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  1 / 2067 MEDLINE  
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[PMID]:29390381
[Au] Autor:Ek S; Rosenborg S
[Ad] Endereço:Department of Obstetrics and Gynecology, Center of Fetal Medicine, Karolinska University Hospital.
[Ti] Título:Mesalazine as a cause of fetal anemia and hydrops fetalis: A case report.
[So] Source:Medicine (Baltimore);96(50):e9277, 2017 Dec.
[Is] ISSN:1536-5964
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:RATIONALE: Mesalazine and its prodrug sulfasalazine are both used for inflammatory bowel disease. Sulfasalazine has been associated with hematological side-effects such as aplastic and hemolytic anemia in patients, but also in fetuses after intrauterine exposure. To our knowledge, we describe the first case of a fetus with severe anemia, and subsequent hydrops, where this drug was found at concentrations in the fetus corresponding to those in the mother and most likely responsible for the fetal condition. PATIENT CONCERNS: A uniparous woman was referred at 31 weeks of gestation due to a hydropic fetus with massive ascites and cardiomegaly. DIAGNOSES: The patient had Crohn's disease and was thus treated with 4 g mesalazine daily. The fetus had severe anemia with an initial hemoglobin level of 51 g/L. INTERVENTIONS: The maternal medication was discontinued and four intrauterine erythrocyte transfusions were given during three weeks. Plasma samples were drawn from mother and fetus during cordocentesis for later analysis of mesalazine. OUTCOMES: A healthy baby was born after 37 full weeks of gestation. Plasma levels of mesalazine were non-conspicuous in neither mother nor fetus. The mesalazine half-life in the fetus (37 h) was half that of the mother (80 h), both considerably longer than previously reported (about 19 h). LESSONS: A causal relationship must be suspected between the fetal anemia and the maternal use of mesalazine. This fetal side-effect should be considered in pregnant women on mesalazine (and its prodrug sulfasalazine).
[Mh] Termos MeSH primário: Anemia/induzido quimicamente
Anti-Inflamatórios não Esteroides/efeitos adversos
Doenças Fetais/induzido quimicamente
Hidropisia Fetal/induzido quimicamente
Mesalamina/efeitos adversos
[Mh] Termos MeSH secundário: Anemia/terapia
Transfusão de Eritrócitos
Feminino
Doenças Fetais/terapia
Seres Humanos
Hidropisia Fetal/terapia
Gravidez
Resultado da Gravidez
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Anti-Inflammatory Agents, Non-Steroidal); 4Q81I59GXC (Mesalamine)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180301
[Lr] Data última revisão:
180301
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:180203
[St] Status:MEDLINE
[do] DOI:10.1097/MD.0000000000009277


  2 / 2067 MEDLINE  
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[PMID]:28453379
[Au] Autor:Han M; Afshar Y; Chon AH; Scibetta E; Rao R; Chmait RH
[Ad] Endereço:a Division of Maternal Fetal Medicine, Department of Obstetrics and Gynecology , University of California , Los Angeles, Los Angeles , California , USA.
[Ti] Título:Pseudoamniotic Band Syndrome Post Fetal Thoracoamniotic Shunting for Bilateral Hydrothorax.
[So] Source:Fetal Pediatr Pathol;36(4):311-318, 2017 Aug.
[Is] ISSN:1551-3823
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:INTRODUCTION: Pseudoamniotic band syndrome (PABS) occurs iatrogenically after fetal surgery or amniocentesis due to chorioamniotic membrane separation. Separation of the amnion from the chorion can expand to form fibrous amniotic bands that can envelope fetal limbs or the umbilical cord, with consequences ranging from limb constriction to fetal demise. CASE REPORT: We report a case of bilateral fetal pleural effusions at 27 weeks' gestation treated by bilateral thoracoamniotic shunts. Following shunt placement, the hydrothorax resolved. However, chorioamniotic membrane separation developed resulting in PABS with subsequent umbilical cord strangulation and fetal demise at 32 weeks' gestation. CONCLUSION: PABS has been previously described in the literature following various fetal interventions. This is the first reported case of pseudoamniotic band syndrome after placement of fetal thoracoamniotic shunts. A high index of suspicion is required to diagnose PABS via postoperative ultrasound. Post intervention chorioamniotic membrane separation warrants close surveillance for sonographic evidence of PABS.
