Base de dados : MEDLINE
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[PMID]:28461004
[Au] Autor:Stevanovic K; Yunus A; Joly-Amado A; Gordon M; Morgan D; Gulick D; Gamsby J
[Ad] Endereço:Department of Molecular Medicine, University of South Florida, Tampa, FL, USA; Byrd Alzheimer's Institute, University of South Florida, Tampa, FL, USA. Electronic address: kstevanovic@health.usf.edu.
[Ti] Título:Disruption of normal circadian clock function in a mouse model of tauopathy.
[So] Source:Exp Neurol;294:58-67, 2017 08.
[Is] ISSN:1090-2430
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Disruption of normal circadian rhythm physiology is associated with neurodegenerative disease, which can lead to symptoms such as altered sleep cycles. In Alzheimer's disease (AD), circadian dysfunction has been attributed to ß-amyloidosis. However, it is unclear whether tauopathy, another AD-associated neuropathology, can disrupt the circadian clock. We have evaluated the status of the circadian clock in a mouse model of tauopathy (Tg4510). Tg4510 mice display a long free-running period at an age when tauopathy is present, and show evidence of tauopathy in the suprachiasmatic nucleus (SCN) of the hypothalamus - the site of the master circadian clock. Additionally, cyclic expression of the core clock protein PER2 is disrupted in the hypothalamus of Tg4510 mice. Finally, disruption of the cyclic expression of PER2 and BMAL1, another core circadian clock protein, is evident in the Tg4510 hippocampus. These results demonstrate that tauopathy disrupts normal circadian clock function both at the behavioral and molecular levels, which may be attributed to the tauopathy-induced neuropathology in the SCN. Furthermore, these results establish the Tg4510 mouse line as a model to study how tauopathy disrupts normal circadian rhythm biology.
[Mh] Termos MeSH primário: Transtornos Cronobiológicos/etiologia
Tauopatias/complicações
[Mh] Termos MeSH secundário: Fatores de Transcrição ARNTL/genética
Fatores de Transcrição ARNTL/metabolismo
Análise de Variância
Animais
Transtornos Cronobiológicos/genética
Modelos Animais de Doenças
Regulação da Expressão Gênica/genética
Locomoção/genética
Camundongos
Camundongos Transgênicos
Mutação/genética
Proteínas Circadianas Period/genética
Proteínas Circadianas Period/metabolismo
Fosforilação/genética
Núcleo Supraquiasmático/metabolismo
Núcleo Supraquiasmático/patologia
Tauopatias/genética
Tauopatias/patologia
Proteínas tau/genética
Proteínas tau/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (ARNTL Transcription Factors); 0 (Arntl protein, mouse); 0 (Per2 protein, mouse); 0 (Period Circadian Proteins); 0 (tau Proteins)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:180218
[Lr] Data última revisão:
180218
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170503
[St] Status:MEDLINE


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[PMID]:28463712
[Au] Autor:Au J; Reece J
[Ad] Endereço:School of Psychological Sciences, Australian College of Applied Psychology, Sydney, Australia. Electronic address: jackyau11@gmail.com.
[Ti] Título:The relationship between chronotype and depressive symptoms: A meta-analysis.
[So] Source:J Affect Disord;218:93-104, 2017 Aug 15.
[Is] ISSN:1573-2517
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Expanding our understanding of the factors that influence depression is crucial for prognosis and treatment. In light of increasing evidence of an association between disrupted circadian rhythms and affective symptoms, a meta-analysis was used to examine the relationship between an eveningness chronotype and depression. METHODS: Electronic searches of the PsycINFO, Medline, Scopus, and Google Scholar databases were conducted in February 2016. Relevant reviews, related journals, and reference lists were manually searched. Statistical data were reported or transformed to a Fisher's z correlational coefficient for effect size analysis. RESULTS: Data from 36 studies (n =15734) met the inclusion criteria and were analysed under a random effects model. Nearly all included studies utilised the Composite Scale of Morningness (CSM) or the Morningness-Eveningness Questionnaire (MEQ) as a measure of chronotype. Overall effect size from 58 effect sizes was small (z=-.20; 95% CI: -.18 to -.23). Effect sizes based on the CSM were significantly larger than those based on the MEQ. There was no evidence of publication bias. LIMITATIONS: The number of studies comparing different mood disorders or the potential moderating effects of gender and age were too few to draw conclusions regarding their respective effect sizes. Future research should utilise longitudinal designs to draw causal inferences on the directionality of this relationship. CONCLUSIONS: Findings from this meta-analysis indicate an eveningness orientation is somewhat associated with more severe mood symptoms. Chronobiological approaches may contribute to the prevention and treatment of depressive disorders.
