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[PMID]:28052890
[Au] Autor:Davies W; Roache R
[Ad] Endereço:Will Davies, DPhil, Oxford Uehiro Centre for Practical Ethics, Oxford; Rebecca Roache, MPhil, PhD, Philosophy at Royal Holloway, Royal Holloway, University of London, Egham, UK wkdavies@hotmail.com.
[Ti] Título:Reassessing biopsychosocial psychiatry.
[So] Source:Br J Psychiatry;210(1):3-5, 2017 Jan.
[Is] ISSN:1472-1465
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:Psychiatry uncomfortably spans biological and psychosocial perspectives on mental illness, an idea central to Engel's biopsychosocial paradigm. This paradigm was extremely ambitious, proposing new foundations for clinical practice as well as a non-reductive metaphysics for mental illness. Perhaps given this scope, the approach has failed to engender a clearly identifiable research programme. And yet the view remains influential. We reassess the relevance of the biopsychosocial paradigm for psychiatry, distinguishing a number of ways in which it could be (re)conceived.
[Mh] Termos MeSH primário: Transtornos Mentais
Psiquiatria
[Mh] Termos MeSH secundário: Psiquiatria Biológica
Seres Humanos
Transtornos Mentais/classificação
Transtornos Mentais/etiologia
Transtornos Mentais/terapia
Teoria Psicológica
[Pt] Tipo de publicação:EDITORIAL
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170904
[Lr] Data última revisão:
170904
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170106
[St] Status:MEDLINE
[do] DOI:10.1192/bjp.bp.116.182873


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[PMID]:28006997
[Au] Autor:Soyka M; Kranzler HR; Hesselbrock V; Kasper S; Mutschler J; Möller HJ; WFSBP Task Force on Treatment Guidelines for Substance Use Disorders
[Ad] Endereço:a Psychiatric Hospital Meiringen , Meiringen , Switzerland.
[Ti] Título:Guidelines for biological treatment of substance use and related disorders, part 1: Alcoholism, first revision.
[So] Source:World J Biol Psychiatry;18(2):86-119, 2017 Mar.
[Is] ISSN:1814-1412
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:These practice guidelines for the biological treatment of alcohol use disorders are an update of the first edition, published in 2008, which was developed by an international Task Force of the World Federation of Societies of Biological Psychiatry (WFSBP). For this 2016 revision, we performed a systematic review (MEDLINE/PUBMED database, Cochrane Library) of all available publications pertaining to the biological treatment of alcoholism and extracted data from national guidelines. The Task Force evaluated the identified literature with respect to the strength of evidence for the efficacy of each medication and subsequently categorised it into six levels of evidence (A-F) and five levels of recommendation (1-5). Thus, the current guidelines provide a clinically and scientifically relevant, evidence-based update of our earlier recommendations. These guidelines are intended for use by clinicians and practitioners who evaluate and treat people with alcohol use disorders and are primarily concerned with the biological treatment of adults with such disorders.
[Mh] Termos MeSH primário: Alcoolismo/terapia
Antipsicóticos/uso terapêutico
Eletroconvulsoterapia
Psicoterapia
[Mh] Termos MeSH secundário: Comitês Consultivos
Antipsicóticos/classificação
Psiquiatria Biológica
Quimioterapia Combinada
Medicina Baseada em Evidências
Seres Humanos
Cooperação Internacional
Guias de Prática Clínica como Assunto
Ensaios Clínicos Controlados Aleatórios como Assunto
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Antipsychotic Agents)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170925
[Lr] Data última revisão:
170925
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161224
[St] Status:MEDLINE
[do] DOI:10.1080/15622975.2016.1246752


