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[PMID]:29458461
[Au] Autor:Ben Ali Gam Z; Thioye A; Cayol JL; Joseph M; Fauque G; Labat M
[Ad] Endereço:1​Laboratoire de Microbiologie IRD, Aix-Marseille Université, Université du Sud Toulon-Var, CNRS/INSU, IRD, MI0 UM110, 163 avenue de Luminy, case 925, F-13288 Marseille cedex 9, France.
[Ti] Título:Characterization of Desulfovibrio salinus sp. nov., a slightly halophilic sulfate-reducing bacterium isolated from a saline lake in Tunisia.
[So] Source:Int J Syst Evol Microbiol;68(3):715-720, 2018 Mar.
[Is] ISSN:1466-5034
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:A novel slightly halophilic sulfate-reducing bacterium, designated strain P1BSR , was isolated from water of a saline lake in Tunisia. Strain P1BSR had motile (single polar flagellum), Gram-negative, rod-shaped, non-spore-forming cells, occurring singly or in pairs. Strain P1BSR grew at temperatures between 15 and 45 °C (optimum 40 °C), and in a pH range between 6 and 8.5 (optimum pH 6.7). The strain required NaCl for growth (1 % w/v), and tolerated high NaCl concentration (up to 12 % w/v) with an optimum of 3 % (w/v). Sulfate, thiosulfate and sulfite served as terminal electron acceptors, but not elemental sulfur, fumarate, nitrate and nitrite. Strain P1BSR utilized lactate, pyruvate, formate, d-fructose and glycerol as carbon and energy sources. The main cellular fatty acid was C16 : 0 (50.8 %). The genomic DNA G+C content was 47.7 mol%. Phylogenetic analysis of 16S rRNA gene sequence similarity indicated that strain P1BSR was affiliated to the genus Desulfovibrio, with the type strains Desulfovibrio salexigens (96.51 %), Desulfovibrio zosterae (95.68 %), Desulfovibrio hydrothermalis (94.81 %) and Desulfovibrio ferrireducens (94.73 %) as its closest phylogenetic relatives. On the basis of genotypic, phenotypic and phylogenetic characteristics, it is proposed to assign strain P1BSR to a novel species of the genus Desulfovibrio, Desulfovibrio salinus sp. nov. The type strain is P1BSR (=DSM 101510 =JCM 31065 ).
[Mh] Termos MeSH primário: Desulfovibrio/classificação
Lagos/microbiologia
Filogenia
Salinidade
[Mh] Termos MeSH secundário: Técnicas de Tipagem Bacteriana
Composição de Bases
DNA Bacteriano/genética
Desulfovibrio/genética
Desulfovibrio/isolamento & purificação
Ácidos Graxos/química
Oxirredução
RNA Ribossômico 16S/genética
Análise de Sequência de DNA
Sulfatos/metabolismo
Tunísia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (DNA, Bacterial); 0 (Fatty Acids); 0 (RNA, Ribosomal, 16S); 0 (Sulfates)
[Em] Mês de entrada:1803
[Cu] Atualização por classe:180308
[Lr] Data última revisão:
180308
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180221
[St] Status:MEDLINE
[do] DOI:10.1099/ijsem.0.002567


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[PMID]:28466260
[Au] Autor:Singh SB; Wilson M; Ritz N; Lin HC
[Ad] Endereço:Section of Gastroenterology, Medicine Service, New Mexico VA Health Care System, 1501 San Pedro SE, Albuquerque, NM, 87108, USA.
[Ti] Título:Autophagy Genes of Host Responds to Disruption of Gut Microbial Community by Antibiotics.
[So] Source:Dig Dis Sci;62(6):1486-1497, 2017 06.