[Mh] Termos MeSH primário: Transtornos Cronobiológicos/psicologia
Ritmo Circadiano/fisiologia
Depressão/fisiopatologia
Depressão/psicologia
Transtornos do Humor/fisiopatologia
[Mh] Termos MeSH secundário: Adulto
Afeto/fisiologia
Transtornos Cronobiológicos/fisiopatologia
Feminino
Seres Humanos
Masculino
Meia-Idade
Transtornos do Humor/psicologia
Fatores de Risco
Inquéritos e Questionários
[Pt] Tipo de publicação:JOURNAL ARTICLE; META-ANALYSIS; REVIEW
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180214
[Lr] Data última revisão:
180214
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170503
[St] Status:MEDLINE


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[PMID]:29244929
[Au] Autor:Osikov MV; Ogneva OI
[Ti] Título:Relationship between the change of ethological status and concentration of certain cytokines in blood in experimental desynchronosis under led lighting.
[So] Source:Patol Fiziol Eksp Ter;60(4):93-100, 2016 Oct-Dec.
[Is] ISSN:0031-2991
[Cp] País de publicação:Russia (Federation)
[La] Idioma:eng
[Ab] Resumo:Changing the natural rhythm of day and night leads to the development of DS, disruption of coordinated muscular activity, adequate behavioral activity, a decrease of attention in the performance of night work by experts in various fields. Changes ethological status may potentiate or weaken the changes in the indices of immune status, contribute to the formation of allostatic load at desynchronosis. The purpose: To investigate the relationship between changes ethological status and concentration of certain cytokines in peripheral blood in experimental desynchronosis under LED lighting. Methods: The study was performed on 158 adult guinea pigs, which were randomly assigned into 2 groups: 1 group- animals in the conditions of standard fixed (12 h light / 12 h dark) LED lighting (SFSDO); 2 group- animals with jet lag in terms of LED lighting (DESSDO). Light desynchronosis created by keeping animals at clock coverage for 30 days. Behavioral activity was studied in the test «open field¼ cognitive function was assessed using aqueous «labyrinth¼ Morris. By ELISA was determined on the apparatus in the peripheral blood concentration of interleukin - 4 (IL-4), interferon-gamma (IFN-g), melatonin, cortisol via specific for guinea pig test systems. Results: It was found that in animals of DS in terms of LED lighting in the dynamics of 10-30 days of observation show signs of anxiety, depression orienting-exploratory behavior, reduce the long-term memory and learning ability, spatial orientation disorders. It found that when a jet lag LED lighting conditions for 10 days, 20 days and 30 days in peripheral blood melatonin concentration decreases, the concentration of cortisol rises. In peripheral blood decreased IL-4 concentrations of 20 and 30 days, reducing the concentration of IFN-g at 30 days. Based on the results of correlation analysis, ethological change status and progress of cognitive function with a decrease in the blood concentration of IL-4 and IFN-g, the concentration of melatonin increase cortisol levels. Conclusion: The results indicate that in experimental conditions in desynchronosis LED lighting changes ethological status are associated with the progression of immune status changes.
