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[PMID]:29366747
[Au] Autor:Camargo-Silva G; Turones LC; da Cruz KR; Gomes KP; Mendonça MM; Nunes A; de Jesus IG; Colugnati DB; Pansani AP; Pobbe RLH; Santos R; Fontes MAP; Guatimosim S; de Castro CH; Ianzer D; Ferreira RN; Xavier CH
[Ad] Endereço:Laboratory of Cardiovascular Physiology and Therapeutics, Department of Physiological Sciences, Institute of Biological Sciences, Federal University of Goiás, Goiania, GO, Brazil.
[Ti] Título:Ghrelin potentiates cardiac reactivity to stress by modulating sympathetic control and beta-adrenergic response.
[So] Source:Life Sci;196:84-92, 2018 Mar 01.
[Is] ISSN:1879-0631
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Prior evidence indicates that ghrelin is involved in the integration of cardiovascular functions and behavioral responses. Ghrelin actions are mediated by the growth hormone secretagogue receptor subtype 1a (GHS-R1a), which is expressed in peripheral tissues and central areas involved in the control of cardiovascular responses to stress. AIMS: In the present study, we assessed the role of ghrelin - GHS-R1a axis in the cardiovascular reactivity to acute emotional stress in rats. MAIN METHODS AND KEY FINDINGS: Ghrelin potentiated the tachycardia evoked by restraint and air jet stresses, which was reverted by GHS-R1a blockade. Evaluation of the autonomic balance revealed that the sympathetic branch modulates the ghrelin-evoked positive chronotropy. In isolated hearts, the perfusion with ghrelin potentiated the contractile responses caused by stimulation of the beta-adrenergic receptor, without altering the amplitude of the responses evoked by acetylcholine. Experiments in isolated cardiomyocytes revealed that ghrelin amplified the increases in calcium transient changes evoked by isoproterenol. SIGNIFICANCE: Taken together, our results indicate that the Ghrelin-GHS-R1a axis potentiates the magnitude of stress-evoked tachycardia by modulating the autonomic nervous system and peripheral mechanisms, strongly relying on the activation of cardiac calcium transient and beta-adrenergic receptors.
[Mh] Termos MeSH primário: Grelina/farmacologia
Coração/efeitos dos fármacos
Receptores Adrenérgicos beta/efeitos dos fármacos
Estresse Psicológico/fisiopatologia
Sistema Nervoso Simpático/efeitos dos fármacos
[Mh] Termos MeSH secundário: Agonistas Adrenérgicos beta/farmacologia
Animais
Pressão Arterial/efeitos dos fármacos
Canais de Cálcio/efeitos dos fármacos
Coração/inervação
Frequência Cardíaca/efeitos dos fármacos
Técnicas In Vitro
Masculino
Agonistas Muscarínicos/farmacologia
Ratos
Ratos Wistar
Receptores de Grelina/efeitos dos fármacos
Restrição Física
Taquicardia/induzido quimicamente
Taquicardia/fisiopatologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Adrenergic beta-Agonists); 0 (Calcium Channels); 0 (Ghrelin); 0 (Muscarinic Agonists); 0 (Receptors, Adrenergic, beta); 0 (Receptors, Ghrelin)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180226
[Lr] Data última revisão:
180226
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180126
[St] Status:MEDLINE


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[PMID]:29293295
[Au] Autor:Gentges J; Schieche C; Williams E; Burns B
[Ti] Título:Case Report: A 24-Year-Old Wide Complex Tachycardia: First Do No Harm.
[So] Source:J Okla State Med Assoc;110(2):72-6, 2017 02.
[Is] ISSN:0030-1876
[Cp] País de publicação:United States
[La] Idioma:eng
[Mh] Termos MeSH primário: Manipulações Musculoesqueléticas
Taquicardia/terapia
[Mh] Termos MeSH secundário: Eletrocardiografia
Seres Humanos
Masculino
Taquicardia/fisiopatologia
Adulto Jovem
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180220
[Lr] Data última revisão:
180220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180103
[St] Status:MEDLINE