[Is] ISSN:1573-2568
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Defective autophagic machinery, such as that in Crohn's disease patients homozygous for ATG16L1 risk allele, is associated with alteration of resident gut bacterial communities. However, whether or not host autophagy responds to changes in the resident gut microbial community is not known. Here, we investigated the effect of antibiotic-induced disruption of the gut microbiome (dysbiosis) on autophagy gene expression and the expression of antimicrobial peptides/protein (AMP) over time. AIM: To test the hypothesis that antibiotic treatment may cause time-dependent changes in gut bacterial density, autophagy genes, and antimicrobial protein/peptide gene expression. METHODS: Mice (n = 8 per group) were treated with antibiotic cocktail and sacrificed at different intervals of recovery (days 3, 7, 10, 14, 21, 28, 35, and 42) post-antibiotics. DNA and RNA were extracted from small intestinal tissues. Bacterial density, expression of host autophagy genes, and AMP genes were analyzed by relative quantitative PCR. Fold change difference in comparison with untreated control group was calculated using 2 method. Statistical analysis was performed using nonparametric Mann-Whitney test. RESULTS: Gut bacterial density changed in a time-dependent fashion in response to antibiotic treatment. These changes were concurrent with upregulation of autophagy genes and antimicrobial peptide/protein gene expression. We further showed that an oral gavage of a resident microbe Desulfovibrio, which bloomed in antibiotic-treated animals, induced Atg5 and lysozyme (Lyz) gene expression. CONCLUSION: Autophagy genes respond to dysbiosis induced by antibiotics. This response may be a host mechanism to detect and possibly correct dysbiosis by activating antimicrobial peptides/proteins that control the microbial load in the gut.
[Mh] Termos MeSH primário: Antibacterianos/farmacologia
Peptídeos Catiônicos Antimicrobianos/genética
Autofagia/genética
Disbiose/microbiologia
Microbioma Gastrointestinal/efeitos dos fármacos
RNA Ribossômico 16S/análise
[Mh] Termos MeSH secundário: Animais
Proteína 5 Relacionada à Autofagia/genética
Proteínas Relacionadas à Autofagia/genética
Bacteroidetes
Células Cultivadas
Desulfovibrio
Desulfovibrio vulgaris
Disbiose/induzido quimicamente
Disbiose/genética
Células Epiteliais/efeitos dos fármacos
Feminino
Firmicutes
Expressão Gênica
Intestino Delgado/citologia
Intestino Delgado/microbiologia
Macrófagos/efeitos dos fármacos
Camundongos
Camundongos Endogâmicos C57BL
Muramidase/genética
Proteínas Associadas a Pancreatite
Proteínas/genética
Fatores de Tempo
Regulação para Cima
alfa-Defensinas/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Anti-Bacterial Agents); 0 (Antimicrobial Cationic Peptides); 0 (Atg5 protein, mouse); 0 (Autophagy-Related Protein 5); 0 (Autophagy-Related Proteins); 0 (Pancreatitis-Associated Proteins); 0 (Proteins); 0 (RNA, Ribosomal, 16S); 0 (Reg3g protein, mouse); 0 (alpha-Defensins); 0 (cryptdin 4, mouse); EC 3.2.1.17 (Muramidase)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:171213
[Lr] Data última revisão:
171213
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170504
[St] Status:MEDLINE
[do] DOI:10.1007/s10620-017-4589-8


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[PMID]:28977605
[Au] Autor:Pichette J; Fynn-Sackey N; Gagnon J
[Ad] Endereço:Laurentian University, Department of Biology, Sudbury, Ontario P3E 2C6, Canada.
[Ti] Título:Hydrogen Sulfide and Sulfate Prebiotic Stimulates the Secretion of GLP-1 and Improves Glycemia in Male Mice.
[So] Source:Endocrinology;158(10):3416-3425, 2017 Oct 01.