[Mh] Termos MeSH primário: Transtornos Cronobiológicos/sangue
Interferon gama/sangue
Interleucina-4/sangue
Iluminação
[Mh] Termos MeSH secundário: Animais
Transtornos Cronobiológicos/fisiopatologia
Cobaias
Masculino
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
207137-56-2 (Interleukin-4); 82115-62-6 (Interferon-gamma)
[Em] Mês de entrada:1801
[Cu] Atualização por classe:180118
[Lr] Data última revisão:
180118
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171216
[St] Status:MEDLINE


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[PMID]:28973223
[Au] Autor:Swanson CM; Shea SA; Wolfe P; Cain SW; Munch M; Vujovic N; Czeisler CA; Buxton OM; Orwoll ES
[Ad] Endereço:Division of Endocrinology and Bone and Mineral Unit, Oregon Health & Science University, Portland, Oregon 97239.
[Ti] Título:Bone Turnover Markers After Sleep Restriction and Circadian Disruption: A Mechanism for Sleep-Related Bone Loss in Humans.
[So] Source:J Clin Endocrinol Metab;102(10):3722-3730, 2017 Oct 01.
[Is] ISSN:1945-7197
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Context: Sleep abnormalities are associated with low bone mineral density. Underlying mechanisms are unknown. Objective: Investigate the impact of sleep restriction with circadian disruption on bone biomarkers. Design: Intervention study. Participants and Methods: Four bone biomarkers [C-terminal cross-linked telopeptide of type I collagen (CTX) = bone resorption, N-terminal propeptide of type I procollagen (P1NP) = bone formation, sclerostin and fibroblast growth factor 23 = osteocyte function] were measured in bihourly serum samples over 24 hours at baseline and after ∼3 weeks of sleep restriction (5.6 hours sleep/24 hours) with concurrent circadian disruption (recurring 28-hour "day" in dim light) in 10 men (age groups: 20 to 27 years, n = 6; 55 to 65 years, n = 4). The effects of sleep/circadian disruption and age on bone biomarker levels were evaluated using maximum likelihood estimation in a mixed model for repeated measures. Results: P1NP levels were lower after intervention compared with baseline (P < 0.001); the decrease in P1NP was greater for younger compared with older men (28.0% vs 18.2%, P < 0.001). There was no change in CTX (Δ = 0.03 ± 0.02 ng/mL, P = 0.10). Sclerostin levels were higher postintervention in the younger men only (Δ = 22.9% or 5.64 ± 1.10 pmol/L, P < 0.001). Conclusions: These data suggest that 3 weeks of circadian disruption with concurrent sleep restriction can lead to an uncoupling of bone turnover wherein bone formation is decreased but bone resorption is unchanged. Circadian disruption and sleep restriction may be most detrimental to bone in early adulthood.
[Mh] Termos MeSH primário: Biomarcadores/sangue
Remodelação Óssea/fisiologia
Reabsorção Óssea/etiologia
Transtornos Cronobiológicos/metabolismo
Privação do Sono/metabolismo
Sono/fisiologia
[Mh] Termos MeSH secundário: Adulto
Idoso
Reabsorção Óssea/metabolismo
Reabsorção Óssea/fisiopatologia
Transtornos Cronobiológicos/etiologia
Transtornos Cronobiológicos/fisiopatologia
Ritmo Circadiano/fisiologia
Seres Humanos
Masculino
Meia-Idade
Polissonografia
Privação do Sono/complicações
Privação do Sono/fisiopatologia
Adulto Jovem
[Pt] Tipo de publicação:CLINICAL TRIAL; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Biomarkers)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171031
[Lr] Data última revisão:
171031
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:171004
[St] Status:MEDLINE
[do] DOI:10.1210/jc.2017-01147


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[PMID]:28851750
[Au] Autor:Báez-Ruiz A; Guerrero-Vargas NN; Cázarez-Márquez F; Sabath E; Basualdo MDC; Salgado-Delgado R; Escobar C; Buijs RM
[Ad] Endereço:Department of Physiology and Cellular BiologyBiomedical Research Institute, UNAM, DF, México.
[Ti] Título:Food in synchrony with melatonin and corticosterone relieves constant light disturbed metabolism.
[So] Source:J Endocrinol;235(3):167-178, 2017 Dec.
