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[PMID]:28834867
[Au] Autor:Lalji A; Izbudak I; Birnbaum J
[Ad] Endereço:aThe Johns Hopkins University School of Medicine bDivision of Neuroradiology, Department of Radiology and Radiological Sciences cDivision of Rheumatology and Department of Neurology, The Johns Hopkins University School of Medicine, Baltimore, MD.
[Ti] Título:Cortical blindness and not optic neuritis as a cause of vision loss in a Sjögren's syndrome (SS) patient with the neuromyelitis optica spectrum disorder (NMOSD): Challenges of ascribing demyelinating syndromes to SS: a case report.
[So] Source:Medicine (Baltimore);96(34):e7454, 2017 Aug.
[Is] ISSN:1536-5964
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:RATIONALE: The conception that multiple sclerosis may be challenging to distinguish from demyelinating manifestations of Sjögren's syndrome (SS) was introduced more than 30 years ago. However, it is now recognized that the neuromyelitis optica spectrum disorder (NMOSD) may occur more frequently in SS as opposed to multiple sclerosis. Characteristic NMOSD features can include severe attacks of optic neuritis, myelitis which is frequently longitudinally-extensive (spanning at least three vertebral segments on magnetic resonance imaging [MRI]), and an association with anti-aquaporin-4 antibodies. In addition, whereas NMOSD was initially thought to spare the brain, it is now recognized that brain lesions occur in a majority of NMOSD patients. Therefore, it is important for the multi-disciplinary team of physicians who care for SS patients to understand this widening spectrum of NMOSD as encompassing brain lesions. In this case-report we describe clinical features, radiographic findings, and treatment of a SS NMOSD patient presenting with severely decreased visual acuity, visual hallucinations, and encephalopathy. PATIENT CONCERNS: The SS NMOSD patient presented with rapid, bilateral onset of severely decreased visual acuity and was therefore suspected as having bilateral optic neuritis. DIAGNOSIS: However, the patient lacked stigmata of optic neuritis, instead had visual hallucinations and encephalopathy suggestive of cortical blindness, and was noted to have occipital lobe lesions on brain MRI. Other radiographic findings included simultaneous enhancement of brainstem and periventricular lesions. INTERVENTIONS: The patient was initially treated with methylprednisolone with no change in her neurological deficits. She was then treated with plasma exchange therapy. OUTCOMES: The patient had resolution of decreased visual acuity, visual hallucinations, encephalopathy, and contrast-enhancing brain lesions in response to plasma exchange therapy. LESSON: We provide the first example of severely decreased visual acuity in a NMOSD patient due to cortical blindness and not bilateral optic neuritis. This finding expands the spectrum of central nervous system syndromes and brain lesions which may occur in NMOSD. The synchronous enhancement of a brainstem lesion (known to occur in NMOSD) with occipital lobe lesions also suggests that our patient's occipital lobe findings were due to NMOSD. All of our patient's findings had an excellent clinical and radiographic response to plasma exchange therapy.
[Mh] Termos MeSH primário: Cegueira Cortical/diagnóstico
Cegueira Cortical/etiologia
Neuromielite Óptica/complicações
Síndrome de Sjogren/complicações
[Mh] Termos MeSH secundário: Idoso
Cegueira Cortical/diagnóstico por imagem
Feminino
Seres Humanos
Imagem por Ressonância Magnética
Neurite Óptica/diagnóstico
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170911
[Lr] Data última revisão:
170911
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170824
[St] Status:MEDLINE
[do] DOI:10.1097/MD.0000000000007454


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[PMID]:28404802
[Au] Autor:Cavanaugh MR; Huxlin KR
[Ad] Endereço:From the Flaum Eye Institute, University of Rochester, NY.
[Ti] Título:Visual discrimination training improves Humphrey perimetry in chronic cortically induced blindness.
[So] Source:Neurology;88(19):1856-1864, 2017 May 09.
