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Pesquisa : C04.588.322 [Categoria DeCS]
Referências encontradas : 793 [refinar]
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[PMID]:29361619
[Au] Autor:Saeki T
[Ad] Endereço:Dept. of Breast Oncology, Comprehensive Cancer Center, Saitama Medical University International Medical Center.
[Ti] Título:[Breast and Endocrine Tumor Supportive Care for Febrile Neutropenia in Chemotherapy - Current Situation and Future Perspective in Breast Cancer Treatment -].
[So] Source:Gan To Kagaku Ryoho;44(13):2072, 2017 Dec.
[Is] ISSN:0385-0684
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Mh] Termos MeSH primário: Antineoplásicos/uso terapêutico
Neoplasias da Mama/tratamento farmacológico
Neoplasias das Glândulas Endócrinas/tratamento farmacológico
Neutropenia Febril/prevenção & controle
[Mh] Termos MeSH secundário: Neutropenia Febril/induzido quimicamente
Seres Humanos
Guias de Prática Clínica como Assunto
[Pt] Tipo de publicação:INTRODUCTORY JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Antineoplastic Agents)
[Em] Mês de entrada:1803
[Cu] Atualização por classe:180302
[Lr] Data última revisão:
180302
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180124
[St] Status:MEDLINE


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[PMID]:28347154
[Au] Autor:Decmann Á; Perge P; Nagy Z; Butz H; Patócs A; Igaz P
[Ad] Endereço:II. Belgyógyászati Klinika, Semmelweis Egyetem, Általános Orvostudományi Kar Budapest, Ülloi út 26., 1085.
[Ti] Título:[Circulating microRNAs in the diagnostics of endocrine neoplasms].
[Ti] Título:Keringo mikroRNS-ek az endokrin daganatok diagnosztikájában..
[So] Source:Orv Hetil;158(13):483-490, 2017 Apr.
[Is] ISSN:0030-6002
[Cp] País de publicação:Hungary
[La] Idioma:hun
[Ab] Resumo:MicroRNAs (miRNA, miR) are short - 19-25 nucleotide long - single stranded (in their mature form), non-coding RNA molecules that regulate gene expression mostly at the posttranscriptional level. microRNAs are involved in the regulation of various physiological processes such as cell differentiation and proliferation, development, haematopoesis, cell death, while their aberrant expression is observed in numerous diseases, like autoimmune disorders, inflammations, vascular diseases or tumorigenesis. microRNAs are expressed in a tissue specific fashion. Beyond their appearance in tissues, they can be found in body fluids as well. microRNAs are present in blood, mother milk, semen, saliva, urine, etc. MicroRNAs in body fluids, especially the blood-borne circulating microRNAs can be exploited as minimally invasive biomarkers of tumor diagnosis. The number of endocrine tumor-associated circulating microRNA alterations is relatively low, mostly described for papillary thyroid cancer, adrenocortical cancer, ovarian and neuroendocrine tumors. As the histological diagnosis including the establishment of malignancy of some of these neoplasms is difficult, studies on circulating microRNAs might have great perspectives. Orv. Hetil., 2017, 158(13), 483-490.
[Mh] Termos MeSH primário: Biomarcadores Tumorais/sangue
Neoplasias das Glândulas Endócrinas/sangue
Regulação Neoplásica da Expressão Gênica
MicroRNAs/sangue
[Mh] Termos MeSH secundário: Neoplasias das Glândulas Endócrinas/diagnóstico
Neoplasias das Glândulas Endócrinas/genética
Perfilação da Expressão Gênica
Seres Humanos
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Biomarkers, Tumor); 0 (MicroRNAs)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170808
[Lr] Data última revisão:
170808
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170329
[St] Status:MEDLINE
[do] DOI:10.1556/650.2017.30708


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[PMID]:28220018
[Au] Autor:De Sousa SM; McCabe MJ; Wu K; Roscioli T; Gayevskiy V; Brook K; Rawlings L; Scott HS; Thompson TJ; Earls P; Gill AJ; Cowley MJ; Dinger ME; McCormack AI
[Ad] Endereço:Hormones and Cancer GroupGarvan Institute of Medical Research, Sydney, Australia.
