Base de dados : MEDLINE
Pesquisa : F03.625 [Categoria DeCS]
Referências encontradas : 653 [refinar]
Mostrando: 1 .. 10   no formato [Detalhado]

página 1 de 66 ir para página                         

  1 / 653 MEDLINE  
              next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:29237598
[Au] Autor:Iacobucci G
[Ad] Endereço:The BMJ.
[Ti] Título:Extra cash for child mental health is being diverted, report warns.
[So] Source:BMJ;359:j5785, 2017 12 13.
[Is] ISSN:1756-1833
[Cp] País de publicação:England
[La] Idioma:eng
[Mh] Termos MeSH primário: Serviços de Saúde da Criança/economia
Acesso aos Serviços de Saúde/economia
Necessidades e Demandas de Serviços de Saúde/economia
Serviços de Saúde Mental/economia
[Mh] Termos MeSH secundário: Adolescente
Criança
Pré-Escolar
Inglaterra
Seres Humanos
Saúde Mental
Transtornos do Neurodesenvolvimento
[Pt] Tipo de publicação:NEWS
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180226
[Lr] Data última revisão:
180226
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:171215
[St] Status:MEDLINE
[do] DOI:10.1136/bmj.j5785


  2 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:28463237
[Au] Autor:Weber-Stadlbauer U
[Ad] Endereço:Institute of Pharmacology and Toxicology, University of Zurich-Vetsuisse, Zurich, Switzerland.
[Ti] Título:Epigenetic and transgenerational mechanisms in infection-mediated neurodevelopmental disorders.
[So] Source:Transl Psychiatry;7(5):e1113, 2017 May 02.
[Is] ISSN:2158-3188
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Prenatal infection is an environmental risk factor for various brain disorders with neurodevelopmental components, including autism spectrum disorder and schizophrenia. Modeling this association in animals shows that maternal immune activation negatively affects fetal brain development and leads to the emergence of behavioral disturbances later in life. Recent discoveries in these preclinical models suggest that epigenetic modifications may be a critical molecular mechanism by which prenatal immune activation can mediate changes in brain development and functions, even across generations. This review discusses the potential epigenetic mechanisms underlying the effects of prenatal infections, thereby highlighting how infection-mediated epigenetic reprogramming may contribute to the transgenerational transmission of pathological traits. The identification of epigenetic and transgenerational mechanisms in infection-mediated neurodevelopmental disorders appears relevant to brain disorders independently of existing diagnostic classifications and may help identifying complex patterns of transgenerational disease transmission beyond genetic inheritance. The consideration of ancestral infectious histories may be of great clinical interest and may be pivotal for developing new preventive treatment strategies against infection-mediated neurodevelopmental disorders.
[Mh] Termos MeSH primário: Encéfalo/fisiopatologia
Epigênese Genética/imunologia
Transtornos do Neurodesenvolvimento/imunologia
Efeitos Tardios da Exposição Pré-Natal/imunologia
[Mh] Termos MeSH secundário: Animais
Transtorno do Espectro Autista
Encéfalo/crescimento & desenvolvimento
Modelos Animais de Doenças
Exposição Ambiental/efeitos adversos
Epigenômica
Feminino
Desenvolvimento Fetal/imunologia
Desenvolvimento Fetal/fisiologia
Seres Humanos
Transmissão Vertical de Doença Infecciosa/prevenção & controle
Transtornos do Neurodesenvolvimento/prevenção & controle
Fenótipo
Gravidez
Efeitos Tardios da Exposição Pré-Natal/epidemiologia
Efeitos Tardios da Exposição Pré-Natal/patologia
Fatores de Risco
Esquizofrenia/imunologia
Esquizofrenia/patologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180220
[Lr] Data última revisão:
180220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170503
[St] Status:MEDLINE
[do] DOI:10.1038/tp.2017.78