[Mh] Termos MeSH primário: Síndrome de Bandas Amnióticas/etiologia
Quilotórax/congênito
Terapias Fetais/efeitos adversos
Hidropisia Fetal/cirurgia
[Mh] Termos MeSH secundário: Quilotórax/cirurgia
Feminino
Morte Fetal
Feto
Seres Humanos
Derrame Pleural/cirurgia
Gravidez
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180222
[Lr] Data última revisão:
180222
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170429
[St] Status:MEDLINE
[do] DOI:10.1080/15513815.2017.1313915


  3 / 2067 MEDLINE  
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[PMID]:28076990
[Au] Autor:Takita H; Hasegawa J; Arakaki T; Hamada S; Tokunaka M; Nakamura M; Matsuoka R; Sekizawa A
[Ad] Endereço:a Department of Obstetrics and Gynecology , Showa University School of Medicine , Tokyo , Japan.
[Ti] Título:Outcomes in the absence of the ductus venosus diagnosed in the first trimester.
[So] Source:J Matern Fetal Neonatal Med;31(2):253-257, 2018 Jan.
[Is] ISSN:1476-4954
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:PURPOSE: To clarify the outcomes of the absence of the ductus venosus (DV) diagnosed in fetuses suspected to have a structural abnormality during a morphological assessment in the first trimester. METHODS: Infants in whom ultrasound fetal morphological assessments were attempted in the first trimester (11 to 13-6 weeks of gestation) and who were subsequently delivered between 2013 and 2015 at Showa University Hospital were enrolled. In cases in which the absence of the DV was diagnosed in the first trimester, the prognosis was assessed. RESULTS: First-trimester ultrasound screening was performed in a total of 2610 cases between 2013 and 2015. Fetal edema (n = 38), hydrops (n = 16), abnormal four-chamber view findings (n = 2), and tricuspid regurgitation (n = 1) were observed in a total of 52 cases (2.0%). In 4 of the 52 cases with abnormal ultrasound findings, the absence of the DV was detected. CONCLUSION: If fetal edema or hydrops in early pregnancy is found without any other structural abnormalities, not only chromosomal abnormalities should be suspected but also an evaluation for the absence of the DV should be included. In addition, absence of the DV with fetal edema may be associated with the outcomes of cardiac dysfunction, chromosome abnormalities, and intrauterine sudden death. Severe fetal edema is associated with a poor prognosis, and the family must be carefully informed of the potential outcomes.
[Mh] Termos MeSH primário: Resultado da Gravidez/epidemiologia
Primeiro Trimestre da Gravidez
Veias Umbilicais
[Mh] Termos MeSH secundário: Estudos de Coortes
Feminino
Seres Humanos
Hidropisia Fetal/diagnóstico por imagem
Gravidez
Ultrassonografia Pré-Natal
Veias Umbilicais/anormalidades
Veias Umbilicais/diagnóstico por imagem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171128
[Lr] Data última revisão:
171128
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170113
[St] Status:MEDLINE
[do] DOI:10.1080/14767058.2017.1281241


  4 / 2067 MEDLINE  
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Texto completo SciELO Brasil
[PMID]:28767982
[Au] Autor:Noya BA; Pérez-Chacón G; Díaz-Bello Z; Dickson S; Muñoz-Calderón A; Hernández C; Pérez Y; Mauriello L; Moronta E
[Ad] Endereço:Universidad Central de Venezuela, Facultad de Medicina, Instituto de Medicina Tropical, Sección de Inmunología, Caracas, Venezuela.
[Ti] Título:Description of an oral Chagas disease outbreak in Venezuela, including a vertically transmitted case.
[So] Source:Mem Inst Oswaldo Cruz;112(8):569-571, 2017 Aug.
[Is] ISSN:1678-8060
[Cp] País de publicação:Brazil
[La] Idioma:eng
[Ab] Resumo:We describe the eleventh major outbreak of foodborne Trypanosoma cruzi transmission in urban Venezuela, including evidence for vertical transmission from the index case to her fetus. After confirming fetal death at 24 weeks of gestation, pregnancy interruption was performed. On direct examination of the amniotic fluid, trypomastigotes were detected. T. cruzi specific-polymerase chain reaction (PCR) also proved positive when examining autopsied fetal organs. Finally, microscopic fetal heart examination revealed amastigote nests. Acute orally transmitted Chagas disease can be life threatening or even fatal for pregnant women and unborn fetuses owing to vertical transmission. There is therefore an urgent need to improve national epidemiologic control measures.
