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[PMID]:29331375
[Au] Autor:Ma L; Wang Q; Yuan M; Zou T; Yin P; Wang S
[Ad] Endereço:National Key Laboratory of Crop Genetic Improvement, National Center of Plant Gene Research (Wuhan), Huazhong Agricultural University, Wuhan 430070, China.
[Ti] Título:Xanthomonas TAL effectors hijack host basal transcription factor IIA α and γ subunits for invasion.
[So] Source:Biochem Biophys Res Commun;496(2):608-613, 2018 02 05.
[Is] ISSN:1090-2104
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The Xanthomonas genus includes Gram-negative plant-pathogenic bacteria, which infect a broad range of crops and wild plant species, cause symptoms with leaf blights, streaks, spots, stripes, necrosis, wilt, cankers and gummosis on leaves, stems and fruits in a wide variety of plants via injecting their effector proteins into the host cell during infection. Among these virulent effectors, transcription activator-like effectors (TALEs) interact with the γ subunit of host transcription factor IIA (TFIIAγ) to activate the transcription of host disease susceptibility genes. Functional TFIIA is a ternary complex comprising α, ß and γ subunits. However, whether TALEs recruit TFIIAα, TFIIAß, or both remains unknown. The underlying molecular mechanisms by which TALEs mediate host susceptibility gene activation require full elucidation. Here, we show that TALEs interact with the α+γ binary subcomplex but not the α+ß+γ ternary complex of rice TFIIA (holo-OsTFIIA). The transcription factor binding (TFB) regions of TALEs, which are highly conserved in Xanthomonas species, have a dominant role in these interactions. Furthermore, the interaction between TALEs and the α+γ complex exhibits robust DNA binding activity in vitro. These results collectively demonstrate that TALE-carrying pathogens hijack the host basal transcription factors TFIIAα and TFIIAγ, but not TFIIAß, to enhance host susceptibility during pathogen infection. The uncovered mechanism widens new insights on host-microbe interaction and provide an applicable strategy to breed high-resistance crop varieties.
[Mh] Termos MeSH primário: Interações Hospedeiro-Patógeno
Oryza/microbiologia
Doenças das Plantas/microbiologia
Proteínas de Plantas/metabolismo
Efetores Semelhantes a Ativadores de Transcrição/metabolismo
Fator de Transcrição TFIIA/metabolismo
Xanthomonas/fisiologia
[Mh] Termos MeSH secundário: Resistência à Doença
Regulação da Expressão Gênica de Plantas
Genes de Plantas
Oryza/genética
Oryza/metabolismo
Doenças das Plantas/genética
Ligação Proteica
Subunidades Proteicas/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Plant Proteins); 0 (Protein Subunits); 0 (Transcription Activator-Like Effectors); 0 (Transcription Factor TFIIA)
[Em] Mês de entrada:1803
[Cu] Atualização por classe:180309
[Lr] Data última revisão:
180309
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180115
[St] Status:MEDLINE


  2 / 4611 MEDLINE  
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[PMID]:28453680
[Au] Autor:Holtz Y; David JL; Ranwez V
[Ad] Endereço:Montpellier Supagro, Department BE, UMR AGAP, 34060 Montpellier, France.
[Ti] Título:The genetic map comparator: a user-friendly application to display and compare genetic maps.
[So] Source:Bioinformatics;33(9):1387-1388, 2017 05 01.
[Is] ISSN:1367-4811
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Motivation: Marker-assisted selection strongly relies on genetic maps to accelerate breeding programs. High-density maps are now available for numerous species. Dedicated tools are required to compare several high-density maps on the basis of their key characteristics, while pinpointing their differences and similarities. Results: We developed the Genetic Map Comparator-a web-based application for easy comparison of different maps according to their key statistics and the relative positions of common markers. Availability and Implementation: The Genetic Map Comparator is available online at: http://bioweb.supagro.inra.fr/geneticMapComparator. The source code is freely available on GitHub under the under the CeCILL general public license: https://github.com/holtzy/GenMap-Comparator. Contact: Holtz@supagro.fr; Ranwez@supagro.fr.