[Ti] Título:Germline variants in familial pituitary tumour syndrome genes are common in young patients and families with additional endocrine tumours.
[So] Source:Eur J Endocrinol;176(5):635-644, 2017 May.
[Is] ISSN:1479-683X
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: Familial pituitary tumour syndromes (FPTS) account for 5% of pituitary adenomas. Multi-gene analysis via next-generation sequencing (NGS) may unveil greater prevalence and inform clinical care. We aimed to identify germline variants in selected patients with pituitary adenomas using a targeted NGS panel. DESIGN: We undertook a nationwide cross-sectional study of patients with pituitary adenomas with onset ≤40 years of age and/or other personal/family history of endocrine neoplasia. A custom NGS panel was performed on germline DNA to interrogate eight FPTS genes. Genome data were analysed via a custom bioinformatic pipeline, and validation was performed by Sanger sequencing. Multiplex ligation-dependent probe amplification (MLPA) was performed in cases with heightened suspicion for , and mutations. The main outcomes were frequency and pathogenicity of rare variants in , , , , , , and . RESULTS: Forty-four patients with pituitary tumours, 14 of whom had a personal history of other endocrine tumours and/or a family history of pituitary or other endocrine tumours, were referred from endocrine tertiary-referral centres across Australia. Eleven patients (25%) had a rare variant across the eight FPTS genes tested: (p.A299V, p.R106C, p.F269F, p.R304X, p.K156K, p.R271W), (p.R176Q), (p.A2V, p.S8S), (p.E110Q) and (p.G12S), with two patients harbouring dual variants. Variants were classified as pathogenic or of uncertain significance in 9/44 patients (20%). No deletions/duplications were identified in , or . CONCLUSIONS: A high yield of rare variants in genes implicated in FPTS can be found in selected patients using an NGS panel. It may also identify individuals harbouring more than one rare variant.
[Mh] Termos MeSH primário: Biomarcadores Tumorais/genética
Neoplasias das Glândulas Endócrinas/epidemiologia
Neoplasias das Glândulas Endócrinas/genética
Variação Genética/genética
Mutação em Linhagem Germinativa/genética
Neoplasias Hipofisárias/genética
[Mh] Termos MeSH secundário: Adulto
Austrália/epidemiologia
Estudos Transversais
Neoplasias das Glândulas Endócrinas/diagnóstico
Feminino
Seres Humanos
Masculino
Neoplasias Hipofisárias/epidemiologia
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Biomarkers, Tumor)
[Em] Mês de entrada:1704
[Cu] Atualização por classe:170413
[Lr] Data última revisão:
170413
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170222
[St] Status:MEDLINE
[do] DOI:10.1530/EJE-16-0944


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[PMID]:28135773
[Au] Autor:Evers M; Rechnitzer C; Graem N; Skov Wehner P; Schroeder H; Rosthoej S; Mosbech CH; Hoei-Hansen CE; Sehested A; Treger TD; Brok J
[Ad] Endereço:Department of Paediatric and Adolescent Haematology and Oncology, Rigshospitalet, Copenhagen University Hospital, Copenhagen, Denmark.
[Ti] Título:Epidemiological study of paediatric germ cell tumours revealed the incidence and distribution that was expected, but a low mortality rate.
[So] Source:Acta Paediatr;106(5):779-785, 2017 May.
[Is] ISSN:1651-2227
[Cp] País de publicação:Norway
[La] Idioma:eng
[Ab] Resumo:AIM: Germ cell tumours (GCTs) are a rare heterogeneous tumour group derived from primordial germ cells, which can be benign or malignant and occur in the gonads or extragonadally. This study mapped the paediatric GCTs in Denmark from 1984 to 2013 to study the incidence and outcome. METHODS: We identified paediatric GCTs from the Danish Childhood Cancer and National Pathology Registries and reviewed the case records for patient characteristics, tumour characteristics and clinical outcome. RESULTS: We identified 403 (71% female) paediatric GCTs and the crude incidence was 1.43 per 100 000. Of these, 79 (20%) were malignant, 39 (10%) were potentially malignant and 285 (70%) were benign. Extragonadal GCTs (39%) were mainly observed in early childhood and were predominately sacrococcygeal teratomas. Gonadal GCTs (61%) in late childhood were most frequently mature teratomas in the ovaries. Nearly all patients underwent surgery. Of the malignant tumours, 62% were treated with chemotherapy. Radiotherapy was only administered to intracranial GCTs. In the cohort, 12 patients died (3%). CONCLUSION: Paediatric GCTs in Denmark were mainly benign and mortality was low, even for malignant tumours. We identified a peak of extragonadal GCTs in early childhood and a peak of gonadal GCTs in late childhood, which was comparable to previous reports.