[Mh] Termos MeSH primário: Doença de Chagas/transmissão
Morte Fetal/etiologia
Parasitologia de Alimentos
Doenças Transmitidas por Alimentos/parasitologia
Transmissão Vertical de Doença Infecciosa
[Mh] Termos MeSH secundário: Adolescente
Adulto
Doença de Chagas/complicações
Doença de Chagas/epidemiologia
Surtos de Doenças
Feminino
Seres Humanos
Hidropisia Fetal/parasitologia
Reação em Cadeia da Polimerase
Gravidez
População Urbana
Venezuela/epidemiologia
Adulto Jovem
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171004
[Lr] Data última revisão:
171004
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170803
[St] Status:MEDLINE


  5 / 2067 MEDLINE  
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[PMID]:28716860
[Au] Autor:Glogowska E; Schneider ER; Maksimova Y; Schulz VP; Lezon-Geyda K; Wu J; Radhakrishnan K; Keel SB; Mahoney D; Freidmann AM; Altura RA; Gracheva EO; Bagriantsev SN; Kalfa TA; Gallagher PG
[Ad] Endereço:Department of Pediatrics and.
[Ti] Título:Novel mechanisms of PIEZO1 dysfunction in hereditary xerocytosis.
[So] Source:Blood;130(16):1845-1856, 2017 Oct 19.
[Is] ISSN:1528-0020
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Mutations in PIEZO1 are the primary cause of hereditary xerocytosis, a clinically heterogeneous, dominantly inherited disorder of erythrocyte dehydration. We used next-generation sequencing-based techniques to identify mutations in individuals from 9 kindreds referred with suspected hereditary xerocytosis (HX) and/or undiagnosed congenital hemolytic anemia. Mutations were primarily found in the highly conserved, COOH-terminal pore-region domain. Several mutations were novel and demonstrated ethnic specificity. We characterized these mutations using genomic-, bioinformatic-, cell biology-, and physiology-based functional assays. For these studies, we created a novel, cell-based in vivo system for study of wild-type and variant PIEZO1 membrane protein expression, trafficking, and electrophysiology in a rigorous manner. Previous reports have indicated HX-associated PIEZO1 variants exhibit a partial gain-of-function phenotype with generation of mechanically activated currents that inactivate more slowly than wild type, indicating that increased cation permeability may lead to dehydration of PIEZO1-mutant HX erythrocytes. In addition to delayed channel inactivation, we found additional alterations in mutant PIEZO1 channel kinetics, differences in response to osmotic stress, and altered membrane protein trafficking, predicting variant alleles that worsen or ameliorate erythrocyte hydration. These results extend the genetic heterogeneity observed in HX and indicate that various pathophysiologic mechanisms contribute to the HX phenotype.
[Mh] Termos MeSH primário: Anemia Hemolítica Congênita/genética
Hidropisia Fetal/genética
Canais Iônicos/genética
[Mh] Termos MeSH secundário: Adulto
Anemia Hemolítica Congênita/metabolismo
Criança
Estudos de Coortes
Análise Mutacional de DNA
Desidratação/genética
Desidratação/metabolismo
Eritrócitos/metabolismo
Família
Feminino
Células HEK293
Seres Humanos
Hidropisia Fetal/metabolismo
Mutação INDEL
Recém-Nascido
Canais Iônicos/metabolismo
Cinética
Masculino
Mutação de Sentido Incorreto
Pressão Osmótica/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Ion Channels); 0 (PIEZO1 protein, human)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171104
[Lr] Data última revisão:
171104
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170719
[St] Status:MEDLINE
[do] DOI:10.1182/blood-2017-05-786004


  6 / 2067 MEDLINE  
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[PMID]:28580721
[Au] Autor:Li M; Blaustein JC
[Ad] Endereço:Blood Systems, Inc, Scottsdale, Arizona.
