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  1 / 4017 MEDLINE  
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[PMID]:29212061
[Au] Autor:Yin P; Chen R; Wang L; Meng X; Liu C; Niu Y; Lin Z; Liu Y; Liu J; Qi J; You J; Zhou M; Kan H
[Ad] Endereço:National Center for Chronic and Noncommunicable Disease Control and Prevention, Chinese Center for Disease Control and Prevention, Beijing, China.
[Ti] Título:Ambient Ozone Pollution and Daily Mortality: A Nationwide Study in 272 Chinese Cities.
[So] Source:Environ Health Perspect;125(11):117006, 2017 11 21.
[Is] ISSN:1552-9924
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Few large multicity studies have been conducted in developing countries to address the acute health effects of atmospheric ozone pollution. OBJECTIVE: We explored the associations between ozone and daily cause-specific mortality in China. METHODS: We performed a nationwide time-series analysis in 272 representative Chinese cities between 2013 and 2015. We used distributed lag models and over-dispersed generalized linear models to estimate the cumulative effects of ozone (lagged over 0-3 d) on mortality in each city, and we used hierarchical Bayesian models to combine the city-specific estimates. Regional, seasonal, and demographic heterogeneity were evaluated by meta-regression. RESULTS: At the national-average level, a 10-µg/m increase in 8-h maximum ozone concentration was associated with 0.24% [95% posterior interval (PI): 0.13%, 0.35%], 0.27% (95% PI: 0.10%, 0.44%), 0.60% (95% PI: 0.08%, 1.11%), 0.24% (95% PI: 0.02%, 0.46%), and 0.29% (95% PI: 0.07%, 0.50%) higher daily mortality from all nonaccidental causes, cardiovascular diseases, hypertension, coronary diseases, and stroke, respectively. Associations between ozone and daily mortality due to respiratory and chronic obstructive pulmonary disease specifically were positive but imprecise and nonsignificant. There were no statistically significant differences in associations between ozone and nonaccidental mortality according to region, season, age, sex, or educational attainment. CONCLUSIONS: Our findings provide robust evidence of higher nonaccidental and cardiovascular mortality in association with short-term exposure to ambient ozone in China. https://doi.org/10.1289/EHP1849.
[Mh] Termos MeSH primário: Poluição do Ar/estatística & dados numéricos
Exposição Ambiental/estatística & dados numéricos
Mortalidade/tendências
Ozônio
[Mh] Termos MeSH secundário: Poluentes Atmosféricos/análise
Doenças Cardiovasculares/mortalidade
China
Seres Humanos
Dióxido de Nitrogênio/análise
Material Particulado/análise
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Particulate Matter); 66H7ZZK23N (Ozone); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1801
[Cu] Atualização por classe:180206
[Lr] Data última revisão:
180206
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171207
[St] Status:MEDLINE
[do] DOI:10.1289/EHP1849


  2 / 4017 MEDLINE  
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[PMID]:28963089
[Au] Autor:Doiron D; de Hoogh K; Probst-Hensch N; Mbatchou S; Eeftens M; Cai Y; Schindler C; Fortier I; Hodgson S; Gaye A; Stolk R; Hansell A
[Ad] Endereço:Research Institute of the McGill University Health Centre , Montreal, Quebec, Canada.
[Ti] Título:Residential Air Pollution and Associations with Wheeze and Shortness of Breath in Adults: A Combined Analysis of Cross-Sectional Data from Two Large European Cohorts.
[So] Source:Environ Health Perspect;125(9):097025, 2017 Sep 29.
