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Pesquisa : C05.116.132 [Categoria DeCS]
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  1 / 28 MEDLINE  
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[PMID]:25105995
[Au] Autor:Siwamogsatham O; Alvarez JA; Tangpricha V
[Ad] Endereço:aDivision of Endocrinology, Metabolism and Lipids, Department of Medicine, Emory University School of Medicine bSamitivej Srinakarin Hospital, Bangkok Hospital Group, Bangkok, Thailand cEmory Adult Cystic Fibrosis Program, Emory Healthcare dSection of Endocrinology, Atlanta VA Medical Center, Decatur, Georgia, USA.
[Ti] Título:Diagnosis and treatment of endocrine comorbidities in patients with cystic fibrosis.
[So] Source:Curr Opin Endocrinol Diabetes Obes;21(5):422-9, 2014 Oct.
[Is] ISSN:1752-2978
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:PURPOSE OF REVIEW: The aim of this review is to provide an update on various relevant endocrine aspects of care in adolescents and adults with cystic fibrosis. RECENT FINDINGS: As life expectancy in cystic fibrosis has continuously improved, endocrine complications have become more apparent. The common endocrine complications include cystic fibrosis related diabetes, cystic fibrosis related bone disease, vitamin D deficiency and poor growth and pubertal development. Thyroid and adrenal disorders have also been reported, although the prevalence appears to be less common. SUMMARY: Endocrine diseases are an increasingly recognized complication that has a significant impact on the overall health of individuals with cystic fibrosis. This review summarizes the updated screening and management of endocrine diseases in the cystic fibrosis population.
[Mh] Termos MeSH primário: Doenças Ósseas Endócrinas/diagnóstico
Fibrose Cística/complicações
Diabetes Mellitus/diagnóstico
Transtornos do Crescimento/diagnóstico
Doenças da Glândula Tireoide/diagnóstico
Deficiência de Vitamina D/diagnóstico
[Mh] Termos MeSH secundário: Absorciometria de Fóton
Estatura
Doenças Ósseas Endócrinas/epidemiologia
Doenças Ósseas Endócrinas/terapia
Comorbidade
Fibrose Cística/epidemiologia
Fibrose Cística/terapia
Diabetes Mellitus/epidemiologia
Diabetes Mellitus/terapia
Feminino
Hemoglobina A Glicada/metabolismo
Transtornos do Crescimento/epidemiologia
Transtornos do Crescimento/terapia
Seres Humanos
Masculino
Programas de Rastreamento/métodos
Estado Nutricional
Prevalência
Doenças da Glândula Tireoide/epidemiologia
Doenças da Glândula Tireoide/terapia
Deficiência de Vitamina D/epidemiologia
Deficiência de Vitamina D/terapia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Glycated Hemoglobin A); 0 (hemoglobin A1c protein, human)
[Em] Mês de entrada:1505
[Cu] Atualização por classe:171116
[Lr] Data última revisão:
171116
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:140809
[St] Status:MEDLINE
[do] DOI:10.1097/MED.0000000000000096


  2 / 28 MEDLINE  
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[PMID]:24824804
[Au] Autor:Elabd SK; Sabry I; Mohasseb M; Algendy A
[Ti] Título:Oxytocin as a novel therapeutic option for type I diabetes and diabetic osteopathy.
[So] Source:Endocr Regul;48(2):87-102, 2014 Apr.
[Is] ISSN:1210-0668
[Cp] País de publicação:Germany
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: The aim of the present study was to highlight the newly discovered metabolic role of oxytocin (OT) in the type I diabetic rats. Previous studies have demonstrated that OT has a beneficial role on bone physiology and therefore, the OT effect on the diabetic osteopathy will be assessed as well. METHODS: Induction of the type I diabetes was carried out by an intraperitoneal injection of 60 mg/kg body weight of streptozotocin. The metabolic role of OT on diabetic rats after OT treatment with intramuscular injection of 40 µIU/kg body weight for 6 weeks was assessed. Histological and ultrastructural studies of rat pancreas samples, before and after the OT injection, were performed and compared with the obtained physiological results. RESULTS: Oxytocin treatment had positive metabolic effects in diabetic rats. This is based on the change in glucose metabolism, lipid profile, and insulin sensitivity in experimental animals. In addition, OT treatment showed histological regenerative changes of pancreatic islet cells of diabetic rats. Moreover, OT administration showed that it has an anabolic effect on the bone biology. CONCLUSIONS: The results suggest that activation of the oxytocin receptor (OTR) pathway by infusion of OT, OT analogs, or OT agonists may represent a promising approach for the treatment of diabetes and some of its complications, including diabetic osteopathy.
[Mh] Termos MeSH primário: Doenças Ósseas Endócrinas/tratamento farmacológico
Diabetes Mellitus Tipo 1/tratamento farmacológico
Ocitocina/uso terapêutico
[Mh] Termos MeSH secundário: Animais
Glicemia/efeitos dos fármacos
Glicemia/metabolismo
Doenças Ósseas Endócrinas/etiologia
Osso e Ossos/efeitos dos fármacos
Osso e Ossos/metabolismo
Diabetes Mellitus Experimental/complicações
Diabetes Mellitus Experimental/tratamento farmacológico
Diabetes Mellitus Experimental/metabolismo
Diabetes Mellitus Tipo 1/complicações
Diabetes Mellitus Tipo 1/metabolismo
Insulina/metabolismo
Resistência à Insulina
Metabolismo dos Lipídeos/efeitos dos fármacos
Masculino
Ratos
Ratos Wistar
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Blood Glucose); 0 (Insulin); 50-56-6 (Oxytocin)
[Em] Mês de entrada:1408
[Cu] Atualização por classe:140514
[Lr] Data última revisão:
140514
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:140515
[St] Status:MEDLINE