[Ti] Título:Persistent hemolytic disease of the fetus and newborn (HDFN) associated with passive acquisition of anti-D in maternal breast milk.
[So] Source:Transfusion;57(9):2121-2124, 2017 Sep.
[Is] ISSN:1537-2995
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Anti-D is a well-documented, significant cause of hemolytic disease of the fetus and newborn (HDFN), but its presence in breast milk is not routinely described. Theoretically, breast milk containing anti-D could have the potential to exacerbate HDFN if ingested by the affected infant. STUDY DESIGN AND METHODS: This is a case report of a 28-week premature male neonate with hydrops fetalis born to a 32-year-old woman (gravidity 3/parity 3) with anti-D and anti-G. The male neonate experienced prolonged HDFN due to passive acquisition of anti-D in the mother's breast milk. RESULTS: The mother's breast milk reacted strongly (4+) with the D-positive cells in the antibody screen test. Discontinuation of breast milk feeding and addition of total parenteral nutrition led to the cessation of clinically significant HDFN. CONCLUSION: Although anti-D is a significant cause of HDFN through placental transfer of antibody, exacerbation of the condition through breast milk antibodies is rarely described. The current case highlights the possibility of this occurring. Discontinuation of maternal breast milk feedings should be considered in infants with HDFN who do not respond to standard treatment.
[Mh] Termos MeSH primário: Eritroblastose Fetal/etiologia
Leite Humano/imunologia
Imunoglobulina rho(D)/farmacologia
[Mh] Termos MeSH secundário: Adulto
Eritroblastose Fetal/imunologia
Feminino
Seres Humanos
Hidropisia Fetal
Recém-Nascido
Masculino
Gravidez
Complicações Hematológicas na Gravidez
Imunoglobulina rho(D)/análise
[Pt] Tipo de publicação:CASE REPORTS
[Nm] Nome de substância:
0 (RHO(D) antibody); 0 (Rho(D) Immune Globulin)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171027
[Lr] Data última revisão:
171027
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170606
[St] Status:MEDLINE
[do] DOI:10.1111/trf.14171


  7 / 2067 MEDLINE  
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[PMID]:28533037
[Au] Autor:Steurer MA; Peyvandi S; Baer RJ; MacKenzie T; Li BC; Norton ME; Jelliffe-Pawlowski LL; Moon-Grady AJ
[Ad] Endereço:Department of Pediatrics, University of California, San Francisco, CA; Department of Epidemiology and Biostatistics, University of California, San Francisco, CA.
[Ti] Título:Epidemiology of Live Born Infants with Nonimmune Hydrops Fetalis-Insights from a Population-Based Dataset.
[So] Source:J Pediatr;187:182-188.e3, 2017 Aug.
[Is] ISSN:1097-6833
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: To evaluate the incidence, etiology, and 1-year mortality of nonimmune hydrops fetalis (NIHF) and to identify risk factors for mortality in a contemporary population-based dataset. STUDY DESIGN: The California Office of Statewide Health Planning and Development maintains a database linking maternal and infant hospital discharge, readmissions, and birth and death certificate date from 1 year before to 1 year after birth. We searched the database (2005-2012) for infants with NIHF (identified by the International Classification of Diseases, 9th Revision, Clinical Modification code). Hazard models were used to identify risk factors for mortality in infants with NIHF; results are presented as hazard ratios (HRs, 95% CI). RESULTS: The incidence of NIHF was 2.5 out of 10 000 among live born infants. Neonatal mortality was 35.1% (364 out of 1037) and overall mortality was 43.2% (448 out of 1037) at 1 year of age. Gestational age (GA) was predictive of mortality with a HR of 2.4 (95% CI 1.9-3.2) for preterm compared with term infants. The GA-adjusted HR for mortality was 1.3 (95% CI 1.1-1.6) for polyhydramnios and 1.5 (95% CI 1.2-2.0) for large for gestational age infants compared with appropriate for GA infants. Aneuploid infants with critical congenital heart disease had an adjusted HR of 2.3 (95% CI 1.5-3.6) compared with euploid infants without a structural birth defect. CONCLUSIONS: In this large, population-based study, prematurity, polyhydramnios, and large for gestational age were predictors of increased mortality. Mortality is highly variable among euploid and aneuploid infants with and without structural birth defects and critical congenital heart disease.
