinemia/patologia
Acantócitos/metabolismo
Acantócitos/patologia
Atorvastatina Cálcica/administração & dosagem
Atorvastatina Cálcica/uso terapêutico
Feminino
Seres Humanos
Inibidores de Hidroximetilglutaril-CoA Redutases/administração & dosagem
Inibidores de Hidroximetilglutaril-CoA Redutases/uso terapêutico
Meia-Idade
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Hydroxymethylglutaryl-CoA Reductase Inhibitors); 48A5M73Z4Q (Atorvastatin Calcium)
[Em] Mês de entrada:1606
[Cu] Atualização por classe:160123
[Lr] Data última revisão:
160123
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150707
[St] Status:MEDLINE
[do] DOI:10.1002/ajh.24105


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[PMID]:26814078
[Au] Autor:Ping M; Li L; Deng X; Cong Y
[Ad] Endereço:Department of Clinical Laboratory in South Building, Chinese PLA General Hospital, Beijing 100853, China.
[Ti] Título:[Clinical significance of new urine red blood cell parameter in different kinds of glomerulonephritis].
[So] Source:Zhonghua Yi Xue Za Zhi;95(36):2976-9, 2015 Sep 22.
[Is] ISSN:0376-2491
[Cp] País de publicação:China
[La] Idioma:chi
[Ab] Resumo:OBJECTIVE: To investigate the clinical value of new kinds of urinary erythrocyte morphology parameter in discriminating different pathology types of glomerulonephritis. METHODS: All of the 52 urine samples were from glomerulonephritis patients who had been diagnosed by renal biopsy results. The change of the percentage of acanthocytes, the size of RBC, the shape of RBC between the primary glomerulonephritis (39 cases) and secondary glomerulonephritis (13 cases) urine were detected by AVE-764 fully automatic urine cell analyzer. RESULTS: Acanthocytes could be found in both primary glomerulonephritis and secondary glomerulonephritis. Of the patients whose acanthocytes percentages above 10%, 94.1% had primary glomerulonephritis and 5.9% had secondary glomerulonephritis. The picture of size-shape phase were classified as strip-type, inverted triangle-type and hanging tail-type. 95.2% Strip-type cases were from primary glomerulonephritis patients. Triangle-typenormally cases were all from primary glomerulonephritis patients. Hanging tail-type cases were all from secondary glomerulonephritis. CONCLUSION: High acanthocytes percentage is most common in primary glomerulonephritis, going with the size and shape of RBC can be useful in the differential diagnosis of different pathology types of glomerulonephritis.
[Mh] Termos MeSH primário: Índices de Eritrócitos
Glomerulonefrite
[Mh] Termos MeSH secundário: Acantócitos
Separação Celular
Diagnóstico Diferencial
Eritrócitos
Seres Humanos
Nefrectomia
Urinálise
[Pt] Tipo de publicação:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Mês de entrada:1604
[Cu] Atualização por classe:160127
[Lr] Data última revisão:
160127
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160128
[St] Status:MEDLINE


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[PMID]:26185871
[Au] Autor:Wong RW; Gonsalves MN; Huber ML; Rich L; Strom A
[Ad] Endereço:Animal Specialty Group, Los Angeles, California.
[Ti] Título:Erythrocyte and Biochemical Abnormalities as Diagnostic Markers in Dogs With Hemangiosarcoma Related Hemoabdomen.
[So] Source:Vet Surg;44(7):852-7, 2015 Oct.