  3 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:27770673
[Au] Autor:Sari Gokten E; Saday Duman N; Soylu N; Uzun ME
[Ad] Endereço:Yuksek Ihtisas Training ve Research Hospital, Bursa, Child and Adolescent Psychiatry, Turkey. Electronic address: esgokten@hotmail.com.
[Ti] Título:Effects of attention-deficit/hyperactivity disorder on child abuse and neglect.
[So] Source:Child Abuse Negl;62:1-9, 2016 12.
[Is] ISSN:1873-7757
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:It is known that children with mental and developmental problems are at risk of abuse and neglect. Attention-deficit/hyperactivity disorder is one of the most frequent neurodevelopmental disorders in children and adolescents. The purpose of this study is to examine whether children diagnosed with ADHD are under more risk in terms of child abuse and neglect compared to controls. In this case-control study, 104 children, who applied to Child and Adolescent Psychiatry Unit of Bursa Yuksek Ihtisas Training and Research Hospital between January and June 2015, were diagnosed with ADHD, and had no other psychiatric comorbidity except for disruptive behavior disorders, and 104 healthy children were compared. Abuse Assessment Questionnaire was applied to children after approval of the families was received. It was determined that the children diagnosed with ADHD were exposed to more physical (96.2%) and emotional abuse (87.5%) in a statistically significant way compared to controls (46.2%; 34.6%), they were exposed to physical and emotional neglect (5.8%) at a lower rate compared to healthy children (24.0%), and there was no difference between them and healthy children in terms of witnessing family violence (56.7%; 47.1%) and being exposed to sexual abuse (5.8%; 1.9%). The children diagnosed with ADHD were exposed to physical and emotional abuse at a higher rate; further studies should emphasize the role of parents in this topic and how parental education and treatment programs change the results.
[Mh] Termos MeSH primário: Transtorno do Deficit de Atenção com Hiperatividade/epidemiologia
Transtorno do Deficit de Atenção com Hiperatividade/psicologia
Maus-Tratos Infantis/estatística & dados numéricos
[Mh] Termos MeSH secundário: Transtorno do Deficit de Atenção com Hiperatividade/diagnóstico
Transtornos de Deficit da Atenção e do Comportamento Disruptivo
Estudos de Casos e Controles
Criança
Maus-Tratos Infantis/diagnóstico
Maus-Tratos Infantis/psicologia
Abuso Sexual na Infância/diagnóstico
Abuso Sexual na Infância/psicologia
Abuso Sexual na Infância/estatística & dados numéricos
Comorbidade
Estudos Transversais
Violência Doméstica
Feminino
Seres Humanos
Masculino
Transtornos do Neurodesenvolvimento
Pais/psicologia
Fatores de Risco
Fatores Socioeconômicos
Estatística como Assunto
Inquéritos e Questionários
Turquia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1802
[Cu] Atualização por classe:180216
[Lr] Data última revisão:
180216
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161023
[St] Status:MEDLINE


  4 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:29196536
[Au] Autor:Fontenot MR; Berto S; Liu Y; Werthmann G; Douglas C; Usui N; Gleason K; Tamminga CA; Takahashi JS; Konopka G
[Ad] Endereço:Department of Neuroscience, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA.
[Ti] Título:Novel transcriptional networks regulated by CLOCK in human neurons.
[So] Source:Genes Dev;31(21):2121-2135, 2017 11 01.
[Is] ISSN:1549-5477
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The molecular mechanisms underlying human brain evolution are not fully understood; however, previous work suggested that expression of the transcription factor in the human cortex might be relevant to human cognition and disease. In this study, we investigated this novel transcriptional role for CLOCK in human neurons by performing chromatin immunoprecipitation sequencing for endogenous CLOCK in adult neocortices and RNA sequencing following CLOCK knockdown in differentiated human neurons in vitro. These data suggested that CLOCK regulates the expression of genes involved in neuronal migration, and a functional assay showed that CLOCK knockdown increased neuronal migratory distance. Furthermore, dysregulation of CLOCK disrupts coexpressed networks of genes implicated in neuropsychiatric disorders, and the expression of these networks is driven by hub genes with human-specific patterns of expression. These data support a role for CLOCK-regulated transcriptional cascades involved in human brain evolution and function.
[Mh] Termos MeSH primário: Proteínas CLOCK/genética
Proteínas CLOCK/metabolismo
Regulação da Expressão Gênica no Desenvolvimento/genética
Redes Reguladoras de Genes/genética
Neurônios/fisiologia
[Mh] Termos MeSH secundário: Linhagem Celular
Movimento Celular/genética
Epigênese Genética/genética
Técnicas de Silenciamento de Genes
Seres Humanos
Neocórtex/metabolismo
Transtornos do Neurodesenvolvimento/genética
Neurônios/citologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T; RESEARCH SUPPORT, N.I.H., EXTRAMURAL
[Nm] Nome de substância:
EC 2.3.1.48 (CLOCK Proteins)
[Em] Mês de entrada:1712
[Cu] Atualização por classe:180216
[Lr] Data última revisão:
180216
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171203
[St] Status:MEDLINE
[do] DOI:10.1101/gad.305813.117