[Mh] Termos MeSH primário: Hidropisia Fetal/epidemiologia
Mortalidade Infantil
[Mh] Termos MeSH secundário: California
Bases de Dados Factuais
Feminino
Idade Gestacional
Seres Humanos
Hidropisia Fetal/mortalidade
Incidência
Lactente
Recém-Nascido
Masculino
Gravidez
Fatores de Risco
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170907
[Lr] Data última revisão:
170907
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170524
[St] Status:MEDLINE


  8 / 2067 MEDLINE  
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[PMID]:28497611
[Au] Autor:Singha K; Srivorakun H; Fucharoen G; Fucharoen S
[Ad] Endereço:Centre for Research and Development of Medical Diagnostic Laboratories, Faculty of Associated Medical Sciences, Khon Kaen University, Khon Kaen, Thailand.
[Ti] Título:Co-inheritance of α -thalassemia elevates Hb A level in homozygous Hb E: Diagnostic implications.
[So] Source:Int J Lab Hematol;39(5):508-512, 2017 Oct.
[Is] ISSN:1751-553X
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:INTRODUCTION: Differentiation of homozygous hemoglobin (Hb) E with and without α -thalassemia is subtle on routine hematological ground. We examined in a large cohort of homozygous Hb E if the level of Hb A is helpful. METHODS: A total of 592 subjects with homozygous Hb E were recruited from ongoing thalassemia screening program. Additionally, five couples at risk of having fetuses with Hb Bart's hydrops fetalis who were homozygous Hb E were also investigated. Hb analysis was performed using capillary electrophoresis system. Globin genotypes were defined by DNA analysis. RESULTS: Subjects were classified into four groups including pure homozygous Hb E (n=532), homozygous Hb E/α -thalassemia (n=48), Hb Constant Spring EE Bart's disease (n=8), and Hb EE Bart's disease (n=4). The levels of Hb A were found, respectively, to be 4.97±0.69, 6.64±1.02, 4.86±0.87, and 7.60±1.04%. Among five couples at risk, α -thalassemia was identified in three subjects with Hb A >6.0%. CONCLUSIONS: Increased Hb A level is a useful marker for differentiation of homozygous Hb E with and without α -thalassemia. This should lead to a significant reduction in number of referral cases of homozygous Hb E for molecular testing of α -thalassemia in routine practice.
[Mh] Termos MeSH primário: Hemoglobina A2/genética
Hemoglobina E/genética
Homozigoto
Padrões de Herança
Talassemia alfa/diagnóstico
Talassemia alfa/genética
[Mh] Termos MeSH secundário: Adulto
Biomarcadores
Eletroforese Capilar
Índices de Eritrócitos
Feminino
Genótipo
Seres Humanos
Hidropisia Fetal/diagnóstico
Hidropisia Fetal/genética
Masculino
Mutação
Talassemia alfa/sangue
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Biomarkers); 9034-53-1 (Hemoglobin A2); 9034-61-1 (Hemoglobin E)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171019
[Lr] Data última revisão:
171019
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170513
[St] Status:MEDLINE
[do] DOI:10.1111/ijlh.12677


  9 / 2067 MEDLINE  
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Afonso, Ana Maria Sardinha
Curti, Suely Pires
Texto completo SciELO Brasil
[PMID]:28489127
[Au] Autor:Oliveira MI; Afonso AMS; Curti SP; Silva PE; Barbosa TF; Silva ER; Figueiredo CA
[Ad] Endereço:PhD, Scientific Researcher, Núcleo de Doenças Respiratórias, Instituto Adolfo Lutz, São Paulo, SP, Brazil.
[Ti] Título:Genotype 1 of human parvovirus B19 in clinical cases.
[So] Source:Rev Assoc Med Bras (1992);63(3):224-228, 2017 Mar.