  3 / 561 MEDLINE  
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[PMID]:27419272
[Au] Autor:Bandelow B; Baldwin D; Abelli M; Bolea-Alamanac B; Bourin M; Chamberlain SR; Cinosi E; Davies S; Domschke K; Fineberg N; Grünblatt E; Jarema M; Kim YK; Maron E; Masdrakis V; Mikova O; Nutt D; Pallanti S; Pini S; Ströhle A; Thibaut F; Vaghi MM; Won E; Wedekind D; Wichniak A; Woolley J; Zwanzger P; Riederer P
[Ad] Endereço:a Department of Psychiatry and Psychotherapy , University of Göttingen , Germany.
[Ti] Título:Biological markers for anxiety disorders, OCD and PTSD: A consensus statement. Part II: Neurochemistry, neurophysiology and neurocognition.
[So] Source:World J Biol Psychiatry;18(3):162-214, 2017 Apr.
[Is] ISSN:1814-1412
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:OBJECTIVE: Biomarkers are defined as anatomical, biochemical or physiological traits that are specific to certain disorders or syndromes. The objective of this paper is to summarise the current knowledge of biomarkers for anxiety disorders, obsessive-compulsive disorder (OCD) and posttraumatic stress disorder (PTSD). METHODS: Findings in biomarker research were reviewed by a task force of international experts in the field, consisting of members of the World Federation of Societies for Biological Psychiatry Task Force on Biological Markers and of the European College of Neuropsychopharmacology Anxiety Disorders Research Network. RESULTS: The present article (Part II) summarises findings on potential biomarkers in neurochemistry (neurotransmitters such as serotonin, norepinephrine, dopamine or GABA, neuropeptides such as cholecystokinin, neurokinins, atrial natriuretic peptide, or oxytocin, the HPA axis, neurotrophic factors such as NGF and BDNF, immunology and CO hypersensitivity), neurophysiology (EEG, heart rate variability) and neurocognition. The accompanying paper (Part I) focuses on neuroimaging and genetics. CONCLUSIONS: Although at present, none of the putative biomarkers is sufficient and specific as a diagnostic tool, an abundance of high quality research has accumulated that should improve our understanding of the neurobiological causes of anxiety disorders, OCD and PTSD.
[Mh] Termos MeSH primário: Transtornos de Ansiedade/diagnóstico
Biomarcadores
Transtorno Obsessivo-Compulsivo/diagnóstico
Transtornos de Estresse Pós-Traumáticos/diagnóstico
[Mh] Termos MeSH secundário: Comitês Consultivos
Psiquiatria Biológica
Consenso
Seres Humanos
Ensaios Clínicos Controlados Aleatórios como Assunto
Sociedades Médicas
[Pt] Tipo de publicação:JOURNAL ARTICLE; REVIEW
[Nm] Nome de substância:
0 (Biomarkers)
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170922
[Lr] Data última revisão:
170922
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160716
[St] Status:MEDLINE
[do] DOI:10.1080/15622975.2016.1190867


  4 / 561 MEDLINE  
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Texto completo SciELO Brasil
Texto completo SciELO Saúde Pública
[PMID]:27580236
[Au] Autor:Freitas-Silva LR; Ortega F
[Ad] Endereço:Universidade Federal Rural do Rio de Janeiro, Seropédica, Brasil.
[Ti] Título:Biological determination of mental disorders: a discussion based on recent hypotheses from neuroscience.
[Ti] Título:A determinação biológica dos transtornos mentais: uma discussão a partir de teses neurocientíficas recentes..
[So] Source:Cad Saude Publica;32(8):e00168115, 2016 Aug 29.
[Is] ISSN:1678-4464
[Cp] País de publicação:Brazil
[La] Idioma:eng; por
[Ab] Resumo:Understanding the processes involved in the development of mental disorders has proven challenging ever since psychiatry was founded as a field. Neuroscience has provided new expectations that an explanation will be found for the development of mental disorders based on biological functioning alone. However, such a goal has not been that easy to achieve, and new hypotheses have begun to appear in neuroscience research. In this article we identify epigenetics, neurodevelopment, and plasticity as the principal avenues for a new understanding of the biology of mental phenomena. Genetic complexity, the environment's formative role, and variations in vulnerability involve important changes in the principal hypotheses on biological determination of mental disorders, suggesting a reconfiguration of the limits between the "social" and the "biological" in neuroscience research.
[Mh] Termos MeSH primário: Transtornos Mentais/etiologia
[Mh] Termos MeSH secundário: Psiquiatria Biológica
Brasil
Cérebro/crescimento & desenvolvimento
Epigenômica
Interação Gene-Ambiente
Determinismo Genético
Seres Humanos
Transtornos Mentais/genética
Plasticidade Neuronal
Neurociências
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1704
[Cu] Atualização por classe:170817
[Lr] Data última revisão:
170817
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160901
[St] Status:MEDLINE