[Is] ISSN:1532-950X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: To investigate: 1) acanthocytosis and presence of acanthocytes in peritoneal fluid as a diagnostic marker for hemangiosarcoma (HSA) in dogs with non-traumatic hemoabdomen; and 2) the association between other erythrocyte, biochemical, and hematologic abnormalities as a mean of differentiating HSA from other disease. STUDY DESIGN: Prospective double-blinded cohort study. ANIMALS: Dogs (n = 40) with non-traumatic hemoabdomen. METHODS: Dogs diagnosed with hemoabdomen (January 2012 to May 2013) had cytologic evaluation of abdominal effusion and peripheral blood smears. Peripheral blood CBC, PT, and aPTT, as well as blood and effusion acanthocytes, keratocytes, schistocytes, lactate, glucose, PCV, and TP results were compared using the paired t-test or Fisher's exact test. Based on histologic confirmation of HSA, dogs were divided into 2 groups (HSA, non-HSA) and variables compared. RESULTS: There was no significant difference in erythrocyte morphology in abdominal effusion or peripheral blood between dogs with HSA or non-HSA related hemoabdomen. Platelet concentration and peripheral blood PCV were significantly lower in the HSA group. CONCLUSIONS: A reliable preoperative biochemical or cytologic test to differentiate between HSA and non-HSA related hemoabdomen was not identified.
[Mh] Termos MeSH primário: Neoplasias Abdominais/veterinária
Acantócitos/metabolismo
Líquido Ascítico/patologia
Doenças do Cão/patologia
Hemangiossarcoma/veterinária
Hemorragia/veterinária
[Mh] Termos MeSH secundário: Neoplasias Abdominais/fisiopatologia
Animais
Biomarcadores/análise
Análise Química do Sangue/veterinária
Cães
Método Duplo-Cego
Feminino
Hemangiossarcoma/diagnóstico
Hemorragia/patologia
Hemorragia/fisiopatologia
Masculino
Estudos Prospectivos
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Biomarkers)
[Em] Mês de entrada:1607
[Cu] Atualização por classe:151005
[Lr] Data última revisão:
151005
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150718
[St] Status:MEDLINE
[do] DOI:10.1111/vsu.12361


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[PMID]:25934153
[Au] Autor:Frey BM; Gassner C; Jung HH
[Ad] Endereço:Blood Transfusion Service SRK, 8952 Schlieren, Zurich, Switzerland. Electronic address: bm.frey@zhbsd.ch.
[Ti] Título:Neurodegeneration in the elderly - When the blood type matters: An overview of the McLeod syndrome with focus on hematological features.
[So] Source:Transfus Apher Sci;52(3):277-84, 2015 Jun.
[Is] ISSN:1473-0502
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Multisystem deterioration occurs mainly in older individuals and may be related to physiological tissue degeneration. However, genetic predisposition may be unmasked by inappropriate functional and structural system deficiencies. McLeod syndrome (MLS) is a rare, multisystem disease which is X-chromosomal inherited and belongs to the neuroacanthocytosis syndromes (NAS). The main clinical manifestations contain progressive neuro-psychiatric and cognitive deterioration, choreatic movement disorder, as well as myopathy, sensory motor axonal neuropathy and cardiomyopathy. In addition, MLS patients have red blood cell abnormalities including immune-hematological, morphological and functional impairments of red blood cells. In large deletions, contiguous gene syndrome may arise, including Duchenne muscular dystrophia, cellular immunodeficiency or retinitis pigmentosa. Hematological abnormalities such as blood group abnormalities in Kell- and XK blood group system, formation of anti-public red blood cell alloantibodies, acanthocytosis and elevated creatinine phosphokinase may precede clinical disease manifestation for decades and provide tools for early diagnosis. Patients with unexplained neuro-muscular deterioration and/or neuro-psychological pathologies accompanied with hematological abnormalities should be investigated for MLS.