[Mh] Termos MeSH primário: Genômica/métodos
Análise de Sequência de DNA/métodos
Software
[Mh] Termos MeSH secundário: Resistência à Doença/genética
Genes de Plantas
Doenças das Plantas/genética
Locos de Características Quantitativas
Triticum/genética
Triticum/virologia
Viroses/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Mês de entrada:1801
[Cu] Atualização por classe:180308
[Lr] Data última revisão:
180308
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170429
[St] Status:MEDLINE
[do] DOI:10.1093/bioinformatics/btw816


  3 / 4611 MEDLINE  
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[PMID]:28465342
[Au] Autor:Stefanova D; Raychev A; Arezes J; Ruchala P; Gabayan V; Skurnik M; Dillon BJ; Horwitz MA; Ganz T; Bulut Y; Nemeth E
[Ad] Endereço:Molecular, Cellular, and Integrative Physiology Graduate Program and.
[Ti] Título:Endogenous hepcidin and its agonist mediate resistance to selected infections by clearing non-transferrin-bound iron.
[So] Source:Blood;130(3):245-257, 2017 07 20.
[Is] ISSN:1528-0020
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The iron-regulatory hormone hepcidin is induced early in infection, causing iron sequestration in macrophages and decreased plasma iron; this is proposed to limit the replication of extracellular microbes, but could also promote infection with macrophage-tropic pathogens. The mechanisms by which hepcidin and hypoferremia modulate host defense, and the spectrum of microbes affected, are poorly understood. Using mouse models, we show that hepcidin was selectively protective against siderophilic extracellular pathogens ( O9) by controlling non-transferrin-bound iron (NTBI) rather than iron-transferrin concentration. NTBI promoted the rapid growth of siderophilic but not nonsiderophilic bacteria in mice with either genetic or iatrogenic iron overload and in human plasma. Hepcidin or iron loading did not affect other key components of innate immunity, did not indiscriminately promote intracellular infections ( ), and had no effect on extracellular nonsiderophilic O8 or Hepcidin analogs may be useful for treatment of siderophilic infections.
[Mh] Termos MeSH primário: Infecções Relacionadas a Cateter/imunologia
Hemocromatose/imunologia
Hepcidinas/imunologia
Sobrecarga de Ferro/imunologia
Ferro/metabolismo
Infecções Estafilocócicas/imunologia
[Mh] Termos MeSH secundário: Animais
Ligação Competitiva
Infecções Relacionadas a Cateter/metabolismo
Infecções Relacionadas a Cateter/microbiologia
Infecções Relacionadas a Cateter/mortalidade
Modelos Animais de Doenças
Resistência à Doença
Expressão Gênica
Hemocromatose/metabolismo
Hemocromatose/microbiologia
Hemocromatose/mortalidade
Hepcidinas/agonistas
Hepcidinas/deficiência
Hepcidinas/genética
Seres Humanos
Ferro/imunologia
Sobrecarga de Ferro/metabolismo
Sobrecarga de Ferro/microbiologia
Sobrecarga de Ferro/mortalidade
Camundongos
Camundongos Endogâmicos C57BL
Camundongos Knockout
Mycobacterium tuberculosis/efeitos dos fármacos
Mycobacterium tuberculosis/crescimento & desenvolvimento
Mycobacterium tuberculosis/metabolismo
Oligopeptídeos/farmacologia
Ligação Proteica
Infecções Estafilocócicas/metabolismo
Infecções Estafilocócicas/microbiologia
Infecções Estafilocócicas/mortalidade
Staphylococcus aureus
Análise de Sobrevida
Transferrina/genética
Transferrina/metabolismo
Yersinia enterocolitica/efeitos dos fármacos
Yersinia enterocolitica/crescimento & desenvolvimento
Yersinia enterocolitica/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Hamp1 protein, mouse); 0 (Hepcidins); 0 (Oligopeptides); 0 (Transferrin); E1UOL152H7 (Iron)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:180305
[Lr] Data última revisão:
180305
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170504
[St] Status:MEDLINE
[do] DOI:10.1182/blood-2017-03-772715


  4 / 4611 MEDLINE  
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[PMID]:29272270
[Au] Autor:Badet T; Voisin D; Mbengue M; Barascud M; Sucher J; Sadon P; Balagué C; Roby D; Raffaele S
[Ad] Endereço:LIPM, Université de Toulouse, INRA, CNRS, Castanet-Tolosan, France.
