Base de dados : MEDLINE
Pesquisa : C04.588.614.550.700 [Categoria DeCS]
Referências encontradas : 224 [refinar]
Mostrando: 1 .. 10   no formato [Detalhado]

página 1 de 23 ir para página                         

  1 / 224 MEDLINE  
              next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:27235344
[Au] Autor:Berger B; Stich O; Labeit S; Rauer S
[Ad] Endereço:Department of Neurology and Neurophysiology, University Hospital Freiburg, Breisacher Straße 64, D-79106 Freiburg, Germany. Electronic address: benjamin.berger@uniklinik-freiburg.de.
[Ti] Título:Screening for anti-titin antibodies in patients with various paraneoplastic neurological syndromes.
[So] Source:J Neuroimmunol;295-296:18-20, 2016 06 15.
[Is] ISSN:1872-8421
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Anti-titin antibodies indicate a paraneoplastic etiology pointing towards a thymoma in myasthenia gravis (MG), but their seroprevalence and potential diagnostic value in patients with other paraneoplastic neurological syndromes (PNS) is unknown. Therefore, we screened the sera of 44 PNS patients with well-characterized onconeural antibodies (anti-Hu, Yo, Ri, CV2/CRMP5, Ma1, Ma2/Ta, or amphiphysin) for anti-titin reactivity. Two patients (4.5%) were positive for anti-titin antibodies: both patients differed regarding the PNS (sensorimotor neuropathy and subacute cerebellar degeneration vs. chorea), well-characterized onconeural antibodies (CV2/CRMP5 vs. Ri), and malignoma (small cell lung cancer vs. breast cancer). However, retrospectively, the patients neither showed any symptoms of MG nor a thymoma on a computed tomographic (CT) scan. The results of this study indicate that anti-titin antibodies without a predictive relevance for MG or thymoma may be present in a small proportion of patients with PNS.
[Mh] Termos MeSH primário: Autoanticorpos/sangue
Conectina/imunologia
Polineuropatia Paraneoplásica
[Mh] Termos MeSH secundário: Adolescente
Adulto
Idoso
Idoso de 80 Anos ou mais
Feminino
Seres Humanos
Masculino
Programas de Rastreamento/métodos
Meia-Idade
Polineuropatia Paraneoplásica/diagnóstico
Polineuropatia Paraneoplásica/imunologia
Polineuropatia Paraneoplásica/metabolismo
Timoma/sangue
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Autoantibodies); 0 (Connectin)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:171121
[Lr] Data última revisão:
171121
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160529
[St] Status:MEDLINE


  2 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:26795713
[Au] Autor:Alam T; Barker AS; Alix JJ; Hadjivassiliou M; Rao DG
[Ad] Endereço:1Department of Clinical Neurophysiology,Royal Hallamshire Hospital,Sheffield,UK.
[Ti] Título:Sensory Ganglionopathy and the Blink Reflex: Electrophysiological Features.
[So] Source:Can J Neurol Sci;43(3):385-9, 2016 May.
[Is] ISSN:0317-1671
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Sensory ganglionopathy (SG) is characterised by asymmetrical sensory fibre degeneration, with the primary pathology occurring at the level of the dorsal root ganglion. It is seen in the context of autoimmune, paraneoplastic, and degenerative disorders. There is limited literature examining the electrophysiological correlate of the trigeminal ganglion and associated pathways, the blink reflex (BR), in cases of SG. Previous work has suggested that the BR is preserved in cases of SG associated with paraneoplasia. METHODS: The local clinical neurophysiology database was searched for patients diagnosed with SG from peripheral nerve conduction studies in whom the BR was performed. Twenty-six patients were included in the final analysis. RESULTS: Sjögren's syndrome constituted the most common SG aetiology (8/26), followed by idiopathic cases (7/26) and paraneoplasia (5/26). BR abnormalities were seen in 9 of the 26 patients (34.6%) across all aetiologies. No patients reported sensory disturbance in the distribution of the trigeminal nerve, indicating that the changes noted are subclinical. Three patients showed abnormality of the R1 response; in the remaining six patients, only R2 responses were affected. CONCLUSIONS: Subclinical abnormalities of both R1 and R2 can be seen in the context of SG of varying aetiologies, including paraneoplasia. Performing the BR in patients with suspected of having SG may be helpful in providing additional evidence of patchy sensory fibre involvement that is characteristic of the disease.
[Mh] Termos MeSH primário: Piscadela/fisiologia
Eletromiografia
Polineuropatia Paraneoplásica/fisiopatologia
Transtornos das Sensações/patologia
Transtornos das Sensações/fisiopatologia
Gânglio Trigeminal/patologia
[Mh] Termos MeSH secundário: Adulto
Idoso
Bases de Dados Factuais/estatística & dados numéricos
Feminino
Seres Humanos
Masculino
Meia-Idade
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1702
[Cu] Atualização por classe:170817
[Lr] Data última revisão:
170817
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160123
[St] Status:MEDLINE
[do] DOI:10.1017/cjn.2015.361