[Is] ISSN:1552-9924
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Research examining associations between air pollution exposure and respiratory symptoms in adults has generally been inconclusive. This may be related in part to sample size issues, which also preclude analysis in potentially vulnerable subgroups. OBJECTIVES: We estimated associations between air pollution exposures and the prevalence of wheeze and shortness of breath using harmonized baseline data from two very large European cohorts, Lifelines (2006-2013) and UK Biobank (2006-2010). Our aim was also to determine whether the relationship between air pollution and respiratory symptom prevalence differed between individuals with different characteristics. METHODS: Cross-sectional analyses explored associations between prevalence of self-reported wheeze and shortness of breath and annual mean particulate matter with aerodynamic diameter <2.5µm, 2.5-10µm, and <10µm (PM2.5, PMcoarse, and PM10, respectively) and nitrogen dioxide (NO2) concentrations at place of residence using logistic regression. Subgroup analyses and tests for interaction were performed for age, sex, smoking status, household income, obesity status, and asthma status. RESULTS: All PM exposures were associated with respiratory symptoms based on single-pollutant models, with the largest associations seen for PM2.5 with prevalence of wheezing {odds ratio (OR)=1.16 per 5µg/m³ [95% confidence interval (CI): 1.11, 1.21]} and shortness of breath [OR=1.61 per 5µg/m³ (95% CI: 1.45, 1.78)]. The association between shortness of breath and a 5-µg/m³ increment in PM2.5 was significantly higher for individuals from lower-[OR=1.73 (95% CI: 1.52, 1.97)] versus higher-income households [OR=1.31 (95% CI: 1.11, 1.55); p-interaction=0.005), whereas the association between PM2.5 and wheeze was limited to lower-income participants [OR=1.30 (95% CI: 1.22, 1.38) vs. OR=1.02; (95% CI: 0.96, 1.08); p-interaction<0.001]. Exposure to NO2 also showed positive associations with wheeze and shortness of breath. CONCLUSION: Exposure to PM and NO2 air pollution was associated with the prevalence of wheeze and shortness of breath in this large study, with stronger associations between PM2.5 and both outcomes among lower- versus higher-income participants. https://doi.org/10.1289/EHP1353.
[Mh] Termos MeSH primário: Poluição do Ar/estatística & dados numéricos
Dispneia/epidemiologia
Exposição Ambiental/estatística & dados numéricos
Sons Respiratórios
[Mh] Termos MeSH secundário: Adulto
Poluentes Atmosféricos
Estudos Transversais
Europa (Continente)/epidemiologia
Feminino
Seres Humanos
Modelos Logísticos
Masculino
Dióxido de Nitrogênio/análise
Prevalência
Fatores de Risco
Emissões de Veículos/análise
Adulto Jovem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Vehicle Emissions); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171027
[Lr] Data última revisão:
171027
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171001
[St] Status:MEDLINE
[do] DOI:10.1289/EHP1353


  3 / 4017 MEDLINE  
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[PMID]:28953453
[Au] Autor:Sørensen M; Wendelboe Nielsen O; Sajadieh A; Ketzel M; Tjønneland A; Overvad K; Raaschou-Nielsen O
[Ad] Endereço:Diet, Genes and Environment, Danish Cancer Society Research Center , Copenhagen, Denmark.
[Ti] Título:Long-Term Exposure to Road Traffic Noise and Nitrogen Dioxide and Risk of Heart Failure: A Cohort Study.
[So] Source:Environ Health Perspect;125(9):097021, 2017 Sep 26.
[Is] ISSN:1552-9924
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Although air pollution and road traffic noise have been associated with higher risk of cardiovascular diseases, associations with heart failure have received only little attention. OBJECTIVES: We aimed to investigate whether long-term exposure to road traffic noise and nitrogen dioxide (NO2) were associated with incident heart failure. METHODS: In a cohort of 57,053 people 50-64 y of age at enrollment in the period 1993-1997, we identified 2,550 cases of first-ever hospital admission for heart failure during a mean follow-up time of 13.4 y. Present and historical residential addresses from 1987 to 2011 were found in national registers, and road traffic noise (Lden) and NO2 were modeled for all addresses. Analyses were done using Cox proportional hazard model. RESULTS: An interquartile range higher 10-y time-weighted mean exposure for Lden and NO2 was associated with incidence rate ratios (IRR) for heart failure of 1.14 (1.08-1.21) and 1.11 (1.07-1.16), respectively, in models adjusted for gender, lifestyle, and socioeconomic status. In models with mutual exposure adjustment, IRRs were 1.08 (1.00-1.16) for Lden and 1.07 (1.01-1.14) for NO2. We found statistically significant modification of the NO2-heart failure association by gender (strongest association among men), baseline hypertension (strongest association among hypertensive), and diabetes (strongest association among diabetics). The same tendencies were seen for noise, but interactions were not statistically significant. CONCLUSIONS: Long-term exposure to NO2 and road traffic noise was associated with higher risk of heart failure, mainly among men, in both single- and two-pollutant models. High exposure to both pollutants was associated with highest risk. https://doi.org/10.1289/EHP1272.