[Ti] Título:Parallel evolution of the POQR prolyl oligo peptidase gene conferring plant quantitative disease resistance.
[So] Source:PLoS Genet;13(12):e1007143, 2017 12.
[Is] ISSN:1553-7404
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Plant pathogens with a broad host range are able to infect plant lineages that diverged over 100 million years ago. They exert similar and recurring constraints on the evolution of unrelated plant populations. Plants generally respond with quantitative disease resistance (QDR), a form of immunity relying on complex genetic determinants. In most cases, the molecular determinants of QDR and how they evolve is unknown. Here we identify in Arabidopsis thaliana a gene mediating QDR against Sclerotinia sclerotiorum, agent of the white mold disease, and provide evidence of its convergent evolution in multiple plant species. Using genome wide association mapping in A. thaliana, we associated the gene encoding the POQR prolyl-oligopeptidase with QDR against S. sclerotiorum. Loss of this gene compromised QDR against S. sclerotiorum but not against a bacterial pathogen. Natural diversity analysis associated POQR sequence with QDR. Remarkably, the same amino acid changes occurred after independent duplications of POQR in ancestors of multiple plant species, including A. thaliana and tomato. Genome-scale expression analyses revealed that parallel divergence in gene expression upon S. sclerotiorum infection is a frequent pattern in genes, such as POQR, that duplicated both in A. thaliana and tomato. Our study identifies a previously uncharacterized gene mediating QDR against S. sclerotiorum. It shows that some QDR determinants are conserved in distantly related plants and have emerged through the repeated use of similar genetic polymorphisms at different evolutionary time scales.
[Mh] Termos MeSH primário: Resistência à Doença/genética
Serina Endopeptidases/genética
[Mh] Termos MeSH secundário: Arabidopsis/genética
Ascomicetos/genética
Ascomicetos/patogenicidade
Mapeamento Cromossômico
Regulação da Expressão Gênica de Plantas
Estudo de Associação Genômica Ampla
Doenças das Plantas/genética
Imunidade Vegetal/genética
Proteínas de Plantas/genética
Serina Endopeptidases/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Plant Proteins); EC 3.4.21.- (Serine Endopeptidases); EC 3.4.21.26 (prolyl oligopeptidase)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180227
[Lr] Data última revisão:
180227
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171223
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pgen.1007143


  5 / 4611 MEDLINE  
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[PMID]:28747428
[Au] Autor:Slootweg E; Koropacka K; Roosien J; Dees R; Overmars H; Lankhorst RK; van Schaik C; Pomp R; Bouwman L; Helder J; Schots A; Bakker J; Smant G; Goverse A
[Ad] Endereço:Laboratory of Nematology, Department of Plant Sciences, Wageningen University, 6708 PD Wageningen, The Netherlands.
[Ti] Título:Sequence Exchange between Homologous NB-LRR Genes Converts Virus Resistance into Nematode Resistance, and Vice Versa.
[So] Source:Plant Physiol;175(1):498-510, 2017 Sep.
