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Pesquisa : C17.800.849 [Categoria DeCS]
Referências encontradas : 75 [refinar]
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[PMID]:28225950
[Au] Autor:Mendes AL; Miot HA; Haddad V
[Ad] Endereço:Department of Clinical Medicine - Faculdade de Medicina de Botucatu - Universidade Estadual Paulista "Júlio de Mesquita Filho" (UNESP) - Botucatu (SP), Brazil.
[Ti] Título:Diabetes mellitus and the skin.
[So] Source:An Bras Dermatol;92(1):8-20, 2017 Jan-Feb.
[Is] ISSN:1806-4841
[Cp] País de publicação:Brazil
[La] Idioma:eng
[Ab] Resumo:Several dermatoses are routinely associated with diabetes mellitus, especially in patients with chronic disease. This relationship can be easily proven in some skin disorders, but it is not so clear in others. Dermatoses such necrobiosis lipoidica, granuloma annulare, acanthosis nigricans and others are discussed in this text, with an emphasis on proven link with the diabetes or not, disease identification and treatment strategy used to control those dermatoses and diabetes.
[Mh] Termos MeSH primário: Complicações do Diabetes/complicações
Diabetes Mellitus
Dermatopatias/etiologia
[Mh] Termos MeSH secundário: Acantose Nigricans/etiologia
Acantose Nigricans/patologia
Pé Diabético/patologia
Granuloma Anular/etiologia
Granuloma Anular/patologia
Seres Humanos
Necrobiose Lipoídica/etiologia
Necrobiose Lipoídica/patologia
Psoríase/etiologia
Psoríase/patologia
Dermatopatias/classificação
Dermatopatias/patologia
Dermatopatias Metabólicas
Dermatopatias Vesiculobolhosas/etiologia
Dermatopatias Vesiculobolhosas/patologia
Vitiligo/etiologia
Vitiligo/patologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171004
[Lr] Data última revisão:
171004
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170223
[St] Status:MEDLINE


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[PMID]:27592007
[Au] Autor:Hidaka M; Horikawa K; Akase T; Makihara H; Ogami T; Tomozawa H; Tsubata M; Ibuki A; Matsumoto Y
[Ad] Endereço:Department of Biological Science and Nursing, School of Medicine, Yokohama City University, 3-9 Fukuura, Kanazawa-ku, Yokohama, Kanagawa, 236-0004, Japan.
[Ti] Título:Efficacy of Kaempferia parviflora in a mouse model of obesity-induced dermatopathy.
[So] Source:J Nat Med;71(1):59-67, 2017 Jan.
[Is] ISSN:1861-0293
[Cp] País de publicação:Japan
[La] Idioma:eng
[Ab] Resumo:Obesity results from excessive energy intake and physical inactivity, and predisposes one to various diseases. One of these reasons is that enlargement of adipocytes raises the lipid metabolic abnormalities that affect various organs. The skin is one such organ, and it has been reported that subcutaneous adipocyte cells secrete various factors and these factors are involved in reduction of dermal collagen fibers and fragility of the skin in obesity. The present study explored the efficacy of Kaempferia parviflora (KP) in preventing obesity-induced dermatopathy. We used Tsumura Suzuki obese diabetes (TSOD) mice as an obesity model. TSOD mice were fed a standard diet (MF) mixed with either an ethanol extract from KP (KPE), polymethoxyflavonoid-rich extract from KP (PMF), or polymethoxyflavonoid-poor extract from KP (X). We then evaluated the effect of these three KP fractions on aging-like skin damage induced by UVB irradiation. KPE and PMF caused a significant decrease of mouse body weight, and suppressed the increase in the thickness of the subcutaneous fat layer. In addition, KPE shifted the frequency of subcutaneous adipocyte sizes towards smaller cells possibly via its polypharmacological actions. Scanning electron microscopy revealed that the stereostructure of the collagenous fibers in the dermis was better retained in the KPE and PMF groups, in that order. These results offer the first evidence that KPE can attenuate obesity-induced dermatopathy more effectively than PMF, suggesting that KPE (or KP) might be a candidate supplement for preventing obesity-related skin disorders.
[Mh] Termos MeSH primário: Obesidade/complicações
Extratos Vegetais/farmacologia
Reação em Cadeia da Polimerase em Tempo Real/métodos
Dermatopatias Metabólicas/tratamento farmacológico
Zingiberaceae/química
[Mh] Termos MeSH secundário: Animais
Modelos Animais de Doenças
Masculino
Camundongos
Camundongos Obesos
Dermatopatias Metabólicas/etiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Plant Extracts)
[Em] Mês de entrada:1703
[Cu] Atualização por classe:171104
[Lr] Data última revisão:
171104
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160905
[St] Status:MEDLINE
[do] DOI:10.1007/s11418-016-1027-8