[Mh] Termos MeSH primário: Poluentes Atmosféricos/análise
Poluição do Ar/estatística & dados numéricos
Exposição Ambiental/estatística & dados numéricos
Insuficiência Cardíaca/epidemiologia
Dióxido de Nitrogênio/análise
Ruído dos Transportes/estatística & dados numéricos
[Mh] Termos MeSH secundário: Idoso
Estudos de Coortes
Feminino
Seres Humanos
Masculino
Meia-Idade
Modelos de Riscos Proporcionais
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171027
[Lr] Data última revisão:
171027
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170928
[St] Status:MEDLINE
[do] DOI:10.1289/EHP1272


  4 / 4017 MEDLINE  
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[PMID]:28935613
[Au] Autor:Yue H; Yan W; Ji X; Gao R; Ma J; Rao Z; Li G; Sang N
[Ad] Endereço:College of Environment and Resource, Research Center of Environment and Health, Shanxi University , Taiyuan, Shanxi, People's Republic of China.
[Ti] Título:Maternal Exposure of BALB/c Mice to Indoor NO2 and Allergic Asthma Syndrome in Offspring at Adulthood with Evaluation of DNA Methylation Associated Th2 Polarization.
[So] Source:Environ Health Perspect;125(9):097011, 2017 Sep 13.
[Is] ISSN:1552-9924
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Fetal stress has been proposed to be associated with diseases in both children and adults. Epidemiological studies suggest that maternal exposure to nitrogen dioxide (NO2) contributes to increased morbidity and mortality of offspring with allergic asthma later in life. OBJECTIVES: We aimed to test whether maternal NO2 exposure causes allergic asthma-related consequences in offspring absent any subsequent lung provocation and whether this exposure enhances the likelihood of developing allergic asthma or the intensity of developed allergic airway disease following postnatal allergic sensitization and challenge. In addition, if such consequences and enhancements occurred, we sought to determine the mechanism(s) of these responses. METHODS: Pregnant BALB/c mice were exposed to either NO2 (2.5 ppm, 5 h/day) or air daily throughout the gestation period. Offspring were sacrificed on postnatal days (PNDs) 1, 7, 14, 21, and 42, and remaining offspring were sensitized by ovalbumin (OVA) injection followed by OVA aerosol challenge during postnatal wk 7-9. We analyzed the lung histopathology, inflammatory cell infiltration, airway hyper-responsiveness (AHR), immune responses, and gene methylation under different treatment conditions. RESULTS: Maternal exposure to NO2 caused a striking increase in inflammatory cell infiltration and the release of type 2 cytokines in the lungs of offspring at PNDs 1 and 7; however, these alterations were reversed during postnatal development. Following OVA sensitization and challenge, the exposure enhanced the levels of allergic asthma-characterized OVA-immunoglobulin (Ig) E, AHR, and airway inflammation in adult offspring. Importantly, differentiation of T-helper (Th) 2 cells and demethylation of the interleukin-4 ( gene occurred during the process. CONCLUSIONS: Maternal exposure to indoor environmental NO2 causes allergic asthma-related consequences in offspring absent any subsequent lung provocation and potentiates the symptoms of allergic asthma in adult offspring following postnatal allergic sensitization and challenge; this response is associated with the Th2-based immune response and DNA methylation of the gene. https://doi.org/10.1289/EHP685.
[Mh] Termos MeSH primário: Poluentes Atmosféricos/análise
Asma/imunologia
Metilação de DNA
Exposição Materna/estatística & dados numéricos
Dióxido de Nitrogênio/análise
Células Th2/fisiologia
[Mh] Termos MeSH secundário: Alérgenos/imunologia
Animais
Modelos Animais de Doenças
Feminino
Camundongos
Camundongos Endogâmicos BALB C
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Allergens); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171020
[Lr] Data última revisão:
171020
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170923
[St] Status:MEDLINE
[do] DOI:10.1289/EHP685


  5 / 4017 MEDLINE  
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[PMID]:28930515
[Au] Autor:Clark LP; Millet DB; Marshall JD
[Ad] Endereço:Department of Civil, Environmental, and Geo-Engineering, University of Minnesota , Minneapolis, Minnesota, USA.
