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[PMID]:29215340
[Au] Autor:Yakut K; Erdogan I; Varan B; Atar I
[Ad] Endereço:Department of Pediatric Cardiology, Baskent University Ankara Hospital, Ankara, Turkey.
[Ti] Título:A Report of Brugada Syndrome Presenting with Cardiac Arrest Triggered by Verapamil Intoxication.
[So] Source:Balkan Med J;34(6):576-579, 2017 12 01.
[Is] ISSN:2146-3131
[Cp] País de publicação:Turkey
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Brugada syndrome is a disease characterized by a specific electrocardiographic pattern and an increased risk of sudden cardiac death. We present this case with the updated literature to emphasise the need to consider the diagnosis of Brugada syndrome in patients admitted to the emergency ward with sudden cardiac arrest. CASE REPORT: A 16-year-old female patient was admitted to the emergency ward with complaints of weakness and abdominal pain, and she had four cardiac arrests during her evaluation period. She was referred to our clinic for permanent pacemaker implantation. She was on a temporary pace maker after having had C-reactive protein. Her physical exam was normal except for bilaterally decreased lung sounds. Lung x-ray and computed tomography, which were performed by another institution, revealed minimal pleural effusion and nothing else of significance. Blood and peritoneal fluid samples were sterile. Echocardiographic exam and cardiac enzymes were also in the normal ranges. Electrocardiographic showed incomplete right branch block in leads V1 and V2. An ajmaline test revealed specific electrocardiographic findings of the type I Brugada pattern. We proposed implanting an implantable cardioverter defibrillator to the patient as there were positive findings on the ajmaline test as well as a history of sudden cardiac arrest. After this treatment proposal, the patient's family admitted that she had taken a high dose of verapamil and thus, the encountered bradycardia was associated with verapamil overuse. The ajmaline test was repeated as it was contemplated that the previous positive ajmaline test had been associated with verapamil overuse. Implantable cardioverter defibrillator implantation was proposed again as there was a history of sudden cardiac arrest; however, the family did not consent to implantable cardioverter defibrillator, and the patient was discharged and followed up. CONCLUSION: Brugada syndrome should be considered for patients who are admitted to the emergency ward with sudden cardiac arrest though surface electrocardiographic is normal. If there is a suspicion of Brugada syndrome, repeated electrocardiographic should be performed on different occasions. Diagnosis can be clarified by upper costal electrocardiographic or by administering Na channel blockers during electrocardiographic performance.
[Mh] Termos MeSH primário: Antiarrítmicos/envenenamento
Síndrome de Brugada/induzido quimicamente
Parada Cardíaca/induzido quimicamente
Verapamil/envenenamento
[Mh] Termos MeSH secundário: Adolescente
Ajmalina/farmacologia
Síndrome de Brugada/diagnóstico
Síndrome de Brugada/fisiopatologia
Diagnóstico Diferencial
Eletrocardiografia
Feminino
Testes Genéticos
Parada Cardíaca/fisiopatologia
Seres Humanos
Fatores Desencadeantes
Bloqueadores dos Canais de Sódio/administração & dosagem
Tentativa de Suicídio
Resultado do Tratamento
[Pt] Tipo de publicação:CASE REPORTS; JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Anti-Arrhythmia Agents); 0 (Sodium Channel Blockers); 1PON08459R (Ajmaline); CJ0O37KU29 (Verapamil)
[Em] Mês de entrada:1801
[Cu] Atualização por classe:180308
[Lr] Data última revisão:
180308
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171208
[St] Status:MEDLINE
[do] DOI:10.4274/balkanmedj.2016.1301


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[PMID]:29023487
[Au] Autor:Abhishek A; Valdes AM; Jenkins W; Zhang W; Doherty M
[Ad] Endereço:Academic Rheumatology, University of Nottingham, Clinical Sciences Building, Nottingham City Hospital, Nottingham, United Kingdom.
[Ti] Título:Triggers of acute attacks of gout, does age of gout onset matter? A primary care based cross-sectional study.
[So] Source:PLoS One;12(10):e0186096, 2017.