  5 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:27779093
[Au] Autor:Harrington AJ; Raissi A; Rajkovich K; Berto S; Kumar J; Molinaro G; Raduazzo J; Guo Y; Loerwald K; Konopka G; Huber KM; Cowan CW
[Ad] Endereço:Department of Neurosciences, Medical University of South Carolina, Charleston, United States.
[Ti] Título:MEF2C regulates cortical inhibitory and excitatory synapses and behaviors relevant to neurodevelopmental disorders.
[So] Source:Elife;5, 2016 10 25.
[Is] ISSN:2050-084X
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Numerous genetic variants associated with are linked to autism, intellectual disability (ID) and schizophrenia (SCZ) - a heterogeneous collection of neurodevelopmental disorders with unclear pathophysiology. MEF2C is highly expressed in developing cortical excitatory neurons, but its role in their development remains unclear. We show here that conditional embryonic deletion of in cortical and hippocampal excitatory neurons (Emx1-lineage) produces a dramatic reduction in cortical network activity in vivo, due in part to a dramatic increase in inhibitory and a decrease in excitatory synaptic transmission. In addition, we find that MEF2C regulates E/I synapse density predominantly as a cell-autonomous, transcriptional repressor. Analysis of differential gene expression in mutant cortex identified a significant overlap with numerous synapse- and autism-linked genes, and the mutant mice displayed numerous behaviors reminiscent of autism, ID and SCZ, suggesting that perturbing MEF2C function in neocortex can produce autistic- and ID-like behaviors in mice.
[Mh] Termos MeSH primário: Comportamento Animal
Transtornos do Neurodesenvolvimento/fisiopatologia
Sinapses/fisiologia
[Mh] Termos MeSH secundário: Animais
Transtorno Autístico/fisiopatologia
Córtex Cerebral/embriologia
Técnicas de Silenciamento de Genes
Hipocampo/embriologia
Deficiência Intelectual/fisiopatologia
Fatores de Transcrição MEF2/metabolismo
Camundongos
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T; RESEARCH SUPPORT, N.I.H., EXTRAMURAL
[Nm] Nome de substância:
0 (MEF2 Transcription Factors); 0 (Mef2c protein, mouse)
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171224
[Lr] Data última revisão:
171224
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161104
[St] Status:MEDLINE