[Is] ISSN:1526-632X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: To assess if visual discrimination training improves performance on visual perimetry tests in chronic stroke patients with visual cortex involvement. METHODS: 24-2 and 10-2 Humphrey visual fields were analyzed for 17 chronic cortically blind stroke patients prior to and following visual discrimination training, as well as in 5 untrained, cortically blind controls. Trained patients practiced direction discrimination, orientation discrimination, or both, at nonoverlapping, blind field locations. All pretraining and posttraining discrimination performance and Humphrey fields were collected with online eye tracking, ensuring gaze-contingent stimulus presentation. RESULTS: Trained patients recovered ∼108 degrees of vision on average, while untrained patients spontaneously improved over an area of ∼16 degrees . Improvement was not affected by patient age, time since lesion, size of initial deficit, or training type, but was proportional to the amount of training performed. Untrained patients counterbalanced their improvements with worsening of sensitivity over ∼9 degrees of their visual field. Worsening was minimal in trained patients. Finally, although discrimination performance improved at all trained locations, changes in Humphrey sensitivity occurred both within trained regions and beyond, extending over a larger area along the blind field border. CONCLUSIONS: In adults with chronic cortical visual impairment, the blind field border appears to have enhanced plastic potential, which can be recruited by gaze-controlled visual discrimination training to expand the visible field. Our findings underscore a critical need for future studies to measure the effects of vision restoration approaches on perimetry in larger cohorts of patients.
[Mh] Termos MeSH primário: Cegueira Cortical/etiologia
Cegueira Cortical/reabilitação
Discriminação (Psicologia)
Reabilitação do Acidente Vascular Cerebral
Acidente Vascular Cerebral/complicações
Percepção Visual
[Mh] Termos MeSH secundário: Adulto
Idoso
Cegueira Cortical/patologia
Medições dos Movimentos Oculares
Feminino
Lateralidade Funcional
Seres Humanos
Masculino
Meia-Idade
Estimulação Luminosa
Estudos Retrospectivos
Acidente Vascular Cerebral/patologia
Resultado do Tratamento
Córtex Visual/patologia
Testes de Campo Visual
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1706
[Cu] Atualização por classe:170602
[Lr] Data última revisão:
170602
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170414
[St] Status:MEDLINE
[do] DOI:10.1212/WNL.0000000000003921


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[PMID]:28393793
[Au] Autor:Bharati S; Sharma MK; Chattopadhay A; Das D
[Ad] Endereço:Department of Anaesthesiology, Narayana Superspeciality Hospital, Howrah, Kolkata, West Bengal, India.
[Ti] Título:Transient cortical blindness following intracardiac repair of congenital heart disease in an 11-year-old boy.
[So] Source:Ann Card Anaesth;20(2):256-258, 2017 Apr-Jun.
[Is] ISSN:0974-5181
[Cp] País de publicação:India
[La] Idioma:eng
[Ab] Resumo:Postoperative blindness (PB) primarily involves reception and conductance parts of the visual pathway due to ischemia following cessation of blood supply, for example, retinal vascular occlusion. Although a rare cause of PB, cortical blindness (CB), which results from ischemia/infarction of visual cortex, has a poor outcome due to its mostly nonreversible nature. Ischemic optic neuropathy is the most common cause of PB following cardiac surgeries. CB following cardiac surgeries involving cardiopulmonary bypass has been rarely reported. Only a few of those articles reported partial or complete reversal of CB. We report an incidence of transient CB in an 11-year-old child who was operated for double chambered right ventricle with ventricular septal defect.
[Mh] Termos MeSH primário: Cegueira Cortical/etiologia
Procedimentos Cirúrgicos Cardíacos/efeitos adversos
Comunicação Interventricular/cirurgia
Complicações Pós-Operatórias/etiologia
[Mh] Termos MeSH secundário: Cegueira Cortical/terapia
Criança
Tratamento Conservador/métodos
Seres Humanos
Masculino
Complicações Pós-Operatórias/terapia
[Pt] Tipo de publicação:CASE REPORTS
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171102
[Lr] Data última revisão:
171102
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170411
[St] Status:MEDLINE
[do] DOI:10.4103/aca.ACA_159_16


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[PMID]:28378817
[Au] Autor:O'Brien A; Marshall CR; Blaser S; Ray PN; Yoon G
[Ad] Endereço:Department of Paediatrics, Division of Clinical and Metabolic Genetics, The Hospital for Sick Children, Toronto, Ontario, Canada.
