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[PMID]:29406641
[Au] Autor:Celia T; Freysteinson WW; Frye RE
[Ti] Título:Concurrent Medical Conditions in Autism Spectrum Disorders.
[So] Source:Pediatr Nurs;42(5):230-4, 2016 Sep-Oct.
[Is] ISSN:0097-9805
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Long thought to be purely psychological in origin, current research lends credenceto the idea that autism has a medical basis. Patients with autism can be among themost challenging patients that a healthcare provider may care for. Often the presentingsymptoms of autism make these patients difficult to examine and may alsomask underlying concurrent conditions. This article reviews some of the more commonconditions found concurrently in the autistic population.
[Mh] Termos MeSH primário: Transtorno do Espectro Autista/complicações
Transtorno do Espectro Autista/diagnóstico
Gastroenteropatias/etiologia
Doenças do Sistema Imune/etnologia
Doenças do Sistema Nervoso/etiologia
Transtornos do Sono-Vigília/etiologia
[Mh] Termos MeSH secundário: Adolescente
Transtorno do Espectro Autista/epidemiologia
Transtorno do Espectro Autista/fisiopatologia
Criança
Pré-Escolar
Comorbidade
Feminino
Gastroenteropatias/epidemiologia
Gastroenteropatias/terapia
Seres Humanos
Doenças do Sistema Imune/epidemiologia
Doenças do Sistema Imune/terapia
Lactente
Masculino
Doenças do Sistema Nervoso/epidemiologia
Doenças do Sistema Nervoso/terapia
Transtornos do Sono-Vigília/epidemiologia
Transtornos do Sono-Vigília/terapia
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1803
[Cu] Atualização por classe:180305
[Lr] Data última revisão:
180305
[Sb] Subgrupo de revista:N
[Da] Data de entrada para processamento:180207
[St] Status:MEDLINE


  2 / 4239 MEDLINE  
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[PMID]:29289266
[Au] Autor:Trop-Steinberg S; Azar Y
[Ad] Endereço:Faculty of Life and Health Sciences (ST-S), JCT Lev Academic Institute, Jerusalem, Israel. Electronic address: shivtia@g.jct.ac.il.
[Ti] Título:Is Myc an Important Biomarker? Myc Expression in Immune Disorders and Cancer.
[So] Source:Am J Med Sci;355(1):67-75, 2018 Jan.
[Is] ISSN:1538-2990
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The proto-oncogene Myc serves as a paradigm for understanding the dynamics of transcriptional regulation. Myc protein has been linked to immune dysfunction, cancer development and neoplastic transformation. We review recent research regarding functions of Myc as an important modulator in immune disorders, postallogeneic hematopoietic stem cell transplantation (HSCT) and several cancers. Myc overexpression has been repeatedly linked to immune disorders and specific cancers, such as myasthenia gravis, psoriasis, pemphigus vulgaris, atherosclerosis, long-term allogeneic survival among HSCT patients, (primary) inflammatory breast cancer, (primary) ovarian carcinoma and hematological malignancies: acute myeloid leukemia, chronic myelogenous leukemia, Hodgkin's lymphoma and diffuse large B-cell lymphoma. However, decreased expression of Myc has been observed in HSCT patients who did not survive. Understanding impaired or inappropriate expression of Myc may present a path for the discovery of new targets for therapeutic applications.
[Mh] Termos MeSH primário: Biomarcadores Tumorais/biossíntese
Regulação Neoplásica da Expressão Gênica
Doenças do Sistema Imune/metabolismo
Neoplasias/metabolismo
Proteínas Proto-Oncogênicas c-myc/biossíntese
[Mh] Termos MeSH secundário: Aloenxertos
Intervalo Livre de Doença
Transplante de Células-Tronco Hematopoéticas
Seres Humanos
Doenças do Sistema Imune/patologia
Doenças do Sistema Imune/terapia
Neoplasias/patologia
Neoplasias/terapia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Biomarkers, Tumor); 0 (MYC protein, human); 0 (Proto-Oncogene Proteins c-myc)
[Em] Mês de entrada:1803
[Cu] Atualização por classe:180301
[Lr] Data última revisão:
180301
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:180101
[St] Status:MEDLINE


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[PMID]:29311458
[Au] Autor:Sasaoka S; Hatahira H; Hasegawa S; Motooka Y; Fukuda A; Naganuma M; Umetsu R; Nakao S; Shimauchi A; Ueda N; Hirade K; Iguchi K; Nakamura M
[Ad] Endereço:Laboratory of Drug Informatics, Gifu Pharmaceutical University.