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[PMID]:27169962
[Au] Autor:Boyce PM; Berk M
[Ad] Endereço:Sydney Medical School and Westmead Clinical School, University of Sydney, Sydney, NSW philip.boyce@sydney.edu.au.
[Ti] Título:Biological models of mental illness: implications for therapy development.
[So] Source:Med J Aust;204(9):339-40, 2016 May 16.
[Is] ISSN:1326-5377
[Cp] País de publicação:Australia
[La] Idioma:eng
[Mh] Termos MeSH primário: Psiquiatria Biológica/organização & administração
Transtornos Mentais/diagnóstico
Transtornos Mentais/terapia
Serviços de Saúde Mental/organização & administração
Modelos Biológicos
[Mh] Termos MeSH secundário: Atitude do Pessoal de Saúde
Pesquisa Biomédica/organização & administração
Seres Humanos
Equipe de Assistência ao Paciente/organização & administração
Teoria Psicológica
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1702
[Cu] Atualização por classe:170227
[Lr] Data última revisão:
170227
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160513
[St] Status:MEDLINE


  6 / 561 MEDLINE  
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Texto completo SciELO Brasil
[PMID]:27008078
[Au] Autor:Béhague DP
[Ad] Endereço:Vanderbilt University, Nashville, TN, USA, dominique.behague@vanderbilt.edu.
[Ti] Título:Psychiatry, bio-epistemes and the making of adolescence in southern Brazil.
[So] Source:Hist Cienc Saude Manguinhos;23(1):131-54, 2016 Jan-Mar.
[Is] ISSN:1678-4758
[Cp] País de publicação:Brazil
[La] Idioma:eng
[Ab] Resumo:Drawing on an ethnographic study in southern Brazil, this paper explores how therapists' attempts to "resist bioreductionist" pharmaceutical use both succeed and crumble. Using a comparative framing, I show that pharmaceuticalization can become an anesthetizing "lid" that interacts with young people's polarizing micro-politics and is an outgrowth of multi-generational medico-political family histories. This lid, however, is not air-tight and exceptionalities are born out of these very same histories. I argue that both pharmaceuticalization and exceptions to it emerge not through "resistance" to biopsychiatric logics but from the transformative possibilities that the patterned co-production of social, political, and psychiatric life affords.
[Mh] Termos MeSH primário: Psiquiatria Biológica
Psicologia do Adolescente
[Mh] Termos MeSH secundário: Adolescente
Psiquiatria do Adolescente/história
Antropologia Cultural
Brasil
Criança
Educação Infantil/história
Feminino
História do Século XX
Seres Humanos
Masculino
Transtornos Mentais/tratamento farmacológico
Transtornos Mentais/terapia
Adulto Jovem
[Pt] Tipo de publicação:HISTORICAL ARTICLE; JOURNAL ARTICLE; RESEARCH SUPPORT, NON-U.S. GOV'T
[Em] Mês de entrada:1705
[Cu] Atualização por classe:170515
[Lr] Data última revisão:
170515
[Sb] Subgrupo de revista:QIS
[Da] Data de entrada para processamento:160324
[St] Status:MEDLINE