  6 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:29229141
[Au] Autor:Miranda A; Roque S; Pêgo JM; Correia-Pinto J
[Ad] Endereço:Life and Health Sciences Research Institute (ICVS), School of Medicine, University of Minho, Braga, Portugal; ICVS/3B's - PT Government Associate Laboratory, Braga, Guimarães, Portugal. Electronic address: alicemiranda@med.uminho.pt.
[Ti] Título:Neurodevelopment impact of CO -pneumoperitoneum in neonates: experimental study in a rat model.
[So] Source:J Surg Res;221:293-303, 2018 Jan.
[Is] ISSN:1095-8673
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Laparoscopy is becoming more common in neonates. However, concerns remain about the impact of the carbon-dioxide (CO )-insufflation over the neonatal brain. We aim to evaluate the peripheral (serum) and central (cerebrospinal fluid [CSF]) cytokine response after neonatal CO -pneumoperitoneum and its impact over neurodevelopmental milestones acquisition and long-term behavioral outcomes. MATERIALS AND METHODS: Rats were subjected to a systematic assessment of neurodevelopmental milestones between postnatal day 1 (PND 1) and PND 21. At PND 10, neonatal rats were anesthetized, mechanically ventilated, and exposed to different pressures and times of abdominal CO -insufflation. Immediately after pneumoperitoneum, corticosterone was analyzed in serum. Twenty-four hours after intervention, serum and CSF were collected to assess inflammatory response (interleukin [IL]-10, IL-1ß, tumor necrosis factor [TNF]-α, and interferon [IFN]-γ). In adulthood, animals from each group were submitted to several tests to assess different behavioral domains (locomotion, anxiety, mood, and cognition). RESULTS: The antiinflammatory cytokine IL-10 was significantly increased in CSF in CO -insufflated groups, with no other significant changes in the other biomarkers. Acquisition of neurodevelopmental milestones was maintained in all studied groups. No significant differences were observed in adult behavior in the different CO -insufflation conditions. CONCLUSIONS: Neonatal CO -pneumoperitoneum does not seem to have any negative impact on neurodevelopment or induce behavioral alterations in adulthood. Minimally invasive surgery results in a central antiinflammatory profile, and further studies on the functional consequences of these phenomena are needed.
[Mh] Termos MeSH primário: Inflamação/etiologia
Transtornos do Neurodesenvolvimento/etiologia
Pneumoperitônio Artificial/efeitos adversos
[Mh] Termos MeSH secundário: Animais
Dióxido de Carbono
Citocinas/metabolismo
Feminino
Inflamação/metabolismo
Gravidez
Ratos Sprague-Dawley
Estresse Fisiológico
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Cytokines); 142M471B3J (Carbon Dioxide)
[Em] Mês de entrada:1712
[Cu] Atualização por classe:171219
[Lr] Data última revisão:
171219
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171213
[St] Status:MEDLINE


  7 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo SciELO Chile
[PMID]:29182193
[Au] Autor:Faundes V; Santa María L; Morales P; Curotto B; Alliende MA
[Ad] Endereço:Laboratorio de Citogenética Molecular, Instituto de Nutrición y Tecnología de los Alimentos (INTA), Universidad de Chile, Santiago, Chile.
[Ti] Título:[Microarrays in 236 patients with neurodevelopmental disorders and congenital abnormalities].
[Ti] Título:Microarreglos cromosómicos en 236 pacientes chilenos con trastornos del neurodesarrollo y anomalías congénitas..
[So] Source:Rev Med Chil;145(7):854-861, 2017 Jul.
[Is] ISSN:0717-6163
[Cp] País de publicação:Chile
[La] Idioma:spa
[Ab] Resumo:BACKGROUND: In 20% of neurodevelopmental disorders (NDD) and congenital abnormalities (CA) the cause would be a genomic imbalance detectable only by chromosomal microarrays (CMA). AIM: To analyze the results of CMA performed at the INTA Laboratory of Molecular Cytogenetics, during a period of four years in patients with NDD or CA. MATERIAL AND METHODS: Retrospective study that included all CMA reports of Chilean patients. Age, sex, clinical diagnosis and origin were analyzed, as well as the characteristics of the finding. The percentage of cases diagnosed by CMA was calculated considering all patients with pathogenic (PV) or probably pathogenic variants (VLP). Finally, we studied the association between patients' characteristics and a positive CMA outcome. RESULTS: A total of 236 reports were analyzed. The median age was 5.41 (range 2.25-9.33) years, and 59% were men. Ninety chromosomal imbalances were found, which corresponded mainly to deletions (53.3%), with a median size of 1.662 (range 0.553-6.673) Megabases. The diagnostic rate of CMA in Chilean patients from all over the country was 19.2%. There was a close relationship between the patient's sex and the detection of VLP/VP (p = 0.034). CONCLUSIONS: Our diagnostic rate and the association between female sex and a higher percentage of diagnosed cases are concordant with other international studies. Therefore, CMA is a valid diagnostic tool in the Chilean population.
[Mh] Termos MeSH primário: Anormalidades Congênitas/diagnóstico
Anormalidades Congênitas/genética
Análise em Microsséries/métodos
Transtornos do Neurodesenvolvimento/diagnóstico
Transtornos do Neurodesenvolvimento/genética
[Mh] Termos MeSH secundário: Criança
Pré-Escolar
Chile
Feminino
Seres Humanos
Masculino
Estudos Retrospectivos
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1712
[Cu] Atualização por classe:171214
[Lr] Data última revisão:
171214
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171129
[St] Status:MEDLINE