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[PMID]:28455405
[Au] Autor:Li Kam Wa ME; Taraborrelli P; Hayat S; Lim PB
[Ad] Endereço:Department of Cardiology, Imperial College Healthcare NHS Trust, London, UK.
[Ti] Título:Respiration driven excessive sinus tachycardia treated with clonidine.
[So] Source:BMJ Case Rep;2017, 2017 Apr 28.
[Is] ISSN:1757-790X
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:A 26-year-old man presented to our syncope service with debilitating daily palpitations, shortness of breath, presyncope and syncope following a severe viral respiratory illness 4 years previously. Mobitz type II block had previously been identified, leading to a permanent pacemaker and no further episodes of frank syncope. Transthoracic echocardiography, electophysiological study and repeated urine metanepherines were normal. His palpitations and presyncope were reproducible on deep inspiration, coughing, isometric hand exercise and passive leg raises. We demonstrated rapid increases in heart rate with no change in morphology on his 12 lead ECG. His symptoms were resistant to fludrocortisone, flecainide, ß blockers and ivabradine. Initiation of clonidine in combination with ivabradine led to rapid resolution of his symptoms. We suggest that an excessive respiratory sinus arrhythmia was responsible for his symptoms and achieved an excellent response with the centrally acting sympatholytic clonidine, where previous peripherally acting treatments had failed.
[Mh] Termos MeSH primário: Inalação/fisiologia
Síncope/fisiopatologia
Taquicardia Sinusal/complicações
[Mh] Termos MeSH secundário: Agonistas de Receptores Adrenérgicos alfa 2/administração & dosagem
Agonistas de Receptores Adrenérgicos alfa 2/uso terapêutico
Adulto
Benzazepinas/administração & dosagem
Benzazepinas/uso terapêutico
Fármacos Cardiovasculares/administração & dosagem
Fármacos Cardiovasculares/uso terapêutico
Clonidina/administração & dosagem
Clonidina/uso terapêutico
Tosse/complicações
Tosse/etiologia
Quimioterapia Combinada/métodos
Dispneia/diagnóstico
Dispneia/etiologia
Ecocardiografia/métodos
Eletrocardiografia/métodos
Seres Humanos
Masculino
Síncope/etiologia
Taquicardia/etiologia
Taquicardia/fisiopatologia
Taquicardia Sinusal/diagnóstico por imagem
Taquicardia Sinusal/tratamento farmacológico
Taquicardia Sinusal/fisiopatologia
Resultado do Tratamento
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Adrenergic alpha-2 Receptor Agonists); 0 (Benzazepines); 0 (Cardiovascular Agents); 3H48L0LPZQ (ivabradine); MN3L5RMN02 (Clonidine)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180220
[Lr] Data última revisão:
180220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170430
[St] Status:MEDLINE


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[PMID]:29309108
[Au] Autor:Jovic-Stosic J; Putic V; Perkovic-Vukcevic N; Babic G; Dordevic S; Segrt Z
[Ti] Título:Intravenous lipid emulsion in treatment of cardiocirculatory disturbances caused by glyphosate-surfactant herbicide poisoning.
[So] Source:Vojnosanit Pregl;73(4):390-2, 2016 Apr.
[Is] ISSN:0042-8450
[Cp] País de publicação:Serbia
[La] Idioma:eng
[Ab] Resumo:Introduction: Glyphosate is the first widely used herbicide against weed in genetically modified crops. Though glyphosate itself has a low toxicity, commercial products are more dangerous because of increased toxicity due to surfactants addition. There is no specific antidote for the poisoning with glyphosate-surfactant (Gly-SH). In recent times, the efficacy of intravenous lipid emulsion (ILE) administration for the treatment of acute poisoning caused by Gly- SH has been investigated. Case Report: A 50-year-old man was admitted 3 hours after self-poisoning with herbicide containing glyphosate and polyoxyethyleneamine, as a surfactant. On admission, the patient was in a coma, hypotensive (80/50 mmHg) and without spontaneous breathing. Electrocardiogram showed widecomplex tachycardia, and arterial blood gas (ABG) revealed acidosis (pH 7.07). Conventional treatment included mechanical ventilation, intravenous fluids, bicarbonate and dopamine. As there was no improvement, ILE was started. The patient received 100 mL of 20% Intralipid® bolus followed by infusion of 400 mL over 20 minutes. Prior to expiration of infusion, a gradual rise in blood pressure was noted, and within 2 hours sinus rhythm was restored. Conclusion: This case report suggests that the use of ILE may be an additional option for the treatment of cardiocirculatory disturbances caused by commercial products of glyphosate herbicide.
[Mh] Termos MeSH primário: Emulsões Gordurosas Intravenosas/uso terapêutico
Glicina/análogos & derivados
Herbicidas/envenenamento
Polietilenoglicóis/envenenamento
Taquicardia/induzido quimicamente
Taquicardia/tratamento farmacológico
[Mh] Termos MeSH secundário: Acidose/induzido quimicamente
Acidose/tratamento farmacológico
Eletrocardiografia
Glicina/envenenamento
Seres Humanos
Masculino
Meia-Idade
Tentativa de Suicídio
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Fat Emulsions, Intravenous); 0 (Herbicides); 0 (polyoxyethyleneamine); 30IQX730WE (Polyethylene Glycols); 4632WW1X5A (glyphosate); TE7660XO1C (Glycine)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180213
[Lr] Data última revisão:
180213
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180109
[St] Status:MEDLINE
[do] DOI:10.2298/VSP141017020J