[Is] ISSN:1932-6203
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:OBJECTIVES: To determine the proportion of people with gout who self-report triggers of acute attacks; identify the commonly reported triggers, and examine the disease and demographic features associated with self-reporting any trigger(s) of acute attacks of gout. METHODS: Individuals with gout were asked to fill a questionnaire enquiring about triggers that precipitated their acute gout attacks. Binary logistic regression was used to compute odds ratio (OR) and 95% confidence intervals (CI) to examine the association between having ≥1 self-reported trigger of acute gout and disease and demographic risk factors and to adjust for covariates. All statistical analyses were performed using STATA. RESULTS: 550 participants returned completed questionnaires. 206 (37.5%) reported at least one trigger of acute attacks, and less than 5% reported >2 triggers. Only 28.73% participants reported that their most recent gout attack was triggered by dietary or lifestyle risk factors. The most frequently self-reported triggers were alcohol intake (14.18%), red-meat or sea-food consumption (6%), dehydration (4.91%), injury or excess activity (4.91%), and excessively warm or cold weather (4.36% and 5.45%). Patients who had onset of gout before the age of 50 years were significantly more likely to identify a trigger for precipitating their acute gout attacks (aOR (95%CI) 1.73 (1.12-2.68) after adjusting for covariates. CONCLUSION: Most people with gout do not identify any triggers for acute attacks, and identifiable triggers are more common in those with young onset gout. Less than 20% people self-reported acute gout attacks from conventionally accepted triggers of gout e.g. alcohol, red-meat intake, while c.5% reported novel triggers such as dehydration, injury or physical activity, and weather extremes.
[Mh] Termos MeSH primário: Gota/etiologia
[Mh] Termos MeSH secundário: Idoso
Consumo de Bebidas Alcoólicas/efeitos adversos
Estudos Transversais
Feminino
Alimentos/efeitos adversos
Seres Humanos
Masculino
Meia-Idade
Fatores Desencadeantes
Autorrelato
Inquéritos e Questionários
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171023
[Lr] Data última revisão:
171023
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:171013
[St] Status:MEDLINE
[do] DOI:10.1371/journal.pone.0186096


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[PMID]:28535245
[Au] Autor:VanderWeele TJ
[Ti] Título:Invited Commentary: The Continuing Need for the Sufficient Cause Model Today.
[So] Source:Am J Epidemiol;185(11):1041-1043, 2017 Jun 01.
[Is] ISSN:1476-6256
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:In this commentary, I review the insights that have been gained using Rothman's sufficient cause model (Am J Epidemiol. 1976;104(6):587-592). Discussion pertains to the relations of the model to similar conceptualizations in other fields of study, the advances and extensions that have been made to the model since the paper's publication, and its relation to questions of actual causation, along with questions concerning the use of the model in the future of epidemiology.
[Mh] Termos MeSH primário: Causalidade
Estudos Epidemiológicos
Modelos Teóricos
[Mh] Termos MeSH secundário: Seres Humanos
Fatores Desencadeantes
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170809
[Lr] Data última revisão:
170809
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170524
[St] Status:MEDLINE
[do] DOI:10.1093/aje/kwx083


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[PMID]:28388530
[Au] Autor:Böhmländer D; Dirndorfer T; Al-Bayatti AH; Brandmeier T
[Ad] Endereço:AUDI AG, 85045 Ingolstadt, Germany; De Montfort University, Leicester, United Kingdom. Electronic address: dennis.boehmlaender@audi.de.
[Ti] Título:Context-aware system for pre-triggering irreversible vehicle safety actuators.
[So] Source:Accid Anal Prev;103:72-84, 2017 Jun.