[Is] ISSN:1532-2548
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Plants have evolved a limited repertoire of NB-LRR disease resistance ( ) genes to protect themselves against myriad pathogens. This limitation is thought to be counterbalanced by the rapid evolution of NB-LRR proteins, as only a few sequence changes have been shown to be sufficient to alter resistance specificities toward novel strains of a pathogen. However, little is known about the flexibility of NB-LRR genes to switch resistance specificities between phylogenetically unrelated pathogens. To investigate this, we created domain swaps between the close homologs and , which confer resistance in potato ( ) to the cyst nematode and , respectively. The genetic fusion of the CC-NB-ARC of Gpa2 with the LRR of Rx1 (Gpa2 /Rx1 ) results in autoactivity, but lowering the protein levels restored its specific activation response, including extreme resistance to in potato shoots. The reciprocal chimera (Rx1 /Gpa2 ) shows a loss-of-function phenotype, but exchange of the first three LRRs of Gpa2 by the corresponding region of Rx1 was sufficient to regain a wild-type resistance response to in the roots. These data demonstrate that exchanging the recognition moiety in the LRR is sufficient to convert extreme virus resistance in the leaves into mild nematode resistance in the roots, and vice versa. In addition, we show that the CC-NB-ARC can operate independently of the recognition specificities defined by the LRR domain, either aboveground or belowground. These data show the versatility of NB-LRR genes to generate resistance to unrelated pathogens with completely different lifestyles and routes of invasion.
[Mh] Termos MeSH primário: Resistência à Doença/genética
Doenças das Plantas/imunologia
Proteínas de Plantas/metabolismo
Potexvirus/fisiologia
Solanum tuberosum/genética
Tylenchoidea/fisiologia
[Mh] Termos MeSH secundário: Animais
Mutação com Perda de Função
Fenótipo
Doenças das Plantas/parasitologia
Doenças das Plantas/virologia
Folhas de Planta/genética
Folhas de Planta/imunologia
Folhas de Planta/parasitologia
Folhas de Planta/virologia
Proteínas de Plantas/genética
Raízes de Plantas/genética
Raízes de Plantas/imunologia
Raízes de Plantas/parasitologia
Raízes de Plantas/virologia
Brotos de Planta/genética
Brotos de Planta/imunologia
Brotos de Planta/parasitologia
Brotos de Planta/virologia
Domínios Proteicos
Proteínas/genética
Proteínas/metabolismo
Receptores Imunológicos/genética
Receptores Imunológicos/metabolismo
Proteínas Recombinantes de Fusão
Solanum tuberosum/imunologia
Solanum tuberosum/parasitologia
Solanum tuberosum/virologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Plant Proteins); 0 (Proteins); 0 (Receptors, Immunologic); 0 (Recombinant Fusion Proteins); 0 (leucine-rich repeat proteins)
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180226
[Lr] Data última revisão:
180226
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170728
[St] Status:MEDLINE
[do] DOI:10.1104/pp.17.00485


  6 / 4611 MEDLINE  
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[PMID]:28453414
[Au] Autor:Dolega P; Szulc-Dabrowska L; Bossowska M; Mielcarska M; Nowak Z; Toka FN
[Ad] Endereço:1 Division of Immunology, Department of Preclinical Sciences, Faculty of Veterinary Medicine, Warsaw University of Life Sciences , Warsaw, Poland .
[Ti] Título:Innate Immune Gene Transcript Level Associated with the Infection of Macrophages with Ectromelia Virus in Two Different Mouse Strains.
[So] Source:Viral Immunol;30(5):315-329, 2017 06.
