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  1 / 2997 MEDLINE  
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[PMID]:29380886
[Au] Autor:Rogers J
[Ad] Endereço:Department of Molecular and Human Genetics and Human Genome Sequencing Center, Baylor College of Medicine, Houston, Texas 77030.
[Ti] Título:The behavioral genetics of nonhuman primates: Status and prospects.
[So] Source:Am J Phys Anthropol;165 Suppl 65:23-36, 2018 01.
[Is] ISSN:1096-8644
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:The complexity and diversity of primate behavior have long attracted the attention of ethologists, psychologists, behavioral ecologists, and neuroscientists. Recent studies have advanced our understanding of the nature of genetic influences on differences in behavior among individuals within species. A number of analyses have focused on the genetic analysis of behavioral reactions to specific experimental tests, providing estimates of the degree of genetic control over reactivity, and beginning to identify the genes involved. Substantial progress is also being made in identifying genetic factors that influence the structure and function of the primate brain. Most of the published studies on these topics have examined either cercopithecines or chimpanzees, though a few studies have addressed these questions in other primate species. One potentially important line of research is beginning to identify the epigenetic processes that influence primate behavior, thus revealing specific cellular and molecular mechanisms by which environmental experiences can influence gene expression or gene function relevant to behavior. This review summarizes many of these studies of non-human primate behavioral genetics. The primary focus is on analyses that address the nature of the genes and genetic processes that affect differences in behavior among individuals within non-human primate species. Analyses of between species differences and potential avenues for future research are also discussed.
[Mh] Termos MeSH primário: Comportamento Animal
Encéfalo
Primatas
[Mh] Termos MeSH secundário: Animais
Encéfalo/anatomia & histologia
Encéfalo/fisiologia
Epigênese Genética/genética
Epigênese Genética/fisiologia
Feminino
Genética Comportamental
Relações Interpessoais
Masculino
Primatas/anatomia & histologia
Primatas/genética
Primatas/fisiologia
[Pt] Tipo de publicação:JOURNAL ARTICLE; RESEARCH SUPPORT, N.I.H., EXTRAMURAL
[Em] Mês de entrada:1803
[Cu] Atualização por classe:180302
[Lr] Data última revisão:
180302
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:180131
[St] Status:MEDLINE
[do] DOI:10.1002/ajpa.23384


  2 / 2997 MEDLINE  
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[PMID]:28617822
[Au] Autor:Pinsonneault JK; Frater JT; Kompa B; Mascarenhas R; Wang D; Sadee W
[Ad] Endereço:Center for Pharmacogenomics, Department of Cancer Biology and Genetics, College of Medicine and Public Health, Ohio State University, Columbus, Ohio, United States of America.
[Ti] Título:Intronic SNP in ESR1 encoding human estrogen receptor alpha is associated with brain ESR1 mRNA isoform expression and behavioral traits.
[So] Source:PLoS One;12(6):e0179020, 2017.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Genetic variants of ESR1 have been implicated in multiple diseases, including behavioral disorders, but causative variants remain uncertain. We have searched for regulatory variants affecting ESR1 expression in human brain, measuring allelic ESR1 mRNA expression in human brain tissues with marker SNPs in exon4 representing ESR1-008 (or ESRα-36), and in the 3'UTR of ESR1-203, two main ESR1 isoforms in brain. In prefrontal cortex from subjects with bipolar disorder, schizophrenia, and controls (n = 35 each; Stanley Foundation brain bank), allelic ESR1 mRNA ratios deviated from unity up to tenfold at the exon4 marker SNP, with large allelic ratios observed primarily in bipolar and schizophrenic subjects. SNP scanning and targeted sequencing identified rs2144025, associated with large allelic mRNA ratios (p = 1.6E10-6). Moreover, rs2144025 was significantly associated with ESR1 mRNA levels in the Brain eQTL Almanac and in brain regions in the Genotype-Tissue Expression project. In four GWAS cohorts, rs2104425 was significantly associated with behavioral traits, including: hypomanic episodes in female bipolar disorder subjects (GAIN bipolar disorder study; p = 0.0004), comorbid psychological symptoms in both males and females with attention deficit hyperactivity disorder (GAIN ADHD, p = 0.00002), psychological diagnoses in female children (eMERGE study of childhood health, subject age ≥9, p = 0.0009), and traits in schizophrenia (e.g., grandiose delusions, GAIN schizophrenia, p = 0.0004). The first common ESR1 variant (MAF 12-33% across races) linked to regulatory functions, rs2144025 appears conditionally to affect ESR1 mRNA expression in the brain and modulate traits in behavioral disorders.
[Mh] Termos MeSH primário: Encéfalo/metabolismo
Receptor alfa de Estrogênio/genética
Transtornos Mentais/genética
Polimorfismo de Nucleotídeo Único
Isoformas de RNA/metabolismo
[Mh] Termos MeSH secundário: Adulto
Transtorno Bipolar/genética
Transtorno Bipolar/metabolismo
Encéfalo/patologia
Feminino
Estudos de Associação Genética
Predisposição Genética para Doença
Genética Comportamental/métodos
Seres Humanos
Masculino
Transtornos Mentais/metabolismo
Meia-Idade
Locos de Características Quantitativas
Isoformas de RNA/genética
Esquizofrenia/genética
Esquizofrenia/metabolismo
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Estrogen Receptor alpha); 0 (RNA Isoforms); 0 (estrogen receptor alpha, human)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170914
[Lr] Data última revisão:
170914
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170616
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0179020