[Is] ISSN:1879-2057
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:New vehicle safety systems have led to a steady improvement of road safety and a reduction in the risk of suffering a major injury in vehicle accidents. A huge leap forward in the development of new vehicle safety systems are actuators that have to be activated irreversibly shortly before a collision in order to mitigate accident consequences. The triggering decision has to be based on measurements of exteroceptive sensors currently used in driver assistance systems. This paper focuses on developing a novel context-aware system designed to detect potential collisions and to trigger safety actuators even before an accident occurs. In this context, the analysis examines the information that can be collected from exteroceptive sensors (pre-crash data) to predict a certain collision and its severity to decide whether a triggering is entitled or not. A five-layer context-aware architecture is presented, that is able to collect contextual information about the vehicle environment and the actual driving state using different sensors, to perform reasoning about potential collisions, and to trigger safety functions upon that information. Accident analysis is used in a data model to represent uncertain knowledge and to perform reasoning. A simulation concept based on real accident data is introduced to evaluate the presented system concept.
[Mh] Termos MeSH primário: Acidentes de Trânsito/prevenção & controle
Condução de Veículo/estatística & dados numéricos
Equipamentos de Proteção
Segurança
[Mh] Termos MeSH secundário: Acidentes de Trânsito/estatística & dados numéricos
Condução de Veículo/psicologia
Seres Humanos
Modelos Teóricos
Fatores Desencadeantes
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171030
[Lr] Data última revisão:
171030
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170408
[St] Status:MEDLINE


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[PMID]:28377021
[Au] Autor:Tofler GH; Kopel E; Klempfner R; Eldar M; Buckley T; Goldenberg I; National Israel Survey of Acute Coronary Syndrome Investigators
[Ad] Endereço:Cardiology Department, Royal North Shore Hospital, Sydney, New South Wales, Australia; University of Sydney, Sydney, Australia. Electronic address: Geoffrey.Tofler@health.nsw.gov.au.
[Ti] Título:Triggers and Timing of Acute Coronary Syndromes.
[So] Source:Am J Cardiol;119(10):1560-1565, 2017 May 15.
[Is] ISSN:1879-1913
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Previous studies have shown that an acute coronary syndrome (ACS) may be triggered by external activities; however, their frequency, predictors, and significance are uncertain. We evaluated data from the National Israel Survey of Acute Coronary Syndromes, which was conducted in 2004 (February to March) in all 25 coronary care units and cardiac wards in Israel. Demographic and clinical data were recorded for consecutive participants, including potential triggers and time of symptom onset of ACS. Among the 1,849 patients who completed the trigger question, 1/4 (25.9%) reported a possible trigger, comprising heavy physical exertion (15.2%), emotional stress (8.3%), anger (1.1%), heavy meal (1.3%), and sexual activity (0.5%). Predictors of a triggered ACS were age <65 years, previous angina, no previous angiotensin-converting enzyme inhibitors/angiotensin 2 receptor blockers, impaired functional class, not having typical chest pain on admission, and a final diagnosis of unstable angina. The highest proportion of triggered ACS was between noon and 6 p.m. Physical exertion as a trigger was associated with reduced in-hospital mortality (0.4% vs 2.8%, p <0.05) and 1-year mortality. Emotional stress as a trigger did not influence in-hospital or 1-year mortality; however among those discharged from hospital, it was associated with increased 30-day rehospitalization (27.6% vs 19.3%, p <0.05) and a trend toward increased mortality (4.1% vs 2.0%, p = 0.10).
[Mh] Termos MeSH primário: Síndrome Coronariana Aguda/etiologia
Vigilância da População
Estresse Psicológico/complicações
[Mh] Termos MeSH secundário: Síndrome Coronariana Aguda/diagnóstico
Síndrome Coronariana Aguda/epidemiologia
Fatores Etários
Idoso
Ira
Progressão da Doença
Eletrocardiografia
Feminino
Seguimentos
Mortalidade Hospitalar/tendências
Seres Humanos
Incidência
Israel/epidemiologia
Masculino
Fatores Desencadeantes
Prognóstico
Estudos Prospectivos
Fatores de Risco
Fatores Sexuais
Estresse Psicológico/psicologia
Taxa de Sobrevida/tendências
Fatores de Tempo
[Pt] Tipo de publicação:JOURNAL ARTICLE; MULTICENTER STUDY
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170809
[Lr] Data última revisão:
170809
[Sb] Subgrupo de revista:AIM; IM
[Da] Data de entrada para processamento:170406
[St] Status:MEDLINE


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[PMID]:28298151
[Au] Autor:Taheri S
[Ad] Endereço:Children's Hospital of Western Ontario, Department of Academic Paediatrics, London, Canada.