  3 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:26789908
[Au] Autor:Zalewski NL; Lennon VA; Lachance DH; Klein CJ; Pittock SJ; Mckeon A
[Ad] Endereço:Department of Neurology, College of Medicine, Mayo Clinic, Rochester, Minnesota, USA.
[Ti] Título:P/Q- and N-type calcium-channel antibodies: Oncological, neurological, and serological accompaniments.
[So] Source:Muscle Nerve;54(2):220-7, 2016 Aug.
[Is] ISSN:1097-4598
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:INTRODUCTION: Voltage-gated calcium-channel autoimmunity (VGCC-P/Q and VGCC-N types) occurs beyond Lambert-Eaton syndrome and lung cancer. METHODS: We reviewed records for 236 Mayo Clinic patients with VGCC antibodies found in evaluation for paraneoplastic neurological autoimmunity (generally without myasthenic syndromes). RESULTS: VGCC autoantibodies were detected in 3.4% of neurological patients, 1.7% of healthy controls, and 4% of neurologically asymptomatic lung cancer controls. Fifty neurological patients (21%) had ≥ 1 neoplasm, historically (46) or detected prospectively [small-cell lung carcinoma (2), breast adenocarcinoma (2), lymphoma (1), and suspected tonsillar carcinoma (1)]. Autoimmune neurological diagnosis frequencies (encephalopathy, ataxia, myelopathy, neuropathy, neuromuscular junction disorder, and myopathy) among patients with medium values (24%; 0.10-0.99 nmol/L) or low values (19%; 0.03-0.10 nmol/L) were fewer than among patients with antibody values exceeding 1.00 nmol/L (71%; P = 0.02 and 0.004, respectively). CONCLUSIONS: Among neuronal VGCC-autoantibody-seropositive patients, autoimmune neurological phenotypes and cancer types are diverse. Cautious interpretation of results (particularly medium and low values) is advised. Muscle Nerve, 2016 Muscle Nerve 54: 220-227, 2016.
[Mh] Termos MeSH primário: Autoanticorpos/sangue
Canais de Cálcio Tipo N/imunologia
Neoplasias Pulmonares/sangue
Doenças do Sistema Nervoso/sangue
Polineuropatia Paraneoplásica/sangue
[Mh] Termos MeSH secundário: Adolescente
Adulto
Idoso
Idoso de 80 Anos ou mais
Autoanticorpos/líquido cefalorraquidiano
Transtornos Cognitivos/sangue
Transtornos Cognitivos/imunologia
Feminino
Seres Humanos
Neoplasias Pulmonares/líquido cefalorraquidiano
Neoplasias Pulmonares/imunologia
Masculino
Meia-Idade
Doenças do Sistema Nervoso/líquido cefalorraquidiano
Doenças do Sistema Nervoso/imunologia
Polineuropatia Paraneoplásica/líquido cefalorraquidiano
Polineuropatia Paraneoplásica/imunologia
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Autoantibodies); 0 (Calcium Channels, N-Type)
[Em] Mês de entrada:1706
[Cu] Atualização por classe:170801
[Lr] Data última revisão:
170801
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160121
[St] Status:MEDLINE
[do] DOI:10.1002/mus.25027