  3 / 2997 MEDLINE  
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[PMID]:28504703
[Au] Autor:Weiner DJ; Wigdor EM; Ripke S; Walters RK; Kosmicki JA; Grove J; Samocha KE; Goldstein JI; Okbay A; Bybjerg-Grauholm J; Werge T; Hougaard DM; Taylor J; Skuse D; Devlin B; Anney R; Sanders SJ; Bishop S; Mortensen PB; Børglum AD; Smith GD; Daly MJ; Robinson EB; iPSYCH-Broad Autism Group; Psychiatric Genomics Consortium Autism Group
[Ad] Endereço:Analytic and Translational Genetics Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA.
[Ti] Título:Polygenic transmission disequilibrium confirms that common and rare variation act additively to create risk for autism spectrum disorders.
[So] Source:Nat Genet;49(7):978-985, 2017 Jul.
[Is] ISSN:1546-1718
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Autism spectrum disorder (ASD) risk is influenced by common polygenic and de novo variation. We aimed to clarify the influence of polygenic risk for ASD and to identify subgroups of ASD cases, including those with strongly acting de novo variants, in which polygenic risk is relevant. Using a novel approach called the polygenic transmission disequilibrium test and data from 6,454 families with a child with ASD, we show that polygenic risk for ASD, schizophrenia, and greater educational attainment is over-transmitted to children with ASD. These findings hold independent of proband IQ. We find that polygenic variation contributes additively to risk in ASD cases who carry a strongly acting de novo variant. Lastly, we show that elements of polygenic risk are independent and differ in their relationship with phenotype. These results confirm that the genetic influences on ASD are additive and suggest that they create risk through at least partially distinct etiologic pathways.
[Mh] Termos MeSH primário: Transtorno do Espectro Autista/genética
Variação Genética
Herança Multifatorial
[Mh] Termos MeSH secundário: Adulto
Transtorno do Espectro Autista/epidemiologia
Criança
Estudos de Coortes
Escolaridade
Grupos Étnicos/genética
Saúde da Família
Feminino
Estudos de Associação Genética
Predisposição Genética para Doença
Genética Comportamental
Seres Humanos
Deficiência Intelectual/genética
Inteligência/genética
Masculino
Fenótipo
Fatores de Risco
Esquizofrenia/genética
Deleção de Sequência
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171119
[Lr] Data última revisão:
171119
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170516
[St] Status:MEDLINE
[do] DOI:10.1038/ng.3863


  4 / 2997 MEDLINE  
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[PMID]:28004961
[Au] Autor:Barbaro N; Boutwell BB; Barnes JC; Shackelford TK
[Ad] Endereço:Department of Psychology, Oakland University.
[Ti] Título:Rethinking the transmission gap: What behavioral genetics and evolutionary psychology mean for attachment theory: A comment on Verhage et al. (2016).
[So] Source:Psychol Bull;143(1):107-113, 2017 01.
[Is] ISSN:1939-1455
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Traditional attachment theory posits that attachment in infancy and early childhood is the result of intergenerational transmission of attachment from parents to offspring. Verhage et al. (2016) present meta-analytic evidence addressing the intergenerational transmission of attachment between caregivers and young children. In this commentary, we argue that their appraisal of the behavioral genetics literature is incomplete. The suggested research focus on shared environmental effects may dissuade the pursuit of profitable avenues of research and may hinder progress in attachment theory. Specifically, further research on the "transmission gap" will continue to limit our understanding of attachment etiology. We discuss recent theoretical developments from an evolutionary psychological perspective that can provide a valuable framework to account for the existing behavioral genetic data. (PsycINFO Database Record
[Mh] Termos MeSH primário: Genética Comportamental
Pais
[Mh] Termos MeSH secundário: Evolução Biológica
Seres Humanos
Apego ao Objeto
[Pt] Tipo de publicação:JOURNAL ARTICLE; COMMENT
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170809
[Lr] Data última revisão:
170809
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161223
[St] Status:MEDLINE
[do] DOI:10.1037/bul0000066