[Ti] Título:Effect of exclusion of frequently consumed dietary triggers in a cohort of children with chronic primary headache.
[So] Source:Nutr Health;23(1):47-50, 2017 Mar.
[Is] ISSN:0260-1060
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:BACKGROUND: Although dietary factors are known to trigger headaches, the relationship between food and headache in children remains unclear. This prospective, observational case series aimed to evaluate the effect of exclusion of frequently-consumed foods in a cohort of children with headache. METHODS: One hundred and fifteen children aged 3-15 (mean 10.5) years with primary headache were followed in a paediatric outpatient clinic. Patients who frequently consumed foods or food additives known to trigger headaches were advised to exclude them for six weeks and to return for follow-up with headache and food diary. RESULTS: One hundred patients attended follow-up. Of these 13 (13%) did not respond to dietary exclusion; 87 (87%) achieved complete resolution of headaches by exclusion of 1-3 of the identified food(s). Caffeine was the most common implicated trigger (28), followed by monosodium glutamate (25), cocoa (22), aspartame (13), cheese (13), citrus (10) and nitrites (six). One patient was sensitive to tomatoes. CONCLUSIONS: This study demonstrates the potential scale and significance of seven frequently consumed foods or food additives as triggers for primary headache in children. Also this is the first study to show that headaches can be triggered by the cumulative effect of a food that is frequently consumed, rather than by single time ingestion.
[Mh] Termos MeSH primário: Dieta/efeitos adversos
Comportamento Alimentar
Cefaleia/dietoterapia
Transtornos de Enxaqueca/dietoterapia
[Mh] Termos MeSH secundário: Adolescente
Aspartame/administração & dosagem
Aspartame/efeitos adversos
Cacau/efeitos adversos
Cafeína/administração & dosagem
Cafeína/efeitos adversos
Queijo/efeitos adversos
Criança
Pré-Escolar
Chocolate/efeitos adversos
Citrus/efeitos adversos
Registros de Dieta
Feminino
Aditivos Alimentares/administração & dosagem
Aditivos Alimentares/efeitos adversos
Cefaleia/etiologia
Transtornos da Cefaleia/dietoterapia
Transtornos da Cefaleia/etiologia
Seres Humanos
Masculino
Transtornos de Enxaqueca/etiologia
Nitritos/administração & dosagem
Nitritos/efeitos adversos
Fatores Desencadeantes
Estudos Prospectivos
Glutamato de Sódio/administração & dosagem
Glutamato de Sódio/efeitos adversos
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Nm] Nome de substância:
0 (Food Additives); 0 (Nitrites); 3G6A5W338E (Caffeine); W81N5U6R6U (Sodium Glutamate); Z0H242BBR1 (Aspartame)
[Em] Mês de entrada:1708
[Cu] Atualização por classe:170807
[Lr] Data última revisão:
170807
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:170317
[St] Status:MEDLINE
[do] DOI:10.1177/0260106016688699


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[PMID]:27790819
[Au] Autor:Arrigo M; Gayat E; Parenica J; Ishihara S; Zhang J; Choi DJ; Park JJ; Alhabib KF; Sato N; Miro O; Maggioni AP; Zhang Y; Spinar J; Cohen-Solal A; Iwashyna TJ; Mebazaa A; GREAT Network
[Ad] Endereço:INSERM UMR-S 942, Université Paris Diderot, PRES Sorbonne Paris Cité, and Department of Anaesthesiology and Critical Care Medicine, APHP-Saint Louis Lariboisière University Hospitals, Paris, France.
[Ti] Título:Precipitating factors and 90-day outcome of acute heart failure: a report from the intercontinental GREAT registry.
[So] Source:Eur J Heart Fail;19(2):201-208, 2017 Feb.