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[PMID]:27221411
[Au] Autor:Stiefelhagen P
[Ad] Endereço:.
[Ti] Título:[The skin is a mirror of glucose metabolism].
[Ti] Título:Die Haut als Spiegel des Zuckerstoffwechsels..
[So] Source:MMW Fortschr Med;158(10):22, 2016 May 25.
[Is] ISSN:1438-3276
[Cp] País de publicação:Germany
[La] Idioma:ger
[Mh] Termos MeSH primário: Complicações do Diabetes/diagnóstico
Dermatopatias Metabólicas/diagnóstico
[Mh] Termos MeSH secundário: Complicações do Diabetes/tratamento farmacológico
Diagnóstico Diferencial
Seres Humanos
Sistemas de Infusão de Insulina/efeitos adversos
[Pt] Tipo de publicação:NEWS
[Em] Mês de entrada:1608
[Cu] Atualização por classe:160525
[Lr] Data última revisão:
160525
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160526
[St] Status:MEDLINE
[do] DOI:10.1007/s15006-016-8252-7


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[PMID]:26475684
[Au] Autor:Saardi KM; Schwartz RA
[Ad] Endereço:Dermatology, Rutgers University New Jersey Medical School, Newark, NJ, USA.
[Ti] Título:Uremic frost: a harbinger of impending renal failure.
[So] Source:Int J Dermatol;55(1):17-20, 2016 Jan.
[Is] ISSN:1365-4632
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Uremic frost is a striking cutaneous finding seen in patients with severe kidney disease. Familiarity with this condition can be a life-saving signal to initiate urgent dialysis. Uremic frost generally occurs at blood urea nitrogen levels of approximately 200 mg/dl, although it may arise with less severe uremia. Recently confirmed urea transporters in the skin may play a role in the development of uremic frost. Alternatively, damage to the cutaneous microvasculature and pilosebaceous units, as seen in chronic kidney disease, could account for the high levels of urea deposited outside the skin. The treatment of uremic frost is largely aimed at correcting the underlying cause of uremia and the other life-threatening conditions associated with renal failure.
[Mh] Termos MeSH primário: Nitrogênio da Ureia Sanguínea
Falência Renal Crônica/fisiopatologia
Dermatopatias Metabólicas/etiologia
Uremia/complicações
[Mh] Termos MeSH secundário: Adulto
Idoso
Progressão da Doença
Feminino
Seres Humanos
Testes de Função Renal
Masculino
Meia-Idade
Prognóstico
Diálise Renal/métodos
Medição de Risco
Dermatopatias Metabólicas/patologia
Dermatopatias Metabólicas/fisiopatologia
Uremia/diagnóstico
Uremia/terapia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1611
[Cu] Atualização por classe:161230
[Lr] Data última revisão:
161230
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151018
[St] Status:MEDLINE
[do] DOI:10.1111/ijd.12963


  5 / 75 MEDLINE  
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[PMID]:26290127
[Au] Autor:Carter A; Ortega-Loayza AG; Barrett J; Nunley J
[Ad] Endereço:Department of Dermatology, New York Medical College, New York, NY, USA.
[Ti] Título:Calciphylaxis with evidence of hypercoagulability successfully treated with unfractionated heparin: a multidisciplinary approach.
[So] Source:Clin Exp Dermatol;41(3):275-8, 2016 Apr.
[Is] ISSN:1365-2230
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Calciphylaxis is characterized by abnormal calcification of vessels and skin; however, its aetiology and pathogenesis remain unclear. Entities frequently associated with calciphylaxis are end-stage renal disease, diabetes mellitus, hypercalcaemia, hyperphosphataemia, elevated calcium-phosphate product, hyperparathyroidism and possible hypercoagulable states. Skin lesions may remain quiescent or may develop suddenly and progress rapidly. They are more common on the legs. Treatment of calciphylaxis is very challenging and requires interdisciplinary management. We present a case that highlights the difficulty of treating calciphylaxis. A multidisciplinary approach was vital for the proper treatment of our patient. This case also demonstrates the importance of searching for underlying hypercoagulable states, especially in recalcitrant cases. In cases of calciphylaxis with vessel occlusion from microthrombi, heparin therapy would be a logical next step. The effect of anticoagulation may be rapid and impressive.
[Mh] Termos MeSH primário: Anticoagulantes/uso terapêutico
Calciofilaxia/tratamento farmacológico
Heparina/uso terapêutico
Dermatopatias Metabólicas/tratamento farmacológico
Trombofilia/tratamento farmacológico
[Mh] Termos MeSH secundário: Feminino
Seres Humanos
Perna (Membro)
Meia-Idade
Resultado do Tratamento
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Anticoagulants); 9005-49-6 (Heparin)
[Em] Mês de entrada:1701
[Cu] Atualização por classe:170107
[Lr] Data última revisão:
170107
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150821
[St] Status:MEDLINE
[do] DOI:10.1111/ced.12729


