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Referências encontradas : 597 [refinar]
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[PMID]:28002071
[Au] Autor:Smith SS; Cannon DL; Fagan K; Weis CP
[Ad] Endereço:Fort Belvoir Community Hospital, Fort Belvoir, VA, steph.sayre@gmail.com Occupational Safety and Health, Administration, Washington, DC National Institute of Environmental, Health Sciences, Bethesda, MD.
[Ti] Título:Occupational Hydrogen Sulfide Fatalities and Thiosulfate Levels.
[So] Source:Am J Forensic Med Pathol;38(1):47-48, 2017 Mar.
[Is] ISSN:1533-404X
[Cp] País de publicação:United States
[La] Idioma:eng
[Mh] Termos MeSH primário: Acidentes de Trabalho
Poluentes Atmosféricos/envenenamento
Intoxicação por Gás/diagnóstico
Sulfeto de Hidrogênio/envenenamento
Esterco
[Mh] Termos MeSH secundário: Adulto
Animais
Seres Humanos
Masculino
Tiossulfatos/sangue
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Manure); 0 (Thiosulfates); YY9FVM7NSN (Hydrogen Sulfide)
[Em] Mês de entrada:1703
[Cu] Atualização por classe:170330
[Lr] Data última revisão:
170330
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161222
[St] Status:MEDLINE
[do] DOI:10.1097/PAF.0000000000000285


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[PMID]:27226017
[Au] Autor:Senthilkumar CS; Sah NK; Ganesh N
[Ad] Endereço:1 Department of Research, Clinical Cytogenetics Laboratory, Jawaharlal Nehru Cancer Hospital & Research Centre (JNCHRC), Bhopal, Madhya Pradesh, India.
[Ti] Título:On the long-term effects of methyl isocyanate on cell-mediated immunity in Bhopal gas-exposed long-term survivors and their offspring.
[So] Source:Toxicol Ind Health;33(4):318-331, 2017 Apr.
[Is] ISSN:1477-0393
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Methyl isocyanate (MIC) is a toxic industrial chemical that is documented as a potent respiratory toxicant. We investigated cell-mediated immunity (CMI) in the MIC-exposed long-term survivors and their offspring born after the Bhopal gas-leak tragedy in 1984. Several earlier reports show inconsistency in the assessment of immunological effects of MIC on the human population. In these studies, important factors including lifestyle attributes were overlooked. We incorporated these factors also in our study of the basic cell-mediated immune function in the Bhopal MIC-affected population. Twenty-seven years after exposure, we assessed the circulating T-lymphocyte frequency using E-Rosette assay. A total of 46 MIC-exposed healthy long-term survivors and their offspring were studied vis-a-vis parallel gender-age group-matched unexposed controls from Bhopal and various other regions of India. The influence of several lifestyle variabilities (smoking, alcohol intake, and tobacco chewing) on T-lymphocyte frequency was also taken into consideration. Our observations suggest that Erythrocyte-Rosette-forming cell (E-RFC) distribution frequency is largely insignificant in the MIC-affected population as compared to controls ( p > 0.05). In the MIC-affected tobacco chewers, there was a trend of suppression in CMI (relative decrease = 10.3%) as compared to nonchewers. Overall, our results show negligible long-term effect of MIC on CMI measured in terms of E-RFC frequency. These observations are not in agreement with earlier findings that immunosuppressive effects of MIC exposure persist in the T-cells of the affected population. However, atypical lymphocytes were frequently observed as E-RFC in the exposed females when compared to all other subgroups. Hematopoietic disorders (atypical lymphocytosis) in the MIC-affected population along with previous reports on the cytogenetic and humoral immune system linking cancer risk and chronic obstructive pulmonary disease (COPD) are important.