[Ti] Título:Severe neurodegeneration, progressive cerebral volume loss and diffuse hypomyelination associated with a homozygous frameshift mutation in CSTB.
[So] Source:Eur J Hum Genet;25(6):775-778, 2017 Jun.
[Is] ISSN:1476-5438
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Mutations of the cystatin B gene (CSTB; OMIM 601145) are known to cause Unverricht-Lundborg disease or progressive myoclonic epilepsy-1A (EPM1A, MIM #254800). Most patients are homozygous for an expanded (>30) dodecamer repeat in the promoter region of CSTB, or are compound heterozygotes for the dodecamer repeat and a point mutation. We report two adolescent sisters born to consanguineous parents of Sri Lankan descent who presented with profound global developmental delay, microcephaly, cortical blindness and axial hypotonia with appendicular hypertonia. Neither sibling ever developed head control, independent sitting or ambulation, and never developed speech. The elder sister had a seizure disorder. Both sisters had profound microcephaly and distinct facial features. On serial brain imaging, they had progressive atrophy of the corpus callosum and supratentorial brain, and diffuse hypomyelination with progressive loss of myelin signal. Exome sequencing revealed both siblings to be homozygous for a c.218dupT (p.His75Serfs*2) mutation in exon 3 of CSTB. The neuroimaging features of our patients are consistent with those observed in Cstb-knockout mice, which supports the hypothesis that disease severity is inversely correlated with the amount of residual functional cystatin B protein.
[Mh] Termos MeSH primário: Cegueira Cortical/genética
Cistatina B/genética
Deficiências do Desenvolvimento/genética
Mutação da Fase de Leitura
Doenças Desmielinizantes Hereditárias do Sistema Nervoso Central/genética
Microcefalia/genética
[Mh] Termos MeSH secundário: Adolescente
Cegueira Cortical/diagnóstico
Criança
Corpo Caloso/diagnóstico por imagem
Corpo Caloso/patologia
Deficiências do Desenvolvimento/diagnóstico
Feminino
Doenças Desmielinizantes Hereditárias do Sistema Nervoso Central/diagnóstico
Homozigoto
Seres Humanos
Masculino
Microcefalia/diagnóstico
Bainha de Mielina/patologia
Linhagem
Síndrome
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (CSTB protein, human); 88844-95-5 (Cystatin B)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170912
[Lr] Data última revisão:
170912
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170406
[St] Status:MEDLINE
[do] DOI:10.1038/ejhg.2017.39


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[PMID]:28357826
[Au] Autor:Azzopardi CP; Matity L; Muscat S
[Ad] Endereço:Hyperbaric Unit, Mater Dei Hospital, Msida, Malta MSD 2090, charles.a.azzopardi@gov.mt.
[Ti] Título:Anton's syndrome as a presentation of decompression illness.
[So] Source:Diving Hyperb Med;47(1):59-60, 2017 Mar.
[Is] ISSN:1833-3516
[Cp] País de publicação:Australia
[La] Idioma:eng
[Ab] Resumo:We present a case of a patient with Anton's syndrome due to decompression illness (DCI) after recreational scuba diving. Visual anosognosia, or denial of loss of vision, which is associated with lack of awareness regarding visual loss in the setting of cortical blindness, is known as Anton's syndrome (also termed Anton-Babinski syndrome). Our patient presented with progressive neurological DCI treated with repeated recompression. The anosogosia resolved after 48 h. Subsequent echocardiography revealed a persistent (patent) foramen ovale.