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[PMID]:26844651
[Au] Autor:Decker HS
[Ad] Endereço:University of Houston.
[Ti] Título:Cyclical swings: The bête noire of psychiatry.
[So] Source:Hist Psychol;19(1):52-6, 2016 Feb.
[Is] ISSN:1939-0610
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Progress in psychiatry in the West has been retarded by the proclivity of the discipline to swing violently between 2 approaches to viewing mental illness; that is, emphasizing-to the exclusion of the other-the material-somatic vs the psychical-experiential avenues to knowledge. Each time a shift occurs, the leaders of the new dominant approach emotionally denounce the principles and ideas that came before. We can examine this phenomenon historically by looking at Romantic psychiatry, mid-/late-19th century empirical psychiatry, psychoanalysis, and modern biological psychiatry. Looking at the 2 approaches in treatment today, the gold standard of patient care involves combining empirical/psychological care in 1 person (the psychiatrist) or shared between 2 clinicians working intimately with each other (psychiatrist with psychologist or social worker.) Yet as regards psychiatrists, they are discouraged from paying full attention to the psychological side by the way managed care and third-party payment have combined to remunerate them. Finally, how do we account for the intense swings and denunciations in psychiatry? The author speculates on possible explanations but leaves the question open for her readers.
[Mh] Termos MeSH primário: Transtornos Mentais/psicologia
Psiquiatria/história
[Mh] Termos MeSH secundário: Psiquiatria Biológica/história
História do Século XIX
História do Século XX
Seres Humanos
Psicanálise/história
[Pt] Tipo de publicação:HISTORICAL ARTICLE; JOURNAL ARTICLE
[Em] Mês de entrada:1701
[Cu] Atualização por classe:170107
[Lr] Data última revisão:
170107
[Sb] Subgrupo de revista:QIS
[Da] Data de entrada para processamento:160205
[St] Status:MEDLINE
[do] DOI:10.1037/hop0000017


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[PMID]:26802772
[Au] Autor:Hafeman DM
[Ad] Endereço:University of Pittsburgh, Pittsburgh, PA. Electronic address: hafemand@upmc.edu.
[Ti] Título:Here/In This Issue and There/Abstract Thinking: Risk Calculators in Child Psychiatry.
[So] Source:J Am Acad Child Adolesc Psychiatry;55(2):87-8, 2016 Feb.
[Is] ISSN:1527-5418
[Cp] País de publicação:United States
[La] Idioma:eng
[Mh] Termos MeSH primário: Psiquiatria Biológica/métodos
Psiquiatria Infantil/métodos
[Mh] Termos MeSH secundário: Psiquiatria Biológica/tendências
Criança
Psiquiatria Infantil/tendências
Previsões
Seres Humanos
Fatores de Risco
[Pt] Tipo de publicação:INTRODUCTORY JOURNAL ARTICLE
[Em] Mês de entrada:1702
[Cu] Atualização por classe:170202
[Lr] Data última revisão:
170202
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160124
[St] Status:MEDLINE


  9 / 561 MEDLINE  
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[PMID]:26595752
[Au] Autor:Thibaut F; Bradford JM; Briken P; De La Barra F; Häßler F; Cosyns P; WFSBP Task Force on Sexual Disorders
[Ad] Endereço:a University Hospital Cochin, Faculty of Medicine Paris Descartes, INSERM U 894 CPN , Paris , France .
[Ti] Título:The World Federation of Societies of Biological Psychiatry (WFSBP) guidelines for the treatment of adolescent sexual offenders with paraphilic disorders.
[So] Source:World J Biol Psychiatry;17(1):2-38, 2016.
[Is] ISSN:1814-1412
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:The primary aim of these guidelines was to evaluate the role of pharmacological agents in the treatment of adolescents with paraphilic disorders who are also sexual offenders or at-risk of sexual offending. Psychotherapeutic and psychosocial treatments were also reviewed. Adolescents with paraphilic disorders specifically present a different therapeutic challenge as compared to adults. In part, the challenge relates to adolescents being in various stages of puberty and development, which may limit the use of certain pharmacological agents due to their potential side effects. In addition, most of the published treatment programmes have used cognitive behavioural interventions, family therapies and psychoeducational interventions. Psychological treatment is predicated in adolescents on the notion that sexually deviant behaviour can be controlled by the offender, and that more adaptive behaviours can be learned. The main purposes of these guidelines are to improve the quality of care and to aid physicians in their clinical decisions. These guidelines brought together different expert views and involved an extensive literature research. Each treatment recommendation was evaluated and discussed with respect to the strength of evidence for efficacy, safety, tolerability and feasibility. An algorithm is proposed for the treatment of paraphilic disorders in adolescent sexual offenders or those who are at risk.
[Mh] Termos MeSH primário: Terapia Cognitiva
Antiandrógenos não Esteroides/uso terapêutico
Transtornos Parafílicos/diagnóstico
Transtornos Parafílicos/terapia
Inibidores da Captação de Serotonina/uso terapêutico
Delitos Sexuais/psicologia
[Mh] Termos MeSH secundário: Adolescente
Psiquiatria Biológica
Seres Humanos
Medição de Risco
Fatores de Risco
Sociedades Médicas
[Pt] Tipo de publicação:JOURNAL ARTICLE; PRACTICE GUIDELINE; REVIEW
[Nm] Nome de substância:
0 (Nonsteroidal Anti-Androgens); 0 (Serotonin Uptake Inhibitors)
[Em] Mês de entrada:1610
[Cu] Atualização por classe:170220
[Lr] Data última revisão:
170220
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151124
[St] Status:MEDLINE
[do] DOI:10.3109/15622975.2015.1085598