[Ti] Título:Changes in Transportation-Related Air Pollution Exposures by Race-Ethnicity and Socioeconomic Status: Outdoor Nitrogen Dioxide in the United States in 2000 and 2010.
[So] Source:Environ Health Perspect;125(9):097012, 2017 Sep 14.
[Is] ISSN:1552-9924
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Disparities in exposure to air pollution by race-ethnicity and by socioeconomic status have been documented in the United States, but the impacts of declining transportation-related air pollutant emissions on disparities in exposure have not been studied in detail. OBJECTIVE: This study was designed to estimate changes over time (2000 to 2010) in disparities in exposure to outdoor concentrations of a transportation-related air pollutant, nitrogen dioxide (NO2), in the United States. METHODS: We combined annual average NO2 concentration estimates from a temporal land use regression model with Census demographic data to estimate outdoor exposures by race-ethnicity, socioeconomic characteristics (income, age, education), and by location (region, state, county, urban area) for the contiguous United States in 2000 and 2010. RESULTS: Estimated annual average NO2 concentrations decreased from 2000 to 2010 for all of the race-ethnicity and socioeconomic status groups, including a decrease from 17.6 ppb to 10.7 ppb (-6.9 ppb) in nonwhite [non-(white alone, non-Hispanic)] populations, and 12.6 ppb to 7.8 ppb (-4.7 ppb) in white (white alone, non-Hispanic) populations. In 2000 and 2010, disparities in NO2 concentrations were larger by race-ethnicity than by income. Although the national nonwhite-white mean NO2 concentration disparity decreased from a difference of 5.0 ppb in 2000 to 2.9 ppb in 2010, estimated mean NO2 concentrations remained 37% higher for nonwhites than whites in 2010 (40% higher in 2000), and nonwhites were 2.5 times more likely than whites to live in a block group with an average NO2 concentration above the WHO annual guideline in 2010 (3.0 times more likely in 2000). CONCLUSIONS: Findings suggest that absolute NO2 exposure disparities by race-ethnicity decreased from 2000 to 2010, but relative NO2 exposure disparities persisted, with higher NO2 concentrations for nonwhites than whites in 2010. https://doi.org/10.1289/EHP959.
[Mh] Termos MeSH primário: Poluentes Atmosféricos/análise
Poluição do Ar/estatística & dados numéricos
Exposição Ambiental/estatística & dados numéricos
Dióxido de Nitrogênio/análise
Emissões de Veículos/análise
[Mh] Termos MeSH secundário: Adulto
Grupos de Populações Continentais
Demografia
Grupos Étnicos
Feminino
Seres Humanos
Masculino
Classe Social
Transportes
Estados Unidos
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Vehicle Emissions); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171020
[Lr] Data última revisão:
171020
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170921
[St] Status:MEDLINE
[do] DOI:10.1289/EHP959


  6 / 4017 MEDLINE  
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[PMID]:28888242
[Au] Autor:Saini R; Taneja A; Singh P
[Ad] Endereço:Department of Chemistry, Dr. Bhim Rao Ambedkar University, Agra 282002, India. Electronic address: renukasaini@gmail.com.
[Ti] Título:Surface ozone scenario and air quality in the north-central part of India.
[So] Source:J Environ Sci (China);59:72-79, 2017 Sep.