[Is] ISSN:1479-6805
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Circadian disruption is associated with metabolic disturbances such as hepatic steatosis (HS), obesity and type 2 diabetes. We hypothesized that HS, resulting from constant light (LL) exposure is due to an inconsistency between signals related to food intake and endocrine-driven suprachiasmatic nucleus (SCN) outputs. Indeed, exposing rats to LL induced locomotor, food intake and hormone arrhythmicity together with the development of HS. We investigated whether providing temporal signals such as 12-h food availability or driving a corticosterone plus melatonin rhythm could restore rhythmicity and prevent the metabolic disturbances under LL conditions in male rats. Discrete metabolic improvements under these separate treatments stimulated us to investigate whether the combination of hormone treatment together with mealtime restriction (12-h food during four weeks) could prevent the metabolic alterations. LL exposed arrhythmic rats, received daily administration of corticosterone (2.5 µg/kg) and melatonin (2.5 mg/kg) in synchrony or out of synchrony with their 12-h meal. HS and other metabolic alterations were importantly ameliorated in LL-exposed rats receiving hormonal treatment in synchrony with 12-h restricted mealtime, while treatment out of phase with meal time did not. Interestingly, liver bile acids, a major indication for HS, were only normalized when animals received hormones in synchrony with food indicating that disrupted bile acid metabolism might be an important mechanism for the HS induction under LL conditions. We conclude that food-elicited signals, as well as hormonal signals, are necessary for liver synchronization and that HS arises when there is conflict between food intake and the normal pattern of melatonin and corticosterone.
[Mh] Termos MeSH primário: Transtornos Cronobiológicos/complicações
Corticosterona/administração & dosagem
Fígado Gorduroso/etiologia
Métodos de Alimentação
Melatonina/administração & dosagem
Núcleo Supraquiasmático/fisiopatologia
[Mh] Termos MeSH secundário: Adiposidade/efeitos dos fármacos
Animais
Transtornos Cronobiológicos/fisiopatologia
Transtornos Cronobiológicos/prevenção & controle
Fígado Gorduroso/metabolismo
Fígado Gorduroso/prevenção & controle
Transtornos do Metabolismo de Glucose/etiologia
Transtornos do Metabolismo de Glucose/prevenção & controle
Luz/efeitos adversos
Masculino
Ratos Wistar
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
JL5DK93RCL (Melatonin); W980KJ009P (Corticosterone)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171013
[Lr] Data última revisão:
171013
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170831
[St] Status:MEDLINE
[do] DOI:10.1530/JOE-17-0370


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[PMID]:28368364
[Au] Autor:Tain YL; Lin YJ; Sheen JM; Yu HR; Tiao MM; Chen CC; Tsai CC; Huang LT; Hsu CN
[Ad] Endereço:Department of Pediatrics, Kaohsiung Chang Gung Memorial Hospital and Chang Gung University College of Medicine, Kaohsiung 833, Taiwan. tainyl@hotmail.com.
[Ti] Título:High Fat Diets Sex-Specifically Affect the Renal Transcriptome and Program Obesity, Kidney Injury, and Hypertension in the Offspring.
[So] Source:Nutrients;9(4), 2017 Apr 03.
[Is] ISSN:2072-6643
[Cp] País de publicação:Switzerland
[La] Idioma:eng
[Ab] Resumo:Obesity and related disorders have increased concurrently with an increased consumption of saturated fatty acids. We examined whether post-weaning high fat (HF) diet would exacerbate offspring vulnerability to maternal HF-induced programmed hypertension and kidney disease sex-specifically, with a focus on the kidney. Next, we aimed to elucidate the gene-diet interactions that contribute to maternal HF-induced renal programming using the next generation RNA sequencing (NGS) technology. Female Sprague-Dawley rats received either a normal diet (ND) or HF diet (D12331, Research Diets) for five weeks before the delivery. The offspring of both sexes were put on either the ND or HF diet from weaning to six months of age, resulting in four groups of each sex (maternal diet/post-weaning diet; = 5-7/group): ND/ND, ND/HF, HF/ND, and HF/HF. Post-weaning HF diet increased bodyweights of both ND/HF and HF/HF animals from three to six months only in males. Post-weaning HF diet increased systolic blood pressure in male and female offspring, irrespective of whether they were exposed to maternal HF or not. Male HF/HF offspring showed greater degrees of glomerular and tubular injury compared to the ND/ND group. Our NGS data showed that maternal HF diet significantly altered renal transcriptome with female offspring being more HF-sensitive. HF diet induced hypertension and renal injury are associated with oxidative stress, activation of renin-angiotensin system, and dysregulated sodium transporters and circadian clock. Post-weaning HF diet sex-specifically exacerbates the development of obesity, kidney injury, but not hypertension programmed by maternal HF intake. Better understanding of the sex-dependent mechanisms that underlie HF-induced renal programming will help develop a novel personalized dietary intervention to prevent obesity and related disorders.