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[PMID]:28947249
[Au] Autor:Piccini JP; Sanders P; Shah R; Roberts G; Karst E; Turakhia MP
[Ad] Endereço:Duke University Medical Center, Durham, North Carolina. Electronic address: jonathan.piccini@duke.edu.
[Ti] Título:Impact of Tachyarrhythmia Detection Rate and Time from Detection to Shock on Outcomes in Nationwide US Practice.
[So] Source:Am J Cardiol;120(8):1325-1331, 2017 Oct 15.
[Is] ISSN:1879-1913
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Although higher detection rates and delayed detection improve survival in implantable cardioverter defibrillator clinical trials, their effectiveness in clinical practice has limited validation. To evaluate the effectiveness of programming strategies for reducing shocks and mortality, we conducted a nationwide assessment of patients with implantable cardioverter defibrillators or cardiac resynchronization therapy defibrillators with linked remote monitoring data. We categorized patients based on the presence or absence of high rate detection and delayed detection: higher rate delayed detection (HRDD), higher rate early detection (HRED), lower rate delayed detection (LRDD), and lower rate early detection (LRED). Cox regression was used to compare mortality and shock-free survival. There were 64,769 patients (age 68 ± 12 years; 27% female; 46% cardiac resynchronization therapy defibrillator; follow-up 1.7 ± 1.1 years). In the first year, 13% of HRDD, 14% of HRED, 18% of LRDD, and 20% in the LRED group experienced a shock. After adjustment, HRDD was associated with lower risk of shock than HRED (hazard ratio [HR] 0.93, 95% confidence interval [CI] 0.89 to 0.98, p = 0.002), LRDD (HR 0.63, 95% CI 0.60 to 0.66, p <0.001), and LRED (HR 0.58, 95% CI 0.55 to 0.61, p <0.001). HRDD was also associated with lower risk of mortality than HRED (adjusted HR 0.80, 95% CI 0.75 to 0.86, p <0.001), LRDD (HR 0.76, 95% CI 0.70 to 0.83, p <0.001), and LRED (HR 0.68, 95% CI 0.62 to 0.73, p <0.001). Similar results were observed in patients with or without a shock in the first 6 months after implant. In conclusion, high rate programming is associated with lower risk of shocks or death compared with delayed detection. Optimal outcomes are observed in patients programmed with both high rate and delayed detection.
[Mh] Termos MeSH primário: Taquicardia/diagnóstico
[Mh] Termos MeSH secundário: Adolescente
Adulto
Idoso
Idoso de 80 Anos ou mais
Cardioversão Elétrica/métodos
Seguimentos
Seres Humanos
Incidência
Meia-Idade
Estudos Retrospectivos
Fatores de Risco
Taxa de Sobrevida/tendências
Taquicardia/epidemiologia
Taquicardia/terapia
Fatores de Tempo
Resultado do Tratamento
Estados Unidos/epidemiologia
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE; MULTICENTER STUDY
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171003
[Lr] Data última revisão:
171003
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170927
[St] Status:MEDLINE