[Ti] Título:[Adverse Event Trends Associated with Over-the-counter Combination Cold Remedy: Data Mining of the Japanese Adverse Drug Event Report Database].
[So] Source:Yakugaku Zasshi;138(1):123-134, 2018.
[Is] ISSN:1347-5231
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Ab] Resumo:OTC combination cold remedies are widely used in Japan. In the present study, we aimed to evaluate the adverse event profiles of OTC combination cold remedy based on the components using the Japanese Adverse Drug Event Report (JADER) database. The JADER database contained 430587 reports between April 2004 and November 2016. 1084 adverse events associated with the use of OTC combination cold remedy were reported. Reporting odds ratio (ROR) was used to detect safety signals. The ROR values for "skin and subcutaneous tissue disorders", "hepatobiliary disorders", and "immune system disorders" stratified by system organ class of the Medical Dictionary for Regulatory Activities (MedDRA) were 9.82 (8.71-11.06), 2.63 (2.25-3.07), and 3.13 (2.63-3.74), respectively. OTC combination cold remedy containing acetaminophen exhibited a significantly higher reporting ratio for "hepatobiliary disorders" than OTC combination cold remedy without acetaminophen. We demonstrated the potential risk of OTC combination cold remedy in a real-life setting. Our results suggested that the monitoring of individuals using OTC combination cold remedy is important.
[Mh] Termos MeSH primário: Acetaminofen/efeitos adversos
Sistemas de Notificação de Reações Adversas a Medicamentos
Mineração de Dados
Bases de Dados Factuais
Uso de Medicamentos/estatística & dados numéricos
Efeitos Colaterais e Reações Adversas Relacionados a Medicamentos/epidemiologia
Medicamentos sem Prescrição/efeitos adversos
[Mh] Termos MeSH secundário: Acetaminofen/administração & dosagem
Sistemas de Notificação de Reações Adversas a Medicamentos/estatística & dados numéricos
Doenças Biliares/induzido quimicamente
Doenças Biliares/epidemiologia
Doença Hepática Induzida por Substâncias e Drogas/epidemiologia
Doença Hepática Induzida por Substâncias e Drogas/etiologia
Combinação de Medicamentos
Doenças do Sistema Imune/induzido quimicamente
Doenças do Sistema Imune/epidemiologia
Japão/epidemiologia
Medicamentos sem Prescrição/administração & dosagem
Razão de Chances
Risco
Dermatopatias/induzido quimicamente
Dermatopatias/epidemiologia
Fatores de Tempo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Drug Combinations); 0 (Nonprescription Drugs); 362O9ITL9D (Acetaminophen)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180226
[Lr] Data última revisão:
180226
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180110
[St] Status:MEDLINE
[do] DOI:10.1248/yakushi.17-00172


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[PMID]:28458362
[Au] Autor:Tanaka K; Yoshitomi T; Hirahara K
[Ad] Endereço:Biologics & Immuno-Oncology Laboratories, Daiichi Sankyo Co., Ltd.
[Ti] Título:Elucidation of Distinct Roles of Guinea Pig CXCR1 and CXCR2 in Neutrophil Migration toward IL-8 and GROα by Specific Antibodies.
[So] Source:Biol Pharm Bull;40(5):729-732, 2017.