[Is] ISSN:1945-7170
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Recently, the gastrointestinal microbiome, and its metabolites, has emerged as a potential regulator of host metabolism. However, to date little is known on the precise mechanisms of how this regulation occurs. Hydrogen sulfide (H2S) is abundantly produced in the colon by sulfate-reducing bacteria (SRB). H2S is a bioactive gas that plays regulatory roles in many systems, including metabolic hormone regulation. This gas metabolite is produced in close proximity to the glucagonlike peptide-1 (GLP-1)-secreting cells in the gut epithelium. GLP-1 is a peptide hormone that plays pivotal roles in both glucose homeostasis and appetite regulation. We hypothesized that H2S can directly regulate GLP-1 secretion. We demonstrated that H2S donors (NaHS and GYY4137) directly stimulate GLP-1 secretion in murine L-cells (GLUTag) and that this occurs through p38 mitogen-activated protein kinase without affecting cell viability. We then increased SRB in mice by supplementing the diet with a prebiotic chondroitin sulfate for 4 weeks. Mice treated with chondroitin sulfate had elevated Desulfovibrio piger levels in the feces and increased colonic and fecal H2S concentration. These animals also had enhanced GLP-1 and insulin secretion, improved oral glucose tolerance, and reduced food consumption. These results indicate that H2S plays a stimulatory role in GLP-1 secretion and that sulfate prebiotics can enhance GLP-1 release and its downstream metabolic actions.
[Mh] Termos MeSH primário: Sulfatos de Condroitina/farmacologia
Colo/efeitos dos fármacos
Microbioma Gastrointestinal/efeitos dos fármacos
Peptídeo 1 Semelhante ao Glucagon/efeitos dos fármacos
Sulfeto de Hidrogênio/metabolismo
Mucosa Intestinal/efeitos dos fármacos
Morfolinas/farmacologia
Compostos Organotiofosforados/farmacologia
Prebióticos
Sulfetos/farmacologia
[Mh] Termos MeSH secundário: Animais
Western Blotting
Colo/metabolismo
DNA Bacteriano/análise
Desulfovibrio/efeitos dos fármacos
Ingestão de Alimentos/efeitos dos fármacos
Fezes/química
Microbioma Gastrointestinal/genética
Peptídeo 1 Semelhante ao Glucagon/secreção
Teste de Tolerância a Glucose
Insulina/secreção
Mucosa Intestinal/secreção
Masculino
Camundongos
Reação em Cadeia da Polimerase em Tempo Real
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (DNA, Bacterial); 0 (GYY 4137); 0 (Insulin); 0 (Morpholines); 0 (Organothiophosphorus Compounds); 0 (Prebiotics); 0 (Sulfides); 89750-14-1 (Glucagon-Like Peptide 1); 9007-28-7 (Chondroitin Sulfates); FWU2KQ177W (sodium bisulfide); YY9FVM7NSN (Hydrogen Sulfide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171016
[Lr] Data última revisão:
171016
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:171005
[St] Status:MEDLINE
[do] DOI:10.1210/en.2017-00391


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[PMID]:28912045
[Au] Autor:Figliuolo VR; Dos Santos LM; Abalo A; Nanini H; Santos A; Brittes NM; Bernardazzi C; de Souza HSP; Vieira LQ; Coutinho-Silva R; Coutinho CMLM
[Ad] Endereço:Instituto de Biofísica Carlos Chagas Filho - IBCCF, Universidade Federal do Rio de Janeiro, RJ, Brazil; Laboratório de Inovações em Terapias, Ensino e Bioprodutos - LITEB, Instituto Oswaldo Cruz, Fundação Oswaldo Cruz, Rio de Janeiro, RJ, Brazil.
[Ti] Título:Sulfate-reducing bacteria stimulate gut immune responses and contribute to inflammation in experimental colitis.
[So] Source:Life Sci;189:29-38, 2017 Nov 15.
[Is] ISSN:1879-0631
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:The intestinal microbiota is critical for mammalian immune system development and homeostasis. Sulfate-reducing bacteria (SRB) are part of the normal gut microbiota, but their increased levels may contribute to colitis development, likely in association with hydrogen sulfide (H S) production. Here, we investigated the effects of SRB in the gut immune response in germ-free mice, and in experimental colitis. After 7days of colonization with Desulfovibrio indonesiensis or with a human SRB consortium (from patients with colitis), germ-free mice exhibited alterations in the colonic architecture, with increased cell infiltration in the lamina propria. SRB colonization upregulated the Th17 and Treg profiles of cytokine production/cell activation, in T cells from mesenteric lymph nodes. These alterations were more pronounced in mice colonized with the human SRB consortium, although D. indonesiensis colonization produced higher levels of H S. Importantly, the colon of C57BL/6 mice with colitis induced by TNBS or oxazolone had increased SRB colonization, and the administration of D. indonesiensis to mice with TNBS-induced colitis clearly exacerbated the alterations in colonic architecture observed in the established disease, and also increased mouse weight loss. We conclude that SRB contribute to immune response activation in the gut and play an important role in colitis development.