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[PMID]:28904685
[Au] Autor:Affangla DA; Leye M; Simo AW; D'Almeida F; Sarr TY; Phiri A; Kane A
[Ad] Endereço:Hôpital Saint Jean de Dieu, Thiès, Sénégal.
[Ti] Título:[Poorly tolerated broad QRS complex tachycardia in a newborn].
[Ti] Título:Une tachycardie à QRS large mal tolérée chez un nourrisson..
[So] Source:Pan Afr Med J;27:157, 2017.
[Is] ISSN:1937-8688
[Cp] País de publicação:Uganda
[La] Idioma:fre
[Ab] Resumo:Poorly tolerated broad QRS complex tachycardia in a newborn poses problems with its diagnosis and emergency management. We report the case of a 35-day-old newborn with broad QRS complex tachycardia admitted because of cardiocirculatory distress. Doppler echocardiography showed morphologically normal heart. The patient received a loading dose of amiodarone but it didn't attenuate tachycardia. Normal sinus rhythm was restored after cardioversion through Lifeline semi-automatic external defibrillator. Maintenance therapy was based on oral amiodarone. The patient had normal sinus rhythm at 03 months of follow-up.
[Mh] Termos MeSH primário: Amiodarona/uso terapêutico
Antiarrítmicos/uso terapêutico
Cardioversão Elétrica/métodos
Taquicardia/diagnóstico
[Mh] Termos MeSH secundário: Terapia Combinada
Ecocardiografia Doppler
Eletrocardiografia
Feminino
Seguimentos
Seres Humanos
Recém-Nascido
Taquicardia/terapia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Anti-Arrhythmia Agents); N3RQ532IUT (Amiodarone)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170926
[Lr] Data última revisão:
170926
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170915
[St] Status:MEDLINE
[do] DOI:10.11604/pamj.2017.27.157.10364


  7 / 17609 MEDLINE  
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[PMID]:28873427
[Au] Autor:Kharche SR; Vigmond E; Efimov IR; Dobrzynski H
[Ad] Endereço:Institute of Cardiovascular Sciences, School of Medical Sciences, University of Manchester, Manchester, United Kingdom.
[Ti] Título:Computational assessment of the functional role of sinoatrial node exit pathways in the human heart.
[So] Source:PLoS One;12(9):e0183727, 2017.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:AIM: The human right atrium and sinoatrial node (SAN) anatomy is complex. Optical mapping experiments suggest that the SAN is functionally insulated from atrial tissue except at discrete SAN-atrial electrical junctions called SAN exit pathways, SEPs. Additionally, histological imaging suggests the presence of a secondary pacemaker close to the SAN. We hypothesise that a) an insulating border-SEP anatomical configuration is related to SAN arrhythmia; and b) a secondary pacemaker, the paranodal area, is an alternate pacemaker but accentuates tachycardia. A 3D electro-anatomical computational model was used to test these hypotheses. METHODS: A detailed 3D human SAN electro-anatomical mathematical model was developed based on our previous anatomical reconstruction. Electrical activity was simulated using tissue specific variants of the Fenton-Karma action potential equations. Simulation experiments were designed to deploy this complex electro-anatomical system to assess the roles of border-SEPs and paranodal area by mimicking experimentally observed SAN arrhythmia. Robust and accurate numerical algorithms were implemented for solving the mono domain reaction-diffusion equation implicitly, calculating 3D filament traces, and computing dominant frequency among other quantitative measurements. RESULTS: A centre to periphery gradient of increasing diffusion was sufficient to permit initiation of pacemaking at the centre of the 3D SAN. Re-entry within the SAN, micro re-entry, was possible by imposing significant SAN fibrosis in the presence of the insulating border. SEPs promoted the micro re-entry to generate more complex SAN-atrial tachycardia. Simulation of macro re-entry, i.e. re-entry around the SAN, was possible by inclusion of atrial fibrosis in the presence of the insulating border. The border shielded the SAN from atrial tachycardia. However, SAN micro-structure intercellular gap junctional coupling and the paranodal area contributed to prolonged atrial fibrillation. Finally, the micro-structure was found to be sufficient to explain shifts of leading pacemaker site location. CONCLUSIONS: The simulations establish a relationship between anatomy and SAN electrical function. Microstructure, in the form of intercellular gap junction coupling, was found to regulate SAN function and arrhythmia.
[Mh] Termos MeSH primário: Sistema de Condução Cardíaco/fisiologia
Coração/fisiologia
Nó Sinoatrial/fisiologia
[Mh] Termos MeSH secundário: Potenciais de Ação/fisiologia
Anisotropia
Fibrilação Atrial/fisiopatologia
Simulação por Computador
Difusão
Eletrofisiologia
Fibrose
Junções Comunicantes
Átrios do Coração/anatomia & histologia
Sistema de Condução Cardíaco/anatomia & histologia
Seres Humanos
Imagem Tridimensional
Modelos Cardiovasculares
Modelos Teóricos
Nó Sinoatrial/anatomia & histologia
Taquicardia/fisiopatologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171025
[Lr] Data última revisão:
171025
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170906
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0183727