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[PMID]:26573769
[Au] Autor:Thapar A; Martin J; Mick E; Arias Vásquez A; Langley K; Scherer SW; Schachar R; Crosbie J; Williams N; Franke B; Elia J; Glessner J; Hakonarson H; Owen MJ; Faraone SV; O'Donovan MC; Holmans P
[Ad] Endereço:Institute of Psychological Medicine and Clinical Neurosciences and MRC Centre for Neuropsychiatric Genetics and Genomics, Cardiff University School of Medicine, Cardiff, UK.
[Ti] Título:Psychiatric gene discoveries shape evidence on ADHD's biology.
[So] Source:Mol Psychiatry;21(9):1202-7, 2016 Sep.
[Is] ISSN:1476-5578
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:A strong motivation for undertaking psychiatric gene discovery studies is to provide novel insights into unknown biology. Although attention-deficit hyperactivity disorder (ADHD) is highly heritable, and large, rare copy number variants (CNVs) contribute to risk, little is known about its pathogenesis and it remains commonly misunderstood. We assembled and pooled five ADHD and control CNV data sets from the United Kingdom, Ireland, United States of America, Northern Europe and Canada. Our aim was to test for enrichment of neurodevelopmental gene sets, implicated by recent exome-sequencing studies of (a) schizophrenia and (b) autism as a means of testing the hypothesis that common pathogenic mechanisms underlie ADHD and these other neurodevelopmental disorders. We also undertook hypothesis-free testing of all biological pathways. We observed significant enrichment of individual genes previously found to harbour schizophrenia de novo non-synonymous single-nucleotide variants (SNVs; P=5.4 × 10(-4)) and targets of the Fragile X mental retardation protein (P=0.0018). No enrichment was observed for activity-regulated cytoskeleton-associated protein (P=0.23) or N-methyl-D-aspartate receptor (P=0.74) post-synaptic signalling gene sets previously implicated in schizophrenia. Enrichment of ADHD CNV hits for genes impacted by autism de novo SNVs (P=0.019 for non-synonymous SNV genes) did not survive Bonferroni correction. Hypothesis-free testing yielded several highly significantly enriched biological pathways, including ion channel pathways. Enrichment findings were robust to multiple testing corrections and to sensitivity analyses that excluded the most significant sample. The findings reveal that CNVs in ADHD converge on biologically meaningful gene clusters, including ones now established as conferring risk of other neurodevelopmental disorders.
[Mh] Termos MeSH primário: Transtorno do Deficit de Atenção com Hiperatividade/genética
Psiquiatria Biológica/métodos
[Mh] Termos MeSH secundário: Adolescente
Transtorno Autístico/genética
Canadá
Criança
Pré-Escolar
Variações do Número de Cópias de DNA/genética
Bases de Dados de Ácidos Nucleicos
Europa (Continente)
Feminino
Estudos de Associação Genética/métodos
Predisposição Genética para Doença/genética
Estudo de Associação Genômica Ampla
Seres Humanos
Irlanda
Masculino
Transtornos do Neurodesenvolvimento/genética
Polimorfismo de Nucleotídeo Único/genética
Esquizofrenia/genética
Reino Unido
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170926
[Lr] Data última revisão:
170926
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:151118
[St] Status:MEDLINE
[do] DOI:10.1038/mp.2015.163



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