[Mh] Termos MeSH primário: Neoplasias das Glândulas Endócrinas/mortalidade
Neoplasias Embrionárias de Células Germinativas/mortalidade
[Mh] Termos MeSH secundário: Adolescente
Criança
Pré-Escolar
Dinamarca/epidemiologia
Neoplasias das Glândulas Endócrinas/terapia
Feminino
Seres Humanos
Incidência
Lactente
Masculino
Neoplasias Embrionárias de Células Germinativas/terapia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171016
[Lr] Data última revisão:
171016
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170131
[St] Status:MEDLINE
[do] DOI:10.1111/apa.13767


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[PMID]:28035432
[Au] Autor:Pelage JP; Fohlen A; Mitry E; Lagrange C; Beauchet A; Rougier P
[Ad] Endereço:Department of Diagnostic Imaging and Interventional Radiology, Caen University and Medical Center, Avenue Cote de Nacre, 14033, Caen Cedex 9, France. pelage-jp@chu-caen.fr.
[Ti] Título:Chemoembolization of Neuroendocrine Liver Metastases Using Streptozocin and Tris-acryl Microspheres: Embozar (EMBOsphere + ZAnosaR) Study.
[So] Source:Cardiovasc Intervent Radiol;40(3):394-400, 2017 Mar.
[Is] ISSN:1432-086X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:PURPOSE: The purpose of this prospective observational study was to evaluate the efficacy and tolerability of transarterial chemoembolization (TACE) for neuroendocrine liver metastases using a combination of streptozocin, Lipiodol, and tris-acryl microspheres. PATIENTS AND METHODS: A total of 16 men and 9 women aged 59.6 ± 11.3 years, all with predominant liver disease, underwent 54 courses of TACE using an emulsion of 1.5 g of streptozocin and 10 ml of Lipiodol. Additional embolization was performed using 300-500 µm tris-acryl microspheres. Morphological response was evaluated using the RECIST criteria on multi-detector computed tomography or MRI. Clinical efficacy was evaluated particularly in patients with carcinoid syndrome. RESULTS: The primary tumor was located in the small bowel or pancreas in 21 (84%) patients. Eleven (44%) patients presented with a carcinoid syndrome. Nineteen (76%) patients presented with more than 10 liver nodules. One delayed case of ischemic cholecystitis was treated conservatively. After a median follow-up of 36.1 months, 1 (4%) patient had a complete response, 12 (48%) patients had a partial response, and 7 (28%) patients had a stable disease corresponding to a disease control rate of 80%. All patients with carcinoid syndrome had significant improvement. Median time to progression was 18.8 months and overall survival was 100, 100, and 92% at 1, 2, and 3 years, respectively. Seven patients presented with extrahepatic progression with abdominal lymphadenopathies or metastases to the brain, ovary, adrenal gland, or lung. CONCLUSION: Optimized TACE using a combination of streptozocin, Lipiodol, and tris-acryl microspheres is effective and well tolerated.