[Mh] Termos MeSH primário: Agnosia/etiologia
Cegueira Cortical/etiologia
Doença da Descompressão/complicações
[Mh] Termos MeSH secundário: Agnosia/diagnóstico
Cegueira Cortical/diagnóstico
Encéfalo/diagnóstico por imagem
Doença da Descompressão/diagnóstico por imagem
Forame Oval Patente/diagnóstico por imagem
Seres Humanos
Imagem por Ressonância Magnética
Masculino
Meia-Idade
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170502
[Lr] Data última revisão:
170502
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170331
[St] Status:MEDLINE


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[PMID]:28319878
[Au] Autor:Tipura E; Pegna AJ; de Gelder B; Renaud O
[Ad] Endereço:Methodology and Data Analysis, Department of Psychology, University of Geneva, Switzerland; Distance Learning University, Sierre, Switzerland. Electronic address: eda.tipura@unige.ch.
[Ti] Título:Visual stimuli modulate frontal oscillatory rhythms in a cortically blind patient: Evidence for top-down visual processing.
[So] Source:Clin Neurophysiol;128(5):770-779, 2017 May.
[Is] ISSN:1872-8952
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: We investigated neuronal correlates of faces versus non-faces processing in a cortically blind patient (TN) and a group of healthy age-matched controls in order to test electrophysiological correlates of the processing of pertinent stimuli in this patient. METHODS: An EEG paradigm was used, in which intact and scrambled faces were displayed on a screen. First, time-frequency transforms were conducted on the patients' data alone. These oscillations were then compared to the frontal activity of six control participants. RESULTS: Post stimulus oscillatory modulations (synchronisation in theta and alpha frequency bands) of both intact and scrambled faces at frontal scalp sites were observed in TN. These modulations were different for correct and incorrect responses. A more important increase in the theta band for incorrect responses was observed. The oscillatory rhythms highlighted in blindsight and in frontal regions differ from the ones observed in control participants. CONCLUSION: Despite the destruction of the visual cortex, oscillatory rhythms are not cancelled out but are shifted to anterior regions, revealing the activity of an alternate pathway for residual visual function. SIGNIFICANCE: The results provide evidence for a top-down cognitive control process in blindsight.
[Mh] Termos MeSH primário: Cegueira Cortical/fisiopatologia
Lobo Frontal/fisiopatologia
Ritmo Teta
Percepção Visual
[Mh] Termos MeSH secundário: Cegueira Cortical/diagnóstico
Estudos de Casos e Controles
Seres Humanos
Masculino
Meia-Idade
Vias Visuais/fisiopatologia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170530
[Lr] Data última revisão:
170530
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170321
[St] Status:MEDLINE


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[PMID]:28300737
[Au] Autor:Philip SS
[Ad] Endereço:Department of Ophthalmology, Cerebral Visual Impairment Clinic, Christian Medical College and Hospital, Vellore, Tamil Nadu, India.
[Ti] Título:Setting up of a cerebral visual impairment clinic for children: Challenges and future developments.
[So] Source:Indian J Ophthalmol;65(1):30-34, 2017 Jan.
[Is] ISSN:1998-3689
[Cp] País de publicação:India
[La] Idioma:eng
[Ab] Resumo:AIM: The aim of this study is to describe the setting up of a cerebral visual impairment (CVI) clinic in a tertiary care hospital in South India and to describe the spectrum of cases seen. MATERIALS AND METHODS: The CVI clinic, set up in February 2011, receives interdisciplinary input from a core team involving a pediatrician, neurologist, psychiatrist, occupational therapist, pediatric ophthalmologist, and an optometrist. All children, <18 years of age, with cerebral palsy (CP), learning disability, autism, neurodegenerative diseases, and brain trauma are referred to the clinic for functional vision assessment and opinion for further management. RESULTS: One thousand four hundred and seventy-eight patients were seen in the CVI clinic from February 2011 to September 2015. Eighty-five percent of the patients were from different parts of India. In the clinic, 61% had CP, 28% had seizure disorders, autism was seen in 9.5%, and learning disability, neurodegenerative conditions, and brain injury together constituted 1.5%. Most of the children (45%) had moderate CP. Forty percent of CVI was due to birth asphyxia, but about 20% did not have any known cause for CVI. Seventy percent of patients, who came back for follow-up, were carrying out the habilitation strategies suggested. CONCLUSIONS: Average attendance of over 300 new patients a year suggests a definite need for CVI clinics in the country. These children need specialized care to handle their complex needs. Although difficult to coordinate, an interdisciplinary team including the support groups and voluntary organizations is needed to facilitate the successful implementation of such specialized service.