[Mh] Termos MeSH primário: Neuroacantocitose/sangue
Doenças Neurodegenerativas/sangue
[Mh] Termos MeSH secundário: Acantócitos/citologia
Idoso
Alelos
Antígenos de Grupos Sanguíneos/genética
Cromossomos Humanos X
Análise Mutacional de DNA
Eritrócitos/citologia
Éxons
Deleção de Genes
Geriatria
Seres Humanos
Sistema do Grupo Sanguíneo de Kell/genética
Masculino
Mutação
Neuroacantocitose/genética
Fenótipo
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Blood Group Antigens); 0 (Kell Blood-Group System)
[Em] Mês de entrada:1604
[Cu] Atualização por classe:150613
[Lr] Data última revisão:
150613
[Sb] Subgrupo de revista:T
[Da] Data de entrada para processamento:150503
[St] Status:MEDLINE


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[PMID]:25933379
[Au] Autor:Cluitmans JC; Tomelleri C; Yapici Z; Dinkla S; Bovee-Geurts P; Chokkalingam V; De Franceschi L; Brock R; Bosman GJ
[Ad] Endereço:Department of Biochemistry, Radboud Institute for Molecular Life Sciences, Radboud University Medical Center, Geert Grooteplein 28, 6525 GA, Nijmegen, The Netherlands.
[Ti] Título:Abnormal red cell structure and function in neuroacanthocytosis.
[So] Source:PLoS One;10(5):e0125580, 2015.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Panthothenate kinase-associated neurodegeneration (PKAN) belongs to a group of hereditary neurodegenerative disorders known as neuroacanthocytosis (NA). This genetically heterogeneous group of diseases is characterized by degeneration of neurons in the basal ganglia and by the presence of deformed red blood cells with thorny protrusions, acanthocytes, in the circulation. OBJECTIVE: The goal of our study is to elucidate the molecular mechanisms underlying this aberrant red cell morphology and the corresponding functional consequences. This could shed light on the etiology of the neurodegeneration. METHODS: We performed a qualitative and semi-quantitative morphological, immunofluorescent, biochemical and functional analysis of the red cells of several patients with PKAN and, for the first time, of the red cells of their family members. RESULTS: We show that the blood of patients with PKAN contains not only variable numbers of acanthocytes, but also a wide range of other misshapen red cells. Immunofluorescent and immunoblot analyses suggest an altered membrane organization, rather than quantitative changes in protein expression. Strikingly, these changes are not limited to the red blood cells of PKAN patients, but are also present in the red cells of heterozygous carriers without neurological problems. Furthermore, changes are not only present in acanthocytes, but also in other red cells, including discocytes. The patients' cells, however, are more fragile, as observed in a spleen-mimicking device. CONCLUSION: These morphological, molecular and functional characteristics of red cells in patients with PKAN and their family members offer new tools for diagnosis and present a window into the pathophysiology of neuroacanthocytosis.
[Mh] Termos MeSH primário: Acantócitos/patologia
Membrana Eritrocítica/patologia
Neuroacantocitose/patologia
Neurodegeneração Associada a Pantotenato-Quinase/patologia
[Mh] Termos MeSH secundário: Acantócitos/metabolismo
Acantócitos/ultraestrutura
Adulto
Idoso
Proteína 1 de Troca de Ânion do Eritrócito/genética
Proteína 1 de Troca de Ânion do Eritrócito/metabolismo
Estudos de Casos e Controles
Forma Celular
Criança
Contagem de Eritrócitos
Membrana Eritrocítica/metabolismo
Membrana Eritrocítica/ultraestrutura
Feminino
Expressão Gênica
Heterozigoto
Homozigoto
Seres Humanos
Masculino
Proteínas de Membrana/genética
Proteínas de Membrana/metabolismo
Meia-Idade
Neuroacantocitose/genética
Neuroacantocitose/metabolismo
Neuroacantocitose/fisiopatologia
Fragilidade Osmótica
Neurodegeneração Associada a Pantotenato-Quinase/genética
Neurodegeneração Associada a Pantotenato-Quinase/metabolismo
Neurodegeneração Associada a Pantotenato-Quinase/fisiopatologia
Linhagem
Espectrina/genética
Espectrina/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Anion Exchange Protein 1, Erythrocyte); 0 (Membrane Proteins); 0 (SLC4A1 protein, human); 0 (STOM protein, human); 12634-43-4 (Spectrin)
[Em] Mês de entrada:1602
[Cu] Atualização por classe:170922
[Lr] Data última revisão:
170922
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150502
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0125580


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[PMID]:25915509
[Au] Autor:Schiessl-Weyer J; Roa P; Laccone F; Kluge B; Tichy A; De Almeida Ribeiro E; Prohaska R; Stoeter P; Siegl C; Salzer U
[Ad] Endereço:Department of Medical Biochemistry, Max F. Perutz Laboratories, Medical University of Vienna, Vienna, Austria.