  8 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:29192004
[Au] Autor:Simon TD; Whitlock KB; Haaland W; Wright DR; Zhou C; Neff J; Howard W; Cartin B; Mangione-Smith R
[Ad] Endereço:Department of Pediatrics, University of Washington, Seattle Children's Hospital, Seattle, Washington; and tamara.simon@seattlechildrens.org.
[Ti] Título:Effectiveness of a Comprehensive Case Management Service for Children With Medical Complexity.
[So] Source:Pediatrics;140(6), 2017 Dec.
[Is] ISSN:1098-4275
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:OBJECTIVES: To assess whether children with medical complexity (CMC) exposed to a hospital-based comprehensive case management service (CCMS) experience improved health care quality, improved functional status, reduced hospital-based utilization, and/or reduced overall health care costs. METHODS: Eligible CMC at Seattle Children's Hospital were enrolled in a cluster randomized controlled trial between December 1, 2010, and September 29, 2014. Participating primary care providers (PCPs) were randomly assigned, and CMC either had access to an outpatient hospital-based CCMS or usual care directed by their PCP. The CCMS included visits to a multidisciplinary clinic ≥ every 6 months for 1.5 years, an individualized shared care plan, and access to CCMS providers. Differences between control and intervention groups in change from baseline to 12 months and baseline to 18 months (difference of differences) were tested. RESULTS: Two hundred PCPs caring for 331 CMC were randomly assigned. Intervention group ( = 181) parents reported more improvement in the Consumer Assessment of Healthcare Providers and Systems version 4.0 Child Health Plan Survey global health care quality ratings than control group parents (6.7 [95% confidence interval (CI): 3.5-9.8] vs 1.3 [95% CI: 1.9-4.6] at 12 months). We did not detect significant differences in child functional status and most hospital-based utilization between groups. The difference in change of overall health care costs was higher in the intervention group (+$8233 [95% CI: $1701-$16 937]) at 18 months). CCMS clinic costs averaged $3847 per child-year. CONCLUSIONS: Access to a CCMS generally improved health care quality, but was not associated with changes in child functional status or hospital-based utilization, and increased overall health care costs among CMC.
[Mh] Termos MeSH primário: Administração de Caso/normas
Pesquisas sobre Serviços de Saúde/métodos
Pessoal de Saúde/normas
Hospitais Pediátricos
Transtornos do Neurodesenvolvimento/terapia
Atenção Primária à Saúde/normas
Melhoria de Qualidade
[Mh] Termos MeSH secundário: Criança
Feminino
Seres Humanos
Masculino
Qualidade de Vida
Estudos Retrospectivos
[Pt] Tipo de publicação:JOURNAL ARTICLE; RANDOMIZED CONTROLLED TRIAL
[Em] Mês de entrada:1712
[Cu] Atualização por classe:171211
[Lr] Data última revisão:
171211
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:171202
[St] Status:MEDLINE