[Is] ISSN:1879-0844
[Cp] País de publicação:England
[La] Idioma:eng
[Ab] Resumo:AIMS: Several clinical conditions may precipitate acute heart failure (AHF) and influence clinical outcome. In this study we hypothesized that precipitating factors are independently associated with 90-day risk of death in AHF. METHODS AND RESULTS: The study population consisted of 15 828 AHF patients from Europe and Asia. The primary outcome was 90-day all-cause mortality according to identified precipitating factors of AHF [acute coronary syndrome (ACS), infection, atrial fibrillation (AF), hypertension, and non-compliance]. Mortality at 90 days was 15.8%. AHF precipitated by ACS or by infection showed increased 90-day risk of death compared with AHF without identified precipitants [hazard ratio (HR) 1.69, 95% confidence interval (CI) 1.44-1.97, P < 0.001; and HR 1.51, 95% CI 1.18-1.92, P = 0.001), while AHF precipitated by AF showed lower 90-day risk of death (HR 0.56, 95% CI 0.42-0.75, P < 0.001), after multivariable adjustment. The risk of death in AHF precipitated by ACS was the highest during the first week after admission, while in AHF precipitated by infection the risk of death had a delayed peak at week 3. In AHF precipitated by AF, a trend toward reduced risk of death during the first weeks was shown. At weeks 5-6, AHF precipitated by ACS, infection, or AF showed similar risk of death to that of AHF without identified precipitants. CONCLUSIONS: Precipitating factors are independently associated with 90-day mortality in AHF. AHF precipitated by ACS or infection is independently associated with higher, while AHF precipitated by AF is associated with lower 90-day risk of death.
[Mh] Termos MeSH primário: Síndrome Coronariana Aguda/epidemiologia
Fibrilação Atrial/epidemiologia
Insuficiência Cardíaca/epidemiologia
Hipertensão/epidemiologia
Infecção/epidemiologia
Mortalidade
Cooperação do Paciente/estatística & dados numéricos
Sistema de Registros
[Mh] Termos MeSH secundário: Doença Aguda
Idoso
Idoso de 80 Anos ou mais
Ásia/epidemiologia
Causas de Morte
Europa (Continente)/epidemiologia
Feminino
Insuficiência Cardíaca/mortalidade
Seres Humanos
Masculino
Meia-Idade
Fatores Desencadeantes
Prognóstico
Modelos de Riscos Proporcionais
Estudos Prospectivos
[Pt] Tipo de publicação:JOURNAL ARTICLE; OBSERVATIONAL STUDY
[Em] Mês de entrada:1711
[Cu] Atualização por classe:171109
[Lr] Data última revisão:
171109
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161030
[St] Status:MEDLINE
[do] DOI:10.1002/ejhf.682


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[PMID]:27736659
[Au] Autor:Oguz-Akarsu E; Aydin-Özemir Z; Bebek N; Gürses C; Gökyigit A; Baykan B
[Ad] Endereço:Departments of Neurology and Clinical Neurophysiology, Istanbul University, Istanbul Faculty of Medicine, Istanbul, Turkey. Electronic address: emeloguz@yahoo.com.
[Ti] Título:Status epilepticus in patients with juvenile myoclonic epilepsy: Frequency, precipitating factors and outcome.
[So] Source:Epilepsy Behav;64(Pt A):127-132, 2016 Nov.
[Is] ISSN:1525-5069
[Cp] País de publicação:United States
[La] Idioma:eng
[Ab] Resumo:Status epilepticus (SE) is rarely described in patients with juvenile myoclonic epilepsy (JME), and little is known about its frequency, subtypes, and predictors and the prognosis of these patients. In this retrospective study, we aimed to analyze the incidence of SE in patients with JME and emphasize the risk factors and long-term outcome of SE in an epilepsy outpatient-based cohort. We included patients with JME with a history of predominant myoclonic seizures and at least one diagnostic EEG with normal background activity and bursts of typical ≥3-Hz generalized spike-polyspike and waves. We investigated the electroclinical features of patients with JME who had experienced SE and compared them with patients with JME without SE. Of the 133 patients with JME, only 5 patients were diagnosed with SE (3.8%) according to new criteria. The most common SE subtype was myoclonic SE (MSE, 4 patients), followed by generalized clonic-tonic-clonic SE (1 patient) and nonconvulsive SE (1 patient). One patient had both MSE and generalized clonic-tonic-clonic SE. In three out of five patients, recurrent episodes of SE were observed. Same seizure precipitants including sleep deprivation, inappropriate antiepileptic drug treatment, and noncompliance were identified in patients with JME with and without SE, not reaching a significant difference between the groups. Myoclonia limited to specific body parts (one arm, face, or head) were significantly more common in patients with JME with SE (p: 0.002). We did not find any significant correlation with drug-resistant course and SE. Status epilepticus is rarely observed in patients with JME, and MSE appears to be the most common subtype. Local myoclonia might predict SE in a subgroup of patients with JME. We may suggest that some patients with JME have a liability to SE, in addition to usual seizure precipitating factors of JME. It seems that SE per se does not affect the outcome of JME and the patients with SE did not have drug-resistant course in the final analysis.