[Mh] Termos MeSH primário: Dieta Hiperlipídica/efeitos adversos
Desenvolvimento Fetal
Hipertensão/etiologia
Lactação
Fenômenos Fisiológicos da Nutrição Materna
Obesidade/etiologia
Insuficiência Renal/etiologia
[Mh] Termos MeSH secundário: Animais
Transtornos Cronobiológicos/etiologia
Transtornos Cronobiológicos/metabolismo
Transtornos Cronobiológicos/fisiopatologia
Relógios Circadianos
Feminino
Perfilação da Expressão Gênica
Regulação da Expressão Gênica no Desenvolvimento
Hipertensão/metabolismo
Hipertensão/patologia
Hipertensão/fisiopatologia
Rim/metabolismo
Rim/patologia
Rim/fisiopatologia
Masculino
Obesidade/metabolismo
Obesidade/patologia
Obesidade/fisiopatologia
Estresse Oxidativo
Gravidez
Ratos Sprague-Dawley
Insuficiência Renal/metabolismo
Insuficiência Renal/patologia
Insuficiência Renal/fisiopatologia
Fatores Sexuais
Transcriptoma
Desmame
Ganho de Peso
[Pt] Tipo de publicação:COMPARATIVE STUDY; JOURNAL ARTICLE
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170830
[Lr] Data última revisão:
170830
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170404
[St] Status:MEDLINE


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[PMID]:28321642
[Au] Autor:Alloy LB; Ng TH; Titone MK; Boland EM
[Ad] Endereço:Department of Psychology, Temple University, 1701 N. 13th Street, Philadelphia, PA, 19122, USA. lalloy@temple.edu.
[Ti] Título:Circadian Rhythm Dysregulation in Bipolar Spectrum Disorders.
[So] Source:Curr Psychiatry Rep;19(4):21, 2017 Apr.
[Is] ISSN:1535-1645
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:PURPOSE OF REVIEW: We review recent evidence for circadian rhythm dysregulation in bipolar spectrum disorders (BSDs). We examine evidence for endogenous abnormalities in the biological clock and disruptions in the external entrainment of circadian rhythms in BSDs. We also address whether circadian dysregulation provides vulnerability to onset of BSD and evidence for a new integration of reward and circadian dysregulation in BSD. RECENT FINDINGS: Relative circadian phase delay (e.g., later melatonin peak, evening chronotype) is associated with BSD, particularly in the depressive phase. More consistent evidence supports irregularity of social rhythms, sleep/wake and activity patterns, and disruptions of social rhythms by life events, as stable trait markers of BSD and potential vulnerabilities for BSD onset. Growing research supports an integrative reward/circadian model. Both endogenous abnormalities in the biological clock pacemaking function and disruptions in the external entrainment of circadian rhythms by physical and social cues are involved in BSDs. Circadian dysregulation may provide vulnerability to BSD onset.