[Mh] Termos MeSH primário: Cegueira Cortical/terapia
Paralisia Cerebral/complicações
Gerenciamento Clínico
Centros de Atenção Terciária/estatística & dados numéricos
[Mh] Termos MeSH secundário: Cegueira Cortical/epidemiologia
Cegueira Cortical/etiologia
Paralisia Cerebral/diagnóstico
Criança
Pré-Escolar
Feminino
Seres Humanos
Incidência
Índia/epidemiologia
Imagem por Ressonância Magnética
Masculino
Estudos Retrospectivos
Acuidade Visual
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170809
[Lr] Data última revisão:
170809
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170317
[St] Status:MEDLINE
[do] DOI:10.4103/0301-4738.202303


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[PMID]:28257538
[Au] Autor:Borovac JA; Bozic J; Ticinovic Kurir T; Zaja N; Kolic K; Hrboka V
[Ad] Endereço:From the Department of Pathophysiology, University of Split School of Medicine, Split, the Department of Internal Medicine, University Hospital Center Split, Split, University Psychiatric Hospital Vrapce, University of Zagreb School of Medicine, Zagreb, the Clinical Department of Diagnostic and Inte
[Ti] Título:Multidisciplinary Approach to Complicated Pregnancy.
[So] Source:South Med J;110(3):154-160, 2017 Mar.
[Is] ISSN:1541-8243
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:A nulliparous pregnant woman in her mid-20s and in the 32nd week of gestation presented to the emergency department with severe headache and vomiting. She had an uneventful medical history; however, the physical examination upon hospital admission revealed a hypertensive emergency, papilledema, and 2+ dipstick proteinuria. Upon establishing the diagnosis of preeclampsia, aggressive therapy with corticosteroids, antihypertensive medication, and seizure prophylaxis was initiated. Hemodynamic stability was achieved within 24 hours and the patient remained in the observation unit located within the gynecology clinic. On the ninth day postadmission, however, her condition abruptly deteriorated and advanced to imminent eclampsia, accompanied by transient vision loss, altered mental status, and acute hypertensive crisis. After the patient underwent successful emergent delivery via caesarean section, a laboratory workup revealed hemolysis, elevated liver enzymes, and low platelet count, suggesting HELLP syndrome, a serious complication of eclampsia. This patient concurrently developed posterior reversible encephalopathy syndrome, which was confirmed by magnetic resonance imaging and acute respiratory distress syndrome (the latter presented with diffuse bilateral infiltrates on x-ray and developing pulmonary edema in the absence of cardiac etiology). Because of these life-threatening dynamics, the patient was transferred to the intensive care unit for further treatment. This case is a rare cascade of life-threatening complications that developed in a patient and required skillful multidisciplinary decision making and experienced management within an acute critical care setting. The final outcome of the treatment and intensive care was successful because both the patient and child survived and had no chronic or debilitating sequelae.
[Mh] Termos MeSH primário: Síndrome HELLP/diagnóstico
Síndrome HELLP/terapia
Equipe de Assistência ao Paciente
Pré-Eclâmpsia/diagnóstico
Pré-Eclâmpsia/terapia
[Mh] Termos MeSH secundário: Adulto
Antibacterianos/uso terapêutico
Anticonvulsivantes/uso terapêutico
Anti-Hipertensivos/uso terapêutico
Cegueira Cortical/etiologia
Confusão/etiologia
Feminino
Seres Humanos
Encefalopatia Hipertensiva/diagnóstico por imagem
Encefalopatia Hipertensiva/etiologia
Intubação Intratraqueal
Rim/irrigação sanguínea
Rim/diagnóstico por imagem
Sulfato de Magnésio/uso terapêutico
Gravidez
Síndrome do Desconforto Respiratório do Adulto/etiologia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Anti-Bacterial Agents); 0 (Anticonvulsants); 0 (Antihypertensive Agents); 7487-88-9 (Magnesium Sulfate)
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170525
[Lr] Data última revisão:
170525
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170304
[St] Status:MEDLINE
[do] DOI:10.14423/SMJ.0000000000000618


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[PMID]:28120068
[Au] Autor:Quiñones-Hinojosa A; Raza SM; Lazaridis C; Olivi A
[Ad] Endereço:Department of Neurosurgery, Johns Hopkins School of Medicine, Baltimore, MD, USA.