[Is] ISSN:1001-0742
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Tropospheric pollutants including surface ozone (O ), nitrogen dioxide (NO ), carbon monoxide (CO) and meteorological parameters were measured at a traffic junction (78°2' E and 27°11' N) in Agra, India from January 2012 to December 2012. Temporal analysis of pollutants suggests that annual average mixing ratios of tropospheric pollutants were: O - 22.97±23.36ppbV, NO - 19.84±16.71ppbV and CO - 0.91±0.86ppmV, with seasonal variations of O having maximum mixing ratio during summer season (32.41±19.31ppbV), whereas lowest was found in post-monsoon season (8.74±3.8ppbV). O precursors: NO and CO, showed inverse relationship with O . Seasonal variation and high O episodes during summer are associated with meteorological parameters such as high solar radiation, atmospheric temperature and transboundary transport. The interdependence of these variables showed a link between the daytime mixing ratios of O with the nighttime level of NO . The mixing ratios of CO and NO showed tight correlations, which confirms the influence of vehicular emissions combined with other anthropogenic activities due to office/working hours, shallowing, and widening of boundary layer. FLEXTRA backward trajectories for the O episode days clearly indicate the transport from the NW and W to S/SE and SW direction at Agra in different seasons.
[Mh] Termos MeSH primário: Poluentes Atmosféricos/análise
Monitoramento Ambiental
Ozônio/análise
[Mh] Termos MeSH secundário: Poluição do Ar/estatística & dados numéricos
Monóxido de Carbono/análise
Índia
Dióxido de Nitrogênio/análise
Temperatura Ambiente
Emissões de Veículos/análise
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Vehicle Emissions); 66H7ZZK23N (Ozone); 7U1EE4V452 (Carbon Monoxide); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170915
[Lr] Data última revisão:
170915
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170911
[St] Status:MEDLINE


  7 / 4017 MEDLINE  
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[PMID]:28888198
[Au] Autor:Costa AF; Hoek G; Brunekreef B; Ponce de Leon ACM
[Ad] Endereço:Institute of Social Medicine, Rio de Janeiro State University, Rio de Janeiro, Brazil; Brazilian National Cancer Institute, Rio de Janeiro, Brazil. Electronic address: acosta@inca.gov.br.
[Ti] Título:Effects of NO exposure on daily mortality in São Paulo, Brazil.
[So] Source:Environ Res;159:539-544, 2017 11.
[Is] ISSN:1096-0953
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Recent reports have suggested that air pollution mixtures represented by nitrogen dioxide (NO ) may have effects on human health, which are independent from those of particulate matter mass. We evaluate the association between NO and daily mortality among elderly using one- and multipollutant models. METHODS: This study was a daily time series of non-accidental and cause-specific mortality among the elderly living in São Paulo, Brazil, between 2000 and 2011. Effects of NO , particulate matter smaller than 10µm (PM10), carbon monoxide (CO) and ozone (O ) were estimated in Poisson generalized additive models. The single lag effect at lags 0 and 1 days and the cumulative effect from 0 to lag 10 days were evaluated in one-, two-, three- and four-pollutant models. The cumulative risk index (CRI) recently proposed to analyze associations with health of multiple correlated pollutants was additionally estimated for each multipollutant model. RESULTS: An association between NO , PM10, CO and O exposures and non-accidental and cause-specific deaths was found in one-pollutant models. NO effects remained significant in multipollutant models for non-accidental and circulatory deaths. The estimated CRIs suggested that circulatory deaths were mainly associated with NO , and respiratory deaths mainly with CO and O , regardless the lag. For non-accidental deaths, multipollutant models were associated with the highest CRI, with the main pollutants depending on the chosen lag. CONCLUSIONS: The results suggest that air pollution mixtures represented by NO have an effect on non-accidental and circulatory mortality, which is independent from PM10, CO and O . The CRI was always larger than the risks associated with single pollutants.
[Mh] Termos MeSH primário: Poluentes Atmosféricos/toxicidade
Doenças Cardiovasculares/mortalidade
Exposição Ambiental
Dióxido de Nitrogênio/toxicidade
[Mh] Termos MeSH secundário: Idoso
Idoso de 80 Anos ou mais
Brasil/epidemiologia
Monóxido de Carbono/análise
Doenças Cardiovasculares/induzido quimicamente
Feminino
Seres Humanos
Masculino
Meia-Idade
Ozônio/análise
Tamanho da Partícula
Material Particulado/análise
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Particulate Matter); 66H7ZZK23N (Ozone); 7U1EE4V452 (Carbon Monoxide); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171120
[Lr] Data última revisão:
171120
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170910
[St] Status:MEDLINE


  8 / 4017 MEDLINE  
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[PMID]:28886601
[Au] Autor:Turner MC; Krewski D; Diver WR; Pope CA; Burnett RT; Jerrett M; Marshall JD; Gapstur SM
[Ad] Endereço:McLaughlin Centre for Population Health Risk Assessment, University of Ottawa , Ottawa, Canada.