[Is] ISSN:1557-8976
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Poxviruses have evolved numerous mechanisms to avoid the immune response of the infected host, and many of these mechanisms have not been fully described. Here, we studied the transcriptional response of innate immune genes in BALB/c and C57BL/6 peritoneal macrophages following infection with the Moscow strain of ectromelia virus (ECTV-Mos) with the aim of delineating innate immune genes that contribute to the difference between susceptibility and resistance to lethal infection. We show a generalized downregulation of many genes in four categories (toll-like receptor signaling, NOD-like receptor signaling, RIG-I-like receptor signaling, and type I interferon signaling) of antiviral innate immune receptors, downstream signaling pathways, and responsive components. Two important observations were made. First, 14 innate antiviral genes were differentially expressed with fold change upregulation of two and above occurring in C57BL/6 mice, known to be resistant to ECTV-Mos infection, whereas the same genes were downregulated in BALB/c mice with fold change of two and below. Second, the cathepsin group of genes was downregulated in both strains of mice but with profound fold changes of 17, 38, and 62 downregulation for CtsL, CtsB, and CtsS, respectively, in C57BL/6 mice. We show that a poxvirus profoundly downregulates both the mRNA and protein expression of these three cathepsins and this change appears to support virus replication. Based on these data we propose that the variations in gene expression observed may contribute to the difference in resistance/susceptibility between BALB/c and C57BL/6 mice to lethal infection by ECTV-Mos.
[Mh] Termos MeSH primário: Resistência à Doença
Vírus da Ectromelia/imunologia
Ectromelia Infecciosa/imunologia
Perfilação da Expressão Gênica
Imunidade Inata
Macrófagos Peritoneais/imunologia
[Mh] Termos MeSH secundário: Animais
Masculino
Camundongos Endogâmicos BALB C
Camundongos Endogâmicos C57BL
[Pt] Tipo de publicação:COMPARATIVE STUDY; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180226
[Lr] Data última revisão:
180226
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170429
[St] Status:MEDLINE
[do] DOI:10.1089/vim.2016.0173


  7 / 4611 MEDLINE  
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[PMID]:29209811
[Au] Autor:Panno S; Caruso AG; Davino S
[Ad] Endereço:Department of Agricultural, Food and Forest science, University of Palermo, Viale delle Scienze, bld 5, 90128, Palermo, Italy.
[Ti] Título:The nucleotide sequence of a recombinant tomato yellow leaf curl virus strain frequently detected in Sicily isolated from tomato plants carrying the Ty-1 resistance gene.
[So] Source:Arch Virol;163(3):795-797, 2018 Mar.
[Is] ISSN:1432-8798
[Cp] País de publicação:Austria
[La] Idioma:eng
[Ab] Resumo:In July 2016, an aggressive syndrome of tomato yellow leaf curl disease was reported in Sicily in tomato plants carrying the Ty-1 resistance gene. A total of 34 samples were collected and analyzed. Twenty-seven out of the 34 samples analyzed appeared to contain only recombinant molecules. One full sequence was obtained after cloning. Alignments and plot similarity analysis showed that the genome of the recombinant, named TYLCV-IL[IT:Sic23:16], was mostly derived from tomato yellow leaf curl virus (TYLCV), with a small region of 132 nucleotides in the non-coding region between the stem-loop and the start of the V2 ORF replaced by 124 nucleotides derived from a virus of a different species, tomato yellow leaf curl Sardinia virus. All plants in which the new recombinant was detected belonged to resistant tomato cultivars.
[Mh] Termos MeSH primário: Begomovirus/genética
Genes Virais/genética
Lycopersicon esculentum/virologia
Folhas de Planta/virologia
Recombinação Genética
[Mh] Termos MeSH secundário: Sequência de Bases
Begomovirus/classificação
Begomovirus/isolamento & purificação
Resistência à Doença/genética
Suscetibilidade a Doenças
Sequenciamento de Nucleotídeos em Larga Escala
Lycopersicon esculentum/genética
Lycopersicon esculentum/imunologia
Doenças das Plantas/genética
Doenças das Plantas/imunologia
Doenças das Plantas/virologia
Imunidade Vegetal/genética
Folhas de Planta/genética
Folhas de Planta/imunologia
Plantas Geneticamente Modificadas
Análise de Sequência de DNA
Sicília
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180220
[Lr] Data última revisão:
180220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171207
[St] Status:MEDLINE
[do] DOI:10.1007/s00705-017-3674-9


  8 / 4611 MEDLINE  
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[PMID]:29205105
[Au] Autor:Ojiambo PS; Yuen J; van den Bosch F; Madden LV
[Ad] Endereço:2017 Focus Issue Senior Editors First author: Center for Integrated Fungal Research, Department of Entomology and Plant Pathology, North Carolina State University, Raleigh 27695; second author: Department of Forest Mycology and Plant Pathology, Swedish University of Agricultural Sciences, Uppsala, S
[Ti] Título:Epidemiology: Past, Present, and Future Impacts on Understanding Disease Dynamics and Improving Plant Disease Management-A Summary of Focus Issue Articles.