[Mh] Termos MeSH primário: Resinas Acrílicas/administração & dosagem
Quimioembolização Terapêutica/métodos
Neoplasias das Glândulas Endócrinas/patologia
Gelatina/administração & dosagem
Neoplasias Hepáticas/secundário
Neoplasias Hepáticas/terapia
Estreptozocina/administração & dosagem
[Mh] Termos MeSH secundário: Adulto
Idoso
Antibióticos Antineoplásicos/administração & dosagem
Óleo Etiodado/administração & dosagem
Feminino
Seres Humanos
Fígado/diagnóstico por imagem
Neoplasias Hepáticas/diagnóstico por imagem
Imagem por Ressonância Magnética
Masculino
Meia-Idade
Estudos Prospectivos
Tomografia Computadorizada por Raios X
Resultado do Tratamento
[Pt] Tipo de publicação:JOURNAL ARTICLE; OBSERVATIONAL STUDY
[Nm] Nome de substância:
0 (Acrylic Resins); 0 (Antibiotics, Antineoplastic); 0 (trisacryl gelatin microspheres); 5W494URQ81 (Streptozocin); 8008-53-5 (Ethiodized Oil); 9000-70-8 (Gelatin)
[Em] Mês de entrada:1705
[Cu] Atualização por classe:171114
[Lr] Data última revisão:
171114
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161231
[St] Status:MEDLINE
[do] DOI:10.1007/s00270-016-1535-7


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[PMID]:27943004
[Au] Autor:Stratakis CA
[Ad] Endereço:Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, Building 10, Room East 1330, CRC, 10 Center Dr. MSC1862, Bethesda, MD, 20892-1862, USA. stratakc@mail.nih.gov.
[Ti] Título:Carney complex: A familial lentiginosis predisposing to a variety of tumors.
[So] Source:Rev Endocr Metab Disord;17(3):367-371, 2016 Sep.
[Is] ISSN:1573-2606
[Cp] País de publicação:Germany
[La] Idioma:eng
[Ab] Resumo:Carney complex is a familial lentiginosis syndrome; these disorders cover a wide phenotypic spectrum ranging from a benign inherited predisposition to develop cutaneous spots not associated with systemic disease to associations with several syndromes. Carney complex is caused by PRKAR1A mutations and perturbations of the cyclic AMP-dependent protein kinase (PKA) signaling pathway. In addition to the cutaneous findings, the main tumors associated with Carney complex are endocrine: 1) primary pigmented nodular adrenocortical disease, a bilateral adrenal hyperplasia leading to Cushing syndrome; 2) growth-hormone secreting pituitary adenoma or pituitary somatotropic hyperplasia leading to acromegaly; 3) thyroid and gonadal tumors, including a predisposition to thyroid cancer. Other tumors associated with Carney complex include: 1) myxomas of the heart, breast and other sites; 2) psamommatous melanotic schwannomas which can become malignant; 4) a predisposition to a variety of cancers.
[Mh] Termos MeSH primário: Complexo de Carney/patologia
Neoplasias das Glândulas Endócrinas/patologia
Doenças do Sistema Endócrino/patologia
Lentigo/patologia
[Mh] Termos MeSH secundário: Complexo de Carney/genética
Neoplasias das Glândulas Endócrinas/genética
Doenças do Sistema Endócrino/genética
Seres Humanos
Lentigo/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171004
[Lr] Data última revisão:
171004
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161213
[St] Status:MEDLINE


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[PMID]:27911704
[Au] Autor:O'Toole SM; Chapple JP
[Ad] Endereço:Centre for Endocrinology, William Harvey Research Institute, Barts and the London School of Medicine and Dentistry, Queen Mary University of London, London, U.K.
[Ti] Título:Primary cilia: a link between hormone signalling and endocrine-related cancers?
[So] Source:Biochem Soc Trans;44(5):1227-1234, 2016 Oct 15.
[Is] ISSN:1470-8752
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Primary cilia are sensory organelles that play a role as signalling hubs. Disruption of primary cilia structure and function is increasingly recognised in a range of cancers, with a growing body of evidence suggesting that ciliary disruption contributes to tumourigenesis. This review considers the role of primary cilia in the pathogenesis of endocrine-related cancers.