  3 / 28 MEDLINE  
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[PMID]:23999360
[Au] Autor:Fukumoto S
[Ad] Endereço:Division of Nephrology & Endocrinology, Department of Medicine, University of Tokyo Hospital, Japan.
[Ti] Título:[Secondary osteoporosis or secondary contributors to bone loss in fracture. Endocrinological aspects of bone metabolism].
[So] Source:Clin Calcium;23(9):1259-64, 2013 Sep.
[Is] ISSN:0917-5857
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Ab] Resumo:Bone works to play essential roles in mineral metabolism and hematopoiesis as well as to support our body and protect internal organs as a hard tissue. In order to accomplish these multiple functions, bone needs to communicate with other organs. Endocrine system functions as one of the communication pathways between bone and other organs. It has been known that bone is a target organ of many hormones. In addition, it has been established that bone itself produces hormones and works as an endocrine organ.
[Mh] Termos MeSH primário: Densidade Óssea/fisiologia
Doenças Ósseas Endócrinas/metabolismo
Osso e Ossos/metabolismo
Cálcio/metabolismo
Fraturas Ósseas/etiologia
Osteoporose/etiologia
[Mh] Termos MeSH secundário: Fraturas Ósseas/metabolismo
Seres Humanos
Osteoporose/metabolismo
[Pt] Tipo de publicação:ENGLISH ABSTRACT; JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
SY7Q814VUP (Calcium)
[Em] Mês de entrada:1312
[Cu] Atualização por classe:130903
[Lr] Data última revisão:
130903
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:130904
[St] Status:MEDLINE
[do] DOI:CliCa130912591264


  4 / 28 MEDLINE  
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[PMID]:22361910
[Au] Autor:Prasad HK; Ekbote V; Rustagi V; Borade A; Chiplonkar S; Khadilkar V; Khadilkar AV
[Ad] Endereço:Department of Pediatric Endocrinology, Bharati Vidyapeeth University Medical College, Pune, Maharashtra, India.
[Ti] Título:Performance of WHO growth standards on Indian children with growth related disorders.
[So] Source:Indian J Pediatr;79(7):884-90, 2012 Jul.
[Is] ISSN:0973-7693
[Cp] País de publicação:India
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: To assess performance of WHO 2006 standards on anthropometric measurements of children referred for growth related disorders to a speciality pediatric clinic in Pune, India, from June 2006 through June 2010. METHODS: Data presented in this study were collected retrospectively from case records of all children from birth to 60 mo (n=1840, mean age 2.7±1.3 y) who presented with growth related disorders; healthy age and sex matched children were recruited as controls (n=824, mean age 2.8±1.2 y). Children were divided as per their clinical diagnosis into eight different groups: growth hormone deficiency, bone disorders, syndromic short stature, familial short stature, hypothyroidism, nutritional and systemic disorder, other endocrinopathies and overgrowth disorders. Anthropometric parameters for all study subjects were converted to standard deviation scores (SD scores) using the WHO Anthro 2005. RESULTS: Mean height SD scores of children with growth related disorders were significantly lower than that of the controls, while that of the tall children were significantly higher (p<0.05). All children who were clinically very short were below the 1st percentile, while none of the children with overgrowth or normal children were classified as stunted. Weight for height SD scores of children with nutritional and systemic disorders were the lowest, while those for the obese children were the highest. CONCLUSIONS: The present results suggest that the WHO 2006 growth standards classify children with growth disorders appropriately and the classification is in concordance with the clinical assessment. They provide health practitioners in a clinical setting with an effective tool to assess growth of children.
[Mh] Termos MeSH primário: Gráficos de Crescimento
Transtornos do Crescimento/diagnóstico
Organização Mundial da Saúde
[Mh] Termos MeSH secundário: Estatura
Peso Corporal
Doenças Ósseas Endócrinas/diagnóstico
Estudos de Casos e Controles
Pré-Escolar
Estudos Transversais
Feminino
Seres Humanos
Índia
Masculino
Transtornos Nutricionais/diagnóstico
Estudos Retrospectivos
[Pt] Tipo de publicação:EVALUATION STUDIES; JOURNAL ARTICLE
[Em] Mês de entrada:1211
[Cu] Atualização por classe:120629
[Lr] Data última revisão:
120629
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:120225
[St] Status:MEDLINE
[do] DOI:10.1007/s12098-012-0687-y