  4 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:26633089
[Au] Autor:Hébant B; Miret N; Berthelot L; Jaafar M; Maltête D; Lefaucheur R
[Ad] Endereço:Department of Neurology, Rouen University Hospital and University of Rouen, 76031 Rouen Cedex, France.
[Ti] Título:Generalized pruritus preceding paraneoplastic neuropathy.
[So] Source:J Clin Neurosci;26:156-7, 2016 Apr.
[Is] ISSN:1532-2653
[Cp] País de publicação:Scotland
[La] Idioma:eng
[Ab] Resumo:Paraneoplastic syndromes are a group of rare disorders involving non-metastatic systemic effects accompanying malignancies, and occur remotely from the tumor itself. Chronic pruritus lasting more than 6 weeks can be from paraneoplastic origin. A 65-year-old woman was admitted for generalized pruritus lasting for 1 month, despite treatment with prednisolone, levocetirizine and hydroxyzine. General examination was normal. Biological data and gastroscopy were normal. One month later, the patient was readmitted for worsening of her pruritus and walking impairment, revealing a severe sensory neuropathy. Blood anti-Hu antibodies returned positive at a level of 400 (normal <100). Bronchoscopy and bronchial biopsies revealed small-cell lung carcinoma. To our knowledge, the association of generalized pruritus and paraneoplastic neuropathy has been rarely reported. Our observation raises the question of a pathophysiological continuum between pruritus and neuropathy in a paraneoplastic context.
[Mh] Termos MeSH primário: Neoplasias Pulmonares/complicações
Polineuropatia Paraneoplásica/etiologia
Prurido/etiologia
Carcinoma de Pequenas Células do Pulmão/complicações
[Mh] Termos MeSH secundário: Idoso
Feminino
Seres Humanos
Síndromes Paraneoplásicas/etiologia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1611
[Cu] Atualização por classe:161230
[Lr] Data última revisão:
161230
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151204
[St] Status:MEDLINE


  5 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:26439587
[Au] Autor:Üçeyler N; Necula G; Wagemann E; Toyka KV; Sommer C
[Ad] Endereço:Department of Neurology, University of Würzburg, Josef-Schneider-Str. 11, 97080, Würzburg, Germany.
[Ti] Título:Endoneurial edema in sural nerve may indicate recent onset inflammatory neuropathy.
[So] Source:Muscle Nerve;53(5):705-10, 2016 May.
[Is] ISSN:1097-4598
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:INTRODUCTION: Sural nerve biopsy is an important means of establishing the diagnosis of inflammatory neuropathies. We investigated the diagnostic value of endoneurial edema. METHODS: Diagnostic sural nerve biopsies from 42 patients with inflammatory and 28 patients with noninflammatory neuropathies were re-evaluated for the presence of endoneurial edema. Edema was assessed on hematoxylin-eosin stained paraffin and frozen sections and on azure II-methylene blue stained semithin sections. We determined the area of endoneurial edema on digitized images in relation to the entire endoneurial area of each fascicle. RESULTS: Edema was more extensive in neuropathies with short disease duration (≤12 months) as compared to long duration (>12 months; P < 0.01). Edema in inflammatory neuropathies of ≤12 months duration covered a larger area than in noninflammatory neuropathies (P < 0.01), and the extent of edema correlated negatively with disease duration (P < 0.05). CONCLUSIONS: Endoneurial edema may be a useful additional disease marker in inflammatory neuropathies of recent onset.
[Mh] Termos MeSH primário: Edema/patologia
Doenças do Sistema Nervoso Periférico/patologia
Nervo Sural/patologia
[Mh] Termos MeSH secundário: Adulto
Idoso
Esclerose Amiotrófica Lateral/complicações
Esclerose Amiotrófica Lateral/imunologia
Esclerose Amiotrófica Lateral/patologia
Biópsia
Estudos de Casos e Controles
Neuropatias Diabéticas/complicações
Neuropatias Diabéticas/imunologia
Neuropatias Diabéticas/patologia
Edema/etiologia
Edema/imunologia
Feminino
Seres Humanos
Masculino
Meia-Idade
Atrofia Muscular Espinal/complicações
Atrofia Muscular Espinal/imunologia
Atrofia Muscular Espinal/patologia
Polineuropatia Paraneoplásica/complicações
Polineuropatia Paraneoplásica/imunologia
Polineuropatia Paraneoplásica/patologia
Nervos Periféricos/imunologia
Nervos Periféricos/patologia
Doenças do Sistema Nervoso Periférico/complicações
Doenças do Sistema Nervoso Periférico/imunologia
Polirradiculoneuropatia Desmielinizante Inflamatória Crônica/complicações
Polirradiculoneuropatia Desmielinizante Inflamatória Crônica/imunologia
Polirradiculoneuropatia Desmielinizante Inflamatória Crônica/patologia
Nervo Sural/imunologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Mês de entrada:1609
[Cu] Atualização por classe:160418
[Lr] Data última revisão:
160418
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151007
[St] Status:MEDLINE
[do] DOI:10.1002/mus.24930