[Is] ISSN:1347-5215
[Cp] País de publicação:Japan
[La] Idioma:eng
[Ab] Resumo:Chemokine receptors CXCR1 and CXCR2 are conserved between guinea pigs and humans, but the distinct role of each receptor in chemotactic responses of neutrophils against chemokine ligands has not been elucidated due in part to the lack of specific inhibitors against these receptors in guinea pigs. In this study, we investigated the roles of guinea pig CXCR1 and CXCR2 on neutrophils in chemotactic responses to guinea pig interleukin (IL)-8 and growth-regulated oncogene (GRO)α by using specific inhibitory antibodies against these receptors. Neutrophil migration induced by IL-8 was partially inhibited by either anti-CXCR1 antibody or anti-CXCR2 antibody. In addition, the migration was inhibited completely when both anti-CXCR1 and anti-CXCR2 antibodies were combined. On the other hand, neutrophil migration induced by GROα was not inhibited by anti-CXCR1 antibody while inhibited profoundly by anti-CXCR2 antibody. These results indicated that CXCR1 and CXCR2 mediated migration induced by the IL-8 synergistically and only CXCR2 mediated migration induced by GROα in guinea pig neutrophils. Our findings on ligand selectivity of CXCR1 and CXCR2 in guinea pigs are consistent with those in humans.
[Mh] Termos MeSH primário: Quimiocina CXCL1/farmacologia
Interleucina-8/farmacologia
Neutrófilos/fisiologia
Receptores de Interleucina-8A/fisiologia
Receptores de Interleucina-8B/fisiologia
[Mh] Termos MeSH secundário: Animais
Anticorpos Bloqueadores/farmacologia
Movimento Celular/efeitos dos fármacos
Quimiocina CXCL1/antagonistas & inibidores
Quimiotaxia/efeitos dos fármacos
Feminino
Cobaias
Doenças do Sistema Imune
Interleucina-8/antagonistas & inibidores
Transtornos Leucocíticos
Neutrófilos/imunologia
Receptores de Interleucina-8A/imunologia
Receptores de Interleucina-8B/imunologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Antibodies, Blocking); 0 (Chemokine CXCL1); 0 (Interleukin-8); 0 (Receptors, Interleukin-8A); 0 (Receptors, Interleukin-8B)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180212
[Lr] Data última revisão:
180212
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170502
[St] Status:MEDLINE
[do] DOI:10.1248/bpb.b16-00918


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[PMID]:28059453
[Au] Autor:Abdollahi E; Momtazi AA; Johnston TP; Sahebkar A
[Ad] Endereço:Department of Medical Immunology, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
[Ti] Título:Therapeutic effects of curcumin in inflammatory and immune-mediated diseases: A nature-made jack-of-all-trades?
[So] Source:J Cell Physiol;233(2):830-848, 2018 Feb.
[Is] ISSN:1097-4652
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Curcumin is a dietary polyphenol from turmeric with numerous pharmacological activities. Novel animal and human studies indicate that curcumin can affect different immune cells, such as various T lymphocyte subsets, macrophages, dendritic cells, B lymphocytes and natural killer cells, which results in decreasing severity of various diseases with immunological etiology. The present review provides a comprehensive overview of the effects of curcumin on different immune cells and immune system-related diseases.
[Mh] Termos MeSH primário: Curcumina/uso terapêutico
Doenças do Sistema Imune/tratamento farmacológico
Fatores Imunológicos/uso terapêutico
Inflamação/tratamento farmacológico
[Mh] Termos MeSH secundário: Animais
Seres Humanos
Doenças do Sistema Imune/imunologia
Doenças do Sistema Imune/metabolismo
Inflamação/imunologia
Inflamação/metabolismo
Mediadores da Inflamação/imunologia
Mediadores da Inflamação/metabolismo
Transdução de Sinais/efeitos dos fármacos
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Immunologic Factors); 0 (Inflammation Mediators); IT942ZTH98 (Curcumin)
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171128
[Lr] Data última revisão:
171128
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170107
[St] Status:MEDLINE
[do] DOI:10.1002/jcp.25778


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[PMID]:28910364
[Au] Autor:Schlinzig T; Johansson S; Stephansson O; Hammarström L; Zetterström RH; von Döbeln U; Cnattingius S; Norman M
[Ad] Endereço:Division of Pediatrics, Department of Clinical Science, Intervention and Technology, Karolinska Institutet, Stockholm, Sweden.