  5 / 28 MEDLINE  
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[PMID]:22042036
[Au] Autor:Duval-Sabatier A; Gondouin B; Bouvier C; Bataille S; Berland Y; Brunet P
[Ad] Endereço:Centre de Néphrologie et Transplantation Rénale, Hôpital de la Conception, Assistance Publique Hôpitaux de Marseille, Marseille, France. ariane.duval@ap-hm.fr
[Ti] Título:Brown tumor: still an old disease?
[So] Source:Kidney Int;80(10):1110, 2011 Nov.
[Is] ISSN:1523-1755
[Cp] País de publicação:United States
[La] Idioma:eng
[Mh] Termos MeSH primário: Doenças Ósseas Endócrinas/etiologia
Hiperparatireoidismo Secundário/etiologia
Nefropatias/terapia
Diálise Renal
Vértebras Torácicas
[Mh] Termos MeSH secundário: Adulto
Dor nas Costas/etiologia
Biópsia
Doenças Ósseas Endócrinas/diagnóstico
Doenças Ósseas Endócrinas/cirurgia
Dor no Peito/etiologia
Descompressão Cirúrgica
Seres Humanos
Hiperparatireoidismo Secundário/diagnóstico
Hiperparatireoidismo Secundário/cirurgia
Nefropatias/complicações
Imagem por Ressonância Magnética
Masculino
Paratireoidectomia
Compressão da Medula Espinal/etiologia
Compressão da Medula Espinal/cirurgia
Vértebras Torácicas/patologia
Vértebras Torácicas/cirurgia
Resultado do Tratamento
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1202
[Cu] Atualização por classe:111101
[Lr] Data última revisão:
111101
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:111102
[St] Status:MEDLINE
[do] DOI:10.1038/ki.2011.290


  6 / 28 MEDLINE  
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[PMID]:21865758
[Au] Autor:Bianco P; Sacchetti B; Riminucci M
[Ad] Endereço:Department of Molecular Medicine, Sapienza University of Rome, Viale Regina Elena 324, Rome, Italy. paolo.bianco@uniroma1.it
[Ti] Título:Stem cells in skeletal physiology and endocrine diseases of bone.
[So] Source:Endocr Dev;21:91-101, 2011.
[Is] ISSN:1662-2979
[Cp] País de publicação:Switzerland
[La] Idioma:eng
[Ab] Resumo:Self-renewing, multipotent progenitors of skeletal tissues are found within skeletal segments (skeletal stem cells) and coincide with adventitial reticular cells of bone marrow vessels in situ and with explanted clonogenic stromal cells ex vivo. These cells, which can be identified and prospectively isolated based on a minimal surface phenotype noted for expression of CD146, CD105 and alkaline phosphatase, are established during bone development through interactions with developing sinusoids. They represent a crucial crossroad of skeletal and hematopoietic physiology, as well as of endocrine regulation of bone growth and remodeling. In addition, they are central to major endocrine functions of bone itself, such as regulation of renal phosphate handling. Skeletal stem cells represent a central model system for investigating skeletal diseases, as tools for in vitro and in vivo models, for cell therapy-based strategies, or as targets for drugs.
[Mh] Termos MeSH primário: Doenças Ósseas Endócrinas/etiologia
Osso e Ossos/fisiologia
Células-Tronco/fisiologia
[Mh] Termos MeSH secundário: Animais
Doenças Ósseas Endócrinas/metabolismo
Doenças Ósseas Endócrinas/patologia
Doenças Ósseas Endócrinas/terapia
Osso e Ossos/citologia
Osso e Ossos/patologia
Tecido Conjuntivo/fisiologia
Seres Humanos
Células Mesenquimais Estromais/citologia
Células Mesenquimais Estromais/fisiologia
Modelos Biológicos
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T; REVIEW
[Em] Mês de entrada:1112
[Cu] Atualização por classe:121115
[Lr] Data última revisão:
121115
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:110826
[St] Status:MEDLINE
[do] DOI:10.1159/000328138