[So] Source:Phytopathology;107(10):1092-1094, 2017 Oct.
[Is] ISSN:0031-949X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Epidemiology has made significant contributions to plant pathology by elucidating the general principles underlying the development of disease epidemics. This has resulted in a greatly improved theoretical and empirical understanding of the dynamics of disease epidemics in time and space, predictions of disease outbreaks or the need for disease control in real-time basis, and tactical and strategic solutions to disease problems. Availability of high-resolution experimental data at multiple temporal and spatial scales has now provided a platform to test and validate theories on the spread of diseases at a wide range of spatial scales ranging from the local to the landscape level. Relatively new approaches in plant disease epidemiology, ranging from network to information theory, coupled with the availability of large-scale datasets and the rapid development of computer technology, are leading to revolutionary thinking about epidemics that can result in considerable improvement of strategic and tactical decision making in the control and management of plant diseases. Methods that were previously restricted to topics such as population biology or evolution are now being employed in epidemiology to enable a better understanding of the forces that drive the development of plant disease epidemics in space and time. This Focus Issue of Phytopathology features research articles that address broad themes in epidemiology including social and political consequences of disease epidemics, decision theory and support, pathogen dispersal and disease spread, disease assessment and pathogen biology and disease resistance. It is important to emphasize that these articles are just a sample of the types of research projects that are relevant to epidemiology. Below, we provide a succinct summary of the articles that are published in this Focus Issue .
[Mh] Termos MeSH primário: Resistência à Doença
Epidemias
Doenças das Plantas/prevenção & controle
Patologia Vegetal
[Mh] Termos MeSH secundário: Agricultura
Doenças das Plantas/estatística & dados numéricos
[Pt] Tipo de publicação:INTRODUCTORY JOURNAL ARTICLE
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180215
[Lr] Data última revisão:
180215
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171206
[St] Status:MEDLINE
[do] DOI:10.1094/PHYTO-07-17-0248-FI


  9 / 4611 MEDLINE  
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[PMID]:28745102
[Au] Autor:Aoun M; Kolmer JA; Rouse MN; Chao S; Bulbula WD; Elias EM; Acevedo M
[Ad] Endereço:First author: Department of Plant Pathology, North Dakota State University, Fargo 58108; second and third authors: United States Department of Agriculture-Agricultural Research Service (USDA-ARS), Cereal Disease Laboratory, and Department of Plant Pathology, University of Minnesota, St. Paul 55108;
[Ti] Título:Inheritance and Bulked Segregant Analysis of Leaf Rust and Stem Rust Resistance in Durum Wheat Genotypes.
[So] Source:Phytopathology;107(12):1496-1506, 2017 12.
[Is] ISSN:0031-949X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Leaf rust, caused by Puccinia triticina, and stem rust, caused by P. graminis f. sp. tritici, are important diseases of durum wheat. This study determined the inheritance and genomic locations of leaf rust resistance (Lr) genes to P. triticina race BBBQJ and stem rust resistance (Sr) genes to P. graminis f. sp. tritici race TTKSK in durum accessions. Eight leaf-rust-resistant genotypes were used to develop biparental populations. Accessions PI 192051 and PI 534304 were also resistant to P. graminis f. sp. tritici race TTKSK. The resulting progenies were phenotyped for leaf rust and stem rust response at seedling stage. The Lr and Sr genes were mapped in five populations using single-nucleotide polymorphisms and bulked segregant analysis. Five leaf-rust-resistant genotypes carried single dominant Lr genes whereas, in the remaining accessions, there was deviation from the expected segregation ratio of a single dominant Lr gene. Seven genotypes carried Lr genes different from those previously characterized in durum. The single dominant Lr genes in PI 209274, PI 244061, PI387263, and PI 313096 were mapped to chromosome arms 6BS, 2BS, 6BL, and 6BS, respectively. The Sr gene in PI 534304 mapped to 6AL and is most likely Sr13, while the Sr gene in PI 192051 could be uncharacterized in durum.