  9 / 653 MEDLINE  
              first record previous record next record last record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:29173597
[Au] Autor:Nattel SN; Adrianzen L; Kessler EC; Andelfinger G; Dehaes M; Côté-Corriveau G; Trelles MP
[Ad] Endereço:Department of Psychiatry, Albert Einstein College of Medicine and Seaver Autism Center at Icahn School of Medicine at Mount Sinai, New York, New York, USA.
[Ti] Título:Congenital Heart Disease and Neurodevelopment: Clinical Manifestations, Genetics, Mechanisms, and Implications.
[So] Source:Can J Cardiol;33(12):1543-1555, 2017 Dec.
[Is] ISSN:1916-7075
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Children with congenital heart disease (CHD) are at increased risk of neurodevelopmental disorders (NDDs) and psychiatric conditions. These include cognitive, adaptive, motor, speech, behavioural, and executive functioning deficits, as well as autism spectrum disorder and psychiatric conditions. Structural and functional neuroimaging have demonstrated brain abnormalities in young children with CHD before undergoing surgical repair, likely as a result of an in utero developmental insult. Surgical factors do not seem to play a significant role in neurodevelopmental outcomes. Specific genetic abnormalities, particularly copy number variants, have been increasingly implicated in both CHD and NDDs. Variations in genes involved in apolipoprotein E (APOE) production, the Wnt signalling pathway, and histone modification, as well as in the 1q21.1, 16p13.1-11, and 8p23.1 genetic loci, have been associated with CHD and NDDs and are important targets for future research. Understanding these associations is important for risk stratification, disease classification, improved screening, and pharmacologic management of individuals with CHD.
[Mh] Termos MeSH primário: Diagnóstico por Imagem/métodos
Testes Genéticos/métodos
Cardiopatias Congênitas
Transtornos do Neurodesenvolvimento
[Mh] Termos MeSH secundário: Cardiopatias Congênitas/complicações
Cardiopatias Congênitas/diagnóstico
Cardiopatias Congênitas/genética
Seres Humanos
Transtornos do Neurodesenvolvimento/diagnóstico
Transtornos do Neurodesenvolvimento/etiologia
Transtornos do Neurodesenvolvimento/genética
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1712
[Cu] Atualização por classe:171204
[Lr] Data última revisão:
171204
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171128
[St] Status:MEDLINE


  10 / 653 MEDLINE  
              first record previous record
seleciona
para imprimir
Fotocópia
Texto completo
[PMID]:28471024
[Au] Autor:Bilardo CM
[Ad] Endereço:Fetal Medicine Unit, Department of Obstetrics & Gynaecology, University Medical Center Groningen, University of Groningen, Hanzelplein 1, 9700 RB, Groningen, The Netherlands.
[Ti] Título:Re: Increased nuchal translucency thickness and risk of neurodevelopmental disorders. S. G. Hellmuth, L. H. Pedersen, C. B. Miltoft, O. B. Petersen, S. Kjaergaard, C. Ekelund and A. Tabor. Ultrasound Obstet Gynecol 2017; 49: 592-598.
[So] Source:Ultrasound Obstet Gynecol;49(5):564, 2017 05.
[Is] ISSN:1469-0705
[Cp] País de publicação:England
[La] Idioma:eng
[Mh] Termos MeSH primário: Transtornos do Neurodesenvolvimento
Medição da Translucência Nucal
[Mh] Termos MeSH secundário: Feminino
Seres Humanos
Gravidez
Risco
[Pt] Tipo de publicação:JOURNAL ARTICLE; COMMENT
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171128
[Lr] Data última revisão:
171128
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170505
[St] Status:MEDLINE
[do] DOI:10.1002/uog.17477



página 1 de 66 ir para página                         
   


Refinar a pesquisa
  Base de dados : MEDLINE Formulário avançado   

    Pesquisar no campo  
1  
2
3
 
           



Search engine: iAH v2.6 powered by WWWISIS

BIREME/OPAS/OMS - Centro Latino-Americano e do Caribe de Informação em Ciências da Saúde