[Mh] Termos MeSH primário: Colite/patologia
Desulfovibrio/metabolismo
Inflamação/patologia
Sulfatos/metabolismo
[Mh] Termos MeSH secundário: Animais
Colite/imunologia
Modelos Animais de Doenças
Feminino
Seres Humanos
Inflamação/imunologia
Camundongos
Camundongos Endogâmicos C57BL
Camundongos Transgênicos
Oxazolona/toxicidade
Linfócitos T Reguladores/imunologia
Células Th17/imunologia
Ácido Trinitrobenzenossulfônico/toxicidade
Perda de Peso
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Sulfates); 15646-46-5 (Oxazolone); 8T3HQG2ZC4 (Trinitrobenzenesulfonic Acid)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171023
[Lr] Data última revisão:
171023
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170916
[St] Status:MEDLINE


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[PMID]:28867000
[Au] Autor:Thioye A; Gam ZBA; Mbengue M; Cayol JL; Joseph-Bartoli M; Touré-Kane C; Labat M
[Ad] Endereço:1​Laboratoire de Microbiologie Appliquée et de Génie Industriel, Ecole Supérieure Polytechnique, Université Cheikh Anta Diop, BP 5005 Dakar-Fann, Dakar, Sénégal 2​Laboratoire de Microbiologie IRD, Aix-Marseille Université, Université du Sud Toulon-Var, CNRS/INSU, IRD, MI0 UM110, 163 avenue de Lu
[Ti] Título:Desulfovibrio senegalensis sp. nov., a mesophilic sulfate reducer isolated from marine sediment.
[So] Source:Int J Syst Evol Microbiol;67(9):3162-3166, 2017 Sep.
[Is] ISSN:1466-5034
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Several strains of sulfate-reducing bacteria were isolated from marine sediments recovered from Hann Bay (Senegal). All were related to members of the genus Desulfovibrio. A strictly anaerobic, mesophilic and moderately halophilic strain designated BLaC1T was further characterized. Cells of strain BLaC1T stained Gram-negative and were 0.5 µm wide and 2-4 µm long, motile, rod-shaped and non-spore-forming. The four major fatty acids were anteiso-C15 : 0, iso-C15 : 0, iso-C17 : 0 and anteiso-C17 : 0. Growth was observed from 15 to 45 °C (optimum 40 °C) and at pH 5.5-8 (optimum pH 7.5). The salinity range for growth was 5-65 g NaCl l-1 (optimum 30 g l-1). Yeast extract was required for growth. Strain BLaC1T was able to grow on lactate and acetate in the presence of sulfate as an electron acceptor. Sulfate, thiosulfate and sulfite could serve as terminal electron acceptors, but not fumarate, nitrate or elemental sulfur. The DNA G+C content was 55.8 mol%. 16S rRNA gene sequence analysis assigned strain BLaC1T to the family Desulfovibrionaceae; its closest relative was Desulfovibrio oxyclinae DSM 19275T (93.7 % similarity). On the basis of 16S rRNA gene sequence comparisons and physiological characteristics, strain BLaC1T is proposed as representing a novel species of Desulfovibrio, with the name Desulfovibrio senegalensis sp. nov. The type strain is BLaC1T (=DSM 101509T=JCM 31063T).