[Mh] Termos MeSH primário: Vazamento Acidental em Bhopal
Intoxicação por Gás/imunologia
Imunidade Celular/efeitos dos fármacos
Imunossupressores/toxicidade
Isocianatos/toxicidade
Linfocitose/etiologia
Praguicidas/toxicidade
[Mh] Termos MeSH secundário: Adolescente
Adulto
Biomarcadores/sangue
Criança
Feminino
Intoxicação por Gás/sangue
Intoxicação por Gás/fisiopatologia
Seres Humanos
Índia/epidemiologia
Exposição por Inalação/efeitos adversos
Masculino
Exposição Materna/efeitos adversos
Meia-Idade
Neoplasias/induzido quimicamente
Neoplasias/epidemiologia
Neoplasias/imunologia
Exposição Paterna/efeitos adversos
Doença Pulmonar Obstrutiva Crônica/induzido quimicamente
Doença Pulmonar Obstrutiva Crônica/epidemiologia
Doença Pulmonar Obstrutiva Crônica/imunologia
Risco
Fatores Sexuais
Sobreviventes
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Biomarkers); 0 (Immunosuppressive Agents); 0 (Isocyanates); 0 (Pesticides); C588JJ4BV9 (methyl isocyanate)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170802
[Lr] Data última revisão:
170802
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160527
[St] Status:MEDLINE
[do] DOI:10.1177/0748233716645480


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[PMID]:27456288
[Au] Autor:Padley AP
[Ad] Endereço:Senior Anaesthetist, Westmead Hospital, Westmead, NSW.
[Ti] Título:Gas: the greatest terror of the Great War.
[So] Source:Anaesth Intensive Care;44 Suppl:24-30, 2016 07.
[Is] ISSN:0310-057X
[Cp] País de publicação:Australia
[La] Idioma:eng
[Ab] Resumo:The Great War began just over a century ago and this monumental event changed the world forever. 1915 saw the emergence of gas warfare-the first weapon of mass terror. It is relevant to anaesthetists to reflect on these gases for a number of reasons. Firstly and most importantly we should acknowledge and be aware of the suffering and sacrifice of those soldiers who were injured or killed so that we could enjoy the freedoms we have today. Secondly, it is interesting to consider the overlap between poison gases and anaesthetic gases and vapors, for example that phosgene can be formed by the interaction of chloroform and sunlight. Thirdly the shadow of gas warfare is very long and covers us still. The very agents used in the Great War are still causing death and injury through deployment in conflict areas such as Iraq and Syria. Industrial accidents, train derailments and dumped or buried gas shells are other sources of poison gas hazards. In this age of terrorism, anaesthetists, as front-line resuscitation specialists, may be directly involved in the management of gas casualties or become victims ourselves.
[Mh] Termos MeSH primário: Substâncias para a Guerra Química/história
Gases/história
Militares
Guerra
I Guerra Mundial
[Mh] Termos MeSH secundário: Substâncias para a Guerra Química/envenenamento
Cloro/história
Cloro/envenenamento
Intoxicação por Gás/história
Gases/toxicidade
História do Século XX
Seres Humanos
Gás de Mostarda/história
Gás de Mostarda/envenenamento
Fosgênio/história
Fosgênio/envenenamento
[Pt] Tipo de publicação:BIOGRAPHY; HISTORICAL ARTICLE; JOURNAL ARTICLE; PORTRAITS
[Ps] Nome de pessoa como assunto:Haber F
[Nm] Nome de substância:
0 (Chemical Warfare Agents); 0 (Gases); 117K140075 (Phosgene); 4R7X1O2820 (Chlorine); T8KEC9FH9P (Mustard Gas)
[Em] Mês de entrada:1706
[Cu] Atualização por classe:170616
[Lr] Data última revisão:
170616
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160727
[St] Status:MEDLINE


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[PMID]:26766558
[Au] Autor:Harrison RJ; Retzer K; Kosnett MJ; Hodgson M; Jordan T; Ridl S; Kiefer M
[Ti] Título:Sudden Deaths Among Oil and Gas Extraction Workers Resulting from Oxygen Deficiency and Inhalation of Hydrocarbon Gases and Vapors - United States, January 2010-March 2015.