  6 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:25129419
[Au] Autor:Erro Aguirre ME; Gila L; Olaziregui O
[Ad] Endereço:Servicio de Neurología, Complejo Hospitalario de Navarra, Pamplona, Navarra, España. Electronic address: elena.erro.aguirre@cfnavarra.es.
[Ti] Título:[Paraneoplastic sensory neuropathy associated with squamous cell carcinoma of the larynx].
[Ti] Título:Neuronopatía sensitiva paraneoplásica asociada a carcinoma escamoso de laringe..
[So] Source:Neurologia;31(4):286-8, 2016 May.
[Is] ISSN:1578-1968
[Cp] País de publicação:Spain
[La] Idioma:spa
[Mh] Termos MeSH primário: Carcinoma de Células Escamosas/complicações
Neoplasias Laríngeas/complicações
Polineuropatia Paraneoplásica/etiologia
[Mh] Termos MeSH secundário: Carcinoma de Células Escamosas/diagnóstico por imagem
Carcinoma de Células Escamosas/terapia
Terapia Combinada
Seres Humanos
Neoplasias Laríngeas/genética
Neoplasias Laríngeas/terapia
Masculino
Meia-Idade
Polineuropatia Paraneoplásica/diagnóstico por imagem
Polineuropatia Paraneoplásica/terapia
Tomografia por Emissão de Pósitrons
[Pt] Tipo de publicação:CASE REPORTS; LETTER
[Em] Mês de entrada:1702
[Cu] Atualização por classe:170223
[Lr] Data última revisão:
170223
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:140818
[St] Status:MEDLINE