  5 / 2997 MEDLINE  
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[PMID]:27906529
[Au] Autor:Charney E
[Ad] Endereço:Sanford School of Public Policy, Duke Center for Brain Sciences, Duke University, Durham, NC, USA.
[Ti] Título:Genes, behavior, and behavior genetics.
[So] Source:Wiley Interdiscip Rev Cogn Sci;8(1-2), 2017 Jan.
[Is] ISSN:1939-5086
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:According to the 'first law' of behavior genetics, 'All human behavioral traits are heritable.' Accepting the validity of this first law and employing statistical methods, researchers within psychology, sociology, political science, economics, and business claim to have demonstrated that all the behaviors studied by their disciplines are heritable-no matter how culturally specific these behaviors appear to be. Further, in many cases they claim to have identified specific genes that play a role in those behaviors. The validity of behavior genetics as a discipline depends upon the validity of the research methods used to justify such claims. It also depends, foundationally, upon certain key assumptions concerning the relationship between genotype (one's specific DNA sequences) and phenotype (any and all observable traits or characteristics). In this article, I examine-and find serious flaws with-both the methodologies of behavior genetics and the underlying assumptions concerning the genotype-phenotype relationship. WIREs Cogn Sci 2017, 8:e1405. doi: 10.1002/wcs.1405 For further resources related to this article, please visit the WIREs website.
[Mh] Termos MeSH primário: Genética Comportamental
Modelos Biológicos
Característica Quantitativa Herdável
[Mh] Termos MeSH secundário: Estudos de Associação Genética
Genótipo
Seres Humanos
Fenótipo
Estudos em Gêmeos como Assunto
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Em] Mês de entrada:1703
[Cu] Atualização por classe:170324
[Lr] Data última revisão:
170324
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161202
[St] Status:MEDLINE
[do] DOI:10.1002/wcs.1405


  6 / 2997 MEDLINE  
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[PMID]:27826669
[Au] Autor:Bruni M; Flax JF; Buyske S; Shindhelm AD; Witton C; Brzustowicz LM; Bartlett CW
[Ad] Endereço:The Battelle Center for Mathematical Medicine, The Research Institute at Nationwide Children's Hospital, Columbus, OH, USA.
[Ti] Título:Behavioral and Molecular Genetics of Reading-Related AM and FM Detection Thresholds.
[So] Source:Behav Genet;47(2):193-201, 2017 Mar.
[Is] ISSN:1573-3297
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Auditory detection thresholds for certain frequencies of both amplitude modulated (AM) and frequency modulated (FM) dynamic auditory stimuli are associated with reading in typically developing and dyslexic readers. We present the first behavioral and molecular genetic characterization of these two auditory traits. Two extant extended family datasets were given reading tasks and psychoacoustic tasks to determine FM 2 Hz and AM 20 Hz sensitivity thresholds. Univariate heritabilities were significant for both AM (h  = 0.20) and FM (h  = 0.29). Bayesian posterior probability of linkage (PPL) analysis found loci for AM (12q, PPL = 81 %) and FM (10p, PPL = 32 %; 20q, PPL = 65 %). Bivariate heritability analyses revealed that FM is genetically correlated with reading, while AM was not. Bivariate PPL analysis indicates that FM loci (10p, 20q) are not also associated with reading.
[Mh] Termos MeSH primário: Limiar Auditivo/fisiologia
Dislexia/genética
Leitura
[Mh] Termos MeSH secundário: Estimulação Acústica
Teorema de Bayes
Dislexia/psicologia
Família
Feminino
Genética Comportamental/métodos
Seres Humanos
Masculino
Biologia Molecular/métodos
Linhagem
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170914
[Lr] Data última revisão:
170914
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161110
[St] Status:MEDLINE
[do] DOI:10.1007/s10519-016-9821-3