[Mh] Termos MeSH primário: Transtorno Bipolar
Transtornos Cronobiológicos
[Mh] Termos MeSH secundário: Transtorno Bipolar/diagnóstico
Transtorno Bipolar/fisiopatologia
Transtornos Cronobiológicos/complicações
Transtornos Cronobiológicos/psicologia
Ritmo Circadiano/fisiologia
Seres Humanos
Melatonina/metabolismo
Psicopatologia
Sono/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
JL5DK93RCL (Melatonin)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:171107
[Lr] Data última revisão:
171107
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170322
[St] Status:MEDLINE
[do] DOI:10.1007/s11920-017-0772-z


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[PMID]:28318412
[Au] Autor:Xiao B; Cui LQ; Ding C; Wang H
[Ad] Endereço:1 Key Laboratory for Ecology and Pollution Control of Coastal Wetlands, School of Environmental Science and Engineering, Yancheng Institute of Technology , Yancheng, Jiangsu, China .
[Ti] Título:Effects of Lithium and 2,4-Dichlorophenol on Zebrafish: Circadian Rhythm Disorder and Molecular Effects.
[So] Source:Zebrafish;14(3):209-215, 2017 Jun.
[Is] ISSN:1557-8542
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The aim of this study was to investigate lithium and 2,4-dichlorophenol (2,4-DCP)-induced circadian rhythm disorder and their genome-wide effects in zebrafish. Zebrafish larvae were exposed to 250 ppm LiCl (n = 40) or 20 ppm 2,4-DCP. RNA was subsequently extracted and determined quantitatively. The mRNA levels of circadian clock-related genes, including clock1a, bmal1b, per2, and per1b, were determined. Microarray datasets were generated and the differentially expressed genes (DEGs) were identified. The mRNA levels of some upregulated and downregulated DEGs were examined by quantitative real-time polymerase chain reaction (RT-PCR). Finally, gene ontology (GO) enrichment analysis was applied to determine the roles of the DEGs. The mRNA expression levels of circadian rhythm-related genes in the daily cycle were significantly affected after incubation of zebrafish with LiCl and 2,4-DCP. Many genes were differentially expressed during the light phase (97 h) and RT-PCR validation tests revealed that the expression patterns of DEGs were in accordance with those obtained by microarray analysis. GO functional enrichment analysis showed that the DEGs in LiCl- and 2,4-DCP-treated groups were associated with signal transduction and development. Collectively, our findings indicate that LiCl and 2,4-DCP could affect signal transduction pathways and immune response, thereby inducing circadian rhythm disorder.
[Mh] Termos MeSH primário: Clorofenóis/toxicidade
Transtornos Cronobiológicos/genética
Ritmo Circadiano/efeitos dos fármacos
Regulação da Expressão Gênica no Desenvolvimento/efeitos dos fármacos
Lítio/toxicidade
Poluentes da Água/toxicidade
Peixe-Zebra/fisiologia
[Mh] Termos MeSH secundário: Animais
Anti-Helmínticos/farmacologia
Transtornos Cronobiológicos/patologia
Perfilação da Expressão Gênica
Ontologia Genética
Larva/efeitos dos fármacos
Larva/metabolismo
RNA Mensageiro/metabolismo
Transdução de Sinais/efeitos dos fármacos
Peixe-Zebra/crescimento & desenvolvimento
Proteínas de Peixe-Zebra/genética
Proteínas de Peixe-Zebra/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Anthelmintics); 0 (Chlorophenols); 0 (RNA, Messenger); 0 (Water Pollutants); 0 (Zebrafish Proteins); 9FN79X2M3F (Lithium); R669TG1950 (2,4-dichlorophenol)
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171102
[Lr] Data última revisão:
171102
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170321
[St] Status:MEDLINE
[do] DOI:10.1089/zeb.2016.1389


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[PMID]:28314121
[Au] Autor:Tu CY; Tseng MM; Chang CH; Lin CC
[Ad] Endereço:Department of Psychiatry, National Taiwan University Hospital, Yunlin Branch, Yunlin, 64041. Electronic address: t70552@hotmail.com.
[Ti] Título:Comparative validity of the Internet and paper-and-pencil versions of the Night Eating Questionnaire.
[So] Source:Compr Psychiatry;75:53-61, 2017 May.