[So] Source:MMWR Morb Mortal Wkly Rep;65(1):6-9, 2016 Jan 15.
[Is] ISSN:1545-861X
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:In 2013, an occupational medicine physician from the University of California, San Francisco, contacted CDC's National Institute for Occupational Safety and Health (NIOSH), and the Occupational Safety and Health Administration (OSHA) about two oil and gas extraction worker deaths in the western United States. The suspected cause of these deaths was exposure to hydrocarbon gases and vapors (HGVs) and oxygen (O2)-deficient atmospheres after opening the hatches of hydrocarbon storage tanks. The physician and experts from NIOSH and OSHA reviewed available fatality reports from January 2010 to March 2015, and identified seven additional deaths with similar characteristics (nine total deaths). Recommendations were made to industry and regulators regarding the hazards associated with opening hatches of tanks, and controls to reduce or eliminate the potential for HGV exposure were proposed. Health care professionals who treat or evaluate oil and gas workers need to be aware that workers might report symptoms of exposure to high concentrations of HGVs and possible O2 deficiency; employers and workers need to be aware of this hazard and know how to limit exposure. Medical examiners investigating the death of oil and gas workers who open tank hatches should consider the contribution of O2 deficiency and HGV exposure.
[Mh] Termos MeSH primário: Morte Súbita Cardíaca/epidemiologia
Hidrocarbonetos/envenenamento
Exposição por Inalação/efeitos adversos
Exposição Ocupacional/efeitos adversos
Indústria de Petróleo e Gás
[Mh] Termos MeSH secundário: Adulto
Morte Súbita Cardíaca/etiologia
Intoxicação por Gás/complicações
Seres Humanos
Hidrocarbonetos/química
Hipóxia/complicações
Masculino
Meia-Idade
Estados Unidos/epidemiologia
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Hydrocarbons)
[Em] Mês de entrada:1605
[Cu] Atualização por classe:161126
[Lr] Data última revisão:
161126
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160115
[St] Status:MEDLINE
[do] DOI:10.15585/mmwr.mm6501a2


  5 / 597 MEDLINE  
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[PMID]:26724695
[Au] Autor:Chang SS; Cheng Q; Lee ES; Yip PS
[Ad] Endereço:Institute of Health Behaviors and Community Sciences, and Department of Public Health, College of Public Health, National Taiwan University, Taipei, Taiwan; Hong Kong Jockey Club Centre for Suicide Research and Prevention, The University of Hong Kong, Hong Kong; Department of Social Work and Social
[Ti] Título:Suicide by gassing in Hong Kong 2005-2013: Emerging trends and characteristics of suicide by helium inhalation.
[So] Source:J Affect Disord;192:162-6, 2016 Mar 01.
[Is] ISSN:1573-2517
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Increased use of lethal suicide methods can have a profound impact on overall suicide incidence; the epidemic of suicide by barbecue charcoal gas poisoning in some East Asian countries is a recent example. There have been concerns about recent rises in suicide using gases in some Western countries. METHODS: We investigated suicide by gassing in Hong Kong (2005-2013) using Coroner's files data. The characteristics were compared between suicide by helium inhalation, charcoal gas poisoning, and other methods. RESULTS: About one sixth (1407/8445, 16.7%) of all suicides used gases. Charcoal-burning suicides constituted the majority (97.5%) of them but showed a reduction over the 9-year period (-33%). Helium suicide was not recorded in 2005-2010 but increased from one in 2011 to three in 2012 and 11 in 2013, accounting for 1.2% of all suicides in 2013. Similar to the profile of charcoal-burning suicides, helium suicides were younger and more likely to have debt problem and less likely to receive psychiatric treatment than other suicides. Internet involvement related to the method was found in one third of cases of helium suicide. LIMITATIONS: The small number of helium suicides (n=15) limits the power to examine their characteristics. CONCLUSION: Suicide by charcoal burning showed a downward trend whilst there was an alarming increase in helium suicide in Hong Kong. Public health measures to prevent an epidemic of helium suicide similar to that of charcoal-burning suicide may include close monitoring of trend, responsible media reporting, and restricting online information about and access to this method.