[Ti] Título:Surge of immune cell formation at birth differs by mode of delivery and infant characteristics-A population-based cohort study.
[So] Source:PLoS One;12(9):e0184748, 2017.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Birth by cesarean section is associated with increased risks of immune disorders. We tested whether establishment of immune function at birth relates to mode of delivery, taking other maternal and infant characteristics into account. METHODS AND FINDINGS: Using a prospectively collected database, we retrieved information on maternal and infant characteristics of 6,014 singleton infants delivered from February to April 2014 in Stockholm, Sweden, with gestational age ≥35 weeks, Apgar scores ≥7, and without congenital malformations or any neonatal morbidity. We linked our data to blood levels of T-cell receptor excision circles (TREC) and κ-deleting recombination excision circles (KREC), determined as part of a neonatal screening program for immune-deficiencies, and representing quantities of newly formed T- and B-lymphocytes. Multivariate logistic regression was used to calculate odds ratios (OR) with 95% confidence intervals (CI) for participants having TREC and KREC levels in the lowest quintile. Multivariate models were adjusted for postnatal age at blood sampling, and included perinatal (mode of delivery, infant sex, gestational age, and birth weight for gestational age), and maternal characteristics (age, parity, BMI, smoking, diabetes, and hypertensive disease). Low TREC was associated with cesarean section before labor (adjusted OR:1.32 [95% CI 1.08-1.62]), male infant sex (aOR:1.60 [1.41-1.83]), preterm birth at 35-36 weeks of gestation (aOR:1.89 [1.21-2.96]) and small for gestational age (aOR:1.67 [1.00-2.79]). Low KREC was associated with male sex (aOR:1.32 [1.15-1.50]), postterm birth at ≥42 weeks (aOR:1.43 [1.13-1.82]) and small for gestational age (aOR:2.89 [1.78-4.69]). Maternal characteristics showed no consistent associations with neonatal levels of either TREC or KREC. CONCLUSION: Cesarean section before labor was associated with lower T-lymphocyte formation, irrespective of maternal characteristics, pregnancy, and neonatal risk factors. The significance of a reduced birth-related surge in lymphocyte formation for future immune function and health remains to be investigated.
[Mh] Termos MeSH primário: Linfócitos B/metabolismo
Parto Obstétrico/métodos
Doenças do Sistema Imune/diagnóstico
Triagem Neonatal/métodos
Linfócitos T/metabolismo
[Mh] Termos MeSH secundário: Índice de Apgar
Estudos de Coortes
Feminino
Idade Gestacional
Seres Humanos
Lactente
Recém-Nascido
Modelos Logísticos
Masculino
Estudos Prospectivos
Fatores de Risco
Suécia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171017
[Lr] Data última revisão:
171017
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170915
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0184748


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[PMID]:28886954
[Au] Autor:Mori AM; Agarwal S; Lee MWM; Rafferty M; Hardy TA; Coles A; Reddel SW; Riminton DS
[Ad] Endereço:Concord Repatriation General Hospital, Hospital Road, Concord, New South Wales 2139, Australia. Electronic address: amelia.m.mori@gmail.com.
[Ti] Título:A systematic checklist approach to immunosuppression risk management: An audit of practice at two clinical neuroimmunology centers.
[So] Source:J Neuroimmunol;312:4-7, 2017 Nov 15.