  7 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:26683964
[Au] Autor:Ni J; Weng L; Liu M; Yang H; Wang Y
[Ad] Endereço:From the Department of Medical Oncology (JN, LW), Department of Neurology (ML), Department of Otolaryngology (HY); and Department of Medical Oncology, Peking Union Medical College Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China (YW).
[Ti] Título:Small Cell Lung Cancer Accompanied by Tonsillar Metastasis and Anti-Hu Antibody-Associated Paraneoplastic Neuropathy: A Rare Case Report With Long-Term Survival.
[So] Source:Medicine (Baltimore);94(50):e2291, 2015 Dec.
[Is] ISSN:1536-5964
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Tonsillar metastatic small cell lung cancer (SCLC) is rare, while anti-Hu antibodies are frequently found in SCLC. A 66-year-old man was admitted to our hospital with painful dysesthesia and muscle weakness in the distal extremities for over 1 year, progressive dysphagia for over 1 month, and severe cough and dyspnea for over 1 week. He was diagnosed with SCLC accompanied by tonsillar metastasis and anti-Hu antibody-associated paraneoplastic sensory neuropathy (PSN). The patient tolerated 6 cycles of sequential chemoradiotherapy and gradually recovered. The patient's disease remained in remission 2 years after the diagnosis with a remarkable reduction of tumor burden and a persisting high titer of anti-Hu antibodies. To our knowledge, this is the first case of tonsillar metastatic SCLC accompanied by anti-Hu antibody-associated PSN, whereby the anticancer immune response was presumed to play a vital role in disease control. Unilateral tonsillar metastasis of SCLC accompanied by anti-Hu antibody-associated PSN can occur and in certain circumstances, may have a favorable prognosis.
[Mh] Termos MeSH primário: Anticorpos/metabolismo
Proteínas ELAV/imunologia
Neoplasias Pulmonares/patologia
Polineuropatia Paraneoplásica/complicações
Carcinoma de Pequenas Células do Pulmão/secundário
Neoplasias Tonsilares/secundário
[Mh] Termos MeSH secundário: Idoso
Seres Humanos
Neoplasias Pulmonares/imunologia
Neoplasias Pulmonares/metabolismo
Masculino
Polineuropatia Paraneoplásica/imunologia
Polineuropatia Paraneoplásica/metabolismo
Carcinoma de Pequenas Células do Pulmão/imunologia
Carcinoma de Pequenas Células do Pulmão/metabolismo
Neoplasias Tonsilares/imunologia
Neoplasias Tonsilares/metabolismo
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Antibodies); 0 (ELAV Proteins)
[Em] Mês de entrada:1604
[Cu] Atualização por classe:170224
[Lr] Data última revisão:
170224
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:151220
[St] Status:MEDLINE
[do] DOI:10.1097/MD.0000000000002291


  8 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
PubMed Central Texto completo
Texto completo
[PMID]:26635447
[Au] Autor:Panis C; Pavanelli WR
[Ad] Endereço:Laboratory of Inflammatory Mediators, State University of West Paraná, UNIOESTE, Campus Francisco Beltrão, Rua Maringá 1200, 85605-010 Francisco Beltrão, PR, Brazil.
[Ti] Título:Cytokines as Mediators of Pain-Related Process in Breast Cancer.
[So] Source:Mediators Inflamm;2015:129034, 2015.
[Is] ISSN:1466-1861
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Pain is a clinical sign of inflammation found in a wide variety of chronic pathologies, including cancer. The occurrence of pain in patients carrying breast tumors is reported and is associated with aspects concerning disease spreading, treatment, and surgical intervention. The persistence of pain in patients submitted to breast surgery is estimated in a range from 21% to 55% and may affect patients before and after surgery. Beyond the physical compression exerted by the metastatic mass expansion and tissue injury found in breast cancer, inflammatory components that are significantly produced by the host-tumor interaction can significantly contribute to the generation of pain. In this context, cytokines have been studied aiming to establish a cause-effect relationship in cancer pain-related syndromes, especially the proinflammatory ones. Few reports have investigated the relationship between pain and cytokines in women carrying advanced breast cancer. In this scenario, the present review analyzes the main cytokines produced in breast cancer and discusses the evidences from literature regarding its role in specific clinical features related with this pathology.
[Mh] Termos MeSH primário: Neoplasias da Mama/fisiopatologia
Citocinas/fisiologia
Dor Intratável/etiologia
[Mh] Termos MeSH secundário: Neoplasias Ósseas/secundário
Neoplasias da Mama/tratamento farmacológico
Feminino
Fator Estimulador de Colônias de Granulócitos/uso terapêutico
Seres Humanos
Dor Intratável/imunologia
Polineuropatia Paraneoplásica/etiologia
Fator de Crescimento Transformador beta/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Cytokines); 0 (Transforming Growth Factor beta); 143011-72-7 (Granulocyte Colony-Stimulating Factor)
[Em] Mês de entrada:1609
[Cu] Atualização por classe:170220
[Lr] Data última revisão:
170220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151205
[St] Status:MEDLINE
[do] DOI:10.1155/2015/129034