[Mh] Termos MeSH primário: Carvão Vegetal/envenenamento
Intoxicação por Gás/epidemiologia
Hélio/envenenamento
Suicídio/tendências
[Mh] Termos MeSH secundário: Adolescente
Adulto
Idoso
Idoso de 80 Anos ou mais
Criança
Feminino
Intoxicação por Gás/psicologia
Hong Kong/epidemiologia
Seres Humanos
Incidência
Masculino
Meia-Idade
Suicídio/psicologia
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
16291-96-6 (Charcoal); 206GF3GB41 (Helium)
[Em] Mês de entrada:1609
[Cu] Atualização por classe:160125
[Lr] Data última revisão:
160125
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160103
[St] Status:MEDLINE


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[PMID]:26975073
[Au] Autor:Moriya F
[Ti] Título:[Poisoning due to carbon monoxide and cyanide gas generated in the occurrence of fire].
[So] Source:Chudoku Kenkyu;28(4):339-45, 2015 Dec.
[Is] ISSN:0914-3777
[Cp] País de publicação:Japan
[La] Idioma:jpn
[Mh] Termos MeSH primário: Intoxicação por Monóxido de Carbono
Cianetos/envenenamento
Fogo
Medicina Legal
Intoxicação por Gás
[Mh] Termos MeSH secundário: Autopsia
Monóxido de Carbono/sangue
Intoxicação por Monóxido de Carbono/sangue
Intoxicação por Monóxido de Carbono/patologia
Carboxihemoglobina
Cianetos/sangue
Intoxicação por Gás/sangue
Intoxicação por Gás/patologia
Hemoglobinas
Seres Humanos
Exposição por Inalação
Metemoglobinemia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Cyanides); 0 (Hemoglobins); 7U1EE4V452 (Carbon Monoxide); 9061-29-4 (Carboxyhemoglobin)
[Em] Mês de entrada:1604
[Cu] Atualização por classe:160315
[Lr] Data última revisão:
160315
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160316
[St] Status:MEDLINE


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[PMID]:26207672
[Au] Autor:Perkins MW; Wong B; Rodriguez A; Devorak J; Sciuto AM
[Ad] Endereço:a Analytical Toxicology Division , US Army Medical Research Institute of Chemical Defense , Aberdeen Proving Ground , MD , USA.
[Ti] Título:Measurement of various respiratory dynamics parameters following acute inhalational exposure to soman vapor in conscious rats.
[So] Source:Inhal Toxicol;27(9):432-9, 2015.
[Is] ISSN:1091-7691
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Respiratory dynamics were investigated in head-out plethysmography chambers following inhalational exposure to soman in untreated, non-anesthetized rats. A multipass saturator cell was used to generate 520, 560 and 600 mg × min/m(3) of soman vapor in a customized inhalational exposure system. Various respiratory dynamic parameters were collected from male Sprague-Dawley rats (300--350 g) during (20 min) and 24 h (10 min) after inhalational exposure. Signs of CWNA-induced cholinergic crisis were observed in all soman-exposed animals. Percentage body weight loss and lung edema were observed in all soman-exposed animals, with significant increases in both at 24 h following exposure to 600 mg × min/m(3). Exposure to soman resulted in increases in respiratory frequency (RF) in animals exposed to 560 and 600 mg × min/m(3) with significant increases following exposure to 560 mg × min/m(3) at 24 h. No significant alterations in inspiratory time (IT) or expiratory time (ET) were observed in soman-exposed animals 24 h post-exposure. Prominent increases in tidal volume (TV) and minute volume (MV) were observed at 24 h post-exposure in animals exposed to 600 mg × min/m(3). Peak inspiratory (PIF) and expiratory flow (PEF) followed similar patterns and increased 24 h post-exposure to 600 mg × min/m(3) of soman. Results demonstrate that inhalational exposure to 600 mg × min/m(3) soman produces notable alterations in various respiratory dynamic parameters at 24 h. The following multitude of physiological changes in respiratory dynamics highlights the need to develop countermeasures that protect against respiratory toxicity and lung injury.