  7 / 28 MEDLINE  
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[PMID]:20878436
[Au] Autor:Sakayama K; Sugawara Y; Kidani T; Fujibuchi T; Kito K; Tanji N; Nakamura A
[Ad] Endereço:Department of Bone and Joint Surgery, Ehime University Graduate School of Medicine, Shitsukawa, Toon, Ehime 791-0204, Japan. kenshi@m.ehime-u.ac.jp
[Ti] Título:Polyostotic fibrous dysplasia with gigantism and huge pelvic tumor: a rare case of McCune-Albright syndrome.
[So] Source:Int J Clin Oncol;16(3):270-4, 2011 Jun.
[Is] ISSN:1437-7772
[Cp] País de publicação:Japan
[La] Idioma:eng
[Ab] Resumo:We report a rare case of polyostotic fibrous dysplasia on endocrine hyperfunction with elevated human growth hormone and normal serum level of prolactin. There were some differential points of gender, gigantism, endocrine function, and GNAS gene from McCune-Albright syndrome. Malignant transformation was suspected in the pelvic tumor from imaging because rapid growth of the tumor by imaging was observed; however, no malignant change occurred in this case.
[Mh] Termos MeSH primário: Displasia Fibrosa Poliostótica/complicações
Displasia Fibrosa Poliostótica/diagnóstico
Hormônio do Crescimento Humano/sangue
Neoplasias Pélvicas/complicações
Neoplasias Pélvicas/cirurgia
[Mh] Termos MeSH secundário: Acromegalia/diagnóstico
Adulto
Doenças Ósseas Endócrinas/complicações
Doenças Ósseas Endócrinas/diagnóstico
Neoplasias Ósseas/diagnóstico
Cromograninas
Diagnóstico Diferencial
Displasia Fibrosa Poliostótica/sangue
Subunidades alfa Gs de Proteínas de Ligação ao GTP/metabolismo
Gigantismo/diagnóstico
Seres Humanos
Masculino
Doenças da Hipófise/complicações
Prolactina/sangue
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Chromogranins); 12629-01-5 (Human Growth Hormone); 9002-62-4 (Prolactin); EC 3.6.1.- (GNAS protein, human); EC 3.6.5.1 (GTP-Binding Protein alpha Subunits, Gs)
[Em] Mês de entrada:1110
[Cu] Atualização por classe:171013
[Lr] Data última revisão:
171013
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:100930
[St] Status:MEDLINE
[do] DOI:10.1007/s10147-010-0127-9


  8 / 28 MEDLINE  
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[PMID]:19816169
[Au] Autor:Sparks AA; McGee SJ; Boone CE; Neuringer IP; Jones SK; Aris RM
[Ad] Endereço:Division of Pulmonary and Critical Care Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina 27599, USA.
[Ti] Título:'Old' bones in young bodies: the tale of cystic fibrosis.
[So] Source:Curr Opin Endocrinol Diabetes Obes;16(6):407-14, 2009 Dec.
[Is] ISSN:1752-2978
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:PURPOSE OF REVIEW: Cystic fibrosis (CF) is the most common genetic disease within the white population and leads to premature respiratory failure. Approximately, 60 000 individuals are currently living with CF in North America and Europe, almost half of whom are adults. RECENT FINDINGS: Dozens of studies across the globe indicate that CF adults have low bone density and increased rates of fractures. This genesis of the problem appears to be in late childhood to adolescence. SUMMARY: Prevention and treatment of CF-related bone disease must address the myriad risk factors (decreased absorption of fat-soluble vitamins due to pancreatic insufficiency, altered sex hormone production, chronic lung infection with increased serum levels of proinflammatory, bone-active cytokines, malnutrition and low body weight, physical inactivity and glucocorticoid therapy) for poor bone health. This review will address the pathogenesis, diagnosis and treatment of bone disease in CF. It will also discuss best practice guidelines for optimizing bone health in patients with CF.
[Mh] Termos MeSH primário: Densidade Óssea
Doenças Ósseas Endócrinas/etiologia
Fibrose Cística/complicações
[Mh] Termos MeSH secundário: Adulto
Doenças Ósseas Endócrinas/diagnóstico
Doenças Ósseas Endócrinas/terapia
Fibrose Cística/fisiopatologia
Fraturas Ósseas/etiologia
Seres Humanos
Guias de Prática Clínica como Assunto
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T; REVIEW
[Em] Mês de entrada:1001
[Cu] Atualização por classe:091027
[Lr] Data última revisão:
091027
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:091010
[St] Status:MEDLINE
[do] DOI:10.1097/MED.0b013e3283329b16