[Mh] Termos MeSH primário: Desulfovibrio/classificação
Sedimentos Geológicos/microbiologia
Filogenia
Água do Mar/microbiologia
[Mh] Termos MeSH secundário: Técnicas de Tipagem Bacteriana
Composição de Bases
DNA Bacteriano/genética
Desulfovibrio/genética
Desulfovibrio/isolamento & purificação
Ácidos Graxos/química
Oxirredução
RNA Ribossômico 16S/genética
Senegal
Análise de Sequência de DNA
Sulfatos/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (DNA, Bacterial); 0 (Fatty Acids); 0 (RNA, Ribosomal, 16S); 0 (Sulfates)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170920
[Lr] Data última revisão:
170920
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170905
[St] Status:MEDLINE
[do] DOI:10.1099/ijsem.0.001997


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[PMID]:28436827
[Au] Autor:Zhou C; Zhou Y; Rittmann BE
[Ad] Endereço:Swette Center for Environmental Biotechnology, Biodesign Institute, Arizona State University, USA. Electronic address: zhou_SCEB@asu.edu.
[Ti] Título:Reductive precipitation of sulfate and soluble Fe(III) by Desulfovibrio vulgaris: Electron donor regulates intracellular electron flow and nano-FeS crystallization.
[So] Source:Water Res;119:91-101, 2017 Aug 01.
[Is] ISSN:1879-2448
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Fully understanding the metabolism of SRB provides fundamental guidelines for allowing the microorganisms to provide more beneficial services in water treatment and resource recovery. The electron-transfer pathway of sulfate respiration by Desulfovibrio vulgaris is well studied, but still partly unresolved. Here we provide deeper insight by comprehensively monitoring metabolite changes during D. vulgaris metabolism with two electron donors, lactate and pyruvate, in presence or absence of citrate-chelated soluble Fe as an additional competing electron acceptor. H was produced from lactate oxidation to pyruvate, but pyruvate oxidation produced mostly formate. Accumulation of lactate-originated H during lag phases inhibited pyruvate transformation to acetate. Sulfate reduction was initiated by lactate-originated H , but MQ-mediated e flow initiated sulfate reduction without delay when pyruvate was the donor. When H -induced electron flow gave priority to Fe reduction over sulfate reduction, the long lag phase before sulfate reduction shortened the time for iron-sulfide crystallite growth and led to smaller mackinawite (Fe S) nanocrystallites. Synthesizing all the results, we propose that electron flow from lactate or pyruvate towards SO reduction to H S are through at least three routes that are regulated by the e donor (lactate or pyruvate) and the presence or absence of another e acceptor (Fe here). These routes are not competing, but complementary: e.g., H or formate production and oxidation were necessary for sulfite and disulfide/trisulfide reduction to sulfide. Our study suggests that the e donor provides a practical tool to regulate and optimize SRB-predominant bioremediation systems.
[Mh] Termos MeSH primário: Desulfovibrio vulgaris
Compostos Férricos/química
Sulfatos/química
[Mh] Termos MeSH secundário: Cristalização
Desulfovibrio
Elétrons
Oxirredução
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Ferric Compounds); 0 (Sulfates)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171019
[Lr] Data última revisão:
171019
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170425
[St] Status:MEDLINE


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[PMID]:28375578
[Au] Autor:Yuan H; He Z
[Ad] Endereço:Department of Civil and Environmental Engineering, Virginia Polytechnic Institute and State University, Blacksburg, VA 24061, USA.
[Ti] Título:Platinum Group Metal-free Catalysts for Hydrogen Evolution Reaction in Microbial Electrolysis Cells.
[So] Source:Chem Rec;17(7):641-652, 2017 07.
[Is] ISSN:1528-0691
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Hydrogen gas is a green energy carrier with great environmental benefits. Microbial electrolysis cells (MECs) can convert low-grade organic matter to hydrogen gas with low energy consumption and have gained a growing interest in the past decade. Cathode catalysts for the hydrogen evolution reaction (HER) present a major challenge for the development and future applications of MECs. An ideal cathode catalyst should be catalytically active, simple to synthesize, durable in a complex environment, and cost-effective. A variety of noble-metal free catalysts have been developed and investigated for HER in MECs, including Nickel and its alloys, MoS , carbon-based catalysts and biocatalysts. MECs in turn can serve as a research platform to study the durability of the HER catalysts. This personal account has reviewed, analyzed, and discussed those catalysts with an emphasis on synthesis and modification, system performance and potential for practical applications. It is expected to provide insights into the development of HER catalysts towards MEC applications.