[Mh] Termos MeSH primário: Inibidores da Colinesterase/toxicidade
Intoxicação por Gás/fisiopatologia
Agentes Neurotóxicos/toxicidade
Intoxicação por Organofosfatos/fisiopatologia
Mucosa Respiratória/efeitos dos fármacos
Sistema Respiratório/efeitos dos fármacos
Soman/toxicidade
[Mh] Termos MeSH secundário: Administração por Inalação
Animais
Biomarcadores
Fluorcarbonetos/administração & dosagem
Fluorcarbonetos/efeitos adversos
Masculino
Veículos Farmacêuticos/administração & dosagem
Veículos Farmacêuticos/efeitos adversos
Edema Pulmonar/etiologia
Ventilação Pulmonar/efeitos dos fármacos
Ratos Sprague-Dawley
Mucosa Respiratória/fisiopatologia
Mucosa Respiratória/secreção
Taxa Respiratória/efeitos dos fármacos
Sistema Respiratório/fisiopatologia
Rinite/etiologia
Convulsões/etiologia
Volume de Ventilação Pulmonar/efeitos dos fármacos
Volatilização
Perda de Peso/efeitos dos fármacos
[Pt] Tipo de publicação:COMPARATIVE STUDY; JOURNAL ARTICLE; RESEARCH SUPPORT, U.S. GOV'T, NON-P.H.S.
[Nm] Nome de substância:
0 (Biomarkers); 0 (Cholinesterase Inhibitors); 0 (Fluorocarbons); 0 (Nerve Agents); 0 (Pharmaceutical Vehicles); 96-64-0 (Soman); FX3WJ41CMX (perflexane)
[Em] Mês de entrada:1606
[Cu] Atualização por classe:150908
[Lr] Data última revisão:
150908
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150725
[St] Status:MEDLINE
[do] DOI:10.3109/08958378.2015.1068890


  8 / 597 MEDLINE  
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[PMID]:26008669
[Au] Autor:Akhtar S; Rehman A; Bano S; Haque A
[Ad] Endereço:Department of Pediatrics and Child Health, The Aga Khan University Hospital, Karachi.
[Ti] Título:Accidental phosphine gas poisoning with fatal myocardial dysfunction in two families.
[So] Source:J Coll Physicians Surg Pak;25(5):378-9, 2015 May.
[Is] ISSN:1681-7168
[Cp] País de publicação:Pakistan
[La] Idioma:eng
[Ab] Resumo:Aluminum phosphide is commonly used as a rodenticide and insecticide and is one of the most fatal poisons. The active ingredient is Phosphine gas which inhibits cytochrome oxidase and cellular oxygen utilization. The clinical symptoms are due to multiorgan involvement including cardiac toxicity which is the most common cause of mortality. Severity of clinical manifestations depends upon the amount of the gas to which a person is exposed. There is no specific antidote available. High index of suspicion and early aggressive treatment is the key to success. We report 2 cases of aluminum phosphide toxicity in 2 families due to incidental exposure after fumigation.