[Ti] Título:Acanthocytosis and the c.680 A>G Mutation in the PANK2 Gene: A Study Enrolling a Cohort of PKAN Patients from the Dominican Republic.
[So] Source:PLoS One;10(4):e0125861, 2015.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Pantothenate Kinase-Associated Neurodegeneration (PKAN) is a form of Neurodegeneration with Brain Iron Accumulation (NBIA) associated with mutations in the pantothenate kinase 2 gene (PANK2). Pantothenate kinases catalyze the rate-limiting step of coenzyme A synthesis and Pank2 is the only pantothenate kinase isoform in humans that is localized to mitochondria. Acanthocytosis, the occurrence of spiculated erythrocytes, is observed in about 10% of the PKAN patients. Therefore PKAN is also classified together with other rare neurodegenerative diseases like Chorea Acanthocytosis (ChAc) and McLeod syndrome (MLS) into the Neuroacanthocytosis (NA) syndromes. It has not been investigated yet whether acanthocytosis in PKAN is associated with a specific subset of Pank2 mutations. In this study, we analyzed acanthocytosis of a cohort of 25 PKAN patients from the Dominican Republic that are homozygous for the c.680 A>G mutation in the PANK2 gene as compared to control donors that are heterozygous or wild-type with respect to this mutation. 3D modeling of this mutation indicated that the replacement of a tyrosine by a cysteine at position 227 in Pank2 disrupts a polar interaction within the A domain of the enzyme. Mean acanthocyte count was elevated in the cohort of patients, however, acanthocytosis varied among the patients with nearly half of them showing high (>20%) or elevated acanthocytosis and the rest showing mild (6-10%) or no (<6%) acanthocytosis. Heterozygous control donors revealed a tendency to mild acanthocytosis. Based on the insight that Pank2 is a normal constituent of red blood cells and de novo biosynthesis of coenzyme A is likely to take place in the erythrocyte cytosol we propose a hypothetical model that accounts for the variability in the occurrence of acanthocytic cells in PKAN.
[Mh] Termos MeSH primário: Abetalipoproteinemia/diagnóstico
Acantócitos/patologia
Neurodegeneração Associada a Pantotenato-Quinase/complicações
Fosfotransferases (Aceptor do Grupo Álcool)/genética
Polimorfismo de Nucleotídeo Único
[Mh] Termos MeSH secundário: Abetalipoproteinemia/genética
Abetalipoproteinemia/patologia
Adolescente
Adulto
Criança
Estudos de Coortes
República Dominicana
Estabilidade Enzimática
Homozigoto
Seres Humanos
Modelos Moleculares
Neurodegeneração Associada a Pantotenato-Quinase/sangue
Neurodegeneração Associada a Pantotenato-Quinase/genética
Fosfotransferases (Aceptor do Grupo Álcool)/química
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
EC 2.7.1.- (Phosphotransferases (Alcohol Group Acceptor)); EC 2.7.1.33 (pantothenate kinase)
[Em] Mês de entrada:1601
[Cu] Atualização por classe:150508
[Lr] Data última revisão:
150508
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150428
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0125861


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[PMID]:25402479
[Au] Autor:Christopher MM; Hawkins MG; Burton AG
[Ad] Endereço:Department of Pathology, Immunology and Microbiology, University of California Davis, Davis, CA, 95616, United States of America.