  7 / 2997 MEDLINE  
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[PMID]:27796609
[Au] Autor:Mark KM; Pike A; Latham RM; Oliver BR
[Ad] Endereço:School of Psychology, University of Sussex, Brighton, East Sussex, BN1 9QH, UK. K.M.Mark@sussex.ac.uk.
[Ti] Título:Using Twins to Better Understand Sibling Relationships.
[So] Source:Behav Genet;47(2):202-214, 2017 Mar.
[Is] ISSN:1573-3297
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:We compared the nature of the sibling relationship in dyads of varying genetic relatedness, employing a behavioural genetic design to estimate the contribution that genes and the environment have on this familial bond. Two samples were used-the Sisters and Brothers Study consisted of 173 families with two target non-twin children (mean ages = 7.42 and 5.22 years respectively); and the Twins, Family and Behaviour study included 234 families with two target twin children (mean age = 4.70 years). Mothers and fathers reported on their children's relationship with each other, via a postal questionnaire (the Sisters and Brothers Study) or a telephone interview (the Twins, Family and Behaviour study). Contrary to expectations, no mean level differences emerged when monozygotic twin pairs, dizygotic twin pairs, and non-twin pairs were compared on their sibling relationship quality. Behavioural genetic analyses also revealed that the sibling bond was modestly to moderately influenced by the genetic propensities of the children within the dyad, and moderately to substantially influenced by the shared environment common to both siblings. In addition, for sibling negativity, we found evidence of twin-specific environmental influence-dizygotic twins showed more reciprocity than did non-twins. Our findings have repercussions for the broader application of results from future twin-based investigations.
[Mh] Termos MeSH primário: Genética Comportamental/métodos
Gêmeos Dizigóticos/genética
Gêmeos Monozigóticos/genética
[Mh] Termos MeSH secundário: Adulto
Criança
Pré-Escolar
Meio Ambiente
Pai/psicologia
Feminino
Seres Humanos
Masculino
Mães/psicologia
Irmãos/psicologia
Comportamento Social
Meio Social
Inquéritos e Questionários
Gêmeos/genética
Gêmeos/psicologia
Gêmeos Dizigóticos/psicologia
Gêmeos Monozigóticos/psicologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170914
[Lr] Data última revisão:
170914
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161101
[St] Status:MEDLINE
[do] DOI:10.1007/s10519-016-9825-z


  8 / 2997 MEDLINE  
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[PMID]:28211559
[Au] Autor:Domaradzki J
[Ad] Endereço:Pracownia Socjologii Zdrowia i Patologii Spolecznych, Katedra Nauk Spolecznych, Uniwersytet Medyczny im. Karola Marcinkowskiego w Poznaniu.
[Ti] Título:Behavioral genetics in Polish print news media between 2000 and 2014.
[Ti] Título:Genetyka behawioralna w polskim czasopismiennictwie w latach 2000­2014..
[So] Source:Psychiatr Pol;50(6):1251-1271, 2016 Dec 23.
[Is] ISSN:2391-5854
[Cp] País de publicação:Poland
[La] Idioma:eng; pol
[Ab] Resumo:OBJECTIVES: The aim of this paper is to describe how Polish print news media frame relations between genetics and human behaviors and what images of behavioral genetics dominate in press discourse. METHODS: A content and frame analysis of 72 print news articles about behavioral genetics published between 2000 and 2014 in four major Polish weekly magazines: "Polityka", "Wprost", "Newsweek" and "Przekrój" was conducted. RESULTS: Twenty one different behaviors were mentioned in the sample and six major analytic frames were identified: essentialist, materialistic, deterministic, probabilistic, optimistic and pessimistic. The most common was the tendency to describe human behaviors in terms of genetic essentialism, reductionism and determinism, as almost one half of the articles was focused solely on genetic determinants of human behaviors and lacked any reference to polygenetic and/or environmental conditioning. Although most of the articles were balanced in tone, benefits were stressed more often than potential risks. Stories that confirmed existence of genetic determinants of human behavior were favored over those that did not. One third of the articles stressed the social or ethical consequences of the development of behavioral genetics. CONCLUSIONS: The complex and abstract character of genetic knowledge results in a simplistic portrayal of behavioral genetics in the press, which may lead to a misunderstood interpretation of the complicated interplay between behavior, genetics and environment by the public. Consequently, print news media contribute to geneticization of behaviors. It is important to improve the quality of science reporting on behavioral genetics and to educate researchers how to communicate with the media more effectively.
[Mh] Termos MeSH primário: Interação Gene-Ambiente
Genética Comportamental
Meios de Comunicação de Massa
Publicações Periódicas como Assunto
[Mh] Termos MeSH secundário: Feminino
Genética
Seres Humanos
Masculino
Processos Mentais
Polônia
Opinião Pública
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170529
[Lr] Data última revisão:
170529
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170218
[St] Status:MEDLINE