[Mh] Termos MeSH primário: Compostos de Alumínio/envenenamento
Cardiomiopatias/induzido quimicamente
Intoxicação por Gás/mortalidade
Inseticidas/envenenamento
Fosfinas/envenenamento
[Mh] Termos MeSH secundário: Criança
Pré-Escolar
Feminino
Seres Humanos
Masculino
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Aluminum Compounds); 0 (Insecticides); 0 (Phosphines); E23DR6L59S (aluminum phosphide); FW6947296I (phosphine)
[Em] Mês de entrada:1607
[Cu] Atualização por classe:161125
[Lr] Data última revisão:
161125
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150527
[St] Status:MEDLINE
[do] DOI:05.2015/JCPSP.378379


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[PMID]:25916033
[Au] Autor:Basharin VA; Grebeniuk AN; Markizova NF; Preobrazhenskaia TN; Sarmanaev SKh; Tolkach PG
[Ti] Título:[Chemicals as fire damaging factor].
[So] Source:Voen Med Zh;336(1):22-8, 2015 Jan.
[Is] ISSN:0026-9050
[Cp] País de publicação:Russia (Federation)
[La] Idioma:rus
[Ab] Resumo:The article provides an overview of published scientific data about toxic chemical compounds formed during thermal degradation of various materials. In case of fire the complex of physical and chemical factors affect the human, along with injuries, thermal burns of the skin and respiratory tract there is a lack of oxygen in the inspired air and the impact of thermal degradation products. The greatest number of deaths in.a fire due to the inhalation by the victims smoke and toxic gases. The impact of the combination of toxic substances leads to the development of various forms of toxic process. The main causes of poisoning at the fires due to the effects of toxic substances and substances which can cause structural and functional disorders of the respiratory organ. Intoxication manifestations by some of them appear already in the fire zone, in other cases, in cases of poisoning by the compounds of the slow motion, there is the latent period of of intoxication. Knowledge of the spectrum of toxic products thermal destruction on the human during the fire, it is necessary to develop approaches to improve medical care and creation of tools of medical protection.
[Mh] Termos MeSH primário: Fogo
Intoxicação por Gás/etiologia
Gases
Lesão por Inalação de Fumaça/etiologia
[Mh] Termos MeSH secundário: Poluição do Ar em Ambientes Fechados/efeitos adversos
Poluição do Ar em Ambientes Fechados/análise
Intoxicação por Gás/diagnóstico
Gases/química
Gases/toxicidade
Temperatura Alta
Seres Humanos
Lesão por Inalação de Fumaça/diagnóstico
[Pt] Tipo de publicação:ENGLISH ABSTRACT; JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Gases)
[Em] Mês de entrada:1505
[Cu] Atualização por classe:150428
[Lr] Data última revisão:
150428
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150429
[St] Status:MEDLINE


  10 / 597 MEDLINE  
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[PMID]:25910669
[Au] Autor:Merin O; Fink D; Fink DL; Shahroor S; Schlesinger Y; Amir G; Birk E
[Ad] Endereço:Department of Cardiothoracic Surgery, Shaare Zedek Medical Center, Jerusalem, Israel 91031.
[Ti] Título:Salvage ECMO deployment for fatal aluminum phosphide poisoning.
[So] Source:Am J Emerg Med;33(11):1718.e1-3, 2015 Nov.
[Is] ISSN:1532-8171
[Cp] País de publicação:United States
[La] Idioma:eng
[Mh] Termos MeSH primário: Compostos de Alumínio/envenenamento
Oxigenação por Membrana Extracorpórea
Fumigação/efeitos adversos
Intoxicação por Gás/terapia
Praguicidas/envenenamento
Fosfinas/envenenamento
[Mh] Termos MeSH secundário: Adulto
Criança
Pré-Escolar
Evolução Fatal
Feminino
Intoxicação por Gás/diagnóstico
Intoxicação por Gás/etiologia
Seres Humanos
Lactente
Masculino
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Aluminum Compounds); 0 (Pesticides); 0 (Phosphines); E23DR6L59S (aluminum phosphide)
[Em] Mês de entrada:1602
[Cu] Atualização por classe:151030
[Lr] Data última revisão:
151030
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:150426
[St] Status:MEDLINE



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