[Is] ISSN:1872-8421
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:There is no consensus approach to safety screening for immune intervention in clinical neuroimmunology. An immunosuppression risk evaluation checklist was used as an audit tool to assess real-world immunosuppression risk management and formulate recommendations for quality improvements in patient safety. Ninety-nine patients from two centres with 27 non-MS diagnoses were included. An average of 1.9 comorbidities with the potential to adversely impact morbidity and mortality associated with immunosuppression were identified. Diabetes and smoking were the most common, however a range of rarer but potentially life-threatening co-morbid disorders in the context of immunosuppression were identified. Inadequate documentation of risk mitigation tasks was common at 40.1% of total tasks across both cohorts. A routine, systematic immunosuppression checklist approach should be considered to improve immunosuppression risk management in clinical neuroimmunology practice.
[Mh] Termos MeSH primário: Lista de Checagem
Auditoria Clínica
Doenças do Sistema Imune/epidemiologia
Doenças do Sistema Imune/fisiopatologia
Imunossupressão
[Mh] Termos MeSH secundário: Austrália
Auditoria Clínica/métodos
Auditoria Clínica/normas
Estudos de Coortes
Estudos Transversais
Feminino
Seres Humanos
Doenças do Sistema Imune/mortalidade
Masculino
Gestão de Riscos
Reino Unido
[Pt] Tipo de publicação:JOURNAL ARTICLE; MULTICENTER STUDY
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171011
[Lr] Data última revisão:
171011
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170910
[St] Status:MEDLINE


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[PMID]:28780942
[Au] Autor:Akin C
[Ad] Endereço:Division of Allergy and Clinical Immunology, University of Michigan, Ann Arbor, Mich. Electronic address: cemakin@med.umich.edu.
[Ti] Título:Mast cell activation syndromes.
[So] Source:J Allergy Clin Immunol;140(2):349-355, 2017 Aug.
[Is] ISSN:1097-6825
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Mast cell activation is common and possibly necessary for maintenance of survival. Disordered mast cell activation occurs when mast cells are pathologically overproduced or if their activation is out of proportion to the perceived threat to homeostasis. Mast cell activation syndrome refers to a group of disorders with diverse causes presenting with episodic multisystem symptoms as the result of mast cell mediator release. Despite introduction of diagnostic criteria and some advances in treatment in the last decade, many areas of mast cell activation syndrome are in need of research. This article reviews our current knowledge about the various types of mast cell activation disorders, their treatment, and areas of uncertainty in need of future investigation.
[Mh] Termos MeSH primário: Mastócitos/imunologia
[Mh] Termos MeSH secundário: Seres Humanos
Doenças do Sistema Imune/imunologia
Síndrome
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170906
[Lr] Data última revisão:
170906
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170808
[St] Status:MEDLINE


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[PMID]:28778586
[Au] Autor:Hayatsu N; Miyao T; Tachibana M; Murakami R; Kimura A; Kato T; Kawakami E; Endo TA; Setoguchi R; Watarai H; Nishikawa T; Yasuda T; Yoshida H; Hori S
[Ad] Endereço:Laboratory for Immune Homeostasis, RIKEN Center for Integrative Medical Sciences, Kanagawa 230-0045, Japan.
[Ti] Título:Analyses of a Mutant Foxp3 Allele Reveal BATF as a Critical Transcription Factor in the Differentiation and Accumulation of Tissue Regulatory T Cells.
[So] Source:Immunity;47(2):268-283.e9, 2017 Aug 15.
[Is] ISSN:1097-4180
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Foxp3 controls the development and function of regulatory T (Treg) cells, but it remains elusive how Foxp3 functions in vivo. Here, we established mouse models harboring three unique missense Foxp3 mutations that were identified in patients with the autoimmune disease IPEX. The I363V and R397W mutations were loss-of-function mutations, causing multi-organ inflammation by globally compromising Treg cell physiology. By contrast, the A384T mutation induced a distinctive tissue-restricted inflammation by specifically impairing the ability of Treg cells to compete with pathogenic T cells in certain non-lymphoid tissues. Mechanistically, repressed BATF expression contributed to these A384T effects. At the molecular level, the A384T mutation altered Foxp3 interactions with its specific target genes including Batf by broadening its DNA-binding specificity. Our findings identify BATF as a critical regulator of tissue Treg cells and suggest that sequence-specific perturbations of Foxp3-DNA interactions can influence specific facets of Treg cell physiology and the immunopathologies they regulate.