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[PMID]:28838549
[Au] Autor:Di Biase L; Gianni C; Bagliani G; Padeletti L
[Ad] Endereço:Texas Cardiac Arrhythmia Institute, St. David's Medical Center, 3000 N IH-35, Austin, TX, 78705, USA; Department of Biomedical Engineering, University of Texas, 107 W Dean Keeton Street, Austin, TX, 78712, USA; Arrhythmia Services, Montefiore Medical Center, Albert Einstein College of Medicine, 111
[Ti] Título:Arrhythmias Involving the Atrioventricular Junction.
[So] Source:Card Electrophysiol Clin;9(3):435-452, 2017 Sep.
[Is] ISSN:1877-9190
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The atrioventricular junction has a central role in electrophysiology, responsible for reentrant and automatic forms of supraventricular tachycardia. During atrioventricular nodal reentry tachycardia, the circuit involves 2 electrophysiologically separate pathways located in the vicinity of the atrioventricular node. Atrioventricular reentry tachycardia is caused by the presence of an accessory pathway located almost anywhere along the atrioventricular groove; the macroreentrant circuit involves the atrioventricular node, the accessory pathway and necessarily portions of atria and ventricles. Junctional tachycardia is a rare form of nonparoxysmal supraventricular tachycardia, secondary to enhanced automaticity or triggered activity. By analyzing a 12-lead electrocardiogram during sinus rhythm and tachycardia, it is possible to accurately diagnose the specific type of supraventricular tachycardia.
[Mh] Termos MeSH primário: Feixe Acessório Atrioventricular/fisiopatologia
Arritmias Cardíacas/fisiopatologia
Nó Atrioventricular/fisiopatologia
Taquicardia/fisiopatologia
[Mh] Termos MeSH secundário: Eletrocardiografia
Seres Humanos
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170920
[Lr] Data última revisão:
170920
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170826
[St] Status:MEDLINE


  9 / 17609 MEDLINE  
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[PMID]:28838547
[Au] Autor:Leonelli F; Bagliani G; Boriani G; Padeletti L
[Ad] Endereço:Cardiology Department James A. Haley Veterans' Hospital, University South Florida, 13000 Bruce B Down Boulevard, Tampa 33612, FL, USA.
[Ti] Título:Arrhythmias Originating in the Atria.
[So] Source:Card Electrophysiol Clin;9(3):383-409, 2017 Sep.
[Is] ISSN:1877-9190
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Atrial flutter, atrial tachycardias, and atrial fibrillation are the main sustained atrial tachycardias. Reentry, increased automaticity, and triggered activity are atrial arrhythmia's main mechanisms. Atrial flutter is the clinical and theoretical model of reentry. Its classification is based on the atrial chamber involved and the arrhythmia's anatomic path. Ablative procedures for atrial fibrillation have created several new reentrant tachycardias. Electrocardiography (ECG) identifies the site of origin of focal atrial tachycardias and the mechanism of these arrhythmias. ECG is fundamental in the diagnosis of atrial fibrillation and often allows understanding of its mechanism of origin and maintenance.
[Mh] Termos MeSH primário: Fibrilação Atrial/fisiopatologia
Flutter Atrial/fisiopatologia
Átrios do Coração/fisiopatologia
Taquicardia/fisiopatologia
[Mh] Termos MeSH secundário: Eletrocardiografia
Seres Humanos
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170920
[Lr] Data última revisão:
170920
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170826
[St] Status:MEDLINE


  10 / 17609 MEDLINE  
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[PMID]:28763820
[Au] Autor:Pätz T; Wolfrum S
[Ad] Endereço:Medizinische Klinik II / Kardiologie, Angiologie, Intensivmedizin, Universitäres Herzzentrum Lübeck.
[Ti] Título:[Tachycardic Arrhythmias in the Emergency Room - State of the Art].
[Ti] Título:Tachykarde Herzrhythmusstörungen in der Notaufnahme ­ Aktuelle Entwicklungen..
[So] Source:Dtsch Med Wochenschr;142(15):1118-1122, 2017 Aug.
[Is] ISSN:1439-4413
[Cp] País de publicação:Germany
[La] Idioma:ger
[Mh] Termos MeSH primário: Serviço Hospitalar de Emergência
Taquicardia
[Mh] Termos MeSH secundário: Seres Humanos
Taquicardia/diagnóstico
Taquicardia/terapia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170914
[Lr] Data última revisão:
170914
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170802
[St] Status:MEDLINE
[do] DOI:10.1055/s-0043-110490



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