[Is] ISSN:1806-9282
[Cp] País de publicação:Brazil
[La] Idioma:eng
[Ab] Resumo:Introduction:: Virus surveillance strategies and genetic characterization of human parvovirus B19 (B19V) are important tools for regional and global control of viral outbreak. In São Paulo, Brazil, we performed a study of B19V by monitoring the spread of this virus, which is an infectious agent and could be mistakenly reported as a rash and other types of infection. Method:: Serum samples were subjected to enzyme immunoassay, real time polymerase chain reaction, and sequencing. Results:: From the 462 patients with suspected cases of exanthematic infections, the results of the 164 serum samples were positive for B19V immunoglobulin M. Among these cases, there were 38 patients with erythema infections and B19-associated with other infections such as encephalitis, hydrops fetalis, chronic anemia, hematological malignancies. These samples were sequenced and identified as genotype 1. Conclusion:: This study showed patients with infections caused by B19V and sequencing genotype 1. Continuous monitoring is necessary to detect all known genotypes, and the emergence of new genotypes of these viruses for case management in public health control activities.
[Mh] Termos MeSH primário: Eritema Infeccioso/virologia
Genótipo
Parvovirus B19 Humano/genética
Parvovirus B19 Humano/isolamento & purificação
[Mh] Termos MeSH secundário: Adolescente
Adulto
Anemia/virologia
Anticorpos Antivirais/sangue
Brasil
Criança
Pré-Escolar
DNA Viral/sangue
Eritema Infeccioso/sangue
Feminino
Seres Humanos
Hidropisia Fetal/virologia
Imunoensaio
Imunoglobulina G/sangue
Imunoglobulina M/sangue
Lactente
Masculino
Meia-Idade
Vigilância da População
Reação em Cadeia da Polimerase Via Transcriptase Reversa
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Antibodies, Viral); 0 (DNA, Viral); 0 (Immunoglobulin G); 0 (Immunoglobulin M)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170829
[Lr] Data última revisão:
170829
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170511
[St] Status:MEDLINE


  10 / 2067 MEDLINE  
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[PMID]:28182812
[Au] Autor:Thong XY; Lee LY; Chia DA; Wong YC; Biswas A
[Ad] Endereço:Department of Paediatrics, Yong Loo Lin School of Medicine, National University Singapore, Singapore.
[Ti] Título:Management and Outcomes of Fetal Hydrops in a Tertiary Care Centre in Singapore.
[So] Source:Ann Acad Med Singapore;46(1):4-10, 2017 Jan.
[Is] ISSN:0304-4602
[Cp] País de publicação:Singapore
[La] Idioma:eng
[Ab] Resumo:: Fetal hydrops is a serious condition which can be caused by immune and non-immune aetiologies. We aimed to review the management of fetal hydrops at our hospital. : A retrospective review of all cases of fetal hydrops diagnosed in our institution from 2006 to 2013 was carried out. : Out of the 30 cases of fetal hydrops diagnosed antenatally, 17 were cases of Bart's hydrops which were all terminated in-utero. Of the remaining 13 cases, 11 cases consisted of non-immune causes of hydrops. Planned antenatal interventions including in-utero blood transfusions (n = 4) and thoracentesis (n = 5) as well as planned caesarean deliveries (n = 11) were performed in the majority of cases. Postnatal neonatal intensive care with interventions including chest drainage and transfusions were also performed. A majority, 92%, of the cases survived the perinatal period following a variable length of hospital stay ranging from a week to 3 months. : Management of fetal hydrops is complex. Close coordination between the obstetric and neonatal teams was the key to good short-term survival of neonates with antenatally diagnosed hydrops, as it allows timely antenatal intervention and anticipation of potential perinatal complications.
[Mh] Termos MeSH primário: Transfusão de Sangue
Cesárea
Terapias Fetais
Hidropisia Fetal/terapia
Toracentese
[Mh] Termos MeSH secundário: Aborto Induzido
Gerenciamento Clínico
Drenagem
Feminino
Hemoglobinas Anormais
Seres Humanos
Hidropisia Fetal/sangue
Hidropisia Fetal/etiologia
Recém-Nascido
Unidades de Terapia Intensiva Neonatal
Gravidez
Diagnóstico Pré-Natal
Estudos Retrospectivos
Singapura
Taxa de Sobrevida
Centros de Atenção Terciária
Talassemia alfa/sangue
Talassemia alfa/complicações
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Hemoglobins, Abnormal); 9056-09-1 (hemoglobin Bart's)
[Em] Mês de entrada:1702
[Cu] Atualização por classe:170221
[Lr] Data última revisão:
170221
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170210
[St] Status:MEDLINE



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