[Mh] Termos MeSH primário: Fatores de Transcrição de Zíper de Leucina Básica/metabolismo
Diabetes Mellitus Tipo 1/congênito
Diarreia/genética
Fatores de Transcrição Forkhead/metabolismo
Doenças Genéticas Ligadas ao Cromossomo X/genética
Doenças do Sistema Imune/congênito
Inflamação/genética
Linfócitos T Reguladores/fisiologia
[Mh] Termos MeSH secundário: Alelos
Animais
Fatores de Transcrição de Zíper de Leucina Básica/genética
Diferenciação Celular
Movimento Celular
Células Cultivadas
Análise Mutacional de DNA
Diabetes Mellitus Tipo 1/genética
Diabetes Mellitus Tipo 1/imunologia
Diarreia/imunologia
Fatores de Transcrição Forkhead/genética
Doenças Genéticas Ligadas ao Cromossomo X/imunologia
Seres Humanos
Doenças do Sistema Imune/genética
Doenças do Sistema Imune/imunologia
Inflamação/imunologia
Camundongos
Camundongos Endogâmicos BALB C
Camundongos Endogâmicos C57BL
Camundongos Knockout
Mutação de Sentido Incorreto/genética
Especificidade de Órgãos/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Basic-Leucine Zipper Transcription Factors); 0 (Batf protein, mouse); 0 (Forkhead Transcription Factors); 0 (Foxp3 protein, mouse)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170928
[Lr] Data última revisão:
170928
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170806
[St] Status:MEDLINE


  10 / 4239 MEDLINE  
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[PMID]:28778450
[Au] Autor:Kumar MM; Venkataswamy MM; Sathyanarayanan G; Thippeswamy H; Chandra PS; Mani RS
[Ad] Endereço:Department of Neurovirology, National Institute of Mental Health and Neurosciences (NIMHANS), Bangalore 560029, India.
[Ti] Título:Immune system aberrations in postpartum psychosis: An immunophenotyping study from a tertiary care neuropsychiatric hospital in India.
[So] Source:J Neuroimmunol;310:8-13, 2017 Sep 15.
[Is] ISSN:1872-8421
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Postpartum psychosis (PP) is associated with significant morbidity to both mother and infant. Immune system dysregulation during PP is reported in recent studies. This study attempted to determine immune signatures associated with first-onset PP by flow cytometry. Peripheral blood showed decreased naive CD4 and CD8 T cells, while activated CD8 and memory regulatory T cells (Tregs) were increased in women with PP as against healthy controls. The CD14 CD16 non-classical monocytes, CD11c+myeloid DCs and cytotoxic CD56 CD16 were reduced, while CD56 CD16 regulatory NK cells were elevated in women with PP. The variations in immune cell subsets highlight the generalized immune dysregulation in PP.
[Mh] Termos MeSH primário: Citocinas/metabolismo
Doenças do Sistema Imune/etiologia
Imunofenotipagem
Período Pós-Parto/imunologia
Transtornos Psicóticos/complicações
[Mh] Termos MeSH secundário: Adulto
Células Dendríticas/metabolismo
Células Dendríticas/patologia
Feminino
Citometria de Fluxo
Hospitais Psiquiátricos/estatística & dados numéricos
Seres Humanos
Doenças do Sistema Imune/patologia
Índia/epidemiologia
Células Matadoras Naturais/metabolismo
Células Matadoras Naturais/patologia
Monócitos/metabolismo
Monócitos/patologia
Subpopulações de Linfócitos T/metabolismo
Subpopulações de Linfócitos T/patologia
Atenção Terciária à Saúde
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Cytokines)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170822
[Lr] Data última revisão:
170822
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170806
[St] Status:MEDLINE



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