[Mh] Termos MeSH primário: Fontes de Energia Bioelétrica/microbiologia
Hidrogênio/metabolismo
Metais/química
[Mh] Termos MeSH secundário: Carbono/química
Catálise
Desulfovibrio/química
Desulfovibrio/metabolismo
Eletrólise
Geobacter/química
Geobacter/metabolismo
Hidrogênio/química
Platina/química
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Metals); 49DFR088MY (Platinum); 7440-44-0 (Carbon); 7YNJ3PO35Z (Hydrogen)
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171113
[Lr] Data última revisão:
171113
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170405
[St] Status:MEDLINE
[do] DOI:10.1002/tcr.201700007


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Martins, Monica
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[PMID]:28365342
[Au] Autor:Mourato C; Martins M; da Silva SM; Pereira IAC
[Ad] Endereço:Instituto de Tecnologia Química e Biológica António Xavier/Universidade Nova de Lisboa (ITQB NOVA), Av. da Republica-EAN, 2780-157 Oeiras, Portugal.
[Ti] Título:A continuous system for biocatalytic hydrogenation of CO to formate.
[So] Source:Bioresour Technol;235:149-156, 2017 Jul.
[Is] ISSN:1873-2976
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:In this work a novel bioprocess for hydrogenation of CO to formate was developed, using whole cell catalysis by a sulfate-reducing bacterium. Three Desulfovibrio species were tested (D. vulgaris Hildenborough, D. alaskensis G20, and D. desulfuricans ATCC 27774), of which D. desulfuricans showed the highest activity, producing 12mM of formate in batch, with a production rate of 0.09mMh . Gene expression analysis indicated that among the three formate dehydrogenases and five hydrogenases, the cytoplasmic FdhAB and the periplasmic [FeFe] HydAB are the main enzymes expressed in D. desulfuricans in these conditions. The new bioprocess for continuous formate production by D. desulfuricans had a maximum specific formate production rate of 14mMg h , and more than 45mM of formate were obtained with a production rate of 0.40mMh . This is the first report of a continuous process for biocatalytic formate production.
[Mh] Termos MeSH primário: Dióxido de Carbono/metabolismo
Formiato Desidrogenases/metabolismo
Formiatos/metabolismo
[Mh] Termos MeSH secundário: Biocatálise
Desulfovibrio/metabolismo
Hidrogenação
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Formates); 142M471B3J (Carbon Dioxide); EC 1.2.1.2 (Formate Dehydrogenases)
[Em] Mês de entrada:1706
[Cu] Atualização por classe:170601
[Lr] Data última revisão:
170601
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170403
[St] Status:MEDLINE


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[PMID]:28264778
[Au] Autor:Qian J; Wang L; Wu Y; Bond PL; Zhang Y; Chang X; Deng B; Wei L; Li Q; Wang Q
[Ad] Endereço:Department of Applied Chemistry, School of Natural and Applied Sciences, Northwestern Polytechnical University, NO. 127 West Youyi Road, Xi'an 710072, China; State Key Laboratory of Geohazard Prevention and Geoenvironment Protection, Chengdu University of Technology, China; Research Center for Ecolo
[Ti] Título:Free sulfurous acid (FSA) inhibition of biological thiosulfate reduction (BTR) in the sulfur cycle-driven wastewater treatment process.
[So] Source:Chemosphere;176:212-220, 2017 Jun.
[Is] ISSN:1879-1298
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:A sulfur cycle-based bioprocess for co-treatment of wet flue gas desulfurization (WFGD) wastes with freshwater sewage has been developed. In this process the removal of organic carbon is mainly associated with biological sulfate or sulfite reduction. Thiosulfate is a major intermediate during biological sulfate/sulfite reduction, and its reduction to sulfide is the rate-limiting step. In this study, the impacts of saline sulfite (the ionized form: HSO + SO ) and free sulfurous acid (FSA, the unionized form: H SO ) sourced from WGFD wastes on the biological thiosulfate reduction (BTR) activities were thoroughly investigated. The BTR activity and sulfate/sulfite-reducing bacteria (SRB) populations in the thiosulfate-reducing up-flow anaerobic sludge bed (UASB) reactor decreased when the FSA was added to the UASB influent. Batch experiment results confirmed that FSA, instead of saline sulfite, was the true inhibitor of BTR. And BTR activities dropped by 50% as the FSA concentrations were increased from 8.0 × 10 to 2.0 × 10 mg H SO -S/L. From an engineering perspective, the findings of this study provide some hints on how to ensure effective thiosulfate accumulation in biological sulfate/sulfite reduction for the subsequent denitrification/denitritation. Such manipulation would result in higher nitrogen removal rates in this co-treatment process of WFGD wastes with municipal sewage.