[Mh] Termos MeSH primário: Epilepsia Mioclônica Juvenil/epidemiologia
Estado Epiléptico
[Mh] Termos MeSH secundário: Adulto
Feminino
Seres Humanos
Masculino
Fatores Desencadeantes
Estudos Retrospectivos
Estado Epiléptico/epidemiologia
Estado Epiléptico/etiologia
Estado Epiléptico/fisiopatologia
[Pt] Tipo de publicação:JOURNAL ARTICLE
[Em] Mês de entrada:1709
[Cu] Atualização por classe:170917
[Lr] Data última revisão:
170917
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161014
[St] Status:MEDLINE


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[PMID]:27721118
[Au] Autor:Madias JE
[Ad] Endereço:Icahn School of Medicine at Mount Sinai, New York, NY, and the Division of Cardiology, Elmhurst Hospital Center, Elmhurst, NY, United States. Electronic address: madiasj@nychhc.org.
[Ti] Título:Cardiac arrest-triggered takotsubo syndrome vs. takotsubo syndrome complicated by cardiac arrest.
[So] Source:Int J Cardiol;225:142-143, 2016 Dec 15.
[Is] ISSN:1874-1754
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Mh] Termos MeSH primário: Parada Cardíaca/complicações
Parada Cardíaca/diagnóstico
Cardiomiopatia de Takotsubo/complicações
Cardiomiopatia de Takotsubo/diagnóstico
[Mh] Termos MeSH secundário: Feminino
Parada Cardíaca/fisiopatologia
Seres Humanos
Fatores Desencadeantes
Cardiomiopatia de Takotsubo/fisiopatologia
[Pt] Tipo de publicação:COMPARATIVE STUDY; LETTER
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171025
[Lr] Data última revisão:
171025
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:161011
[St] Status:MEDLINE


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[PMID]:27497104
[Au] Autor:Madias JE
[Ad] Endereço:Icahn School of Medicine at Mount Sinai, New York, NY, United States; Division of Cardiology, Elmhurst Hospital Center, Elmhurst, NY 11373, United States. Electronic address: madiasj@nychhc.org.
[Ti] Título:What is/are the trigger(s) of takotsubo syndrome in cancer patients receiving chemotherapy?
[So] Source:Int J Cardiol;222:253, 2016 Nov 01.
[Is] ISSN:1874-1754
[Cp] País de publicação:Netherlands
[La] Idioma:eng
[Mh] Termos MeSH primário: Antineoplásicos/efeitos adversos
Cardiomiopatia de Takotsubo/sangue
Cardiomiopatia de Takotsubo/induzido quimicamente
[Mh] Termos MeSH secundário: Biomarcadores/sangue
Seres Humanos
Fatores Desencadeantes
Cardiomiopatia de Takotsubo/diagnóstico
[Pt] Tipo de publicação:LETTER
[Nm] Nome de substância:
0 (Antineoplastic Agents); 0 (Biomarkers)
[Em] Mês de entrada:1710
[Cu] Atualização por classe:171003
[Lr] Data última revisão:
171003
[Sb] Subgrupo de revista:IM
[Da] Data de entrada para processamento:160807
[St] Status:MEDLINE



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