[Ti] Título:Olfactory Groove Meningiomas: Acute Presentation and Potential : Pitfalls in Management and Functional Restoration.
[So] Source:Acta Neurochir Suppl;124:155-158, 2017.
[Is] ISSN:0065-1419
[Cp] País de publicação:Austria
[La] Idioma:eng
[Ab] Resumo:Meningiomas are typically benign, slow-growing lesions that present after an insidious onset of symptoms related to mass effect. The acute presentation of a patient who has suffered a transtentorial herniation event due to a meningioma is rare. There are only few publications describing such a presentation in the absence of hemorrhage [1]. In this case report, a patient with an olfactory groove meningioma presenting with signs and symptoms of transtentorial herniation in the absence of tumor-associated hemorrhage is discussed. This is a unique presentation of such a lesion. The patient developed Anton's syndrome-binocular visual loss with blindness denial. Management considerations for patients with meningiomas that present with acute deterioration are discussed.
[Mh] Termos MeSH primário: Neoplasias Meníngeas/diagnóstico por imagem
Meningioma/diagnóstico por imagem
[Mh] Termos MeSH secundário: Cegueira Cortical/etiologia
Craniotomia
Imagem de Difusão por Ressonância Magnética
Feminino
Seres Humanos
Imagem por Ressonância Magnética
Neoplasias Meníngeas/complicações
Neoplasias Meníngeas/cirurgia
Meningioma/complicações
Meningioma/cirurgia
Meia-Idade
Procedimentos Neurocirúrgicos
Tomografia Computadorizada por Raios X
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171102
[Lr] Data última revisão:
171102
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170126
[St] Status:MEDLINE
[do] DOI:10.1007/978-3-319-39546-3_24


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[PMID]:27789154
[Au] Autor:Garcia Tirado A; Jimenez-Rolando B; Noval S; Martinez Bermejo A
[Ad] Endereço:Department of Ophtalmology, La Paz University Hospital, IdiPaz, Madrid, Spain. Electronic address: a.gartir@gmail.com.
[Ti] Título:Cortical Blindness in a Child Secondary to Mycoplasma pneumoniae Infection.
[So] Source:J Stroke Cerebrovasc Dis;26(1):e12-e13, 2017 Jan.
[Is] ISSN:1532-8511
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Our objective is to present a case of an uncommon complication associated with Mycoplasma pneumoniae infection in a child where cortical blindness was the main clinical feature. Stroke due to an infection by M. pneumoniae is very uncommon. No consensus has been reached on the pathogenesis, although several pathogenic mechanisms have been proposed. Occlusion of posterior cerebral circulation is the most uncommon central nervous system complication of M. pneumoniae infection being reported. Symptoms are usually hemiplegia and dysarthria. We report a case of a 6-year-old boy who suffered cortical blindness due to a stroke 2 days after M. pneumoniae infection. This is the first case of documented cortical blindness due to posterior cerebral arteries occlusion in children after M. pneumoniae infection.
[Mh] Termos MeSH primário: Cegueira Cortical/etiologia
Cegueira Cortical/microbiologia
Pneumonia por Mycoplasma/complicações
[Mh] Termos MeSH secundário: Cegueira Cortical/diagnóstico por imagem
Criança
Angiografia por Tomografia Computadorizada
Seres Humanos
Masculino
Mycoplasma pneumoniae/patogenicidade
Pneumonia por Mycoplasma/diagnóstico por imagem
Tomógrafos Computadorizados
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1704
[Cu] Atualização por classe:170410
[Lr] Data última revisão:
170410
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161030
[St] Status:MEDLINE



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