[Mh] Termos MeSH primário: Biodegradação Ambiental
Esgotos/microbiologia
Enxofre/química
Tiossulfatos/metabolismo
Águas Residuais/química
[Mh] Termos MeSH secundário: Reatores Biológicos/microbiologia
Desnitrificação
Desulfovibrio/metabolismo
Água Doce/microbiologia
Oxirredução
Tiossulfatos/antagonistas & inibidores
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Sewage); 0 (Thiosulfates); 0 (Waste Water); 70FD1KFU70 (Sulfur)
[Em] Mês de entrada:1704
[Cu] Atualização por classe:170428
[Lr] Data última revisão:
170428
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170308
[St] Status:MEDLINE


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[PMID]:28229521
[Au] Autor:Freedman AJE; Tan B; Thompson JR
[Ad] Endereço:Department of Civil and Environmental Engineering, Massachusetts Institute of Technology, Cambridge, MA, USA.
[Ti] Título:Microbial potential for carbon and nutrient cycling in a geogenic supercritical carbon dioxide reservoir.
[So] Source:Environ Microbiol;19(6):2228-2245, 2017 Jun.
[Is] ISSN:1462-2920
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Microorganisms catalyze carbon cycling and biogeochemical reactions in the deep subsurface and thus may be expected to influence the fate of injected supercritical (sc) CO following geological carbon sequestration (GCS). We hypothesized that natural subsurface scCO reservoirs, which serve as analogs for the long-term fate of sequestered scCO , harbor a 'deep carbonated biosphere' with carbon cycling potential. We sampled subsurface fluids from scCO -water separators at a natural scCO reservoir at McElmo Dome, Colorado for analysis of 16S rRNA gene diversity and metagenome content. Sequence annotations indicated dominance of Sulfurospirillum, Rhizobium, Desulfovibrio and four members of the Clostridiales family. Genomes extracted from metagenomes using homology and compositional approaches revealed diverse mechanisms for growth and nutrient cycling, including pathways for CO and N fixation, anaerobic respiration, sulfur oxidation, fermentation and potential for metabolic syntrophy. Differences in biogeochemical potential between two production well communities were consistent with differences in fluid chemical profiles, suggesting a potential link between microbial activity and geochemistry. The existence of a microbial ecosystem associated with the McElmo Dome scCO reservoir indicates that potential impacts of the deep biosphere on CO fate and transport should be taken into consideration as a component of GCS planning and modelling.
[Mh] Termos MeSH primário: Dióxido de Carbono/metabolismo
Clostridiales/metabolismo
Desulfovibrio/metabolismo
Epsilonproteobacteria/metabolismo
Rhizobium/metabolismo
[Mh] Termos MeSH secundário: Carbono/metabolismo
Ciclo do Carbono/fisiologia
Sequestro de Carbono/fisiologia
Clostridiales/classificação
Clostridiales/genética
Colorado
Desulfovibrio/classificação
Desulfovibrio/genética
Ecossistema
Epsilonproteobacteria/classificação
Epsilonproteobacteria/genética
Genoma Bacteriano/genética
Metagenoma
RNA Ribossômico 16S/genética
Rhizobium/classificação
Rhizobium/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (RNA, Ribosomal, 16S); 142M471B3J (Carbon Dioxide); 7440-44-0 (Carbon)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171031
[Lr] Data última revisão:
171031
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170224
[St] Status:MEDLINE
[do] DOI:10.1111/1462-2920.13706



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