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[PMID]:25807990
[Au] Autor:Kong MX; Zhang Q; Cao L; Zhao C; Ru GQ; Bi Q
[Ad] Endereço:Department of Orthopedics and Joint Surgery, Zhejiang Provincial People's Hospital, Hangzhou, Zhejiang, China.
[Ti] Título:Familial hypercholesterolaemia with tuberous and tendinous xanthomas: case report and mutation analysis.
[So] Source:Clin Exp Dermatol;40(7):765-9, 2015 Oct.
[Is] ISSN:1365-2230
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Xanthomas are important clinical manifestations of disordered lipid metabolism, which are mostly found in patients with familial hypercholesterolaemia (FH), an inherited disorder that is predominantly caused by mutations in the low-density lipoprotein receptor gene (LDLR). Tuberous and tendinous xanthomas with wide distribution and large size are rare; however, they may indicate the severity of FH, and tend to be found in homozygous FH. In this study, we investigated the clinical and genetic aspects of a young patient with FH presenting with multiple large masses in various locations. The lesions on the elbows and buttocks were locally excised and subsequently confirmed by biopsy to be xanthomas. Genetic analysis further confirmed that the patient was compound heterozygous for two mutations in both alleles of the LDLR gene. This rare case of compound heterozygous FH presenting with multiple large and widely distributed xanthomas provides a better understanding of FH and xanthomas.
[Mh] Termos MeSH primário: Hiperlipoproteinemia Tipo II/genética
Mutação
Receptores de LDL/genética
Dermatopatias Metabólicas/patologia
Xantomatose/patologia
[Mh] Termos MeSH secundário: Predisposição Genética para Doença
Seres Humanos
Masculino
Dermatopatias Metabólicas/genética
Xantomatose/genética
Adulto Jovem
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (LDLR protein, human); 0 (Receptors, LDL)
[Em] Mês de entrada:1606
[Cu] Atualização por classe:150916
[Lr] Data última revisão:
150916
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150327
[St] Status:MEDLINE
[do] DOI:10.1111/ced.12644


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[PMID]:25787691
[Au] Autor:Choque B; Catheline D; Delplanque B; Guesnet P; Legrand P
[Ad] Endereço:Laboratoire de Biochimie et Nutrition Humaine,INRA USC 2012, Agrocampus Ouest, 65 rue de Saint Brieuc,35042Rennes Cedex,France.
[Ti] Título:Dietary linoleic acid requirements in the presence of α-linolenic acid are lower than the historical 2 % of energy intake value, study in rats.
[So] Source:Br J Nutr;113(7):1056-68, 2015 Apr 14.
[Is] ISSN:1475-2662
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Previous studies on rats and human subjects have established that the linoleic acid (LA) requirement is 2 % of the total energy intake (en%), but is obtained in the absence of α-linolenic acid (ALA) and consequently appear to be overestimated. This raises questions since a recent study including ALA has suggested to divide the historical value by four. However, this recent study has remained inconclusive because the animals used were not totally LA-deficient animals. For the first time, the present study was especially designed using physiological and biochemical markers and performed in two steps: (1) to achieve a specific n-6 fatty acid deficiency model using growing male rats fed either a 0 en% from LA/0 en% from ALA (0LA/0ALA), 0LA/0·5ALA or 2LA/0·5ALA diet, born from female rats fed a 0LA/0·5ALA diet; and (2) to refine the required level of LA in the presence of ALA using rats fed either a 0LA/0ALA, 0·5LA/0·5ALA, 1LA/0·5ALA, 1·5LA/0·5ALA diet, born from female rats fed a 0LA/0·5ALA diet. The first step shows that the best LA deficiency model was obtained using rats fed the 0LA/0ALA diet, born from female rats fed the 0LA/0·5ALA diet. The second step demonstrates that in growing rats, LA deficiency was corrected with an intake of 1-1·5 en% from LA and 0·5 en% from ALA. These data suggest that the requirements in humans should be revisited, considering the presence of ALA to set up the recommendation for LA.
[Mh] Termos MeSH primário: Deficiências Nutricionais/prevenção & controle
Modelos Animais de Doenças
Ingestão de Energia
Ácido Linoleico/uso terapêutico
Necessidades Nutricionais
Ácido alfa-Linolênico/administração & dosagem
[Mh] Termos MeSH secundário: Animais
Biomarcadores
Deficiências Nutricionais/dietoterapia
Deficiências Nutricionais/fisiopatologia
Feminino
Desenvolvimento Fetal
Lactação
Ácido Linoleico/administração & dosagem
Ácido Linoleico/deficiência
Ácido Linoleico/metabolismo
Masculino
Fenômenos Fisiológicos da Nutrição Materna
Gravidez
Distribuição Aleatória
Ratos Wistar
Dermatopatias Metabólicas/etiologia
Dermatopatias Metabólicas/prevenção & controle
Cauda
Desmame
Ganho de Peso
Ácido alfa-Linolênico/deficiência
Ácido alfa-Linolênico/metabolismo
[Pt] Tipo de publicação:COMPARATIVE STUDY; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Biomarkers); 0RBV727H71 (alpha-Linolenic Acid); 9KJL21T0QJ (Linoleic Acid)
[Em] Mês de entrada:1506
[Cu] Atualização por classe:151119
[Lr] Data última revisão:
151119
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150320
[St] Status:MEDLINE
[do] DOI:10.1017/S0007114515000094