[Ti] Título:Poikilocytosis in rabbits: prevalence, type, and association with disease.
[So] Source:PLoS One;9(11):e112455, 2014.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Rabbits (Oryctolagus cuniculus) are a popular companion animal, food animal, and animal model of human disease. Abnormal red cell shapes (poikilocytes) have been observed in rabbits, but their significance is unknown. The objective of this study was to investigate the prevalence and type of poikilocytosis in pet rabbits and its association with physiologic factors, clinical disease, and laboratory abnormalities. We retrospectively analyzed blood smears from 482 rabbits presented to the University of California-Davis Veterinary Medical Teaching Hospital from 1990 to 2010. Number and type of poikilocytes per 2000 red blood cells (RBCs) were counted and expressed as a percentage. Acanthocytes (>3% of RBCs) were found in 150/482 (31%) rabbits and echinocytes (>3% of RBCs) were found in 127/482 (27%) of rabbits, both healthy and diseased. Thirty-three of 482 (7%) rabbits had >30% acanthocytes and echinocytes combined. Mild to moderate (>0.5% of RBCs) fragmented red cells (schistocytes, microcytes, keratocytes, spherocytes) were found in 25/403 (6%) diseased and 0/79 (0%) healthy rabbits (P = 0.0240). Fragmentation and acanthocytosis were more severe in rabbits with inflammatory disease and malignant neoplasia compared with healthy rabbits (P<0.01). The % fragmented cells correlated with % polychromasia, RDW, and heterophil, monocyte, globulins, and fibrinogen concentrations (P<0.05). Echinocytosis was significantly associated with renal failure, azotemia, and acid-base/electrolyte abnormalities (P<0.05). Serum cholesterol concentration correlated significantly with % acanthocytes (P<0.0001), % echinocytes (P = 0.0069), and % fragmented cells (P = 0.0109), but correlations were weak (Spearman ρ <0.02). These findings provide important insights into underlying pathophysiologic mechanisms that appear to affect the prevalence and type of naturally-occurring poikilocytosis in rabbits. Our findings support the need to carefully document poikilocytes in research investigations and in clinical diagnosis and to determine their diagnostic and prognostic value.
[Mh] Termos MeSH primário: Eritrócitos Anormais/patologia
[Mh] Termos MeSH secundário: Acantócitos/patologia
Doenças dos Animais/diagnóstico
Doenças dos Animais/epidemiologia
Doenças dos Animais/patologia
Animais
Feminino
Doenças Hematológicas/veterinária
Masculino
Prevalência
Coelhos
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1507
[Cu] Atualização por classe:170220
[Lr] Data última revisão:
170220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:141118
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0112455


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[PMID]:24756017
[Au] Autor:Zimmer V; Bittenbring J; Fries P; Lammert F
[Ad] Endereço:Department of Medicine II, Saarland University Medical Center, Homburg, Germany.
[Ti] Título:Severe mixed-type iron overload in alcoholic cirrhosis related to advanced spur cell anemia.
[So] Source:Ann Hepatol;13(3):396-8, 2014 May-Jun.
[Is] ISSN:1665-2681
[Cp] País de publicação:Mexico
[La] Idioma:eng
[Mh] Termos MeSH primário: Acantócitos/patologia
Anemia Hemolítica/patologia
Sobrecarga de Ferro/sangue
Cirrose Hepática Alcoólica/sangue
Fígado/patologia
Pâncreas/patologia
[Mh] Termos MeSH secundário: Anemia Hemolítica/etiologia
Seres Humanos
Sobrecarga de Ferro/etiologia
Sobrecarga de Ferro/patologia
Cirrose Hepática Alcoólica/complicações
Cirrose Hepática Alcoólica/patologia
Imagem por Ressonância Magnética
Meia-Idade
Baço/patologia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1501
[Cu] Atualização por classe:140423
[Lr] Data última revisão:
140423
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:140424
[St] Status:MEDLINE



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