  9 / 224 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:26511029
[Au] Autor:Sakurai T; Wakida K; Kimura A; Inuzuka T; Nishida H
[Ad] Endereço:Department of Neurology, Gifu Prefectural General Medical Center.
[Ti] Título:[Anti-Hu antibody-positive paraneoplastic limbic encephalitis with acute motor sensory neuropathy resembling Guillain-Barré syndrome: a case study].
[So] Source:Rinsho Shinkeigaku;55(12):921-5, 2015.
[Is] ISSN:1882-0654
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Ab] Resumo:A 69-year-old man experienced general malaise, weight loss, amnesia, gait disturbance, and restlessness a month prior to admission. Brain MRI showed high intensity areas in the bilateral medial temporal lobes and insular cortices on FLAIR images, and therefore, he was diagnosed with limbic encephalitis. After admission, quadriplegia and respiratory failure progressed rapidly, and he needed ventilatory management. A nerve conduction study revealed low compound muscle action potential amplitude with loss of sensory nerve action potential, which indicated axonal sensorimotor neuropathy. We administered intravenous immunoglobulin and methylprednisolone pulse therapy, but he did not recover. Although no tumor was found on CT, his serum was positive for anti-Hu antibody; therefore, we diagnosed him with paraneoplastic neurological syndrome. An FDG-PET study showed accumulation at lesions on two hilar lymph nodes. Small cell lung carcinoma was detected by endobronchial ultrasound-guided transbronchial needle aspiration. Although paraneoplastic acute sensorimotor neuropathy with respiratory failure resembling Guillain-Barré syndrome is rare, identification of antibodies and servey of tumors aids accurate diagnosis.
[Mh] Termos MeSH primário: Autoanticorpos/sangue
Proteínas ELAV/imunologia
Encefalite Límbica/complicações
Encefalite Límbica/diagnóstico
Neurônios Motores
Polineuropatia Paraneoplásica/diagnóstico
Polineuropatia Paraneoplásica/etiologia
Células Receptoras Sensoriais
[Mh] Termos MeSH secundário: Doença Aguda
Idoso
Biomarcadores/sangue
Diagnóstico Diferencial
Diagnóstico por Imagem
Síndrome de Guillain-Barré
Seres Humanos
Neoplasias Pulmonares/complicações
Neoplasias Pulmonares/diagnóstico
Masculino
Insuficiência Respiratória/etiologia
Carcinoma de Pequenas Células do Pulmão/complicações
Carcinoma de Pequenas Células do Pulmão/diagnóstico
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Autoantibodies); 0 (Biomarkers); 0 (ELAV Proteins)
[Em] Mês de entrada:1612
[Cu] Atualização por classe:161230
[Lr] Data última revisão:
161230
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151030
[St] Status:MEDLINE
[do] DOI:10.5692/clinicalneurol.cn-000772


  10 / 224 MEDLINE  
              first record previous record
seleciona
para imprimir
Fotocópia
[PMID]:26480794
[Au] Autor:Ohyama K; Koike H; Sobue G
[Ti] Título:[Paraneoplastic neuropathy].
[So] Source:Nihon Rinsho;73 Suppl 7:782-7, 2015 Sep.
[Is] ISSN:0047-1852
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Mh] Termos MeSH primário: Polineuropatia Paraneoplásica
[Mh] Termos MeSH secundário: Autoanticorpos/imunologia
Síndrome de Guillain-Barré/imunologia
Seres Humanos
Imagem Multimodal
Polineuropatia Paraneoplásica/diagnóstico
Polineuropatia Paraneoplásica/imunologia
Polineuropatia Paraneoplásica/terapia
Polirradiculoneuropatia Desmielinizante Inflamatória Crônica/imunologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Autoantibodies)
[Em] Mês de entrada:1601
[Cu] Atualização por classe:151020
[Lr] Data última revisão:
151020
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151021
[St] Status:MEDLINE



página 1 de 23 ir para página                         
   


Refinar a pesquisa
  Base de dados : MEDLINE Formulário avançado   

    Pesquisar no campo  
1  
2
3
 
           



Search engine: iAH v2.6 powered by WWWISIS

BIREME/OPAS/OMS - Centro Latino-Americano e do Caribe de Informação em Ciências da Saúde