[Mh] Termos MeSH primário: Cílios/metabolismo
Hormônios/metabolismo
Neoplasias/metabolismo
Transdução de Sinais
[Mh] Termos MeSH secundário: Animais
Axonema/metabolismo
Corpos Basais/metabolismo
Cílios/patologia
Neoplasias das Glândulas Endócrinas/metabolismo
Neoplasias das Glândulas Endócrinas/patologia
Seres Humanos
Modelos Biológicos
Neoplasias/patologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Hormones)
[Em] Mês de entrada:1707
[Cu] Atualização por classe:170714
[Lr] Data última revisão:
170714
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161203
[St] Status:MEDLINE


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[PMID]:27899191
[Au] Autor:Petr EJ; Else T
[Ad] Endereço:Department of Internal Medicine, Division of Metabolism, Endocrinology and Diabetes, University of Michigan Health System, Ann Arbor, MI.
[Ti] Título:Genetic predisposition to endocrine tumors: Diagnosis, surveillance and challenges in care.
[So] Source:Semin Oncol;43(5):582-590, 2016 Oct.
[Is] ISSN:1532-8708
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Endocrine tumor syndromes, eg, multiple endocrine neoplasia types 1 and 2, were among the first recognized hereditary predisposition syndromes to tumor development. Over time, the number of endocrine tumor syndromes has significantly expanded, eg, with the recent inclusion of hereditary paraganglioma syndromes. Associations of non-endocrine tumors with hereditary endocrine tumor syndromes and endocrine tumors with non-classical endocrine tumor syndromes have emerged. These findings have certainly expanded the scope of care, necessitating a multidisciplinary approach by a team of medical professionals and researchers, integrating shared patient decision-making at every step of surveillance, diagnosis, and treatment. In the absence of evidence-based guidelines, multiple aspects of patient care remain individualized, based on a patient's clinical presentation and family pedigree. This is particularly important when determining a surveillance plan for unaffected or disease-free mutation carriers. In this review, we describe the main endocrine tumor manifestations found in familial cancer syndromes in an organ-based approach, focusing on adrenocortical carcinoma, pheochromocytoma and paraganglioma, neuroendocrine tumors, differentiated thyroid cancer, and medullary thyroid cancer. We highlight the challenges in diagnosis, surveillance, and therapy unique to the patient population with hereditary syndromes. Furthermore, we underscore the importance of evaluating for genetic predisposition to tumor development, provide features that can identify index patients, and discuss the approach to screening surveillance for mutation carriers.
[Mh] Termos MeSH primário: Neoplasias das Glândulas Endócrinas/genética
Predisposição Genética para Doença
[Mh] Termos MeSH secundário: Neoplasias das Glândulas Endócrinas/diagnóstico
Seres Humanos
Neoplasia Endócrina Múltipla Tipo 2a/genética
Tumores Neuroendócrinos/genética
PTEN Fosfo-Hidrolase/genética
Feocromocitoma/genética
Succinato Desidrogenase/genética
Neoplasias da Glândula Tireoide/genética
Doença de von Hippel-Lindau/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
EC 1.3.99.1 (Succinate Dehydrogenase); EC 3.1.3.67 (PTEN Phosphohydrolase); EC 3.1.3.67 (PTEN protein, human)
[Em] Mês de entrada:1705
[Cu] Atualização por classe:171024
[Lr] Data última revisão:
171024
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161201
[St] Status:MEDLINE


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[PMID]:27367344
[Au] Autor:Antoniv VF; Koval' IV; Popadyuk VI; Antoniv TV; Aksenov VM
[Ad] Endereço:Russian University of People's Friendship, Moscow, Russia, 117198.
[Ti] Título:[Etiology and pathogenesis of glomus tumours (chemodectomas) of the base of the skull].
[So] Source:Vestn Otorinolaringol;81(3):26-29, 2016.
[Is] ISSN:0042-4668
[Cp] País de publicação:Russia (Federation)
[La] Idioma:rus
[Ab] Resumo:The objective of the present work was to describe the poorly known history of the development of the nomenclature of glomus tumours (chemodectomas) of the base of the skull, elucidate etiology and pathogenesis of these neoplasms. The authors present a chronological table illustrating the progress of etiological and pathogenetic studies as well as the surgical treatment of the tumours. The results of analysis and discussion of the controversial issues of the nomenclature are reported.