[Is] ISSN:1532-8384
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: This study examined the psychometric properties of the Internet and paper-and-pencil versions of the Mandarin Chinese version of the Night Eating Questionnaire (C-NEQ) and compared these measures' validity. METHOD: The C-NEQ was evaluated through two different media: 626 participants completed the C-NEQ on the Internet and 160 participants completed the paper-form C-NEQ at the psychiatric outpatient clinics. A subgroup completed both versions of the C-NEQ (n=50). The Night Eating Syndrome History and Inventory was used to identify individuals with night eating syndrome (NES). RESULTS: The paper-and-pencil and Internet versions of the C-NEQ both showed good internal consistency, reliability, and concurrent validity. Reliability between the Internet and the paper-and-pencil versions of the C-NEQ was excellent (ICC=.96). Diagnostic analysis of the C-NEQ's performance using the Receiver Operation Curve method showed excellent results in both versions; the area under the curve did not differ significantly between the versions. Regarding detecting NES, the Internet version had a higher optimal cutoff point than the paper-and-pencil version (23 and 22, respectively). CONCLUSIONS: The Internet and paper-and-pencil versions of the C-NEQ both showed strong reliability and validity; however the two versions appear to differ marginally regarding usage in NES detection.
[Mh] Termos MeSH primário: Transtornos Cronobiológicos/diagnóstico
Ingestão de Alimentos/psicologia
Transtornos da Alimentação e da Ingestão de Alimentos/diagnóstico
Inquéritos e Questionários
[Mh] Termos MeSH secundário: Transtornos Cronobiológicos/psicologia
Transtornos da Alimentação e da Ingestão de Alimentos/psicologia
Feminino
Seres Humanos
Internet
Linguagem
Masculino
Psicometria
Curva ROC
Reprodutibilidade dos Testes
Traduções
[Pt] Tipo de publicação:JOURNAL ARTICLE; VALIDATION STUDIES
[Em] Mês de entrada:1706
[Cu] Atualização por classe:170607
[Lr] Data última revisão:
170607
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170318
[St] Status:MEDLINE


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[PMID]:28164454
[Au] Autor:Dulloo AG; Miles-Chan JL; Montani JP
[Ad] Endereço:Department of Medicine, Division of Physiology, University of Fribourg, Fribourg, Switzerland.
[Ti] Título:Nutrition, movement and sleep behaviours: their interactions in pathways to obesity and cardiometabolic diseases.
[So] Source:Obes Rev;18 Suppl 1:3-6, 2017 02.
[Is] ISSN:1467-789X
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Among the multitude of dietary and lifestyle behaviours that have been proposed to contribute to the obesity epidemic, those that have generated considerable research scrutiny in the past decade are centred upon sleep behaviours, sedentary behaviours (sitting or lying while awake) and diminished low-level physical activities of everyday life, with each category of behaviours apparently presenting an independent risk for obesity and/or cardiometabolic diseases. These behaviours are highly complex, operate in synergy with each other, disrupt the link between regulation of the circadian clock and metabolic physiology and impact on various components of daily energy expenditure and feeding behaviours to promote obesity and hinder the outcome of obesity therapy. As such, this behavioural triad (nutrition, movement and sleep) presents plenty of scope for intervention and optimization in the context of body weight regulation and lifestyle-related disease prevention. It is against this background that recent advances relevant to the theme of 'Nutrition, Movement & Sleep Behaviors: their interactions in pathways to obesity and cardiometabolic diseases' are addressed in this overview and the nine review articles in this supplement reporting the proceedings of the 8th Fribourg Obesity Research Conference.
[Mh] Termos MeSH primário: Doenças Cardiovasculares/prevenção & controle
Dieta
Exercício
Síndrome Metabólica/prevenção & controle
Obesidade/prevenção & controle
Sono
[Mh] Termos MeSH secundário: Adiposidade
Transtornos Cronobiológicos/prevenção & controle
Comportamentos Relacionados com a Saúde
Seres Humanos
Estilo de Vida
[Pt] Tipo de publicação:INTRODUCTORY JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171116
[Lr] Data última revisão:
171116
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170207
[St] Status:MEDLINE
[do] DOI:10.1111/obr.12513



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