[Ti] Título:Ambient Air Pollution and Cancer Mortality in the Cancer Prevention Study II.
[So] Source:Environ Health Perspect;125(8):087013, 2017 08 21.
[Is] ISSN:1552-9924
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: The International Agency for Research on Cancer classified both outdoor air pollution and airborne particulate matter as carcinogenic to humans (Group 1) for lung cancer. There may be associations with cancer at other sites; however, the epidemiological evidence is limited. OBJECTIVE: The aim of this study was to clarify whether ambient air pollution is associated with specific types of cancer other than lung cancer by examining associations of ambient air pollution with nonlung cancer death in the Cancer Prevention Study II (CPS-II). METHODS: Analysis included 623,048 CPS-II participants who were followed for 22 y (1982-2004). Modeled estimates of particulate matter with aerodynamic diameter <2.5µm (PM ) (1999-2004), nitrogen dioxide (NO ) (2006), and ozone (O ) (2002-2004) concentrations were linked to the participant residence at enrollment. Cox proportional hazards models were used to estimate associations per each fifth percentile-mean increment with cancer mortality at 29 anatomic sites, adjusted for individual and ecological covariates. RESULTS: We observed 43,320 nonlung cancer deaths. PM was significantly positively associated with death from cancers of the kidney {adjusted hazard ratio (HR) per 4.4 µg/m =1.14 [95% confidence interval (CI): 1.03, 1.27]} and bladder [HR=1.13 (95% CI: 1.03, 1.23)]. NO was positively associated with colorectal cancer mortality [HR per 6.5 ppb=1.06 (95% CI: 1.02, 1.10). The results were similar in two-pollutant models including PM and NO and in three-pollutant models with O . We observed no statistically significant positive associations with death from other types of cancer based on results from adjusted models. CONCLUSIONS: The results from this large prospective study suggest that ambient air pollution was not associated with death from most nonlung cancers, but associations with kidney, bladder, and colorectal cancer death warrant further investigation. https://doi.org/10.1289/EHP1249.
[Mh] Termos MeSH primário: Poluição do Ar/estatística & dados numéricos
Exposição Ambiental/estatística & dados numéricos
Neoplasias/mortalidade
[Mh] Termos MeSH secundário: Adulto
Poluentes Atmosféricos/análise
Seres Humanos
Dióxido de Nitrogênio/análise
Ozônio
Material Particulado/análise
Modelos de Riscos Proporcionais
Fatores de Tempo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Air Pollutants); 0 (Particulate Matter); 66H7ZZK23N (Ozone); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171109
[Lr] Data última revisão:
171109
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170909
[St] Status:MEDLINE
[do] DOI:10.1289/EHP1249


  9 / 4017 MEDLINE  
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[PMID]:28811146
[Au] Autor:Li W; Sun Y; Bian R; Wang H; Zhang D
[Ad] Endereço:School of Environmental and Municipal Engineering, Qingdao University of Technology, Qingdao 266033, PR China.
[Ti] Título:N O emissions from an intermittently aerated semi-aerobic aged refuse bioreactor: Combined effect of COD and NH -N in influent leachate.
[So] Source:Waste Manag;69:242-249, 2017 Nov.