[Mh] Termos MeSH primário: Neoplasias das Glândulas Endócrinas/patologia
Tumor Glômico
[Mh] Termos MeSH secundário: Adulto
Idade de Início
Feminino
Tumor Glômico/epidemiologia
Tumor Glômico/etiologia
Tumor Glômico/patologia
Tumor Glômico/fisiopatologia
Tumor Glômico/cirurgia
Serviços de Saúde
Seres Humanos
Masculino
Meia-Idade
Fatores Sexuais
Neoplasias da Base do Crânio/epidemiologia
Neoplasias da Base do Crânio/etiologia
Neoplasias da Base do Crânio/patologia
Neoplasias da Base do Crânio/fisiopatologia
Neoplasias da Base do Crânio/cirurgia
Terminologia como Assunto
[Pt] Tipo de publicação:ENGLISH ABSTRACT; JOURNAL ARTICLE
[Em] Mês de entrada:1609
[Cu] Atualização por classe:170621
[Lr] Data última revisão:
170621
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160702
[St] Status:MEDLINE
[do] DOI:10.17116/otorino201681326-29


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[PMID]:27338155
[Au] Autor:Jakubowski CD; Vertosick EA; Untch BR; Sjoberg D; Wei E; Palmer FL; Patel SG; Downey RJ; Strong VE; Russo P
[Ad] Endereço:Urology Service, Memorial Sloan Kettering Cancer Center, New York, New York.
[Ti] Título:Complete metastasectomy for renal cell carcinoma: Comparison of five solid organ sites.
[So] Source:J Surg Oncol;114(3):375-9, 2016 Sep.
[Is] ISSN:1096-9098
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND AND OBJECTIVES: Patients with metastatic RCC can undergo metastasectomy to improve survival time. Our goal was to provide and compare characteristics and oncological outcomes of RCC patients who underwent complete metastasectomy at a single organ site. METHODS: A total of 138 RCC patients were identified as undergoing complete metastasectomy at a single organ site including adrenal, lung, liver, pancreas, or thyroid. Competing risk regression analysis was used to assess RFS and CSS adjusting for several covariates. RESULTS: In this highly selected cohort, RFS and CSS was 27% and 84% at 5 years following metastasectomy, respectively. Univariate analysis revealed that removal of multiple tumors, younger age, and a shorter interval between nephrectomy and metastasis was associated with worse RFS. Larger tumors and sarcomatoid histology at nephrectomy was associated with worse CSS. We found no evidence that metastases at the time of RCC diagnosis influenced recurrence or survival. Tumor size, number of metastases resected, and time from nephrectomy to first recurrence was significantly different, but recurrence rates were not found to be significantly different, when compared across all organ sites. CONCLUSIONS: These findings inform clinical and surgical management of select RCC patients with isolated metastasis to one of several organ sites. J. Surg. Oncol. 2016;114:375-379. © 2016 Wiley Periodicals, Inc.
[Mh] Termos MeSH primário: Carcinoma de Células Renais/secundário
Carcinoma de Células Renais/cirurgia
Neoplasias das Glândulas Endócrinas/cirurgia
Neoplasias Hepáticas/cirurgia
Neoplasias Pulmonares/cirurgia
Metastasectomia
[Mh] Termos MeSH secundário: Idoso
Carcinoma de Células Renais/mortalidade
Estudos de Coortes
Neoplasias das Glândulas Endócrinas/mortalidade
Neoplasias das Glândulas Endócrinas/secundário
Feminino
Seres Humanos
Neoplasias Renais/mortalidade
Neoplasias Renais/patologia
Neoplasias Renais/cirurgia
Neoplasias Hepáticas/mortalidade
Neoplasias Hepáticas/secundário
Neoplasias Pulmonares/mortalidade
Neoplasias Pulmonares/secundário
Masculino
Meia-Idade
Nefrectomia
Resultado do Tratamento
[Pt] Tipo de publicação:COMPARATIVE STUDY; JOURNAL ARTICLE
[Em] Mês de entrada:1702
[Cu] Atualização por classe:170902
[Lr] Data última revisão:
170902
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160625
[St] Status:MEDLINE
[do] DOI:10.1002/jso.24327



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