[Mh] Termos MeSH primário: Basidiomycota/fisiologia
Resistência à Doença/genética
Doenças das Plantas/imunologia
Triticum/genética
[Mh] Termos MeSH secundário: Mapeamento Cromossômico
Cromossomos de Plantas/genética
Loci Gênicos/genética
Genótipo
Fenótipo
Doenças das Plantas/microbiologia
Folhas de Planta/genética
Folhas de Planta/imunologia
Folhas de Planta/microbiologia
Caules de Planta/genética
Caules de Planta/imunologia
Caules de Planta/microbiologia
Polimorfismo de Nucleotídeo Único/genética
Plântulas/genética
Plântulas/imunologia
Plântulas/microbiologia
Triticum/imunologia
Triticum/microbiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180215
[Lr] Data última revisão:
180215
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170727
[St] Status:MEDLINE
[do] DOI:10.1094/PHYTO-12-16-0444-R


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[PMID]:28742460
[Au] Autor:Garrett KA; Andersen KF; Asche F; Bowden RL; Forbes GA; Kulakow PA; Zhou B
[Ad] Endereço:First and second authors: Plant Pathology Department, Emerging Pathogens Institute, and Institute for Sustainable Food Systems, University of Florida, Gainesville 32611; third author: School of Forest Resources and Conservation and Institute for Sustainable Food Systems, University of Florida, Gaine
[Ti] Título:Resistance Genes in Global Crop Breeding Networks.
[So] Source:Phytopathology;107(10):1268-1278, 2017 10.
[Is] ISSN:0031-949X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Resistance genes are a major tool for managing crop diseases. The networks of crop breeders who exchange resistance genes and deploy them in varieties help to determine the global landscape of resistance and epidemics, an important system for maintaining food security. These networks function as a complex adaptive system, with associated strengths and vulnerabilities, and implications for policies to support resistance gene deployment strategies. Extensions of epidemic network analysis can be used to evaluate the multilayer agricultural networks that support and influence crop breeding networks. Here, we evaluate the general structure of crop breeding networks for cassava, potato, rice, and wheat. All four are clustered due to phytosanitary and intellectual property regulations, and linked through CGIAR hubs. Cassava networks primarily include public breeding groups, whereas others are more mixed. These systems must adapt to global change in climate and land use, the emergence of new diseases, and disruptive breeding technologies. Research priorities to support policy include how best to maintain both diversity and redundancy in the roles played by individual crop breeding groups (public versus private and global versus local), and how best to manage connectivity to optimize resistance gene deployment while avoiding risks to the useful life of resistance genes. [Formula: see text] Copyright © 2017 The Author(s). This is an open access article distributed under the CC BY 4.0 International license .
[Mh] Termos MeSH primário: Resistência à Doença/genética
Manihot/genética
Oryza/genética
Doenças das Plantas/imunologia
Solanum tuberosum/genética
Triticum/genética
[Mh] Termos MeSH secundário: Agricultura
Cruzamento
Clima
Produtos Agrícolas
Abastecimento de Alimentos
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T; RESEARCH SUPPORT, U.S. GOV'T, NON-P.H.S.
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180215
[Lr] Data última revisão:
180215
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170726
[St] Status:MEDLINE
[do] DOI:10.1094/PHYTO-03-17-0082-FI



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