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[PMID]:25267496
[Au] Autor:Noh TK; Han JS; Won CH; Chang S; Choi JH; Moon KC; Lee MW; Yang JH; Soung JH
[Ad] Endereço:Department of Dermatology, Asan Medical Center, University of Ulsan College of Medicine, Seoul, Korea.
[Ti] Título:Characteristic "pebbling" skin eruption as a presenting sign of Hunter syndrome.
[So] Source:Int J Dermatol;53(12):e594-6, 2014 Dec.
[Is] ISSN:1365-4632
[Cp] País de publicação:England
[La] Idioma:eng
[Mh] Termos MeSH primário: Mucopolissacaridose II/diagnóstico
Dermatopatias Metabólicas/diagnóstico
[Mh] Termos MeSH secundário: Criança
Terapia de Reposição de Enzimas
Seres Humanos
Iduronato Sulfatase/uso terapêutico
Masculino
Mucopolissacaridose II/tratamento farmacológico
Proteínas Recombinantes/uso terapêutico
Dermatopatias Metabólicas/tratamento farmacológico
[Pt] Tipo de publicação:CASE REPORTS; LETTER
[Nm] Nome de substância:
0 (Recombinant Proteins); EC 3.1.6.13 (Iduronate Sulfatase); EC 3.1.6.13 (idursulfase)
[Em] Mês de entrada:1507
[Cu] Atualização por classe:141127
[Lr] Data última revisão:
141127
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:141001
[St] Status:MEDLINE
[do] DOI:10.1111/ijd.12206