[Is] ISSN:1879-2456
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The carbon-nitrogen ratio (COD/NH -N) is an important factor affecting nitrification and denitrification in wastewater treatment; this factor also influences nitrous oxide (N O) emissions. This study investigated two simulated intermittently aerated semi-aerobic aged refuse bioreactors (SAARB) filled with 8-year old aged refuse (AR). The research analyzed how differences in and the combination of influent COD and NH -N impact N O emissions in leachate treatment. Experimental results showed that N O emissions increased as the influent COD/NH -N decreased. The influent COD had a greater effect on N O emissions than NH -N at the same influent ratios of COD/NH -N (2.7 and 8.0, respectively). The maximum N O emission accounted for 8.82±2.65% of the total nitrogen removed from the influent leachate; the maximum level occurred when the COD was 2000mg/L. An analysis of differences in influent carbon sources at the same COD/NH -N ratios concluded that the availability of biodegradable carbon substrates (i.e. glucose) is an important factor affecting N O emissions. At a low influent COD/NH -N ratio (2.7), the N O conversion rate was greater when there were more biodegradable carbon substrates. Although the SAARB included the N O generation and reduction processes, N O reduction mainly occurred later in the process, after leachate recirculation. The maximum N O emission rate occurred in the first hour of single-period (24h) experiments, as leachate contacted the surface AR. In practical SAARB applications, N O emissions may be reduced by measures such as reducing the initial recirculation loading of NH -N substrates, adding a later supplement of biodegradable carbon substrates, and/or prolonging hydraulic retention time (HRT) of influent leachate.
[Mh] Termos MeSH primário: Análise da Demanda Biológica de Oxigênio
Reatores Biológicos/microbiologia
Dióxido de Nitrogênio/análise
Eliminação de Resíduos Líquidos/métodos
Poluentes Químicos da Água/análise
[Mh] Termos MeSH secundário: Aerobiose
Amônia
Desnitrificação
Águas Residuais
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Waste Water); 0 (Water Pollutants, Chemical); 7664-41-7 (Ammonia); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171108
[Lr] Data última revisão:
171108
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170817
[St] Status:MEDLINE


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[PMID]:28797886
[Au] Autor:Paulin LM; Williams D'L; Peng R; Diette GB; McCormack MC; Breysse P; Hansel NN
[Ad] Endereço:Johns Hopkins Pulmonary/Critical Care, Baltimore, MD, United States. Electronic address: Lpaulin1@jhmi.edu.
[Ti] Título:24-h Nitrogen dioxide concentration is associated with cooking behaviors and an increase in rescue medication use in children with asthma.
[So] Source:Environ Res;159:118-123, 2017 11.
[Is] ISSN:1096-0953
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Ab] Resumo:Exposure to nitrogen dioxide (NO ), a byproduct of combustion, is associated with poor asthma control in children. We sought to determine whether gas-fueled kitchen appliance use is associated with 24-h indoor NO concentrations and whether these concentrations are associated with asthma morbidity in children. Children aged 5-12 years old with asthma were eligible. Mean 24-h NO concentration was measured in the kitchen over a four-day sampling period and gas stove use was captured in time activity diaries. The relationship between stove and oven use and daily NO concentration was analyzed. Longitudinal analysis assessed the effect of daily NO exposure on symptoms, inhaler use, and lung function. Multivariate models were adjusted for age, sex, season, and maternal education. Thirty children contributed 126 participant days of sampling. Mean indoor 24-h NO concentration was 58(48)ppb with a median (range) of 45(12-276)ppb. All homes had gas stoves and furnaces. Each hour of kitchen appliance use was associated with an 18ppb increase in 24-h NO concentration. In longitudinal multivariate analysis, each ten-fold increase in previous-day NO was associated with increased nighttime inhaler use (OR = 4.9, p = 0.04). There were no associations between NO and lung function or asthma symptoms. Higher previous-day 24-h concentration of NO is associated with increased nighttime inhaler use in children with asthma.
[Mh] Termos MeSH primário: Poluentes Atmosféricos/efeitos adversos
Poluição do Ar em Ambientes Fechados/efeitos adversos
Asma/epidemiologia
Culinária/métodos
Exposição Ambiental
Dióxido de Nitrogênio/efeitos adversos
[Mh] Termos MeSH secundário: Asma/induzido quimicamente
Baltimore/epidemiologia
Criança
Pré-Escolar
Monitoramento Ambiental
Feminino
Seres Humanos
Estudos Longitudinais
Masculino
Morbidade
Análise Multivariada
Dióxido de Nitrogênio/análise
Estações do Ano
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, U.S. GOV'T, NON-P.H.S.; RESEARCH SUPPORT, N.I.H., EXTRAMURAL
[Nm] Nome de substância:
0 (Air Pollutants); S7G510RUBH (Nitrogen Dioxide)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171120
[Lr] Data última revisão:
171120
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170812
[St] Status:MEDLINE



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