  9 / 2997 MEDLINE  
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[PMID]:27748230
[Au] Autor:Hahn E; Gottschling J; Bleidorn W; Kandler C; Spengler M; Kornadt AE; Schulz W; Schunck R; Baier T; Krell K; Lang V; Lenau F; Peters AL; Diewald M; Riemann R; Spinath FM
[Ad] Endereço:Saarland University,Saarbruecken,Germany.
[Ti] Título:What Drives the Development of Social Inequality Over the Life Course? The German TwinLife Study.
[So] Source:Twin Res Hum Genet;19(6):659-672, 2016 Dec.
[Is] ISSN:1832-4274
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:The German twin family study 'TwinLife' was designed to enhance our understanding of the development of social inequalities over the life course. The interdisciplinary project investigates mechanisms of social inequalities across the lifespan by taking into account psychological as well as social mechanisms, and their genetic origin as well as the interaction and covariation between these factors. Main characteristics of the study are: (1) a multidimensional perspective on social inequalities, (2) the assessment of developmental trajectories in childhood, adolescence, and young adulthood in a longitudinal design by using (3) a combination of a multi-cohort cross-sequential and an extended twin family design, while (4) capturing a large variation of behavioral and environmental factors in a representative sample of about 4,000 German twin families. In the present article, we first introduce the theoretical and empirical background of the TwinLife study, and second, describe the design, content, and implementation of TwinLife. Since the data will be made available as scientific use file, we also illustrate research possibilities provided by this project to the scientific community.
[Mh] Termos MeSH primário: Genética Comportamental
Fatores Socioeconômicos
Gêmeos/genética
[Mh] Termos MeSH secundário: Adolescente
Família
Feminino
Alemanha
Seres Humanos
Masculino
Estudos em Gêmeos como Assunto
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171026
[Lr] Data última revisão:
171026
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161018
[St] Status:MEDLINE


  10 / 2997 MEDLINE  
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[PMID]:27748217
[Au] Autor:Wadsworth SJ; DeFries JC; Willcutt EG; Pennington BF; Olson RK
[Ad] Endereço:Institute for Behavioral Genetics,University of Colorado,Boulder,Colorado,USA.
[Ti] Título:Genetic Etiologies of Comorbidity and Stability for Reading Difficulties and ADHD: A Replication Study.
[So] Source:Twin Res Hum Genet;19(6):647-651, 2016 Dec.
[Is] ISSN:1832-4274
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Because of recent concerns about the replication of published results in the behavioral and biomedical sciences (Ioannidis, PLoS Medicine, Vol. 2, 2005, p. e124; Open Science Collaboration, Science, Vol. 349, 2015, p. 943; Pashler & Wagenmakers, Perspectives on Psychological Science, Vol. 7, 2012, pp. 528-530), we have conducted a replication of our recently published analyses of longitudinal reading performance and attention deficit-hyperactivity disorder data from twin pairs selected for reading difficulties (Wadsworth et al., Twin Research and Human Genetics, Vol. 18, 2015, pp. 755-761). Results obtained from univariate and bivariate (DeFries & Fulker, Behavior Genetics, Vol. 15, 1985, pp. 467-473; Acta Geneticae Medicae et Gemellologiae: Twin Research, Vol. 37, 1988, pp. 205-216) analyses of data from a subset of twin pairs tested in the International Longitudinal Twin Study of Early Reading Development at post-4th grade, and its continuation into high school at post-9th grade, were compared to those from our previous report. Similar measures of reading performance, the same measures of inattention and hyperactivity/impulsivity, and similar selection criteria were used in the two studies. In general, the patterns of results obtained from these two independent studies were highly similar. Thus, these results clearly illustrate the principle that findings from studies in quantitative behavioral genetics often replicate (Plomin et al., Perspectives on Psychological Science, Vol. 11, 2016, pp. 3-23).
[Mh] Termos MeSH primário: Transtorno do Deficit de Atenção com Hiperatividade/genética
Dislexia/genética
Leitura
Gêmeos/genética
[Mh] Termos MeSH secundário: Adolescente
Transtorno do Deficit de Atenção com Hiperatividade/fisiopatologia
Criança
Doenças em Gêmeos
Dislexia/fisiopatologia
Feminino
Predisposição Genética para Doença
Genética Comportamental
Seres Humanos
Estudos Longitudinais
Masculino
Instituições Acadêmicas
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171026
[Lr] Data última revisão:
171026
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161018
[St] Status:MEDLINE



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