  9 / 28 MEDLINE  
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[PMID]:19721199
[Au] Autor:Takeshita M; Mizuno Y
[Ad] Endereço:Division of Endocrinology and Metabolism, Kanto Central Hospital.
[Ti] Título:[Effect of glucose and bone metabolism in endocrinal disorder].
[So] Source:Clin Calcium;19(9):1282-90, 2009 Sep.
[Is] ISSN:0917-5857
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Ab] Resumo:It is well known that endocrine disorders; glucocorticoid excess including Cushing syndrome, high dose or long term steroid therapy and hyperthyroidism, induce secondary osteoporosis. These common endocrinal disorders affect not only bone metabolism but also glucose metabolism. Glucose metabolisms also play a important role in the progression of osteoporosis. To prevent secondary osteoporosis, multifocal intervention such as correction of insulin resistance or glucose metabolism, lifestyle, and drug therapy, need to be added to current management of endocrinal system.
[Mh] Termos MeSH primário: Doenças Ósseas Endócrinas/etiologia
Osso e Ossos/metabolismo
Glucose/metabolismo
Osteoporose/etiologia
[Mh] Termos MeSH secundário: Animais
Doenças Ósseas Endócrinas/prevenção & controle
Seres Humanos
Resistência à Insulina
Estilo de Vida
Osteoporose/prevenção & controle
[Pt] Tipo de publicação:ENGLISH ABSTRACT; JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
IY9XDZ35W2 (Glucose)
[Em] Mês de entrada:0911
[Cu] Atualização por classe:131121
[Lr] Data última revisão:
131121
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:090902
[St] Status:MEDLINE
[do] DOI:CliCa090912821290


  10 / 28 MEDLINE  
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[PMID]:19670266
[Au] Autor:Stalvey MS; Flotte TR
[Ad] Endereço:Department of Pediatrics and the Gene Therapy Center, University of Massachusetts Medical School, Worcester, Massachusetts 01655, USA. michael.stalvey@umassmed.edu
[Ti] Título:Endocrine parameters of cystic fibrosis: back to basics.
[So] Source:J Cell Biochem;108(2):353-61, 2009 Oct 01.
[Is] ISSN:1097-4644
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Dramatic changes in the life expectancy of cystic fibrosis (CF) patients are occurring, creating a cohort of aging individuals experiencing long-term complications of this chronic disease. The two most common of these complications include CF-related diabetes and CF bone disease. The clinical implications of each have become better understood, as have potential therapies. However, data obtained from the basic science studies of both diseases have not been widely recognized. In this review, we focus on the known and hypothesized pathogenesis of these two disorders. Additionally, the molecular underpinnings of CF will be explained along with the potential interactions with endocrine disease phenotypes.
[Mh] Termos MeSH primário: Doenças Ósseas Endócrinas/etiologia
Fibrose Cística/complicações
Diabetes Mellitus/etiologia
[Mh] Termos MeSH secundário: Envelhecimento
Animais
Doenças Ósseas Endócrinas/patologia
Doenças Ósseas Endócrinas/fisiopatologia
Fibrose Cística/patologia
Fibrose Cística/fisiopatologia
Diabetes Mellitus/patologia
Diabetes Mellitus/fisiopatologia
Progressão da Doença
Feminino
Seres Humanos
Masculino
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:0912
[Cu] Atualização por classe:090923
[Lr] Data última revisão:
090923
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:090812
[St] Status:MEDLINE
[do] DOI:10.1002/jcb.22284



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