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[PMID]:23928127
[Au] Autor:Radner FP; Fischer J
[Ad] Endereço:Institute for Human Genetics, University Medical Center Freiburg, Freiburg 79106, Germany. Electronic address: franz.radner@uniklinik-freiburg.de.
[Ti] Título:The important role of epidermal triacylglycerol metabolism for maintenance of the skin permeability barrier function.
[So] Source:Biochim Biophys Acta;1841(3):409-15, 2014 Mar.
[Is] ISSN:0006-3002
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Survival in a terrestrial, dry environment necessitates a permeability barrier for regulated permeation of water and electrolytes in the cornified layer of the skin (the stratum corneum) to minimize desiccation of the body. This barrier is formed during cornification and involves a cross-linking of corneocyte proteins as well as an extensive remodeling of lipids. The cleavage of precursor lipids from lamellar bodies by various hydrolytic enzymes generates ceramides, cholesterol, and non-esterified fatty acids for the extracellular lipid lamellae in the stratum corneum. However, the important role of epidermal triacylglycerol (TAG) metabolism during formation of a functional permeability barrier in the skin was only recently discovered. Humans with mutations in the ABHD5/CGI-58 (α/ß hydrolase domain containing protein 5, also known as comparative gene identification-58, CGI-58) gene suffer from a defect in TAG catabolism that causes neutral lipid storage disease with ichthyosis. In addition, mice with deficiencies in genes involved in TAG catabolism (Abhd5/Cgi-58 knock-out mice) or TAG synthesis (acyl-CoA:diacylglycerol acyltransferase-2, Dgat2 knock-out mice) also develop severe skin permeability barrier dysfunctions and die soon after birth due to increased dehydration. As a result of these defects in epidermal TAG metabolism, humans and mice lack ω-(O)-acylceramides, which leads to malformation of the cornified lipid envelope of the skin. In healthy skin, this epidermal structure provides an interface for the linkage of lamellar membranes with corneocyte proteins to maintain permeability barrier homeostasis. This review focuses on recent advances in the understanding of biochemical mechanisms involved in epidermal neutral lipid metabolism and the generation of a functional skin permeability barrier. This article is part of a Special Issue entitled The Important Role of Lipids in the Epidermis and their Role in the Formation and Maintenance of the Cutaneous Barrier. Guest Editors: Kenneth R. Feingold and Peter Elias.
[Mh] Termos MeSH primário: Metabolismo dos Lipídeos/fisiologia
Triglicerídeos/metabolismo
[Mh] Termos MeSH secundário: 1-Acilglicerol-3-Fosfato O-Aciltransferase/genética
1-Acilglicerol-3-Fosfato O-Aciltransferase/metabolismo
Animais
Diacilglicerol O-Aciltransferase/genética
Diacilglicerol O-Aciltransferase/metabolismo
Epiderme
Seres Humanos
Eritrodermia Ictiosiforme Congênita/genética
Eritrodermia Ictiosiforme Congênita/metabolismo
Eritrodermia Ictiosiforme Congênita/patologia
Erros Inatos do Metabolismo Lipídico/genética
Erros Inatos do Metabolismo Lipídico/metabolismo
Erros Inatos do Metabolismo Lipídico/patologia
Camundongos
Camundongos Knockout
Doenças Musculares/genética
Doenças Musculares/metabolismo
Doenças Musculares/patologia
Permeabilidade
Dermatopatias Metabólicas/genética
Dermatopatias Metabólicas/metabolismo
Dermatopatias Metabólicas/patologia
Triglicerídeos/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Triglycerides); EC 2.3.1.20 (DGAT2 protein, human); EC 2.3.1.20 (DGAT2 protein, mouse); EC 2.3.1.20 (Diacylglycerol O-Acyltransferase); EC 2.3.1.51 (1-Acylglycerol-3-Phosphate O-Acyltransferase); EC 2.3.1.51 (ABHD5 protein, human); EC 2.3.1.51 (Abhd5 protein, mouse)
[Em] Mês de entrada:1404
[Cu] Atualização por classe:161126
[Lr] Data última revisão:
161126
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:130810
[St] Status:MEDLINE


  10 / 75 MEDLINE  
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[PMID]:23817304
[Au] Autor:Grossberg AL
[Ad] Endereço:Department of Dermatology, Johns Hopkins University, Baltimore, Maryland 21287, USA. agrossb2@jhmi.edu
[Ti] Título:Update on pediatric photosensitivity disorders.
[So] Source:Curr Opin Pediatr;25(4):474-9, 2013 Aug.
[Is] ISSN:1531-698X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:PURPOSE OF REVIEW: Although rare in the pediatric population, photosensitive dermatoses may begin prior to adulthood. The causes of photosensitivity are diverse, ranging from primary, immunologically mediated disorders of photosensitivity to inherited genetic or metabolic disorders. This review will highlight the key features of these disorders to familiarize the pediatric practitioner with their symptoms and any associated extracutaneous clinical or laboratory findings that may accompany them. RECENT FINDINGS: New developments in the field of pediatric photosensitivity have been scant over recent years. While mechanisms of photosensitivity and genetic underpinnings associated with various conditions such as xeroderma pigmentosum continue to be uncovered, the literature on disorders of photosensitivity has been otherwise without many recent significant advances. SUMMARY: Although the differential diagnosis of pediatric photosensitivity disorders is broad, it is often possible to establish the diagnosis by following an algorithmic approach. Once the correct diagnosis is rendered, this will guide any further workup that needs to be performed as well as specific management strategies.
[Mh] Termos MeSH primário: Transtornos de Fotossensibilidade/diagnóstico
[Mh] Termos MeSH secundário: Idade de Início
Criança
Doenças do Tecido Conjuntivo/complicações
Diagnóstico Diferencial
Seres Humanos
Transtornos de Fotossensibilidade/etiologia
Transtornos de Fotossensibilidade/terapia
Dermatopatias Genéticas/diagnóstico
Dermatopatias Metabólicas/complicações
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1403
[Cu] Atualização por classe:130710
[Lr] Data última revisão:
130710
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:130703
[St] Status:MEDLINE
[do] DOI:10.1097/MOP.0b013e328362c2c3



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